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Books on the topic 'Osteoporosis Osteoporosis Bone density. Ben'

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Published: 4 June 2021
Last updated: 1 February 2022

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1

Foundation, National Osteoporosis. America's bone health: The state of osteoporosis and low bone mass in our nation. The Foundation, 2002.

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2

The bone density test. Berkley Books, 2000.

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3

Kessler, George J. The bone density diet: 6 weeks to a strong body and mind. Ballantine Books, 2000.

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4

Bath Conference on Osteoporosis and Bone Mineral Measurement (5th 1996). Current research in osteoporosis and bone mineral measurement IV, 1996: Proceedings of the Fifth Bath Conference on Osteoporosis and Bone Mineral Measurement, Bath, 24-26 June 1996. British Institute of Radiology, 1996.

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5

1998), Bath Conference on Osteoporosis and Bone Mineral Measurement (6th. Current research in osteoporosis and bone mineral measurement V: 1998: Proceedings of the Sixth Bath Conference on Osteoporosis and Bone Mineral Measurement, Bath, 22-26 June 1998. British Institute of Radiology, 1998.

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6

Bertha, Frisch, and SpringerLink (Online service), eds. Osteoporosis: Diagnosis, Prevention, Therapy. Springer Berlin Heidelberg, 2009.

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7

Exercises for osteoporosis: A safe and effective way to build bone density and muscle strength. HealthyLiving Books, 2005.

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8

Daniels, Dianne. Exercises for osteoporosis: A safe and effective way to build bone density and muscle strength. 3rd ed. HealthyLiving Books, 2008.

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9

Licata, Angelo A. A DXA primer for the practicing clinician: A case-based manual for understanding and interpreting bone densitometry. Springer, 2014.

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10

service), SpringerLink (Online, ed. Bone Densitometry in Clinical Practice: Application and Interpretation. Humana Press, 2010.

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11

Colbin, Annemarie. Whole foods for strong bones: A holistic approach. New Harbinger Publications, 2008.

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12

Magliano, Malgorzata. Osteoporosis. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199550647.003.010006.

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♦ Osteoporotic fractures affect one in two women and one in five men over the age of 50♦ Previous fragility fracture increases future fracture risk and should prompt further assessment and treatment♦ Clinical risk factors in combination with bone mineral density measurement allow identifying patients at risk♦ Screening for secondary causes of osteoporosis is important, particularly in men and younger women♦ Patients at high risk for future fracture should be offered appropriate treatment. Bisphosphonates together with adequate calcium and vitamin D supplementation constitute first-line therapy♦ Compliance with treatment and clinical response need to be monitored.
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13

Wordsworth, B. P. Skeletal dysplasias. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0150.

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Bone is metabolically active throughout life and metabolic disturbances may have wide-ranging consequences that are not restricted to altering its mechanics. The study of some genetic bone diseases has already provided remarkable insights into the normal regulation of bone metabolism. Skeletal dysplasias are developmental disorders of the chondro-osseous tissues commonly resulting in short stature, which is often disproportionate. The underlying mutations are often in the structural genes encoding components of the matrix but may also involve growth factors or cell signalling. In contrast, the dysostoses tend to affect single bones or groups of bones, reflecting the transient nature of the many different signalling factors to which they are responsive during development. Abnormalities of bone density (high or low) may be due to primary deficiency of bone matrix synthesis (e.g. osteogenesis imperfecta and hypophosphatasia) but may also reflect an imbalance between bone formation and resorption. This may be caused by abnormalities of bone formation (e.g. hyperostosis/sclerosteosis and osteoporosis pseudoglioma syndrome) or bone resorption (e.g. classic osteopetrosis and fibrous dysplasia).
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14

Harsch, Igor Alexander. Osteoporosis Update. Nova Science Publishers, Incorporated, 2011.

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15

O’Neal, M. Angela. Pain in the Back. Edited by Angela O’Neal. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190609917.003.0006.

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This case explores how bone health can be affected by antiepileptic drugs (AEDs), and why this is an important issue in women with epilepsy (WWE). AEDs affect bone health through activation of the cytochrome P450 system in the liver, leading to increased metabolism of vitamin D. The enzyme-inducing AEDs include phenytoin, carbamazepine, phenobarbital, and primidone. Risk factors for osteoporosis include female gender, low body mass, inadequate vitamin D intake, and smoking. The specific AEDs used and the length of treatment confer additional risks. Furthermore, in WWE, risks are magnified related to falls, either from seizures or related to medication toxicity. Screening with bone density and measures to promote bone health are extremely important in these at-risk women.
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16

K, Genant Harry, Guglielmi G. 1957-, and Jergas M. 1961-, eds. Bone densitometry and osteoporosis. Springer, 1998.

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17

Holroyd, Christopher R., Nicholas C. Harvey, Mark H. Edwards, and Cyrus Cooper. Environment. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0038.

