Academic literature on the topic 'Original screen plays'
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Journal articles on the topic "Original screen plays"
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Efimova, Natalia Nikolaevna. "Sound Editing in Screen Works." Journal of Flm Arts and Film Studies 7, no. 2 (June 15, 2015): 73–81. http://dx.doi.org/10.17816/vgik7273-81.
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The article pinpoints peculiarities of sound editing in movies basing on analysis of partitions of popular films of40-90s; the most frequent principles of sound track arrangement are examined for the first time. The stuff selection is conditioned by measure of popularity of screen works in question. Due to talent of such famous composers as I. Dunaevsky, S. Prokofiev, A. Khachaturian, A. Pakhmutova, A. Petrov et al and their ability to hear plastic imagery, to comprehend filmic atmosphere music plays an extremely important part in these films. Many songs from these films are still in circulation even now. Thorough sound design and editing are of great significance in film production. The author comes to conclusion, that rondo as a musical form and leit-motif as a principle of musical stuff development form a dominant principle of sound stuff arrangement. The two fundamentally tighten the structure of the film. Since original music affords to accentuate sound effects in the most adequate way, it seems perfect to call to a composer for creating original music. The author assumes, that the choice of sound arrangement principle in cinema depends on deliberate conception of the film, wrought out by the helmer, composer, and supervising sound editor. The screen works property is closely bound with attentive partition editing.
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Anderson, Laura. "Sonic ‘Detheatricalization’: Jean Cocteau, Film Music, and ‘Les Parents Terribles’." Music and Letters 100, no. 4 (November 1, 2019): 654–84. http://dx.doi.org/10.1093/ml/gcz081.
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Abstract Jean Cocteau’s adaptation of his controversial play Les Parents terribles for the screen stands out in his oeuvre as an attempt to reconcile theatre and cinema. It also presented a challenge in preparing a soundscape for a work that did not have any music in its original form. Parents occupies a unique, Janus-like position in the history of French film music, as forward-looking in its anticipation of New Wave treatment of music as material as it is representative of the turn to adapting stage plays for the screen that started in the 1930s. Drawing on production sketchbooks and testimonies, this article considers the development of Cocteau’s working method and his collaboration with Georges Auric, fuelled by the director’s desire to take control of sonic matters. The resulting employment of a monothematic score was not only a new solution to the famous problem of filmed theatre, ‘detheatricalizing’ Parents sonically and visually, it contributed considerably to the development of Cocteau’s status as film auteur—one whose role now extended to adapting musical material. Furthermore, the effect of this compositional technique in Parents suggests that it can be fruitfully situated in relation to recent work in film music studies on issues of anempathetic scoring practices.
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El Maï, Mounir, Serena Janho dit Hreich, Cedric Gaggioli, Armelle Roisin, Nicole Wagner, Jing Ye, Pierre Jalinot, Julien Cherfils-Vicini, and Eric Gilson. "A Novel Screen for Expression Regulators of the Telomeric Protein TRF2 Identified Small Molecules That Impair TRF2 Dependent Immunosuppression and Tumor Growth." Cancers 13, no. 12 (June 15, 2021): 2998. http://dx.doi.org/10.3390/cancers13122998.
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Telomeric repeat-binding factor 2 (TRF2) is a subunit of the shelterin protein complex, which binds to and protects telomeres from unwanted DNA damage response (DDR) activation. TRF2 expression plays a pivotal role in aging and cancer, being downregulated during cellular senescence and overexpressed during oncogenesis. Cancers overexpressing TRF2 often exhibit a poor prognosis. In cancer cells, TRF2 plays multiple functions, including telomere protection and non-cell autonomous roles, promoting neo-angiogenesis and immunosuppression. We present here an original screening strategy, which enables identification of small molecules that decrease or increase TRF2 expression. By screening a small library of Food and Drug Agency (FDA)-approved drugs, we identified two molecules (AR-A014418 and alexidine·2HCl) that impaired tumor growth, neo-angiogenesis and immunosuppression by downregulating TRF2 expression in a mouse xenograft model. These results support the chemotherapeutic strategy of downregulating TRF2 expression to treat aggressive human tumors and validate this cell-based assay capable of screening for potential anti-cancer and anti-aging molecules by modulating TRF2 expression levels.
