Relevant bibliographies by topics / Oral mucosa. Mouth Mucosa

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Oral mucosa. Mouth Mucosa'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Oral mucosa. Mouth Mucosa"

1

Lesch, C. A., C. A. Squier, A. Cruchley, D. M. Williams, and P. Speight. "The Permeability of Human Oral Mucosa and Skin to Water." Journal of Dental Research 68, no. 9 (September 1989): 1345–49. http://dx.doi.org/10.1177/00220345890680091101.

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Specimens from fbur regions of oral mucosa (palate, buccal mucosa, lateral border of the tongue, and the floor of the mouth) and of abdominal skin were taken from 58 individuals at autopsy, for determination of permeability constants (Kp) to tritium-labeled water. Comparisons between fresh specimens and those stored at -80°C revealed no significant effect on Kp as a result of freezing; similar results were found with use of specimens from corresponding regions of the pig. Values for Kp were significantly different for all of the tissue regions examined and ranged from 44 ± 4 × 10-7 cm/min for skin to 973 ± 33 x 10-7 cmlmin for the floor of the mouth, which was the most penneable region. Similar differences were evident among corresponding regions of porcine oral mucosa and skin. Moreover, the Kp values obtained for human tissues were not significantly different from those of the pig, except for the floor of the mouth, which was more permeable in human than in pig tissue. The results reveal interesting differences in the permeability of human oral mucosa that might be related to susceptibility to mucosal disease in those conditions where local extrinsic etiological agents are implicated.
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Primasari, Ameta, and Safriani Sitompul. "THE INCREASED NUMBER OF EPITHELIAL CELLS DETACHED FROM ORAL MUCOSA BECAUSE OF AMALGAM FILLINGS." Dentika Dental Journal 20, no. 2 (December 1, 2017): 62–65. http://dx.doi.org/10.32734/dentika.v20i2.995.

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Amalgam is a filler material containing toxic mercury. One of the main risks comes from the mercury vapor released in the mouth. The presence of amalgams in the oral cavity changes the metabolism of epithelial cells in the oral mucosa. Exfoliative cytology is a fast and easy method because the examination is only performed on the surface of the oral mucosa, and epithelial cells can be released without anesthesia. The research method was an analytical descriptive with a cross-sectional design. Epithelial cells from oral mucosa were collected from 32 samples using the cytobrush method. The epithelial cells were collected on the closest buccal mucosa of the amalgam restoration. The number of epithelial cells in the oral mucosa was seen through micrographs from a microscope with 100x magnification. The data were analyzed using the Mann Whitney U test to determine the number of epithelial cells released. The Mann Whitney U test results showed that there was a significant difference in the number of oral mucosal epithelial cells obtained ​​between subjects with amalgam and subjects without amalgam with p<0.05. In addition, there was a difference in the features of oral mucosal epithelial cells between subjects with amalgam and subjects without amalgam. In conclusion, the number of epithelial cells obtained from amalgam users was higher than the subjects without restorations. The forms of epithelial cells which experience desquamation in the subjects with amalgam were more irregular than those of subjects without restorations.
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Yadav, Nisha Rani, Meena Jain, Ankur Sharma, Roma Yadav, Meetika Pahuja, and Vishal Jain. "Distribution and prevalence of oral mucosal lesions in residents of old age homes in Delhi, India." Nepal Journal of Epidemiology 8, no. 2 (October 31, 2018): 727–34. http://dx.doi.org/10.3126/nje.v8i2.18708.

