Relevant bibliographies by topics / P15

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  5. Conference papers
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Academic literature on the topic 'P15'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 August 2024

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Journal articles on the topic "P15"

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محمد عبد الرزاق الصوفي, إحسان هادي عبيد, and زينب خضير عباس. "تقييم جودة تعبئة المواد الغذائية بوساطة الاكياس البلاستيكية المرنة." Journal of the College of Basic Education 29, no. 120 (September 22, 2023): 466–55. http://dx.doi.org/10.35950/cbej.v29i120.10782.

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جرى تحديد كفاءة تعبئة 20 انموذجا من المواد الغذائية المعبأة في اكياس بلاستيكية مرنة والمتوافرة في اسواق مدينة بغداد، واشارت النتائج الى عدم وجود رقم الوجبة في النماذج P2، P3، P4، P7، P9، P19 وP20 وانها غير واضحة في الانموذج P1، كما يلاحظ اختلاف مدة الصلاحية للنماذج وعدم مطابقة بعضها لمدة الصلاحية التي نصت عليها المواصفات القياسية العراقية 1847 لسنة (2012)، اذ انها كانت 6 اشهر في النماذج P1 وP2، بينما كانت 24 شهرا في الانموذج P5 وكانت 12 شهرا في الانموذج P18 ولم تكن واضحة في الانموذج P7 وانما استعيض عنها بعبارة (صالحة للاستهلاك لمدة خمس سنوات من تاريخ الانتاج)، وبينت نتائج فحص التسرب للمواد الغذائية المعبأة بالأكياس البلاستيكية المرنة تبين وجود تسرب في النماذج P1، P2، P6، P7، P14 وP19، وان نتائج فحص اللحام بينت فشل النماذج P3، P4، P5، P15، وP16 وP17، بينما بلغت 611، 428، 223، 515، 326، 187، 288 و440 ملي بار للنماذج P8، P9، P10، P11، P12، P13، P18 وP20 على التوالي، في حين كان سمك الغلاف 0.048، 0.096، 0.093، 0.069، 0.088، 0.106، 0.110، 0.487، 0.142، 0.135، 0.136، 0.059، 0.099، 0.113، 0.081، 0.083، 0.063، 0.083، 0.127 و 0.094 مايكرومتر للنماذج P1، P2، P3، P4، P5، P6، P7، P8، P9، P10، P11، P12، P13، P14، P15، P16، P17، P18، P19، P20 على التوالي.
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Handayani, Heni Yuli. "The Impact of Covid-19 on Pencak Silat Course Process in STKIP PGRI Bangkalan." STRADA Jurnal Ilmiah Kesehatan 9, no. 2 (November 1, 2020): 1611–16. http://dx.doi.org/10.30994/sjik.v9i2.508.

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The data collection technique used in this study was a survey technique in the form of a questionnaire using google forms. Descriptive test shows that students agree with the percentage of P1 83.3%, P2 80%, P3 80%, P4 90%, P5 90%, P6 83.3%, P7 76.7%, P8 86.7%, P9 83.3%, P10 80%, P11 83.3 %, P12 86.7%, P13 83.3%, P14 80%, P15 83.3%, P16 80%, P17 80%, P18 83.3%, P19 80%, P20 90%. The results of this study indicate that the majority of students agree that COVID-19 has an impact on the pencak silat lecture process, besides that there are also several obstacles experienced by students in the process of lecturing online pencak silat courses. Based on the results of the study, it can be concluded that CIVID-19 has an impact on the learning process of the STKIP PGRI Bangkalan pencak silat course
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Azizah, A. N., E. Yuniastuti, Nandariyah, Supriyono, and I. I. S. Putri. "Morphological characterization of pachira (Pachira aquatica Aubl.)." IOP Conference Series: Earth and Environmental Science 905, no. 1 (November 1, 2021): 012044. http://dx.doi.org/10.1088/1755-1315/905/1/012044.

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Abstract Pachira is one of the plants that have a fairly high and promising selling value. Characterization of morphological properties is important so that the pachira (Pachira aquatica Aubl.) germplasm is more efficient. This study aims to study and characterize diversity and kinship in order to obtain information about the characteristics of pachira groupings. Pachira’s characterization includes qualitative and quantitative characters. Analysis of kinship using the UPGMA method. The results showed that the level of diversity in the morphological characters of pachira 3 districts in East Java reached 0.33. The results of the kinship analysis obtained 4 clusters with a coefficient of 0.74. Group A consisted of samples P1, P3, P15, P4, and P12. Group B consisted of samples P2, P8, P7, P11, P5, P9, P18, P13. Group C consisted of samples P6, P10, and P14. Group D consisted of P16 and P17. The level of diversity of pachira morphological characters reached 0.33.
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Nabila, Ferhiyan, Chomsin Sulistya Widodo, and Didik Rahadi Santoso. "Electrical Impedance Spectroscopic Analysis on Whole Blood Cells to Correlate Severity Level of Ischemic Stroke Patients." Jurnal Penelitian Pendidikan IPA 9, no. 11 (November 25, 2023): 9859–66. http://dx.doi.org/10.29303/jppipa.v9i11.5443.

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Measuring the impedance value of biological materials in the form of blood samples has been widely used to determine a person's health status. Ischemic stroke patients in terms of impedance values and correlates with morphological tests on changes in red blood cells. The Electrical Impedance Spectroscopy (EIS) method is applied by providing a blood sample which is detected by electrodes and read using a BIA tool set so that the results are obtained on a laptop. The results obtained from the EIS test are in the form of Bode graphs, Bode phase graphs, Nyquist graphs, and impedance values. The EIS method uses a frequency of 100 Hz to 100 kHz by injecting a current of 10 μA. In this study, the impedance value obtained for normal people was 1058 Ω to 709 Ω, while for ischemic stroke patients with various levels of condition, it was from 517 Ω to 761 Ω. When counting the number of morphological cells in the patient's blood, changes were found ranging from the most cell changes to the least, namely with a value of 44% to 19%. The severity level of ischemic stroke patients obtained based on impedance tests and morphological tests includes the order P4, P5, P3, P2, P10, P21, P15, P19, P1, P6, P13, P18, P14, P17, P16, P20, P12, P11, P8, P7, P9.
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Küçük, Çiğdem. "Selection of Rhizobium spp. Isolates for Inoculation of Field Pea (Pisum sativum)." International Journal of Agriculture and Biology 25, no. 04 (April 1, 2021): 845–50. http://dx.doi.org/10.17957/ijab/15.1737.

