Relevant bibliographies by topics / Oxidative DNA damage

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Oxidative DNA damage'

Author: Grafiati
Published: 4 June 2021
Last updated: 26 July 2025

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Journal articles on the topic "Oxidative DNA damage"

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Skrypnyk, N. V., and O. O. Maslova. "Oxidative DNA damage." Biopolymers and Cell 23, no. 3 (2007): 202–14. http://dx.doi.org/10.7124/bc.000766.

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Hayes, Robert C., Lynn A. Petrullo, Haimei Huang, Susan S. Wallace, and J. Eugene LeClerc. "Oxidative damage in DNA." Journal of Molecular Biology 201, no. 2 (1988): 239–46. http://dx.doi.org/10.1016/0022-2836(88)90135-0.

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Collins, A. R., and E. Horváthová. "Oxidative DNA damage, antioxidants and DNA repair: applications of the comet assay." Biochemical Society Transactions 29, no. 2 (2001): 337–40. http://dx.doi.org/10.1042/bst0290337.

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Estimates of background levels of oxidative base damage in human white blood cells vary enormously, from 300 down to 0.4 molecules of 8-oxoguanine per 106 guanines. An EC-funded Concerted Action, the European Standards Committee on Oxidative DNA Damage, is currently attempting to resolve the discrepancy and to agree a realistic estimate of basal endogenous oxidation. Oxidation of lymphocyte DNA is a useful marker of oxidative stress, and this can be decreased by supplementation with pure antioxidants or with foods rich in antioxidants. The steady-state level of DNA oxidation is ultimately cont
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Soria-Meneses, Pedro Javier, Alejandro Jurado-Campos, Virgilio Gómez-Rubio, et al. "Determination of Ram (Ovis aries) Sperm DNA Damage Due to Oxidative Stress: 8-OHdG Immunodetection Assay vs. SCSA®." Animals 12, no. 23 (2022): 3286. http://dx.doi.org/10.3390/ani12233286.

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Conventional DNA analysis techniques can hardly detect DNA damage in ruminant spermatozoa due to high DNA compaction in these cells. Furthermore, these techniques cannot discriminate whether the damage is due to oxidative stress. The main purpose of this study was to evaluate the efficacy of two techniques for determining DNA damage in ovine sperm when the source of that damage is oxidative stress. Semen samples from twenty Manchega rams (Ovis aries) were collected and cryopreserved. After thawing, the samples were subjected to different levels of oxidative stress, and DNA oxidation was quanti
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Anderson, Andrew P., Xuemei Luo, William Russell, and Y. Whitney Yin. "Oxidative damage diminishes mitochondrial DNA polymerase replication fidelity." Nucleic Acids Research 48, no. 2 (2019): 817–29. http://dx.doi.org/10.1093/nar/gkz1018.

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Abstract Mitochondrial DNA (mtDNA) resides in a high ROS environment and suffers more mutations than its nuclear counterpart. Increasing evidence suggests that mtDNA mutations are not the results of direct oxidative damage, rather are caused, at least in part, by DNA replication errors. To understand how the mtDNA replicase, Pol γ, can give rise to elevated mutations, we studied the effect of oxidation of Pol γ on replication errors. Pol γ is a high fidelity polymerase with polymerase (pol) and proofreading exonuclease (exo) activities. We show that Pol γ exo domain is far more sensitive to ox
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Kwiatkowski, Dominik, Piotr Czarny, Monika Toma, et al. "Associations between DNA Damage, DNA Base Excision Repair Gene Variability and Alzheimer's Disease Risk." Dementia and Geriatric Cognitive Disorders 41, no. 3-4 (2016): 152–71. http://dx.doi.org/10.1159/000443953.

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Background: Increased oxidative damage to DNA is one of the pathways involved in Alzheimer's disease (AD). Insufficient base excision repair (BER) is in part responsible for increased oxidative DNA damage. The aim of the present study was to assess the effect of polymorphic variants of BER-involved genes and the peripheral markers of DNA damage and repair in patients with AD. Material and Methods: Comet assays and TaqMan probes were used to assess DNA damage, BER efficiency and polymorphic variants of 12 BER genes in blood samples from 105 AD patients and 130 controls. The DNA repair efficacy (
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Jeng, Hueiwang Anna, Chih-Hong Pan, Mu-Rong Chao, and Wen-Yi Lin. "Sperm DNA oxidative damage and DNA adducts." Mutation Research/Genetic Toxicology and Environmental Mutagenesis 794 (December 2015): 75–82. http://dx.doi.org/10.1016/j.mrgentox.2015.09.002.