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Background: It has been seen that very less attention has been given to the oral health of the geriatric population residing in old age homes and as the oral mucosal lesions are a matter of concern for this growing population. Therefore, a study was done with the objective of finding the prevalence of oral mucosal lesions and the distribution of oral mucosal lesions among 65-74 year old residents of old age homes in Delhi, India. Materials and Methods: A cross sectional study was done on 65-74 year old age group elders of old age homes in Delhi. A total of 464 subjects participated in the study. Oral Health Assessment Form, WHO was used for assessing oral mucosa. Clinical examination was performed using two mouth mirrors under natural illumination in a systematic manner. Data was processed and analyzed using SPSS version 23. Results: Out of a total of 464 subjects, 291 (62.70%) were males and 173 (37.30%) were females. Oral mucosal lesions seen in the study subjects were malignant tumours, leukoplakia, lichen planus, ulcerations, ANUG, Abscess and candidiasis. Leukoplakia was seen in 70 subjects (15%) and was present on buccal mucosa in the majority. A malignant tumour was seen in 7 subjects (1.5%) and commonly seen area is floor of mouth. Conclusion: Prevalence of oral mucosal lesions among residents of old age homes shows the need for increased preventive and diagnostic measures for prevention and early identification of oro-mucosal lesions. Taking adequate care for oro-mucosal health of elderly people residing in old age homes is necessary.
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Compilato, Domenico, Giuseppina Campisi, Luca Pastore, and Antonio Carroccio. "The Production of the Oral Mucosa of Antiendomysial and Anti—Tissue-Transglutaminase Antibodies in Patients with Celiac Disease: A Review." Scientific World JOURNAL 10 (2010): 2385–94. http://dx.doi.org/10.1100/tsw.2010.228.

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Celiac disease (CD) is a lifelong, T cell—mediated enteropathy, triggered by the ingestion of gluten and related prolamins in genetically susceptible subjects, resulting in minor intestinal mucosal injury, including villous atrophy with crypt hyperplasia and intraepithelial lymphocytosis, and subsequent nutrient malabsorption. Although serological tests for antiendomysial (EMA) and anti—tissue transglutaminase (anti-tTG) autoantibodies are used to screen and follow up on patients with CD, diagnostic confirmation is still based on the histological examination of the small intestinal mucosa. Although the small intestinal mucosa is the main site of the gut involved in CD, other mucosal surfaces (such as gastric, rectal, ileal, and esophageal) belonging to the gastrointestinal tract and the gut-associated lymphoid tissue (GALT) can also be involved. A site that could be studied less invasively is the mouth, as it is the first part of the gastrointestinal system and a part of the GALT. Indeed, not only have various oral ailments been reported as possible atypical aspects of CD, but it has been also demonstrated that inflammatory changes occur after oral supramucosal application and a submucosal injection of gliadin into the oral mucosa of CD patients. However, to date, only two studies have assessed the capacity of the oral mucosa of untreated CD patients to EMA and anti-tTG antibodies. In this paper, we will review studies that evaluate the capacity of the oral mucosa to produce specific CD autoantibodies. Discrepancies in sensitivity from the two studies have revealed that biopsy is still not an adequate procedure for the routine diagnostic purposes of CD patients, and a more in-depth evaluation on a larger sample size with standardized collection and analysis methods is merited. However, the demonstration of immunological reactivity to the gluten ingestion of the oral mucosa of CD, in terms of IgA EMA and anti-tTG production, needs to be further evaluated in order to verify whether the oral mucosa is colonized by lymphocytes activated in the intestine or if gluten could stimulate naïve lymphocytes directly in the oral mucosa. This would have important implications for the pathogenesis, diagnosis, and treatment of CD.
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Vuckovic, Nada, Marija Bokor-Bratic, Dejan Vuckovic, and Ivana Picuric. "Presence of Candida albicans in potentially malignant oral mucosal lesions." Archive of Oncology 12, no. 1 (2004): 51–54. http://dx.doi.org/10.2298/aoo0401051v.

