Relevant bibliographies by topics / Ovaries – Cancer – Pathophysiology

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  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters

Academic literature on the topic 'Ovaries – Cancer – Pathophysiology'

Author: Grafiati
Published: 4 June 2021
Last updated: 13 February 2022

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Journal articles on the topic "Ovaries – Cancer – Pathophysiology"

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Laviolette, Laura A., Kenneth Garson, Elizabeth A. Macdonald та ін. "17β-Estradiol Accelerates Tumor Onset and Decreases Survival in a Transgenic Mouse Model of Ovarian Cancer". Endocrinology 151, № 3 (2010): 929–38. http://dx.doi.org/10.1210/en.2009-0602.

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Epithelial ovarian cancer is thought to be derived from the ovarian surface epithelium (OSE) but often goes undetected in the early stages, and as a result, the factors that contribute to its initiation and progression remain poorly understood. Epidemiological studies have suggested that the female steroid hormones are involved in ovarian carcinogenesis and that women who use hormone replacement therapy are at increased risk of developing the disease. A novel transgenic mouse model of ovarian cancer (tgCAG-LS-TAg) was developed to examine the role of the female reproductive steroid hormones [1
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Bulun, Serdar E., Bahar D. Yilmaz, Christia Sison, et al. "Endometriosis." Endocrine Reviews 40, no. 4 (2019): 1048–79. http://dx.doi.org/10.1210/er.2018-00242.

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AbstractPelvic endometriosis is a complex syndrome characterized by an estrogen-dependent chronic inflammatory process that affects primarily pelvic tissues, including the ovaries. It is caused when shed endometrial tissue travels retrograde into the lower abdominal cavity. Endometriosis is the most common cause of chronic pelvic pain in women and is associated with infertility. The underlying pathologic mechanisms in the intracavitary endometrium and extrauterine endometriotic tissue involve defectively programmed endometrial mesenchymal progenitor/stem cells. Although endometriotic stromal c
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Hojnik, Marko, Nataša Kenda Šuster, Špela Smrkolj, Snježana Frković Grazio, Ivan Verdenik, and Tea Lanišnik Rižner. "AKR1C3 Is Associated with Better Survival of Patients with Endometrial Carcinomas." Journal of Clinical Medicine 9, no. 12 (2020): 4105. http://dx.doi.org/10.3390/jcm9124105.

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The aldo-keto reductase (AKR) superfamily is gaining attention in cancer research. AKRs are involved in important biochemical processes and have crucial roles in carcinogenesis and chemoresistance. The enzyme AKR1C3 has many functions, which include production of prostaglandins, androgens and estrogens, and metabolism of different chemotherapeutics; AKR1C3 is thus implicated in the pathophysiology of different cancers. Endometrial and ovarian cancers represent the majority of gynecological malignancies in developed countries. Personalized treatments for these cancers depend on identification o
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Wong, Raymond R. Y., Michael J. Worley, Tony K. H. Chung, and Yick Fu Wong. "An update on Müllerian-inhibiting substance: its potential application against ovarian cancer." Endocrine-Related Cancer 21, no. 3 (2014): R227—R233. http://dx.doi.org/10.1530/erc-14-0030.

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Each year, ∼25 000 women are newly diagnosed with ovarian cancer in the USA. The vast majority (>90%) of cases are of epithelial origin. This highly lethal cancer carries a mortality rate of >50% and a high risk of recurrence after conventional, first-line chemotherapy. Müllerian-inhibiting substance (MIS) is a gonadal hormone that causes regression of the Müllerian ducts. A series of studies have demonstrated that MIS also has multiple extra-Müllerian functions including inhibition of epithelial ovarian cancer cells in vitro and in vivo. Accumulating evidence has shown that many human c
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Halkia, Evgenia, George Chrelias, Charalambos Chrelias, and Jesus Esquivel. "2017 Update on Ovarian Cancer Peritoneal Carcinomatosis Multimodal-Treatment Considerations." Gastroenterology Research and Practice 2018 (2018): 1–6. http://dx.doi.org/10.1155/2018/5284814.

