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Relevant bibliographies by topics / P53 Arg

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Academic literature on the topic 'P53 Arg'

Author: Grafiati

Published: 4 June 2021

Last updated: 4 February 2022

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Journal articles on the topic "P53 Arg"

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Dong, Huajie, Cheng Fang, Lei Fan, et al. "Pro72 Allele Potentially Increases the Poor Prognostic Value of p53 Mutations In Chinese Patients with Chronic Lymphocytic Leukemia." Blood 116, no. 21 (2010): 4612. http://dx.doi.org/10.1182/blood.v116.21.4612.4612.

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Abstract Abstract 4612 Background: The poor prognosis of chronic lymphocytic leukemia (CLL) patients with del(17p13) is well established. Association of p53 mutations and its codon 72 polymorphism with CLL prognosis has been studied. However, there is no study on p53 mutations in Chinese patients with CLL to date, and the joint effect of p53 mutations and p53 codon 72 polymorphism on the prognosis of CLL remains uncertain. Methods: The frequency of p53 codon 72 genotype and the p53 mutations status were assessed by direct sequencing and correlated with clinical outcome in 180 CLL patients. p53

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Gasco, M., N. Syed, P. Smith, et al. "A multi-gene algorithm as predictor of response to chemo-radiotherapy in head and neck cancer." Journal of Clinical Oncology 24, no. 18_suppl (2006): 10086. http://dx.doi.org/10.1200/jco.2006.24.18_suppl.10086.

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10086 Background: Chemo-radiotherapy results in clinical cure for stage III/IV head and neck cancer in approximately 40% of cases, with significant treatment-associated morbidity and mortality. Molecular genetic factors predictive of treatment outcome would clearly be of value in selection of patients with highest probability of response. We have analysed the structure and epigenetic regulation of specific genes as possible predictors of outcome to chemo-radiotherapy. The genes analyzed were: p53 (single nucleotide polymorphism (SNP) and mutation), MDM2 (SNP), Chfr (methylation), CRABP1 (methy

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Min-Min, H., X. Ming-Rong, C. Ze-Yi, Y. Kai-Xuan, and S. Zhi-Lin. "Analysis of p53 codon 72 polymorphism and its association with human papillomavirus 16 and 18 E6 in Chinese cervical lesions." International Journal of Gynecologic Cancer 16, no. 6 (2006): 2004–8. http://dx.doi.org/10.1111/j.1525-1438.2006.00733.x.

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The aim of this study was to analysis the relationship between p53 codon 72 polymorphism with human papillomavirus (HPV) 16 and 18 E6 in Chinese cervical cancer. A total of 81 cervical squamous cancer (specimens of G1, G2, and G3 are 13, 24, and 44, respectively; and of stage IB, IIA, IIB, and IIIA are 15, 37, 24, and 5, respectively), 18 cervical adenocarcinoma, 88 cervical intraepithelial neoplasm (CIN) (specimens of CIN II and III are 30 and 58), and 60 normal cervical specimens were included in this study. Polymerase chain reaction was used to examine p53 genotypes and HPV 16 and 18 E6. Th

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Pandima Devi, K., B. Sivamaruthi, PV Kiruthiga, and S. Karutha Pandian. "Study of p53 codon 72 polymorphism and codon 249 mutations in Southern India in relation to age, alcohol drinking and smoking habits." Human & Experimental Toxicology 29, no. 6 (2009): 451–58. http://dx.doi.org/10.1177/0960327109354938.