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Recently, an interest in the study of oral candidiasis has markedly increased mainly because of its association with viral infections due to human immunodeficiency, but also because of its relation with potentially malignant lesions of oral mucosa. These lesions belong to the wide group of leukoplakia. Leukoplakia is a clinical term used to describe a range of nonspecific white lesions, whose appearance does not generally correlate well with histopathologic changes; therefore, biopsy should be performed in all cases to determine which are precancerous or potentially malignant ones. In order to study the association of Candida albicans and the types of mucosal lesions, we took 30 consecutive biopsies of oral mucosa and the smears for microbiologic examination from the changed surface of mucosa and from the rest of oral cavity. The study group consisted of 30 patients, 21 women and 9 men, with the average age of 50.23 years (range, 25-77 years). In 6 cases Candida was diagnosed in mucosal biopsy. In the smear from the lesion, it was present in 3 cases, and 2 cases were found in the smear from an unchanged oral mucosa. In 9/30 cases (30%) Candida was positive regardless of the smear area or mode of diagnosis. The most common lesion is leukoplakia, diagnosed in 12/30 cases (40%), in 6 female and 6 male patients. The average age of those patients was 52.42 years. The lesions were located as follows: cheek mucosa - 5 cases; gingival mucosa - 2; lower lip - 2; floor of the mouth - 2; soft palate - 1; Candida was present in 3/12 cases. The lesion with the second highest incidence is lichen planus (9 cases), with positive Candida infection in 4/9 (44.44%). Epithelial dysplasia, although diagnosed in a very small number of cases (1/30 or 3.3%) with leukoplakia, was associated with a Candida infection. Generally, Candida is present in potentially malignant oral mucosal lesions (in 3/12 or 25% of leukoplakia cases, in 4/9 or 44.44% of lichen planus cases, and 1/1 squamous papilloma), with an increasing incidence in lesions with serious dysplastic epithelial changes.
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Fitriasari, Nuri, Eko Rotary Nurtito, Nanan Nur’aeny, and Indah Suasani Wahyuni. "THE IMPORTANT ROLE OF ORAL MEDICINE SPECIALIST IN MANAGEMENT OF STEVENS-JOHNSON SYNDROME PATIENT." Dentino : Jurnal Kedokteran Gigi 5, no. 2 (August 15, 2020): 165. http://dx.doi.org/10.20527/dentino.v5i2.8969.

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ABSTRACTIntroduction: Stevens-Johnson Syndrome (SJS) is an acute hypersensitivity reaction that manifests on the skin, oral mucosa, ocular, gastrointestinal, genital and anal area. It is also potentially life-threatening in concern of dehydration and infection. Oral mucosal lesions due to SJS resulted in a significant decrease of patient’s quality of life. When the oral mucosa involved, the intake of nutrients and fluids is disrupted contributing to electrolyte imbalance that aggravates dehydration. Moreover, oral mucosal lesions have become an entry point for infection. Purpose: This case report describes the important role of oral medicine specialists in the management of oral mucosal lesions in SJS patient. Review: A 26-year-old female patient was referred from the Department of Dermatology and Venereology with a diagnosis of SJS et causa suspected paracetamol and/or amoxycillin. The complaints comprised of pain on the lips and oral cavity, difficulty in mouth opening, and pain when swallowing. The management for oral lesions included: history taking, external and intra oral examinations, dexamethasone mouthwash, nystatin oral suspension, and sodium chloride (NaCl) 0.9% solution. The patient showed improvement in oral mucosal lesions within 3 weeks of treatment that was provided by oral medicine specialist and medical team collaboration. Conclusion: Based on this case report, the role of oral medicine specialist is very important as part of the management team for SJS patient. Oral medicine specialist can reduce morbidity that results from oral mucosal involvement. Collaboration with oral medicine specialist since the beginning of treatment is the key to success in SJS management. Keywords: Oral medicine specialist, Oral mucosal lesion, Stevens-Johnson Syndrome.
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Şenel, Sevda. "An Overview of Physical, Microbiological and Immune Barriers of Oral Mucosa." International Journal of Molecular Sciences 22, no. 15 (July 22, 2021): 7821. http://dx.doi.org/10.3390/ijms22157821.

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The oral mucosa, which is the lining tissue of the oral cavity, is a gateway to the body and it offers first-line protection against potential pathogens, exogenous chemicals, airborne allergens, etc. by means of its physical and microbiological-immune barrier functions. For this reason, oral mucosa is considered as a mirror to the health of the individual as well as a guard or early warning system. It is organized in two main components: a physical barrier, which consists of stratified epithelial cells and cell–cell junctions, and a microbiological-immune barrier that keeps the internal environment in a condition of homeostasis. Different factors, including microorganism, saliva, proteins and immune components, have been considered to play a critical role in disruption of oral epithelial barrier. Altered mucosal structure and barrier functions results in oral pathologies as well as systemic diseases. About 700 kinds of microorganisms exist in the human mouth, constituting the oral microbiota, which plays a significant role on the induction, training and function of the host immune system. The immune system maintains the symbiotic relationship of the host with this microbiota. Crosstalk between the oral microbiota and immune system includes various interactions in homeostasis and disease. In this review, after reviewing briefly the physical barriers of oral mucosa, the fundamentals of oral microbiome and oral mucosal immunity in regard to their barrier properties will be addressed. Furthermore, their importance in development of new diagnostic, prophylactic and therapeutic strategies for certain diseases as well as in the application for personalized medicine will be discussed.
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Andabak Rogulj, Ana, Ivana Škrinjar, Danica Vidović Juras, Vanja Vučićević Boras †, and Božana Lončar Brzak. "Burning mouth syndrome – a burning enigma." Medicina Fluminensis 57, no. 1 (March 1, 2021): 4–16. http://dx.doi.org/10.21860/medflum2021_365333.