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Del Priore, V., C. Heath, C. Snay, A. MacMillan, L. Gorsch, S. Dagher, and C. Cole. "A structure/function analysis of Rat7p/Nup159p, an essential nucleoporin of Saccharomyces cerevisiae." Journal of Cell Science 110, no. 23 (December 1, 1997): 2987–99. http://dx.doi.org/10.1242/jcs.110.23.2987.
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Rat7p/Nup159p is an essential nucleoporin of Sac-charomyces cerevisiae originally isolated in a genetic screen designed to identify yeast temperature-sensitive mutants defective in mRNA export. Here we describe a detailed structural-functional analysis of Rat7p/Nup159p. The mutation in the rat7-1 ts allele, isolated in the original genetic screen, was found to be a single base pair change that created a stop codon approximately 100 amino acids upstream of the actual stop codon of this 1,460 amino acid polypeptide, thus eliminating one of the two predicted coiled-coil regions located near the carboxyl terminus of the protein. These coiled-coil regions are essential since an allele lacking both coiled-coil regions was unable to support growth under any conditions. In contrast, no other region of the protein was absolutely required. The SAFG/PSFG repeat region in the central third of the protein was completely dispensable for growth at temperatures between 16 degrees C and 37 degrees C and cells expressing this mutant allele were indistinguishable from wild type. Deletion of the amino-terminal third of the protein, upstream from the repeat region, or the portion between the repeat region and the coiled-coils resulted in temperature-sensitivity, but the two alleles showed distinct phenotypes with respect to the behavior of nuclear pore complexes (NPCs). Taken together, our data suggest that Rat7p/Nup159p is anchored within the NPC through its coiled-coil region and adjacent sequences. In addition, we postulate that the N-terminal third of Rat7p/Nup159p plays an important role in mRNA export.
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Kruger, Jan-Louis. "Ideology and Subtitling: South African Soap Operas." Broadcasting with Intent 57, no. 2 (February 4, 2013): 496–509. http://dx.doi.org/10.7202/1013958ar.
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This article investigates the ideological component of patronage in the subtitling of four South African soap operas: Generations, 7de Laan, Muvhango, and Isidingo. Taking the concepts introduced by Lefevere as point of departure, the article first discusses the various ways in which audiovisual translation (AVT) is subject to manipulation. This manipulation is shown to be a result of the fact that subtitles, as text superimposed onto the image during post-editing, thereby obscuring a small part of the screen, constantly foregrounds itself to the audience. This foregrounding is also affected by the linguistic background of the audience – whether or not they understand the original dialogue. The argument then turns to a discussion of AVT, and specifically subtitling, as rewriting. The link between language and ideology is discussed as it pertains to issues of power, particularly related to the role of English in the media, also in South Africa, where, in Gottlieb’s terminology, South Africa can be described as a multilingual anglophile context. The language policy of the South African Broadcasting Corporation is then discussed in terms of patronage and ideology followed by a discussion of the role of ideology in these four locally-produced soap operas. In this discussion the different ways in which the subtitling practices of the soap operas reflect ideology are investigated. The article concludes that accessibility plays a smaller role in subtitling in South Africa than the ideology of multilingualism and multiculturalism.
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Gozdecka, Malgorzata, Monika Dudek, Konstantinos Tzelepis, Aristi Damaskou, Vijay Baskar, Penny Wright, Graham Duddy, et al. "Genetic Vulnerabilities of DNMT3AR882H in Myeloid Malignancies." Blood 134, Supplement_1 (November 13, 2019): 111. http://dx.doi.org/10.1182/blood-2019-126505.
