Academic literature on the topic 'Ovulation Induction'

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Journal articles on the topic "Ovulation Induction"

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Ginther, O. J., M. O. Gastal, E. L. Gastal, J. C. Jacob, and M. A. Beg. "Induction of haemorrhagic anovulatory follicles in mares." Reproduction, Fertility and Development 20, no. 8 (2008): 947. http://dx.doi.org/10.1071/rd08136.

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A follicular wave and luteolysis were induced in mares by ablation of follicles ≥6 mm and treatment with prostaglandin F2α (PGF) on Day 10 (where ovulation = Day 0). The incidence of haemorrhagic anovulatory follicles (HAFs) in the induced waves (20%) was greater (P < 0.007) than in preceding spontaneous waves (2%). Hormone and follicle dynamics were compared between induced follicular waves that ended in ovulations (ovulating group; n = 36) v. HAFs (HAF group; n = 9). The day of the first ovulation or the beginning of HAF formation at the end of an induced wave was designated as post-treatment Day 0. The mean 13-day interval from Day 10 (PGF and ablation) to the post-treatment ovulation was normalised into Days 10 to 16, followed by Day –6 to Day 0 relative to the post-treatment ovulation. Concentrations of LH were greater (P < 0.05) in the HAF group than in the ovulating group on Days 10, 11, 12, 14, –3 and –2. The HAF group had greater (P < 0.003) LH concentrations on Day 10 of the preceding oestrous cycle with spontaneous ovulatory waves. The diameter of the largest follicle was less (P < 0.05) in the HAF group on most days between Day 13 and Day –1 and this was attributable to later (P < 0.002) emergence of the future largest follicle at 6 mm in the HAF group (Day 12.4 ± 0.5) than in the ovulating group (Day 11.3 ± 0.1). The results indicate that the high incidence of HAFs after PGF and ablation was associated with later follicle emergence and immediate and continuing greater LH concentration after PGF treatment, apparently augmented by an inherently high pretreatment LH concentration.
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Kennedy, Janet L., and Eli Y. Adashi. "Ovulation Induction." Obstetrics and Gynecology Clinics of North America 14, no. 4 (December 1987): 831–64. http://dx.doi.org/10.1016/s0889-8545(21)00591-x.

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WOLF, LYNDA J. "Ovulation Induction." Clinical Obstetrics and Gynecology 43, no. 4 (December 2000): 902–15. http://dx.doi.org/10.1097/00003081-200012000-00020.

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Homburg, Roy. "Ovulation induction." Expert Opinion on Pharmacotherapy 4, no. 11 (November 2003): 1995–2004. http://dx.doi.org/10.1517/14656566.4.11.1995.

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Smith, Pamela M. "Ovulation Induction." Journal of Obstetric, Gynecologic & Neonatal Nursing 14 (November 1985): S37—S43. http://dx.doi.org/10.1111/j.1552-6909.1985.tb02798.x.

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Von Hofe, Johanna, and G. Wright Bates. "Ovulation Induction." Obstetrics and Gynecology Clinics of North America 42, no. 1 (March 2015): 27–37. http://dx.doi.org/10.1016/j.ogc.2014.09.007.

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SETHIA, DEEPA. "Ovulation Induction." International Journal of Scientific and Research Publications 12, no. 10 (October 24, 2022): 147–48. http://dx.doi.org/10.29322/ijsrp.12.10.2022.p13022.

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Balen, Adam. "Ovulation induction." Current Obstetrics & Gynaecology 14, no. 4 (August 2004): 261–68. http://dx.doi.org/10.1016/j.curobgyn.2004.04.005.

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Fanchin, R., H. Fernandez, F. Olivennes, and R. Frydman. "Ovulation induction: Ovulation induction in 1995: a new policy." Human Reproduction 10, no. 9 (September 1, 1995): 2224–25. http://dx.doi.org/10.1093/oxfordjournals.humrep.a136272.

