Relevant bibliographies by topics / Pancreas Pancreatitis

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Pancreas Pancreatitis'

Author: Grafiati
Published: 4 June 2021
Last updated: 16 February 2022

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Journal articles on the topic "Pancreas Pancreatitis"

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Baldiwala, Aafrin S., and Rajesh G. Chandnani. "A case report of portal vein thrombosis and common bile duct compression in the patient having pseudocyst of pancreas and its management by percutaneous trans-hepatic biliary drainage." International Surgery Journal 8, no. 5 (2021): 1643. http://dx.doi.org/10.18203/2349-2902.isj20211849.

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Pancreatitis is a systemic disease owing to release of inflammatory mediators and digestive enzymes. Acute pancreatitis is sudden inflammation of the pancreas. Alcohol and gallstones are main cause of acute pancreatitis. Chronic pancreatitis is the persistent inflammation and irreversible fibrosis associated with atrophy of pancreatic parenchyma. There are various complications associated with pancreatitis such as strictures, pancreatic necrosis, pseudo-cyst of pancreas, pancreato-cutaneous fistulas, venous thrombosis, arterial aneurysm in various arteries around pancreas etc. Common bile duct (CBD) strictures are a common complication in patients with advanced chronic pancreatitis and have a variable clinical presentation ranging from an incidental finding to overt jaundice and cholangitis. CBD strictures occur as a consequence of recurrent acute inflammatory episodes which may ultimately result in a periductal fibrotic stricture. CBD can be compressed as a result of extrinsic compression by large pseudocyst or aneurysm. The diagnosis is mostly made during investigations for abdominal pain but jaundice may be the initial clinical presentation. The jaundice is typically transient but may be recurrent with a small risk of secondary biliary cirrhosis in longstanding cases. Vascular complications in chronic pancreatitis are rare. Venous thrombosis is the most common complication of pancreatitis affecting venous system. It occurs as consequences of an inflammatory mass in head of pancreas, and splenic vein thrombosis occurs in association with chronic pancreatitis in 4-8% cases. Present case is a case of acute pancreatic collection in head of pancreas with aneurysmal small bleeding causing complete CBD compression and extensive venous thrombosis involving superior mesenteric vein, portal vein, splenic.
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Kovalchuk, O. V., L. P. Goralskyi, and I. M. Sokulskyi. "Pathomorphology of cat pancreas under chronic pancreatitis." Scientific Messenger of LNU of Veterinary Medicine and Biotechnologies 23, no. 102 (2021): 87–92. http://dx.doi.org/10.32718/nvlvet10213.

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The paper deals with studying the pathomorphology of cat pancreas under chronic pancreatitis. This paper is a component of a research mix of the Department of Anatomy and Histology, it goes under the title “The development, morphology and histo-chemistry of animal organs in health and in disease”, (state registration number № 0120U100796). A pancreas is an azygos parenchymatous organ which refers to the endoexocrine glands, includes exocrine and endocrine pancreas, is involved in the processes of digestion and regulation of carbohydrate metabolism, protein metabolism and lipid exchange in tissues. Pancreatic juice, which is rich in enzymes (trypsin, lipase, amylase), is produced in an exocrine pancreas, and hormones (insulin, somatostatin, glucagon (vasoactive intestinal polypeptide), pancreatic polypeptide) are produced in endocrine pancreas. This galand is involved in the process of digestion while producing digestive enzymes, which get into the duodenum and hydrolyze practically all parts of feeds which enter the body. It is located in an abdominal cavity, anatomically connected with a stomach, liver and duedenum. It has been found that pathomorphological changes in pancreas under chronic pancreatits manifest themselves depending on the disease stage and are revealed by insignificant progress of the pathological process. Herewith, morphological parameters of pancreas width and length in cats under chronical pancreatitis did not significantly change, but these indices tended to decrease. Its absolute weight in cats under chronical pancreatitis, as compared with clinically healthy cats, did not change and equalled 9.12 ± 2.03 g. But pancreas relative weight in sick cats increased by 1.4 (Р ≤ 0.01) and equalled 0.51 ± 0.08 %, as compared with control 0.38 ± 0.06 %. Under histological analysis of pancreas histology specimen stained with hematoxylin Corazzi and eosin, some distortion in a microscopic structure of a pancreas was observed, it manifested itself in thickening of interparticle tissue-connective layers which spread like desmogenous bands. Some destructive changes in acini in exocrinal pancreas, which manifested themselves in losing their characteristic form, were noticed. The cytoplasm of such acinous cells was in a state of plasmorrhexis, the pycnosis was observed.
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Hernandez, Genaro, Ting Luo, Tanveer A. Javed, et al. "Pancreatitis is an FGF21-deficient state that is corrected by replacement therapy." Science Translational Medicine 12, no. 525 (2020): eaay5186. http://dx.doi.org/10.1126/scitranslmed.aay5186.