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Rhizobium isolates from wild pea nodules were characterized on the basis of microbiological characteristics. P4, P7, P12, P14, P16, P19, P20, P22, P23 and P24 isolates grew at the 4.5 pH, P5, P6, P11, P12, P13, P14, P16, P17, P19, P20 and P21 isolates grew at 4% NaCl and P7, P8, P10, P11, P12, P14, P19, P20, P22, P23, P24 and P25 isolates grew at 40°C. Resistance to antibiotics (μg mL-1) was investigated in a large propotion of isolates; streptomycin sulphate (80), rifampisin (40), erythromycin (30), chloramphenicol (100), Penicillin (40). In this study, local Rhizobium bacterial isolates were isolated from wild pea root nodules and their efficacy was investigated. Isolates significantly increased plant dry matter weight. The highest nitrogen fixation was achieved with P4 inoculation. Glutamine synthetase and leghemoglobin content of the nodules were determined in the inoculated with the highest P4 isolate. © 2021 Friends Science Publishers
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Li, Lu Qi, Cheng Zhi Liu, and Yan Chun Liu. "Gulong Oilfield Putaohua Oil Layer in the Gu 83 Block Sequence and Sandstone Characteristics Research." Applied Mechanics and Materials 395-396 (September 2013): 459–62. http://dx.doi.org/10.4028/www.scientific.net/amm.395-396.459.

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Gu 83 wellblock is the center of the Gulong reserves, the well test oil yield is high, the oil-bearing area shall, as reserves and production increase nine oil production company of Daqing oil field replacement area. On the basis of high resolution sequence stratigraphy theory, strata as the instruction, the trunk section and the well combination method is used to contrast layer and multiple well profile compared to small layer, establish high resolution stratigraphic framework. The Putaohua reservoir is divided into three sandstone under the group, which, subdivided three eight small layers for sandstone. Through well combine to determine the sequence division of the interface, divided into Putaohua reservoir. Considering lithology, curve characteristics of shaft vibration, even the well profile correlation research area according to the principle, method, sequence division division, in the Putaohua oil layer, three sets of sandstone under the group; On the segmentation, each group sandstone, upper sandstone will eventually set is divided into two layers of P11, P12; middle sandstone is divided into P13, P14, P15 three small layer; under sandstone under group is divided into P16, P17, P18 three small layer.
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Kurniasari, Indah Fitriana, Ida Dwijayanti, Fenny Roshayanti, and Susi Handayani. "Implementasi Culturally Responsive Teaching pada Materi Bentuk Bangun Ruang Kelas 1 SDN Pandean Lamper 04 Semarang." JIIP - Jurnal Ilmiah Ilmu Pendidikan 6, no. 7 (July 7, 2023): 5364–67. http://dx.doi.org/10.54371/jiip.v6i7.2403.

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Tujuan dari penelitian ini adalah untuk menganalisis bagaimana Implementasi Culturally Responsive Teaching Pada Materi Bentuk Bangun Ruang Kelas 1 SDN Pandean Lamper 04 Semarang. Penelitian ini menggunakan metode deskriptif analitis dengan pendekatan kualitatif. Tahapan-tahapan penelitian adalah melakukan observasi motivasi belajar siswa, mengisi jurnal refleksi guru dan siswa. Hasil dari analisis perencanaan memperoleh hasil (P1) 85,19%, (P2) 72,22%, (P3) 85,19%, (P4) 84,26%, (P5) 97,22%, (P6) 84,26%, (P7) 98,15% (P8) 90,74% (P9) 81,48% (P10) 87,96 % (P11) 96,30% (P12) 95,37% (P13) 89,81% (P14) 76,85% (P15) 81,48%. Implementasi Culturally Responsive Teaching dilakukan dengan tahap penelitian dan tahap pembelajaran di kelas.
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Rosyidi, Moh Ririn, and Nailul Izzah. "Analisis Kualitas Pelayanan Kinerja Bengkel Berdasarkan Persepsi Pengunjung dengan Metode IPA di Bengkel X." Jurnal Optimalisasi 8, no. 1 (April 13, 2022): 16. http://dx.doi.org/10.35308/jopt.v8i1.4817.

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Many organizations exist and make different decisions for the purpose of accommodation and compliance with a given administration so that the buyer continues to use a given administration, the vital nature of administration is the general component and nature of a thing or organization that can satisfy an expressed or proposed need. workshop X is located in Jalan Raya Sembayat, Kec. Manyar Kab. Gresik busy with service administration for buyers and also until now has many clients, but sometimes errors occur in work, this is because the help system is still manual so it becomes wasteful. The X workshop provided is also not ideal for the various services that exist to support customer improvement, therefore a top-down review is needed to provide a surprisingly better improvement using the Importance Performance Analysis method. Then at that time the value of the respondent's level of similarity was 75%, for the IPA value at the level of conformity, which was 91% on the question of the ease of reaching the workshop. Result of the Importance Performance Analysis, obtained quadrant I (Main Priority), namely P20, P4, P2, this shows that the client anticipates a significant level of significance, but the provision of assistance is not optimal so that there needs to be improvements that pay more attention to rules, Quadrant II (Maintain Achievement) in this area i.e. P5, P6, P7, P8, P9, P10, P11, P12, P13, P14, P15, P16, P17, P19, server friendliness in serving clients, which implies that the client have considered the characteristics, Quadrant III (Low Priority), there are two namely P1, P3, it is necessary to improve the attributes of the assistance after focusing on improving the characteristics that are basic needs, such as the neatness of employees' clothes and waiting room support facilities, Quadrant IV (Exaggerated) P18, respondents consider this quality to have a low level of significance but have truly elite exhibits. While the PGCV value is 8.97 with the question of giving the most important discount according to the customer.
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Kang, Seong-Ho, Dong Soon Lee, Tae Young Kim, Hyun Jung Min, Bo Ra Oh, Han-Ik Cho, and Sung-Soo Yoon. "Methylation Profiles of p16, p15 and p14 Genes in Korean Patients with Multiple Myeloma." Blood 110, no. 11 (November 16, 2007): 1504. http://dx.doi.org/10.1182/blood.v110.11.1504.1504.

