Academic literature on the topic 'Orthostatic stress'

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Journal articles on the topic "Orthostatic stress"

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Watenpaugh, Donald E., Deborah D. O'Leary, Suzanne M. Schneider, et al. "Lower body negative pressure exercise plus brief postexercise lower body negative pressure improve post-bed rest orthostatic tolerance." Journal of Applied Physiology 103, no. 6 (2007): 1964–72. http://dx.doi.org/10.1152/japplphysiol.00132.2007.

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Orthostatic intolerance follows actual weightlessness and weightlessness simulated by bed rest. Orthostasis immediately after acute exercise imposes greater cardiovascular stress than orthostasis without prior exercise. We hypothesized that 5 min/day of simulated orthostasis [supine lower body negative pressure (LBNP)] immediately following LBNP exercise maintains orthostatic tolerance during bed rest. Identical twins (14 women, 16 men) underwent 30 days of 6° head-down tilt bed rest. One of each pair was randomly selected as a control, and their sibling performed 40 min/day of treadmill exerc
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Bronzwaer, Anne-Sophie G. T., Jasper Verbree, Wim J. Stok, et al. "The cerebrovascular response to lower-body negative pressure vs. head-up tilt." Journal of Applied Physiology 122, no. 4 (2017): 877–83. http://dx.doi.org/10.1152/japplphysiol.00797.2016.

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Lower-body negative pressure (LBNP) has been proposed as a MRI-compatible surrogate for orthostatic stress. Although the effects of LBNP on cerebral hemodynamic behavior have been considered to reflect those of orthostatic stress, a direct comparison with actual orthostasis is lacking. We assessed the effects of LBNP (−50 mmHg) vs. head-up tilt (HUT; at 70°) in 10 healthy subjects (5 female) on transcranial Doppler-determined cerebral blood flow velocity (CBF v) in the middle cerebral artery and cerebral perfusion pressure (CPP) as estimated from the blood pressure signal (finger plethysmograp
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Sonkodi, Balázs, Tamás Radovits, Emese Csulak, Bence Kopper, Nóra Sydó, and Béla Merkely. "Orthostasis Is Impaired Due to Fatiguing Intensive Acute Concentric Exercise Succeeded by Isometric Weight-Loaded Wall-Sit in Delayed-Onset Muscle Soreness: A Pilot Study." Sports 11, no. 11 (2023): 209. http://dx.doi.org/10.3390/sports11110209.

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The aim of the study was to investigate any indication of diminished orthostatic tolerance as a result of fatiguing intensive acute concentric exercise with a successive isometric wall-sit followed by an orthostatic stress test, with a special focus on any distinguishable alterations due to a delayed-onset muscle soreness effect. The exercise protocol was carried out among nineteen (10 female, 9 male) junior swimmers from the Hungarian National Swim Team. All athletes showed a positive orthostatic stress test right after our exercise protocol. The diastolic blood pressure was significantly low
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Taylor, J. A., G. A. Hand, D. G. Johnson, and D. R. Seals. "Sympathoadrenal-circulatory regulation of arterial pressure during orthostatic stress in young and older men." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 263, no. 5 (1992): R1147—R1155. http://dx.doi.org/10.1152/ajpregu.1992.263.5.r1147.

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Our purpose was to test the hypothesis that human aging alters sympathoadrenal-circulatory control of arterial blood pressure during orthostasis. Plasma catecholamine and hemodynamic adjustments to two different forms of orthostatic stress, lower body suction (-10 to -50 mmHg) and standing, were determined in 14 young (26 +/- 1 yr) and 13 older (64 +/- 1) healthy, normally active men. During quiet supine rest, cardiac output tended to be lower and systemic vascular resistance higher in the older men, but no other differences were observed. On average, arterial blood pressure was well maintaine
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Javorka, Michal, Fatima El-Hamad, Barbora Czippelova, et al. "Role of respiration in the cardiovascular response to orthostatic and mental stress." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 314, no. 6 (2018): R761—R769. http://dx.doi.org/10.1152/ajpregu.00430.2017.

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The objective of this study was to determine the response of heart rate and blood pressure variability (respiratory sinus arrhythmia, baroreflex sensitivity) to orthostatic and mental stress, focusing on causality and the mediating effect of respiration. Seventy-seven healthy young volunteers (46 women, 31 men) aged 18.4 ± 2.7 yr underwent an experimental protocol comprising supine rest, 45° head-up tilt, recovery, and a mental arithmetic task. Heart rate variability and blood pressure variability were analyzed in the time and frequency domain and modeled as a multivariate autoregressive proce
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Fu, Qi, Sarah Witkowski, Kazunobu Okazaki, and Benjamin D. Levine. "Effects of gender and hypovolemia on sympathetic neural responses to orthostatic stress." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 289, no. 1 (2005): R109—R116. http://dx.doi.org/10.1152/ajpregu.00013.2005.

