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Dissertations / Theses on the topic 'Orthostatic stress'

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1

Serrador, Jorge M. "Cerebral autoregulation during orthostatic stress." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape2/PQDD_0017/NQ58188.pdf.

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2

Howden, Reuben. "Tolerance to orthostatic stress and human cardiovascular control." Thesis, De Montfort University, 2002. http://hdl.handle.net/2086/4812.

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3

Heldt, Thomas 1972. "Computational models of cardiovascular response to orthostatic stress." Thesis, Massachusetts Institute of Technology, 2004. http://hdl.handle.net/1721.1/28761.

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Thesis (Ph. D.)--Harvard-MIT Division of Health Sciences and Technology, 2004.<br>Includes bibliographical references (p. 163-185).<br>The cardiovascular response to changes in posture has been the focus of numerous investigations in the past. Yet despite considerable, targeted experimental effort, the mechanisms underlying orthostatic intolerance (OI) following spaceflight remain elusive. The number of hypotheses still under consideration and the lack of a single unifying theory of the pathophysiology of spaceflight-induced OI testify to the difficulty of the problem. In this investigation, w
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4

Ramsey, Michael W., Bradley J. Behnke, Rhonda D. Prisby, and Michael D. Delp. "Aging Alters Regional Vascular Conductance and Arterial Pressure During Orthostatic Stress." Digital Commons @ East Tennessee State University, 2007. https://dc.etsu.edu/etsu-works/4098.

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5

Berry, Narelle Margaret, and narelle berry@unisa edu au. "Acute and long term interventions to assess the adaptability of the cardiovascular responses to orthostatic stress." RMIT University. Medical Sciences, 2006. http://adt.lib.rmit.edu.au/adt/public/adt-VIT20070228.123618.

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This thesis comprises of four experiments from which related but independent analyses were undertaken. The interventions employed were designed to investigate the effect of cardiovascular adaptation, both in the short and long term on the cardiovascular responses to orthostatic stress. The first study, described in Chapter 3, tested the hypothesis that the cardiovascular system (CVS) could adapt to repeated orthostatic challenges in a single session. 14 subjects were exposed to ten +75° head-up tilts (HUT) over 70 mins. Each tilt involved a 5 min supine period (SUPINE) followed b
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6

Diehl, Ursula Anne. "The role of the hydrostatic indifferent point in governing splachnic blood pooling during orthostatic stress." Thesis, University of Iowa, 2011. https://ir.uiowa.edu/etd/948.

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The response of the circulatory system to gravity and hydrostatic forces has been well studied, for example the hydrostatic indifferent point (the location at which pressure does not change with posture) of the venous system has been established to be an important determinant of orthostatic responses and it has been found to be located near the diaphragm. However, the role of the abdomen has been less researched; for example, it appears that the concept that the abdominal compartment may have its own hydrostatic indifferent point has been overlooked. The goal of the present study was to establ
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7

Pawelczyk, James A. (James Anthony). "Interactions between Carotid and Cardiopulmonary Baroreceptor Populations in Men with Varied Levels of Maximal Aerobic Power." Thesis, University of North Texas, 1989. https://digital.library.unt.edu/ark:/67531/metadc331205/.

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Reductions in baroreflex responsiveness have been thought to increase the prevalence of orthostatic hypotension in endurance trained athletes. To test this hypothesis, cardiovascular responses to orthostatic stress, cardiopulmonary and carotid baroreflex responsiveness, and the effect of cardiopulmonary receptor deactivation on carotid baroreflex responses were examined in 24 men categorized by maximal aerobic power (V02max) into one of three groups: high fit (HF, V0-2max=67.0±1.9 ml•kg^-1•min^-1), moderately fit (MF, V0-2max=50.9±1.4 ml•kg^-1•min^-1), and low fit (LF, V0-2max=38.9±1.5 ml•kg^-
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8

Stevens, Glen Harold John. "Blood Pressure Regulation During Simulated Orthostatism Prior to and Following Endurance Exercise Training." Thesis, University of North Texas, 1992. https://digital.library.unt.edu/ark:/67531/metadc277914/.

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Cardiovascular responses and tolerance to an orthostatic stress were examined in eight men before and after eight months of endurance exercise training. Following training, maximal oxygen consumption and blood volume were increased, and resting heart rate reduced. Orthostatic tolerance was reduced following training in all eight subjects. It was concluded that prolonged endurance training decreased orthostatic tolerance and this decrease in tolerance appeared associated with attenuated baroreflex sensitivity and alterations in autonomic balance secondary to an increased parasympathetic tone no
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9

Zhang, Qingguang. "HUMAN CARDIOVASCULAR RESPONSES TO SIMULATED PARTIAL GRAVITY AND A SHORT HYPERGRAVITY EXPOSURE." UKnowledge, 2015. http://uknowledge.uky.edu/cbme_etds/30.