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Abstract Dysruption of cell cycle control genes (p16, p15 and p14) is known to be involved in the tumorigenesis of multiple myeloma (MM). We investigated the inactivation status of p16, p15 and p14 genes using promoter methylation study and fluorescent in situ hybridization (FISH) study in patients with MM and analyzed the association of inactivation of those genes and clinical prognosis.MM Promoter methylation study of p16, p14 and p15 gene was done with newly diagnosed 52 patients with by bisulfite modification and methylation specific PCR. Two sets of primers were used for p16 methylation study and one set of primer was used for p15, p14 methylation study. Deletion of p16, p14 and p15 gene was detected by dual color FISH (Vysis, Downers GroveIL, USA). Overall survival was analyzed by Kaplan Meier Method and Cox’s proportional hazard model. Methylation of p16, p15 and p14 promotor was detected in 36/52 (69.2%), 15/52(28.8%) and 7/52 (13.5%) patients with MM, respectively. Methylation of any of p16, p15 or p14 promotor was observed in 44/52 (84.6%) of MM patients Deletion of p16, p15 and p14 was detected in only one of the patients with MM. Of note, in the cases (15/52, 28.8%) showing that in cases with two positive methylation specific PCR of the p16 promotor with two sets of primers, significant lower overall survival rate (p<0.036) were observed compared with only one positive methylation specific PCR. Heavy methylation of p16 protomor was a independent predictive variable for overall survival [Hazard Ratio, 3.74; 95% CI, 1.44–9.68, P = 0.007]. Other adverse prognostic factors by univariate analysis were old age (p=0.014), β2-microglobuline (p=0.026), high serum creatinine levels (≥2.0 mg/dL, p=0.014). More than 80% of MM patients showed the methylation of any of p16, p15 or p14 promotor, which suggest the potential applicability of hypomethlyating agents to MM. The promoter methylation of p16, p14 or p15 was a major contributor to the disruption of cell cycle regulation in MM, whereas both the deletion and/or the promoter methylation of p16, p14, and p15 contributed to the disruption of cell cycle regulation in ALL. In our study, methylation of two positive methylation specific PCR of the p16 promotor was an independent adverse prognostic factor in MM. We infer that quantitative methylation study for p16 promotor is helpful for the evaluation of prognosis. Figure 1. Overall survival analysis of patients with according to p16 methylation amount (P = 0.036) Figure 1. Overall survival analysis of patients with according to p16 methylation amount (P = 0.036)
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Takeuchi, S., CR Bartram, T. Seriu, CW Miller, A. Tobler, JW Janssen, A. Reiter, WD Ludwig, M. Zimmermann, and J. Schwaller. "Analysis of a family of cyclin-dependent kinase inhibitors: p15/MTS2/INK4B, p16/MTS1/INK4A, and p18 genes in acute lymphoblastic leukemia of childhood." Blood 86, no. 2 (July 15, 1995): 755–60. http://dx.doi.org/10.1182/blood.v86.2.755.bloodjournal862755.

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A newly recognized family of proteins that inhibit cyclin-dependent kinases (CDKs) termed cyclin-dependent kinase inhibitors (CDKI) have an important role in regulation of cell-cycle progression. A subfamily of these CDKIs (p15INK4B/MTS2, p16INK4/MTS1, and p18) have a high degree of structural and functional homology and are candidate tumor- suppressor genes. We evaluated the mutational status of the p15, p16, and p18 genes in 103 childhood acute lymphoblastic leukemia (ALL) samples and correlated these results with both their clinical data and additional results concerning their loss of heterozygosity in the region of the p15/p16 genes. Homozygous deletions of the p16 gene occurred extremely frequently in T-ALLs (17/22; 77%), and it was also frequent in precursor-B ALLs (12/81; 15%). Homozygous deletions of the p15 gene were also very frequent in T-ALLs (9/22; 41%), and it occurred in 5 of 81 (6%) precursor-B ALL samples. No deletions of p18 was found in any of the 103 ALL samples. Also, no point mutations of the p15, p16, and p18 genes were detected. We correlated p15/p16 alterations at diagnosis with their clinical characteristics as compared with 2,927 other patients treated similarly. Those with p15/p16 alterations were older; had higher white blood cell counts, often with T-cell ALL phenotype; and more frequently had a mediastinal mass at presentation; but they had the same nonremission, relapse, and survival rates at 5 years as did those patients whose blast cells did not have a p15/p16 deletion. To better understand the extent of alterations affecting chromosome 9p21 (location of the p15/p16 genes), loss of heterozygosity (LOH) was examined at D9S171, which is about 1 megabase proximal to the p15/p16 genes. LOH was detected in 15 of 37 (41%) informative samples. Interestingly, of the 24 informative samples that had no detectable alteration of the p15/p16 genes, 7 samples (29%) had LOH at D9S171. In summary, we show in a very large study that p15 and p16, but not p18, CDKI genes are very frequently altered in ALL; those with p15/p16 alterations are more frequently older children, have higher white blood cells at presentation, and often have a T-cell ALL phenotype. The LOH analysis suggests that another tumor-suppressor gene important in ALL also is present on chromosome 9p21.
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Dissertations / Theses on the topic "P15"

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Thullberg, Minna. "The cell cycle regulators p15, p16, p18 and p19 : functions and regulation during normal cell cycle and in multistep carcinogenesis /." Stockholm, 2000. http://diss.kib.ki.se/2000/91-628-4432-6/.

