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Books on the topic 'Oxidative DNA damage'

Author: Grafiati

Published: 4 June 2021

Last updated: 26 July 2025

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1

1962-, Evans Mark D., and Cooke Marcus S, eds. Oxidative damage to nucleic acids. Landes Bioscience, 2007.

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2

J, Lunec, ed. Measuring in vivo oxidative damage: A practical approach. Wiley, 2000.

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3

Jana, Varvaøovská, ed. Impact of oxidative stress on diabetes mellitus and inflammatory bowel diseases. Nova Science, 2007.

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4

D, Szabó Csaba M., ed. Cell death: The role of poly(ADP-ribose) polymerase. CRC Press, 2000.

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5

Kennedy, Gayle. The effects of dietary antioxidant supplementation on cellular sensitivity to ionising radiation and oxidative DNA damage. The Author], 1997.

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6

G, Cutler Richard, and International Conference on Oxidative Stress and Aging (1st : 1994 : Hawaii), eds. Oxidative stress and aging. Birkhauser Verlag, 1995.

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7

1945-, Favier Alain, ed. Analysis of free radicals in biological systems: Edited by A. E. Favier ... [et al.]. Birkhäuser Verlag, 1995.

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8

Barry, Halliwell, and Aruoma Okezie I. 1951-, eds. DNA and free radicals. Ellis Horwood, 1993.

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9

Evans, Mark D., and Marcus S. Cooke. Oxidative Damage to Nucleic Acids. Springer London, Limited, 2007.

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10

Wagner, J. Richard. Oxidative DNA Damage: Formation, Measurement and Biochemical Features. World Scientific Publishing Co Pte Ltd, 2013.

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11

(Editor), J. Lunec, and H. R. Griffiths (Editor), eds. Measuring In Vivo Oxidative Damage: A Practical Approach. Wiley, 2000.

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12

Leanderson, Per. Mineral Fibers, Cigarette Smoke, and Oxidative DNA Damage: An experimental study. 1992.

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13

DNA Damage, Oxidative Stress and Related Metabolic By-Products in Cancer and Environmental Studies. MDPI, 2021. http://dx.doi.org/10.3390/books978-3-0365-1271-6.

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14

Szabo, Csaba. Cell Death: The Role of PARP. Taylor & Francis Group, 2000.

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15

Szabo, Csaba. Cell Death: The Role of PARP. Taylor & Francis Group, 2000.

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16

Gray, Doug, Carole Proctor, and Tom Kirkwood. Biological aspects of human ageing. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199644957.003.0001.

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At the molecular and cellular levels human ageing is characterized by the accumulation of unrepaired random damage, and an accompanying loss of function. A major source of damage is oxidative stress caused by the generation of reactive oxygen species as a by-product of respiration. DNA and proteins are both susceptible to damage but whereas DNA damage repair systems exist, faulty proteins are generally removed by protein degradation systems. During ageing these systems become less efficient and the subsequent accumulation of damaged protein promotes protein aggregation, a process which is espe

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17

Dodds, Chris, Chandra M. Kumar, and Frédérique Servin. Pathophysiological changes of ageing and their relevance to anaesthesia. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198735571.003.0002.

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The molecular basis of ageing is reviewed. This includes the concept of a summation of DNA damage over a lifetime causing genome instability. Epigenetic alterations, telomeric shortening, and the possibility of their modification are discussed. Oxidative and mitochondrial DNA damage and the resulting dysfunction leading to senescence are briefly described. Systemic problems and resultant behavioural adaptation may mask the decline in functional reserve and cause some of the difficulties in identifying its presence in ill elderly patients. Specific organ system changes are then described in som

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18

Monari, Antonio, Elise Dumont, and Chryssostomos Chatgilialoglu, eds. Radiation-induced and oxidative DNA damages. Frontiers Media SA, 2015. http://dx.doi.org/10.3389/978-2-88919-660-9.

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19

Jeng, Winnie. Free radical determinants of endogenous and amphetamine-enhanced neurodegenerative disease: Prostaglandin H synthase-catalyzed free radical formation, reactive oxygen species-mediated oxidative DNA damage and glucose-6-phosphate dehydrogenase-catalyzed neuroprotection. 2004.

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20

Gapstur, Susan M., and Philip John Brooks. Alcohol and Cancer Risk. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0012.

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In 2010, alcoholic beverage consumption caused an estimated 3.3 million deaths worldwide, and contributed to injuries, violence, liver cirrhosis, social disruption and at least seven different types of cancer. The International Agency for Research on Cancer (IARC) classifies exposure to both ethanol in alcoholic beverages and acetaldehyde, the primary metabolite of ethanol, as carcinogenic to humans (Group 1) based on “sufficient” evidence that alcoholic beverage consumption is causally related to cancers of the oral cavity, pharynx, larynx, esophagus, liver, colorectum and female breast. The

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21

Arsenic in Drinking Water and Skin Cancer. Exon Publications, 2024. https://doi.org/10.36255/arsenic-drinking-water-skin-cancer.

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Arsenic in Drinking Water and Skin Cancer is a comprehensive guide that explains the relationship between arsenic-contaminated water and the development of skin cancer.The article is organized into distinct sections that provide a clear understanding of how arsenic enters drinking water, the health risks associated with exposure, and strategies for prevention. It begins by defining arsenic as a naturally occurring element found in rocks, soil, and water, while also highlighting how human activities like mining and industrial waste contribute to water contamination. The article emphasizes that

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