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Mutations affecting the gene for the de novo DNA Methyltransferase 3A (DNMT3A) are the most common drivers of clonal hematopoiesis (CH) and amongst the most common somatic events in acute myeloid leukaemia (AML). Approximately two thirds of AML-associated DNMT3A mutations are heterozygous substitutions affecting codon R882, located within the methytransferase domain and are correlated with global hypomethylation. DNMT3A mutations are initiating events in leukemogenesis and leukemic progression relies on the acquisition of additional mutations in genes such as NPM1, TET2, IDH1/2, FLT3 andNRAS. AMLs harboring DNMT3A-R882 mutations display increased resistance to chemotherapy and carry a worse prognosis for patients, but the molecular basis of this is not well understood. To improve our understanding of the molecular effects of mutant DNMT3A, we developed a conditional model of DNMT3A-R882H. Our conditional Dnmt3a-flox-R882H allele retains the native locus intact, but generates the Dnmt3a-R882H mutant upon Cre-loxP recombination (Mx1-Cre). We observed that Dnmt3aR882H/+ mutant bone marrow (BM) cells had markedly enhanced self-renewal potential in serial re-plating assays and increased BM repopulation ability in competitive transplants. Approximately 30% of Dnmt3aR882H/+ mice developed leukaemia after a long latency (median of 532 days). Introduction of Flt3-ITD mutation significantly accelerated spontaneous disease, in agreement with other published reports, with majority of mice succumbing to AML after a median of 192 days. Due to the frequent co-occurrence and strong cooperation between Dnmt3aR882H/+ with Flt3ITD/+, we sought to identify potential genetic vulnerabilities of this combination, to facilitate patient treatment. We therefore generated Dnmt3aR882H/+/Flt3ITD/+/Mx1-Cre+/Rosa-Cas9+ mice, induced Dnmt3aR882H mutation with pIpC and isolated hematopoietic stem and progenitor cells (HSPCs). We cultured HSPCs in vitro and performed whole-genome CRISPR dropout screens to identify genetic vulnerabilities. This identified 526 dropout genes at a genome-wide FDR of 20%, including pan-essential genes such as components of the ribosome, proteasome or spliceosome. To identify essential genes specific to the Dnmt3aR882H/+/Flt3ITD/+ genotype, we performed analogous screens in the non-leukemic mouse hematopoietic precursor cell-7 (HPC-7) and used published data indicating essential genes for normal human CD34 cells. Overlap of these screens revealed 196 dropouts present only in Dnmt3aR882H/+/Flt3ITD/+/Mx1-Cre+/Rosa-Cas9+ cells. Among these were genes coding for "druggable" candidates such as kinases and histone modifying enzymes, including SET Domain Containing 1B (Setd1b), one of the six mammalian proteins (also Setd1a and Mll1-4) that can catalyze methylation of H3K4. Each of these six proteins can interact with obligate proteins as a part of the methyltransferase complex, but also form distinct complexes with non-redundant function. We firstly validated that the knockout of Setd1b induced by gRNA significantly reduced the growth of pre-leukemic and leukemic HSPCs from both Dnmt3aR882H/+/Flt3ITD/+/Mx1-Cre+/Rosa-Cas9+ and Dnmt3aR882H/+/Mx1-Cre+/Rosa-Cas9+ mice. Importantly, Setd1b deletion in normal HSPC had no major effect on cell growth. To investigate the molecular role of Setd1b in Dnmt3aR882H context, we asked if Setd1b catalytic activity and linked transcriptional programs were responsible for the observed phenotypes. For this, we performed histone profiling and RNA-sequencing in Dnmt3aR882H/+/Flt3ITD/+/Mx1-Cre+/Rosa-Cas9+HSPC upon Setd1b KO vs control and observed a striking reduction of H3K4me3 marks at particular loci in association with loss of expression of corresponding genes. These included several known to be required for cellular proliferation and transformation, some of which were themselves dropouts in our original screen. Importantly the majority of genes were also significantly upregulated when gene expression from Dnmt3aR882H/+/Flt3ITD/+ HSPC were compared with Flt3ITD/+ HSPC. Our data propose that the catalytic activity of Setd1b plays a mechanistic role in driving the growth/fitness-promoting effects of Dnmt3aR882H/+. Blocking the catalytic activity of SETD1B may therefore represent a plausible therapeutic option for the treatment of AML of even CH patients harboring mutations inDNMT3A. Disclosures Mazan: Selvita S.A.: Employment. Vassiliou:Kymab Ltd: Consultancy, Other: Minor Stockholder; Oxstem Ltd: Consultancy; Celgene: Research Funding.