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Burning Mouth Syndrome (BMS) is a chronic pain condition characterized by an intraoral burning sensation and an absence of oral mucosal lesions and disturbances in laboratory findings. Burning symptoms usually affect the anterior two-thirds of the tongue, its lateral borders, hard palate and labial mucosa, but other oral cavity sites may also be affected. Taste alterations and a decrease in the salivary flow rate frequently accompany the burning symptoms. This condition mostly affects peri- and postmenopausal women. To date, the etiology of BMS remains unclear. This unknown etiology means that no appropriate treatment is currently available. A large number of the treatments and medications have been tried for BMS, but treatment management remains unsatisfactory in some patients. The purpose of this article is to present current knowledge on the treatment of BMS.
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Javali, MA. "Burning Mouth Syndrome: An Enigmatic Disorder." Kathmandu University Medical Journal 11, no. 2 (May 3, 2015): 175–78. http://dx.doi.org/10.3126/kumj.v11i2.12498.

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Burning mouth syndrome (BMS) is a chronic oral pain or burning sensation affecting the oral mucosa, often unaccompanied by mucosal lesions or other evident clinical signs. It is observed principally in middle-aged patients and postmenopausal women and may be accompanied by xerostomia and altered taste. Burning mouth syndrome is characterized by an intense burning or stinging sensation, preferably on the tongue or in other areas of mouth. This disorder is one of the most common, encountered in the clinical practice. This condition is probably of multifactorial origin; however the exact underlying etiology remains uncertain. This article discusses several aspects of BMS, updates current knowledge about the etiopathogenesis and describes the clinical features as well as the diagnosis and management of BMS patients. DOI: http://dx.doi.org/10.3126/kumj.v11i2.12498 Kathmandu University Medical Journal Vol.11(2) 2013: 175-178
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Bretsztajn, S., T. Leturc, E. Euvrard, and A. G. Bodard. "Lymphomatoid papulosis localized to the oral mucosa: case report and literature review." Journal of Oral Medicine and Oral Surgery 25, no. 3 (2019): 30. http://dx.doi.org/10.1051/mbcb/2019021.

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Introduction: Lymphomatoid papulosis is a primary CD 30+ cutaneous lymphoproliferation. Observation: We report the case of a 39-year-old patient who presented with ulcers on the back of the tongue, gums, buccal mucosa, and soft palate, which evolved as spontaneously regressive flare-ups. The diagnosis of inflammatory bowel disease was initially proposed. Several years later, the patient presented an ulcer on the left middle finger. Histological examination confirmed the diagnosis of lymphomatoid papulosis. Discussion: This chronic dermatosis manifests a single rash or multiple papulonodular rashes, evolving as spontaneously regressive flare-ups. Mucosal involvement is rare, and no prognostic factor for this location has been highlighted to this date. Pathological examination is essential. Conclusion: The mucosal involvement of lymphomatoid papulosis is one of the diagnoses to be considered for recurrent mouth ulcers.
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Dissertations / Theses on the topic "Oral mucosa. Mouth Mucosa"

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Rodriguez, Francisco Jose. "Oral mucositis in children receiving bone marrow transplantation." Thesis, Birmingham, Ala. : University of Alabama at Birmingham, 2008. https://www.mhsl.uab.edu/dt/2008m/rodriguez.pdf.

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Wickström, Claes. "MUC5B from the oral cavity identification of 'insoluble' assemblies and putative regulatory proteolytic events /." [Malmö] : Faculty of Odontology, Malmö University, 2002. http://catalog.hathitrust.org/api/volumes/oclc/51310732.html.

