Relevant bibliographies by topics / Organe lymphoïde tertiaire

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Organe lymphoïde tertiaire'

Author: Grafiati
Published: 7 December 2024
Last updated: 31 July 2025

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Journal articles on the topic "Organe lymphoïde tertiaire"

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Shomer, Nirah H., James G. Fox, Amy E. Juedes, and Nancy H. Ruddle. "Helicobacter-Induced Chronic Active Lymphoid Aggregates Have Characteristics of Tertiary Lymphoid Tissue." Infection and Immunity 71, no. 6 (2003): 3572–77. http://dx.doi.org/10.1128/iai.71.6.3572-3577.2003.

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ABSTRACT Susceptible strains of mice that are naturally or experimentally infected with murine intestinal helicobacter species develop hepatic inflammatory lesions that have previously been described as chronic active hepatitis. The inflammatory infiltrates in some models of chronic autoimmunity or inflammation resemble tertiary lymphoid organs hypothesized to arise by a process termed lymphoid organ neogenesis. To determine whether hepatic inflammation caused by infection with helicobacter could give rise to tertiary lymphoid organs, we used fluorescence-activated cell sorting, immunohistoche
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Barone, Francesca, Saba Nayar, Joana Campos, et al. "IL-22 regulates lymphoid chemokine production and assembly of tertiary lymphoid organs." Proceedings of the National Academy of Sciences 112, no. 35 (2015): 11024–29. http://dx.doi.org/10.1073/pnas.1503315112.

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The series of events leading to tertiary lymphoid organ (TLO) formation in mucosal organs following tissue damage remain unclear. Using a virus-induced model of autoantibody formation in the salivary glands of adult mice, we demonstrate that IL-22 provides a mechanistic link between mucosal infection, B-cell recruitment, and humoral autoimmunity. IL-22 receptor engagement is necessary and sufficient to promote differential expression of chemokine (C-X-C motif) ligand 12 and chemokine (C-X-C motif) ligand 13 in epithelial and fibroblastic stromal cells that, in turn, is pivotal for B-cell recru
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Erlich, Emma, Rafael Czepielewski, Shashi Kumar, et al. "B cells drive tertiary lymphoid organ formation in ileal inflammation." Journal of Immunology 208, no. 1_Supplement (2022): 113.18. http://dx.doi.org/10.4049/jimmunol.208.supp.113.18.

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Abstract Crohn’s disease [CD] is one of the two most common forms of inflammatory bowel disease, affecting over half a million Americans. Like many other diseases with chronic inflammation, some patients with CD develop tertiary lymphoid organs [TLO] in areas of the gastrointestinal tract with active disease. TLOs are organized clusters of lymphocytes, similar in structure to secondary lymphoid organs, though they develop after birth and their contribution to pathogenesis in CD, or other diseases, is unclear. We, and others, have also found B cell rich lymphoid aggregates in the mesenteric fat
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Ruddle, Nancy H. "Lymphatic vessels and tertiary lymphoid organs." Journal of Clinical Investigation 124, no. 3 (2014): 953–59. http://dx.doi.org/10.1172/jci71611.

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Feizi, Neda, Neda Feizi, Gang Zhang, Latha Halesha, Khodor Abou Daya, and Martin H. Oberbarnscheidt. "Tertiary Lymphoid Organs promote allograft rejection." Journal of Immunology 212, no. 1_Supplement (2024): 0321_5062. http://dx.doi.org/10.4049/jimmunol.212.supp.0321.5062.

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Abstract Tertiary lymphoid organs (TLOs) are ectopic lymphoid structures that arise in non-lymphoid tissues and are frequently observed in tissue affected by non-resolving chronic inflammation. If TLOs are beneficial or detrimental in transplantation is controversial. We investigate the role of TLO and LTbR in transplantation by manipulating the LTa-LTbR pathway. Using a mouse allo kidney transplantation model, we found that preformed intragraft TLO are sufficient to precipitate rejection in recipients lacking all secondary lymphoid organs (SLO)(LTbRko). In WT recipients with a complete set of
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Kirsh, Andrew L., Sharon L. Cushing, Eunice Y. Chen, Stephen M. Schwartz, and Jonathan A. Perkins. "Tertiary Lymphoid Organs in Lymphatic Malformations." Lymphatic Research and Biology 9, no. 2 (2011): 85–92. http://dx.doi.org/10.1089/lrb.2010.0018.

