Relevant bibliographies by topics / Oxidative stess

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  2. Dissertations / Theses
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Academic literature on the topic 'Oxidative stess'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Oxidative stess"

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Horton, Amanda, Kim Boggess, Kevin Moss, James Beck, and Steven Offenbacher. "401: Maternal periodontal infection, oxidative stess, and preeclampsia risk." American Journal of Obstetrics and Gynecology 199, no. 6 (December 2008): S121. http://dx.doi.org/10.1016/j.ajog.2008.09.430.

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Sarvazyan, Narine, Luther M. Swift, Pamela J. Kell, and Jame McHowat. "Oxidant-induced inhibition of phospholipase Ag2 activity and its role in potentiation of oxidative stess." Journal of Molecular and Cellular Cardiology 33, no. 6 (June 2001): A105. http://dx.doi.org/10.1016/s0022-2828(01)90419-5.

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Maharaj, Deepa S., Beverley D. Glass, and Santy Daya. "Melatonin: New Places in Therapy." Bioscience Reports 27, no. 6 (November 20, 2007): 299–320. http://dx.doi.org/10.1007/s10540-007-9052-1.

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The fact that the full extent of the function of the pineal gland has not yet been elucidated, has stimulated melatonin research worldwide. This review introduces melatonin's mechanism of action, direct and indirect antioxidant actions as well as the antioxidant properties of its metabolites, 6-hydroxymelatonin (6-OHM) and N-acetyl-N-formyl-5-methoxykynurenamine (AFMK). At present the mechanism of action is proposed to be receptor-, protein- and nonprotein-mediated. From its popular role in the treatment of jetlag, melatonin is now implicated in the reduction of oxidative stess, both as a free radical scavenger and antioxidant. Melatonin's direct scavenging action in respect of the following will be discussed: superoxide anions, hydrogen peroxide, hydroxyl radicals, singlet oxygen, peroxy radicals and nitric oxide/peroxy nitrite anions. In addition melatonin also possesses indirect antioxidant activity and the role of its metabolites, AFMK and 6-OHM will be presented. It is these free radical scavenging and antioxidant properties of melatonin that has shifted the focus from that of merely strengthening circadian rhythms to that of neuroprotectant: a new place in therapy.
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Sun, Qi-An, Nageswara Madamanchi, and Marschall Runge. "Oxidative stress, NADPH oxidases, and arteries." Hämostaseologie 36, no. 02 (2016): 77–88. http://dx.doi.org/10.5482/hamo-14-11-0076.

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ZusammenfassungDie Atherosklerose und ihre wichtigsten Komplikationen – Myokardinfarkt und Schlaganfall – sind die Hauptursachen für Tod und Behinderung in den USA und weltweit. Eine dramatische Zunahme bei Adipositas und Diabetes mellitus wird wahrscheinlich auch in Zukunft zu einer hohen Prävalenz kardiovaskulärer Erkrankungen (CVD) und deren Auswirkungen auf das Gesundheitswesen führen. Große Fortschritte gibt es bei der Entwicklung neuer Therapien zur Senkung der Inzidenz von Atherosklerose und CVD, besonders bei der Behandlung der Hypercholesterinämie und Hypertonie. Der gemeinsame mechanistische Nenner bei vielen Risikofaktoren für CVD ist oxidativer Stress. Erst seit kurzem verfügen wir über Methoden, um die Schnittstelle zwischen oxidativem Stress und CVD im Tiermodell zu untersuchen. Die wichtigste Quelle für reaktive Sauerstoffspezies (und damit für oxidativen Stress) in vaskulären Zellen sind die Formen der Nicotin - amidadenindinukleotidphosphat-Oxidase (NADPH-Oxidase). Die jüngsten Studien belegen eindeutig, dass 1. NADPH-Oxidasen im Tiermodell von grundlegender Bedeutung für Atherosklerose und Hypertonie sind und 2. der vaskuläre oxidative Stress, angesichts der gewebespezifischen Expression wichtiger Bestandteile der NADPH-Oxidase, ein Ziel bei der Prävention der CVD sein könnte.
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Lozano-Picazo, Carmen María, and Francisco Fernández-Belda. "Especies reactivas de oxígeno y su implicación en Biomedicina." Anales de Veterinaria de Murcia 34 (December 16, 2020): 17–26. http://dx.doi.org/10.6018/analesvet.332621.

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Las especies reactivas de oxígeno (ROS) actúan como regulador intracelular cuando se generan de forma controlada en puntos concretos de la célula. Modifican la función de proteínas mediante la oxidación reversible de cisteínas. Hay quinasas y fosfatasas de proteínas, factores de transcripción y canales iónicos que están regulados por ROS. Estrés oxidativo y daño celular aparecen cuando los mecanismos antioxidantes de protección son incapaces de mantener bajo el nivel intracelular de ROS. En estas condiciones, ROS inducen pérdida de viabilidad celular en patologías degenerativas de corazón y cerebro y promueven proliferación celular ilimitada en procesos tumorales. La alteración de la función mitocondrial juega un papel clave en la generación del estrés oxidativo y por tanto es una diana terapéutica preferente para evitar o aminorar los daños oxidativos producidos por ROS. Reactive oxygen species (ROS) act as intracellular regulator when they are generated under control in specific cell spots. They modify proteins function by cysteine reversible oxidation. There are protein kinases and phosphatases, transcription factors and ionic channels that are regulated by ROS. Oxidative stress and cell damage arise when the protection antioxidant mechanisms are unable to keep low the intracellular ROS level. Under these conditions, ROS induce cell viability loss in heart and brain degenerative pathologies and promote unlimited cell proliferation in tumor processes. Alteration of the mitochondrial function is a key player in the oxidative stress generation and therefore it is preferential therapeutic target for prevention or attenuation of the ROS-induced oxidative damage.
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Rosa, Eloi Francisco, and Vanessa Cristina Coimbra. "Câncer de cólon e estresse oxidativo." O Mundo da Saúde 33, no. 4 (December 4, 2009): 415–18. http://dx.doi.org/10.15343/0104-7809.20094415418.