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Gutierrez, Alejandro, Hui Feng, Prochownik Edward, John Kanki, and A. Thomas Look. "A Tamoxifen-Dependent Conditional Model of MYC-Induced T Cell Acute Lymphoblastic Leukemia in the Zebrafish." Blood 110, no. 11 (November 16, 2007): 2808. http://dx.doi.org/10.1182/blood.v110.11.2808.2808.
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Abstract The MYC oncogene plays a central role in the pathogenesis of human T cell acute lymphoblastic leukemia (T-ALL), and our laboratory has previously developed a zebrafish model of Myc-induced T-ALL. The primary strength of the zebrafish as a model system for human disease lies in is its suitability for unbiased forward genetic and small molecule screens. Our central hypothesis is that forward screens performed using our zebrafish model of MYC-induced T-ALL will lead to the identification of entirely novel genes and pathways that play critical roles in MYC-induced leukemogenesis. However, zebrafish from our original line develop rapidly progressive T-ALL prior to achieving reproductive maturity, making this line poorly suited for the performance of large-scale screens. Therefore, a conditional model was required. We have now generated a transgenic zebrafish line that expresses a human MYC-estrogen receptor fusion construct under the control of the zebrafish recombination activating gene 2 (Rag2) promoter, which is lymphocyte-specific. When mated against fish transgenic for a Rag2-GFP transgene, the development and progression of T-ALL can be readily tracked in live fish by fluorescent microscopy. Upon treatment with 4-hydroxytamoxifen (4HT), zebrafish from this line develop fully penetrant T-ALL, with a mean time to tumor onset of 8 weeks. Additionally, removal from 4HT invariably led to complete morphologic remission in leukemic zebrafish from this line, and all of these fish remained alive and were able to mate successfully for greater than 6 months after removal from 4HT. This conditional zebrafish model of MYC-induced T-ALL will now allow the successful performance of forward genetic and small molecule screens to identify known and novel genes and pathways that play critical roles in T-ALL leukemogenesis and MYC-induced transformation. Figure Figure
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Chen, Lindsey N. "Cross-Linguistic Comparison of Screen Translations of Names in Hayao Miyazaki’s Animated Films." Вопросы Ономастики 18, no. 1 (2021): 195–208. http://dx.doi.org/10.15826/vopr_onom.2021.18.1.009.
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This article discusses the screen translation, into English and Chinese, of some of the names in Hayao Miyazaki’s animated films. In particular, in drawing onomastic examples from Miyazaki’s six animated fantasies, this study provides insights into the naming practices and strategies adopted by the screen translators into languages of distinct linguistic families. The analysis of names sorted into four categories yields the following results: (a) each screen translator used diff erent approaches to the translation of names, and (b) shared similarities with the source language and culture play a crucial role in the translation task. In brief, the first category concerns the films’ protagonists, for which the strategy of diminution is observed in Chinese but not in English translation. The second concerns the names of supporting human characters. Here, screen translators adopt several strategies, including direct phonetic transfer and incorporation of courtesy titles. The third comprises names of anthropomorphic and non-human creatures, and translated samples are shown more likely to be denotative and descriptive. Finally, there is no loss in translation with respect to the symbolic implication of location names. In general, screen translators utilized various linguistic strategies to produce onomastic substitutes that are acceptable to the local audience. Concurrently, they strived not to deviate too much from the original character names, in form and meaning.
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Natalie Winteringham, Louise, Raelene Endersby, Jennifer Beaumont, Jean-Philippe Lalonde, Merlin Crossley, and Svend Peter Klinken. "Hls5, a Novel Ubiquitin E3 Ligase, Modulates Levels of Sumoylated GATA-1." Blood 114, no. 22 (November 20, 2009): 253. http://dx.doi.org/10.1182/blood.v114.22.253.253.