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Quesnel, Bruno. "Gene p16 ink4a , p15 ink4b, et hemopathies malignes." Lille 2, 1997. http://www.theses.fr/1997LIL2T009.

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文偉倫 and Wai-lun Matthew Man. "p15 and p16 genes in head and neck carcinoma." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2001. http://hub.hku.hk/bib/B31224945.

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Man, Wai-lun Matthew. "P15 and p16 genes in head and neck carcinoma /." Hong Kong : University of Hong Kong, 2001. http://sunzi.lib.hku.hk:8888/cgi-bin/hkuto%5Ftoc%5Fpdf?B23436001.

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Bourassa, Nancy. "Étude des gènes p16 et p15 dans la prédisposition au cancer colorectal héréditaire sans polypose." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/mq33581.pdf.

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Le, Frère-Belda Marie-Aude. "Les inhibiteurs de la transition G1-Sp14arf, p15 et p16 dans le cancer de vessie." Paris 11, 2002. http://www.theses.fr/2002PA11T063.

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Chicano, Lavilla María. "Estudio de metilación en los genes CDH1, P15, P16 y BIK en pacientes afectos de mieloma múltiple." Doctoral thesis, Universitat Autònoma de Barcelona, 2017. http://hdl.handle.net/10803/458532.

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El Mieloma Múltiple (MM) es una neoplasia hematológica, que en la gran mayoría de casos, la enfermedad viene precedida de una alteración de células plasmáticas (CP) asintomática, la Gammapatía monoclonal de significado incierto (GMSI). Así mismo se ha descrito un estadio intermedio entre GMSI y MM denominado Smoldering Multiple Myeloma (SMM). Se desconoce por qué solo algunas GMSI progresan a MM. Se han elaborado guías de estratificación de los pacientes con marcadores de progresión clínico-biológicos, como los niveles de componente monoclonal, el tipo de cadenas ligeras libres y el porcentaje y atipias de las CP. Sin embargo, y debido a la gran heterogeneidad que presenta esta enfermedad, no existen marcadores de progresión fiables. La inactivación por metilación de los promotores de genes supresores de tumor podría estar implicada en la progresión y/o evolución de la enfermedad. Estudios de metilación global en MM de la literatura describen un aumento del porcentaje de pacientes con metilación, en algunos de estos genes a lo largo de la evolución de la enfermedad. El estudio del patrón de metilación de determinados genes supresores de tumor podría ayudar a comprender los mecanismos implicados en la evolución del MM. En el presente estudio se ha analizado la presencia de metilación en el promotor de los genes supresores de tumor CDH1, P15, P16 y BIK en una serie de 103 pacientes afectos de MM, GMSI o SMM. El análisis de metilación se ha realizado mediante MS-PCR con DNA obtenido del cultivo celular de médula ósea tras fijación con Carnoy, siendo el primero en MM que utiliza este tipo de muestras. Se ha establecido la frecuencia de casos con metilación en estadios asintomáticos y en MM. Se ha comparado la frecuencia de metilación en pacientes con MM estudiados al diagnóstico con la de pacientes estudiados durante el seguimiento y resistentes al tratamiento o en recaída. Asimismo se ha evaluado la posible relación entre la metilación de estos genes y variables clínico-biológicas para establecer su posible valor pronóstico. Las frecuencias de casos con metilación en P15 y P16 mostraron diferencias significativas entre estadios premalignos y MM. Es de destacar que en cuatro de los pacientes, de los que se disponían muestras en dos estadios diferentes de la enfermedad, se observó metilación en BIK únicamente en aquellas correspondientes a la enfermedad resistente a tratamiento. Ello sugiere que la metilación de algunos genes supresores de tumor podría tener un papel importante en la progresión de determinados clones de CP resistentes a tratamiento. Al analizar la relación entre las variables clínico-biológicas y metilación, solamente se encontró correlación en toda la cohorte de pacientes (pre-MM y MM) entre metilación en P15 y niveles elevados de LDH así como entre P16 y niveles elevados de b2-microglobulina y porcentaje elevado de CP. Además la presencia de tres o cuatro genes metilados estaba asociada a una supervivencia inferior en los pacientes. Ello sugiere que la metilación de varios genes supresores de tumor podría conferir un fenotipo más agresivo. Es de destacar que la metilación concomitante en P16 y BIK en pacientes afectos de MM tenía un impacto más adverso en la SG que roza la significación como factor pronóstico independiente. Éste hecho apoyaría la hipótesis de que la metilación de determinados genes supresores de tumor podría tener un papel importante en la supervivencia de determinados clones de CP porque serían resistentes al tratamiento.
Multiple Myeloma is a hematologic neoplasm included in the called plasma cell (PC) dyscrasias. In most of the cases the disease is preceded by an asymptomatic premalignant entity, the MGUS, an intermediate stage called Smoldering Multiple Myeloma (SMM) has also been described. Only some of the MGUS progress to MM, but the reason is unknown. The research of progression markers in MM has brought to the incorporation of some clinical variables in stratification guides as monoclonal component levels, free light chain type and PC percentage and atypia. However, due to the heterogeneity of the MM, there is no clear progression marker, specifically in the context of MGUS to MM progression. Promoter methylation of tumour suppressor genes could lead to an inactivation and could have an effect in the progression and/or evolution of the disease. Global methylation studies in MM have observed an increase in the methylation of some of these genes all along the evolution of the disease. For that reason, changes in methylation pattern of some genes could help into a better comprehension of the evolution of the disease. In this study the presence of promoter methylation in tumour suppressor genes CDH1, P15, P16 and BIK has been analysed in 103 patients of MM, SMM or MGUS. The frequency of methylation has been compared between asymptomatic stages and MM, as well as in MM patients at diagnostic or in the follow up of the disease. Additionally, the relation between methylation of these genes and clinical variables has been evaluated in order to observe a possible implication in the prognosis. Methylation analysis has been done by MS-PCR with DNA obtained from bone marrow cell culture after fixation with Carnoy. Only methylation frequencies in P15 and P16 showed significant differences between asymptomatic stages and MM, however, they did not show a correlation with adverse prognosis variables. In relation to methylation and the evolution of the disease, a tendency to a greater BIK methylation frequency in the follow up MM patients compared to the diagnostic patients was observed. Likewise, the presence of BIK gene methylation in refractory samples of four patients which didn’t showed methylation in a previous study, could suggest that methylation of certain genes could play a role in the progression of drug resistant PC clones. In the analysis of the relation between clinical variables and methylation, just a correlation of P15 methylation and high levels of LDH as well as a correlation of P16 methylation and high levels of b2-microglobulin and CP were found. This results were observed only in the total of the series but not when only MM patients were analysed. In overall survival analysis, the presence of methylation in three or more genes showed lower survival than the rest of the group, which would suggest that hypermethylation could confer a more aggressive phenotype. A tendency to a lower survival was observed in refractory or relapsed patients which showed methylation in P16. The combined methylation in P16 and BIK had an adverse impact on overall survival in MM patients that was near the significance when was studied as an independent prognosis factor. This result would sustain the hypothesis that methylation in certain genes could have an important paper in the survival of some PC clones resistant to treatment.
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Patel, Shyamal. "MC1R and p15/RB pathways in melanocyte differentiation and melanoma progression." Thesis, St George's, University of London, 2015. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.677181.

