Relevant bibliographies by topics / Oxidative Stress – drug therapy

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Oxidative Stress – drug therapy'

Author: Grafiati
Published: 4 June 2021
Last updated: 17 February 2022

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Journal articles on the topic "Oxidative Stress – drug therapy"

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Hanikoglu, Aysegul, Hakan Ozben, Ferhat Hanikoglu, and Tomris Ozben. "Hybrid Compounds & Oxidative Stress Induced Apoptosis in Cancer Therapy." Current Medicinal Chemistry 27, no. 13 (2020): 2118–32. http://dx.doi.org/10.2174/0929867325666180719145819.

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: Elevated Reactive Oxygen Species (ROS) generated by the conventional cancer therapies and the endogenous production of ROS have been observed in various types of cancers. In contrast to the harmful effects of oxidative stress in different pathologies other than cancer, ROS can speed anti-tumorigenic signaling and cause apoptosis of tumor cells via oxidative stress as demonstrated in several studies. The primary actions of antioxidants in cells are to provide a redox balance between reduction-oxidation reactions. Antioxidants in tumor cells can scavenge excess ROS, causing resistance to ROS i
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Geisser, P. "Iron Therapy and Oxidative Stress." Metal-Based Drugs 4, no. 3 (1997): 137–52. http://dx.doi.org/10.1155/mbd.1997.137.

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Coppolino, Giuseppe, Giuseppe Leonardi, Michele Andreucci, and Davide Bolignano. "Oxidative Stress and Kidney Function: A Brief Update." Current Pharmaceutical Design 24, no. 40 (2019): 4794–99. http://dx.doi.org/10.2174/1381612825666190112165206.

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In this review we summarized some information regarding the link between kidney and oxidative stress. Accruing evidences indicated the kidney as a fundamental organ in reactive oxygen species (ROS) production. ROS are highly reactive and cause in single cells: protein alteration, DNA damage, cellular senescence and apoptosis; while the effect of ROS in biological tissues leads to a harmful oxidation effect on all their biochemical components: lipids, proteins, carbohydrates, and nucleic acids. Oxidative stress plays a role in the pathophysiology of renal impairment and is a mediator of CKD pro
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Yamada, Yuma, Yuta Takano, Satrialdi, Jiro Abe, Mitsue Hibino, and Hideyoshi Harashima. "Therapeutic Strategies for Regulating Mitochondrial Oxidative Stress." Biomolecules 10, no. 1 (2020): 83. http://dx.doi.org/10.3390/biom10010083.

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There have been many reports on the relationship between mitochondrial oxidative stress and various types of diseases. This review covers mitochondrial targeting photodynamic therapy and photothermal therapy as a therapeutic strategy for inducing mitochondrial oxidative stress. We also discuss other mitochondrial targeting phototherapeutic methods. In addition, we discuss anti-oxidant therapy by a mitochondrial drug delivery system (DDS) as a therapeutic strategy for suppressing oxidative stress. We also describe cell therapy for reducing oxidative stress in mitochondria. Finally, we discuss t
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Barrera, Giuseppina. "Oxidative Stress and Lipid Peroxidation Products in Cancer Progression and Therapy." ISRN Oncology 2012 (October 17, 2012): 1–21. http://dx.doi.org/10.5402/2012/137289.

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The generation of reactive oxygen species (ROS) and an altered redox status are common biochemical aspects in cancer cells. ROS can react with the polyunsaturated fatty acids of lipid membranes and induce lipid peroxidation. The end products of lipid peroxidation, 4-hydroxynonenal (HNE), have been considered to be a second messenger of oxidative stress. Beyond ROS involvement in carcinogenesis, increased ROS level can inhibit tumor cell growth. Indeed, in tumors in advanced stages, a further increase of oxidative stress, such as that occurs when using several anticancer drugs and radiation the
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Zhu, Jinghan, Yixiao Xiong, Yuxin Zhang, et al. "The Molecular Mechanisms of Regulating Oxidative Stress-Induced Ferroptosis and Therapeutic Strategy in Tumors." Oxidative Medicine and Cellular Longevity 2020 (December 21, 2020): 1–14. http://dx.doi.org/10.1155/2020/8810785.

