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Books on the topic 'Osteoarthritis Drugs'

Author: Grafiati

Published: 2 June 2025

Last updated: 31 July 2025

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1

Bales, Peter. Osteoarthritis: Preventing and healing without drugs. Prometheus Books, 2008.

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2

G, Lombardino J., ed. Nonsteroidal antiinflammatory drugs. Wiley, 1985.

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3

Brandt, Kenneth D. Diagnosis and nonsurgical management of osteoarthritis. 2nd ed. Professional Communications, Inc., 2000.

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4

1951-, Reid David, and Miller Colin G. 1960-, eds. Clinical trials in rheumatoid arthritis and osteoarthritis. Springer, 2008.

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5

L, Hardy Mary, Southern California Evidence-Based Practice Center/RAND., and United States. Agency for Healthcare Research and Quality., eds. S-adenosyl-L-methionine for treatment of depression, osteoarthritis, and liver disease. Agency for Healthcare Research and Quality, 2002.

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6

R, Cook Allan, ed. Arthritis sourcebook: Basic information about specific forms of arthritis and related disorders including rheumatoid arthritis, osteoarthritis, gout, polymyalgia rheumatica, psoriatic arthritis, spondyloarthropathies, juvenile rheumatoid arthritis, and juvenile ankylosing spondylitis along with treatment options from over-the-counter and prescription drugs to surgery and alternative measures and coping strategies to ease pain, fatigue, and stress. Omnigraphics, 1998.

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7

Maetzel, Andreas. The cost-effectiveness of celecoxib and rofecoxib in patients with osteoarthritis or rheumatoid arthritis. Canadian Coordinating Office for Health Technology Assessment, 2002.

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8

Brenda, Adderly, and Fox Barry, eds. The arthritis cure: The medical breakthrough that can halt, reverse, and even cure osteoarthritis. Century, 1997.

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9

Brenda, Adderly, and Fox Barry, eds. The arthritis cure: The medical miracle that can halt, reverse, and may even cure osteoarthritis. St. Martin's Press, 1997.

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10

Brenda, Adderly, and Fox Barry, eds. The arthritis cure: The medical miracle that can halt, reverse, and may even cure osteoarthritis. St. Martin's Paperbacks, 1997.

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11

Theodosakis, Jason. The arthritis cure: The medical miracle that can halt, reverse, and may even cure osteoarthritis. St. Martin's Press, 2004.

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12

Theodosakis, Jason. The arthritis cure: The medical miracle that can halt, reverse, and may even cure osteoarthritis. Wheeler Pub., 1997.

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13

W, Moskowitz Roland, Hirohata Kazushi, and SEAPAL Congress of Rheumatology (6th : 1988 : Tokyo, Japan), eds. Diclofenac (Voltaren) and cartilage in osteoarthritis: An international symposium held during the 6th SEAPAL Congress of Rheumatology, Tokyo, Japan, September 1988. Hogrefe & Huber Publishers, 1990.

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14

1920-, Muir Helen, Hirohata Kazushi, Shichikawa Kanji, and SEAPAL Congress of Rheumatology (6th : 1988 : Tokyo, Japan), eds. Mechanisms of articular cartilage damage and repair in osteoarthritis: An international symposium held during the 6th SEAPAL Congress of Rheumatology, Tokyo, Japan, September 1988. Hogrefe & Huber, 1990.

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15

Brenda, Adderly, and Fox Barry, eds. Guan jie yen liao fa: The Arthritis cure. Di teng chu ban tu shu yu xian gong si, 1998.

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16

Theodosakis, Jason. The arthritis cure. St. Martin's Paperbacks, 2004.

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17

Bales, Peter. Osteoarthritis: Preventing and Healing Without Drugs. Prometheus Books, 2008.

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18

Wenham, Claire Y. J., and Philip G. Conaghan. Osteoarthritis—management. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0140.

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Osteoarthritis (OA) is a common condition which often causes pain and functional limitation, significantly impacting on a person's quality of life. A comprehensive assessment of the impact of OA should be performed before selecting therapies and treatment goals. Current recommended therapies include a combination of pharmacological and non-pharmacological therapies, which should be considered for all people with OA, regardless of anatomical site of involvement. Non-pharmacological treatments include education, muscle strengthening and aerobic exercises, weight loss if appropriate, splints and

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19

Simon, Lee S., and Marc C. Hochberg. Non-steroidal anti-inflammatory drugs. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0030.