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Abstract Abstract 253 Hemopoietic lineage commitment is controlled, in part, by transcription factors that regulate specific genes required for the formation of mature blood cells. Differentiation along particular hemopoietic lineages is dependant not only on the presence of particular transcription factors, but also on appropriate concentrations - altering transcription factor levels can force cells into different hemopoietic pathways. Transcription factors undergo numerous post-translational modifications and are controlled spatially via sub-cellular localisation. De-regulation of transcription factors can result in leukemias, or other blood disorders. GATA-1 is an example of a key lineage-determining gene, essential for erythropoiesis. Increasing GATA-1 levels promotes maturation along the erythroid pathway, whereas reducing GATA-1 concentrations favours myelopoiesis. GATA-1 regulation occurs at multiple levels including transcription, translation and post-translational modifications such as phosphorylation, acetylation, ubiquitination and sumoylation. Although GATA-1 ubiquitination modifies the protein for proteasomal degradation, the effect of adding small ubiquitin-like modier (Sumo) to GATA-1 is unclear. Several examples of hemopoietic differentiation plasticity have been observed. We reported a lineage switch by erythroleukemic J2E cells which spontaneously developed a monoblastoid phenotype. Two genes (Hls5 and Hls7/Mlf1) were isolated from this lineage switch with potential lineage-determining features. Hls5 is a member of the RBCC (Ring finger, B-box, Coiled-coil) family of proteins, which includes PML. Ectopic expression of Hls5 impedes erythroid differentiation by reducing GATA-1 levels, and suppressing hemoglobin synthesis. Significantly, Hls5 relocates from the cytoplasm to associate with GATA-1 in the nucleus, where it interferes with DNA binding and transactivation of GATA-1. Several members of the RBCC family are ubiquitin E3 ligases, catalysing the final step in the ubiquitination process - these molecules play a vital role in regulating the levels of target proteins. Here we show that Hls5 is a bona fide ubiquitin E3 ligase, in partnership with several ubiquitin E2 enzymes. The Ring finger is critical for Hls5 ligase activity as mutation of key residues within the Ring finger ablates catalytic activity. Interestingly, a yeast 2 hybrid screen for Hls5 interactors identified Ubc9 and Pias1, which act as E2 and E3 enzymes in the sumoylation cascade. Co-immunoprecipitation, BRET and co-localization experiments confirmed the Hls5 association with Ubc9 and Pias1. Moreover, Hls5 binds Sumo-1 (but not Sumo-2 or 3), and co-localizes with Sumo-1 in discrete nuclear bodies. Thus, Hls5 interacts with several components of the intracellular sumoylation machinery. Hls5 can also reduce sumoylated proteins globally, indicating it may target these modified proteins for degradation. Recently, a new family of ubiquitin E3 ligases has been described which specifically mark sumoylated proteins for degradation. These Sumo-targeted ubiquitin ligases (STUbL) are found primarily in yeast, and only one mammalian STUbL has been identified. We postulated that Hls5 may be a STUbL, capable of regulating sumoylated GATA-1. Our data demonstrate that while Hls5 is able to bind GATA-1 via the B-box and Coiled-coil domains, it preferentially associates with sumoylated GATA-1 through a canonical Sumo interacting motif (SIM). This results in increased GATA-1 ubiquitination and, as a consequence, levels of sumoylated GATA-1 are reduced substantially. Since mutation of the lysine necessary for Sumo attachment does not affect GATA-1 transactivation, sumoylation may act as a prelude to ubiquitination and protein turn-over. We propose, therefore, that GATA-1 mediates transcription of target genes, and is subsequently sumoylated by Pias1 and Ubc9 – addition of Sumo moieties to GATA-1 enhance binding to Hls5, which in turn impedes GATA-1 DNA binding, and promotes ubiquitination for proteasomal degradation. This model is consistent with decreased levels of GATA-1 in erythroid cells ectopically expressing Hls5, and with the original isolation of Hls5 as a potential lineage-determining gene involved with the erythroid to monoblastoid lineage switch. Thus, Hls5 is a novel STUbL which plays a role in hemopoietic lineage commitment by modulating GATA-1 activity and content. Disclosures: No relevant conflicts of interest to declare.