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Germline polymorphisms of genes involved in different steps of tumorigenesis like p53, the tumor suppressor gene, are reported to determine the individual susceptibility to cancer. Lung cancer is one of the most common and lethal cancers and tobacco smoking remains its most important etiologic factors. The most frequently p53 mutated codons of lung cancer are 72 (exon 4) and 249 (exon 7). Since mutations in the p53 gene are present in ∼40% of all human lung cancers and are more common in smokers than in nonsmokers, we aimed to detect the status of p53 at codon 72 for Arg/Arg or Arg/Pro or Pro/

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Yi, Ke, LingYun Yang, Zhu Lan, and MingRong Xi. "The Association Between p53 Codon 72 Polymorphism and Endometrial Cancer Risk: A System Review and Meta-analysis." International Journal of Gynecologic Cancer 26, no. 6 (2016): 1121–28. http://dx.doi.org/10.1097/igc.0000000000000725.

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AbstractPolymorphism of p53 codon 72 plays an important role in pathogenesis and development of cancer. Published data on the association between the p53 codon 72 polymorphism and endometrial cancer risk are controversial. A meta-analysis was performed to assess whether the polymorphism of p53 codon 72 is associated with endometrial cancer risk. Medline, Embase, China National Knowledge Infrastructure, and Chinese Biomedicine Databases were searched to identify eligible studies. Pooled odds ratios (ORs) and 95% confidence intervals (CIs) for p53 codon 72 polymorphism and endometrial cancer wer

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Das, Mandakini, Santanu Kumar Sharma, Gaganpreet Singh Sekhon, Jagadish Mahanta, Rup Kumar Phukan, and Bimal Kumar Jalan. "p16 gene silencing along with p53 single-nucleotide polymorphism and risk of esophageal cancer in Northeast India." Tumor Biology 39, no. 5 (2017): 101042831769838. http://dx.doi.org/10.1177/1010428317698384.

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The high incidence of esophageal cancer in Northeast India and the unique ethnic background and dietary habits provide a great opportunity to study the molecular genetics behind esophageal squamous cell carcinoma in this part of the region. We hypothesized that in addition to currently known environmental risk factors for esophageal cancer, genetic and epigenetic factors are also involved in esophageal carcinogenesis in Northeast India. Therefore, in this study, we explored the possible association between the two important G1 cell cycle regulatory genes p16 and p53 and environmental risk fact

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Lee, J. E., S. J. Lee, S. E. Namkoong, et al. "Gene–gene and gene–environmental interactions of p53, p21, and IRF-1 polymorphisms in Korean women with cervix cancer." International Journal of Gynecologic Cancer 14, no. 1 (2004): 118–25. http://dx.doi.org/10.1136/ijgc-00009577-200401000-00016.

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BackgroundThe aim of this study was to identify gene–gene and gene–environmental factors affecting cervix carcinogenesis in Korean women.MethodsWe evaluated 530 subjects composed of 185 female cervix cancer patients and 345 normal healthy women. The single nucleotide polymorphisms (SNPs) of p53 codon 72, p21 codon 31, and interferon regulatory factor-1 (IRF-1) intron 6 were evaluated from extracted DNA of peripheral blood with an automatic DNA sequencer. The differences of each SNP, gene–gene and gene–environmental interactions between normal controls and patients were evaluated in the adjuste

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Ørsted, David Dynnes, Stig Egil Bojesen, Anne Tybjærg-Hansen, and Børge Grønne Nordestgaard. "Tumor suppressor p53 Arg72Pro polymorphism and longevity, cancer survival, and risk of cancer in the general population." Journal of Experimental Medicine 204, no. 6 (2007): 1295–301. http://dx.doi.org/10.1084/jem.20062476.

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p53 is an important tumor suppressor, normally preventing cancer development via apoptosis. A genomic Arg72Pro substitution in the p53 protein has important influence on cell death via apoptosis, which could be beneficial. We therefore tested the hypotheses that this polymorphism influences longevity, survival after a cancer diagnosis, and risk of cancer in the general population. We examined a cohort of 9,219 participants ages 20–95 from the Danish general population with 100% follow-up. The overall 12-yr survival was increased in p53 Arg/Pro heterozygotes with 3% (P = 0.003) and in Pro/Pro h

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Hayati, Lusia, and Siska Delvia. "Polymorphism of p53 Codon 72 Gene on Cervical Cancer Incidence in Malay Population." Archives of The Medicine and Case Reports 1, no. 1 (2020): 25–30. http://dx.doi.org/10.37275/amcr.v1i1.5.