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We tested the hypothesis that women have blunted sympathetic neural responses to orthostatic stress compared with men, which may be elicited under hypovolemic conditions. Muscle sympathetic nerve activity (MSNA) and hemodynamics were measured in eight healthy young women and seven men in supine position and during 6 min of 60° head-up tilt (HUT) under normovolemic and hypovolemic conditions (randomly), with ∼4-wk interval. Acute hypovolemia was produced by diuretic (furosemide) administration ∼2 h before testing. Orthostatic tolerance was determined by progressive lower body negative pressure
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Kimmerly, Derek S., and J. Kevin Shoemaker. "Hypovolemia and neurovascular control during orthostatic stress." American Journal of Physiology-Heart and Circulatory Physiology 282, no. 2 (2002): H645—H655. http://dx.doi.org/10.1152/ajpheart.00535.2001.

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Humans exposed to real or simulated microgravity experience decrements in blood pressure regulation during orthostatic stress that may be related to autonomic dysregulation and/or hypovolemia. We examined the hypothesis that hypovolemia, without the deconditioning effects of bed rest or spaceflight, would augment the sympathoneural and vasomotor response to graded orthostatic stress. Radial artery blood pressure (tonometry), stroke volume (SV), brachial blood flow (Doppler ultrasound), heart rate (electrocardiogram), peroneal muscle sympathetic nerve activity (MSNA; microneurography), and esti
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Kawai, Mio, Nobuyuki Miyai, and Mikio Arita. "The prevalence of orthostatic dysregulation among newly graduated female nurses after employment and its associations with autonomic nervous function, stress, and depressive symptoms." SAGE Open Medicine 9 (January 2021): 205031212110121. http://dx.doi.org/10.1177/20503121211012180.

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Objectives: We aimed to examine the prevalence of orthostatic dysregulation among newly graduated female nurses after employment and its associations with autonomic nervous function, stress, and depressive symptoms. Methods: This follow-up study included 48 newly graduated female nurses (aged 22 ± 3 years) employed in acute care hospitals. The orthostatic dysregulation symptoms were evaluated using a screening checklist. A sit-to-stand test was conducted to assess the autonomic nervous function. Subjective stress and depressive symptoms were determined using a self-administered questionnaire.
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Wilson, Thad E., Jian Cui, Rong Zhang, and Craig G. Crandall. "Heat stress reduces cerebral blood velocity and markedly impairs orthostatic tolerance in humans." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 291, no. 5 (2006): R1443—R1448. http://dx.doi.org/10.1152/ajpregu.00712.2005.

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Orthostatic tolerance is reduced in the heat-stressed human. This study tested the following hypotheses: 1) whole body heat stress reduces cerebral blood velocity (CBV) and increases cerebral vascular resistance (CVR); and 2) reductions in CBV and increases in CVR in response to an orthostatic challenge will be greater while subjects are heat stressed. Fifteen subjects were instrumented for measurements of CBV (transcranial ultrasonography), mean arterial blood pressure (MAP), heart rate, and internal temperature. Whole body heating increased both internal temperature (36.4 ± 0.1 to 37.3 ± 0.1
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Raj, Satish R. "What is the optimal orthostatic stress to diagnose orthostatic hypotension?" Clinical Autonomic Research 15, no. 2 (2005): 67–68. http://dx.doi.org/10.1007/s10286-005-0265-8.

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Dissertations / Theses on the topic "Orthostatic stress"

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Serrador, Jorge M. "Cerebral autoregulation during orthostatic stress." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape2/PQDD_0017/NQ58188.pdf.

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Howden, Reuben. "Tolerance to orthostatic stress and human cardiovascular control." Thesis, De Montfort University, 2002. http://hdl.handle.net/2086/4812.

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Heldt, Thomas 1972. "Computational models of cardiovascular response to orthostatic stress." Thesis, Massachusetts Institute of Technology, 2004. http://hdl.handle.net/1721.1/28761.