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Skin melanocytes are cells that produce pigment in melanosomes and protect us from the damaging effects of UV radiation. This project covered two facets of melanocyte biology: melanocyte differentiation and transformation. Hermansky-Pudlak syndrome (HPS), a rare autosomal recessive disorder, is characterised by oculocutaneous albinism, a bleeding diathesis and other variable symptoms due to impaired formation of lysosome-related organelles, including melanosomes. We showed that cAMP agonists modestly restore pigmentation in HPS-mutant melanocytes and that this effect correlates with pigment formation in lysosomes instead of melanosomes, shown for the first time by colocalisation studies. These findings build upon our current understanding of melanosomal protein trafficking in HPS-mutant melanocytes and may have important implications for the treatment of hypopigmentation in patients with HPS or other forms of albinism. The other section of this project focused on melanoma. The majority of cancer cells have centrosome numerical abnormalities including extra or supernumerary centrosomes. This correlates with aneuploidy and genetic instability and may affect tumour aggressiveness and clinical outcome. The tumour suppressors p 15INK4B and p16INK4A (encoded by the familial melanoma CDKN2 locus) inhibit CDK4/6 activity and have important roles in cellular senescence. p 16INK4A and its downstream regulators are also associated with suppressing centrosome overduplication; however, the role of p15INK4B in centrosome amplification is unknown. We showed that normal human melanocyte lines did not exhibit centrosome number abnormalities whereas those from later stages of melanoma did. Additionally, under conditions of S-phase block, p 15INK4B and p 16INK4A status determined whether centrosome overduplication would occur. Indeed, removal of p 151NK4B from p 161NK4A negative cells from various stages of melanoma progression changed lines that previously would not overduplicate their centrosomes into cells that did. These data suggest that, during melanoma progression, sequential loss of p15INK4B and p16INK4A provides the conditions to deregulated centrosome duplication with consequences for genome instability,
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Hussey, Damian J. "An investigation of the (4;11)(q21;p15) translocation in acute lymphocytic leukaemia /." Title page, contents and abstract only, 2000. http://web4.library.adelaide.edu.au/theses/09PH/09phh9725.pdf.

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Thesis (Ph.D.) -- University of Adelaide, Dept. of Medicine, 2000.
Copies of author's previously published articles inserted. Errata pasted onto verso of back end-paper. Bibliography: leaves 163-189.
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Wadsworth, Santiago. "Genetic and biochemical study of P15, the RNA silencing suppressor of Peanut clump virus." Strasbourg, 2010. http://www.theses.fr/2010STRA6233.

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Le RNA silencing est un mécanisme de régulation négative de l’expression des gènes, retrouvé chez la plupart des organismes eucaryotes et basé sur des interactions d’ARN séquence-spécifiques. Il est déclenché par de l’ARN double brin qui est découpé par Dicer en de petits ARNs de 21 ‡ 24nt, nommés short-interfering (si)RNAs ou microRNAs. Un brin du duplex est incorporé dans un complexe appelé RNA-induced silencing complex (RISC), pour guider le clivage endonucléolytique ou l’inhibition traductionnelle d’ARNm présentant une homologie de séquence. Chez les plantes, le RNA silencing joue un rôle important dans la défense antivirale, le développement et l’adaptation aux stress biotiques et abiotiques. Pour contrer ce mécanisme de défense, les virus de plantes ont élaboré des protéines dites suppresseurs de RNA silencing. Au cours de cette thèse, nous avons analysé les interactions entre le RNA silencing et le Peanut clump virus (PCV). Nous avons démontré que les siRNA dérivés de PCV sont produits par Dicer-like 4 (DCL4) et dans une moindre mesure par DCL2, se traduisant par l’accumulation de siRNA de 21 et 22nt respectivement. La P15, suppresseur de RNA silencing du PCV, provoque une réduction drastique des niveaux de siRNA provenant des molécules d’ARN exogène de type tige-boucle ; et stabilise le brin non incorporé dans RISC. Dans une deuxième partie, nous avons étudié les interactions de P15 et ces protéines cibles. Nous avons ainsi identifié DCL1 parmi les protéines co-immunoprécipitées par P15 et montré que P15 affecte le niveau d’accumulation de DCL1, suggèrant ainsi que P15 pourrait déclencher une régulation négative DCL1-dépendante du système antiviral
RNA silencing refers to a set of RNA-based eukaryotic processes that regulate gene expression in a sequence-specific manner. It initiates with the processing of dsRNA by Dicer (RNase type III) proteins into small molecules of dsRNA of 21- to 24-nt in length, generally referred as short-interfering (si)RNAs or microRNAs, depending on their origin. One brand of the small duplex is incorporated into an effector complex, the RNA-Induced Silencing Complex, to guide the regulation of gene expression through different mechanisms, including endonucleolytic cleavage, translational repression or histone- and DNA-modifications. In plants, siRNAs and miRNAs are involved in viral defense, development and adaptation to biotic and abiotic stresses. As natural targets of RNA silencing, plant viruses have produced highly diverse proteins called silencing suppressors. First, we have studied the genetic requirements of Peanut clump virus (PCV) viral-si(vs)RNA production in Arabidopsis and shown that PCV RNA is primary processed by Dicer-like 4 (DCL4) to produce 21-nt vsRNAs, with DCL2 playing a minor role in the production of 22-nt PCV viRNAs. We also observed that P15, the PCV-encoded silencing suppressor, drastically reduces the accumulation of siRNAs derived only from transgenic hairpins of perfect dsRNA and stabilizes the non-incorporated brand of the miRNA duplex. Another part of this work consisted in the identification of plant proteins that may interact with P15. Among others, we identified DCL1 as P15 co-immunoprecipitated protein and showed that P15 alters the level of DCL1, suggesting that P15 may mediate a DCL1-dependent negative regulation of the antiviral plant defense
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Books on the topic "P15"

