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Relevant bibliographies by topics / Osteoclast

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Academic literature on the topic 'Osteoclast'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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Journal articles on the topic "Osteoclast"

1

Yu, Anna Xiao-Dan, Jian Xiao, Shi-Zheng Zhao, et al. "Biological Evaluation and Transcriptomic Analysis of Corylin as an Inhibitor of Osteoclast Differentiation." International Journal of Molecular Sciences 22, no. 7 (2021): 3540. http://dx.doi.org/10.3390/ijms22073540.

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Corylin, a flavonoid isolated from the fruit of Psoralea corylifolia, has an osteogenic effect on osteoblasts in vitro and bone micromass ex vivo. However, the effect and mechanism of corylin in regulating osteoclastogenesis remain unknown. By using murine bone marrow macrophages as the osteoclast precursor, corylin was found to inhibit the receptor activator of nuclear factor (NF) κB ligand (RANKL)-induced osteoclast differentiation via down-regulating osteoclastic marker genes. In parallel, F-actin formation and osteoclast migration were diminished in corylin-treated cultured osteoclasts, an

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Alatalo, Sari L., Jussi M. Halleen, Teuvo A. Hentunen, Jukka Mönkkönen, and H. Kalervo Väänänen. "Rapid Screening Method for Osteoclast Differentiation in Vitro That Measures Tartrate-resistant Acid Phosphatase 5b Activity Secreted into the Culture Medium." Clinical Chemistry 46, no. 11 (2000): 1751–54. http://dx.doi.org/10.1093/clinchem/46.11.1751.

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Abstract Background: Osteoclasts secrete tartrate-resistant acid phosphatase (TRAP; EC 3.1.3.2) 5b into the circulation. We studied the release of TRAP 5b from osteoclasts using a mouse in vitro osteoclast differentiation assay. Methods: We developed and characterized a polyclonal antiserum in rabbits, using purified human osteoclastic TRAP 5b as antigen. The antiserum was specific for TRAP in Western analysis of mouse osteoclast culture medium and was used to develop an immunoassay. We cultured mouse bone marrow-derived osteoclast precursor cells for 3–7 days with or without clodronate in the

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Combs, Charlotte E., Karen Fuller, Hashethra Kumar, et al. "Urocortin is a novel regulator of osteoclast differentiation and function through inhibition of a canonical transient receptor potential 1-like cation channel." Journal of Endocrinology 212, no. 2 (2011): 187–97. http://dx.doi.org/10.1530/joe-11-0254.

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This study investigated the role of urocortin (UCN), a member of the corticotrophin-releasing factor (CRF) family of peptides, in osteoclast maturation and function. We found that 10−7 M UCN significantly (P<0.05) suppressed osteoclast differentiation from bone marrow precursor cells in culture and reduced the expression of several osteoclastic markers. Furthermore, UCN potently suppressed osteoclast bone resorption, by significantly inhibiting both the plan area of bone resorbed by osteoclasts and actin ring formation within osteoclasts at 10−9 M (P<0.05), with complete inhibition at 10

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Fong, E. L. S., E. L. Prabha, and T. Carney. "POS0348 DEVELOPING A WHOLE MOUNT FLUORESCENT OSTEOCLAST ACTIVITY ASSAY USING THE ELF97 PHOSPHATASE SUBSTRATE TO VISUALISE AND QUANTIFY IN SITU OSTEOCLAST ACTIVITY IN ZEBRAFISH (DANIO RERIO)." Annals of the Rheumatic Diseases 81, Suppl 1 (2022): 427.3–428. http://dx.doi.org/10.1136/annrheumdis-2022-eular.5402.

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BackgroundOsteoporosis is a mineral bone disease arising from the predominance of osteoclastic bone resorption. Bisphosphonates which inhibit osteoclasts are commonly used in osteoporosis treatment, but are not without severe adverse effects like osteonecrosis of the jaw. The mechanisms behind the development of such phenomena is not well understood. Bone homeostasis is achieved through an intimate cross-talk between osteoclasts and osteoblasts. Thus, it is important to visualise activities of these cells simultaneously in situ. Currently, there are means to visualise osteoclast shape and numb

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Niida, Shumpei, Masato Kaku, Hitoshi Amano, et al. "Vascular Endothelial Growth Factor Can Substitute for Macrophage Colony-Stimulating Factor in the Support of Osteoclastic Bone Resorption." Journal of Experimental Medicine 190, no. 2 (1999): 293–98. http://dx.doi.org/10.1084/jem.190.2.293.