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The exocrine pancreas expresses the highest concentrations of fibroblast growth factor 21 (FGF21) in the body, where it maintains acinar cell proteostasis. Here, we showed in both mice and humans that acute and chronic pancreatitis is associated with a loss of FGF21 expression due to activation of the integrated stress response (ISR) pathway. Mechanistically, we found that activation of the ISR in cultured acinar cells and mouse pancreata induced the expression of ATF3, a transcriptional repressor that directly bound to specific sites on the Fgf21 promoter and resulted in loss of FGF21 expression. These ATF3 binding sites are conserved in the human FGF21 promoter. Consistent with the mouse studies, we also observed the reciprocal expression of ATF3 and FGF21 in the pancreata of human patients with pancreatitis. Using three different mouse models of pancreatitis, we showed that pharmacologic replacement of FGF21 mitigated the ISR and resolved pancreatitis. Likewise, inhibition of the ISR with an inhibitor of the PKR-like endoplasmic reticulum kinase (PERK) also restored FGF21 expression and alleviated pancreatitis. These findings highlight the importance of FGF21 in preserving exocrine pancreas function and suggest its therapeutic use for prevention and treatment of pancreatitis.
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Yegorov, V. I., V. A. Koubyshkin, G. G. Karmazanovsky, et al. "Cystic duodenal dystrophy. Typical case as an example of diagnosticsand surgical tactics." Bulletin of Siberian Medicine 6, no. 3 (2007): 65–70. http://dx.doi.org/10.20538/1682-0363-2007-3-65-70.

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Duodenal dystrophy, a chronic inflammation of the aberrant pancreatic tissue in the duodenal wall, is a relatively rare disease in the practice of physicians. The heterotopic pancreas is usually functioning, and the development of acute and chronic pancreatitis in it is even more probable than in the orthotopic gland as a result of an underdeveloped duct system. The progression of ectopic pancreatitis associated with increasing cystic formation could lead to a blockade of the major or minor duodenal papilla and subsequent chronic pancreatitits in the pancreas proper. Furthermore, a malignant transformation of the aberrant pancreas is not a rare occurrence. It is essential to carry out a timely and sharp diagnosis of this condition as it often defines the surgical tactics. The purpose of this report is to present a typical case of cystic duodenal dystrophy.
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Igaz, Péter, and Zsolt Tulassay. "Autoimmune pancreatitis." Orvosi Hetilap 149, no. 19 (2008): 873–76. http://dx.doi.org/10.1556/oh.2008.28319.