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Hansson, Annette. "Modeling of multi-step oral carcinogenesis in vitro : assessment of growth, differentiation and apoptosis markers /." Stockholm, 2003. http://diss.kib.ki.se/2003/91-7349-445-3/.

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Vondracek, Martin. "Toxicity of smokeless tobacco in human oral epithelium with emphasis on carcinogen metabolism and regulation of programmed cell death /." Stockholm, 2002. http://diss.kib.ki.se/2002/91-7349-335-X/.

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Ng, William Man Fai. "The effect of volatile thiol compounds on permeability of oral mucosa." Thesis, University of British Columbia, 1986. http://hdl.handle.net/2429/26508.

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Cumulative clinical and experimental evidence indicates that volatile sulphur compounds (VSC) the principal components of oral malodour, may play an important role in the pathogenesis of periodontal disease. As their (H₂S and CH₃SH) concentrations in gingival sulci increase with the severity of periodontal involvement, the objective of this investigation is to ascertain if they exert an effect on the permeability of oral mucosa. Permeability determinations were performed on excised porcine sublingual mucosal specimens which consisted of non-keratinized epithelium, basal membrane and connective tissue layers mounted in a two compartment perfusion apparatus. Using radioactive and fluorescent-labelled penetrants, it was found that exposure of the epithelial surface to an atmosphere containing physiological concentrations of both thiols (15 ng H₂S or CH₃SH / ml of 95% air - 5% C0₂) increased the permeability of the mucosa to (³⁵S)-S0₄⁻², (³H)-prostaglandin E₂ (PGE₂) and fluorescein isothiocyanate labelled E. coli lipopolysaccharide (F-LPS). A three hour exposure of the mucosa to H₂S and CH₃SH resulted in a 75% and 103% increase respectively in permeability to (³⁵S)-labelled sulphate ion. Similarly, the mercaptan induced up to a 70% increase in permeability of the mucosa to (³H)-prostaglandin E₂. The magnitude of changes in the permeability were found to depend on duration of exposure to the thiols and to their concentration. Studies using (³⁵S)-H₂S suggest that the observed changes in the tissue permeability are related to the reaction of the thiols with tissue components. In addition, the (³⁵S)-H₂S is capable of perfusing through all three layers of the mucosa at 12.3 ng / cm². In contrast to H₂S , the CH₃SH effect was irreversible in control air / C0₂ environment. This infers that CH₃SH is potentially a more deleterious agent to the tissue barrier. However, its effect can also be reversed by treatment of tissues with 0.22% ZnCl₂ either prior to or after exposure to mercaptan. This suggests that Zn⁺² ion may be useful in preventing the potentially harmful effects of VSC. Fluorescent studies with F-LPS indicate that thiols can also potentiate the penetration of endotoxin. Whereas the fluorescence of the F-LPS in control systems was confined to the superficial epithelial layer in contact with the endotoxin, the CH₃SH- exposed mucosa exhibited fluorescence throughout the epithelial and connective tissue layers. Fluorescent staining of the mucosal specimens with fluorescein diacetate followed by counter staining with ethidium bromide provides evidence of membrane impairment to some cells by CH₃SH. Collectively these observations provide strong experimental evidence that the VSC, products of putrefaction produced in the gingival sulcus by oral microflora, may adversely affect the integrity of the crevicular barrier to deleterious agents and thus contribute to the etiology of periodontal disease.
Dentistry, Faculty of
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Nilsson, Jan Anders. "Aldehyde toxicity in human oral epithelium /." Stockholm, 2004. http://diss.kib.ki.se/2004/91-7140-141-5/.

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Nicander, Ingrid. "Electrical impedance related to experimentally induced changes of human skin and oral mucosa /." Stockholm, 1998. http://diss.kib.ki.se/search/diss.se.cfm?19980907nica.

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Mörée, Agnes. "Gold and mercury in the oral mucosa in patch-tested patients with oral lichen lesions." Thesis, Malmö högskola, Odontologiska fakulteten (OD), 2013. http://urn.kb.se/resolve?urn=urn:nbn:se:mau:diva-19701.

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Rantanen, Irma. "Betaine in oral hygiene with special attention to dry and sensitive mucosa." Turku : Turun Yliopisto, 2003. http://books.google.com/books?id=qcJpAAAAMAAJ.