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Ferroptosis is an atypical form of regulated cell death, which is different from apoptosis, necrosis, pyroptosis, and autophagy. Ferroptosis is characterized by iron-dependent oxidative destruction of cellular membranes following the antioxidant system’s failure. The sensitivity of ferroptosis is tightly regulated by a series of biological processes, the metabolism of iron, amino acids, and polyunsaturated fatty acids, and the interaction of glutathione (GSH), NADPH, coenzyme Q10 (CoQ10), and phospholipids. Elevated oxidative stress (ROS) level is a hallmark of cancer, and ferroptosis serves a
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Kim, Su Ji, Hyun Soo Kim, and Young Rok Seo. "Understanding of ROS-Inducing Strategy in Anticancer Therapy." Oxidative Medicine and Cellular Longevity 2019 (December 20, 2019): 1–12. http://dx.doi.org/10.1155/2019/5381692.

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Redox homeostasis is essential for the maintenance of diverse cellular processes. Cancer cells have higher levels of reactive oxygen species (ROS) than normal cells as a result of hypermetabolism, but the redox balance is maintained in cancer cells due to their marked antioxidant capacity. Recently, anticancer therapies that induce oxidative stress by increasing ROS and/or inhibiting antioxidant processes have received significant attention. The acceleration of accumulative ROS disrupts redox homeostasis and causes severe damage in cancer cells. In this review, we describe ROS-inducing cancer
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Pauletti, Alberto, Gaetano Terrone, Tawfeeq Shekh-Ahmad, et al. "Targeting oxidative stress improves disease outcomes in a rat model of acquired epilepsy." Brain 142, no. 7 (2019): e39-e39. http://dx.doi.org/10.1093/brain/awz130.

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AbstractEpilepsy therapy is based on antiseizure drugs that treat the symptom, seizures, rather than the disease and are ineffective in up to 30% of patients. There are no treatments for modifying the disease—preventing seizure onset, reducing severity or improving prognosis. Among the potential molecular targets for attaining these unmet therapeutic needs, we focused on oxidative stress since it is a pathophysiological process commonly occurring in experimental epileptogenesis and observed in human epilepsy. Using a rat model of acquired epilepsy induced by electrical status epilepticus, we s
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Tangvarasittichai, Orathai, and Surapon Tangvarasittichai. "Oxidative Stress, Ocular Disease and Diabetes Retinopathy." Current Pharmaceutical Design 24, no. 40 (2019): 4726–41. http://dx.doi.org/10.2174/1381612825666190115121531.

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Background: Oxidative stress is caused by free radicals or oxidant productions, including lipid peroxidation, protein modification, DNA damage and apoptosis or cell death and results in cellular degeneration and neurodegeneration from damage to macromolecules. Results: Accumulation of the DNA damage (8HOdG) products and the end products of LPO (including aldehyde, diene, triene conjugates and Schiff’s bases) were noted in the research studies. Significantly higher levels of these products in comparison with the controls were observed. Oxidative stress induced changes to ocular cells and tissue
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Pastore, Donatella, Francesca Pacifici, Giampaolo Ciao, Valentina Bedin, Guido Pasquantonio, and David Della-Morte. "Far Infrared Technology (FIT) Therapy Patches, Protects from Inflammation, Oxidative Stress and Promotes Cellular Vitality." Current Pharmaceutical Design 26, no. 34 (2020): 4323–29. http://dx.doi.org/10.2174/1381612826666200427112023.

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Background: It is known from the most recent literature that far-infrared (FIR) radiations promote a broad spectrum of therapeutic benefits for cells and tissues. Objective: To identify molecular mechanisms by which FIT patches, as a far infrared technology, protects against damage caused by inflammatory process and oxidative stress. Methods: Endothelial cells (HUVEC, Human Umbilical Vein Endothelial Cells) were used as in vitro experimental model. HUVEC were stimulated with a pro-inflammatory cytokine, TNF-α, or hydrogen peroxide (H2O2) to induce oxidative stress. As markers of inflammation w
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Dissertations / Theses on the topic "Oxidative Stress – drug therapy"

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Iyer, Gokulakrishnan Seshadri. "Superoxide dismutase delivery and cardiac progenitor cell characterization for myocardial regeneration applications." Diss., Georgia Institute of Technology, 2011. http://hdl.handle.net/1853/42846.