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Ferreira-Silva, José Carlos, Pábola Santos Nascimento, Marcelo Tigre Moura, Sarah Romini Lima Basto, Marlon Vasconcelos Azevedo, Jorge Motta Rocha, José Pompeu Santos Filho, and Marcos Antonio Lemos Oliveira. "Induction of Ovulation in Mangalarga Marchador Mares by hCG or GnRH." Acta Scientiae Veterinariae 46, no. 1 (March 10, 2018): 6. http://dx.doi.org/10.22456/1679-9216.86667.

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Background: Induction of ovulation is a key procedure for horse assisted reproduction technologies, such as for artificial insemination (AI) and embryo transfer. The application of hCG remains as the primary ovulation-inducing agent for horse assisted reproduction, but alternatives are in demand to avoid its adverse effects, such as loss of ovulation-inducing efficiency over multiple applications by hCC-antibody production. Despite reports on alternative ovulation-inducing agents, pair-wise comparisons of such agents under similar conditions have been limited. Under such scenario, the work was aimed to determine the efficiency of both hCG and Buserelin at inducing ovulation in Mangalarga Marchador mares raised in the Northeast of Brazil under an AI program.Materials, Methods & Results: Mares were initially selected based on their reproductive performance, the absence of clinical-reproductive alterations and adequate body condition score. Mares in diestrus were randomly distributed in three experimental conditions, received 5 mg of Dinoprost and were monitored daily for estrus detection. After estrus detection, ovaries were monitored by ultrasonography, in 12-h intervals, until the follicle reached 35 mm. At this time-point, ovulation was induced with 0.042 mg of Buserelin endovenously, with 3,000 IU hCG by an intramuscular shot, and control mares received 2 mL of saline solution, also by an intramuscular shot. Both hCG and Buserelin displayed similar efficiencies (P > 0.05) for induction of ovulation and that both agents were effective (P < 0.05) for such purpose, since greater percentages (P < 0.05) of induction on mares treated from those of the control. Moreover, the total number of ovulations in mares treated at the end of the experiment was not different (P > 0.05) from those found in the Control. All ovulations occurred within a 72-h period after treatment. It can be observed that in mares treated with hCG or Buserelin, ovulations occurred both in more mares (P < 0.05) and at earlier time-points than mares from the control. It is also possible to note that pregnancy was not different (P > 0.05) between hCG and Buserelin groups, and that pregnancy of mares treated with ovulation-inducing factors was similar to the control.Discussion: The majority of ovulations in mares occurred within initial 48-h after treatment for both hCG and GnRH, suggesting a similar potential for horse assisted reproduction. Both hCG and Buserelin are two commonly used agents for induction of ovulation in mares. As described here, the majority of ovulations occurred within initial 48-h after treatment, a fact which can be attributed to hCG and GnRH activity, since it can happen in intervals from 36 to 48-h after treatment. Pregnancy rates did not differ among groups. These results are under the working hypothesis that hCG and Buserelin would display similar efficiencies on pregnancy rates. Despite the understanding of hCG activity on induction of ovulation due to its high specificity toward LH receptors and results from a direct effect on diminishing estradiol concentration, increasing LH, and further inducing ovulation within 48-h after treatment. In contrast, Buserelin has a similar functional property but acts upon LH synthesis and its release. In conclusion, ovulation in mares can be induced with both hCG and Buserelin, and both ovulation-inducing agents do not affect pregnancy rates.
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Dissertations / Theses on the topic "Ovulation Induction"

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Wely, Madelon van. "Treatment regimens in ovulation induction and ovarian hyperstimulation." [S.l. : Amsterdam : s.n.] ; Universiteit van Amsterdam [Host], 2004. http://dare.uva.nl/document/77513.

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Moore, Kelly Renee. "Estrus induction and maintenance of cycles in gilts with PG-600 and boar exposure." Diss., Columbia, Mo. : University of Missouri-Columbia, 2007. http://hdl.handle.net/10355/4937.