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Shorter, Edward. "Still tilting at windmills." Psychiatrist 35, no. 5 (May 2011): 183–84. http://dx.doi.org/10.1192/pb.bp.111.034108.
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SummaryThomas Szasz's essay misses several key points about the undoubted changes that psychiatry has undergone since he wrote his original screed against the discipline in 1961. Szasz fails to recognise that the discipline today acknowledges a neurological basis for much psychiatric illness. Thus, his fulminations against psychiatry for treating ‘mental illness' is off-base. Szasz's original diatribe was heavily against psychoanalysis. Yet today Freud's doctrines can scarcely be said to play even a marginal role in psychiatry, and it is absurd to keep levelling the same old charges of 50 years ago. One has the feeling of looking at one of the last veterans of the Esperanto movement in confronting Szasz: lunacy at the time, bizarrely outdated today.
Dissertations / Theses on the topic "Original screen plays"
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Buchholz, Robert Henry. "Triathlon: an Original Screenplay." Thesis, North Texas State University, 1987. https://digital.library.unt.edu/ark:/67531/metadc504205/.
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A young man, out of college and work, sets out to make his mark on the world, by winning the endurance sport of the eighties: the Hawaiian Ironman Triathlon. As he eats, sleeps and breaths "Ironman," he shuts others out of his life because he feels that he must do it alone for the victory to be genuine; a philosophy that has been dogging him all his life.
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Kopchick, Laura A. (Laura Ann). "Scam King." Thesis, University of North Texas, 1994. https://digital.library.unt.edu/ark:/67531/metadc500605/.
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"Scam King" is a full-length feature screenplay and follows standard script format. The idea behind "Scam King" came originally from the James Joyce short story "Two Gallants" in Dubliners. "Scam King" is, however, not an adaption of Joyce's story, but rather was inspired by the gaps in his story pertaining to the characters' way of life on the street.
Books on the topic "Original screen plays"
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Simon, Neil. The odd couple I and II: The original screen plays. New York, NY: Simon & Schuster, 2000.
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Enright, Nick. Blackrock: Original Screenplay (Currency Screen Plays). Currency Press, 1998.
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Vidal, Gore. The Best Man: A Screen Adaptation of the Original Play Directed by Franklin Schaffner, Garrett. Irvington Publishers, 1989.
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McHugh, Dominic, ed. The Oxford Handbook of Musical Theatre Screen Adaptations. Oxford University Press, 2019. http://dx.doi.org/10.1093/oxfordhb/9780190469993.001.0001.
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This book examines the phenomenon of adapting musicals originally written for the Broadway or West End (London) stage into Hollywood movies. It highlights tensions between live and recorded media, between the culture of the East and West Coasts of America, and between producers on Hollywood and Broadway. The book is divided into sections dealing with identity, technology, audiences, music, stars and multiple adaptations of single works. A range of methodologies is used, including film studies and musicology, and archival research has informed original readings in various chapters. Some chapters also look at how the stage musical concerned is already an adaptation, e.g from a play or novel. Overall, the book reflects on stage-to-screen adaptations and offers an introduction to the scholarship on the subject, often offering the first-ever scholarship on various important films.
Book chapters on the topic "Original screen plays"
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Ivory, Yvonne. "Prussian Discipline and Lesbian Vulnerability: Christa Winsloe’s Children in Uniform at the Gate." In Cultural Convergence, 193–216. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-57562-5_8.
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Abstract This chapter examines the Dublin production and critical reception of Christa Winsloe’s Children in Uniform, which ran to full houses at the Gate for three weeks in April 1934. The play, which deals with the love between a Prussian schoolgirl and her female teacher, had premiered in Leipzig (1930), run successfully in Berlin (1931), and been adapted for the screen as Mädchen in Uniform (1931) before it was translated into English for a successful London run in 1932-1933. Edwards and mac Liammóir probably saw the original German play in Berlin in 1931. Using the prompt copy, lighting plots, photographs and reviews, the chapter shows how Edwards used expressionistic lighting and sonic leitmotifs to underscore the authoritarian regime within which the relationship between the women develops. In following the Berlin staging, Edwards produced a more subversive version of the play than that seen by London audiences or cinema goers.