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Thesis (Ph. D.)--Harvard-MIT Division of Health Sciences and Technology, 2004.<br>Includes bibliographical references (p. 163-185).<br>The cardiovascular response to changes in posture has been the focus of numerous investigations in the past. Yet despite considerable, targeted experimental effort, the mechanisms underlying orthostatic intolerance (OI) following spaceflight remain elusive. The number of hypotheses still under consideration and the lack of a single unifying theory of the pathophysiology of spaceflight-induced OI testify to the difficulty of the problem. In this investigation, w
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Ramsey, Michael W., Bradley J. Behnke, Rhonda D. Prisby, and Michael D. Delp. "Aging Alters Regional Vascular Conductance and Arterial Pressure During Orthostatic Stress." Digital Commons @ East Tennessee State University, 2007. https://dc.etsu.edu/etsu-works/4098.

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Berry, Narelle Margaret, and narelle berry@unisa edu au. "Acute and long term interventions to assess the adaptability of the cardiovascular responses to orthostatic stress." RMIT University. Medical Sciences, 2006. http://adt.lib.rmit.edu.au/adt/public/adt-VIT20070228.123618.

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This thesis comprises of four experiments from which related but independent analyses were undertaken. The interventions employed were designed to investigate the effect of cardiovascular adaptation, both in the short and long term on the cardiovascular responses to orthostatic stress. The first study, described in Chapter 3, tested the hypothesis that the cardiovascular system (CVS) could adapt to repeated orthostatic challenges in a single session. 14 subjects were exposed to ten +75° head-up tilts (HUT) over 70 mins. Each tilt involved a 5 min supine period (SUPINE) followed b
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Diehl, Ursula Anne. "The role of the hydrostatic indifferent point in governing splachnic blood pooling during orthostatic stress." Thesis, University of Iowa, 2011. https://ir.uiowa.edu/etd/948.

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The response of the circulatory system to gravity and hydrostatic forces has been well studied, for example the hydrostatic indifferent point (the location at which pressure does not change with posture) of the venous system has been established to be an important determinant of orthostatic responses and it has been found to be located near the diaphragm. However, the role of the abdomen has been less researched; for example, it appears that the concept that the abdominal compartment may have its own hydrostatic indifferent point has been overlooked. The goal of the present study was to establ
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Pawelczyk, James A. (James Anthony). "Interactions between Carotid and Cardiopulmonary Baroreceptor Populations in Men with Varied Levels of Maximal Aerobic Power." Thesis, University of North Texas, 1989. https://digital.library.unt.edu/ark:/67531/metadc331205/.

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Reductions in baroreflex responsiveness have been thought to increase the prevalence of orthostatic hypotension in endurance trained athletes. To test this hypothesis, cardiovascular responses to orthostatic stress, cardiopulmonary and carotid baroreflex responsiveness, and the effect of cardiopulmonary receptor deactivation on carotid baroreflex responses were examined in 24 men categorized by maximal aerobic power (V02max) into one of three groups: high fit (HF, V0-2max=67.0±1.9 ml•kg^-1•min^-1), moderately fit (MF, V0-2max=50.9±1.4 ml•kg^-1•min^-1), and low fit (LF, V0-2max=38.9±1.5 ml•kg^-
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Stevens, Glen Harold John. "Blood Pressure Regulation During Simulated Orthostatism Prior to and Following Endurance Exercise Training." Thesis, University of North Texas, 1992. https://digital.library.unt.edu/ark:/67531/metadc277914/.

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Cardiovascular responses and tolerance to an orthostatic stress were examined in eight men before and after eight months of endurance exercise training. Following training, maximal oxygen consumption and blood volume were increased, and resting heart rate reduced. Orthostatic tolerance was reduced following training in all eight subjects. It was concluded that prolonged endurance training decreased orthostatic tolerance and this decrease in tolerance appeared associated with attenuated baroreflex sensitivity and alterations in autonomic balance secondary to an increased parasympathetic tone no
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Zhang, Qingguang. "HUMAN CARDIOVASCULAR RESPONSES TO SIMULATED PARTIAL GRAVITY AND A SHORT HYPERGRAVITY EXPOSURE." UKnowledge, 2015. http://uknowledge.uky.edu/cbme_etds/30.

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Orthostatic intolerance (OI), i.e., the inability to maintain stable arterial pressure during upright posture, is a major problem for astronauts after spaceflight. Therefore, one important goal of spaceflight-related research is the development of countermeasures to prevent post flight OI. Given the rarity and expense of spaceflight, countermeasure development requires ground-based simulations of partial gravity to induce appropriate orthostatic effects on the human body, and to test the efficacy of potential countermeasures. To test the efficacy of upright lower body positive pressure (LBPP)
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Samin, Azfar. "Neuronal modelling of baroreflex response to orthostatic stress." 2005. http://link.library.utoronto.ca/eir/EIRdetail.cfm?Resources__ID=232699&T=F.