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Ovarian cancer peritoneal carcinomatosis requires a multimodal-treatment approach. Current treatment considerations are analyzed in this update and include the management of recurrent malignant ascites and the understanding of its pathophysiology, the role of peritoneal washing cytology in detecting peritoneal metastases, capsular invasion and ovarian cancer histologic type, interpretation of pretreatment Ca-125 levels at different time points of ovarian cancer therapeutic management, characteristics of 10-year survivors of high-grade ovarian cancer, and the role of lymphadenectomy in ovarian
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Furuya, Mitsuko. "Ovarian Cancer Stroma: Pathophysiology and the Roles in Cancer Development." Cancers 4, no. 3 (2012): 701–24. http://dx.doi.org/10.3390/cancers4030701.

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Ness, Roberta B. "Endometriosis and ovarian cancer: Thoughts on shared pathophysiology." American Journal of Obstetrics and Gynecology 189, no. 1 (2003): 280–94. http://dx.doi.org/10.1067/mob.2003.408.

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Andersen Hammond, Elizabeth, Marshall Pitz, and Barbara Shay. "Neuropathic Pain in Taxane-Induced Peripheral Neuropathy: Evidence for Exercise in Treatment." Neurorehabilitation and Neural Repair 33, no. 10 (2019): 792–99. http://dx.doi.org/10.1177/1545968319860486.

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One in 2 Canadians is expected to acquire cancer in their lifetime. Many cancers, including breast, ovarian, and lung cancer, are treated using taxane chemotherapy with curative intent. A major adverse effect with the use of taxane chemotherapeutic agents is taxane-induced peripheral neuropathy (TIPN). Both positive (spontaneous pain, heightened sensitivity with light touch, tingling, itching, burning) and negative (loss of touch, loss of hot/cold sensations, and loss of pain) sensory symptoms can be experienced in the hands and feet and worsen with increasing dose and treatment duration. The
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Rickard, Brittany P., Christina Conrad, Aaron J. Sorrin, et al. "Malignant Ascites in Ovarian Cancer: Cellular, Acellular, and Biophysical Determinants of Molecular Characteristics and Therapy Response." Cancers 13, no. 17 (2021): 4318. http://dx.doi.org/10.3390/cancers13174318.

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Ascites refers to the abnormal accumulation of fluid in the peritoneum resulting from an underlying pathology, such as metastatic cancer. Among all cancers, advanced-stage epithelial ovarian cancer is most frequently associated with the production of malignant ascites and is the leading cause of death from gynecologic malignancies. Despite decades of evidence showing that the accumulation of peritoneal fluid portends the poorest outcomes for cancer patients, the role of malignant ascites in promoting metastasis and therapy resistance remains poorly understood. This review summarizes the curren
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Pal, Purab, Kara Nicole Starkweather, Karen Held Hales, and Dale Buchanan Hales. "A Review of Principal Studies on the Development and Treatment of Epithelial Ovarian Cancer in the Laying Hen Gallus gallus." Comparative Medicine 71, no. 4 (2021): 271–84. http://dx.doi.org/10.30802/aalas-cm-20-000116.

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Often referred to as the silent killer, ovarian cancer is the most lethal gynecologic malignancy. This disease rarely shows any physical symptoms until late stages and no known biomarkers are available for early detection. Because ovarian cancer is rarely detected early, the physiology behind the initiation, progression, treatment, and prevention of this disease remains largely unclear. Over the past 2 decades, the laying hen has emerged as a model that naturally develops epithelial ovarian cancer that is both pathologically and histologically similar to that of the human form of the disease.
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Dissertations / Theses on the topic "Ovaries – Cancer – Pathophysiology"

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Kodigepalli, Madhav Karthik. "Role and Regulation of SnoN/SkiL and PLSCR1 Located at 3q26.2 and 3q23, Respectively, in Ovarian Cancer Pathophysiology." Scholar Commons, 2014. https://scholarcommons.usf.edu/etd/5426.