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Goyal, Girija, Lucas Barck, Yunhao Zhai, et al. "Human implantable tertiary lymphoid organs (TLO) for solid tumor therapy: from organ chips to the clinic?" Journal of Immunology 212, no. 1_Supplement (2024): 0731_7312. http://dx.doi.org/10.4049/jimmunol.212.supp.0731.7312.

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Abstract We previously created an in vitro human TLO model from patient-derived, circulating immune cells using organ-on-a-chip devices and 3D culture in extracellular matrix (ECM). B cells in these TLOs express activation induced cytidine deaminase, which is only expressed in lymphoid tissues and is required for class switching and somatic hypermutation (SHM). When challenged by vaccines, these TLO undergo SHM and produce antigen-specific antibodies and CD8 T cells. To model cancer associated TLOs, we integrated human pancreatic and lung cancer cell lines into these TLO Chips to understand ho
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Lau, Aden, Susan Lester, Sophia Moraitis, et al. "Tertiary lymphoid organs in recalcitrant chronic rhinosinusitis." Journal of Allergy and Clinical Immunology 139, no. 4 (2017): 1371–73. http://dx.doi.org/10.1016/j.jaci.2016.08.052.

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Neyt, Katrijn, Frédéric Perros, Corine H. GeurtsvanKessel, Hamida Hammad, and Bart N. Lambrecht. "Tertiary lymphoid organs in infection and autoimmunity." Trends in Immunology 33, no. 6 (2012): 297–305. http://dx.doi.org/10.1016/j.it.2012.04.006.

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Shipman, William D., Dragos C. Dasoveanu, and Theresa T. Lu. "Tertiary lymphoid organs in systemic autoimmune diseases: pathogenic or protective?" F1000Research 6 (February 28, 2017): 196. http://dx.doi.org/10.12688/f1000research.10595.1.

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Tertiary lymphoid organs are found at sites of chronic inflammation in autoimmune diseases such as systemic lupus erythematosus and rheumatoid arthritis. These organized accumulations of T and B cells resemble secondary lymphoid organs and generate autoreactive effector cells. However, whether they contribute to disease pathogenesis or have protective functions is unclear. Here, we discuss how tertiary lymphoid organs can generate potentially pathogenic cells but may also limit the extent of the response and damage in autoimmune disease.
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Dissertations / Theses on the topic "Organe lymphoïde tertiaire"

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Veber, Romain. "Néogenèse lymphoïde au cours du lupus : mécanismes fondamentaux et pistes thérapeutiques." Thesis, Strasbourg, 2018. http://www.theses.fr/2018STRAJ092.

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Le lupus érythémateux disséminé est une maladie auto-immune systémique chronique dont les atteintes sont multiples, l’atteinte rénale constituant la plus sévère. Une conjonction de facteurs génétiques et environnementaux conduit au développement de la maladie qui se traduit par une rupture de tolérance au soi. L’un des signes biologiques majeurs est la production d’auto-anticorps dirigés contre des composants nucléaires qui, en se déposant dans divers tissus dont les reins, génèrent une inflammation chronique conduisant au dysfonctionnement de l’organe. Le dépôt d’autoanticorps s’accompagne d’
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Fouet, Morgane. "Modulation du microenvironnement tumoral et de la réponse immunitaire par des virus oncolytiques modifiés." Electronic Thesis or Diss., Nantes Université, 2024. http://www.theses.fr/2024NANU1008.

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Les virus présentant un tropisme pour les cellules tumorales et une capacité à induire leur lyse, sont appelé des virus oncolytiques. Ils sont utilisés dans le cadre de la virothérapie et permettent de stimuler la réponse immunitaire antitumorale par le recrutement et l’activation de cellules immunitaires innées et adaptatives. Les travaux issus de ma thèse ont permis d'approfondir la compréhension du rôle des lymphocytes Tϒ9δ2 dans le cadre des infections oncolytiques, en mettant notamment en lumière leur activation spécifique par la souche Schwarz du virus de la rougeole. J’ai ensuite dévelo
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Motallebzadeh, Reza. "Tertiary lymphoid organogenesis in solid organ transplantation." Thesis, University of Cambridge, 2013. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.608121.

