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Relevant bibliographies by topics / Ovarian Follicular Development

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Journal articles

Academic literature on the topic 'Ovarian Follicular Development'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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Journal articles on the topic "Ovarian Follicular Development"

1

Leung, Peter C. K. "Ovarian follicular development and regression." Canadian Journal of Physiology and Pharmacology 67, no. 8 (1989): 953. http://dx.doi.org/10.1139/y89-149.

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Many exciting developments in mammalian reproductive research with far-reaching consequences have occurred in recent years. To highlight the significance of some of these developments, a symposium on the theme of ovarian follicular development and regression was organized, and held at the joint meeting of the American Physiological Society and the Canadian Physiological Society, in Montréal in October 1988. Several leading researchers, from both Canada and the U.S.A., in various aspects of ovarian research, participated in the symposium. The topics of discussion ranged from the role of growth

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2

Sobinoff, A. P., V. Pye, B. Nixon, S. D. Roman, and E. A. McLaughlin. "153. XENOBIOTICS; INFLUENCE ON OVARIAN FOLLICULAR DEVELOPMENT." Reproduction, Fertility and Development 21, no. 9 (2009): 71. http://dx.doi.org/10.1071/srb09abs153.

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The mammalian female reproductive lifespan is largely defined by a finite pool of ovarian follicles established around the time of birth. It is now understood that certain synthetic chemical compounds, known as xenobiotics, can cause premature ovarian senescence through the destruction of small ovarian follicles. Although the ovotoxic effects of these chemicals are well documented, the exact molecular mechanisms behind their action are only just becoming understood. Recent evidence suggests that bioactivation of xenobiotics by Phase I detoxifying enzymes may lead to the generation of free oxyg

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3

Segino, Miwa, Mario Ikeda, Fumiki Hirahara, and Kahei Sato. "In vitro follicular development of cryopreserved mouse ovarian tissue." Reproduction 130, no. 2 (2005): 187–92. http://dx.doi.org/10.1530/rep.1.00515.

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In a previous report, we showed that follicles isolated from frozen/thawed mouse ovarian tissues reached the mature follicle stage on the 12th day of culture. However, the developmental ability was lower than that of fresh ovarian tissue. The purpose of this study was to define a culture system with some technical modification for preantral follicles isolated from frozen/thawed ovarian tissue and to confirm cell injury. Ovaries obtained from three-week-old female mice were cryopreserved by the rapid freezing method. Preantral follicles isolated from frozen/thawed ovarian tissues were cultured

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4

van der Schoot, P., and W. J. de Greef. "Development of ovarian follicles during lactation in rats." Acta Endocrinologica 112, no. 2 (1986): 247–52. http://dx.doi.org/10.1530/acta.0.1120247.

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Abstract. Ovarian follicular development was studied during lactation in rats. At an early stage of lactation (day 7) the ovaries showed only small follicles in agreement with the expected 'follicular quiescence' during lactation. However, at a more advanced stage of lactation (day 14), there were large follicles which were capable of ovulation in response to exogenous gonadotropins. Unilateral ovariectomy early during lactation (day 2) resulted in compensatory follicular development in the remaining ovary. However, doubling of the number of large follicles per ovary had not yet occurred by da

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5

Kim, Yu Jin, YoungJoon Park, Yeo Reum Park, et al. "Role of RGMc as a Neogenin Ligand in Follicular Development in the Ovary." Biomedicines 9, no. 3 (2021): 280. http://dx.doi.org/10.3390/biomedicines9030280.

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There is currently no cure for infertility in women with a poor ovarian response (POR). Neogenin is reported to be abundantly expressed in the ovary; however, its role in mammalian follicular development is unclear and its ligand and signaling pathway remain uncertain. We systematically investigated the role of neogenin and the ligand repulsive guidance molecule c (RGMc) during follicular development. We treated hyperstimulated mouse ovaries with RGMc and analyzed follicular development. Furthermore, we investigated clusters of up/downregulated genes in RGMc-treated ovaries using whole-transcr

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6

Rodgers, R. J., T. C. Lavranos, I. L. van Wezel, and H. F. Irving-Rodgers. "Development of the ovarian follicular epithelium." Molecular and Cellular Endocrinology 151, no. 1-2 (1999): 171–79. http://dx.doi.org/10.1016/s0303-7207(99)00087-8.

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7

Maddineni, Sreenivasa R., Olga M. Ocón-Grove, Susan M. Krzysik-Walker, Gilbert L. Hendricks, and Ramesh Ramachandran. "Gonadotropin-inhibitory hormone (GnIH) receptor gene is expressed in the chicken ovary: potential role of GnIH in follicular maturation." REPRODUCTION 135, no. 2 (2008): 267–74. http://dx.doi.org/10.1530/rep-07-0369.

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Gonadotropin-inhibitory hormone (GnIH), an RFamide peptide, has been found to inhibit pituitary LH secretion in avian and mammalian species. The gene encoding a putative receptor for GnIH (GnIHR) was recently identified in the chicken and Japanese quail brain and pituitary gland. GnIHR appears to be a seven-transmembrane protein belonging to a family of G-protein-coupled receptors. In the present study, we have characterized the expression of GnIHR mRNA in the chicken ovary and demonstrate that GnIHR may exert an inhibitory effect on ovarian follicular development. By RT-PCR, we detected GnIHR

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8

Cha, KY, BR Do, HJ Chi, et al. "Viability of Human Follicular Oocytes Collected from Unstimulated Ovaries and Matured and Fertilized in vitro." Reproduction, Fertility and Development 4, no. 6 (1992): 695. http://dx.doi.org/10.1071/rd9920695.

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Immature human follicular oocytes were collected from unstimulated ovaries, matured and fertilized in vitro and then transferred to patients with no ovarian dysfunction such as premature ovarian failure. From 11 1 consenting donors, 422 immature oocytes were collected from 97 ovaries between January 1990 and October 1991. The number of oocytes collected from ovaries and their development were recorded so that comparisons could be made among donors of different ages and ovarian condition, such as menstrual cycle, cyclic and non-cyclic ovaries. The rate of fertilization in vitro showed a peak in

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9

Shi, Xuan, Tao Tang, Qiyuan Lin, et al. "Efficient generation of bone morphogenetic protein 15-edited Yorkshire pigs using CRISPR/Cas9†." Biology of Reproduction 103, no. 5 (2020): 1054–68. http://dx.doi.org/10.1093/biolre/ioaa138.

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Abstract Bone morphogenetic protein 15 (BMP15), a member of the transforming growth factor beta superfamily, plays an essential role in ovarian follicular development in mono-ovulatory mammalian species. Studies using a biallelic knockout mouse model revealed that BMP15 potentially has just a minimal impact on female fertility and ovarian follicular development in polyovulatory species. In contrast, our previous study demonstrated that in vivo knockdown of BMP15 significantly affected porcine female fertility, as evidenced by the dysplastic ovaries containing significantly decreased numbers of

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10

Zhou, Jiawei, Xianwen Peng, and Shuqi Mei. "Autophagy in Ovarian Follicular Development and Atresia." International Journal of Biological Sciences 15, no. 4 (2019): 726–37. http://dx.doi.org/10.7150/ijbs.30369.

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