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Az autoimmun pancreatitis a krónikus pancreatitis ritka oka. Klinikai jelentősége mégsem lebecsülendő, mivel részben a rosszindulatú hasnyálmirigyráktól történő elkülönítése nehézséget okozhat, részben pedig az idült pancreatitisek többségétől eltérően jól kezelhető, szteroidkezelésre teljes regressziója is előfordulhat. Klinikai képe nem jellegzetes, az elzáródásos sárgaság, hasi fájdalom, fogyás gyakori. A képalkotó vizsgálatok jellemzően a pancreas diffúz megnagyobbodását és a Wirsung-vezeték egyenetlen szűkületét mutatják. Autoimmun pancreatitisben szenvedőkben az IgG4-immunglobulin növekedett szérumkoncentrációját, autoantitesteket és IgG4-pozitív immunsejtek jelenlétét mutatták ki más szövettani jellegzetességek mellett. A hasnyálmirigy érintettsége mellett egyéb szervek is megbetegedhetnek, így pl. sclerotisáló cholangitisszel, sialoadenitisszel, retroperitonealis fibrosissal, Riedel-strumával és gyulladásos bélbetegségekkel való társulását is leírták. Mindezek alapján az autoimmun pancreatitis rendszerbetegségnek tartható, szisztémás IgG4-asszociált sclerotisáló kórkép egyik megjelenési formájaként.
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Tulassay, Zsolt, János Pap, and Ivan E. Farkas. "Pancreas divisum and pancreatitis." Gastroenterology 91, no. 1 (1986): 267. http://dx.doi.org/10.1016/0016-5085(86)90492-0.

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Staritz, Martin, Thomas Hütteroth, and Karl-Hermann Meyer Zum Büschenfelde. "Pancreas divisum and pancreatitis." Gastroenterology 91, no. 2 (1986): 525–26. http://dx.doi.org/10.1016/0016-5085(86)90615-3.

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Crișu, Georgiana, Monica Grigore, V. Balaban, et al. "Groove Pancreatitis - Cause of Recurrent Pancreatitis." Internal Medicine 16, no. 3 (2019): 71–77. http://dx.doi.org/10.2478/inmed-2019-0071.

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AbstractBackground. Groove pancreatitis or paraduodenal pancreatitis represents a rare type of pancreatitis, and can be classified into cystic dystrophy of the duodenal wall in heterotopic pancreas, paraduodenal cyst or myoadenomatosis.Case presentation. We present a case of a 58 year old man, drinker and smoker who was admitted in the Department of Gastroenterology for abdominal pain, weight loss and nausea. From his history we have noticed frequent presentations of recurrent acute pancreatitis in the last two years. Laboratory tests have revealed cholestasis, high value of lipase and high value of amylase, with normal value of CA 19.9. The magnetic resonance from the last two years showed the same appearances: a large and edematous head of pancreas, a thickening of the wall of adjacent duodenum and an inhomogeneous area with cystic transformation in the head of the pancreas. We performed endoscopic ultrasound with fine needle aspiration. The histopathological result showed only inflammatory cells. We have established the diagnosis of groove pancreatitis.Conclusion. Groove pancreatitis represents a rare condition, with an incidence of 0.4%-14% on biopsies. Endoscopic ultrasound is the best method for diagnosis, it could evaluate also the duodenal wall.
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Bu, Heng-Fu, Saravanan Subramanian, Hua Geng, et al. "MFG-E8 Plays an Important Role in Attenuating Cerulein-Induced Acute Pancreatitis in Mice." Cells 10, no. 4 (2021): 728. http://dx.doi.org/10.3390/cells10040728.

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Milk fat globule-EGF factor 8 (MFG-E8) is a secreted glycoprotein that regulates tissue homeostasis, possesses potent anti-inflammatory properties, and protects against tissue injury. The human pancreas expresses MFG-E8; however, the role of MFG-E8 in the pancreas remains unclear. We examined the expression of MFG-E8 in the pancreas at baseline and during cerulein-induced acute pancreatitis in mice and determined whether MFG-E8 attenuates the progression of pancreatitis, a serious inflammatory condition that can be life-threatening. We administered cerulein to wild-type (WT) and Mfge8 knockout (KO) mice to induce pancreatitis. Immunoblot analysis showed that MFG-E8 is constitutively expressed in the murine pancreas and is increased in mice with cerulein-induced acute pancreatitis. In situ hybridization revealed that ductal epithelial cells in the mouse pancreas express Mfge8 transcripts at baseline. During pancreatitis, Mfge8 transcripts were abundantly expressed in acinar cells and endothelial cells in addition to ductal epithelial cells. Knocking out Mfge8 in mice exacerbated the severity of cerulein-induced acute pancreatitis and delayed its resolution. In contrast, administration of recombinant MFG-E8 attenuated cerulein-induced acute pancreatitis and promoted repair of pancreatic injury in Mfge8 KO mice. Taken together, our study suggests that MFG-E8 protects the pancreas against inflammatory injury and promotes pancreatic tissue repair. MFG-E8 may represent a novel therapeutic target in acute pancreatitis.
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Shrikhande, Shailesh, Helmut Friess, Claudia Issenegger, et al. "Fluconazole Penetration into the Pancreas." Antimicrobial Agents and Chemotherapy 44, no. 9 (2000): 2569–71. http://dx.doi.org/10.1128/aac.44.9.2569-2571.2000.