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Nayar, Saba. "Lymphoid like stromal cells in a model of tertiary lymphoid organ formation." Thesis, University of Birmingham, 2014. http://etheses.bham.ac.uk//id/eprint/5245/.

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Tertiary lymphoid organs (TLOs) are a hallmark of many chronic immune-mediated inflammatory diseases. However, till date the series of events leading to stromal cell activation in TLOs and their role in the inflammatory process remain unclear. Using a model of inducible TLO formation in the salivary glands of mice we explored the role of gp38+LTβR+ lymphoid-like stromal cells (LLSc) during TLO development and show that they acquire the capability to produce lymphoid chemokines (CKs)/cytokine and drive lymphocyte compartmentalization. In this thesis, we provide evidence that stromal cell activa
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Le, Coz Carole. "Quelle contribution du centre germinatif et de ses composants moléculaires et cellulaires dans la physiopathologie du lupus ?" Thesis, Strasbourg, 2014. http://www.theses.fr/2014STRAJ077.

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Le lupus érythémateux disséminé est une maladie auto-immune systémique très invalidante dont les atteintes sont multiples, les plus fréquentes étant cutanées, articulaires et rénales. Dans ce type de maladie, le système immunitaire, hyperactif, ne se limite pas à lutter contre des agents extérieurs mais s'attaque à ses propres cellules, entre autres par le biais d'auto-anticorps. Ces anticorps délétères sont produits par des plasmocytes, cellules issus de la différenciation des lymphocytes B. Ce processus se déroule principalement au sein des centres germinatifs (GC) dans les organes lymphoïde
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Browne, Eleanor. "Tertiary lymphoid organ neogenesis in grey matter pathology in multiple sclerosis." Thesis, Imperial College London, 2015. http://hdl.handle.net/10044/1/30813.

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Chronic meningeal inflammation is suggested to contribute to the progression of secondary progressive multiple sclerosis (SPMS) in part by driving cortical grey matter pathology. The presence of tertiary lymphoid organ-like (TLO) structures in a large proportion of SPMS cases is associated with faster clinical progression and more severe cortical pathology, suggesting that TLO neogenesis and chronic meningeal inflammation contribute to progression. Gene expression of the cytokine lymphotoxin-alpha (LTα), implicated in TLO formation and cytotoxicity, and the lymphoid chemokines CXCL13 and CCL21
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Dias, De Campos Joana. "The pleomorphic role of stromal cells in the formation and maintenance of tertiary lymphoid organs." Thesis, University of Birmingham, 2016. http://etheses.bham.ac.uk//id/eprint/6994/.

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A large body of evidence supports the role of activated stromal cells in the persistence of inflammation. The switch from resting to pathogenic stroma appears to be associated with the development of tertiary lymphoid organs (TLOs) within sites of chronic inflammation. However little is known about the immunological function of the stromal component. We utilised a murine model of inducible TLO formation in inflamed salivary glands to investigate the role of activated stromal cells characterised by the expression of gp38 and FAP during TLO development. We demonstrated that during inflammation,
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Winter, Susann. "Funktionale Bedeutung der homöostatischen Chemokinrezeptoren CCR7 und CXCR5 im Verlauf von mukosalen Immunantworten." Doctoral thesis, Humboldt-Universität zu Berlin, Mathematisch-Naturwissenschaftliche Fakultät I, 2011. http://dx.doi.org/10.18452/16328.

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Die kontinuierliche Rezirkulation von Immunzellen durch periphere und sekundäre lymphatische Organe (SLOs) ist Bestandteil der Immunüberwachung und wichtig für die Aufrechterhaltung und Funktionsbereitschaft des Immunsystems. Der homöostatische Chemokinrezeptor CCR7 vermittelt dabei nicht nur die Rezirkulation von Lymphozyten durch SLOs, sondern scheint auch an der homöostatischen Rezirkulation von Lymphozyten durch nicht-lymphoide periphere Gewebe beteiligt zu sein. Im Rahmen dieser Arbeit wurde mithilfe von CCR7-defizienten Mäusen die funktionale Bedeutung von CCR7 für die homöostatische Rez
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Hörnblad, Andreas. "Imaging the pancreas : new aspects on lobular development and adult constitution." Doctoral thesis, Umeå universitet, Umeå centrum för molekylär medicin (UCMM), 2011. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-50601.