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Non-steroidal anti-inflammatory drugs (NSAIDs) are a chemically diverse group of compounds that share three cardinal characteristics: they are anti-inflammatory, analgesic, and antipyretic. They are approved by regulatory authorities for the treatment of patients with osteoarthritis, rheumatoid arthritis, ankylosing spondylitis, acute gout, and some forms of juvenile idiopathic arthritis. There are at least 20 chemically different NSAIDs currently available in Europe and the United States. These include not only the ‘traditional’ non-selective cyclooxygenase (COX) inhibitors that inhibit both

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20

Bennell, Kim L., Ans Van Ginckel, Fiona Dobson, and Rana S. Hinman. Exercise for the person with osteoarthritis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0022.

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Because of its beneficial effects on pain and physical dysfunction commonly reported by afflicted individuals, all clinical guidelines of osteoarthritis (OA) advocate exercise therapy as a vital component of conservative management strategies. Although the optimal exercise modalities in terms of dosage, exercise type, or delivery mode are not yet known, clinical benefits can be achieved with a wide range of exercise types. While treatment effect sizes may be considered small to moderate, they are similar to those of common analgesic drugs or oral non-steroidal anti-inflammatories but seem to e

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21

McDougall, Jason J., and Joel A. Vilensky. The innervation of the joint and its role in osteoarthritis pain. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0007.

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Diarthrodial joints possess an extensive network of sensory and sympathetic nerve fibres whose physiological functions are varied and complex. Nerves are primarily located in the synovium but also innervate the subchondral bone, the outer third of menisci, and the superficial surface of tendons and ligaments. Large-diameter, myelinated neurons are involved in joint position sense while small-diameter neurons with thin or no myelin typically sense pain. The small-diameter nerves in conjunction with sympathetic fibres control synovial blood flow and maintain joint homeostasis. In patients with o

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22

Michet, Clement J., Kenneth G. Moder, and William W. Ginsburg. Rheumatology. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199755691.003.0681.

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Major rheumatologic diseases are reviewed in detail, including symptoms, diagnosis, and treatment. Rheumatoid arthritis, Sjogren syndrome, Felty syndrome, osteoarthritis, fibromyalgia, and vasculitic syndromes like giant cell arteritis, polyarteritis nodosa, Takayasu arteritis, and Wegener granulomatosis are included. Drugs used to treat rheumatic disease are also highlighted. They include NSAIDs, antimalarials, and glucocorticosteroids. Crystalline arthropathies, such as hyperuricemia and gout, are another class of rheumatologic disease, as are spondyloarthropathies, systemic lupus erythemato

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23

Stengler, Mark, James Balch, Balch James F. Jr, and Robin Young-Balch. Osteoarthritis and Osteoporosis Drug Alternatives. Wiley & Sons, Incorporated, John, 2011.

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24

Stengler, Mark, James Balch, Balch James F. Jr, and Robin Young-Balch. Osteoarthritis and Osteoporosis Drug Alternatives. Wiley & Sons, Incorporated, John, 2011.

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25

Roddy, Edward, and Michael Doherty. Calcium pyrophosphate crystal deposition (CPPD). Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0142.

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Calcium pyrophosphate crystal deposition (CPPD) in articular cartilage is a common age-related phenomenon. Recent important advances in our understanding of the pathophysiology of pyrophosphate metabolism include the identification of a mutation within the ANK gene which associates with familial CPPD, and elucidation of the interleukin-1β‎ (IL-1β‎)-dependent mechanisms by which crystals invoke an inflammatory response. Risk factors for CPPD include age, prior joint damage and osteoarthritis, genetic factors, and occasionally metabolic diseases (hyperparathyroidism, haemochromatosis, hypomagnes

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26

Stengler, Mark, James F. Balch, and Robin Young-Balch. AARP Osteoarthritis and Osteoporosis Drug Alternatives. Wiley & Sons, Incorporated, John, 2011.

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27

Platt, Philip, and Ismael Atchia. Injection therapy. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0087.