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Pérez Bowie, José Antonio. "Más allá de la adaptación." In El teatro clásico español en el cine. Venice: Edizioni Ca' Foscari, 2019. http://dx.doi.org/10.30687/978-88-6969-330-4/003.
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The relationship between cinema and literature is not limited to the phenomenon of adaptation: literary texts can be present on the screen through other channels that do not involve the complete transfer of textual contents but are limited to citing it, to take advantage of some of its thematic materials, to proceed to a thorough reordering of its elements, etc. This paper tries to classify and describe some of those ways through the analysis of some plays of the Spanish Golden Age that have served as basis for very different films but that cannot be qualified stricto sensu as adaptations due to interventions made on the original texts or the irrelevant presence of them in the final result.
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Dwyer, Tessa. "Vanishing Subtitles: The Invisible Cinema (1970–4)." In Speaking in Subtitles. Edinburgh University Press, 2017. http://dx.doi.org/10.3366/edinburgh/9781474410946.003.0003.
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This case-study chapter focuses on the blanket rejection of both subtitling and dubbing at New York’s short-lived Invisible Cinema, established by Anthology Film Archives in the early 1970s. In resurrecting the silent-era dream of non-translation, the Invisible Cinema drew attention, paradoxically, to translation’s centrality for screen culture generally and Anthology in particular – pinpointing the re-evaluative role that translation plays in screen culture by keeping ‘originals’ in circulation and contention. This point is affirmed by Anthology’s present-day operations and the residual legacy of its translation ban. Additionally, the chapter explores how the Invisible Cinema’s excessive zero-tolerance approach to translation actually takes certain pro-subtitling arguments to their logical conclusion and is hence ripe for deconstruction. Hence, this chapter outlines a route for revaluation developed further in subsequent chapters, identifying the flaws and failures of screen translation as necessary to the preservation and destabilisation of screen culture.
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Dwyer, Tessa. "Dubbing Undone: Can Dialectics Break Bricks? (1973)." In Speaking in Subtitles. Edinburgh University Press, 2017. http://dx.doi.org/10.3366/edinburgh/9781474410946.003.0004.
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This chapter explores how dubbing has been deployed as a mode of deliberate, self-reflexive mistranslation. Can Dialectics Break Bricks? flaunts translation dysfunction as a deliberate strategy of political or aesthetic intervention, challenging the authority of authorship and ‘originals’ in the process. Engaging extensively with the notion of ‘abusive translation’ developed by Derrida and updated by Abe Markus Nornes, it demonstrates how errant forms of screen translation evade theoretical containment, and indicate a path for revaluation firmly grounded by the ‘practical’. Parodic mistranslation or deconstructive dubbing, it proposes, presents an overly abusive example of screen translation that indicates how quality considerations are insufficient for engaging with improper modes of practice. It also introduces issues relating to translation censorship and media piracy foreshadowed by the parody dynamics at play in Can Dialectics Break Bricks?
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Ali Yüksel, Kamer. "Gestural Interaction with Mobile Devices Based on Magnetic Field." In Advances in Wireless Technologies and Telecommunication, 203–22. IGI Global, 2014. http://dx.doi.org/10.4018/978-1-4666-4446-5.ch011.
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The theory of around device interaction (ADI) has recently gained a lot of attention in the field of human computer interaction (HCI). As an alternative to the classic data entry methods, such as keypads and touch screens, ADI founds a 3D user interface that extends to the peripheral area of a device. In this chapter, the authors introduce a revolutionary interaction framework that is based on the idea of ADI. The proposed method constitutes a touchless data entry system that is based on the interaction between the magnetic fields around a device and a properly shaped magnet. The magnetic field that surrounds the device is generated by a magnetic sensor (compass) that is embedded in the new generation of mobile phones such as Apple’s iPhone 3GS and 4G, and Google’s Nexus one. The user movements of the properly shaped magnet in front of the device, then, deforms the sensor’s original magnetic field pattern whereby we can constitute a new means of communication between the user and the device. Thus, the magnetic field encompassing the device plays the role of a communication channel and encodes the hand-movement patterns of the user into temporal changes of the sensor’s magnetic field. In the back-end of the communication, an engine samples the momentary status of the field during a trial and recognizes the user’s pattern by matching it against some pre-recorded templates. The proposed method has been tested in a variety of applications such as handwriting recognition, user authentication, gesture recognition, and some entertainment applications. The experimental results show that the proposed interface not only elevates the convenience of user-device interactions, but also shows very promising accuracies in a wide range of applications requiring user interactions.
