Relevant bibliographies by topics / Pain Pain Prostaglandins Nitric Oxide

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Academic literature on the topic 'Pain Pain Prostaglandins Nitric Oxide'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Pain Pain Prostaglandins Nitric Oxide"

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Minami, Toshiaki, Masato Sakai, Naoki Hara, Masahiko Onaka, Hidemaro Mori, and Seiji Ito. "Nitric Oxide Mediates Hyperalgesia Induced by Intrathecal Administration of Prostaglandin E2 in Conscious Mice." PAIN RESEARCH 11, no. 2 (1996): 63–70. http://dx.doi.org/10.11154/pain.11.63.

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Toda, Noboru, Hiroshi Toda, Yoshio Hatano, and David C. Warltier. "Nitric Oxide." Anesthesiology 107, no. 5 (2007): 822–42. http://dx.doi.org/10.1097/01.anes.0000287213.98020.b6.

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There has been an explosive increase in the amount of interesting information about the physiologic and pathophysiologic roles of nitric oxide in cardiovascular, nervous, and immune systems. The possible involvement of the nitric oxide-cyclic guanosine monophosphate pathway in the effects of anesthetic agents has been the focus of many investigators. Relaxations of cerebral and peripheral arterial smooth muscle as well as increases in cerebral and other regional blood flows induced by anesthetic agents are mediated mainly via nitric oxide released from the endothelium and/or the nitrergic nerv
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de Larminat, V., J. Boczkowski, B. Dureuil, M. Aubier, and J. M. Desmonts. "Role of prostaglandins and nitric oxide (NO) in arteriolar haiothane-induced vasodilation." Anesthesiology 77, Supplement (1992): A683. http://dx.doi.org/10.1097/00000542-199209001-00683.

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Chen, Xiaojie, and Jon D. Levine. "NOS Inhibitor Antagonism of PGE2-Induced Mechanical Sensitization of Cutaneous C-Fiber Nociceptors in the Rat." Journal of Neurophysiology 81, no. 3 (1999): 963–66. http://dx.doi.org/10.1152/jn.1999.81.3.963.

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NOS inhibitor antagonism of PGE2-induced mechanical sensitization of cutaneous C-fiber nociceptors in the rat. Prostaglandins, metabolites of arachidonic acid, released during tissue injury and inflammation sensitize primary afferent nociceptors. While it has been suggested that this effect on nociceptors is mediated mainly via the cAMP second messenger system, recent evidence suggests that nitric oxide (NO) is also involved in peripheral pain mechanisms. To test the hypothesis that NO contributes to the sensitization of nociceptors to mechanical stimuli induced by hyperalgesic prostaglandins,
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Honoré, Per Hartvig, Anna Basnet, Pernille Kristensen, et al. "Predictive validity of pharmacologic interventions in animal models of neuropathic pain." Scandinavian Journal of Pain 2, no. 4 (2011): 178–84. http://dx.doi.org/10.1016/j.sjpain.2011.06.002.

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AbstractIntroductionThe pathophysiologic and neurochemical characteristics of neuropathic pain must be considered in the search for new treatment targets. Breakthroughs in the understanding of the structural and biochemical changes in neuropathy have opened up possibilities to explore new treatment paradigms. However, long term sequels from the damage are still difficult to treat.Aim of the studyTo examine the validity of pharmacological treatments in humans and animals for neuropathic pain.MethodAn overview from the literature and own experiences of pharmacological treatments employed to inte
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Hefferan, Michael P., and Christopher W. Loomis. "Interaction of Spinal Nitric Oxide and Prostaglandins after L5–L6 Spinal Nerve Ligation in the Rat." Anesthesiology 100, no. 6 (2004): 1611–14. http://dx.doi.org/10.1097/00000542-200406000-00040.

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Nakahata, Katsutoshi, Hiroyuki Kinoshita, Keiko Hama-Tomioka, et al. "Cholinesterase Inhibitor Donepezil Dilates Cerebral Parenchymal Arterioles via the Activation of Neuronal Nitric Oxide Synthase." Anesthesiology 109, no. 1 (2008): 124–29. http://dx.doi.org/10.1097/aln.0b013e31817c0316.