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Cardiovascular diseases are the leading cause of death throughout the world and various estimates predict that heart disease will remain the number one killer in the world. Pharmacotherapies have not shown significant long term survival benefits to the patients, therefore alternate therapeutic strategies such as bioactive agent delivery and cell therapy based approaches are being investigated. One of the major causes of heart failure is the disease progression after an ischemic event and any successful therapy will be needed over the course of several days/weeks. Oxidative stress is greatly i
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DHALIWAL, PARVIN. "PARKINSON'S GDNF THERAPY AND OXIDATIVE STRESS." Thesis, The University of Arizona, 2008. http://hdl.handle.net/10150/190438.

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Homer, Natalie. "Erythocyte oxidative stress : focus on hormone replacement therapy." Thesis, University of Strathclyde, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.273852.

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Vyas, Piyush Manhur. "Sulfonamide-induced cutaneous drug reactions: role of bioactivation, oxidative stress and folate deficiency." Diss., University of Iowa, 2006. https://ir.uiowa.edu/etd/81.

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Sulfonamide- and sulfone-induced hypersensitivity reactions are thought to be mediated through bioactivation of parent drug molecule(s) to their respective reactive metabolite(s). In order to explain the cutaneous drug reactions caused by sulfonamides and sulfone, a mechanism can be proposed by which the bioactivation of these drugs in keratinocytes of the skin forms reactive hydroxylamine metabolites that can covalently bind to cellular proteins, which in turn act as antigens leading to the cascade of immune reactions resulting in a cutaneous drug reaction.
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Linden, Melissa. "The effects of exercise and fish oil on oxidative stress." Diss., Columbia, Mo. : University of Missouri-Columbia, 2006. http://hdl.handle.net/10355/5909.

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Thesis (M.A.)--University of Missouri-Columbia, 2006.<br>The entire dissertation/thesis text is included in the research.pdf file; the official abstract appears in the short.pdf file (which also appears in the research.pdf); a non-technical general description, or public abstract, appears in the public.pdf file. Title from title screen of research.pdf file (viewed on August 30, 2007) Includes bibliographical references.
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Sappy, Immaculate. "Ribonucleic Acids in Disease Etiology and Drug Discovery." University of Toledo Health Science Campus / OhioLINK, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=mco1566562465233197.

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Johansson, Katarina. "Microsomal glutathione transferase 1 in anti-cancer drug resistance and protection against oxidative stress." Stockholm, 2010. http://diss.kib.ki.se/2010/978-91-7409-832-7/.

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Tang, Jingjie. "Innovative imaging systems and novel drug candidates for cancer therapy." Thesis, Aix-Marseille, 2016. http://www.theses.fr/2016AIXM4021.

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Le cancer est l'une des principales causes de décès dans le monde et reste une maladie difficile à traiter du fait des difficultés de pronostic, du développement rapide de métastases et de la résistance aux médicaments. Il en résulte une forte demande en méthodologies d'imagerie innovantes pour le diagnostic précoce et précis ainsi qu’en nouveaux agents anticancéreux possédant de nouveaux mécanismes pour surmonter la résistance aux médicaments. Le but de mon projet de recherche de doctorat était donc de contribuer à cet objectif.La première partie de ma thèse de doctorat a porté sur la créatio
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Turbyville, Thomas Jefferson. "Using Phylogenetically Conserved Stress Responses to Discover Natural Products with Anticancer Activity." Diss., Tucson, Arizona : University of Arizona, 2005. http://etd.library.arizona.edu/etd/GetFileServlet?file=file:///data1/pdf/etd/azu%5Fetd%5F1078%5F1%5Fm.pdf&type=application/pdf.

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Kanase, Nilesh. "The impact of oxidative stress and potential antioxidant therapy on function and survival of cultured pancreatic β-islet cells". Thesis, University of the Highlands and Islands, 2011. https://pure.uhi.ac.uk/portal/en/studentthesis/the-impact-of-oxidative-stress-and-potential-antioxidant-therapy-on-function-and-survival-of-cultured-pancreatic-islet-cells(ec0cd703-3902-4410-8c58-e7c7e49f33e7).html.