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Guan, Zhe, Hongjie Song, and Rui Liu. "Adventures of DNA: Oxidative Damage." Daxue Huaxue 37, no. 9 (2022): 2204087–0. http://dx.doi.org/10.3866/pku.dxhx202299999.

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Takeuchi, Toru, Kanehisa Morimoto, Yasuhiro Nakaya, Naoki Kato, and Kunitomo Watanabe. "Oxidative DNA damage in anaerobes." Free Radical Biology and Medicine 25 (January 1998): S79. http://dx.doi.org/10.1016/s0891-5849(98)90254-4.

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Hartmann, A., and A. M. Niess. "Oxidative DNA damage in exercise." Pathophysiology 5 (June 1998): 112. http://dx.doi.org/10.1016/s0928-4680(98)80719-0.

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Dissertations / Theses on the topic "Oxidative DNA damage"

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Farooq, Sabya. "Free radical induced oxidative DNA damage." Thesis, University of Leicester, 1997. http://hdl.handle.net/2381/30749.

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Oxidative DNA damage has been implicated in processes such as carcinogenesis, mutagenesis, ageing and cell death. Reactive oxygen species (ROS) such as superoxide (O2), hydrogen peroxide (H2O 2) and hydroxyl radical (OH*) are produced in mammalian cells as a result of aerobic metabolism. However excess generation of these species by endogenous or exogenous sources can result in damage to DNA, producing a large number of sugar and base lesions. In order to understand the biological consequences of such free radical induced damage it is essential to characterise and quantitate this damage. This
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Soman, Sony. "OXIDATIVE DAMAGE TO DNA IN ALZHEIMER'S DISEASE." UKnowledge, 2013. http://uknowledge.uky.edu/chemistry_etds/28.

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Previous studies from our laboratory and others show a significant increase in levels of both nuclear and mitochondrial DNA and RNA oxidation in vulnerable brain regions in the progression of Alzheimer’s disease (AD). Although total DNA oxidation is increased in AD it remains unclear whether oxidative damage is widespread throughout the genome or is concentrated to specific genes. To test the hypothesis that specific genes are more highly oxidized in the progression of AD, we propose to quantify the percent oxidative damage in genes coding for proteins shown to be altered in the progression of
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Jansson, Kristina. "Oxidative damage and the DNA glycosylase MutYH /." Göteborg : Department of Cell and Molecular Biology, University of Gothenburg, 2010. http://hdl.handle.net/2077/22092.

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Ndlebe, Thabisile S. "Oxidative Damage in DNA: an Exploration of Various DNA Structures." Diss., Available online, Georgia Institute of Technology, 2006, 2006. http://etd.gatech.edu/theses/available/etd-05112006-154326/.

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Thesis (Ph. D.)--Chemistry and Biochemistry, Georgia Institute of Technology, 2007.<br>Donald F. Doyle, Committee Member ; Bridgette Anne Barry, Committee Member ; Dr. Gary B. Schuster, Committee Chair ; Nicholas V. Hud, Committee Member ; Roger M. Wartell, Committee Member.
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Cao, Huachuan. "Probe Oxidative Damage in DNA Charge Transfer Process." Diss., Georgia Institute of Technology, 2005. http://hdl.handle.net/1853/6983.

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As a hydrophilic biopolymer, a DNA molecule is surrounded by water molecules in aqueous solution. The charge hopping mechanism indicates the competition between radical cation quenching by water molecules and migration along DNA partially determines the distance and efficiency of charge transport in DNA. Lipid can effectively bind DNA to induce hydrophobic environment around the DNA helix and reduce the water contact with bases in the DNA duplex. Therefore, the effect of water molecules on charge transport can be studied by comparison between nature DNA and DNA-lipid complexes. We synthesized
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Abdallah, Suaad Audat. "Investigation of Oxidative DNA Damage from Ionizing Radiation." University of Toledo Health Science Campus / OhioLINK, 2012. http://rave.ohiolink.edu/etdc/view?acc_num=mco1349377002.

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Rehman, Almas. "Factors affecting oxidative DNA damage in the human body." Thesis, King's College London (University of London), 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.251611.

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Xu, Meng. "Oxidative DNA Damage Modulates Trinucleotide Repeat Instability Via DNA Base Excision Repair." FIU Digital Commons, 2014. http://digitalcommons.fiu.edu/etd/1576.