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Đỗ, Xuân Mão. Mối quan hệ dân số và phát triển: Lồng ghép các biến dân số vào kế hoạch hóa phát triển ở Việt Nam. Hà Nội: UNFPA, 2005.

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C, Swann William, Wang Chih-ming, and National Institute of Standards and Technology (U.S.), eds. Hydrogen cyanide Hp13sCp14sN absorption reference for 1530 nm to 1560 nm wavelength calibration: SRM 2519. Gaithersburg, MD: U.S. Dept. of Commerce, Technology Administration, National Institute of Standards and Technology, 1998.

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Muniz, Fernandes Ricardo, and Filho Antunes, eds. PP1, PP2, PP3, PP4, PP5--. São Paulo: SESC São Paulo, 2004.

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Educational Resources Information Center (U.S.), ed. Patterns for success: Communication (P10). [Washington, DC]: U.S. Dept. of Education, Office of Educational Research and Improvement, Educational Resources Information Center, 1996.

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Nam, UNDP Viet. Mid-term review of Programme P135-II. Ha Noi: Committee for Ethnic Minority Affairs, 2009.

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1942-, Dow Jim, ed. Ph15: Fotografías por chicos de ciudad oculta. Buenos Aires: Fundación ph15, 2005.

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Patmanidi, Alexandra. Ba culovirus pathogenesis: The role of Chitinase and P10. Oxford: Oxford Brookes University, 2003.

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Cash Management & Credit Control P15 (Aat Textbooks). Ftc Foulks Lynch, 2003.

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Cash Management & Credit Control P15 (Aat Workbooks). Ftc Foulks Lynch, 2003.

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Revue/. P15 politica hermetica 2002 deus ex machina. Age d'homme, 2002.

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Book chapters on the topic "P15"

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Urbschat, Steffi, Silke Wemmert, and Ralf Ketter. "Glioblastoma Patients: p15 Methylation as a Prognostic Factor." In Tumors of the Central Nervous System, Volume 1, 399–404. Dordrecht: Springer Netherlands, 2011. http://dx.doi.org/10.1007/978-94-007-0344-5_42.

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Bruhn, Karen. "Das stille Gedächtnis der Universität." In Universitätskultur(en) jenseits der Fachgeschichte, 63–80. Kiel: Universitätsverlag Kiel | Kiel University Publishing, 2023. http://dx.doi.org/10.38072/2701-5122/p15.

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Der Beitrag Das stille Gedächtnis der Universität: Lebenswelt und Selbstverständnis der Sekretärinnen des Historischen Seminars von Karen Bruhn stellt eine erste Spurensuche zu dieser spannenden Akteurinnengruppe dar. Jahrzehntelang wurden die Frauen, die diesem Beruf nachgingen, als ›Vorzimmerdamen‹ oder ›gute Geister‹ mit einer Mischung aus Hochachtung und Abwertung angesehen und behandelt. Der Beitrag zeigt, dass das Berufsfeld jedoch seit jeher – für das Historische Seminar zumindest seit der Nachkriegszeit – hohe Anforderungen an die Qualifikation der Frauen stellte. Mit dem technischen Wandel und der Digitalisierung veränderte sich das Berufsbild nachhaltig. Eine Sache bleibt jedoch von höchster Priorität und oberstes Gebot: die Loyalität zur vorgesetzten Person.
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Štrop, P., L. Pavlíčková, D. Štys, M. Souček, J. Urban, O. Hrušková, F. Kaprálek, P. Ječmen, J. Sedláček, and V. Kostka. "p15 gag Proteinase of Myeloblastosis Associated Virus: Specificity Studies with Substrate Based Inhibitors." In Advances in Experimental Medicine and Biology, 519–23. Boston, MA: Springer US, 1991. http://dx.doi.org/10.1007/978-1-4684-6012-4_69.

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Bogiatzis, Petros, George Vargemezis, Gregory Tsokas, Ilias Fikos, Eftychia Amanatidou, Nikolaos Kordatos, Prodromos Louvaris, Konstantinos Polydoropoulos, and Alexandra Karamitrou. "Internal structure of the great tumulus of Apollonia as revealed by seismic tomography." In Advances in On- and Offshore Archaeological Prospection, 141–50. Kiel: Universitätsverlag Kiel | Kiel University Publishing, 2023. http://dx.doi.org/10.38072/978-3-928794-83-1/p15.