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Books on the topic "Orthostatic stress"

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Urquhart, Nathan Alexander. The cardiovascular response to acute, repeated orthostatic stress. National Library of Canada, 2003.

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Livingstone, Kristina. The cardiovascular hemodynamic responses to various levels of orthostatic stress in children. Brock University, Faculty of Applied Health Sciences, 2007.

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Samin, Azfar. Neuronal modelling of baroreflex response to orthostatic stress. 2005.

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TWAM, S. M. R. Pot Journal: Postural Orthostatic Tachycardia Syndrome , 120 Pages Beautiful Journal for Postural Orthostatic Tachycardia Syndrome Management with Stress and Energy Trackers, POTS Symptom and More. Independently Published, 2020.

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Book chapters on the topic "Orthostatic stress"

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Nobuaki, Yutaka, Akira Amano, Takao Shimayoshi, Jianyin Lu, Eun B. Shim, and Tetsuya Matsuda. "A Model for Simulation of Infant Cardiovascular Response to Orthostatic Stress." In Functional Imaging and Modeling of the Heart. Springer Berlin Heidelberg, 2007. http://dx.doi.org/10.1007/978-3-540-72907-5_20.

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Kaňa, M., M. Jiřina, and J. Holčík. "Estimation of Sympathetic and Parasympathetic Level during Orthostatic Stress Using Artificial Neural Networks." In Recent Advances in Mechatronics. Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-05022-0_73.

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Ottesen, Johnny T., Vera Novak, and Mette S. Olufsen. "Development of Patient Specific Cardiovascular Models Predicting Dynamics in Response to Orthostatic Stress Challenges." In Lecture Notes in Mathematics. Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-32882-4_10.

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Novak, Peter. "Autonomic Tests." In Autonomic Testing, edited by Peter Novak. Oxford University Press, 2019. http://dx.doi.org/10.1093/med/9780190889227.003.0004.

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Autonomic tests are focused on the cardiovascular and sudomotor systems. Established cardiovascular reflex function tests are heart rate variability during paced deep breathing, Valsalva maneuver, and tilt test. Transcranial Doppler is essential to assess cerebral vasculature and blood flow regulation to orthostatic stress. Skin biopsy also assesses small sensory and sudomotor fibers. The test results can be graded by a quantitative scale for grading of cardiovascular reflex tests, transcranial Doppler, quantitative sudomotor axon reflex test, and small fiber (epidermal sensory and sweat gland
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Deharo, Jean-Claude. "Reflex syncope." In ESC CardioMed. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0470.

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Reflex syncope, also called neurally mediated syncope, accounts for 56–73% of the aetiologies of syncope, with a balanced incidence over the various age categories. The most common form is ‘vasovagal syncope’ where the trigger is pain, fever, instrumentation, emotion, or orthostatic stress; ‘situational syncope’ refers to syncope triggered by a specific situation, that is, micturition, defecation, swallowing, cough; and ‘carotid sinus syncope’, which may be triggered by carotid sinus manipulation or diagnosed in patients with syncope and positive carotid sinus massage. The term ‘atypical refle
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Campen, C. (Linda) MC Van, Peter C. Rowe, and Frans C. Visser. "Cerebral Blood Flow is reduced in Severe Myalgic EncephalomyelitisChronic Fatigue Syndrome Patients during Mild Orthostatic Stress Testing An Exploratory Study at 20 Degrees of Head-Up Tilt Testing." In Prime Archives in Medicine. Vide Leaf, Hyderabad, 2021. http://dx.doi.org/10.37247/pamed2ed.3.2021.2.

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Rowell, Loring B. "Neural-Humoral Adjustments To Orthostasis And Long-Term Control." In Human Cardiovascular Control. Oxford University PressNew York, NY, 1993. http://dx.doi.org/10.1093/oso/9780195073621.003.0003.