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In Indonesia, the cases of cervical cancer are estimated at around 50 per 100.000 people. It wasestimatedthattherearemore than1 millionwomenworldwidewho have cervical cancer,andmostofthemhavenot been diagnosed yet or do not have access to screening and medical treatment. P53 codon 72polymorphism can affect the risk of cervical cancerthrough the regulationofproliferationandcellapoptosis.Thepurpose of this research was to investigate the association between p53 codon 72 polymorphism and cases ofcervical cancer. This research was observational analytic research. The research was done by examining

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Gomez-Sanchez, Jose C., Maria Delgado-Esteban, Irene Rodriguez-Hernandez, et al. "The human Tp53 Arg72Pro polymorphism explains different functional prognosis in stroke." Journal of Experimental Medicine 208, no. 3 (2011): 429–37. http://dx.doi.org/10.1084/jem.20101523.

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The functional outcome after stroke is unpredictable; it is not accurately predicted by clinical pictures upon hospital admission. The presence of apoptotic neurons in the ischemic penumbra and perihematoma area may account for poor prognosis, but whether the highly variable stroke outcome reflects differences in genetic susceptibility to apoptosis is elusive. The p53 tumor suppressor protein, an important transcriptional regulator of apoptosis, naturally occurs in humans in two variants with single nucleotide polymorphisms resulting in Arg or Pro at residue 72. We show that poor functional ou

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Dissertations / Theses on the topic "P53 Arg"

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Almeida, Priscilla Silva Rosa de. "POLIMORFISMO DO GENE TP53 EM SARCOMAS DE PARTES MOLES NO ADULTO." Pontifícia Universidade Católica de Goiás, 2008. http://localhost:8080/tede/handle/tede/2412.

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Made available in DSpace on 2016-08-10T10:39:18Z (GMT). No. of bitstreams: 1 PRISCILLA SILVA ROSA DE ALMEIDA.pdf: 3720637 bytes, checksum: b39a9c071d90058d47a3a8c7aec2c7f3 (MD5) Previous issue date: 2008-07-21<br>Soft tissue sarcomas (STS) are tumors with mesodermical origin, comprising about 1% of all adult neoplasms. Because of its effect on the p53 protein coding sequence, and its association with an increased risk for some cancer types, TP53 codon 72 polymorphism has been investigated in several studies. TP53 codon 72 codes for either Arginine (p53Arg), or Proline (p53Pro) at the p53 pro

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Crawford, Lauren Anne. "Are All P53s Created Equal? Uncovering the Function of Soft-shell Clam (Mya arenaria) p53." Fogler Library, University of Maine, 2005. http://www.library.umaine.edu/theses/pdf/CrawfordLA2005.pdf.

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Marques, Candeias Marco. "RNA-Dependent regulation of p53." Paris 7, 2007. http://www.theses.fr/2007PA077077.

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La protéine suppresseur de tumeur p53 est impliquée dans la régulation de la croissance et de la survie des cellules en réponse à une multitude de facteurs de stress cellulaires. Son activation a pour conséquence des modifications de l'expression d'un grand nombre de gènes. Son régulateur principal est la protéine Mdm2, une E3 ubiquitine-ligase qui lie p53 et conduit à sa dégradation par la voie protéasomale. Cependant, la façon dont p53 peut reconnaître spécifiquement différents stimuli de stress et y répondre en induisant des voies alternatives de régulation menant soit à un arrêt du cycle c

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Fourtouna, Argyro. "Function of the anterior gradient protein family in cancer." Thesis, University of Edinburgh, 2009. http://hdl.handle.net/1842/4302.