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Qin, Bei, Kuan Yang, and Ruijun Cao. "Synthesis and Antioxidative Activity of Piperine Derivatives Containing Phenolic Hydroxyl." Journal of Chemistry 2020 (July 21, 2020): 1–9. http://dx.doi.org/10.1155/2020/2786359.

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Piperine was used in this study in its raw form, and different steps, such as amide hydrolysis and amidation, were used to synthesize piperine derivatives containing a phenolic hydroxyl group. DPPH and ABTS free radical scavenging assays were used to assess piperine derivative antioxidant activities. We constructed an AAPH oxidative stress erythrocyte model to study the effect of piperine derivatives on the hemolysis rate of oxidatively damaged erythrocytes as well as the hemoglobin oxidation rate. This AAPH model was also used to determine piperine derivative effects on antioxidant enzyme activity and malondialdehyde (MDA) content. Results showed that spectroscopic methods could synthesize and identify piperine derivatives containing phenolic hydroxyl groups (H-1∼H-3). Moreover, DPPH and ABTS assay results showed that piperine derivative free radical clearance rates were higher compared with the parent compound. Additionally, piperine derivatives (H-1∼H-3) were found to provide protection to AAPH oxidatively damaged erythrocytes in their ability to inhibit AAPH-induced erythrocyte lysis, while hemoglobin oxidation was higher compared with the parent compound. Piperine derivatives may protect intracellular glutathione peroxidase (GSH-Px) antioxidant enzyme system activities, safeguarding against oxidative damage. This study synthesized novel piperine derivatives for use as potential antioxidant agent candidates.
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Dorovskikh, V. A., N. V. Simonova, E. Yu Yurtaeva, R. A. Anokhina, and M. A. Shtarberg. "PHYTOCORRECTION OF OXIDATIVE STRESS IN EXPERIMENT." Amur Medical Journal, no. 15-16 (2016): 35–37. http://dx.doi.org/10.22448/amj.2016.15-16.35-37.

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Chen, Chuck T., Marc-Olivier Trépanier, Kathryn E. Hopperton, Anthony F. Domenichiello, Mojgan Masoodi, and Richard P. Bazinet. "Inhibiting Mitochondrial β-Oxidation Selectively Reduces Levels of Nonenzymatic Oxidative Polyunsaturated Fatty Acid Metabolites in the Brain." Journal of Cerebral Blood Flow & Metabolism 34, no. 3 (December 11, 2013): 376–79. http://dx.doi.org/10.1038/jcbfm.2013.221.

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Schönfeld and Reiser recently hypothesized that fatty acid β-oxidation is a source of oxidative stress in the brain. To test this hypothesis, we inhibited brain mitochondrial β-oxidation with methyl palmoxirate (MEP) and measured oxidative polyunsaturated fatty acid (PUFA) metabolites in the rat brain. Upon MEP treatment, levels of several nonenzymatic auto-oxidative PUFA metabolites were reduced with few effects on enzymatically derived metabolites. Our finding confirms the hypothesis that reduced fatty acid β-oxidation decreases oxidative stress in the brain and β-oxidation inhibitors may be a novel therapeutic approach for brain disorders associated with oxidative stress.
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Shang, F., and A. Taylor. "Oxidative stress and recovery from oxidative stress are associated with altered ubiquitin conjugating and proteolytic activities in bovine lens epithelial cells." Biochemical Journal 307, no. 1 (April 1, 1995): 297–303. http://dx.doi.org/10.1042/bj3070297.

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Roles for ubiquitin (an 8.5 kDa polypeptide) involve its conjugation to proteins as a signal to initiate degradation and as a stress protein. We investigated ubiquitin conjugation and ubiquitin-dependent proteolytic activities in cultured bovine lens epithelial cells (BLECs) upon oxidative challenge. A 44% decrease in intracellular glutathione confirmed oxidative stress upon incubation with 1 mM H2O2. After 30 min incubation, endogenous high-molecular-mass ubiquitin conjugates decreased 73%, and intracellular proteolysis decreased about 50%. In the supernatants of the oxidatively treated BLECs, the ability to form high-molecular-mass ubiquitin conjugates with exogenous 125I-labelled ubiquitin decreased 28%, and ATP-dependent degradation of oxidized alpha-crystallin decreased 36%. When the H2O2-treated BLECs were allowed to recover for 60 min, intracellular proteolysis returned to the level of control cells. There was also a subsequent transient enhancement of intracellular proteolysis and a simultaneous recovery of endogenous high-molecular-mass ubiquitin conjugates. In parallel cell-free experiments, conjugating activity with exogenous 125I-labelled ubiquitin and ATP-dependent degradation of oxidized alpha-crystallin increased 35% and 72% respectively compared with non-oxidatively treated BLECs. ATP-independent proteolysis showed little response to exposure or removal of H2O2. These results indicate that (1) the rate of intracellular proteolysis in BLECs is associated with the level of endogenous high-molecular-mass ubiquitin conjugates and (2) oxidative stress may inactivate the ubiquitin conjugation activity with coordinate depression of proteolytic capability. Enhancement in ubiquitin conjugation and proteolytic activities during recovery from oxidative stress may be important in removal of damaged proteins and restoration of normal function of BLECs. The inactivation of ubiquitin-dependent proteolysis by oxidation may be involved in the accumulation of altered proteins and other adverse sequelae in the oxidatively challenged aging lens.
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Dissertations / Theses on the topic "Oxidative stess"

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Barber, Drew. "Selenium In Thioredoxin Reductase: Resistance To Oxidative Inactivation, Oxidation States, And Reversibility Of Chemical Reactions." ScholarWorks @ UVM, 2018. https://scholarworks.uvm.edu/graddis/943.