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Thesis (M.S.)--University of Missouri-Columbia, 2007.
The entire dissertation/thesis text is included in the research.pdf file; the official abstract appears in the short.pdf file (which also appears in the research.pdf); a non-technical general description, or public abstract, appears in the public.pdf file. Title from title screen of research.pdf file (viewed on October 31, 2007) Includes bibliographical references.
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LONGELIN, CHRISTINE. "Induction de l'ovulation par administration pulsatile de gnrh : six ans d'experience lilloise." Lille 2, 1991. http://www.theses.fr/1991LIL2M292.

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WINTZ, SOPHIE. "L'induction de l'ovulation par la gonadoliberine et ses analogues." Strasbourg 1, 1987. http://www.theses.fr/1987STR10727.

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Ghobadi, Cyrus. "Individualizing clomiphene citrate for induction of ovulation : searching for covariates." Thesis, University of Sheffield, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.500091.

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Clomiphene citrate (CC) has been the first line of treatment for induction of ovulation in patients with anovulatory infertility since 1962. It is administered as a mixture of two geometric isomers, Enclomiphene (En) and Zu-clomiphene (Zu) citrate in the ratio 62%:38%. The En isomer is perceived to be more active. A prediction model of response to CC was developed utilizing physical and biochemical characteristics of patients but this model has not been vahdated in a set of patients independent of those used to develop the model. It examines an existent nomogram for the identification of patient resistant to CC therapy and indicates that the nomogram is not sufficiently sensitive and specific to be useful in clinical practice. It shows that obesity could change the pharmacokinetics of Zu but not En. It provides early indications for a hypothesis that therapeutic outcome may be linked to interindividual variation in the exposure to the metabolite formed by CYP2D6. This may explain the lack of any direct relationship between the concentrations of the parent compound and response.
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Chan, Po-heung, and 陳寶香. "Use of letrozole versus clomiphene citrate for superovulation in patients undergoing intrauterine insemination : a systematic review." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2014. http://hdl.handle.net/10722/206904.

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Background: Intrauterine Insemination (IUI)is one of the common first-line assisted reproductive technologies (ART) for couples suffering infertility. Controlled ovarian stimulation (COH) with clomiphene citrate (CC) or aromatase inhibitor (like Letrozole) is often used in adjunct to IUI to increase the pregnancy outcome. Both CC and letrozole can be given alone as a single ovulation induction agent or they can be combined with injectable gonadotropin for purpose of superovulation. Study objectives: To systematically review the efficacy and adverse outcomes of letrozole and CC for supervulation in infertility patients undergoing IUI. Method: Systematic review of pertinent randomised controlled trials (RCT) using the bibliographic databases EMBASE, PubMed, Cochrane, Medline (OVID), Academic Search Premier and CINAHL. References of selected articles identified were hand-searched for additional relevant citations. RCTs that have compared the pharmacological performance of CC and letrozole as a single agent or combination with equal dose of gonadotropins were included. Results: Ten published randomized controlled trials were included in this review. The mean age, infertility diagnosis and duration of infertility of the recruited participants were comparable. Pregnancy rate was found to be comparable in clomiphene citrate (CC) group and letrozole (L) group. Higher peak estrogen concentration and greater number of dominant follicles were reported in CC group. Endometrial thickness was found significantly greater in L group. Adverse outcomes of rate of miscarriage, multiple pregnancies, ectopic pregnancies, OHSS and fetal anomalies were not significantly different between the two intervention groups. Conclusion: Letrozole and CC, considered equally patient-friendly agent due to oral route administration. Both agents achieved similar pregnancy rates without any increased risk of adverse events in either group. Letrozole can be used as alternative first-line OI agent to CC in reproductive treatments. Drug selection for patients should be done according to the cost effectiveness, duration of therapy, characteristics and compliance of patients.
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PRADAT, ERIC. "Fentes faciales et grossesses induites : registres de malformations, outil de recherche epidemiologique." Lyon 1, 1990. http://www.theses.fr/1990LYO1M346.

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DELETTRE, PATRICE. "Les grossesses et accouchements plurigemellaires : a propos de 14 cas a la maternite du c.h.u. de reims (service du professeur p. wahl)." Reims, 1988. http://www.theses.fr/1988REIMM007.