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ABSTRACT Because of antibiotic prophylaxis for necrotizing pancreatitis, the frequency of fungal superinfection in patients with pancreatic necrosis is increasing. In this study we analyzed the penetration of fluconazole into the human pancreas and in experimental acute pancreatitis. In human pancreatic tissues, the mean fluconazole concentration was 8.19 ± 3.38 μg/g (96% of the corresponding concentration in serum). In experimental edematous and necrotizing pancreatitis, 88 and 91% of the serum fluconazole concentration was found in the pancreas. These data show that fluconazole penetration into the pancreas is sufficient to prevent and/or treat fungal contamination in patients with pancreatic necrosis.
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Dissertations / Theses on the topic "Pancreas Pancreatitis"

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Liu, Chi-leung. "The role of endoscopic ultrasonography in the management of acute pancreatitis." Click to view the E-thesis via HKUTO, 2005. http://sunzi.lib.hku.hk/hkuto/record/B42577354.

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Cherelyn, Vella. "Coxsackie B4 virus infection of the pancreas - a murine model." Thesis, Queen Mary, University of London, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.281620.

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Fernandes, Cátia Conceição da Encarnação. "Clínica médica e cirúrgica em animais de companhia: pancreatite em animais de companhia." Master's thesis, Universidade de Évora, 2015. http://hdl.handle.net/10174/18224.

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Este relatório apresenta a conclusão de seis meses de estágio curricular incorporado no Mestrado Integrado em Medicina Veterinária e descreve as atividades médico-veterinárias realizadas e assistidas num Hospital e Clínica de referência. Tem como parte integrante uma revisão bibliográfica médica atual do estado de arte da Pancreatite em Animais de Companhia e posterior discussão de casos clínicos. A pancreatite é uma doença inflamatória de particular importância. O seu comportamento dinâmico e as espécies animais atingidas determinam a sua imprevisibilidade. Nos gatos, a possível associação de outras co morbilidades inflamatórias toma, por vezes, a pancreatite um desafio. Estudos recentes conferem uma nova visão sobre esta doença que até então era turva, pouco compreendida e assente em alguns aspetos que hoje se confirmam totalmente paradoxais e erráticos. O foco dirige-se para a nutrição enteral precoce e à combinação da mensuração da lipase pancreática e ecografia como os meios de diagnóstico de eleição; ABSTRACT: This Report presents the conclusion of a six months of internship incorporated into Veterinary Medicine Master's degree and is intended to describe the medical and Veterinary activities done and assisted in a hospital and reference clinic. lt includes a review of current medical literature on state of the science regarding Pancreatitis in Companion Animals and further discussion of clinical cases. Pancreatitis is an inflammatory disease of particular importance. lts dynamic behavior and the affected animal species determine its unpredictability. In cats due to the possible association of the other comorbidities of inflammatory nature make sometimes pancreatitis a challenge. Recent studies provide new insight into this disease, which until now was blurred, poorly understood and based on some aspects that today are considered completely paradoxical and erratic. Currently the focus is directed to the early enteral nutrition and in what concerns the diagnosis of pancreatitis, measurement of pancreatic lipase and ultrasound are preferred tools/methods.
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Smithies, Alison Marie. "A study of the role of cytokines in acute pancreatitis in man." Thesis, University of Plymouth, 2001. http://hdl.handle.net/10026.1/1988.