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Selenium is a required trace element which was originally discovered by the Swedish chemist Jons Jacob Berzelius in 1817. It was initially believed to be a toxin as it was identified as being the cause of hoof maladies and excessive hair loss in horses that feed upon plants with high selenium content. It wasn’t until 1957 that the potential contributions of selenium to physiology were first demonstrated. Selenium is now known to play a critical role in the maintenance of human health. Interestingly, unlike other trace metals/semi-metals, selenium is directly incorporated into proteins in the form of the amino acid selenocysteine (Sec) in a very complicated and energetically costly fashion. Though rare, being found in only 25 human proteins, Sec proteins are involved in numerous vital biological processes including maintenance of redox homeostasis and anti-oxidant defense. Even though Sec is essential, the reason that Sec replaces its structural analog cysteine (Cys) in only 25 proteins is not widely agreed upon. A previous model suggests that the replacement of Cys with Sec provides enzymes with a type of catalytic advantage. The presence of Cys-containing orthologs of mammalian Sec-enzymes in other eukaryotes argues against this model. A newer model to explain the use of Sec is that the gain of function imparted to an enzyme by replacing Cys with Sec is the ability of Sec to impart chemical reversibility. Building on previous results from our lab demonstrating the ability of Sec to confer proteins with the ability to resist over oxidation we have elucidated the mechanism by which Sec containing thioredoxin reductase (TrxR) resists over oxidation. The ability of Sec-TrxR to resist oxidative inactivation is due to the greater electrophilicity of Sec relative to Cys. This allows for quicker resolution and prevents over oxidation. Based on these findings we also investigate the utility of the alkylating agent dimedone to probe the oxidation state of Sec. Interestingly, it was discovered that dimedone will react with seleneninic acid with the resulting adduct being labile. Additonally it was discovered that dimedone will also react with seleninic acid, resulting in the formation of a dimedone dimer. These results call into question the usefulness of dimedone in deteremining the oxidation state of Sec. Finally, we provide evidence that Sec-TrxR enzymes are able to catalyze single electron reductions. This is most likely due to the formation of a stable Sec radical intermediate. As a whole this project provides support for the theory that Sec was selected for due to its ability to convey chemical reversiablity to proteins.
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Landim, Emanuel Rocha. "Efeito do prÃ-tratamento com l-alanil glutamina e precondicionamento isquÃmico em modelo de isquemia / reperfusÃo de membros pÃlvicos em ratos." Universidade Federal do CearÃ, 2008. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=7077.

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No presente trabalho, estudaram-se os efeitos da l-alanil glutamina (Ala-Gln), do precondicionamento isquÃmico (PCI) e das duas tÃcnicas concomitantemente sobre a lesÃo pulmonar provocada por isquemia e reperfusÃo (I/R) causada por pinÃamento da aorta infra-renal em ratos. Foram utilizados 60 ratos machos Wistar, randomizados em cinco grupos (n = 12) divididos em dois tempos (n = 6): Grupo Simulado, Grupo I/R, Grupo PCI + I/R, Grupo Ala-Gln + I/R, Grupo Ala-Gln + PCI + I/R. Tempos: T1 (4h de isquemia) e T2 (4 horas de isquemia e 1h de reperfusÃo). Todos os grupos receberam soluÃÃo salina previamente, menos os grupos prÃ-tratados com Ala-Gln que receberam o dipeptÃdeo e soluÃÃo salina em igual volume. Foi utilizado o modelo de pinÃamento da aorta infra-renal com 4 horas de isquemia e 1 hora de reperfusÃo. Determinaram-se as concentraÃÃes de mieloperoxidase (MPO) pulmonar, substÃncias reativas ao Ãcido tiobarbitÃrico (TBARS) e glutationa reduzida (GSH) no sangue e pulmÃo para avaliar os grupos em estudo. O teste de Kolmogorov-Smirnoff mostrou distribuiÃÃo normal dos dados. Dados expressos como mÃdia acompanhada pelo seu desvio padrÃo (MÃdia  DPM) sendo realizado teste t de Student. Para anÃlise comparativa simultÃnea de trÃs grupos utilizou-se o teste Anova com pÃs-teste de Tukey. Em todos os casos foi adotado o nÃvel de significÃncia de p<0,05. Houve elevaÃÃo das concentraÃÃes de MPO pulmonar tanto no grupo submetido à isquemia quanto no grupo que realizou a I/R. Ocorreu reduÃÃo significante das concentraÃÃes de MPO pulmonar nos grupos submetidos à isquemia prÃ-tratados com Ala-Gln e com PCI. Na avaliaÃÃo dos grupos que sofreram I/R nÃo foi observada alteraÃÃo nas concentraÃÃes de MPO nos grupos prÃ-tratados Ala-Gln ou PCI. O grupo prÃ-tratado com as duas tÃcnicas apresentou aumento significante da MPO nos tempos estudados. A Ala-Gln como prÃ-tratamento isolado reduziu TBARS plasmÃtico na isquemia e o aumentou no pulmÃo na I/R. Jà no pulmÃo durante isquemia e no plasma na I/R houve reduÃÃo da GSH. O PCI como prÃ-tratamento isolado elevou o TBARS pulmonar na I/R e reduziu a GSH pulmonar na I/R. A associaÃÃo da Ala-Gln e PCI acresceu o TBARS plasmÃtico na isquemia, tambÃm o elevando no pulmÃo e mÃsculo na I/R. Jà a GSH, com os dois prÃ-tratamentos, sofre reduÃÃo plasmÃtica na isquemia e pulmonar na I/R, com elevaÃÃo plasmÃtica na I/R. O presente estudo demonstra que tanto o prÃ-tratamento com Ala-Gln como o PCI protegem contra a lesÃo isquÃmica à distÃncia, em modelo murino de pinÃamento da aorta infra-renal quando avaliado MPO pulmonar. O mesmo nÃo ocorre na lesÃo por I/R. NÃo hà benefÃcio, e sim agravamento de lesÃo à distÃncia pulmonar, na associaÃÃo dos dois prÃ-tratamentos ao mensurar a MPO pulmonar.
The present work determined the effects of pre-treatment with L-alanyl glutamine (Ala-Gln) and ischemic preconditioning (IPC), alone and in combination, against lesions caused by I/R by clamping the infrarenal aorta in rats. Sixty Wistar rats were distributed into five groups (n = 12) divided into two times (n = 6): Control, Group I/R, Group IPC + I/R, Group Ala-Gln + I/R, Group Ala-Gln + IPC + I/R. Times: T1 (infrarenal-aorta clamping ischemia-4h); T2 (ischemia-4h plus reperfusion-1h). Pulmonary myeloperoxidase (MPO) and plasma TBARS concentrations were measured. Data expressed as mean  standard-deviation, analyzed by Studentâs t-test and ANOVA/Tukeyâs post-test. P-values < 0,05 were considered significant. Increased MPO concentrations in ischemic group and in I/R group occurred as compared to control. Reduction in MPO concentrations happened in ischemic groups pre-treated with either Ala-Gln or IPC. I/R induced no change in MPO concentrations in groups pre-treated with either Ala-Gln or IPC. Pre-treating with the two procedures showed increased MPO at both times studied. Reduction in TBARS concentrations occurred in Ala-Gln pre-treated group, whereas significant elevation was observed when Ala-Gln and IPC were associated in ischemic animals. Ischemia/reperfusion induced elevation of plasma TBARS. Pre-treatment with either Ala-Gln or IPC protects against distant pulmonary lesion due to ischemia. The same did not occur in I/R lesion. Combining the two procedures aggravated inflammation indicated by increased MPO concentrations. Elevated TBARS concentrations in ischemic animals pre-treated with the two procedures indicate increased lipid peroxidation, whereas pre-treatment with Ala-Gln induced decreased TBARS concentrations.
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Nälsén, Cecilia. "Measurement and evaluation of antioxidant status and relation to oxidative stress in humans /." Uppsala : Acta Universitatis Upsaliensis : Univ.-bibl. [distributör], 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-6742.