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We demonstrated previously that a single injection of recombinant human macrophage colony-stimulating factor (rhM-CSF) is sufficient for osteoclast recruitment and survival in osteopetrotic (op/op) mice with a deficiency in osteoclasts resulting from a mutation in M-CSF gene. In this study, we show that a single injection of recombinant human vascular endothelial growth factor (rhVEGF) can similarly induce osteoclast recruitment in op/op mice. Osteoclasts predominantly expressed VEGF receptor 1 (VEGFR-1), and activity of recombinant human placenta growth factor 1 on osteoclast recruitment was

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Moreaux, Jerome, Dirk Hose, Alboukadel Kassambara, et al. "Osteoclast-gene expression profiling reveals osteoclast-derived CCR2 chemokines promoting myeloma cell migration." Blood 117, no. 4 (2011): 1280–90. http://dx.doi.org/10.1182/blood-2010-04-279760.

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Abstract Multiple myeloma is characterized by the clonal expansion of malignant plasma cells (multiple myeloma cells [MMCs]), in the bone marrow. Osteolytic bone lesions are detected in 80% of patients because of increased osteoclastic bone resorption and reduced osteoblastic bone formation. MMCs are found closely associated with sites of increased bone resorption. Osteoclasts strongly support MMC survival in vitro. To further elucidate the mechanisms involved in osteoclast/MMC interaction, we have identified 552 genes overexpressed in osteoclasts compared with other bone marrow cell subpopula

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Fuller, K., J. M. Owens, and T. J. Chambers. "Macrophage inflammatory protein-1 alpha and IL-8 stimulate the motility but suppress the resorption of isolated rat osteoclasts." Journal of Immunology 154, no. 11 (1995): 6065–72. http://dx.doi.org/10.4049/jimmunol.154.11.6065.

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Abstract Cells of the osteoblastic lineage play a major role in the regulation of osteoclastic bone resorption. Recent studies have demonstrated production of chemokines by osteoblastic cells. Although these phagocyte-stimulating and proinflammatory cytokines act as chemoattractants and activators for other members of the hemopoietic lineage, their actions on osteoclasts have not been characterized. We found that macrophage inflammatory protein-1 alpha (MIP-1 alpha) and IL-8 inhibited bone resorption by rat osteoclasts, primarily through reduction in the proportion of osteoclasts resorbing bon

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Perkins, S. L., and S. J. Kling. "Local concentrations of macrophage colony-stimulating factor mediate osteoclastic differentiation." American Journal of Physiology-Endocrinology and Metabolism 269, no. 6 (1995): E1024—E1030. http://dx.doi.org/10.1152/ajpendo.1995.269.6.e1024.

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Macrophage colony-stimulating factor (M-CSF) is essential for differentiation of osteoclasts and macrophages from a common bone marrow precursor. Using ST-2 stromal cell/murine bone marrow coculture, we studied the effects of increasing amounts of M-CSF on differentiation of macrophages and osteoclasts. Addition of exogenous M-CSF caused a dose-dependent 98% decrease in tartrate-resistant acid phosphatase (TRAP)-positive multinucleated cells, accompanied by a 2.5-fold increase in nonspecific esterase-staining macrophages. Similar decrease in osteoclastic functional activity, including 125I-lab

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Cheng, Yin, Haixia Liu, Jing Li, et al. "Evaluation of culture conditions for osteoclastogenesis in RAW264.7 cells." PLOS ONE 17, no. 11 (2022): e0277871. http://dx.doi.org/10.1371/journal.pone.0277871.

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Osteoclasts are the only multinucleated cells in vivo responsible for bone resorption and are vital for regulating bone remodeling and maintaining bone mass. The RAW264.7 cell line is widely used to study osteoclastic differentiation and biological molecular mechanism. However, protocols for inducing osteoclast formation in RAW264.7 cells vary considerably between laboratories, hindering the replication of results. Therefore, we tested the influence of culture conditions on osteoclast differentiation, including cell density and receptor activator of nuclear factor kappa-B ligand (RANKL) concen

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Kameda, Takashi, Hiroshi Mano, Tatsuhisa Yuasa, et al. "Estrogen Inhibits Bone Resorption by Directly Inducing Apoptosis of the Bone-resorbing Osteoclasts." Journal of Experimental Medicine 186, no. 4 (1997): 489–95. http://dx.doi.org/10.1084/jem.186.4.489.