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Abstract Without rapid constriction of resistance vessels, upright posture would not be possible without severe arterial hypotension in any animal whose heart lies considerable distance above the remainder of the circulation. The most rapid vasoconstriction is initiated by sympathetic vasomotor fibers. If the stress is prolonged, blood pressure is supported by circulating hormones that modify renal salt and water excretion and, in higher concentrations, are also vasoactive. Both neural and humoral effectors can act as efferent arms of the cardiopulmonary and arterial baroreflexes.
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Conference papers on the topic "Orthostatic stress"

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Borovik, Anatoly S., Vladimir O. Negulyaev, Olga S. Tarasova, and Olga L. Vinogradova. "Estimation of Time Characteristics of Baroreflex Resetting During Orthostatic Stress." In 2020 11th Conference of the European Study Group on Cardiovascular Oscillations (ESGCO). IEEE, 2020. http://dx.doi.org/10.1109/esgco49734.2020.9158012.

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Reulecke, S., S. Charleston-Villalobos, T. Aljama-Corrales, et al. "Temporal cardiovascular causality during orthostatic stress by extended partial directed coherence." In 2019 41st Annual International Conference of the IEEE Engineering in Medicine & Biology Society (EMBC). IEEE, 2019. http://dx.doi.org/10.1109/embc.2019.8857021.

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Javorka, Michal, Barbora Czippelova, Lenka Chladekova, et al. "Cardiovascular control during orthostatic and mental stress: Conditional entropy based analysis." In 2014 8th Conference of the European Study Group on Cardiovascular Oscillations (ESGCO). IEEE, 2014. http://dx.doi.org/10.1109/esgco.2014.6847495.

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MITSIS, GEORGIOS D., RONG ZHANG, BENJAMIN D. LEVINE, and VASILIS Z. MARMARELIS. "NONLINEAR PHYSIOLOGICAL SYSTEMS IDENTIFICATION: APPLICATION TO CEREBRAL HEMODYNAMICS UNDER ORTHOSTATIC STRESS." In Proceedings of the Seventh International Workshop. WORLD SCIENTIFIC, 2006. http://dx.doi.org/10.1142/9789812773197_0035.

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Alvarado-Alvarez, N., S. Charleston-Villalobos, S. Reulecke, et al. "Time-Frequency Analysis of Cardiovascular Variability during an Orthostatic Stress by Complete EMD." In 2020 42nd Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC) in conjunction with the 43rd Annual Conference of the Canadian Medical and Biological Engineering Society. IEEE, 2020. http://dx.doi.org/10.1109/embc44109.2020.9176709.

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Verma, Ajay K., Amanmeet Garg, Andrew Blaber, Reza Fazel-Rezai, and Kouhyar Tavakolian. "Analysis of causal cardio-postural interaction under orthostatic stress using convergent cross mapping." In 2016 38th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2016. http://dx.doi.org/10.1109/embc.2016.7591194.

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Etter, Karen E., and M. Keith Sharp. "Modeling of Orthostatic Intolerance During Lower Body Negative Pressure." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19053.

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Postflight orthostatic intolerance (POI) afflicts a significant fraction of male astronauts and nearly all female astronauts when they first stand on Earth after spaceflight. Symptoms include dizziness and fainting, which can impact their abilities to perform critical tasks during the post-landing period. On the Moon or Mars, poor performance or accidents resulting from POI may have potentially catastrophic consequences due to the more hazardous conditions and lack of medical facilities. In addition, the long duration flights necessary to reach Mars may elicit adaptations that increase the ris
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Valente, Martina, Michal Javorka, Zuzana Turianikova, et al. "Cardiovascular and respiratory variability during orthostatic and mental stress: A comparison of entropy estimators." In 2017 39th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2017. http://dx.doi.org/10.1109/embc.2017.8037606.

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Johnny, Ottesen,. "On the Track of Syncope Induced by Orthostatic Stress - Feedback Mechanisms Regulating the Cardiovascular System." In Modeling and Control in Biomedical Systems, edited by Rees, Stephen, chair Andreassen, Steen and Andreassen, Steen. Elsevier, 2009. http://dx.doi.org/10.3182/20090812-3-dk-2006.00032.

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Blanco, Igor, Peyman Zirak, Ana Fortuna, et al. "The effect of obstructive sleep apnea on the cerebral blood flow response to orthostatic stress." In Biomedical Optics. OSA, 2014. http://dx.doi.org/10.1364/biomed.2014.bm3a.10.

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Reports on the topic "Orthostatic stress"

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Self, David A., Curtis D. White, Robert M. Shaffstall, Benjamin L. Mtinangi, and Jennifer S. Croft. Differences in Mechanism Between Syncope Resulting from Rapid Onset Acceleration and Orthostatic Stress. Defense Technical Information Center, 1996. http://dx.doi.org/10.21236/ada333371.

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