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Silva, Andreza Amaral da [UNESP]. "Estudo clínico, hemagasométrico e do estresse oxidativo em ovinos clinicamente sadios portadores de pneumonia." Universidade Estadual Paulista (UNESP), 2012. http://hdl.handle.net/11449/101285.

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Made available in DSpace on 2014-06-11T19:31:11Z (GMT). No. of bitstreams: 0 Previous issue date: 2012-02-09Bitstream added on 2014-06-13T20:41:36Z : No. of bitstreams: 1 silva_aa_dr_botfmvz.pdf: 1478521 bytes, checksum: 3590a41144246c2ed3a4619905b51919 (MD5)
Fundação de Amparo à Pesquisa do Estado de São Paulo (FAPESP)
Nas espécies domésticas as pneumonias cursam com intensa resposta inflamatória e acúmulo de células fagocíticas nos pulmões, levando a danos expressivos das estruturas do trato respiratório e à função pulmonar devido ao estresse oxidativo decorrente da liberação de grandes quantidades de Espécies Reativas do Oxigênio (ERO) durante a explosão respiratória. O objetivo deste estudo foi analisar o status oxidativo, a resposta inflamatória e a gasometria arterial, de ovinos sadios (n=20) e com diagnóstico clínico de pneumonia (n=20). Inicialmente os animais foram submetidos ao exame clínico e divididos em dois grupos: I) G1/controle, composto pelos animais clinicamente sadios e II) G2, composto pelos animais portadores de pneumonia. O status oxidativo foi avaliado por determinação indireta da atividade enzimática da Superóxido Dismutase (SOD) e Glutationa Peroxidase (GSH-Px) e das concentrações de Glutationa total (GSH-t) e Substâncias Reativas ao Ácido Tiobarbitúrico (TBARS) no sangue periférico por método colorimétrico. A resposta inflamatória foi avaliada pelo hemograma e proteína total e fibrinogênio plasmáticos e a função pulmonar pela determinação das variáveis hemogasométricas Pressão Arterial de Oxigênio (PaO2), Pressão Arterial de Gás Carbônico (PaCO2), Hidrogeniônico (pH), Saturação de Oxigênio (SO2), Bicarbonato (HCO3¯), Dióxido de Carbono Total (TCO2) e Excesso de Bases (EB), avaliados em sangue arterial. O leucograma revelou leucocitose com neutrofilia, eosinofilia, monocitose e linfopenia nos animais doentes (p<0,05). Com relação aos parâmetros bioquímicos, os ovinos portadores de pneumonia apresentaram aumento significativo (p>0,05) da concentração de fibrinogênio e proteína plasmática total. Os animais portadores de pneumonia apresentaram diminuição estatisticamente...
In domestic species, pneumonia is accompanied by intense inflammatory response and accumulation of phagocytic cells in the lungs, causing structural damage of the respiratory tract due to oxidative stress resulting from the release of large amounts of Reactive Oxygen Species (ROS) during the respiratory burst. The aim of this study was to analyze the oxidative status, inflammatory response and arterial blood gases values in healthy sheep (n=20) and animals with a clinically diagnosed pneumonia (n = 20). After physical examination the animals were divided into two groups: i) G1/control, composed of clinically healthy animals and ii) G2, composed of animals with pneumonia. The oxidative status was assessed by indirect determinations of enzymatic activity of superoxide dismutase (SOD) and glutathione peroxidase (GSH-Px) and concentrations of total glutathione (GSH-t) and thiobarbituric acid reactive substances (TBARS) in peripheral blood by a colorimetric method. The inflammatory response was evaluated by complete blood count and total protein and plasma fibrinogen. The lung function was evaluated by determinations of blood gas parameters in arterial blood: Oxygen Pressure (PaO2) Pressure of Carbon Dioxide (PaCO2), Pressure Hydrogen (pH), Oxygen Saturation (SO2), Bicarbonate (HCO3¯), Total Carbon Dioxide (TCO2) and Base Excess (EB). The leucogram results showed Leukocytosis with neutrophilia, eosinophilia, monocytosis and lymphopenia in sick animals (p<0,05). With regard to biochemical parameters, sheep with pneumonia showed a significant increase (p<0,05) of fibrinogen and total plasma protein concentrations. The animals from group G2 had a statistically significant reduction (p<0,05) in SOD and GSH-Px enzymatic activity and GSH-t concentration, while TBARS concentration was significantly higher (p<0,05). Arterial blood... (Complete abstract click electronic access below)
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Silva, Andreza Amaral da. "Estudo clínico, hemagasométrico e do estresse oxidativo em ovinos clinicamente sadios portadores de pneumonia /." Botucatu, 2012. http://hdl.handle.net/11449/101285.