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Joint and soft tissue injections with glucocorticoids and other agents remain a critical aspect of the management of musculoskeletal conditions. Injection therapy has previously consisted mainly of glucocorticoid and local anaesthetic, but other agents such as hyaluronic acid, radioactive agents, plasma-rich products, and biologics have also been introduced in the practice of musculoskeletal clinicians. Overall glucocorticoid injection remains the most widely performed procedure, and is an effective treatment for an inflamed joint or soft tissue. This procedure has been widely used for at leas

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28

Abhishek, Abhishek, Adrian Jones, and Michael Doherty. Topical pharmacological treatments. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0028.

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Topical pharmacological agents such as non-steroidal anti-inflammatory drugs (NSAIDs) and capsaicin are widely recommended as first-line analgesics in the treatment of osteoarthritis (OA) of the knee, hand, and potentially other peripheral joints in view of their safety and efficacy. Although initial studies were short in duration (2–4 weeks), recent randomized controlled trials have confirmed the efficacy of topical NSAIDs over longer (12-week) study periods. Systematic reviews demonstrate that their efficacy can be equivalent to oral NSAIDs for OA pain, but they have a significantly better s

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29

Clunie, Gavin P. R., Nick Wilkinson, Elena Nikiphorou, and Deepak Jadon, eds. Oxford Handbook of Rheumatology. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198728252.001.0001.

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The Oxford Handbook of Rheumatology, 4th edition, has been expanded and improved to incorporate paediatric and adolescent rheumatology. The format of the book is retained. The first four chapters offer a pragmatic guide to evaluating rheumatic and musculoskeletal diseases, showing how a differential diagnosis can be formed on the basis of symptoms, examination, and investigation findings, both for regional musculoskeletal and systemic generalized conditions. Part II comprises chapters on all the major rheumatic and bone diseases and autoimmune connective tissue diseases, such as rheumatoid art

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30

Brandt, Kenneth D. Diagnosis And Nonsurgical Management of Osteoarthritis. 4th ed. Professional Communications, Inc., 2005.

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31

The arthritis handbook: The essential guide to a pain-free, drug-free life. DiaMedica, 2008.

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32

Doherty, Michael, Johannes Bijlsma, Nigel Arden, David J. Hunter, and Nicola Dalbeth. Introduction: the comprehensive approach. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0020.

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This introductory chapter to the section on management of osteoarthritis (OA) emphasizes the need for a full assessment of the patient, not just in terms of joint symptoms and examination findings but a full holistic assessment of the person, including the impact of OA on their life, their illness perceptions of OA, and the presence of comorbidities. An individualized package of care can then be developed. Patients should be fully informed about OA and fully involved in all management decisions. Apart from education, which is an ongoing not one-off process, other core treatments to be consider

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33

Zhang, Weiya, and Michael Doherty. Guidelines. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0037.

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A number of treatment guidelines have been developed to optimize the treatment of osteoarthritis, some of which were recently updated. Fifty-one non-pharmacological, pharmacological, and surgical treatments are addressed in these guidelines but only two (oral opioid and intra-articular steroid injection) reach the minimal clinically important difference above placebo. Recommendations for these treatments vary depending on joint sites, risk:benefit ratio, and population. Exercise, self-management, and weight reduction if obese are universally recommended. While topical non-steroidal anti-inflam

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34

Kapoor, Mohit, and Nizar N. Mahomed. Osteoarthritis: Pathogenesis, Diagnosis, Available Treatments, Drug Safety, Regenerative and Precision Medicine. Adis, 2016.

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35

Kapoor, Mohit, and Nizar N. Mahomed. Osteoarthritis: Pathogenesis, Diagnosis, Available Treatments, Drug Safety, Regenerative and Precision Medicine. Adis International, Limited, 2015.

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36

Kapoor, Mohit, and Nizar N. Mahomed. Osteoarthritis: Pathogenesis, Diagnosis, Available Treatments, Drug Safety, Regenerative and Precision Medicine. Adis, 2015.

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37

Grifka, J. Osteoarthritis: Fundamentals and Strategies for Joint-Preserving Treatment. Edited by J. Grifka. SPRINGER-VERLAG, 2000.

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38

Clinical Trials in Rheumatoid Arthritis and Osteoarthritis (Clinical Trials). Springer, 2008.

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39

Zoe, Anne. My Problem and How I Solved It : Osteoarthritis: Drug-Free Relief from OA Pain. Independent Publisher, 2022.

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40

Bannwarth, Bernard, and Francis Berenbaum. Systemic analgesics (including paracetamol and opioids). Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0029.