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Background An acetylcholinesterase inhibitor donepezil currently is used to treat patients with Alzheimer disease. However, its direct effect on cerebral blood vessels has not been evaluated. The present study was designed to examine whether donepezil induces acute cerebral arteriolar dilation and whether neuronal nitric oxide synthase contributes to this vasodilator response. Methods Brain slices were obtained from neuronal nitric oxide synthase knock-out or C57BL/6J strain (control) mice as well as Wistar rats. Parenchymal arterioles were monitored using videomicroscopy. During constriction
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Toriyabe, Masaki, Keiichi Omote, Tomoyuki Kawamata, and Akiyoshi Namiki. "Contribution of Interaction between Nitric Oxide and Cyclooxygenases to the Production of Prostaglandins in Carrageenan-induced Inflammation." Anesthesiology 101, no. 4 (2004): 983–90. http://dx.doi.org/10.1097/00000542-200410000-00025.

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Background Nitric oxide (NO) and prostaglandins (PGs) are crucial mediators contributing to generation of inflammatory responses and pain. This study was designed to investigate the effects of peripherally released NO on cyclooxygenase (COX) expression/activation and production of PGs in carrageenan-induced inflammation. Methods A microdialysis probe was implanted subcutaneously into the skin of hind paws of rats. The concentrations of NO metabolites, PGE2, and 6-keto-PGF1alpha (metabolite of PGI2) in the dialysate were measured. Carrageenan was injected into the plantar surface of the hind pa
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Singh, Sumitra, and Bhagwati Devi. "ETHANOMEDICINAL PLANTS WITH ANTI-INFLAMMATORY EFFECT FROM SOUTHERN HARYANA, INDIA: A REVIEW." International Journal of Advanced Research 8, no. 12 (2020): 1013–34. http://dx.doi.org/10.21474/ijar01/12248.

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Inflammation is a physiological host response to external challenges or cellular injury such as pathogens, damaged cells or irritants leading to the release of a complex array of inflammatory mediators and aiding the recovery of tissue structure and function.All inflammatory processes develop along a known sequence: locally increased blood supply, leakage of fluid, small molecule or proteins and infiltration of cells.Inflammation is not a synonym for infection, even in case where inflammation is caused by infection, response includes clinical signs of erythema, edema, hyperalgesia and pain. Si
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Kehl, Franz, Hui Shen, Carol Moreno, et al. "Isoflurane-induced Cerebral Hyperemia Is Partially Mediated by Nitric Oxide and Epoxyeicosatrienoic Acids in Mice In Vivo." Anesthesiology 97, no. 6 (2002): 1528–33. http://dx.doi.org/10.1097/00000542-200212000-00027.

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Background Despite intense investigation, the mechanism of isoflurane-induced cerebral hyperemia is unclear. The current study was designed to determine the contributions of neuronal nitric oxide synthase, prostaglandins, and epoxyeicosatrienoic acids to isoflurane-induced cerebral hyperemia. Methods Regional cerebral cortical blood flow was measured with laser Doppler flowmetry during stepwise increases of isoflurane from 0.0 to 1.2, 1.8, and 2.4 vol% end-tidal concentration in alpha-chloralose-urethane-anesthetized, C57BL/6 mice before and 45 min after administration of the neuronal nitric o
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Dissertations / Theses on the topic "Pain Pain Prostaglandins Nitric Oxide"

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Levy, Dan. "Local nitric oxide synthase expression and nitric oxide action in an animal model of neuropathic pain." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape7/PQDD_0034/NQ38484.pdf.

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Osborne, Michael G. "The role of nitric oxide in carrageenan-induced hyperalgesia." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape7/PQDD_0025/MQ50848.pdf.

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Handy, Rachel Louisa Charlotte. "Isoform selective inhibitors of nitric oxide synthase as tools to study the role of nitric oxide in pain and inflammation in rodents." Thesis, King's College London (University of London), 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.267295.

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GirÃo, VirgÃnia ClÃudia Carneiro. "ParticipaÃÃo do Ãxido nÃtrico (NO) na modulaÃÃo central da hiperalgesia na artrite induzida por zymozan (AZy) em ratos." Universidade Federal do CearÃ, 2006. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=57.

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CoordenaÃÃo de AperfeiÃoamento de Pessoal de NÃvel Superior<br>Conselho Nacional de Desenvolvimento CientÃfico e TecnolÃgico<br>We investigate in this work the role of the central nervous system in the modulation of the inflammatory peripheral pain in the zymosan induced arthritis (AZy) in rats. Cerebrospinal fluid (CSF) and sinovial fluid were collected from animals at different AZy times (1, 3, 6, 12 and 14 hours) in order to determine the nitrite level. Different groups of male, Wistar rats (n=6), 250 to 300g weight, were then submitted to surgery in order to place a cannula in the subarach
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Franchin, Marcelo 1987. "Avaliação do potencial anti-inflamatório e antinociceptivo da geoprópolis de Melipona scutellaris." [s.n.], 2012. http://repositorio.unicamp.br/jspui/handle/REPOSIP/288502.