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Musculoskeletal disease covers a broad spectrum of conditions whose aetiology comprises variable genetic and environmental contributions. More recently it has become clear that, particularly early in life, the interaction of gene and environment is critical to the development of later disease. Additionally, only a small proportion of the variation in adult traits such as bone mineral density has been explained by specific genes in genome-wide association studies, suggesting that gene-environment interaction may explain a much larger part of the inheritance of disease risk than previously thought. It is therefore critically important to evaluate the environmental factors which may predispose to diseases such as osteorthritis, osteoporosis, and rheumatoid arthritis both at the individual and at the population level. In this chapter we describe the environmental contributors, across the whole life course, to osteoarthritis, osteoporosis and rheumatoid arthritis, as exemplar conditions. We consider factors such as age, gender, nutrition (including the role of vitamin D), geography, occupation, and the clues that secular changes of disease pattern may yield. We describe the accumulating evidence that conditions such as osteoporosis may be partly determined by the early interplay of environment and genotype, through aetiological mechanisms such as DNA methylation and other epigenetic phenomena. Such studies, and those examining the role of environmental influences across other stages of the life course, suggest that these issues should be addressed at all ages, starting from before conception, in order to optimally reduce the burden of musculoskeletal disorders in future generations.
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18

Holroyd, Christopher R., Nicholas C. Harvey, Mark H. Edwards, and Cyrus Cooper. Environment. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199642489.003.0038_update_001.

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Musculoskeletal disease covers a broad spectrum of conditions whose aetiology comprises variable genetic and environmental contributions. More recently it has become clear that, particularly early in life, the interaction of gene and environment is critical to the development of later disease. Additionally, only a small proportion of the variation in adult traits such as bone mineral density has been explained by specific genes in genome-wide association studies, suggesting that gene-environment interaction may explain a much larger part of the inheritance of disease risk than previously thought. It is therefore critically important to evaluate the environmental factors which may predispose to diseases such as osteorthritis, osteoporosis, and rheumatoid arthritis both at the individual and at the population level. In this chapter we describe the environmental contributors, across the whole life course, to osteoarthritis, osteoporosis and rheumatoid arthritis, as exemplar conditions. We consider factors such as age, gender, nutrition (including the role of vitamin D), geography, occupation, and the clues that secular changes of disease pattern may yield. We describe the accumulating evidence that conditions such as osteoporosis may be partly determined by the early interplay of environment and genotype, through aetiological mechanisms such as DNA methylation and other epigenetic phenomena. Such studies, and those examining the role of environmental influences across other stages of the life course, suggest that these issues should be addressed at all ages, starting from before conception, in order to optimally reduce the burden of musculoskeletal disorders in future generations.
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19

Hill, Uta, Jane Ashbrook, and Charles Haworth. Metabolic and musculoskeletal effects of cystic fibrosis. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780198702948.003.0009.

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This chapter provides a comprehensive update on the prevention, recognition, and treatment of low bone mineral density in people with CF. As life expectancy improves, the extra-pulmonary complications of CF are becoming increasingly important to quality of life. Up to 25 per cent of CF patients have reduced bone mineral density in adulthood, leading to the development of fragility fractures which cause pain, thereby interfering with airway clearance and predisposing to pulmonary infection. Osteoporosis can be a relative contraindication for lung transplantation. Other important musculoskeletal issues including CF arthropathy, growth, and urinary incontinence are covered. CF arthropathy is a non-erosive episodic sero-negative arthritis, often difficult to treat and which may require specialist input. Urinary incontinence is common girls and women with CF and has a negative impact on quality of life and ability to complete therapies. The pathophysiology and management of urinary incontinence are discussed.
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20

Afrooz, Afghani, ed. Family history of osteoporosis. Nova Science Publishers, 2009.

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21

Hypertension and Bone Loss. Nova Science Publishers, 2011.

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22

Skeletal Aging And Osteoporosis Biomechanics And Mechanobiology. Springer, 2012.

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23

(Contributor), C. Bartl, ed. OSTEOPOROSIS: Diagnosis, Prevention, Therapy. Springer, 2004.

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24

Ring, E. F. J., D. M. Elvins, and A. K. Bhalla. Current Research in Osteoporosis and Bone Mineral Measurement V. The British Institute of Radiology, 1998.

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25

Kapklein, Colleen, and George Dr Kessler. The Bone Density Diet: 6 Weeks to a Strong Body and Mind. Ballantine Books, 2000.

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26

Hutchison, Alastair J., and Michael L. Picton. Fractures in patients with chronic kidney disease. Edited by David J. Goldsmith. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0121.