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Apart from non-steroidal anti-inflammatory drugs (NSAIDs), there are only two categories of systemic analgesics, namely paracetamol (acetaminophen) and opioids, that are currently available worldwide for clinical use. Paracetamol is poorly effective in relieving pain and improving function in patients with symptomatic osteoarthritis (OA). Furthermore, its safety profile is less favourable than classically thought. In fact, there is evidence paracetamol acts as a weak inhibitor of the cyclooxygenase enzymes. Given that paracetamol poses a lower risk of severe adverse events than NSAIDs while be

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41

Stengler, Mark, James F. Balch, Balch James F. Jr, Robin Young-Balch, and Robin Young-Balch. AARP Osteoarthritis and Osteoporosis Drug Alternatives: All-Natural Options for Better Health Without the Side Effects. Wiley & Sons, Incorporated, John, 2011.

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42

Merkesdal, Sonja, and Wilfried Mau. Health economics. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0031.

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The economic burden of rheumatic diseases for society, various payers, and last but not least the individual patient has been increasingly recognized. In addition to the well-known impact of back pain and osteoarthritis, the upcoming new and expensive therapies have made this issue especially intriguing in the treatment of rheumatoid arthritis (RA). A mean international estimate of the total annual costs of RA, mainly consisting of direct resource consumption and indirect costs due to productivity losses relating to paid work, comes to about €5600. Other inflammatory rheumatologic diseases (an

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43

Buff, Sheila, and Jason Theodosakis. Arthritis Cure: The Medical Miracle That Can Halt, Reverse, and May Even Cure Osteoarthritis. St. Martin's Press, 2004.

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44

(Editor), R. W. Moskowitz, and K. Hirohata (Editor), eds. Diclofenac ( Voltaren ) and Ca. Hogrefe & Huber Publishers, 1991.

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45

Cheng, Jerry, and David Madigan. Bayesian approaches to aspects of the Vioxx trials: Non-ignorable dropout and sequential meta-analysis. Edited by Anthony O'Hagan and Mike West. Oxford University Press, 2018. http://dx.doi.org/10.1093/oxfordhb/9780198703174.013.3.

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This article discusses Bayesian approaches to aspects of the Vioxx trials study, with a focus on non-ignorable dropout and sequential meta-analysis. It first provides a background on Vioxx, a COX-2 selective, non-steroidal anti-inflammatory drug (NSAID) approved by the FDA in May 1999 for the relief of the signs and symptoms of osteoarthritis, the management of acute pain in adults, and for the treatment of menstrual symptoms. However, Vioxx was found to cause an array of cardiovascular side-effects such as myocardial infarction, stroke, and unstable angina. As a result, Vioxx was withdrawn fr

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46

Buff, Sheila, and Jason Theodosakis. The Arthritis Cure, Revised and Updated: The Medical Miracle That Can Halt, Reverse, And May Even Cure Osteoarthritis. St. Martin's Paperbacks, 2003.

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47

Abhishek, Abhishek, and Michael Doherty. Treatment of calcium pyrophosphate deposition. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0052.

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The treatment of calcium pyrophosphate crystal deposition (CPPD) is mainly symptomatic. Acute calcium pyrophosphate (CPP) crystal synovitis should be treated with rest, local application of ice packs, joint aspiration, and/or intra-articular corticosteroid injection (once joint sepsis has been excluded). Oral colchicine or prednisolone may be used if joint aspiration and/or injection are not feasible. Anti-inflammatory agents (with proton pump inhibitors) may be used but in general these should be avoided as most patients with acute CPP crystal arthritis are elderly, and at a high risk of gast

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48

Abhishek, Abhishek, and Michael Doherty. Placebo, nocebo, and contextual effects. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0027.

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Placebo effect is an example of ‘contextual’ effect and is the symptomatic improvement experienced by patients who have unknowingly received a placebo (inactive treatment) instead of an active drug. It occurs due to patient-specific factors such as expectation of improvement and is influenced by the context in which the treatment is delivered. Nocebo effect is the opposite of placebo effect and includes worsening of symptoms or incident adverse effects due to expectancy or negative contextual or practitioner influence. Placebo effect has been demonstrated in a range of musculoskeletal conditio

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49

Adderly, Brenda, Barry Fox, and Jason Theodosakis. The Arthritis Cure. Century, 1997.

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