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Proteomic technologies verified Anterior Gradient 2, AGR-2, as a protein over-expressed in human cancers, including breast, prostate and oesophagus cancers, with the ability to inhibit the tumour suppressor protein p53. AGR-2 gene is a hormone responsive gene with an unexpected induction by the anti-cancer drug tamoxifen highlighting the proto-oncogenic role of this protein. Anterior Gradient-2 encodes one protein that gives rise to two forms· the full length and the mature one. Full length bears a leader sequence that leads the protein to secretion. Localization studies of both forms of AGR-2

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Pontes, Flavia Sirotheau Corrêa. "Avaliação in vitro da expressão das proteínas PTEN, Akt, Mdm2 e p53 em células de carcinoma epidermóide de cabeça e pescoço submetidas a ação de EGF e 17-AAG." Universidade de São Paulo, 2007. http://www.teses.usp.br/teses/disponiveis/23/23141/tde-02012008-151120/.

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O carcinoma epidermóide de cabeça e pescoço é responsável por 90% das neoplasias malignas, nesta região. Molecularmente, inúmeras vias de sinalização, ainda não muito bem compreendidas, são responsáveis pelo seu crescimento e invasão para tecidos vizinhos, além de metástases para órgãos distantes. Este trabalho destinou-se a avaliar o crosstalk entre as vias de sinalização do PTEN, Akt, Mdm2 e p53 em quatro linhagens de células de carcinoma epidermóide (HN6, HN19, HN30 e HN31) e queratinócitos imortalizados (HaCat), estimulados com EGF (fator de crescimento epitelial) e 17-AAG. Para observar a

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Bourougaa, Karima. "Diversifying the p53 pathway in response to Endoplasmic Reticulum stress via alternative translation initiation of the p53 mRNA." Paris 7, 2009. http://www.theses.fr/2009PA077128.

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La protéine p53 est le produit d'un gène muté dans plus de 50% des cancers humains. P53 est un facteur de transcription capable d'induire, en réponse à différents stress, l'arrêt du cycle cellulaire en Gl et en G2, la réparation de l'ADN, ou, si le dommage est trop sévère, la mort cellulaire. Un grand nombre de gènes répondent à p53 mais il reste quand même difficile de comprendre comment les cellules parviennent à différencier et à intégrer l'activation de p53 en une réponse bien spécifique. Récemment, différents isoformes de p53 ont été identifiés mais leur rôle physiologique reste encore in

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Gajjar, Madhavsai K. "Stress dependent regulation of p53 and Mdm2 mRNA translation." Paris 7, 2011. http://www.theses.fr/2011PA077184.

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Plus de 50% des cancers chez l'homme sont dus à des mutations de la protéine p53. Mdm2 joue un rôle majeur dans le contrôle de l'activité de p53, en induisant soit sa dégradation, soit au contraire sa synthèse. Récemment, notre équipe a montré que Mdm2 induit la synthèse de p53 en se liant avec PARNm de p53. Cependant, les mécanismes moléculaires responsables de cette dualité du rôle de Mdm2 ne sont pas encore élucidés. Les résultats présentés dans ce travail montrent qu'en cas de stress génotoxique, p53 induit la synthèse de Mdm2. Il s'ensuit également une phosphorylation ATM-dépendante de Md

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Todd, Ann Radha. "Molecular pathology of the p53-MDM2-p14 ARF pathway in soft tissue sarcomas." Thesis, University of Newcastle Upon Tyne, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.417433.

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Overkamp, Tim. "Bedeutung des p53-Signalwegs für Apoptoseaktivierung und Zellzyklusarrestregulation durch das p14 ARF Tumorsuppressorgen." Doctoral thesis, Humboldt-Universität zu Berlin, Mathematisch-Naturwissenschaftliche Fakultät I, 2012. http://dx.doi.org/10.18452/16619.