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Introduction: Acute pancreatitis is an inflammatory disease with a diverse aetiology and variable clinical course. The IL-l gene cluster has been implicated in this disease. Aims: The aims of the study were to investigate polymorphisms of the genes encoded within the IL-l gene cluster in patients with acute pancreatitis and normal controls and to determine the relationship between the polymorphisms and protein levels. Methods: Genotype and allele frequencies were determined in controls (n=217) and patients with acute pancreatitis (n=137) using the polymerase chain reaction (PCR) followed by digestion with restriction endonucleases where applicable. Protein levels were determined using in vitro stimulation of PBMCs followed by Enzyme Linked Immunosorbent Assay (ELISA). Patients were categorised according to severity, organ failure scores and aetiology. Results: Allele l of the VNTR86 polymorphism in the IL-l RN gene was significantly increased in the severe group of patients compared to controls (81.9% vs 63.0%, x2=9.38, p=0.002, Pc=0.004) and in the idiopathic group compared to controls (82.4% vs 63.0%, x2=9.33, p=0.002, Pc=0.004). The polymorphisms within the genes and between the genes were strongly linked. Significantly more of the Mspl-VLP-VNTR86-Sspl 2-3-2-2 haplotype was observed in the control (15.7% vs 0.1%, x2 =2528.11, p<0.000000l, Pc<0.0000001) and patient (14.0% VS 0.1%, x2 =4368.10, p<0.000000l, Pc<0.0000004) populations than expected. Significantly more of the Pstl-Aval-Alul-Taql 2-2-2-1 haplotype was observed in controls (27.7% vs 9.7%, x2 =31.39, p<0.000000l, Pc<0.0000005) and patients (12.5% vs 2.0%, x2=53.69, p<0.000000l, Pc=0.0000007) than expected. Preferential combinations of the genotypes existed within controls and patients. The median IL-lα and IL-lβ protein levels from unstimulated PBMCs were significantly increased in patients compared to controls: median values (interquartile range). In the IL-lα study, significant differences were found at 24 hours: 193.5 (127.5-363.5) pg/ml vs 1.0 (0.0-3.0) pg/ml, p=0.005, 48 hours: 256.5 (171.5-417.0) pg/ml vs 6.5 (2.0-16.0) pg/ml, p=0.006 and at 72 hours: 210.5 (138-427) pg/ml vs 0.5 (0-7) pg/ml, p=0.005. In the IL-l β study, significant differences were found at 24 hours: 663 (507-782) pg/ml vs 12 (5-53) pg/ml, p=0.004, 48 hours: 620 (570-1080) pg/ml vs 14.5 (11-36) pg/ml, p=0.004 and at 72 hours: 545.5 (442-771) pg/ml vs 12.5 (2-43) pg/ml, p=0.006. Conclusion: Polymorphisms of the IL-l gene cluster are associated with susceptibility to and/or severity of the acute pancreatitis. Polymorphisms within the IL-l gene cluster are in linkage disequilibrium. Unstimulated PBMCs from patients with acute pancreatitis secrete significantly more IL-la and IL-IP protein levels compared to those from controls. The (AC)n, Alu I and VNTR86 polymorphisms do not correspond to differences in functional protein levels.
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Vonlaufen, Alain Clinical School South Western Sydney Faculty of Medicine UNSW. "Alcohol, endotoxin and the pancreas (induction, progression and reversibility of alcoholic pancreatitis)." Publisher:University of New South Wales. Clinical School - South Western Sydney, 2009. http://handle.unsw.edu.au/1959.4/43721.