Conference papers on the topic "Original screen plays"
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Hoernig, T., and C. Friedrich. "Thread Reinforcement of Screw Connections in Lightweight Design." In ASME 2014 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2014. http://dx.doi.org/10.1115/imece2014-39465.
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Design engineers today desire to develop more lighter and compact components with higher performance capabilities against the background of saving resources. For this purpose, low strength and lightweight metals such Aluminum or Magnesium are preferred on one hand. On the other hand there is an increase in load requirements which needs higher clamp force between these components. In such applications length of thread engagement requires special focus. At the same time from industry perspective it is essential to have effective dimensioning methods to develop new products in shorter time and at a reasonable cost (economics). Besides the increase in length of thread engagement, the tribology of mechanical contacts (i.e. increased friction coefficients and limited loading capacity due to higher contact pressure) plays an important role in the tightening process. The result is that the desired preload is often not achieved with a standardized tightening specification (reasons: high friction decreases preload for same tightening torque by high torsional stresses). In this contribution experimental results of tightening screws with nut components made from aluminum alloys are analyzed for their frictional behavior by using a multi-channel screw assembly test stand. Risks and dangers associated with tightening process are highlighted and suggestions are made for improvement in thread reinforcement. An established method to realize thread reinforcement is using wired thread inserts. For these elements several standards concerning geometry and strength exist but no dimensioning method is available. These thread inserts themselves have a higher strength than the nut thread material and transmit the preload force of the screw with a greater surface area than the original nut component. This leads theoretically to an increase in the load capacity of the nut thread. The approach of existing dimensioning methods by established rules and regulations for the design of bolted connections, e.g. VDI 2230, are not able to predict the behavior of reinforced thread engagements. Hence, an extension for the dimensioning of threaded engagement is essential for modern requirements. Experiments of tightening screw joints with wired thread inserts and with normal thread engagement are comparatively analyzed along with numerical calculations using Finite Element Analysis (FEA). Conclusions are drawn from experimental and FEA results with the perspective of formulating an extended dimensioning method for thread reinforcement of screw connections in lightweight design. These points are interesting for every design engineer, especially in the field of lightweight design.
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Johnson, Michael D., Elif Ozturk, Lauralee Valverde, Bugrahan Yalvac, Prentiss McGary, and Xiaobo Peng. "A Methodology for Examining the Role of Adaptive Expertise on CAD Modeling." In ASME 2013 International Design Engineering Technical Conferences and Computers and Information in Engineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/detc2013-12779.
Full textAbstract:
Computer-aided design (CAD) tools play a significant role in the modern product commercialization environment. As CAD and general CAx technology advances, it becomes more important to understand how engineers adapt their expertise to new environments and problems. This work examines a methodology consisting of a set of surveys, interviews, and exercises with a small group of practicing engineers to assess adaptive expertise (AE) and relate this AE to CAD modeling performance and procedures. Results detail AE survey results, a modeling and alteration exercise, and an exercise where engineers are asked to model a component using a CAD platform they are unfamiliar with. Interview and time use (from screen capture videos) results from this exercise are presented along with other data. Correlations among AE survey and interview variables and model analysis variables are presented. The epistemology dimension of the AE survey was found to be negatively correlated with both original modeling and alteration time. Overall modeling time in the different platform was positively correlated with the percentage of time a participant spent engaging in trial and error; modeling time in the different platform was negatively correlated with percentage of time spent doing actual modeling and the time spent thinking.