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Dietary antioxidant curcumin derived from turmeric has been suggested to decrease the risk of many chronic diseases. Much of the existing data for curcumin stem from experiments performed at supra-physiological concentrations (μM-mM) that are impossible to attain through oral ingestion. It was therefore hypothesized that curcumin at low plasma achievable concentration, though itself not acting as a direct antioxidant might up-regulate the intracellular antioxidants and thus helping combat oxidative stress and protect β-islet cells. The results indicated that Curcumin, DMC and BDMC were able to
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Books on the topic "Oxidative Stress – drug therapy"

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Andreescu, Silvana. Oxidative stress: Diagnostics, prevention, and therapy. Edited by American Chemical Society. Division of Analytical Chemistry. American Chemical Society, 2011.

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Andreescu, Silvana, and Maria Hepel, eds. Oxidative Stress: Diagnostics, Prevention, and Therapy. American Chemical Society, 2011. http://dx.doi.org/10.1021/bk-2011-1083.

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Spitz, Douglas R., Kenneth J. Dornfeld, Koyamangalath Krishnan, and David Gius, eds. Oxidative Stress in Cancer Biology and Therapy. Humana Press, 2012. http://dx.doi.org/10.1007/978-1-61779-397-4.

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Hepel, Maria, and Silvana Andreescu, eds. Oxidative Stress: Diagnostics, Prevention, and Therapy Volume 2. American Chemical Society, 2015. http://dx.doi.org/10.1021/bk-2015-1200.

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Geisser, Peter. Iron therapy: With special emphasis on oxidative stress. P. Geisser, 1998.

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Kong, Ah-Ng Tony. Inflammation, oxidative stress, and cancer: Dietary approaches for cancer prevention. Taylor & Francis, 2014.

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1947-, Martin John E., and Dubbert Patricia M, eds. Non-drug treatments for essential hypertension. Pergamon Press, 1988.

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1935-, Benson Herbert, O'Neill Brian E, and Harvard Medical School, eds. The Harvard Medical School guide to lowering your blood pressure. McGraw-Hill, 2006.

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The oxytocin factor: Tapping the hormone of calm, love, and healing. Da Capo Press, 2003.

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Stahl, S. M. Stahl's illustrated anxiety, stress, and PTSD. Cambridge University Press, 2010.

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Book chapters on the topic "Oxidative Stress – drug therapy"

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Xie, Na, and Canhua Huang. "Drug Repurposing: An Avenue Toward Stress Medicine in Cancer Therapy." In Oxidative Stress. Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-16-0522-2_10.

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Zamboni, William C., and Ninh M. La-Beck. "Carrier-Mediated and Targeted Cancer Drug Delivery." In Oxidative Stress in Cancer Biology and Therapy. Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61779-397-4_21.

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Nin, Dawn Sijin, Shabana Binte Idres, and Lih-Wen Deng. "Cysteine Metabolism in Cancer Progression and Therapy Resistance." In Oxidative Stress. Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-16-0522-2_7.

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Bocci, Velio. "How is Oxidative Stress Assessed?" In Oxygen-Ozone Therapy. Springer Netherlands, 2002. http://dx.doi.org/10.1007/978-94-015-9952-8_10.

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Brazeau, G. A. "Drug-induced muscle damage." In Oxidative Stress in Skeletal Muscle. Birkhäuser Basel, 1998. http://dx.doi.org/10.1007/978-3-0348-8958-2_19.

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Sawyer, D. B., and W. S. Colucci. "Oxidative stress in heart failure." In Molecular Approaches to Heart Failure Therapy. Steinkopff, 2000. http://dx.doi.org/10.1007/978-3-642-57710-9_18.

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Cullen, Joseph J. "Oxidative Stress and Pancreatic Cancer." In Oxidative Stress in Cancer Biology and Therapy. Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61779-397-4_13.

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Shan, Weihua, Weixiong Zhong, Jamie Swanlund, and Terry D. Oberley. "Oxidative Stress in Prostate Cancer." In Oxidative Stress in Cancer Biology and Therapy. Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61779-397-4_15.

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Bocci, Velio. "The Adaptation to Chronic Oxidative Stress (COS)." In Oxygen-Ozone Therapy. Springer Netherlands, 2002. http://dx.doi.org/10.1007/978-94-015-9952-8_22.