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Matheus, Paula Daniele. "Cell proliferation rate in clinically healthy oral mucosa of crack cocaine users." reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2012. http://hdl.handle.net/10183/143365.

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Objetivo: Avaliar a taxa proliferativa de células esfoliadas da mucosa bucal clinicamente saudável de usuários de crack. Material e Métodos: esfregaços orais foram coletados de língua e assoalho bucal de 87 indivíduos, divididos em três grupos: os usuários de crack (CRCO), n = 26; fumantes / etilistas (SA), n = 26 e controles (C ), n = 35. Lâminas histológicas foram submetidas à técnica de impregnação pela prata para quantificação do número de AgNORs/núcleo. As imagens foram obtidas por um sistema de captura de imagem adaptado a um microscópio de luz em x1000 ampliação. A média AgNOR por núcleo (mAgnor) e a percentagem de células com mais de 1,2,3 e 4 AgNORs por núcleo (pAgNOR> 1,> 2> 3 um> 4) foram calculados. Resultados: As células esfoliadas de mucosa da língua SA (3,34 ± 0,51 AgNOR / núcleo) exibiram maior taxa de proliferação celular (p <0,05) quando comparado com C (2,81 ± 0,773 AgNORs / núcleo) e CRCO (2,87 ± 0,51 AgNORs / núcleo) . Um aumento (p <0,05) da mAgnor também foi observada nas células do assoalho bucal (3,55 ± 0,57) em comparação com SA C (3,18 ± 0,53) e CRCO (3,28 ± 0,39). Dados semelhantes foram encontrados usando pAgNOR>1,>2,>3 e > 4. Conclusão: usuários de crack não apresentaram alterações na taxa proliferativa celular da mucosa bucal. Diante dos dados apresentados, o consumo de cigarro, em combinação com o consumo de álcool, continua sendo o maior fator prejudicial à mucosa bucal.
Objective: The aim of this study was to evaluate cell proliferation rate of cells exfoliated from clinically healthy mucosa of crack cocaine users. Material and Methods: Oral smears were collected from tongue and floor of the mouth mucosa of 87 individuals divided into three groups: crack cocaine users (CrCo), n=26; smokers/alcohol drinkers (SA), n=26 and controls (C), n=35. Histological slides were silver-stained using AgNOR technique to evaluate cell proliferation rate. Images were obtained by an image capturing system adapted to a light microscope at x1000 magnification. Quantification considered 50 cells by smear in which the number of AgNOR dots was visually counted. Mean AgNOR numbers per nucleus (mAgNOR) and the percentage of cells with more than 1,2,3 and 4 AgNORs per nucleus (pAgNOR>1,>2>3 an>4) were calculated. Results: Cells exfoliated from tongue mucosa of SA (3.34±0.51 AgNOR/nucleus) exhibit higher cell proliferation rate (p<0.05) when compared to C (2.81±0.773 AgNORs/nucleus) and to CrCo (2.87±0.51 AgNORs/nucleus). An increase (p<0.05) in mAgNOR was also observed in floor of the mouth cells (3.55±0.57) in SA when compared to C (3.18±0.53) and CrCo (3.28±0.39). Similar findings were found using pAgNOR>1,>2,>3 e >4. Conclusion: Crack cocaine users did not present changes in cell proliferation rate of oral mucosa. Between the expositions studied here, cigarette smoking in combination with alcohol consumption remain as the most harmful factors to oral mucosa.
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Books on the topic "Oral mucosa. Mouth Mucosa"

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Squier, Christopher A. Human oral mucosa: Development, structure, and function. Chichester, West Sussex, UK: Wiley-Blackwell, 2011.

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Pindborg, J. J. Atlas of diseases of the oral mucosa. 4th ed. Copenhagen: Munksgaard, 1985.

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Pindborg, J. J. Atlas of diseases of the oral mucosa. 5th ed. Copenhagen: Munksgaard, 1992.

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Günther, Veltman, Loevy Hannelore Taschini, and Taschini Pierangelo, eds. Differential diagnosis of diseases of the oral mucosa. Chicago: Quintessence Pub. Co., 1989.

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Diseases of the oral mucosa: A color atlas. 2nd ed. Chicago: Quintessence Pub. Co., 1994.