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Patients with any degree of chronic kidney disease (CKD) have a much higher risk of fractures than the general population, and the risk of death at 1 year post hip fracture in a dialysis patient is over 60%, compared to less than 20% for a non-CKD patient. The assessment of fracture risk and diagnosis of the underlying skeletal pathology in CKD patients is a significant clinical challenge. Non-invasive imaging techniques are not totally reliable in the general population, and the presence of advanced CKD (stages 4, 5, and 5D) renders them largely useless. Bone strength is not determined only by quantity of bone, and renal osteodystrophy can significantly affect bone quality, rendering it liable to fracture even in the presence of a normal bone density measurement. Currently, the only reliable method of assessing both quantity and quality of bone is the examination of trans-iliac bone biopsy, which is generally, but probably incorrectly, perceived to be overly invasive. However, identifying the cause of reduced bone strength and fractures may influence the choice of therapy. For example, in the presence of low-turnover states such as adynamic bone, antiresorptive agents may be ineffective. Pharmaceuticals licensed for the treatment of osteoporosis in the general population can be used similarly in patients with CKD 1–3 without dosage alteration. In CKD 4, post-hoc analyses suggest denosumab is effective and safe, based on a 3-year study that included 73 such patients. In CKD 5 and 5D no dependable data exists to guide therapy, and it should probably be reserved for patients who have already suffered and survived a fracture, and are therefore at high risk of death from a second event.
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27

Daniels, Dianne Ma. Exercises for Osteoporosis: A Safe and Effective Way to Build Bone Density and Muscle Strength, Revised Edition. Hatherleigh Press, 2004.

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28

Javaid, Kassim. Osteoporosis and fragility fracture. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0275.

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Osteoporosis is defined as a systemic bone disease with reduction in both bone density and microarchitectural integrity, resulting in an increase in fragility fracture risk. It is a multifactorial disease which, through effects on bone formation and resorption, reduces the peak bone mass achieved during early adulthood and increases the rate of bone loss in later adulthood. Osteoporosis is clinically silent until a fragility fracture occurs. There are 3 million patients with osteoporosis in the UK, with over 200 000 fractures per year and 80 000 hip fractures. This chapter addresses the causes, clinical features, diagnosis, and management of osteoporosis.
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29

Lineback, Karena Thek. Osteopilates: Increase Bone Density Reduce Fracture Risk Look and Feel Great. New Page Books, 2003.

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30

Lineback, Karena Thek. Osteo Pilates: Increase Bone Density, Reduce Fracture Risk, Look and Feel Great. Red Wheel/Weiser, 2003.

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31

Ridd, Stephanie. Osteoporosis Treatment: How To Reverse or Prevent It Naturally With Osteoporosis Diet And Osteoporosis Exercise To Maintain Healthy Bone Mineral Density Even In Old Age Today! CreateSpace Independent Publishing Platform, 2017.

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32

1956-, Green C. J., and British Columbia Office of Health Technology Assessment., eds. Bone mineral density testing: Does the evidence support its selective use in well women? BC Office of Health Technology Assessment, 1997.

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33

Exercises for Osteoporosis: A Safe and Effective Way to Build Bone Density and Muscle Strength and Improve Posture and Flexibility for Women and Men (Exercises for). Hatherleigh Press, 2008.

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34

The relationship between bone mineral density and selected variables: An epidemiological perspective. 1990.

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35

Kapklein, Colleen, and George Dr Kessler. The Bone Density Program: 6 Weeks to Strong Bones and a Healthy Body. Ballantine Books, 2001.

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36

Knopf, Chris (Personal trainer), author, ed. Beat osteoporosis with exercise: A low-impact program for building strength, increasing bone density and improving posture. Ulysses Press, 2016.

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37

Dr. Lani's no-nonsense bone health guide: The truth about density testing, osteoporosis drugs, and building bone quality at any age. Hunter House, 2014.

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38

Diaz-Granados, Natalia. Predictors of low bone mineral density among Canadian men: Data from the Canadian multicentre osteoporosis study. 2003.

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39

Lee, Christoph I. Repeat Bone Mineral Density Screening and Osteoporotic Fracture Prediction. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190223700.003.0035.

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This chapter, found in the bone, joint, and extremity pain section of the book, provides a succinct synopsis of a key study examining the need for repeat bone densitometry screening and prediction of fractures from osteoporosis. This summary outlines the study methodology and design, major results, limitations and criticisms, related studies and additional information, and clinical implications. The study showed that a repeat bone mineral density test within 4 years adds little additional value beyond the baseline test when assessing hip fracture risk. Moreover, a repeat test within 4 years may not improve fracture risk stratification used for clinical management of osteoporosis. In addition to outlining the most salient features of the study, a clinical vignette and imaging example are included in order to provide relevant clinical context.
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40

Heath, Jan. The effects of exercise on high density lipoprotein cholestrol and bone alkaline phophates: Implications for coronary artery disease and osteoporosis in postmenopausal women. 1987.

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41

Ulrich, Cornelia M. Relationship between total, axial and peripheral bone mineral density, lifetime milk consumption and lifetime physical activity in elderly mothers and their premenopausal daughters. 1992.

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42

Eddins, William C. A comparison of bone mineral density between active and nonactive men with spinal cord injuries. 1994.

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43

The effect of a weight training program on the bone density of women aged 40-50 years. 1994.

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44

The effects of high impact exercise versus low impact exercise on bone density in postmenopausal women. 1990.

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45

Takahashi, H. E. Mechanical Loading Of Bones And Joints. Edited by H. E. Takahashi. Springer, 1999.

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