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Trinucleotide repeat (TNR) expansion is the cause of more than 40 types of human neurodegenerative diseases such as Huntington’s disease. Recent studies have linked TNR expansion with oxidative DNA damage and base excision repair (BER). In this research, we provided the first evidence that oxidative DNA damage can induce CAG repeat deletion/contraction via BER. We found that BER of an oxidized DNA base lesion, 8-oxoguanine in a CAG repeat tract, resulted in the formation of a CTG hairpin at the template strand. DNA polymerase β (pol b) then skipped over the hairpin creating a 5’-flap that was
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Cannan, Wendy J. "Mechanisms and Dynamics of Oxidative DNA Damage Repair in Nucleosomes." ScholarWorks @ UVM, 2016. http://scholarworks.uvm.edu/graddis/628.

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DNA provides the blueprint for cell function and growth, as well as ensuring continuity from one cell generation to the next. In order to compact, protect, and regulate this vital information, DNA is packaged by histone proteins into nucleosomes, which are the fundamental subunits of chromatin. Reactive oxygen species, generated by both endogenous and exogenous agents, can react with DNA, altering base chemistry and generating DNA strand breaks. Left unrepaired, these oxidation products can result in mutations and/or cell death. The Base Excision Repair (BER) pathway exists to deal with damage
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Mikhed, Yuliya [Verfasser]. "The role of DNA damage in the pathogenesis of nitrate tolerance - nitrosative versus oxidative DNA damage / Yuliya Mikhed." Mainz : Universitätsbibliothek Mainz, 2015. http://d-nb.info/1080187529/34.

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Books on the topic "Oxidative DNA damage"

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1962-, Evans Mark D., and Cooke Marcus S, eds. Oxidative damage to nucleic acids. Landes Bioscience, 2007.

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J, Lunec, ed. Measuring in vivo oxidative damage: A practical approach. Wiley, 2000.

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Jana, Varvaøovská, ed. Impact of oxidative stress on diabetes mellitus and inflammatory bowel diseases. Nova Science, 2007.

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D, Szabó Csaba M., ed. Cell death: The role of poly(ADP-ribose) polymerase. CRC Press, 2000.

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Kennedy, Gayle. The effects of dietary antioxidant supplementation on cellular sensitivity to ionising radiation and oxidative DNA damage. The Author], 1997.

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G, Cutler Richard, and International Conference on Oxidative Stress and Aging (1st : 1994 : Hawaii), eds. Oxidative stress and aging. Birkhauser Verlag, 1995.

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1945-, Favier Alain, ed. Analysis of free radicals in biological systems: Edited by A. E. Favier ... [et al.]. Birkhäuser Verlag, 1995.

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Barry, Halliwell, and Aruoma Okezie I. 1951-, eds. DNA and free radicals. Ellis Horwood, 1993.

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Evans, Mark D., and Marcus S. Cooke. Oxidative Damage to Nucleic Acids. Springer London, Limited, 2007.

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Wagner, J. Richard. Oxidative DNA Damage: Formation, Measurement and Biochemical Features. World Scientific Publishing Co Pte Ltd, 2013.

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Book chapters on the topic "Oxidative DNA damage"

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Guengerich, F. Peter. "Oxidative DNA Damage." In Molecular Life Sciences. Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-6436-5_441-1.

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Collins, Andrew R. "Oxidative DNA Damage." In Encyclopedia of Cancer. Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-16483-5_4306.

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Guengerich, Frederick Peter. "Oxidative DNA Damage." In Molecular Life Sciences. Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4614-1531-2_441.

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Cadet, J., M. Berger, B. Morin, S. Raoul, and J. R. Wagner. "Oxidative damage to DNA." In Analysis of Free Radicals in Biological Systems. Birkhäuser Basel, 1995. http://dx.doi.org/10.1007/978-3-0348-9074-8_5.

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Williamson, Josh, and Gareth W. Davison. "Exercise and DNA Damage." In Oxidative Eustress in Exercise Physiology. CRC Press, 2022. http://dx.doi.org/10.1201/9781003051619-9.

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Stuart, Jeffrey A., and Melissa M. Page. "DNA Oxidative Damage and Cancer." In Oxidative Stress in Aging. Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-420-9_12.

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Rose, Sarah, Sarah Wong, and Cecilia Giulivi. "Mitochondrial DNA Damage in Autism." In Biochemistry of Oxidative Stress. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-45865-6_21.

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Olinski, Ryszard, Marek Foksinski, and Barbara Tudek. "Oxidative DNA Damage and Carcinogenesis." In Oxidative Damage to Nucleic Acids. Springer New York, 2007. http://dx.doi.org/10.1007/978-0-387-72974-9_12.