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We investigate the tumulus of Apollonia using the NSTomo3D seismic tomography software. Results reveal P-wave velocities of >850 m/s at the perimeter and <650 m/s in the middle. High velocities are associated with travertine rocks that were used as building materials. Low velocities correspond to unconsolidated sediments used as filling material. Several structures revealed that are of potential archaeological interest.
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Gilsbach, Anna, and Hannah Vos. "Weitere Ablehnungsgründe." In Handbuch Informationsfreiheitsrecht, 297–306. Kiel: Universitätsverlag Kiel | Kiel University Publishing, 2023. http://dx.doi.org/10.38072/978-3-910591-01-1/p15.

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Das Kapitel stellt Aspekte dar, die informationspflichtige Stellen einem Antrag auf Informationszugang neben den »klassischen« Ablehnungsgründen entgegenhalten können. Im Einzelnen handelt es sich dabei um den Einwand, der Antrag sei rechtsmissbräuchlich gestellt worden (A.), der Bearbeitung des Antrags stehe ein unverhältnismäßiger Verwaltungsaufwand entgegen (B.) oder die antragstellende Person verfüge bereits über die Informationen beziehungsweise diese seien allgemein zugänglich (C.).
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"Coastal morphodynamics." In River, Coastal and Estuarine Morphodynamics: RCEM 2007, Two Volume Set, 689. CRC Press, 2007. http://dx.doi.org/10.1201/noe0415453639-p15.

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Faill, Rodger T. "The Acadian orogeny and the Catskill Delta." In The Catskill Delta, 15–38. Geological Society of America, 1985. http://dx.doi.org/10.1130/spe201-p15.

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Okaya, David A., and George A. Thompson. "Involvement of deep crust in extension of Basin and Range province." In Geological Society of America Special Papers, 15–22. Geological Society of America, 1986. http://dx.doi.org/10.1130/spe208-p15.

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Gryta, Jeffrey J., and Mervin J. Bartholomew. "Factors influencing the distribution of debris avalanches associated with the 1969 Hurricane Camille in Nelson County, Virginia." In Geological Society of America Special Papers, 15–28. Geological Society of America, 1989. http://dx.doi.org/10.1130/spe236-p15.

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Farrand, William R. "Origins of Quaternary-Pleistocene-Holocene stratigraphic terminology." In Geological Society of America Special Papers, 15–22. Geological Society of America, 1990. http://dx.doi.org/10.1130/spe242-p15.

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Conference papers on the topic "P15"

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Kerr, Rachel, Zac Dolan, Nicola Fearnley, and Sophie Brady. "P15 Expansion of sexual health outreach during COVID." In BASHH 2022 Abstracts. BMJ Publishing Group Ltd, 2022. http://dx.doi.org/10.1136/sextrans-bashh-2022.62.

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Welch, Joseph, Fazle Roghani, Shih-Wei Chen, Cristina Kinsella, Ulises Zanetto, and Anil George. "P15 Oncological outcomes following malignant colorectal polyp resection." In BSG LIVE’24, 17-20 June 2024, ICC Birmingham. BMJ Publishing Group Ltd and British Society of Gastroenterology, 2024. http://dx.doi.org/10.1136/gutjnl-2024-bsg.97.

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Kotake, Y. "PO-050 Long noncoding RNA, ANRIL positively regulates cell proliferation in a P15/P16-dependent and -independent manners." In Abstracts of the 25th Biennial Congress of the European Association for Cancer Research, Amsterdam, The Netherlands, 30 June – 3 July 2018. BMJ Publishing Group Ltd, 2018. http://dx.doi.org/10.1136/esmoopen-2018-eacr25.94.

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Clayton, Michelle, Lynda Greenslade, and Kate Jones. "P15 Development of clinical professional standards for liver transplant nursing." In BASL Abstracts, 21–23 September, 2020. BMJ Publishing Group Ltd and British Society of Gastroenterology, 2020. http://dx.doi.org/10.1136/gutjnl-2020-basl.26.

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Tao, Catharina Jialing, Ashleigh Gibby, Katrina Burrows, Jo-Anne Johnson, Sachin Patil, and Theofilos Polychronakis. "P15 Predictors of obstructive sleep apnoea in children with obesity." In BSS Sleep 2023 – Biennial Scientific Meeting of the British Sleep Society, Leeds, UK. British Thoracic Society, 2023. http://dx.doi.org/10.1136/bmjresp-2023-bssconf.26.

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Craig, Antonia, Eleanor Murray, and Tomasz Guzik. "P15 Tools to assess vascular function and phenotype in early hypertension." In Scottish Cardiovascular Forum – 23rd annual meeting, University of Strathclyde, Saturday 1st February 2020. BMJ Publishing Group Ltd and British Cardiovascular Society, 2020. http://dx.doi.org/10.1136/heartjnl-2020-scf.25.

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AZEVEDO, B., R. A. SILVA, S. S. OLIVEIRA, R. P. VELOSO, S. B. F. SANTOS, and L. S. C. OLIVEIRA. "PRODUÇÃO DE ENDO-Β-1,4-GLUCANASE UTILIZANDO SORGO SACARINO IPA P15." In XXII Congresso Brasileiro de Engenharia Química. São Paulo: Editora Blucher, 2018. http://dx.doi.org/10.5151/cobeq2018-pt.0606.

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Ho, Pei-Yin, Yu-Chieh Tsai, Chao-Yuan Huang, Shiu-Dong Chung, Hsin-Yi Yang, Hsiang-Chi Peng, Ching-Hung Lin, and Ming-Kuen Lai. "Abstract 4564: Dysregulated p15 in urothelial carcinoma of the upper urinary tract." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-4564.

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Giurgola, Laura, Claudio Gatto, Umberto Rodella, and Jana D’Amato Tóthová. "P15-A112 Descemet’s membrane endothelial keratoplasty (DMEK) graft preparation in porcine cornea." In Abstracts from the 2023 Annual Meeting of the European Eye Bank Association (Aachen, Germany - 2-4 March 2023). BMJ Publishing Group Ltd, 2023. http://dx.doi.org/10.1136/bmjophth-2023-eeba.14.

