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Books on the topic 'Oocystes'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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1

Clancy, Jennifer L. Recovery of Cryptosporidium oocysts from high-volume water samples. Denver, CO: Awwa Research Foundation, 2003.

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2

Norton, William D. Survey of open finished water reservoirs for Giardia cysts and Cryptosporidium oocysts. [Trenton, N.J.]: Division of Science and Research, NJ Dept. of Environmental Protection, 1997.

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3

Radziminski, Christopher Zygmunt. Inactivation of cryptosporidium parvum oocysts and bacillus subtilis spores by chlorine dioxide in laboratory reagent and natural waters. Ottawa: National Library of Canada, 2000.

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4

LeChevallier, Mark W. Short-term variability of Giardia cyst and Cryptosporidium oocyst concentrations in a surface water source used for potable water. Trenton, N.J: New Jersey Dept. of Environmental Protection, Division of Science and Research, 1998.

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5

Biswas, Kaushik. Synergistic Inactivation of Cryptosporidium Oocysts in Natural Waters. American Water Works Association, 2002.

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6

Dubey, J. P. Toxoplasmosis, sarcocystosis, isosporosis, and cyclosporosis. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0054.

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Toxoplasmosis is a protozoan disease caused by Toxoplasma gondii. It is widely prevalent in humans and animals throughout the world, especially in the western hemisphere. Virtually all warm-blooded animals can act as intermediate hosts but the life cycle is completed only in cats, the definitive host. Cats excrete the resistant stage of T. gondii (oocysts) in faeces, and oocysts can survive in the environment for months. Humans become infected congenitally, by ingesting undercooked infected meat, or by ingesting food and water contaminated with oocysts from cat faeces. It can cause mental retardation and loss of vision in congenitally infected children and deaths in immunosuppressed patients, especially those with AIDS. There is no vaccine to control toxoplasmosis in humans at the present time but one is available for reduction of fetal losses in sheep.

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7

Widmer, Giovanni. Molecular Mechanisms of Chemical Inactivation of Cryptosporidium Oocysts and Giardia Cysts. American Water Works Association, 2003.

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8

NJDEP survey of source water for Giardia cysts and Cryptosporidium oocysts. [Trenton, N.J.]: New Jersey Dept. of Environmental Protection, Division of Science and Research, 1995.

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9

Sequential Disinfection Design Criteria for Inactivation of Cryptosporidium Oocysts in Drinking Water. Amer Water Works Assn, 2001.

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10

Honorine, Ward, and AWWA Research Foundation, eds. Structural physiology of the cryptosporidium oocyst wall. Denver, CO: AWWA Research Foundation, 2004.

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11

Development of a test to assess Cryptosporidium parvum oocysts viability: Correlation with infectivity potential. Denver, CO: The Foundation and American Water Works Association, 1993.

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12

Dept.of Environment. Isolation and Identification of Giardia Cysts, Cryptosporidium Oocysts and Free Living Pathogenic Amoebae in Water, Etc. Stationery Office Books, 1990.

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13

United States. Environmental Protection Agency. WSTB., ed. ICR protozoan method for detecting Giardi cysts and Cryptosporidium oocysts in water by a fluorescent antibody procedure. Cincinnati, OH (26 Martin Luther King Dr., Cincinnati 45268): U.S. Environmental Protection Agency, OGWDW/TSD/WSTB, 1995.

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14

Standing Committee of Analysts., ed. Isolation and identification of giardia cysts, cryptosporidium oocysts and free living pathogenic amoebae in water etc 1989. London: HMSO, 1990.

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15

United States. Environmental Protection Agency. WSTB., ed. ICR protozoan method for detecting Giardi cysts and Cryptosporidium oocysts in water by a fluorescent antibody procedure. Cincinnati, OH (26 Martin Luther King Dr., Cincinnati 45268): U.S. Environmental Protection Agency, OGWDW/TSD/WSTB, 1995.

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16

United States. Environmental Protection Agency. WSTB, ed. ICR protozoan method for detecting Giardi cysts and Cryptosporidium oocysts in water by a fluorescent antibody procedure. Cincinnati, OH (26 Martin Luther King Dr., Cincinnati 45268): U.S. Environmental Protection Agency, OGWDW/TSD/WSTB, 1995.

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17

Ward, H., N. Bhat, and R. O'Connora. Structural Physiology of the Cryptosporidium Oocyst Wall (Awwa Research Foundation Reports). AwwaRF, 2005.

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18

Awwa Research Foundation (Corporate Author), Ontario Ministry of Environment and Energy (Corporate Author), and Syed A. Sattar (Editor), eds. Giardia Cyst and Cryptosporidium Oocyst in Watersheds and Factors Affecting Inactivation. Amer Water Works Assn, 1999.

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19

Giardia cyst and Cryptosporidium oocyst survival in watersheds and factors affecting inactivation. Denver, CO: AWWA Research Foundation and American Water Works Association, 1999.

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20

Chartered Institute of Water and Environmental Management., ed. CIWEM National Workshop on the Removal/ Inactivation of Cryptosporidium Oocysts During Drinking Water Treatment: Technical papers from the CIWEM Workshop. Terence Dalton for the Chartered Institute of Water & Environmental Management, 2001.

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21

R, Archer J., and Wisconsin. Dept. of Natural Resources., eds. Cryptosporidium spp. oocyst and Giardia spp. cyst occurrence, concentrations, and distribution in Wisconsin waters. Madison, WI: Wisconsin Dept. of Natural Resources, 1995.

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22

CIWEM national workshop on the removal/inactivation of cryptosporidium oocysts during drinking water treatment: Technical papers from the CIWEM workshop held 25 March 1999. [S.l.]: Terence Dalton Publishers, 1999.

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23

Jex, Aaron R., Rachel M. Chalmers, Huw V. Smith, Giovanni Widmer, Vincent McDonald, and Robin B. Gasser. Cryptosporidiosis. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0053.

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Cryptosporidium species represent a genus of parasitic protozoa (Apicomplexa) that are transmitted via the faecal-oral route and commonly infect the epithelial tissues of the gastric or intestinal (or sometimes the respiratory) tract of many vertebrates, including humans. Infection occurs following the ingestion of viable and resistant oocysts, through direct host-to-host contact or in contaminated food, drinking or recreational water. Infection can be transmitted via anthroponotic (human-to-human, human-to-animal) or zoonotic (animal-to-human or animal-to-animal) pathways, depending upon the species of Cryptosporidium. Although infection can be asymptomatic, common symptoms of disease (cryptosporidiosis) include diarrhoea, colic (abdominal pain), nausea or vomiting, dehydration and/or fever. In humans, cryptosporidial infection in immunocompetent patients is usually short-lived (days to weeks) and eliminated following the stimulation of an effective immune response. However, infection in immunodeficient individuals (e.g., those with HIV/AIDS) can be chronic and fatal (in the absence of immunotherapy), as there are few effective anti-cryptosporidial drugs and no vaccines available. The present chapter provides an account of the history, taxonomy and biology, genomics and genetics of Cryptosporidium, the epidemiology, pathogenesis, treatment and control of cryptosporidiosis and the advances in tools for the identification and characterisation of Cryptosporidium species and the diagnosis of cryptosporidiosis.

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24

J. Garcés-Gudiño, R. Merino-Guzmán*, and A.L. Cevallos-Gordón. Litter reuse reduces Eimeria spp oocyst counts and improves the performance in broiler chickens reared in a tropical zone in Ecuador. Verlag Eugen Ulmer, 2018. http://dx.doi.org/10.1399/eps.2018.220.

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