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DELAOUSTRE, BERTRAND, and TREFFEL LAURENCE DELAOUSTRE. "Complications des inductions de l'ovulation, hors fiv : a propos de 5 cas, revue de la litterature." Lille 2, 1991. http://www.theses.fr/1991LIL2M338.

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ALBERSAMMER, JOEL. "Nanisme hypophysaire et grossesse : induction de l'ovulation et grossesse chez une femme presentant une insuffisance somatotrope et gonadotrope : a propos d'une observation." Saint-Etienne, 1988. http://www.theses.fr/1988STET6068.

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Books on the topic "Ovulation Induction"

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Collins, Robert L., ed. Ovulation Induction. New York, NY: Springer New York, 1991. http://dx.doi.org/10.1007/978-1-4612-3026-7.

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Cohlen, Ben J. Ovulation Induction. Boca Raton : Taylor & Francis, 2017. | Series: Reproductive: CRC Press, 2016. http://dx.doi.org/10.1201/9781315381459.

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L, Collins Robert, ed. Ovulation induction. New York: Springer-Verlag, 1991.

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World Conference on Ovulation Induction (2nd 1997 Bologna, Italy). Ovulation induction: Update '98 : the proceedings of the 2nd World Conference on Ovulation Induction, Bologna, Italy, 12-13 September 1997. New York: Parthenon Pub. Group, 1998.

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Shlomo, Mashiach, and Lunenfeld Bruno, eds. Ovulation induction and in vitro fertilization. Chicago: Year Book Medical Publishers, 1986.

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Homburg, Roy. Ovulation Induction and Controlled Ovarian Stimulation. Cham: Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-05612-8.

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M, Filicori, and Flamigni C, eds. Ovulation induction: Basic science and clinical advances : proceedings of the Symposium on Ovulation Induction: Basic Science and Clinical Advances, 20-22 January 1994, Palm Beach, Florida, USA. Amsterdam: Elsevier Science, 1994.

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P, Dickey Richard. Manual of intrauterine insemination and ovulation induction. Cambridge: Cambridge University Press, 2010.

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P, Dickey Richard. Manual of intrauterine insemination and ovulation induction. Cambridge: Cambridge University Press, 2010.

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Dickey, Richard P., Peter R. Brinsden, and Roman Pyrzak, eds. Manual of Intrauterine Insemination and Ovulation Induction. Cambridge: Cambridge University Press, 2009. http://dx.doi.org/10.1017/cbo9780511642159.

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Book chapters on the topic "Ovulation Induction"

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Gibson, Mark. "Ovulation Induction." In Reproductive Endocrinology and Infertility, 525–43. New York, NY: Springer New York, 2010. http://dx.doi.org/10.1007/978-1-4419-1436-1_35.

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Tulandi, Togas, and Hélène S. Weibel. "Ovulation Induction." In Clinical Reproductive Medicine and Surgery, 289–98. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-52210-4_16.

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Sel, Görker. "Ovulation Induction." In Practical Guide to Oral Exams in Obstetrics and Gynecology, 269–73. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-29669-8_42.

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Mills, Ginevra, and Togas Tulandi. "Ovulation Induction." In Clinical Reproductive Medicine and Surgery, 353–66. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-99596-6_16.

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Martinez, Alan M., and Steven R. Lindheim. "Induction of Ovulation." In Clinical Reproductive Medicine and Surgery, 209–19. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-6837-0_14.

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Taymor, Melvin L. "Induction of Ovulation." In Infertility, 217–35. Boston, MA: Springer US, 1990. http://dx.doi.org/10.1007/978-1-4613-0627-6_26.

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Franks, S. "Induction of Ovulation." In Infertility, 237–50. London: Springer London, 1992. http://dx.doi.org/10.1007/978-1-4471-1962-3_15.

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Baker, Marsha B., and Kristin A. Bendikson. "Induction of Ovulation." In Management of Common Problems in Obstetrics and Gynecology, 433–37. Oxford, UK: Wiley-Blackwell, 2010. http://dx.doi.org/10.1002/9781444323030.ch98.

