Relevant bibliographies by topics / Organophosphate induced delayed neuropathy

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  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters

Academic literature on the topic 'Organophosphate induced delayed neuropathy'

Author: Grafiati
Published: 4 June 2025
Last updated: 1 August 2025

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Journal articles on the topic "Organophosphate induced delayed neuropathy"

1

Eyer, P. "Review : Neuropsychopathological changes by organophosphorus compounds — a review." Human & Experimental Toxicology 14, no. 11 (1995): 857–64. http://dx.doi.org/10.1177/096032719501401101.

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1 Available literature dealing with neuropsychopathologi cal changes after exposure to organophosphate insecti cides is reviewed. 2 Subacute neurological sequelae following acute organophosphate intoxication include the 'intermediate syndrome', probably a myopathy elicited by excess acetylcholine, and the 'organophosphate-induced delayed neuropathy' (OPIDN), which is caused by particularly neurotoxic organophosphates that inhibit neuropathy target esterase. 3 Long-term toxic effects affecting behaviour as well as mental and visual functions are occasionally observed after exposure to high dose
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Vasconcellos, Luiz Felipe R., Ana Cláudia Leite, and Osvaldo J. M. Nascimento. "Organophosphate-induced delayed neuropathy: case report." Arquivos de Neuro-Psiquiatria 60, no. 4 (2002): 1003–7. http://dx.doi.org/10.1590/s0004-282x2002000600022.

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Organophosphate induced delayed neuropathy (OPIDN) is an uncommon clinical condition. It occurs in association with the ingestion of great amounts of organophosphate after the stimulation of cholinergic receptor. The clinical picture is characterized by a distal paresis in lower limbs associated with sensitive symptoms. Electrodiagnostic studies show a motor axonal neuropathy. Involvement of the central nervous system may occur. We describe a 39 years-old female patient who developed hyperesthesia associated with lower limbs paresis, fourteen days after she had ingested a Dichlorvos-based inse
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Basak, PM, MM Begum, MJ Islam, FA Khan, HS Das, and KI Jahan. "Organophosphate Induced Delayed Neuropathy (OPIDN)." TAJ: Journal of Teachers Association 27, no. 1 (2018): 62–63. http://dx.doi.org/10.3329/taj.v27i1.37617.

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Shukla, Abhiranjan, Dharampal Anand, and Ravindra Urkude. "Organophosphate induced delayed pure motor neuropathy." Journal of Neurology, Neurosurgery & Psychiatry 88, no. 5 (2017): e1.43-e1. http://dx.doi.org/10.1136/jnnp-2017-316074.45.

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Prasai, Pratik, Jitendra Thakur, and Karuna Tamrakar Karki. "Organophosphorus Poisoning Induced Delayed Poly-axonal Motor Neuropathy: A Case Report." Eastern Green Neurosurgery 4, no. 1 (2022): 27–29. http://dx.doi.org/10.3126/egn.v4i1.51012.

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Organophosphate being the lethal compound and easily available insecticide in the developing countries has significant potential health risk. The morbidity related to organophosphate poisoning as delayed peripheral neuropathy, extra pyramidal syndromes and neuropsychiatric manifestations are still the major consequences of secondary neuronal damage. Herein, we present a case of 23 year old lady who ingested organophosphate in the context of suicidal attempt and presented with motor neuropathy after 4 weeks.
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International, Journal of Medical Science and Advanced Clinical Research (IJMACR). "Organophosphorus (Chlorpyrifos) Toxicity Causing Delayed Induced Neuropathy and Myelopathy - Rare Case Report." International Journal of Medical Science and Advanced Clinical Research (IJMACR) 8, no. 2 (2025): 42–47. https://doi.org/10.5281/zenodo.15239883.

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<strong>Abstract</strong> Organophosphate (OP) poisoning is the most common poisoning in India, accounting for almost half of the hospital admissions due to poisoning. Delayed neuropathy is initiated by an attack on a nervous tissue esterase. Although uncommon, delayed neurotoxicity has been consistently reported in literature. This mechanism is implicated not only in damaging peripheral nervous system but also in causing central processes leading to myelopathy. We report a case report of 35year old male who came to our hospital with delayed neurological manifestations of organophosphorus pois
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Akçay Yalbuzdag, Şeniz. "Organophosphate induced delayed neuropathy: a case report." Türkiye Fiziksel Tıp ve Rehabilitasyon Dergisi 63, no. 1 (2017): 88–91. http://dx.doi.org/10.5606/tftrd.2017.13549.

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Gao, Zhaobing, Qiang Ding, Xin Xie, Bernard Attali, and Jian Li. "TRPA1 channel mediates organophosphate-induced delayed neuropathy." Proceedings for Annual Meeting of The Japanese Pharmacological Society WCP2018 (2018): PO1–1–124. http://dx.doi.org/10.1254/jpssuppl.wcp2018.0_po1-1-124.

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GLYNN, P. "A mechanism for organophosphate-induced delayed neuropathy." Toxicology Letters 162, no. 1 (2006): 94–97. http://dx.doi.org/10.1016/j.toxlet.2005.10.012.

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Qiang, D., X. Xie, and Z. Gao. "New insights into the organophosphate-induced delayed neuropathy." Journal of the Neurological Sciences 381 (October 2017): 153–54. http://dx.doi.org/10.1016/j.jns.2017.08.451.

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Dissertations / Theses on the topic "Organophosphate induced delayed neuropathy"

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Massicotte, Christiane II. "Neuropathologic Effects of Phenylmethylsulfonyl Fluoride (PMSF)-Induced Promotion and Protection inn Organophosphorus Ester-Induced Delayed Neuropathy(Opidn) in Hens." Thesis, Virginia Tech, 1998. http://hdl.handle.net/10919/10100.