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This thesis pertains to the pathogenesis of alcoholic pancreatitis, a considerable burden in terms of morbidity, mortality and health related costs. It has long been known that only a minority of alcoholics develop clinically evident pancreatitis, suggesting that (an) additional trigger factor(s) is required to elicit overt disease. Endotoxin (lipopolysaccharide LPS), from gut-derived gram negative bacteria may be one such trigger factor, since alcoholics exhibit increased levels of serum endotoxin. In addition, the degree of endotoxinaemia has been reported to correlate with the severity of pancreatitis. Studies described in this thesis report, i) the development of a novel rodent model of alcoholic pancreatitis produced by challenging alcohol-fed animals with single or repeated doses of LPS. The animals exhibit features of both acute (acinar vacuolisation, necrosis, pancreatic oedema, haemorrhage and inflammatory infiltration) and chronic (acinar atrophy and pancreatic fibrosis) pancreatitis; ii) the reversion of pancreatic injury (including fibrosis) upon withdrawal of alcohol in the model and the persistence of pancreatic damage with continuation of alcohol feeding; iii) activation of pancreatic stellate cells (PSCs, known to play a central role in fibrogenesis) in vivo and in vitro by alcohol and LPS; iv) the inhibition of PSC apoptosis in vivo and in vitro upon exposure to alcohol and LPS and the induction of PSC apoptosis in vivo upon withdrawal of alcohol from the diet and v) the presence of LPS receptors TLR4 and CD14 on PSCs, which would explain the responsiveness of PSCs to LPS. Thus the work in this thesis provides strong evidence in support of endotoxin as a clinically relevant trigger factor for the initiation of alcoholic pancreatitis and as a factor that promotes disease progression. The thesis also provides the first experimental evidence to support the clinical reports of a beneficial effect of abstinence on chronic pancreatitis. Delineation of the mechanisms mediating the induction, progression and reversibility of alcoholic pancreatitis has the potential to direct the development of new therapeutic interventions for alcohol-related pancreatic injury.
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Nunes, Quentin. "The role of heparin-binding proteins in normal pancreas and acute pancreatitis." Thesis, University of Liverpool, 2015. http://livrepository.liverpool.ac.uk/2009329/.

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Acute pancreatitis (AP) is a leading cause for hospitalisation and has significant quality of life implications for the patient and cost implications for the National Health Service. Although most episodes of AP are mild and self-limiting, the severe form of the disease is associated with a high mortality. In the absence of definitive treatment, management is mainly supportive. There is an urgent need to develop more effective biomarkers and drugs to manage AP. Genome-wide studies have demonstrated that proteins that bind to heparin (HBPs) form highly interconnected networks which are functionally important in health and disease. It was hypothesized that this is true in the pancreas and in AP. Testing this hypothesis, using mRNA as a proxy for protein, it was shown that HBPs constitute an important extracellular sub-proteome within the normal pancreas and in major pancreatic diseases that is likely to provide a rich repository of potential biomarkers and drug targets. Building upon this work, a proteomic analysis of HBPs in normal pancreas (NP) and in caerulein-induced mouse AP was undertaken. This has more than doubled the number of HBPs to 883, with 460 new HBPs identified. These may represent the most interconnected set of extracellular proteins and therefore with the greatest regulatory potential. Non canonical HBPs such as NDUFS4, NDUFS6, NDUFS7, NDUFS8, NDUFA9, NDUFA10, NDUFA9 and NDUFA10 were identified and found to be underexpressed in AP as compared to NP. These may have potential moonlighting roles, not previously known. By virtue of being extracellular and binding to heparin, HBPs are accessible and are potential biomarkers and drug targets in AP. In addition to identifying existing biomarkers in AP such as pancreatic amylase, a number of HBPs with biomarkers potential such as HRG, CD14 and FN1 were identified and need further investigation. HBPs such as SERPINC1, VEGFA and PIP5K1C need further evaluation in drug development. These along with modified heparins, heparin mimetics and matrix therapy in AP provide exciting areas for future research.
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Liu, Chi-leung, and 廖子良. "The role of endoscopic ultrasonography in the management of acute pancreatitis." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2004. http://hub.hku.hk/bib/B42577354.

