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Books on the topic 'Oxidative Stress – drug therapy'

Author: Grafiati

Published: 4 June 2021

Last updated: 17 February 2022

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1

Andreescu, Silvana. Oxidative stress: Diagnostics, prevention, and therapy. Edited by American Chemical Society. Division of Analytical Chemistry. American Chemical Society, 2011.

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2

Andreescu, Silvana, and Maria Hepel, eds. Oxidative Stress: Diagnostics, Prevention, and Therapy. American Chemical Society, 2011. http://dx.doi.org/10.1021/bk-2011-1083.

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3

Spitz, Douglas R., Kenneth J. Dornfeld, Koyamangalath Krishnan, and David Gius, eds. Oxidative Stress in Cancer Biology and Therapy. Humana Press, 2012. http://dx.doi.org/10.1007/978-1-61779-397-4.

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4

Hepel, Maria, and Silvana Andreescu, eds. Oxidative Stress: Diagnostics, Prevention, and Therapy Volume 2. American Chemical Society, 2015. http://dx.doi.org/10.1021/bk-2015-1200.

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5

Geisser, Peter. Iron therapy: With special emphasis on oxidative stress. P. Geisser, 1998.

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6

Kong, Ah-Ng Tony. Inflammation, oxidative stress, and cancer: Dietary approaches for cancer prevention. Taylor & Francis, 2014.

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7

1947-, Martin John E., and Dubbert Patricia M, eds. Non-drug treatments for essential hypertension. Pergamon Press, 1988.

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8

1935-, Benson Herbert, O'Neill Brian E, and Harvard Medical School, eds. The Harvard Medical School guide to lowering your blood pressure. McGraw-Hill, 2006.

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9

The oxytocin factor: Tapping the hormone of calm, love, and healing. Da Capo Press, 2003.

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10

Stahl, S. M. Stahl's illustrated anxiety, stress, and PTSD. Cambridge University Press, 2010.

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11

Cancer: Oxidative Stress and Dietary Antioxidants. Elsevier Science & Technology Books, 2014.

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12

Diabetes: Oxidative Stress and Dietary Antioxidants. Elsevier Science & Technology Books, 2013.

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13

Carotenoids and Vitamin a in Translational Medicine Oxidative Stress and Disease. CRC Press, 2012.

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14

Preedy, Victor R., and Vinood B. Patel. Cancer: Oxidative Stress and Dietary Antioxidants. Elsevier Science & Technology Books, 2021.

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15

Preedy, Victor R. Cancer: Oxidative Stress and Dietary Antioxidants. Elsevier Science & Technology Books, 2014.

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16

Oxidative Stress In Cancer Biology And Therapy. Humana Press, 2011.

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17

Fuchs, Jürgen, 1957 June 28- and Ochsendorf F. R, eds. Oxidative stress in male infertility. Gardez Verlag, 1997.

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18

Oxidative Stress: Diagnostics, Prevention, and Therapy Volume 2. American Chemical Society, 2016.

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19

Carvalho, André F., Gilberto S. Alves, Cristiano A. Köhler, and Roger S. McIntyre. Cognitive Enhancement in Major Depressive Disorder. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190214401.003.0010.

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Major depressive disorder (MDD) is a chronic and disabling illness often associated with elevated rates of non-recovery and substantial psychosocial burden. Cognitive impairment is a common residual manifestations of MDD. Overactivation of the hypothalamic–pituitary–adrenal axis, along with immune–inflammatory imbalances, a decrease in neurotrophin signaling, and an increase in oxidative and nitrosative stress, leads to neuroprogression and cognitive deterioration in MDD. “Cognitive remission” has been proposed as a novel treatment target for MDD. Cognitive remediation therapy has provided enc

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20

Free radicals in ophthalmic disorders. Informa Healthcare, 2008.

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21

(Editor), Manfred Zierhut, Enrique Cadenas (Editor), and Narsing Rao (Editor), eds. Free Radicals in Ophthalmic Disorders. Informa Healthcare, 2008.

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22

(Editor), Klaus Kramer, Peter-Paul Hoppe (Editor), and Lester Packer (Editor), eds. Nutraceuticals in Health and Disease Prevention (Oxidative Stress and Disease). CRC, 2001.

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23

(Editor), Young-Joon Surh, Zigang Dong (Editor), Enrique Cadenas (Editor), and Lester Packer (Editor), eds. Dietary Modulation of Cell Signaling Pathways (Oxidative Stress and Disease). CRC, 2008.

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24

Reddy, Ravinder M. D. Fatty Acids and Oxidative Stress in Neuropsychiatric Disorders. Nova Science Pub Inc, 2007.

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25

Kong, Ah-Ng Tony. Inflammation, Oxidative Stress, and Cancer: Dietary Approaches for Cancer Prevention. Taylor & Francis Group, 2016.

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26

Farooqui, Akhlaq A. Inflammation and Oxidative Stress in Neurological Disorders: Effect of Lifestyle, Genes, and Age. Springer, 2014.

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27

Nutrition and diabetes: Pathophysiology and management. CRC/Taylor & Francis, 2006.

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28

C, Opara Emmanuel, ed. Nutrition & diabetes: Pathophysiology & management. Taylor and Francis, 2005.

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29

Opara, Emmanuel. Nutrition and Diabetes: Pathophysiology and Management. CRC, 2005.