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Cadet, J., M. Bardet, M. Berger, et al. "Oxidative Base Damage to DNA." In Advances in DNA Damage and Repair. Springer US, 1999. http://dx.doi.org/10.1007/978-1-4615-4865-2_5.

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Dedon, Peter C., and Marita C. Barth. "Oxidatively Damaged DNA and Inflammation." In Oxidative Damage to Nucleic Acids. Springer New York, 2007. http://dx.doi.org/10.1007/978-0-387-72974-9_15.

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Conference papers on the topic "Oxidative DNA damage"

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Tekutskaya, E. E., and I. S. Raybova. "OXIDATIVE DAMAGE TO DNA, IONIZING RADIATION AND MAGNETIC FIELDS." In International conference New technologies in medicine, biology, pharmacology and ecology (NT +M&Ec ' 2020). Institute of information technology, 2020. http://dx.doi.org/10.47501/978-5-6044060-0-7.21.

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In this work, we studied the mechanisms of damage to the structure of DNA extracted from human&#x0D; whole blood after exposure to blood samples of various types of ionizing radiation and a lowfrequency magnetic field. Oxidative damage of nitrogenous bases and single-stranded DNA breaks are caused by the formation of reactive oxygen species generated by radiation.
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Xuena Zhu, P. Shah, and Chenzhong Li. "Paper-based Immunosensor for Oxidative DNA Damage Biomarker Detection." In 2013 29th Southern Biomedical Engineering Conference (SBEC 2013). IEEE, 2013. http://dx.doi.org/10.1109/sbec.2013.71.

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Turkiewicz, Szymon, Marcin Sochal, Marta Ditmer, Filip Franciszek Karuga, and Agata Gabryelska. "Evaluation of DNA/RNA oxidative damage in Obstructive Sleep Apnea patients." In ERS Sleep and Breathing 2023 abstracts. European Respiratory Society, 2023. http://dx.doi.org/10.1183/23120541.sleepandbreathing-2023.111.

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Kant, S., N. Dikshit, A. K. Verma, et al. "To estimate oxidative stress and DNA damage in post-COVID patients." In ERS International Congress 2022 abstracts. European Respiratory Society, 2022. http://dx.doi.org/10.1183/13993003.congress-2022.2458.

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Ding, Ning, Emily M. Bonham, Brooke E. Hannon, Stephen B. Baylin, and Heather M. O'Hagan. "Abstract A18: Mismatch repair proteins recruit DNA methyltransferase 1 to sites of oxidative DNA damage." In Abstracts: AACR Special Conference: Chromatin and Epigenetics in Cancer; September 24-27, 2015; Atlanta, GA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.chromepi15-a18.

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Tekutskaya, E. E., I. I. Pavlyuchenko, and L. R. Gusaruk. "rs652438 POLYMORPHISM OF THE MMP-12 GENE AND ANALYSIS OF DEGREE OF OXIDATIVE DAMAGE TO GENOMIC DNA." In NOVEL TECHNOLOGIES IN MEDICINE, BIOLOGY, PHARMACOLOGY AND ECOLOGY. Institute of information technology, 2022. http://dx.doi.org/10.47501/978-5-6044060-2-1.219-224.

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The article provides a comparative analysis of the degree of oxidative DNA damage in epi-dermolysis bullosa (EB) and bronchial asthma (BA). It was found that the concentration of the modified base 8-oxoguanine in patients with BE is 1.9 times higher than in the control group. Different concentrations of 8-oxoguanine in BE and BE indicate the severity of struc-tural DNA damage in BE and the almost absence of oxidative modification of DNA in AD.
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Wu, Yang, Qing Chang, and Xu Yang. "Evaluation of DNA Oxidative Damage in Visceral Cells Using 8-OHdG Level." In 2010 4th International Conference on Bioinformatics and Biomedical Engineering (iCBBE 2010). IEEE, 2010. http://dx.doi.org/10.1109/icbbe.2010.5515243.

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Soulitzis, Nikolaos, Eirini Neofytou, Nikoletta Rovina, Eleftherios Zervas, Dimitra Papandrinopoulou, and Nikolaos Siafakas. "Cytotoxic activity and oxidative DNA damage in smoking asthmatics versus COPD patients." In Annual Congress 2015. European Respiratory Society, 2015. http://dx.doi.org/10.1183/13993003.congress-2015.pa3923.