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Charrier, Alyssa. "Connective Tissue Growth Factor in Pancreatitis." The Ohio State University, 2013. http://rave.ohiolink.edu/etdc/view?acc_num=osu1366025057.

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Gestic, Martinho Antonio. "Tratamento cirúrgico da pancreatite crônica com a técnica de Frey = análise dos resultados." [s.n.], 2010. http://repositorio.unicamp.br/jspui/handle/REPOSIP/309057.

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Orientadores: Elinton Adami Chaim, José Carlos Pareja<br>Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Ciências Médicas<br>Made available in DSpace on 2018-08-16T22:56:32Z (GMT). No. of bitstreams: 1 Gestic_MartinhoAntonio_M.pdf: 3381516 bytes, checksum: 2ac6e6bb2938cf49388dd92cfe4fb63c (MD5) Previous issue date: 2010<br>Resumo: O tratamento cirúrgico da pancreatite crônica é indicado na falência do tratamento clínico da dor e na presença de complicações da doença. O emprego da melhor técnica ainda é um desafio e, ao longo do último século, várias técnicas foram desenvolvidas determinando três padrões de procedimento: descompressivos, ressecativos e mistos. A técnica de Frey é do tipo mista, recentemente desenvolvida e que apresenta excelentes resultados no alívio da dor secundária à pancreatite crônica. Seu princípio propõe baixas taxas de morbidade e mortalidade pós-operatórias e menor dano às funções pancreáticas comparáveis às cirurgias descompressivas (Partington-Rochelle, Puestow) com a mesma efetividade das cirurgias ressecativas (duodenopancreatectomias) no controle da dor. O objetivo deste trabalho é descrever a casuística e analisar os resultados de uma série consecutiva de pacientes com pancreatite crônica submetidos à técnica de Frey no Hospital de Clínicas da UNICAMP. Foram analisados retrospectivamente 73 pacientes consecutivos de janeiro de 1991 a dezembro de 2007, sem tratamento cirúrgico prévio para pancreatite crônica e com pelo menos um ano de seguimento pós-operatório. Estudou-se o perfil da população, indicação cirúrgica, complicações pós-operatórias e resultados a longo prazo no controle da dor e das complicações. Os pacientes apresentaram idade média de 40,6 anos, sendo a maioria homens (97,3%) e a etiologia alcoólica foi a mais freqüente (95,9%). A dor abdominal acometia todos os pacientes, 98,8% com intensidade moderada ou severa. A taxa de morbidade global foi de 28,7% e as complicações mais freqüentes foram as infecciosas (13,7%), dentre elas as pneumonias; a prevalência de fístulas da anastomose pancreática foi de 6,8%. Não houve mortalidade cirúrgica. Em seguimento médio de 77,0 meses, 91,4% dos pacientes apresentavam remissão dolorosa completa e houve aumento do IMC no pós-operatório (p < 0,001). Insuficiência exócrina nova apareceu em 49% dos pacientes e diabetes de novo em 36,7%. A recidiva de ingestão alcoólica ocorreu em 32,9% dos pacientes, os quais apresentaram menor expectativa de vida em relação àqueles que se mantiveram abstêmios (p = 0,02). As principais causas de mortalidade tardia foram as neoplasias do trato aéreo-digestivo superior e complicações de cirrose hepática. Identificaram-se associações estatisticamente significativas entre abstinência alcoólica pré-operatória com menor taxa de complicações infecciosas e fistulas; a quantidade de ingestão de álcool e o tempo de aparecimento de diabetes nova pós-operatória; o calibre do ducto pancreático com o surgimento de diabetes pós-operatória; e níveis elevados de amilase sérica e no liquido do dreno abdominal no primeiro dia pós-operatório com fístulas. A técnica de Frey mostrou-se uma opção segura e eficaz para o tratamento cirúrgico da pancreatite crônica, proporciononando melhora da sintomatologia dolorosa e reganho de peso e não interrompeu a deterioração das funções exócrina e endócrina do pâncreas. A recidiva do abuso da ingestão de etanol é um problema freqüente nesses pacientes e interfere na sobrevida deles<br>Abstract: Surgical treatment of chronic pancreatitis is indicated for failure in clinical pain management of the disease. Frey's surgery is one of the techniques available for intervention in this process. Application of the best technique is still a challenge today and, over