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Estrogen deficiency causes bone loss, which can be prevented by estrogen replacement therapy. Using a recently developed technique for isolation of highly purified mammalian osteoclasts, we showed that 17 β-estradiol (E2) was able to directly inhibit osteoclastic bone resorption. At concentrations effective for inhibiting bone resorption, E2 also directly induced osteoclast apoptosis in a dose- and time-dependent manner. ICI164,384 and tamoxifen, as pure and partial antagonists, respectively, completely or partially blocked the effect of E2 on both inhibition of osteoclastic bone resorption an

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Dissertations / Theses on the topic "Osteoclast"

1

O'Brien, Elizabeth Ann. "Regulation of osteoclast activity : differential adhesion of osteoclasts to the bone surface." Thesis, University of Liverpool, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.343930.

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Stephens, Sebastien. "Novel Osteoclast Signalling." Thesis, Griffith University, 2010. http://hdl.handle.net/10072/365823.

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When you say osteoporosis, people think of their grandma’s brittle bones, but scientists think of the osteoclast. When you say cancer, people think of death, but before this, many succumb to the osteoclast. The fact is all these things are true yet it is even truer to say that each disease in fact also has more of the other – osteoporosis and death, and cancer and brittle bones. However, the commonality is undeniably the osteoclast. Scratching the surface of the osteoclast reveals that it is the basis of a diversity of bone-related disorders yet the osteoclast itself is, even given the large a

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Wilkinson, Debbie Isabelle. "Visualisation of osteoclast membrane domains." Thesis, University of Aberdeen, 2010. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=158808.

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Osteoclasts polarise upon activation and form four distinct membrane domains; the basolateral domain, the sealing zone, the functional secretory domain and the ruffled border. The ruffled border is the resorptive organelle of the cell and provides a large surface area for the release of protons and enzymes into the space beneath the osteoclast. Defects in osteoclast formation or function can lead to diseases such as osteopetrosis. Ruffled border formation is a critical event in osteoclast function but the process by which it and other membrane domains form is only partially understood. Vesicul

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Zaidi, Mone. "Novel mechanisms of osteoclast regulation." Thesis, University of London, 1991. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.411434.

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Gu, Xiaomei Everett Eric T. "Physiopathology of osteoclast in bone." Chapel Hill, N.C. : University of North Carolina at Chapel Hill, 2008. http://dc.lib.unc.edu/u?/etd,1870.

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Thesis (M.S.)--University of North Carolina at Chapel Hill, 2008.<br>Title from electronic title page (viewed Dec. 11, 2008). "... in partial fulfillment of the requirements for the degree of Master of Science in the Curriculum of Oral Biology." Discipline: Oral Biology; Department/School: Dentistry.

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Barnes, Calvin Langston Toure. "C-mpl Expression in Osteoclast Progenitors: A Novel Role for Thrombopoietin in Regulating Osteoclast Development." Yale University, 2006. http://ymtdl.med.yale.edu/theses/available/etd-06262006-123750/.

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A new paradigm has evolved in which multiple regulatory interactions between the skeletal and hematopoietic systems have been identified. Previous studies have demonstrated that megakaryocytes (MK) play a dual role in skeletal homeostasis by stimulating osteoblast proliferation and simultaneously inhibiting osteoclast (OC) development. Here we identify a novel regulatory pathway in which the main MK growth factor, thrombopoietin (TPO), directly regulates osteoclastogenesis. To study the role of TPO in OC development, spleen or bone marrow (BM) cells (2x10[exponent]6 cells/ml) or BM macrophages

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Hale, Annette Julie. "The characterisation of the Pagetic osteoclast." Thesis, University of Nottingham, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.359919.

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Lång, Johanna. "CCL11 and Effects on Pre-osteoclast Migration." Thesis, Umeå universitet, Institutionen för odontologi, 2017. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-143797.