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Dows, Symposium (2nd 1985 Iowa City Iowa). Oral mucosal diseases: Biology, etiology and therapy: proceedings of the second Dows Symposium. Denmark: Published for the Dows Institute for Dental Research by Laegeforeningens forlag, 1987.

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Scully, Crispian. Oral diseases: An illustrated guide to diagnosis and management of diseases of the oral mucosa, gingivae, teeth, salivary glands, bones and joints. 2nd ed. London: Martin Dunitz, 1996.

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Scully, Crispian. Oral diseases: An illustrated guide to diagnosis and management of diseases of the oral mucosa, gingivae, teeth, salivary glands, bones and joints. 2nd ed. London: Martin Dunitz, 1996.

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Mattsson, Torsten. Oral side effects of allogeneic bone marrow transplantation. [S.l: s.n.], 1991.

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Mattsson, Torsten. Oral side effects of allogeneic bone marrow transplantation. Stockholm: Departments of Oral Surgery, Clinical Immunology and Transplantation Surgery, Karolinska Institute, 1991.

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Book chapters on the topic "Oral mucosa. Mouth Mucosa"

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Lucas, R. B., and J. W. Eveson. "The Oral Mucosa." In Atlas of Oral Pathology, 47–65. Dordrecht: Springer Netherlands, 1985. http://dx.doi.org/10.1007/978-94-009-5580-6_5.

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Squier, Christopher, and Kim A. Brogden. "Oral Epithelium." In Human Oral Mucosa, 19–52. West Sussex, UK: John Wiley & Sons, Ltd., 2013. http://dx.doi.org/10.1002/9781118710470.ch3.

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Squier, Christopher, and Kim A. Brogden. "The Functions of Oral Mucosa." In Human Oral Mucosa, 1–7. West Sussex, UK: John Wiley & Sons, Ltd., 2013. http://dx.doi.org/10.1002/9781118710470.ch1.

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Squier, Christopher, and Kim A. Brogden. "The Organization of Oral Mucosa." In Human Oral Mucosa, 9–17. West Sussex, UK: John Wiley & Sons, Ltd., 2013. http://dx.doi.org/10.1002/9781118710470.ch2.

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Squier, Christopher, and Kim A. Brogden. "The Interface Between Epithelium and Connective Tissue." In Human Oral Mucosa, 53–58. West Sussex, UK: John Wiley & Sons, Ltd., 2013. http://dx.doi.org/10.1002/9781118710470.ch4.

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Squier, Christopher, and Kim A. Brogden. "Connective Tissue." In Human Oral Mucosa, 59–75. West Sussex, UK: John Wiley & Sons, Ltd., 2013. http://dx.doi.org/10.1002/9781118710470.ch5.

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Squier, Christopher, and Kim A. Brogden. "Regional Differences in the Oral Mucosa." In Human Oral Mucosa, 77–98. West Sussex, UK: John Wiley & Sons, Ltd., 2013. http://dx.doi.org/10.1002/9781118710470.ch6.

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Squier, Christopher, and Kim A. Brogden. "Development and Aging of the Oral Mucosa." In Human Oral Mucosa, 99–111. West Sussex, UK: John Wiley & Sons, Ltd., 2013. http://dx.doi.org/10.1002/9781118710470.ch7.

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Squier, Christopher, and Kim A. Brogden. "Barrier Functions of Oral Mucosa." In Human Oral Mucosa, 113–44. West Sussex, UK: John Wiley & Sons, Ltd., 2013. http://dx.doi.org/10.1002/9781118710470.ch8.

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Squier, Christopher, and Kim A. Brogden. "Homologies in Structure and Function Among Mucosae: Oral, Esophageal, and Vaginal Mucosa." In Human Oral Mucosa, 145–57. West Sussex, UK: John Wiley & Sons, Ltd., 2013. http://dx.doi.org/10.1002/9781118710470.ch9.

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Conference papers on the topic "Oral mucosa. Mouth Mucosa"

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Majumdar, S. K., A. Uppal, and P. K. Gupta. "Autofluorescence spectroscopy of oral mucosa." In BiOS '98 International Biomedical Optics Symposium, edited by Alexander V. Priezzhev, Toshimitsu Asakura, and J. D. Briers. SPIE, 1998. http://dx.doi.org/10.1117/12.311884.