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Homburg, Roy. "Physiology of Ovulation." In Ovulation Induction and Controlled Ovarian Stimulation, 7–23. Cham: Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-05612-8_2.

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Schenker, Joseph G. "Ovulation Induction: Adverse Effects." In ISGE Series, 103–18. Cham: Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-03494-2_10.

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Conference papers on the topic "Ovulation Induction"

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Hamáčková, Jitka, Jan Kouřil, Tomislav Barth, Andrea Lepičová, Pavel Kozák, and Pavel Lepič. "Induction of ovulation in rudd (Scardinius erythrophthalmus) using hormone preparations." In VIIth Conference Biologically Active Peptides. Prague: Institute of Organic Chemistry and Biochemistry, Academy of Sciences of the Czech Republic, 2001. http://dx.doi.org/10.1135/css200104087.

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Ionescu, Ruxandra C., Babar A. Khan, and Mark O. Farber. "Dyspnea, Nausea And Vomiting Following Ovulation Induction, A Case Of Severe Ovarian Hyperstimulation Syndrome." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a5402.

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Ohlsson, M., A. J. W. Hsueh, and T. Ny. "HORMONE REGULATION OF THE FIBRINOLYTIC SYSTEM IN THE OVARY." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644389.

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In the ovary, the release of oocytes from graafian follicles during hormone-induced ovulation has been found to be associated with substantial increases in follicular plasminogen activator (PA) activity. Most of the PA activity comes from the granulosa cells that have been shown to produce tPA, uPA as well as the type-1 PA-inhibitor,(PAI-1).We have studied the molecular mechanism of follicle stimulating hormone (FSH) and gonadotropin releasing hormone (GnRH) on the synthesis of tPA in primary cultures of rat granulosa cells. FSH and GnRH were both found to induce tPA in granulosa cells in a time and dose dependent manner. The effect of FSH and GnRH on the levels of tPA mRNA was also studied by northern and slot blot hybridizations. FSH and GnRH were both found to increase the level of tPA mRNA. The stimulation was up to 18 -fold compared to untreated cells.The induction of tPA mRNA by FSH and GnRH was additive and the time courses of the stimulation by the hormones differed, suggesting that different cellular mechanisms are involved. Consistent with the ability of FSH to activate the cAMP dependent protein kinase A pathway, the phosphodiesterase inhibitor 1-methyl-3-isobutylxanthine further enhanced the FSH induction of tPA mRNA.GnRH is known to activate the phospholipid-dependent protein kinase C pathway. Likewise the effect of GnRH can be mimicked by the kinase C activator, phorbol myristate acetate.It is concluded that FSH and GnRH regulates tPA production by differnt molecular mechanisms, and that the increase in tPA activity is mediated via an increase in the levels tPA mRNA. Since both gonadotropins and GnRH cause ovulation in hyposectomized animals, similar stimulatory actions of these hormones on the tPA activity suggest a correlative relationship between this enzyme and the ovulatory process.
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Reports on the topic "Ovulation Induction"

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Benton, Jennifer, Peggy A. Miller-Auwerda, Curtis R. Youngs, and Lawrence E. Evans. Induction of Ovulation and LH Response in Cyclic Mares Treated with Gonadorelin Diacetate Tetrahydrate. Ames (Iowa): Iowa State University, January 2010. http://dx.doi.org/10.31274/ans_air-180814-667.

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Youngs, Curtis R., Shelby L. Pattern, Cassie D. Krebill, Kacey L. Klemesrud, Zach A. Thompson, Benjamin J. Duran, and Jessica R. Juarez. Preliminary Evaluation of Administration Site of TwoManufacturer’s Reproductive Hormones on Induction of Ovulation in Postpartum Dairy Cows. Ames (Iowa): Iowa State University, January 2018. http://dx.doi.org/10.31274/ans_air-180814-400.