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ABSTRACT Periodontitis is a chronic inflammatory disease due to dental bacteria, and the disease is highly prevalent worldwide. Both environmental factors and genetic variation are confounding factors. Characteristic for disease development is degradation of gingival tissue and resorption of the alveolar bone due to inflammation. The cells that are capable to resorb bone is named osteoclasts and those are recruited and activated by numerous cytokines. Cytokines are small signal proteins responsible for cell communication and cell recruitment. Cytokines with chemotactic capacity are called che

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Combs, Charlotte Emma. "The role of urocortin in osteoclast physiology." Thesis, St George's, University of London, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.517193.

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MacQuarrie, Robyn Melanie. "Arthroplasty-derived wear particles effect osteoclast differentiation." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2001. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp05/MQ63539.pdf.

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Books on the topic "Osteoclast"

1

Holt, Ian. Control of osteoclast activity. University of Manchester, 1996.

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R, Rifkin Barry, and Gay Carol V, eds. Biology and physiology of the osteoclast. CRC Press, 1992.

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Mattsson, Jan P. The osteoclast H⁺-ATPase: Isolation and initial characterization. Department of Biochemistry and Biophysics, Department of Cell Biology, University of Göteborg and Chalmers University of Technology, 1995.

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4

Green, Philip Macey. An experimental study of the osteoclast and its role in bone remodelling. University of Birmingham, 1988.

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5

Tai, Victoria. The effects of leukotriene Bb4s on osteoclast formation and osteoclastic bone resorption and the role of osteoblastic cells in these processes. National Library of Canada = Bibliothèque nationale du Canada, 1999.

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6

Sutton, Michael Mark. The Influence of Microtubules and Microtubule-Based Structures on Osteoclast and CD4+ T Cell Function. [publisher not identified], 2022.

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Maria, Bijvoet Olav Leonardus, Lipton Allan, and International Cancer Congress (15th : 1990 : Hamburg, Germany), eds. Osteoclast inhibition in the management of malignancy-related bone disorders: An international symposium held during the 15th International Cancer Congress, Hamburg, Germany, August 1990. Hogrefe & Huber, 1993.

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D, Rubens R., and European Conference on Clinical Oncology (5th : 1989 : London, England), eds. The Management of bone metastases and hypercalcaemia by osteoclast inhibition: An international symposium held during the 5th European Conference on Clinical Oncology (ECCO 5), London, September 1989. Hogrefe & Huber, 1990.

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9

Yu, Hesheng. P2 purinoceptor-linked Ca2+ signaling and pH changes in osteoclasts. University of Toronto, Faculty of Dentistry], 1996.

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10

Rodionova, N. V. Funkt͡s︡ionalʹnai͡a︡ morfologii͡a︡ kletok v osteogeneze. Nauk. dumka, 1989.

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Book chapters on the topic "Osteoclast"

1

Pavelka, Margit, and Jürgen Roth. "Osteoclast." In Functional Ultrastructure. Springer Vienna, 2010. http://dx.doi.org/10.1007/978-3-211-99390-3_153.

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Schwab, Manfred. "Osteoclast." In Encyclopedia of Cancer. Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-27841-9_4277-2.

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Paiwal, Kapil, Niranzena Panneer Selvam, Abrar Ahmed Alamoudi, Jitin Makker, and Anirudha Agnihotry. "Osteoclast." In Handbook of Oral and Maxillofacial Giant Cell Lesions. Springer Nature Singapore, 2024. https://doi.org/10.1007/978-981-97-2863-3_4.

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Crockett, Julie C., David J. Mellis, and Adam Taylor. "Transfection of Osteoclasts and Osteoclast Precursors." In Methods in Molecular Biology. Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61779-415-5_14.

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Reddy, Sakamuri V., and G. David Roodman. "Osteoclast Differentiation." In The Skeleton. Humana Press, 2004. http://dx.doi.org/10.1007/978-1-59259-736-9_14.

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Krstić, R. V. "Knochengewebe. Osteoclast." In Die Gewebe des Menschen und der Säugetiere. Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-61380-7_111.

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Chambers, T. J. "The Osteoclast." In Handbook of Experimental Pharmacology. Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/978-3-642-55742-2_19.

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Krstić, Radivoj V. "Bony Tissue. Osteoclast." In General Histology of the Mammal. Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-70420-8_111.

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Orriss, Isabel R., and Timothy R. Arnett. "Rodent Osteoclast Cultures." In Methods in Molecular Biology. Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61779-415-5_8.