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Orientador: Roberto Calderon Gonçalves
Banca: Simone Biagio Chiacchio
Banca: Raimundo de Souza Lopes
Banca: Fernando José Benesi
Banca: Débora Cristina Damasceno
Resumo: Nas espécies domésticas as pneumonias cursam com intensa resposta inflamatória e acúmulo de células fagocíticas nos pulmões, levando a danos expressivos das estruturas do trato respiratório e à função pulmonar devido ao estresse oxidativo decorrente da liberação de grandes quantidades de Espécies Reativas do Oxigênio (ERO) durante a explosão respiratória. O objetivo deste estudo foi analisar o status oxidativo, a resposta inflamatória e a gasometria arterial, de ovinos sadios (n=20) e com diagnóstico clínico de pneumonia (n=20). Inicialmente os animais foram submetidos ao exame clínico e divididos em dois grupos: I) G1/controle, composto pelos animais clinicamente sadios e II) G2, composto pelos animais portadores de pneumonia. O status oxidativo foi avaliado por determinação indireta da atividade enzimática da Superóxido Dismutase (SOD) e Glutationa Peroxidase (GSH-Px) e das concentrações de Glutationa total (GSH-t) e Substâncias Reativas ao Ácido Tiobarbitúrico (TBARS) no sangue periférico por método colorimétrico. A resposta inflamatória foi avaliada pelo hemograma e proteína total e fibrinogênio plasmáticos e a função pulmonar pela determinação das variáveis hemogasométricas Pressão Arterial de Oxigênio (PaO2), Pressão Arterial de Gás Carbônico (PaCO2), Hidrogeniônico (pH), Saturação de Oxigênio (SO2), Bicarbonato (HCO3¯), Dióxido de Carbono Total (TCO2) e Excesso de Bases (EB), avaliados em sangue arterial. O leucograma revelou leucocitose com neutrofilia, eosinofilia, monocitose e linfopenia nos animais doentes (p<0,05). Com relação aos parâmetros bioquímicos, os ovinos portadores de pneumonia apresentaram aumento significativo (p>0,05) da concentração de fibrinogênio e proteína plasmática total. Os animais portadores de pneumonia apresentaram diminuição estatisticamente... (Resumo completo, clicar acesso eletrônico abaixo)
Abstract: In domestic species, pneumonia is accompanied by intense inflammatory response and accumulation of phagocytic cells in the lungs, causing structural damage of the respiratory tract due to oxidative stress resulting from the release of large amounts of Reactive Oxygen Species (ROS) during the respiratory burst. The aim of this study was to analyze the oxidative status, inflammatory response and arterial blood gases values in healthy sheep (n=20) and animals with a clinically diagnosed pneumonia (n = 20). After physical examination the animals were divided into two groups: i) G1/control, composed of clinically healthy animals and ii) G2, composed of animals with pneumonia. The oxidative status was assessed by indirect determinations of enzymatic activity of superoxide dismutase (SOD) and glutathione peroxidase (GSH-Px) and concentrations of total glutathione (GSH-t) and thiobarbituric acid reactive substances (TBARS) in peripheral blood by a colorimetric method. The inflammatory response was evaluated by complete blood count and total protein and plasma fibrinogen. The lung function was evaluated by determinations of blood gas parameters in arterial blood: Oxygen Pressure (PaO2) Pressure of Carbon Dioxide (PaCO2), Pressure Hydrogen (pH), Oxygen Saturation (SO2), Bicarbonate (HCO3¯), Total Carbon Dioxide (TCO2) and Base Excess (EB). The leucogram results showed Leukocytosis with neutrophilia, eosinophilia, monocytosis and lymphopenia in sick animals (p<0,05). With regard to biochemical parameters, sheep with pneumonia showed a significant increase (p<0,05) of fibrinogen and total plasma protein concentrations. The animals from group G2 had a statistically significant reduction (p<0,05) in SOD and GSH-Px enzymatic activity and GSH-t concentration, while TBARS concentration was significantly higher (p<0,05). Arterial blood... (Complete abstract click electronic access below)
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Ferreira, Paula Souza. "Atividade anti-inflamatória e antioxidante de flavonoides cítricos em camundongos submetidos a dieta indutora do estado pró-inflamatório /." Araraquara, 2014. http://hdl.handle.net/11449/113840.