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Hatcher, Heather C., Frank M. Torti, and Suzy V. Torti. "Curcumin, Oxidative Stress, and Cancer Therapy." In Oxidative Stress in Cancer Biology and Therapy. Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61779-397-4_12.

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Conference papers on the topic "Oxidative Stress – drug therapy"

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Ghanian, Zahra, Ganesh Kondouri, and Mahsa Ranji. "Optical Studies of Oxidative Stress in Persistent Pulmonary Hypertension Cells." In Optical Molecular Probes, Imaging and Drug Delivery. OSA, 2015. http://dx.doi.org/10.1364/omp.2015.ow3d.6.

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Alcazar Ramirez, J., M. Murri Pierri, F. Linde de Luna, R. Garcia-Delgado, F. Cardona, and F. Tinahones. "CPAP Therapy Reduce Oxidative Stress without Affecting Insulin-Resistance." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a5315.

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Walter, Alec B., Eric P. Skaar, and E. Duco Jansen. "Effects of 690nm light on the response of Staphylococcus aureus to oxidative stress (Conference Presentation)." In Mechanisms of Photobiomodulation Therapy XV, edited by Michael R. Hamblin, James D. Carroll, and Praveen Arany. SPIE, 2020. http://dx.doi.org/10.1117/12.2544401.

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Huang, Ying-Ying, Clark E. Tedford, Thomas McCarthy, and Michael R. Hamblin. "Low level laser therapy reduces oxidative stress in cortical neurons in vitro." In SPIE BiOS, edited by Michael R. Hamblin, Juanita Anders, and James D. Carroll. SPIE, 2012. http://dx.doi.org/10.1117/12.912948.

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Aryal, Baikuntha P., Jean-Pierre Gillet, and V. Ashutosh Rao. "Abstract 5842: Oxidative stress and protein carbonylation towards multi-drug resistance in cancer." In Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-5842.

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Gillis, Katherine, Julie Clor, Kimvan Tran, Asima Khan, and Kamala Tyagarajan. "Abstract 1738: Multiparametric approaches to evaluate mitochondrial toxicity, oxidative stress and apoptosis in drug discovery studies." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-1738.

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Terasaki, Yasuhiro, Kang Dedong, Naomi Kuwahara, et al. "Hydrogen Therapy Attenuates Radiation-induced Pulmonary Injury In C57/Bl 6 Mice Through Reducing Oxidative Stress." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a6356.

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KULKARNI, RASHMI. "A SIMPLE LOGISTIC SIGMOIDAL MODEL PREDICTS OXIDATIVE STRESS THRESHOLDS IN NEWLY DIAGNOSED DIABETICS ON GLUCOSE CONTROL THERAPY." In 15th International Symposium on Mathematical and Computational Biology. WORLD SCIENTIFIC, 2016. http://dx.doi.org/10.1142/9789813141919_0024.

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Okagu, Innocent, Joseph Ndefo, Christian Chibwuogwu, Emmanuel Aham, and Amarachukwu Onoh. "Modulation of Hydrogen Peroxide-Induced Oxidative Stress in Rats by Deep Root Herbal Mixture®—A Nigerian Branded Polyherbal Drug." In The 1st International E-Conference on Antioxidants in Health and Disease. MDPI, 2020. http://dx.doi.org/10.3390/cahd2020-08562.

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Yu, Miao, and Alisa Morss Clyne. "Dextran and PEG Coating Reduced Nanoparticle Toxicity to Cells." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80819.

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Iron oxide nanoparticles are of interest for drug delivery, since they can be targeted using a magnetic field. However, prior to using nanoparticles in vivo, they must be shown as relatively non-toxic to cells. We and others have shown that bare iron oxide nanoparticles are readily taken up by cells, where they catalyze production of highly toxic reactive oxygen species (ROS). This oxidative stress disrupts the cell cytoskeleton and alters cell mechanics. [1] Iron oxide nanoparticles under current development for in vivo biomedical applications are often coated with a polysaccharide (eg. dextr
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Reports on the topic "Oxidative Stress – drug therapy"

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Gomer, Charles J. Photodynamic Therapy Oxidative Stress as a Molecular Switch Controlling Therapeutic Gene Expression for the Treatment of Locally Recurrent Breast Carcinoma. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada396793.

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