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Yang, Jingxuan, Xiaohong Bi, and Min Li. "Osteoclast Differentiation Assay." In Methods in Molecular Biology. Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-8879-2_12.

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Conference papers on the topic "Osteoclast"

1

Ravi, Vilupanur, Wilyson Wantah, Steven Castenada, et al. "Corrosion and Immune Response of Boron-Modified Titanium Alloys in Biological Environments." In CORROSION 2009. NACE International, 2009. https://doi.org/10.5006/c2009-09466.

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Abstract Titanium alloys containing boron additions have shown remarkable improvements in processing and mechanical behavior. Significant reductions in grain size are observed even with trace additions of boron (&lt; 0.1 wt% B) leading to benefits in processing. When the boron levels are increased to modest levels (~ 1 wt% B), the stiffness and strength of the Ti-6 Al-4 V base alloy increase by 20 – 30% with no loss in ductility. Keeping these advantages in mind, the aqueous corrosion behavior of these unique materials was investigated in this study to establish a baseline for performance

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Reddy Manne, Sai Kumar, Brendan Martin, Tyler Roy, et al. "NOISe: Nuclei-Aware Osteoclast Instance Segmentation for Mouse-to-Human Domain Transfer." In 2024 IEEE/CVF Conference on Computer Vision and Pattern Recognition Workshops (CVPRW). IEEE, 2024. http://dx.doi.org/10.1109/cvprw63382.2024.00686.

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Ravi, Vilupanur A., Roviden Enriquez, Carlos Beecher, et al. "Stability of Advanced Titanium Alloys in Saline Solution and Characterization of Osteoblast Activation." In CORROSION 2012. NACE International, 2012. https://doi.org/10.5006/c2012-01709.

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Abstract Electrochemical characterization of titanium alloys with different levels of boron was carried out in phosphate buffered saline (PBS) at body temperature and in 0.9 wt% sodium chloride (saline) at room temperature. Two types of cyclic potentiodynamic polarization tests were conducted - one based on ASTM F 2129-08 and the other with a broader voltage scan with higher peak potentials that further distinguished the behavior of the different titanium alloys. Break-down (pitting) potentials for the alloys in the latter test were in the 4.9 – 6.5 V range. The susceptibility of osteoblast ce

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McCann, Kristen, Travis Voorhees, Gamer Margoosian, Janice Miguel, and Vilupanur Ravi. "Electrochemical Evaluation of Titanium-Boron Alloys for Potential Biomedical Applications." In CORROSION 2016. NACE International, 2016. https://doi.org/10.5006/c2016-07842.

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Abstract Structural biomedical implant alloys are predominantly titanium-based with UNS R56400 (Ti-6Al-4V; Ti64) being the most commonly used. These alloys demonstrate excellent corrosion resistance and biocompatibility; however, there is an ongoing need to have longer lasting implants given the rapid increase in the longevity of the world’s population. A critical issue concerning the durability of the implants is aseptic loosening, a phenomenon initiated by the release of metallic cations from the alloy that alters the equilibrium between osteoclasts (bone-consuming cells) and osteoblasts (bo

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Daydar, Akshay, Alik Pramanick, Arijit Sur, and Subramani Kanagaraj. "MedCAM-OsteoCls: Medical Context Aware Multimodal Classification of Knee Osteoarthritis." In ICASSP 2025 - 2025 IEEE International Conference on Acoustics, Speech and Signal Processing (ICASSP). IEEE, 2025. https://doi.org/10.1109/icassp49660.2025.10889060.

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Xuan, W., X. Feng, Y. Shi, F. Wang, M. Zhang, and W. Tan. "THU0066 Osteoclast differentiation gene expression profiling reveals ccl4 mediates rankl-induced osteoclast migration." In Annual European Congress of Rheumatology, 14–17 June, 2017. BMJ Publishing Group Ltd and European League Against Rheumatism, 2017. http://dx.doi.org/10.1136/annrheumdis-2017-eular.4255.

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Maccataio, A., G. Schett, and U. Steffen (Née Harre). "POS0349 SINGLE OSTEOCLAST RESORPTION ANALYSIS REVEALS AN INCREASED INTRINSIC OSTEOCLAST ACTIVITY IN RA PATIENTS." In EULAR 2024 European Congress of Rheumatology, 12-15 June. Vienna, Austria. BMJ Publishing Group Ltd and European League Against Rheumatism, 2024. http://dx.doi.org/10.1136/annrheumdis-2024-eular.673.