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Orientador: Pedro Luiz Rosalen<br>Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Odontologia de Piracicaba<br>Made available in DSpace on 2018-08-19T23:06:36Z (GMT). No. of bitstreams: 1 Franchin_Marcelo_M.pdf: 1716627 bytes, checksum: 9d137d15b27856e3799f2681ac713d50 (MD5) Previous issue date: 2012<br>Resumo: O objetivo deste estudo foi avaliar a atividade anti-inflamatória e antinociceptiva do extrato etanólico de geoprópolis (EEGP) de Melipona scutellaris e frações químicas, bem como possíveis mecanismos de ação. Além disso, caracterizar quimicamente o EEGP e fra
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Rocha, Marcelo Gondim. "Mediadores inflamatórios na dor pélvica crônica identificação de possíveis marcadores séricos da doença." Universidade de São Paulo, 2010. http://www.teses.usp.br/teses/disponiveis/17/17145/tde-13092010-175740/.

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ROCHA, MG. Mediadores inflamatórios na dor pélvica crônica Identificação de possíveis marcadores séricos da doença. Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo, Ribeirão Preto, 2010. Introdução: Dor pélvica crônica é uma doença de elevada prevalência e fisiopatologia complexa. Os métodos diagnósticos muitas vezes são insuficientes e, em decorrência, o tratamento e seguimento das mulheres é difícil. Inúmeras doenças que se apresentam com dor crônica tem um perfil inflamatório, que ainda não foi investigado para o tema em questão. Objetivos: Quantificar os níveis de óxido
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Hervera, Abad Arnau. "Role of gaseous neurotransmitters in the effects and expression of opioid and cannabinoid receptors during neuropathic pain." Doctoral thesis, Universitat Autònoma de Barcelona, 2012. http://hdl.handle.net/10803/117583.

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El dolor neuropàtic es causat per una lesió o malaltia del sistema nerviós somatosensiorial, i es caracteritza per la presencia d'alodínia e hiperalgèsia. Actualment el seu tractament es basa en la teràpia amb opioids, però són necessàries altes dosis per alleujar el símptomes, les quals van acompanyades de nombrosos efectes secundaris. Aleshores es necessari investigar noves dianes així com nous mecanismes per tal de millorar els tractaments amb opioids a fi d’evitar l’administració de dosis altes i els efectes no desitjats. En aquest estudi hem investigat el paper que juguen dos dels princip
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Filho, Marcus VinÃcius Ponte de Sousa. "Inhibition of neutrophil migration and hypernociception by remote ischemic preconditioning: participation of the L-arginine-NO-cGMP-CHANNELS K + ATP." Universidade Federal do CearÃ, 2005. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=6432.

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FundaÃÃo de Amparo à Pesquisa do Estado do CearÃ<br>CoordenaÃÃo de AperfeiÃoamento de NÃvel Superior<br>CoordenaÃÃo de AperfeiÃoamento de Pessoal de NÃvel Superior<br>A lesÃo de reperfusÃo (LR) ocorre em diversos orgÃos e tecidos durante o restabelecimento do fluxo sangÃÃneo apÃs episÃdios de isquemia prolongada. Em diversas Ãreas clÃnicas esta lesÃo à temida pelas complicaÃÃes decorrentes da demora no restabelecimento do fluxo vascular. Nos transplantes de ÃrgÃos, nos retalhos microcirÃrgicos e nas cirurgias de revascularizaÃÃo miocÃrdica, a LR, dependendo de sua intensidade, pode prejudicar
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Hoffmeister, Carin Gorete Hendges. "PAPEL DO TRPV1 EM UM MODELO ARTICULAR DE GOTA AGUDA EM RATOS." Universidade Federal de Santa Maria, 2014. http://repositorio.ufsm.br/handle/1/3838.

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Conselho Nacional de Desenvolvimento Científico e Tecnológico<br>The gout is an extremely painful type of arthritis. Despite the large number of drugs available for its treatment, they usually cause many adverse effects that limit their use. Then, further investigations are necessary for a better understanding the different mechanisms involved in gout. It was found previously that TRPV1 receptor, an ion channel modulated by various inflammatory mediators mediated edema and the nociceptive responses induced by subcutaneous injection of MSU in rats. In this plantar model, activation of TRPV1 dep
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Fabri, Gisele Maria Campos. "Doença periodontal grave em pacientes com e sem queixa de dor crônica crânio-facial: correlação dos aspectos clínicos com a análise quantitativa da substância P e do óxido nítrico do tecido gengival inflamado." Universidade de São Paulo, 2007. http://www.teses.usp.br/teses/disponiveis/5/5160/tde-11032008-154135/.