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Orientador: Thais Borges Cesar
Banca: Amanda Martins Baviera
Banca: Luis Carlos Spolidorio
Resumo: O estresse oxidativo e a inflamação na obesidade estão associados ao desenvolvimento de doenças crônicas, tais como o diabetes mellitus e as doenças cardiovasculares. A ingestão de dietas com alto teor de gorduras saturadas e açúcar, mas deficientes em compostos antioxidantes, contribui para o acúmulo de tecido adiposo e podem levar ao aumento de marcadores inflamatórios no sangue e tecidos. Os flavonoides cítricos possuem propriedades biológicas capazes de atenuar o estresse oxidativo e a inflamação, protegendo contra as desordens metabólicas decorrentes da obesidade e excesso de tecido adiposo. Neste trabalho foi avaliado o efeito da hesperidina, eriocitrina e eriodictiol sobre a inflamação, o estresse oxidativo e alterações no soro, fígado, coração e baço de camundongos induzidos à obesidade com dieta hiperlipídica, contendo 45% de calorias derivadas de lipídios, por quatro semanas. A hesperidina, eriocitrina e eriodictiol inibiram eficientemente o aumento dos níveis séricos de IL-6, MCP-1, proteína C-reativa, e de TBARS no fígado causado pelo consumo da dieta hiperlipídica e excesso de gordura visceral, impedindo o aumento da massa do baço e aumentando a capacidade antioxidante total no soro. A eriocitrina e eriodictiol reduziram também os níveis de TBARS no soro, enquanto o acúmulo de gordura e danos no fígado foram reduzidos pela hesperidina e eriocitrina, e a massa do coração pela hesperidina e eriodictiol. Esses resultados mostram que a hesperidina, eriocitrina e eriodictiol protegem contra a inflamação e estresse oxidativo causados pelo consumo de dieta hiperlipídica e acúmulo de gordura visceral, como indicado pela diminuição dos marcadores inflamatórios, da peroxidação lipídica, esteatose e danos hepáticos, e da massa do baço e coração, sendo bons candidatos para o tratamento das alterações primárias da obesidade, nas quais eles poderiam ajudar a prevenir o desenvolvimento de ...
Abstract: Oxidative stress and inflammation in obesity are associated with the development of chronic diseases such as diabetes mellitus and cardiovascular diseases. The ingestion of diets rich in saturated fatty acids and sugar, but deficient in antioxidants, contributes to adipose tissue accumulation and may lead to increased inflammatory markers in the blood and tissues. Citrus flavonoids have biological properties capable of attenuating oxidative stress and inflammation, protecting against metabolic disorders resulting from obesity and adipose tissue excess. In the present work we assessed the effect of hesperidin, eriocitrin and eriodictyol over inflammation, oxidative stress and the changes resulting from these process in the blood serum, liver, heart and spleen of mice fed a high-fat diet, which contained 45% of calories from fat, for a period of four weeks. Hesperidin, eriocitrin and eriodictyol supplementation efficiently inhibited the increase of serum IL-6, MCP-1 and C-reactive protein, and also the TBARS levels of the liver, caused by high-fat diet ingestion and excessive visceral fat, thus preventing the increase in spleen weight and increasing serum total antioxidant capacity. Eriocitrin and eriodictyol also reduced TBARS levels in the blood serum, while liver fat accumulation and damage were reduced by hesperidin and eriocitrin, and heart weight by hesperidin and eriodictyol. These results show that hesperidin, eriocitrin and eriodictiol have protective effect against inflammation and oxidative stress caused by high-fat diet feeding and visceral obesity, as indicated by reduced liver damage and fat accumulation, and reduced heart and spleen weight, making them good candidates for use in such conditions, in which they could possibly help to prevent cardiovascular diseases ...
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Ferreira, Paula Souza [UNESP]. "Atividade anti-inflamatória e antioxidante de flavonoides cítricos em camundongos submetidos a dieta indutora do estado pró-inflamatório." Universidade Estadual Paulista (UNESP), 2014. http://hdl.handle.net/11449/113840.

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O estresse oxidativo e a inflamação na obesidade estão associados ao desenvolvimento de doenças crônicas, tais como o diabetes mellitus e as doenças cardiovasculares. A ingestão de dietas com alto teor de gorduras saturadas e açúcar, mas deficientes em compostos antioxidantes, contribui para o acúmulo de tecido adiposo e podem levar ao aumento de marcadores inflamatórios no sangue e tecidos. Os flavonoides cítricos possuem propriedades biológicas capazes de atenuar o estresse oxidativo e a inflamação, protegendo contra as desordens metabólicas decorrentes da obesidade e excesso de tecido adiposo. Neste trabalho foi avaliado o efeito da hesperidina, eriocitrina e eriodictiol sobre a inflamação, o estresse oxidativo e alterações no soro, fígado, coração e baço de camundongos induzidos à obesidade com dieta hiperlipídica, contendo 45% de calorias derivadas de lipídios, por quatro semanas. A hesperidina, eriocitrina e eriodictiol inibiram eficientemente o aumento dos níveis séricos de IL-6, MCP-1, proteína C-reativa, e de TBARS no fígado causado pelo consumo da dieta hiperlipídica e excesso de gordura visceral, impedindo o aumento da massa do baço e aumentando a capacidade antioxidante total no soro. A eriocitrina e eriodictiol reduziram também os níveis de TBARS no soro, enquanto o acúmulo de gordura e danos no fígado foram reduzidos pela hesperidina e eriocitrina, e a massa do coração pela hesperidina e eriodictiol. Esses resultados mostram que a hesperidina, eriocitrina e eriodictiol protegem contra a inflamação e estresse oxidativo causados pelo consumo de dieta hiperlipídica e acúmulo de gordura visceral, como indicado pela diminuição dos marcadores inflamatórios, da peroxidação lipídica, esteatose e danos hepáticos, e da massa do baço e coração, sendo bons candidatos para o tratamento das alterações primárias da obesidade, nas quais eles poderiam ajudar a prevenir o desenvolvimento de ...
Oxidative stress and inflammation in obesity are associated with the development of chronic diseases such as diabetes mellitus and cardiovascular diseases. The ingestion of diets rich in saturated fatty acids and sugar, but deficient in antioxidants, contributes to adipose tissue accumulation and may lead to increased inflammatory markers in the blood and tissues. Citrus flavonoids have biological properties capable of attenuating oxidative stress and inflammation, protecting against metabolic disorders resulting from obesity and adipose tissue excess. In the present work we assessed the effect of hesperidin, eriocitrin and eriodictyol over inflammation, oxidative stress and the changes resulting from these process in the blood serum, liver, heart and spleen of mice fed a high-fat diet, which contained 45% of calories from fat, for a period of four weeks. Hesperidin, eriocitrin and eriodictyol supplementation efficiently inhibited the increase of serum IL-6, MCP-1 and C-reactive protein, and also the TBARS levels of the liver, caused by high-fat diet ingestion and excessive visceral fat, thus preventing the increase in spleen weight and increasing serum total antioxidant capacity. Eriocitrin and eriodictyol also reduced TBARS levels in the blood serum, while liver fat accumulation and damage were reduced by hesperidin and eriocitrin, and heart weight by hesperidin and eriodictyol. These results show that hesperidin, eriocitrin and eriodictiol have protective effect against inflammation and oxidative stress caused by high-fat diet feeding and visceral obesity, as indicated by reduced liver damage and fat accumulation, and reduced heart and spleen weight, making them good candidates for use in such conditions, in which they could possibly help to prevent cardiovascular diseases ...
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Collins, Tracey Helen. "Investigation into the Effects of Oxidative Stress on Reproductive Development." The University of Waikato, 2007. http://hdl.handle.net/10289/2364.