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BH3-only Proteine, eine pro-apoptotische Untergruppe der Bcl-2 Proteinfamilie, sind zentrale Mediatoren von apoptotischen Signalen durch die Regulierung intrinsischer Apoptose-signalwege. Unsere Arbeitsgruppe hat vor kurzem gezeigt, dass Apoptose, die durch den p14ARF Tumorsuppressor induziert wird über die p53-abhängige Aktivierung des BH3-only Proteins Puma/Bbc3 vermittelt wird. Interessanterweise induziert p14ARF aber auch in p53 defizienten Zellen Zellzyklusarrest und Apoptose. Die dahinterliegenden Signalwege sind jedoch nicht bekannt. In dieser Arbeit berichten wir, dass das BH3-only Pro

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Courtois-Cox, Stéphanie. "DeltaNp53, une isoforme du suppresseur de tumeur p53 : découverte, caractérisation et fonctions biologiques." Paris 7, 2003. http://www.theses.fr/2003PA077207.

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Books on the topic "P53 Arg"

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Building The P51 Mustang The Story Of Manufacturing North Americans Legendary Wwii Fighter In Original Photos. Specialty Press (MN), 2011.

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Generative Design: Visualize, Program, and Create with JavaScript in p5.js. Princeton Architectural Press, 2018.

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Gross, Wolfgang L., and Julia U. Holle. Clinical features of ANCA-associated vasculitis. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0131.

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The primary ANCA-associated vasculitides are granulomatosis with polyangiitis (Wegener's, GPA), microscopic polyangiitis (MPA), and eosinophilic granulomatosis with polyangiitis (EGPA, Churg-Strauss syndrome, CSS). They predominantly affect small (and medium-sized) vessels and share a variable association with ANCA (anti-neutrophil cytoplasm antibody) directed against neutrophil proteinase 3 (PR3, mainly in GPA) and myeloperoxidase (MPO, mainly in MPA and CSS). Crescentic necrotizing glomerulonephritis and alveolar haemorrhage due to pulmonary capillaritis represent classical (vasculitic) orga

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Morris, Justin, and Nicholas Wheeler. The Responsibility Not to Veto. Edited by Alex J. Bellamy and Tim Dunne. Oxford University Press, 2016. http://dx.doi.org/10.1093/oxfordhb/9780198753841.013.13.

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The responsibility to protect (R2P) and the question of UN Security Council veto constraint are intimately linked, but whilst the R2P has become increasingly embedded in diplomatic discourse and practice, the idea that in relation to it the Council’s five permanent members should recognize a ‘responsibility not to veto’ (RN2V) has fared less well. This chapter examines why this should be so. In its assessment of the prospects for, and pros and cons of, veto-restriction, the chapter argues that opposition amongst the P5 to the idea of a RN2V is unlikely to change in the foreseeable future, and

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Book chapters on the topic "P53 Arg"

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Hontz, Robert D., and Maureen E. Murphy. "p53, ARF, and the Control of Autophagy." In Cell Cycle Deregulation in Cancer. Springer New York, 2010. http://dx.doi.org/10.1007/978-1-4419-1770-6_6.

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Ponten, F., C. Berg, M. Uhlen, and J. Ponten. "Multiple p53 Mutations are Common in Basal Cell Carcinoma." In Skin Cancer and UV Radiation. Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-642-60771-4_85.

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Quartin, Robin S., and Arnold J. Levine. "The Two Amino Terminal Transforming Functions of the SV40 Large T-Antigen are Required to Overcome P53 Mediated Growth Arrest." In The Cell Cycle. Springer US, 1994. http://dx.doi.org/10.1007/978-1-4615-2421-2_36.

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Hyvönen, Mats, Maria Karlsson, and Madeleine Eriksson. "The Politics of True Crime: Vulnerability and Documentaries on Murder in Swedish Public Service Radio’s P3 Documentary." In Vulnerability in Scandinavian Art and Culture. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-37382-5_14.