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Ovarian cancer is one of the most common causes of gynecological cancer related deaths in women. In 2014, the estimated number of deaths due to ovarian cancer is 14,270 with occurrence of over 22, 240 new cases (National Cancer Institute, http://seer.cancer.gov/statfacts/html/ovary.html). Despite improvement in treatment strategies, the 5-year survival rate is still below 50% mainly due to chemoresistance and relapse. Amplification of chromosomal region 3q26 is a common characteristic in various epithelial cancers including ovarian cancer. This region harbors various oncogenes including the TG
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"Role of nitric oxide (NO), NO synthases and soluble guanylyl cyclase/cGMP/protein kinase G signaling pathway in the regulation of apoptosis and cell proliferation in pancreatic islets and ovarian cancer cells." Thesis, 2006. http://library.cuhk.edu.hk/record=b6074229.

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In the studies about ovarian cancer cells, basal iNOS expression in the chemosensitive OV2008 cells was significantly higher than in the chemoresistant C13* cells. Cisplatin further increased iNOS expression in OV2008 cells, but had no effect in C13* cells. Furthermore, cisplatin dramatically reduced the expression levels of eNOS and nNOS, but again only in OV2008 cells. The data suggest that failure of cisplatin to upregulate iNOS and downregulate eNOS and nNOS in C13* cells could be an etiological factor in chemoresistance. Addition of exogenous NO at high levels, using SNAP, significantly i
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Guimarães, Joana Isabel Sá Mesquita. "Fisiopatologia da Prevenção do Cancro do Ovário." Master's thesis, 2018. http://hdl.handle.net/10316/82421.

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Trabalho Final do Mestrado Integrado em Medicina apresentado à Faculdade de Medicina<br>O cancro do ovário apresenta uma elevada taxa de mortalidade. Assim, é importante compreender como se desenvolve esta neoplasia para que se possam adoptar estratégias preventivas de forma a diminuir a incidência da mesma e, consequentemente, a sua mortalidade. Nos dias de hoje, os autores são unânimes em considerar que, nos últimos anos tem havido uma mudança quanto à fisiopatologia do cancro do ovário. Atualmente, os tumores do ovário são divididos em Tipo I e Tipo II. Os de tipo I são menos comuns, aprese
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Book chapters on the topic "Ovaries – Cancer – Pathophysiology"

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Mills, Gordon B., Astrid Eder, Xianjun Fang, et al. "Critical Role of Lysophospholipids in the Pathophysiology, Diagnosis, and Management of Ovarian Cancer." In Ovarian Cancer. Springer US, 2002. http://dx.doi.org/10.1007/978-1-4757-3587-1_12.

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Legro, Richard S. "Polycystic ovary syndrome: metabolic aspects." In Oxford Textbook of Endocrinology and Diabetes. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199235292.003.0857.

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Polycystic ovary syndrome (PCOS) is thought to be primarily a disorder that affects women during their reproductive years. The diagnostic criteria reflect ovarian dysfunction, i.e. hyperandrogenism, anovulation, and polycystic ovaries. However, women with PCOS appear to be uniquely insulin resistant, are frequently obese, and may be at risk for a variety of long-term health disorders including diabetes, cardiovascular disease, and cancers. Although the endocrine and reproductive features of the disorder improve with age, the associated metabolic abnormalities, particularly components of the metabolic syndrome, may actually worsen. This chapter will explore the pathophysiology of aberrant insulin action in women with PCOS, recognition of long-term risks, and preventive strategies.
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Nath, Banashree, and Hirok Roy. "Antioxidants in Female Reproductive Biology." In Antioxidants - Benefits, Sources, Mechanisms of Action. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.95937.

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Human female reproductive biology is a complex system and its pathologies are varied. However, majority of the pathologic processes involves the role of reactive oxygen species (ROS). Imbalance between the ROS and antioxidants results in oxidative stress (OS). OS is the pathognomonic factor in various female reproductive system ailments. OS contributes to the pathophysiology of infertility, pregnancy related complications, endometriosis, ovarian cancers, etc. Evidence of elevated oxidative stress biomarkers can be found in various inflammatory conditions. Numerous strategies have been postulated for management of OS related pathologic conditions. Antioxidants supplementation may play a crucial in prevention and management of these conditions. However, robust evidence is needed to support the role of antioxidants supplementation in various female reproductive disorders.
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Journal articles
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