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Nuclear transfer (NT), or cloning, which is the transfer of a donor nucleus to a recipient enucleated oocyte, has been successfully achieved to produce viable offspring in many species. The process is very inefficient, as reprogramming of the donor nucleus is required, and losses are high throughout development. Placentation abnormalities are a common feature amongst cloned animals. Incomplete nuclear reprogramming and erroneous epigenetic imprinting may contribute to aberrant protein transcription and DNA mutations, affecting mitochondrial metabolism and inducing cellular stress. In vitro produced embryos under high oxygen culture conditions may also suffer oxidative stress, with the resulting reactive oxygen species causing mitochondrial DNA mutations and cellular stress similar to clones. In this study, expression of oxidative stress protein markers (Hsp60, SOD2, Hsp70) in NT cotyledons were compared to artificial insemination (AI) at different time points of gestation (days 50, 100, and 150). As a continuum of the oxidative stress investigation in cloned cotyledons, in vitro produced embryos were cultured under 20% oxygen compared to the control 7% oxygen laboratory standard culture, with oxidative stress protein markers examined between the groups at blastocyst stage (day 7) and day 15. Embryo morphology was also observed to determine apparent physiological differences between the treatment and control embryos. No previous studies to date have investigated the developmental effects of oxidative stress in day 15 bovine embryos. The significant differences in oxidative stress proteins observed at several time points in the NT and AI groups were not repeatable, possibly due to sample freeze/thaw degradation. Morphological differences observed between embryos cultured in 20% oxygen and control groups were visually apparent, although not quantified. At day 15 manganese superoxide dismutase expression was significantly lower in the 20% group compared to control. The 20% oxygen group did not show higher heat shock protein 60 expression than control, however the same results have been observed in another study at blastocyst stage. The results of this study suggest that the effect of oxidative stress on embryonic development is evident yet inconclusive in bovine NT cotyledons, however does not appear apparent in day 15 embryos following culture in 20% oxygen.
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Rajaraman, Gnana Oli [Verfasser], and Helga [Akademischer Betreuer] Stopper. "Oxidative stress : role in genomic damage and disease = Oxidativer Stress / Gnana Oli Rajaraman. Betreuer: Helga Stopper." Würzburg : Universitätsbibliothek der Universität Würzburg, 2012. http://d-nb.info/1024851885/34.

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Plant, Stuart D. "The response of human umbilical vein endothelial cells and blood platelets to modified NiTi surfaces." Thesis, University of Nottingham, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.275630.

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Books on the topic "Oxidative stess"

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Huang, Canhua, and Yuanyuan Zhang, eds. Oxidative Stress. Singapore: Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-16-0522-2.

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Molecular basis of oxidative stress: Chemistry, mechanisms, and disease pathogenesis. Hoboken, New Jersey: Wiley, 2013.

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Andreescu, Silvana. Oxidative stress: Diagnostics, prevention, and therapy. Edited by American Chemical Society. Division of Analytical Chemistry. Washington, DC: American Chemical Society, 2011.

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Brett, Blackman, and Jo Hanjoong, eds. Hemodynamics and mechanobiology of endothelium. New Jersey: World Scientific, 2010.

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Advanced protocols in oxidative stress II. New York, N.Y: Humana Press, 2009.

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Cutler, R. G., L. Packer, J. Bertram, and A. Mori, eds. Oxidative Stress and Aging. Basel: Birkhäuser Basel, 1995. http://dx.doi.org/10.1007/978-3-0348-7337-6.

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Parvez, H., and P. Riederer, eds. Oxidative Stress and Neuroprotection. Vienna: Springer Vienna, 2006. http://dx.doi.org/10.1007/978-3-211-33328-0.

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Gelpi, Ricardo Jorge, Alberto Boveris, and Juan José Poderoso, eds. Biochemistry of Oxidative Stress. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-45865-6.

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Armstrong, Donald, and Dhruba J. Bharali, eds. Oxidative Stress and Nanotechnology. Totowa, NJ: Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-475-3.

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Miwa, Satomi, Kenneth B. Beckman, and Florian L. Muller, eds. Oxidative Stress in Aging. Totowa, NJ: Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-420-9.

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Book chapters on the topic "Oxidative stess"

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Li, Jingyi, and Rui Liu. "Autophagy in Cellular Stress Responses." In Oxidative Stress, 133–54. Singapore: Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-16-0522-2_6.

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He, Ming-Liang, Qianya Wan, Dan Song, and Betsy He. "Stress Proteins: Biological Functions, Human Diseases, and Virus Infections." In Oxidative Stress, 77–102. Singapore: Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-16-0522-2_4.

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Yang, Ying, Yue Wu, Xiao-Dong Sun, and Yuanyuan Zhang. "Reactive Oxygen Species, Glucose Metabolism, and Lipid Metabolism." In Oxidative Stress, 213–35. Singapore: Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-16-0522-2_9.

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Sun, Meng-Juan, Hai-Yan Yin, Xin Cao, Liang Kang, Yu-Shi Hu, and Yong Tang. "Stress and Circadian Rhythms." In Oxidative Stress, 193–212. Singapore: Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-16-0522-2_8.

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Li, Ting, and Yi Zhun Zhu. "Stress and Inflammation." In Oxidative Stress, 277–91. Singapore: Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-16-0522-2_12.

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Ren, Yi, Charlie Cheong, and Han-Ming Shen. "Oxidative Stress in Cell Signaling and Cell Fate Determination Under Glucose Starvation." In Oxidative Stress, 293–323. Singapore: Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-16-0522-2_13.

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Xie, Na, and Canhua Huang. "Drug Repurposing: An Avenue Toward Stress Medicine in Cancer Therapy." In Oxidative Stress, 237–62. Singapore: Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-16-0522-2_10.

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Nin, Dawn Sijin, Shabana Binte Idres, and Lih-Wen Deng. "Cysteine Metabolism in Cancer Progression and Therapy Resistance." In Oxidative Stress, 155–91. Singapore: Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-16-0522-2_7.

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Lee, Yew Mun, Dongxue Hu, and Yih-Cherng Liou. "Importance of Mitochondrial Quality Control in Parkinson’s Disease: The Potential Interplay of Mitochondrial Unfolded Protein Response and Mitophagy." In Oxidative Stress, 103–31. Singapore: Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-16-0522-2_5.

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Yuan, Kefei, and Yuanyuan Zhang. "Oxidative Stress and Antioxidant Strategies in Human Diseases." In Oxidative Stress, 1–26. Singapore: Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-16-0522-2_1.