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Kolomeyer, Natasha Nayak, and Marlene R. Moster. "New Modalities of Cycloablation and High-Intensity-Focused Ultrasound." In Minimally Invasive Glaucoma Surgery. Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-5632-6_9.

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Abstract Cycloablative or cyclodestructive procedures aim to lower intraocular pressure (IOP) by decreasing the function of the ciliary body and thereby decreasing the rate of aqueous production. Cycloablative procedures were typically used in refractory glaucoma in eyes with poor visual potential; however, more focused energy and targeted destruction of the ciliary body have led to an increase in cyclodestructive treatment options that are now an important adjunct to our surgical armamentarium. This chapter highlights the history of these procedures while focusing on current modalities including transscleral diode cyclophotocoagulation (TSCPC), micropulse transscleral diode cyclophotocoagulation (MP-TSCPC, MicroPulse P3, IRIDEX IQ810 Laser System, Mountain View, CA, USA), and High-Intensity Focused Ultrasound (HIFU). Specifically, this chapter discusses the protocols, indications, results, and complications of each featured procedure.

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Kamagata, Kiyoto. "A Study of p53 Action on DNA at the Single Molecule Level." In P53 - A Guardian of the Genome and Beyond [Working Title]. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.96163.

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The transcription factor p53 searches for and binds to target sequences within long genomic DNA, to regulate downstream gene expression. p53 possesses multiple disordered and DNA-binding domains, which are frequently observed in DNA-binding proteins. Owing to these properties, p53 is used as a model protein for target search studies. It counters cell stress by utilizing a facilitated diffusion mechanism that combines 3D diffusion in solution, 1D sliding along DNA, hopping/jumping along DNA, and intersegmental transfer between two DNAs. Single-molecule fluorescence microscopy has been used to characterize individual motions of p53 in detail. In addition, a biophysical study has revealed that p53 forms liquid-like droplets involving the functional switch. In this chapter, the target search and regulation of p53 are discussed in terms of dynamic properties.

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Raimondo, Salvatore, Mariacira Gentile, Tommaso Gentile, and Luigi Montano. "Presence of p53 Protein on Spermatozoa DNA: A Novel Environmental Bio-Marker and Implications for Male Fertility." In P53 - A Guardian of the Genome and Beyond [Working Title]. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.99559.

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Many studies suggest a direct relationship between toxic effects and an increase in the p53 protein on cellular DNA. For our studies, we used sperm DNA as an indicator of environmental toxic effects, dosing p53 quantitatively. To assess possible variations, we used semen samples from two homogeneous male groups living permanently in areas with different environmental impact. The toxic effects of the selected high environmental impact area are caused by both soil and air pollution, while the selected low environmental impact area is a nature reserve where there are no landfills, but only rural factories. As we work with reproductive cells, our interest was inevitably focused on sperm DNA damage and whether this damage could affect their fertilizing capacity. The length of telomeres and the quantification of protamines are being studied to better define the possible damage.

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Lydiate, Henry. "Authorship and authentication." In The Art Business. Routledge, 2008. http://dx.doi.org/10.4324/9780203885611.pt3.

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"Sensors for weld-seam tracking." In Arc Welding Control. CRC Press, 2003. http://dx.doi.org/10.1201/9780203491126.pt3.

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Ji, Suntae, Young Bae Sohn, and Sung Yoon Cho. "LIN28BPolymorphisms Are Associated with Central Precocious Puberty in Girls." In CLINICAL/TRANSLATIONAL - Pediatric Endocrinology: Puberty. The Endocrine Society, 2011. http://dx.doi.org/10.1210/endo-meetings.2011.part4.p13.p3-713.

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Conference papers on the topic "P53 Arg"

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Rivera, Bianca L., and Adriana Baez. "Abstract 727: Homozygosity for Arg of p53 codon 72 correlates with poor prognosis in Puerto Rican patients with head and neck squamous cell carcinoma." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-727.