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Junaid, Ebraheem. "P15 Paediatric prescribing test at a tertiary children’s hospital – observations and reflections." In Abstracts from the NPPG Conference 2023. BMJ Publishing Group Ltd, 2024. http://dx.doi.org/10.1136/bmjpo-2024-nppg.25.

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Reports on the topic "P15"

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Allen, J., R. Goldstone, S. Instenes, and B. Lawver. Force10 P10 Evaluation. Office of Scientific and Technical Information (OSTI), June 2007. http://dx.doi.org/10.2172/919959.

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Splitter, Gary, Zeev Trainin, and Yacov Brenner. Lymphocyte Response to Genetically Engineered Bovine Leukemia Virus Proteins in Persistently Lymphocytic Cattle from Israel and the U.S. United States Department of Agriculture, July 1995. http://dx.doi.org/10.32747/1995.7570556.bard.

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The goal of this proposal was to identify proteins of BLV recognized by lymphocyte subpopulations and determine the contribution of these proteins to viral pathogenesis. Our hypothesis was that BLV pathogenesis is governed by the T-cell response and that the immune system likely plays an important role in controlling the utcome of infection. Our studies presented in ths final report demonstrate that T cell competency declines with advancing stages of infection. Dramatic differences were observed in lymphocyte proliferation to recombinant proteins encoded by BLV gag (p12, p15, and p24) and env (gp30 and gp15) genes in different disease stages. Because retroviruses are known to mutate frequently, examinatin of infected cattle from both Israel and the United States will likely detect variability in the immune response. This combined research approach provides the first opportunity to selectively address the importance of T-cell proliferation to BLV proteins and cytokines produced during different stages of BLV infection. Lack of this information regarding BLV infection has hindered understanding lympocyte regulation of BLV pathogenesis. We have developed the essential reagents necessary to determine the prominence of different lymphocyte subpopulations and cytokines produced during the different disease stages within the natural host. We found that type 1 cytokines (IL-2 and IFN-g) increased in PBMCs from animals in early disease, and decreasd in PBMCs from animals in late disease stages of BLV infection, while IL-10, increased with disease progression. Recently, a dichotomy between IL-12 and IL-10 has emerged in regards to progression of a variety of diseases. IL-12 activates type 1 cytokine production and has an antagonistic effect on type 2 cytokines. Here, using quantitative competitive PCR, we show that peripheral blood mononuclear cells from bovine leukemia virus infected animals in the alymphocytotic disease stage express increased amount of IL-12 p40 mRNA. In contrast, IL-12 p40 mRNA expression by PL animals was significantly decreased compared to normal and alymphocytotic animals. To examine the functions of these cytokines on BLV expression, BLV tax and pol mRNA expression and p24 protein production were quantified by competitive PCR, and by immunoblotting, respectively. IL-10 inhibited BLV tax and pol mRNA expression by BLV-infected PBMCs. In addition, we determined that macrophages secret soluble factor(s) that activate BLV expression, and that secretion of the soluble factor(s) could be inhibited by IL-10. In contrast, IL-2 increased BLV tax and pol mRNA, and p24 protein production. These findings suggest that macrophages have a key role in regulating BLV expression, and IL-10 produced by BLV-infected animals in late disease stages may serve to control BLV expression, while IL-2 in the early stage of disease may activate BLV expression. PGE2 is an important immune regulator produced only by macrophages, and is known to facilitate HIV replication. We hypothesized that PGE2 may regulate BLV expression. Here, we show that cyclooxygenase-2 (COX-2) mRNA expression was decreased in PBMCs treated with IL-10, while IL-2 enhanced COX-2 mRNA expression. In contrast, addition of PGE2 stimulated BLV tax and pol mRNA expression. In addition, the specific COX-2 inhibitor, NS-398, inhibited BLV expression, while addition of PGE2 increased BLV tax expression regardless of NS-398. These findings suggest that macrophage derived cyclooxygenase -2 products, such as PGE2, may regulate virus expression and disease rogression in BLV infection, and that cytokines (IL-2 and IL-10) may regulate BLV expression through PGE2 production.
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Lam, H. L. Forecasts of Pc5 magnetic pulsations. Natural Resources Canada/ESS/Scientific and Technical Publishing Services, 1988. http://dx.doi.org/10.4095/122733.

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PARKMAN, D. B. Process Test Plan Shutdown P16 Exhauster. Office of Scientific and Technical Information (OSTI), January 2000. http://dx.doi.org/10.2172/801150.

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Cardone, Antonio, R. Wayne Albers, Ram D. Sriram, and Harish C. Pant. Study of interaction of the cyclin-dependent kinase 5 with its activator, p25 and with the p25-derived inhibitor, CIP. Gaithersburg, MD: National Institute of Standards and Technology, 2009. http://dx.doi.org/10.6028/nist.ir.7552.

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Aldaz, Claudio M. The p16 Pathway in Breast Cancer and Senescence Control. Fort Belvoir, VA: Defense Technical Information Center, September 2000. http://dx.doi.org/10.21236/ada391851.

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Aldaz, Claudio M. The p16 Pathway in Breast Cancer and Senescence Control. Fort Belvoir, VA: Defense Technical Information Center, September 1999. http://dx.doi.org/10.21236/ada383067.

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Zhu, LIang. P16 Axie in Androgen-Dependent Proliferation of Prostate Cancer Cells. Fort Belvoir, VA: Defense Technical Information Center, April 2001. http://dx.doi.org/10.21236/ada397994.

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Zhu, Liang. P16 Axis in Androgen-Dependent Proliferation of Prostate Cancer Cells. Fort Belvoir, VA: Defense Technical Information Center, April 2002. http://dx.doi.org/10.21236/ada406854.

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Zhu, Liang. P16 Axis in Androgen-Dependent Proliferation of Prostate Cancer Cells. Fort Belvoir, VA: Defense Technical Information Center, April 2004. http://dx.doi.org/10.21236/ada427114.

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