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The serine/cysteine protease inhibitor phenylmethylsulfonyl fluoride (PMSF) has been used both to promote and to protect against neuropathic events of organophosphorus-induced delayed neuropathy (OPIDN) in hens (Lotti et al., 1991; Veronesi et al., 1985; Pope and Padilla, 1990; Pope et al., 1993). This study expands upon this work by correlating clinical and neuropathological findings in these modifications of OPIDN. To provide appropriate models of OPIDN, single phenyl saligenin phosphate (PSP) dosages of 0.5, 1.0, or 2.5 mg/kg were administered to adult hens. PMSF (90 mg/kg) was given either
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McCain, Wilfred C. "Effects of organophosphate esters on blood vessels: a physiological, pharmacological, and histological assessment of involvement in organophosphorus-induced delayed neuropathy (OPIDN)." Diss., Virginia Tech, 1994. http://hdl.handle.net/10919/39396.

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McCain, Wilfred C. "Cardiovascular components of organophosphorus-induced delayed neuropathy." Thesis, Virginia Tech, 1991. http://hdl.handle.net/10919/41691.

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The focus of this study was to assess the cardiovascular effects in hens of a single 2.5 mg/kg intramuscular injection of phenyl saligenin phosphate (PSP) into the breast muscle. Parameters were measured at 1, 3, 7, and 20 days post treatment. All hens developed clinical signs of delayed neuropathy by day 10 and these signs were maximal by day 20. Alterations of measured parameters were observed prior to the onset of clinical signs of organophosphorus-induced delayed neuropathy (OPIDN) (days 1, 3, and 7) as well as when maximal clinical signs were evident (days 15-21). Significant decreases in
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Carboni, Deborah Ann. "Comparative evolution of mipafox-induced delayed neuropathy in the rat and hen." Thesis, This resource online, 1993. http://scholar.lib.vt.edu/theses/available/etd-12052009-020159/.

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El-Fawal, Hassan Ahmed Naguib. "Involvement of calcium in organophosphorus-induced delayed neuropathy: a functional morphological, and biochemical study." Diss., Virginia Polytechnic Institute and State University, 1989. http://hdl.handle.net/10919/54520.

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Organophosphorus compounds are widely used in agriculture as pesticides and in industry as petroleum additives and modifiers of plastics. Some of these compounds are capable of inducing an irreversible neuropathy developing weeks to months after exposure, yet there is no effective treatment. This may be due in part to the lack of knowledge of how this neuropathy develops. In this dissertation, it is proposed that as a consequence of a triggering event, peripheral nerves may be predisposed to an increase in calcium (Ca⁺⁺) mobilization and the neuronal accumulation of this cation. This increase
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Book chapters on the topic "Organophosphate induced delayed neuropathy"

1

Ehrich, Marion, and Bernard S. Jortner. "Organophosphate-Induced Delayed Neuropathy." In Handbook of Neurotoxicology. Humana Press, 2002. https://doi.org/10.1007/978-1-59259-132-9_2.

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WILSON, BARRY W., MICHAEL HOOPER, EDWARD CHOW, JAMES N. SEIBER, and JAMES B. KNAAK. "Assessment of Farmworker Risk from Organophosphate-Induced Delayed Neuropathy." In ACS Symposium Series. American Chemical Society, 1985. http://dx.doi.org/10.1021/bk-1985-0273.ch034.

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Johnson, M. K. "Organophosphate-induced Delayed Neuropathy: Anomalous Data Lead to Advances in Understanding." In Selectivity and Molecular Mechanisms of Toxicity. Palgrave Macmillan UK, 1987. http://dx.doi.org/10.1007/978-1-349-08759-4_2.

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Jortner, Bernard S. "Organophosphorus Ester-Induced Delayed Neuropathy, Rat." In Nervous System. Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-83516-2_7.

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Ehrich, Marion. "Neurotoxic Esterase Inhibition: Predictor of Potential for Organophosphorus-Induced Delayed Neuropathy." In ACS Symposium Series. American Chemical Society, 1996. http://dx.doi.org/10.1021/bk-1996-0643.ch006.

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Tanaka, Duke, Steven J. Bursian, and Ellen J. Lehning. "Silver Impregnation of Organophosphorus-Induced Delayed Neuropathy in the Central Nervous System." In The Vulnerable Brain and Environmental Risks. Springer US, 1992. http://dx.doi.org/10.1007/978-1-4615-3330-6_12.

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Veronesi, Bellina, and Stephanie Padilla. "Rodent Models of Organophosphorus-Induced Delayed Neuropathy." In Organophosphates Chemistry, Fate, and Effects. Elsevier, 1992. http://dx.doi.org/10.1016/b978-0-08-091726-9.50022-7.

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Veronesi, Bellina. "Validation of a rodent model of organophosphorus-induced delayed neuropathy." In Clinical and Experimental Toxicology of Organophosphates and Carbamates. Elsevier, 1992. http://dx.doi.org/10.1016/b978-0-7506-0271-6.50017-6.

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"Organophosphate-Induced Delayed Polyneuropathy." In Dictionary of Toxicology. Springer Nature Singapore, 2024. http://dx.doi.org/10.1007/978-981-99-9283-6_2000.

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"Organophosphate-Induced Delayed Polyneuropathy (OPIDP)." In Dictionary of Toxicology. Springer Nature Singapore, 2024. http://dx.doi.org/10.1007/978-981-99-9283-6_1987.

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