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Fujita, Kaori, Abdul M. Mondal, Izumi Horikawa та ін. "Abstract 2915: p53 isoforms Δ133p53 and p53β are endogenous regulators of replicative cellular senescence". У Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-2915.

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Yi, Han-Jie, Xiang-Lei Yan, Qiu-Yun Luo, et al. "Abstract 314: A novel MDM2-p53 antagonist APG-115 induces p53-mediated apoptosis and enhances radiosensitivity in colorectal cancer." In Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-314.

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Li, G., S. S. Nair, S. J. Lees, and F. W. Booth. "Regulation of G2/M Transition in Mammalian Cells by Oxidative Stress." In ASME 2005 International Mechanical Engineering Congress and Exposition. ASMEDC, 2005. http://dx.doi.org/10.1115/imece2005-82349.

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The regulation of the G2/M transition for the mammalian cell cycle has been modeled using 19 states to investigate the G2 checkpoint dynamics in response to oxidative stress. A detailed network model of G2/M regulation is presented and then a “core” subsystem is extracted from the full network. An existing model of Mitosis control is extended by adding two important pathways regulating G2/M transition in response to DNA damage induced by oxidative stress. Model predictions indicate that the p53 dependent pathway is not required for initial G2 arrest as the Chk1/Cdc25C pathway can arrest the ce

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Kai, Fumitake, Robert D. Kendig, Donna P. Frazier, et al. "Abstract 1096: Arf-independent activation of p53 by the Dmp1 tumor suppressor." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-1096.

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Zhang, Shengliang, Lanlan Zhou, David Dicker, and Wafik S. El-Deiry. "Abstract 3816: Reactive oxygen species and ERK2 phosphorylation are required for NSC59984 to induce mutant p53 protein degradation and restore p53 signaling." In Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-3816.

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Moela, Pontsho, Lesetja Motadi, and Marcia Lekganyane. "Abstract 3040: The expressional effects of RBBP6 in breast cancer cells are p53-dependent." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-3040.

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Li, Zhongyou, Olga Mejia, and Carlos Caulin. "Abstract 479: p53 mutations and Ink4a/Arf deletion cooperate to induce metastatic skin carcinomas." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-479.

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Reed, Sara, Van Tompkins, Jussara Hagen, et al. "Abstract 215: Mechanisms of p53 activation by NIAM, nuclear interactor of ARF and MDM2." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-215.

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Moela, Pontsho, Lesetja Motadi, and Marcia Lekganyane. "Abstract 3040: The expressional effects of RBBP6 in breast cancer cells are p53-dependent." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-3040.

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Reports on the topic "P53 Arg"

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Nahle, Zaher A., and Scott Lowe. Analysis of the ARF/p53 Pathway During Oncogenic Stimulation. Defense Technical Information Center, 2003. http://dx.doi.org/10.21236/ada424043.

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Kandel, Rita A. Are p53 Mutations Associated With Increased Risk of Developing Breast Cancer? A Molecular Epidermiological Study. Defense Technical Information Center, 2003. http://dx.doi.org/10.21236/ada424156.

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Maglic, Dejan. MEKK1 is a Novel Regulator of the Dmp1-Arf-p53 Pathway and Prognostic Indicator in Breast Cancer. Defense Technical Information Center, 2012. http://dx.doi.org/10.21236/ada574517.

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Mehraein-Ghomi, Farideh. Exploring AR-NFkappaB/p52-Targeted Inhibitors as Novel Therapy Against Castration-Resistant Prostate Cancer Progression. Defense Technical Information Center, 2014. http://dx.doi.org/10.21236/ada604317.

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OCOMA, E. C. Seismic Adequacy Review of PC012 SCEs that are Potential Seismic Hazards with PC3 SCEs at Cold Vacuum Dryer (CVD) Facility. Office of Scientific and Technical Information (OSTI), 1999. http://dx.doi.org/10.2172/797683.

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