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Orthostatic intolerance (OI), i.e., the inability to maintain stable arterial pressure during upright posture, is a major problem for astronauts after spaceflight. Therefore, one important goal of spaceflight-related research is the development of countermeasures to prevent post flight OI. Given the rarity and expense of spaceflight, countermeasure development requires ground-based simulations of partial gravity to induce appropriate orthostatic effects on the human body, and to test the efficacy of potential countermeasures. To test the efficacy of upright lower body positive pressure (LBPP)
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10

Samin, Azfar. "Neuronal modelling of baroreflex response to orthostatic stress." 2005. http://link.library.utoronto.ca/eir/EIRdetail.cfm?Resources__ID=232699&T=F.

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11

Mardimae, Melina May Alexandra. "The hemodynamic effects of negative inspiratory pressure during orthostatic stress." 2008. http://link.library.utoronto.ca/eir/EIRdetail.cfm?Resources__ID=772059&T=F.

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12

Lee, Joshua Floyd. "Mechanisms of heat stress- and obesity-induced reductions in orthostatic tolerance." Thesis, 2013. http://hdl.handle.net/2152/26848.

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These studies investigated 1) mechanisms underlying the well-established reduction in orthostatic tolerance (OT) that occurs in humans during heat stress (HS) relative to normothermia (NT) with particular focus on determining factors contributing to the high degree of inter-individual variability in this phenomenon; and 2) influence of obesity on OT, and mechanisms underlying reduced OT, should it exist. In Study #1, OT was assessed during lower body negative pressure (LBNP), and quantified with a cumulative stress index (CSI). Differences in CSI (CSIdiff) between thermal conditions were used
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13

Esch, Ben Thomas Alexander. "Pericardial-mediated diastolic ventricular interactions in endurance-trained athletes during orthostatic stress." Thesis, 2005. http://hdl.handle.net/2429/16735.

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Purpose: To assess diastolic ventricular interaction (DVI) and its consequences in endurance athletes and normally active individuals during lower body positive (LBPP) and negative pressure (LBNP). Methods: Eight male endurance athletes (VO₂ max 65.4 ± 5.7 mL•kg⁻¹•min⁻¹) and eight normally active individuals (VO₂ max 45.1 ± 6.0 mL•kg⁻¹•min⁻¹) underwent three experimental days: 1) assessment of VO₂ max 2) a negative orthostatic tolerance test, and 3) LBPP (0 to 60 mmHg) and LBNP (0 to -80 mmHg) during which time ventricular volumes were examined via echocardiography. Results: All nor
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14

Edgell, Heather. "Cardiovascular responses of women to orthostatic stress, the effects of the menstrual cycle and age, and a comparison to men." Thesis, 2010. http://hdl.handle.net/10012/5334.

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Young women are known to exhibit a greater incidence of orthostatic hypotension than men. The exact mechanisms for this are unclear and it has been proposed to be related to cardiac filling, peripheral resistance, and/or regional blood pooling. The sexually dimorphic effects of lower body negative pressure (LBNP) or upright posture were investigated throughout this study. Women could experience these changes due to effects of the sex hormones estrogen and progesterone. Chapters 3 and 4 in this thesis investigated the responses of women to LBNP in both the follicular and the luteal phase of the
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15

Lange, Andrew Peter. "Assessment of various mechanisms involved in heat-stress induced reductions in orthostatic tolerance." 2013. http://hdl.handle.net/2152/22733.

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Purpose: This study aimed to expand our knowledge of the underlying mechanisms of orthostatic tolerance. First, cerebral perfusion was compared with reductions in orthostatic tolerance between normal thermic and heated conditions. The researchers' hypothesized that subjects with the greatest reduction in orthostatic tolerance will experience the largest drop in cerebral blood flow. Additionally, ANG II was measured in order to identify if during passive heating, the elevation in plasma ANG II is negatively correlated with heat-stress induced reductions in orthostatic tolerance. Lastly, orthost
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16

Gagne, Nathalie. "Developing a Non-Invasive Method to Monitor Cardiovascular Control during Orthostatic Challenge Considering the Limitation of the FinometerTM." Thesis, 2009. http://hdl.handle.net/10012/4299.

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Sensations of dizziness or fainting (pre-syncope or syncope) on standing up from a lying or a seated position are usually associated with impaired blood pressure regulation leading to inadequate perfusion of the brain. The purpose of this project was to develop a simple method to provide scientists and doctors a convenient way to monitor cardiovascular control during orthostatic stress with the non-invasive FinometerTM device. This apparatus provides a continuous estimate of arterial blood pressure (BP) contour from the finger and computes brachial blood pressure contours (systolic (SBP) and d
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17

Beavers, Keith. "Investigating the efficacy of the NASA fluid loading protocol for astronauts: The role of hormonal blood volume regulation in orthostasis after bed rest." Thesis, 2009. http://hdl.handle.net/10012/4532.

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Despite years of research, the role that hypovolemia plays in orthostatic intolerance after head down bed rest (BR) and spaceflight remains unclear. Additionally, the efficacy of oral saline countermeasures, employed in an attempt to restore plasma volume (PV) after BR is questionable. Several previous studies have suggested that a new homeostatic set point is achieved in space or during BR, making attempts to restore PV temporary at best. We tested the hypotheses that one day of BR would induce a transient increase in PV followed by hypovolemia and new hormonal balance; that a salt tablet and
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