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Objetivos: Avaliar a implicação da doença periodontal (DP) avançada, e da expressão da NOS e sP dos tecidos gengivais inflamados, na intensidade de dor e na qualidade de vida de pacientes com dor crônica crânio-facial. Casuística e Métodos: foram avaliados e tratados 20 pacientes com queixas de dores crônicas crânio-faciais e DP (Grupo de Estudo), comparativamente com 20 pacientes que tinham somente DP (Grupo Controle). Todos os pacientes receberam tratamento cirúrgico periodontal. A avaliação foi realizada pré e pós-tratamento periodontal (7, 30 e 180 dias). Instrumentos de avaliação: ficha c
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Books on the topic "Pain Pain Prostaglandins Nitric Oxide"

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Takao, Kumazawa, Kruger Lawrence, and Mizumura Kazue, eds. The polymodal receptor: A gateway to pathological pain. Elsevier, 1996.

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(Editor), T. Kumazawa, L. Kruger (Editor), and K. Mizumura (Editor), eds. The Polymodal Receptor - A Gateway to Pathological Pain (Progress in Brain Research). Elsevier Science, 1996.

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Book chapters on the topic "Pain Pain Prostaglandins Nitric Oxide"

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Handy, Rachel L. C. "Nitric oxide and inflammatory pain." In Pain and Neurogenic Inflammation. Birkhäuser Basel, 1999. http://dx.doi.org/10.1007/978-3-0348-8753-3_5.

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Willis, William D. "Spinothalamic Tract Neurons, Role of Nitric Oxide." In Encyclopedia of Pain. Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-28753-4_4177.

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Schmidtko, Achim. "Nitric Oxide-Mediated Pain Processing in the Spinal Cord." In Pain Control. Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-662-46450-2_6.

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Zhang, H., T. E. Stewart, and J. L. Vincent. "Nitric Oxide in Sepsis and Nitric Oxide in Sepsis and ARDS." In Anaesthesia, Pain, Intensive Care and Emergency Medicine — A.P.I.C.E. Springer Milan, 1999. http://dx.doi.org/10.1007/978-88-470-2145-7_53.

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Auler, J. O. C. "Nitric Oxide in Pulmonary Hypertension." In Anaesthesia, Pain, Intensive Care and Emergency Medicine — A.P.I.C.E. Springer Milan, 1996. http://dx.doi.org/10.1007/978-88-470-2203-4_66.

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Schlunt, Michelle. "Nitric Oxide and Pulmonary Vasodilators." In Essentials of Pharmacology for Anesthesia, Pain Medicine, and Critical Care. Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-8948-1_17.

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Zhang, H., and J. L. Vincent. "Role of Nitric Oxide in Septic Shock." In Anaesthesia, Pain, Intensive Care and Emergency Medicine - A.P.I.C.E. Springer Milan, 1998. http://dx.doi.org/10.1007/978-88-470-2278-2_2.

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Hedenstierna, G., K. Hambraeus Jonzon, and F. Fredén. "Regional Lung Blood Flow and Inhalation of Nitric Oxide." In Anesthesia, Pain, Intensive Care and Emergency Medicine — A.P.I.C.E. Springer Milan, 2000. http://dx.doi.org/10.1007/978-88-470-2286-7_14.

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Lachmann, B., S. Verbrugge, and D. Gommers. "Combining Exogenous Surfactant or Perfluorocarbons with Inhaled Nitric Oxide to Improve Lung Function in Acute Respiratory Failure." In Anaesthesia, Pain, Intensive Care and Emergency Medicine — A.P.I.C.E. Springer Milan, 1997. http://dx.doi.org/10.1007/978-88-470-2296-6_28.

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Ferreira, Sergio H., Femardo Q. Cunha, and Stephen Hyslop. "Role of the inducible forms of cyclooxygenase and nitric oxide synthase in inflammatory pain." In Inducible Enzymes in the Inflammatory Response. Birkhäuser Basel, 1999. http://dx.doi.org/10.1007/978-3-0348-8747-2_7.

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Conference papers on the topic "Pain Pain Prostaglandins Nitric Oxide"

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Halasa, Salaheldin, and Eva Dickinson. "Combination of nitric oxide therapy, anti-oxidative therapy, low level laser therapy, plasma rich platelet therapy and stem cell therapy as a novel therapeutic application to manage the pain and treat many clinical conditions." In SPIE BiOS, edited by Michael R. Hamblin, James D. Carroll, and Praveen Arany. SPIE, 2014. http://dx.doi.org/10.1117/12.2038329.

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