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Conference papers on the topic "Oxidative stess"

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Sridharan, Sriram, Ritwik Layek, Aniruddha Datta, and Jijayanagaram Venkatraj. "Modelling oxidative stress response pathways." In 2011 IEEE International Workshop on Genomic Signal Processing and Statistics (GENSIPS). IEEE, 2011. http://dx.doi.org/10.1109/gensips.2011.6169471.

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Liu, Junhua, Zhenhua Wang, Bao Ju, and Qiusheng Zheng. "Heroin-Induced Hepatotoxicity: Involved Oxidative Stress." In 2008 2nd International Conference on Bioinformatics and Biomedical Engineering (ICBBE '08). IEEE, 2008. http://dx.doi.org/10.1109/icbbe.2008.272.

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Gea, J., S. Mas, E. Balcells, J. Sauleda, F. Gomez, J. Galdiz, E. Monso, et al. "Systemic Oxidative Stress and COPD Phenotypes." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a4329.

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Shikov, A. E., V. V. Lastochkin, T. V. Chirkova, and V. V. Emelyanov. "Oxidative damage to plant lipids and proteins bynatural and artificial oxidative stress." In IX Congress of society physiologists of plants of Russia "Plant physiology is the basis for creating plants of the future". Kazan University Press, 2019. http://dx.doi.org/10.26907/978-5-00130-204-9-2019-481.

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Avramouli, Antigoni, Georgia Theocharopoulou, and Panayiotis Vlamos. "Detection of oxidative stress in neurodegenerative diseases." In 2015 IEEE International Symposium on Signal Processing and Information Technology (ISSPIT). IEEE, 2015. http://dx.doi.org/10.1109/isspit.2015.7394366.

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Xu, Weiling, Suzy A. A. Comhair, Deepa George, Nayra Cardenes, Sruti Shiva, Christina Kao, Samuel H. Wedes, et al. "Mitochondrial Function And Oxidative Stress In Asthma." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a5653.

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Jassal, R., D. Talwar, D. Mcmillan, and S. Tovey. "Biomarkers of Oxidative Stress in Breast Cancer." In Abstracts: Thirty-Second Annual CTRC‐AACR San Antonio Breast Cancer Symposium‐‐ Dec 10‐13, 2009; San Antonio, TX. American Association for Cancer Research, 2009. http://dx.doi.org/10.1158/0008-5472.sabcs-09-2141.

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Keyhani, J., E. Keyhani, F. Attar, and M. Hadizadeh. "Anti-oxidative stress enzymes in Pleurotus ostreatus." In Proceedings of the II International Conference on Environmental, Industrial and Applied Microbiology (BioMicroWorld2007). WORLD SCIENTIFIC, 2009. http://dx.doi.org/10.1142/9789812837554_0001.

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Kawai, K., Y.-S. Li, Y. Kawasaki, H. Kasai, S. Watanabe, H. Yamato, T. Honda, and M. Ohta. "1218 Salivary oxidative stress biomarker: 8-hydroxyguanosine." In 32nd Triennial Congress of the International Commission on Occupational Health (ICOH), Dublin, Ireland, 29th April to 4th May 2018. BMJ Publishing Group Ltd, 2018. http://dx.doi.org/10.1136/oemed-2018-icohabstracts.1158.

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Bayer, Rainer, and Gerd Wasser. "Effects of oxidative stress on erythrocyte deformability." In Photonics West '96, edited by Daniel L. Farkas, Robert C. Leif, Alexander V. Priezzhev, Toshimitsu Asakura, and Bruce J. Tromberg. SPIE, 1996. http://dx.doi.org/10.1117/12.239518.

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Reports on the topic "Oxidative stess"

1

Paul, Satashree. Oxidative Stress: A Cause of Male Infertility. Science Repository OÜ, October 2020. http://dx.doi.org/10.31487/sr.blog.10.

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Zhao, Hua. Oxidative Stress, DNA Repair and Prostate Cancer Risk. Fort Belvoir, VA: Defense Technical Information Center, August 2009. http://dx.doi.org/10.21236/ada518882.

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Zhao, Hua. Oxidative Stress, DNA Repair, and Prostate Cancer Risk. Fort Belvoir, VA: Defense Technical Information Center, August 2011. http://dx.doi.org/10.21236/ada561087.

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zhao, Hua. Oxidative Stress, DNA Repair and Prostate Cancer Risk. Fort Belvoir, VA: Defense Technical Information Center, August 2010. http://dx.doi.org/10.21236/ada542698.

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Madaeva, I. M., N. A. Kurashova, N. V. Semenova, E. B. Uhinov, S. I. Kolesnikov, and L. I. Kolesnikova. HSP70 HEAT SHOCK PROTEIN IN OXIDATIVE STRESS APNEA PATIENTS. Publishing house of the Russian Academy of Medical Sciences, 2020. http://dx.doi.org/10.18411/1695-1978-2020-62730.

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Li, Jiaxiao, Suling Liu, and Yang Cui. Oxidative and Anti-oxidative stress-linked biomarkers in Ankylosing Spondylitis: a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, May 2020. http://dx.doi.org/10.37766/inplasy2020.5.0066.

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Carey, Hannah V. The Adaptive Response to Intestinal Oxidative Stress in Mammalian Hibernation. Fort Belvoir, VA: Defense Technical Information Center, January 2006. http://dx.doi.org/10.21236/ada442363.

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Madaev, I. M., N. A. Kurashova, N. V. Semenova, E. B. Ukhinov, S. I. Kolesnikov, and L. I. Kolesnikova. Heat shock protein HSP70 for oxidative stress in patients with apnea. Federal State Budgetary Institution Scientific Center, 2020. http://dx.doi.org/10.18411/1695-2608-2020-62730.

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Irene, E. A., and E. Kobeda. In-Situ Stress Measurements during Thermal Oxidation of Silicon. Fort Belvoir, VA: Defense Technical Information Center, March 1989. http://dx.doi.org/10.21236/ada206546.

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Wright, Austin. Small RNA MgrR Regulates Sensitivity of Escherichia fergusonii to Oxidative Stress. Portland State University Library, January 2000. http://dx.doi.org/10.15760/etd.6617.

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