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The mouse pancreas is a mixed exocrine and endocrine glandconsisting of three lobular compartments: the splenic, duodenal and gastric lobes. During embryogenesis, the pancreas forms from two progenitor populations located on the dorsal and ventral side of the primitive gut tube. These anlagen are brought in close proximity as the gut elongates and rotates, and fuse to form a single organ. The splenic and duodenal lobes develop from the dorsal and ventral anlagen, respectively. In the adult pancreas, exocrine tissue secretes digestive enzymes intothe gut lumen to support nutrient uptake. The en
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Books on the topic "Organe lymphoïde tertiaire"

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Yin, Changjun, Andreas J. R. Habenicht, Sarajo Mohanta, and Pasquale Maffia, eds. Tertiary Lymphoid Organs (TLOs): Powerhouses of Disease Immunity. Frontiers Media SA, 2017. http://dx.doi.org/10.3389/978-2-88945-180-7.

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Book chapters on the topic "Organe lymphoïde tertiaire"

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Kobayashi, Yuka, Koichi Kato, Makoto Nakamura, and Takeshi Watanabe. "Synthesis of Functional Tertiary Lymphoid Organs." In Synthetic Immunology. Springer Japan, 2016. http://dx.doi.org/10.1007/978-4-431-56027-2_7.

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Rivellese, Felice, Elena Pontarini, and Costantino Pitzalis. "Tertiary Lymphoid Organs in Rheumatoid Arthritis." In Current Topics in Microbiology and Immunology. Springer International Publishing, 2020. http://dx.doi.org/10.1007/82_2020_216.

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Conference papers on the topic "Organe lymphoïde tertiaire"

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Touzani, Fahd, and Agnieszka Pozdzik. "O21 Analysis of B-cell infiltrates and tertiary lymphoid organ in lupus nephritis." In 12th European Lupus Meeting. Lupus Foundation of America, 2020. http://dx.doi.org/10.1136/lupus-2020-eurolupus.32.

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Conlon, T. M., G. John-Schuster, M. Lehmann, et al. "Therapeutic Targeting of B Cell Mediated Tertiary Lymphoid Organs (TLO) Reverts Chronic Obstructive Pulmonary Disease." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a7674.

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Lehmann, J., M. Thelen, S. Schran, et al. "P02.03 Organization, function and gene expression of tertiary lymphoid structures in pancreatic cancer resembles lymphoid follicles in secondary lymphoid organs and their abundance is related to superior survival." In iTOC9 – 9th Immunotherapy of Cancer Conference, September 22–24, 2022 – Munich, Germany. BMJ Publishing Group Ltd, 2022. http://dx.doi.org/10.1136/jitc-2022-itoc9.22.

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Wennhold, K., J. Lehmann, M. Thelen, et al. "03.03 Tertiary lymphoid structures in pancreatic cancer resemble lymphoid follicles in secondary lymphoid organs as sites for initiation and maintenance of anti-tumor T- and B-cell responses." In iTOC10 - 10th Immunotherapy of Cancer Conference, March 21 – 23, 2024 – Munich, Germany. BMJ Publishing Group Ltd, 2024. http://dx.doi.org/10.1136/jitc-2024-itoc10.1.

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Liu, Xinning, Yun Qiu, Ren Mao, and Ziyin Ye. "IDDF2022-ABS-0268 Subserosal tertiary lymphoid organs is a risk factor for postoperative recurrence in Crohn’s disease." In Abstracts of the International Digestive Disease Forum (IDDF), Hong Kong, 2–4 September 2022. BMJ Publishing Group Ltd and British Society of Gastroenterology, 2022. http://dx.doi.org/10.1136/gutjnl-2022-iddf.248.

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Hiraoka, Nobuyoshi. "Abstract B21: Tertiary lymphoid organs within pancreatic ductal carcinoma tissue are a favorable prognosticator, being strongly associated with blood vessels not infiltrated by cancer cells." In Abstracts: AACR Special Conference: Tumor Angiogenesis and Vascular Normalization: Bench to Bedside to Biomarkers; March 5-8, 2015; Orlando, FL. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-8514.tumang15-b21.

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