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Wolfenson, David, William W. Thatcher, and James E. Kinder. Regulation of LH Secretion in the Periovulatory Period as a Strategy to Enhance Ovarian Function and Fertility in Dairy and Beef Cows. United States Department of Agriculture, December 2003. http://dx.doi.org/10.32747/2003.7586458.bard.

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The general research objective was to increase herd pregnancy rates by enhancing corpus luteum (CL) function and optimizing follicle development, in order to increase conception rate and embryo survival. The specific objectives were: to determine the effect of the duration of the preovulatory LH surge on CL function; to determine the function of LH during the postovulatory period on CL development; to optimize CL differentiation and follicle development by means of a biodegradable GnRH implant; to test whether optimization of CL development and follicle dynamics in timed- insemination protocols would improve fertility in high-yielding dairy cows. Low fertility in cattle results in losses of hundreds of millions of dollars in the USA and Israel. Two major causes of low fertility are formation of a functionally impaired CL, and subsequent enhanced ovarian follicle development. A functionally impaired CL may result from suboptimal LH secretion. The two major causes of low fertility in dairy cattle in US and Israel are negative energy status and summer heat stress; in both situations, low fertility is associated with reductions in LH secretion and impaired development of the ovulatory follicle and of the CL. In Florida, the use of 450-mg deslorelin (GnRH analogue) implants to induce ovulation, under the Ovsynch protocol resulted in a higher pregnancy rates than use of 750-mg implants, and pregnancy losses tended to decrease compared to controls, due probably to decrease in follicular development and estradiol secretion at the time of conceptus signaling to maintain the CL. An alternative strategy to enhance progesterone concentrations involved induction of an accessory CL by injection of hCG on day 5 after the cows were inseminated. Treatment with hCG resulted in 86% of the cows having two CLs, compared with 23% of the control cows. Conception rates were higher among the hCG-treated cows than among the controls. Another approach was to replace the second injection of GnRH analogue, in a timed-insemination protocol, with estradiol cypionate (ECP) injected 24 h after the injection of PGF₂ₐ Pregnancy rates were comparable with those obtained under the regular Ovsynch (timed- AI) program. Use of ECP induced estrus, and cows inseminated at detected estrus are indeed more fertile than those not in estrus at the time of insemination. Collectively, the BARD-supported programs at the University of Florida have improved timed insemination programs. In Ohio, the importance of the frequency of LH episodes during the early stages of the estrous cycle of cattle, when the corpus luteum is developing, was studied in an in vivo experiment in which cows were subjected to various episodic exposures to exogenous bovine LH. Results indicate that the frequent LH episodes immediately following the time of ovulation are important in development of the corpus luteum, from the points of view of both size and functionality. In another study, rates of cell proliferation and numbers of endothelial cells were examined in vitro in CLs collected from cows that received post-ovulation pulsatile LH treatment at various frequencies. The results indicate that the corpora lutea growth that results from luteal cell proliferation is enhanced by the episodes of LH release that occur immediately after the time of ovulation in cattle. The results also show that luteal endothelial cell numbers did not differ among cows treated with different LH doses. In Israel. a longer duration of the preovulatory LH surge stimulated the steroidogenic capacity of granulosa-derived luteal cells, and might, thereby, contribute to a higher progesterone output from the bovine corpus luteum. In an in vivo study, a subgroup of high-yielding dairy cows with extended estrus to ovulation interval was identified. Associated with this extended interval were: low plasma progesterone and estradiol concentrations and a low preovulatory LH surge prior to ovulation, as well as low post- ovulation progesterone concentration. In experiments based on the above results, we found that injection of GnRH at the onset of estrus increased the LHpeak, prevented late ovulation, decreased the variability between cows and elicited high and uniform progesterone levels after ovulation. GnRH at estrus onset increased conception rates, especially in the summer, and among primiparous cows and those with low body condition. Another study compared ovarian functions in multiparous lactating cows with those in nulliparous non-lactating heifers. The results revealed differences in ovarian follicular dynamics, and in plasma concentrations of steroids and gonadotropins that may account for the differences in fertility between heifers and cows.
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