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Intemann, J., C. Wehmeyer, V. Kracke, et al. "P081 Sclerostin affects rankl-mediated osteoclast differentiation." In 38th European Workshop for Rheumatology Research, 22–24 February 2018, Geneva, Switzerland. BMJ Publishing Group Ltd and European League Against Rheumatism, 2018. http://dx.doi.org/10.1136/annrheumdis-2018-ewrr2018.98.

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Hasegawa, Y., N. M. Nikolaidis, Y. Uehara, et al. "Osteoclast-Like Differentiation in Mouse Model of Silicosis." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a5413.

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Strini, T., S. Preinstorfer, H. Haidl, S. Tokic, S. Gallistl, and A. Schlagenhauf. "PAR4 activation promotes osteoclast formation and resorptive function." In GTH Congress 2024 – 68th Annual Meeting of the Society of Thrombosis and Haemostasis Research – Building Bridges in Coagulation. Georg Thieme Verlag, 2024. http://dx.doi.org/10.1055/s-0044-1779244.

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Reports on the topic "Osteoclast"

1

Ampornaramveth, Ruchanee. IL-1β mediate cementoblasts and osteoclast precursors interaction. Chulalongkorn University, 2016. https://doi.org/10.58837/chula.res.2016.17.

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An injury of the periodontium followed by an inflammatory response often leads to root resorption. Resorption is accomplished by osteoclasts and their generation may depend on an interaction with the cells in direct contact with the root, the cementoblasts. Our study aimed to investigate the role of human cementoblasts in the formation of osteoclasts and the effect of IL-1β hereupon. Extracted teeth from healthy volunteers were subjected to sequential digestion by type I collagenase and trypsin. The effect of enzymatic digestion on the presence of cells on the root surface was analyzed by hist

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Reddy, Sakamuri. Osteoclast Inhibitory Peptide-1 Therapy for Paget's Disease. Defense Technical Information Center, 2010. http://dx.doi.org/10.21236/ada539193.

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Reddy, Sakamuri V. Osteoclast Inhibitory Peptide-1 Therapy for Paget's Disease. Defense Technical Information Center, 2012. http://dx.doi.org/10.21236/ada567774.

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Reddy, Sakamuri. Osteoclast Inhibitory Peptide-1 Therapy for Paget's Disease. Defense Technical Information Center, 2011. http://dx.doi.org/10.21236/ada553287.

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Reddy, Sakamuri V. Measles Virus Nucleocapsid (MJVNP) Gene Expression and RANK Receptor Signaling in Osteoclast Precursors. Osteoclast Inhibitors Peptide Therapy for Pagets Disease. Defense Technical Information Center, 2005. http://dx.doi.org/10.21236/ada484715.

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Reddy, Sakamuri V. Measles Virus Nucleocapsid (MVNP) Gene Expression and RANK Receptor Signaling in Osteoclast Precursors, Osteoclast Inhibitors Peptide Therapy for Pagets Disease. Defense Technical Information Center, 2008. http://dx.doi.org/10.21236/ada500887.

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Reddy, Sakamuri V. Measles Virus Nucleocapsid (MVNP) Gene Expression and RANK Receptor Signaling in Osteoclast Precursors, Osteoclast Inhibitors Peptide Therapy for Pagets Disease. Defense Technical Information Center, 2006. http://dx.doi.org/10.21236/ada462833.

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Reddy, Sakamuri V. Measles Virus Nucleocapsid (MVNP) Gene Expression and RANK Receptor Signaling in Osteoclast Precursors,Osteoclast Inhibitors Peptide Therapy for Pagets Disease. Defense Technical Information Center, 2004. http://dx.doi.org/10.21236/ada482539.

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Morgans, Alicia K., David F. Penson, Amy J. Graves, Parul Chaudhuri, and Daniel Sonnenburg. Osteoclast inhibitor treatment among men with metastatic castration-resistant prostate cancer. Science Repository OÜ, 2018. http://dx.doi.org/10.31487/j.cor.2018.03.001.

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Morfin, C., and G. G. Loots. Characterizing the role of Mef2c in regulating osteoclast differentiation and energy metabolism. Office of Scientific and Technical Information (OSTI), 2018. http://dx.doi.org/10.2172/1459127.

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