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30

Viegas, Claudio Jr, Carlos Alberto Manssour Fraga, Maria Emília Sousa, and Andrea Tarozzi, eds. Oxidative Stress: How has it been Considered in the Design of New Drug Candidates for Neurodegenerative Diseases? Frontiers Media SA, 2021. http://dx.doi.org/10.3389/978-2-88966-417-7.

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31

Antioxidants in Food, Vitamins and Supplements: Prevention and Treatment of Disease. Elsevier Science & Technology Books, 2014.

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32

R, Connor James, ed. Metals and oxidative damage in neurological disorders. Plenum Press, 1997.

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33

Friedman, Matthew J., American Psychological Association Staff, and Nancy C. Bernardy. Practical Guide to PTSD Treatment: Pharmacological and Psychotherapeutic Approaches. American Psychological Association, 2014.

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34

C, Pappas A., ed. Cadmium toxicity and the antioxidant system. Nova Science, 2010.

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35

1943-, Giller Earl L., and American Psychiatric Association Meeting, eds. Biological assessment and treatment of posttraumatic stress disorder. American Psychiatric Press, 1990.

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36

Studies On Arthritis And Joint Disorders. Springer-Verlag New York Inc., 2012.

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37

D, Armstrong, and International Symposium on Free Radicals in Diagnostic Medicine (1st : 1993 : Buffalo, N.Y.), eds. Free radicals in diagnostic medicine: A systems approach to laboratory technology, clinical correlations, and antioxidant therapy. Plenum Press, 1994.

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38

A, Courtois Christine, and Ford Julian D. 1951-, eds. Treating complex traumatic stress disorders: An evidence-based guide. Guilford Press, 2009.

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39

Free Radicals in Diagnostic Medicine: A Systems Approach to Laboratory Technology, Clinical Correlations and Antioxidant Therapy (Advances in Experimental Medicine and Biology). Springer, 1995.

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40

Bessel A. Van der Kolk. Treating Complex Traumatic Stress Disorders: Scientific Foundations and Therapeutic Models. Guilford Publications, 2013.

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41

Freeman, Marcus. Overcome Anxiety: Improve Social Skills, Stress Relief, and Wellness with News Habits, and Massage Stimulation. 3 in 1 Vagus Nerve+Panic Attacks+CBT for Anxiety. Reboot Your Brain with Drug-Free Therapy. Independently Published, 2020.

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42

Salem, Harry, and Steven Baskin. Oxidants, Antioxidants and Free Radicals. Taylor & Francis Group, 2017.

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43

Salem, Harry, and Steven Baskin. Oxidants, Antioxidants and Free Radicals. Taylor & Francis Group, 2017.

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44

Salem, Harry, and Steven Baskin. Oxidants, Antioxidants and Free Radicals. Taylor & Francis Group, 2017.

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45

I, Baskin Steven, and Salem Harry 1929-, eds. Oxidants, antioxidants, and free radicals. Taylor & Francis, 1997.

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46

Salem, Harry, and Steven Baskin. Oxidants, Antioxidants and Free Radicals. Taylor & Francis Group, 2017.

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47

Vogelnest, Larry, and Timothy Portas, eds. Current Therapy in Medicine of Australian Mammals. CSIRO Publishing, 2019. http://dx.doi.org/10.1071/9781486307524.

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Current Therapy in Medicine of Australian Mammals provides an update on Australian mammal medicine. Although much of the companion volume, Medicine of Australian Mammals, is still relevant and current, there have been significant advances in Australian mammal medicine and surgery since its publication in 2008. The two texts together remain the most comprehensive source of information available in this field. This volume is divided into two sections. The first includes comprehensive chapters on general topics and topics relevant to multiple taxa. Several new topics are presented including

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48

Madl, Ulrike. Pathophysiology of glucose control. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0258.

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Hyperglycaemia is a frequent phenomenon in critically-ill patients, associated with increased morbidity and mortality. Hyperglycaemia results in cellular glucose overload and toxic adverse effects of glycolysis and oxidative phosphorylation, especially in tissues with insulin-independent glucose uptake, and acute hyperglycaemia can exert a variety of negative effects. It is the main side effect of intensive insulin therapy. Both severe and moderate hypoglycaemia are independent risk factors of mortality in critically-ill patients. Prolonged hypoglycaemia induces neuronal damage, but may also h

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49

Becker, Jeffrey. Nutraceutical Treatments for Addiction Recovery. Edited by Shahla J. Modir and George E. Muñoz. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190275334.003.0020.

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Current medical treatment in substance-use disorders and addiction recovery often fails to address the underlying molecular pathophysiologic mechanisms of addiction morbidity. Psychopharmacology and behavioral interventions do not directly address the cellular patterns of dysfunction in addiction but natural treatments can and should be employed in a research-based manner to support existing treatment protocols. Research into addiction pathophysiology is clear: removing the offending agent through sobriety is often not enough to restore natural premorbid physiology. Drug-induced oxidative stre

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50

Annane, Djillali, and B. Jérôme Aboab. Management of carbon monoxide poisoning. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0328.

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CO poisoning is the commonest cause of toxic death. Carbon monoxide is colourless, odourless, and tasteless, and is produced under various conditions. When people inhale CO, the gas diffuses rapidly to the body and replaces oxygen at the level of haemoglobin, myoglobin, and other oxygen carriers. Subsequently, CO causes oxygen deprivation of all body tissues. CO also induces oxidative stress and systemic inflammation. After CO poisoning a broad variety of symptoms may occur. Survivors of CO poisoning often present with persistent neurological sequels or develop delayed neurological symptoms. T

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