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Roy, Krishnendu, Ian Bottrill, Duncan R. Ingrams, Michail M. Pankratov, Elie E. Rebeiz, Peak Woo, Sadru Kabani, et al. "Diagnostic fluorescence spectroscopy of oral mucosa." In Photonics West '95, edited by R. Rox Anderson, Graham M. Watson, Rudolf W. Steiner, Douglas E. Johnson, Stanley M. Shapshay, Michail M. Pankratov, George S. Abela, et al. SPIE, 1995. http://dx.doi.org/10.1117/12.209094.

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Rubins, Uldis, Zbignevs Marcinkevics, Robert Andrianirina Muckle, Ieva Henkuzena, Andris Roze, and Andris Grabovskis. "Remote photoplethysmography for assessment of oral mucosa." In Preclinical and Clinical Optical Diagnostics, edited by J. Quincy Brown and Ton G. van Leeuwen. SPIE, 2019. http://dx.doi.org/10.1117/12.2526979.

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Behl, Isha, Hitesh Mamgain, Atul Deshmukh, Lekha Kukreja, Arti R. Hole, and C. Murali Krishna. "Raman microspectroscopic study of oral buccal mucosa." In SPIE BiOS, edited by Robert R. Alfano and Stavros G. Demos. SPIE, 2014. http://dx.doi.org/10.1117/12.2033933.

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ZHUO, SHUANGMU, JIANXIN CHEN, XINGSHAN JIANG, ZUFANG HUANG, and SHUSEN XIE. "NONLINEAR OPTICAL MICROSCOPY OF MOUSE ORAL MUCOSA." In Proceedings of the 6th International Conference on Photonics and Imaging in Biology and Medicine (PIBM 2007). WORLD SCIENTIFIC, 2008. http://dx.doi.org/10.1142/9789812832344_0040.

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de Veld, Diana C. G., Max Witjes, Jan L. Roodenburg, Willem M. Star, and Hericus J. C. M. Sterenborg. "Optical detection of (pre-)malignant lesions of the oral mucosa: autofluorescence characteristics of healthy mucosa." In European Conference on Biomedical Optics, edited by Theodore G. Papazoglou and Georges A. Wagnieres. SPIE, 2001. http://dx.doi.org/10.1117/12.447135.

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Edward, Kert, Tuya Shilagard, Suimin Qiu, and Gracie Vargas. "Two-photon autofluorescence spectroscopy of oral mucosa tissue." In SPIE BiOS, edited by Ammasi Periasamy, Karsten König, and Peter T. C. So. SPIE, 2011. http://dx.doi.org/10.1117/12.875049.

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Selezneva, O. V. "Lesions of the oral mucosa in various diseases in children." In SCIENCE OF RUSSIA: TARGETS AND GOALS. LJournal, 2019. http://dx.doi.org/10.18411/sr-10-06-2019-34.

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Ourutina, M. N., Natalia D. Gladkova, Felix I. Feldchtein, Grigory V. Gelikonov, Valentin M. Gelikonov, Roman V. Kuranov, and Alexander M. Sergeev. "In-vivo optical coherent tomography of teeth and oral mucosa." In BiOS Europe '98, edited by Marco Dal Fante, Hans-Jochen Foth, Neville Krasner, Renato Marchesini, and Halina Podbielska. SPIE, 1999. http://dx.doi.org/10.1117/12.339175.

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Nuebler-Moritz, Michael, Norbert Gutknecht, Hermann F. Sailer, Peter Hering, and Wilhelm Prettl. "Ablation of oral mucosa by erbium:YAG and holmium:YAG laser radiation." In BiOS '97, Part of Photonics West, edited by Harvey A. Wigdor, John D. B. Featherstone, and Peter Rechmann. SPIE, 1997. http://dx.doi.org/10.1117/12.273587.

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Reports on the topic "Oral mucosa. Mouth Mucosa"

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Feinberg, Stephen E. Phase 2 Clinical Trial of Intraoral Grafting of Human Tissue-Engineered Oral Mucosa. Fort Belvoir, VA: Defense Technical Information Center, October 2013. http://dx.doi.org/10.21236/ada591622.

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