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Kawai, Kazuaki, Yun-Shan Li, Yuya Kawasaki, and Yasuo Morimoto. "Oxidative DNA damage in rat lung after exposure to cerium oxide nanoparticles." In Annual Congress 2015. European Respiratory Society, 2015. http://dx.doi.org/10.1183/13993003.congress-2015.pa4115.

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Alli, Elizabeth, and James M. Ford. "Abstract 1761: Breast cancers with compromised DNA repair exhibit selective sensitivity to elesclomol-induced oxidative DNA damage." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-1761.

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Reports on the topic "Oxidative DNA damage"

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Wu, Guangyu. Preventive Role of Specific Dietary Factors and Natural Compounds Against DNA Damage and Oxidative Stress. Defense Technical Information Center, 1999. http://dx.doi.org/10.21236/ada377925.

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Patil, Bhimanagouda S., Ron Porat, G. K. Jayaprakasha, and K. N. C. Murthy. Optimization of Postharvest Storage Conditions to Maintain Fruit Quality and Health Maintaining Properties of Grapefruit. United States Department of Agriculture, 2010. http://dx.doi.org/10.32747/2010.7613879.bard.

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Antioxidant activity of fruits is gaining wide interest among consumers due to its importance in counteracting oxidative stress, free radicals and preventing DNA damage. Oxygen radical absorbance capacity (ORAC) assay is one of the commonly used assays to measure the antioxidant activity, which is based on hydrogen atom transfer mechanism. Furocoumarins present in grapefruit are reported to have antiproliferative activity, induce GST activity, inhibit biofilm formation and increase bioavailability of drugs. In the present project ORAC values were measured of Star Ruby grapefruit undergone ethy
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Di Pasquale, Alice, Kerri Palmer-Quinn, and Hannah Moody. Pesticides, organic food and breast cancer. Breast Cancer UK, 2025. https://doi.org/10.71450/95822655.

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Pesticides are chemicals used in agriculture and residential areas to repel or control certain forms of plant or animal life that are considered to be pests. Many of these chemicals are harmful and may damage the DNA, alter gene expression, induce oxidative stress, or act as Endocrine Disrupting Chemicals (EDCs). Some pesticides have also been classified as potentially promoting cancer by international regulatory bodies. People working with pesticides may be exposed to higher levels, whilst the rest of the population is primarily exposed through the diet or when pesticides are used in resident
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Louisek Prakash. Repair of DNA damaged by ionizing radiation and other oxidative agents in yeast and human. Office of Scientific and Technical Information (OSTI), 2000. http://dx.doi.org/10.2172/807355.

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Louise Prakash. Repair of DNA damaged by ionizing radiation and other oxidative agents in yeast and human. Office of Scientific and Technical Information (OSTI), 2000. http://dx.doi.org/10.2172/807356.

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Cahaner, Avigdor, Sacit F. Bilgili, Orna Halevy, Roger J. Lien, and Kellye S. Joiner. effects of enhanced hypertrophy, reduced oxygen supply and heat load on breast meat yield and quality in broilers. United States Department of Agriculture, 2014. http://dx.doi.org/10.32747/2014.7699855.bard.

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Original objectivesThe objectives of this project were to evaluate the growth performance, meat yield and quality attributes of broiler strains widely differing in their genetic potential under normal temperature vs. warm temperature (short and long-term) conditions. Strain differences in breast muscle accretion rate, metabolic responses under heat load and, gross and histopathological changes in breast muscle under thermal load was also to be characterized. BackgroundTremendous genetic progress has been made in broiler chicken growth rate and meat yield since the 1950s. Higher growth rate is
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Watkins, Chris B., Susan Lurie, Amnon Lers, and Patricia L. Conklin. Involvement of Antioxidant Enzymes and Genes in the Resistance Mechanism to Postharvest Superficial Scald Development. United States Department of Agriculture, 2004. http://dx.doi.org/10.32747/2004.7586539.bard.

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The objective of this research project was to evaluate the involvement of antioxidant enzymes and genes in the resistance mechanism to postharvest superficial scald development using two primary systems: 1. Resistant and susceptible progenies of an apple cross between a scald resistant crab apple, ‘White Angel’ and a scald susceptible cultivar, ‘Rome Beauty’; 2. Heat-treatment of ‘Granny Smith’, which is known to reduce scald development in this cultivar. In 2002 we asked for, and received (October 14), permission to revise our initial objectives. The US side decided to expand their results to
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Oxidative DNA damage leading to chromosomal aberrations and mutations. Organisation for Economic Co-Operation and Development (OECD), 2023. http://dx.doi.org/10.1787/399d2c34-en.

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