the last century, several techniques were developed by determining three patterns of treatment: decompression, resection and mixed. Frey's procedure is a mixed technique, which was recently developed and shows excellent results in pain relief. Its principle suggests low rates of morbidity and mortality after surgery and less damage to pancreatic function comparable to surgical decompression (Partington-Rochelle, Puestow) with the same effectiveness of the resection procedures (pancreatoduodenectomy) in pain control. The aim of this paper is to describe and analyze the results of a consecutive series of patients with chronic pancreatitis underwent Frey technique at the Hospital de Clínicas of UNICAMP. Seventy-three consecutive patients were retrospectively analyzed from January 1991 to December 2007, with no previous surgical treatment for chronic pancreatitis and with at least one year of follow-up. We studied the profile of the population, surgical indication, postoperative complications and long-term results in controlling pain and complications. Patients had mean age of 40.6 years, most were men (97.3%) and alcoholic etiology was the most frequent (95.9%). Abdominal pain gripped all patients, 98.8% with moderate or severe intensity. The overall morbidity rate was 28.7% and the most frequent complications were infections (13.7%), among them pneumonia. Fistulas of pancreatic anastomosis were 6.8%. There was no surgical mortality. At mean follow-up of 77.0 months, 91.4% of patients had complete pain remission and there was increased of BMI postoperatively (p < 0.001). New exocrine insufficiency appeared in 49% and new diabetes in 36.7%. Recurrence of alcohol consumption occurred in 32.9% of patients, which showed a lower life expectancy than those who remained abstinent (p = 0.02). The main causes of late death were neoplasm of the upper aero-digestive tract and complications of liver cirrhosis. We identified significant associations between preoperative alcohol abstinence with a lower rate of infectious complications and fistulas; the amount of alcohol intake and time of onset of postoperative diabetes; diameter of the pancreatic duct with the onset of postoperative diabetes; and elevated levels of amylase in blood and abdominal drain fluid on the first postoperative day with fistulas. Frey procedure proved to be a safe and effective option for the surgical treatment of disabling pain caused by chronic pancreatitis provided regained weight but did not stop the deterioration of exocrine and endocrine functions of the pancreas. Recurrence of ethanol abuse is a frequent problem in these patients and interferes with their life expectancy<br>Mestrado<br>Fisiopatologia Cirúrgica<br>Mestre em Ciências da Cirurgia
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Dawson, Amanda Caroline St Vincent???s Hospital Clinical School UNSW. "Evaluation of novel molecular markers from the WNT pathway : a stepwise regression model for pancreatic cancer survival." Awarded by:University of New South Wales. St Vincent???s Hospital Clinical School, 2007. http://handle.unsw.edu.au/1959.4/31528.

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Optimisation of the conventional tripartite of pancreatic cancer (PC) treatment have led to significant improvements in mortality, however further knowledge of the underlying molecular processes is still required. Transcript profiling of mRNA expression of over 44K genes with microarray technology demonstrated upregulation of secreted frizzled related protein 4 (sFRP4) and ??-catenin in PC compared to normal pancreata. Their pathway ??? Wnt signalling is integral to transcriptional regulation and aberrations in these molecules are critical in the development of many human malignancies. Immunohistochemistry protocols were evaluated by two independent blinded examiners for antigen expression differences associated with survival patterns in 140 patients with biopsy verified PC and a subset of 23 normal pancreata with substantial observer agreement (kappa value 0.6-0.8). A retrospective cohort was identified from 6 Sydney hospitals between 1972-2003 and archival formalin fixed tissue was collected together with clinicopathological data. Three manual stepwise regression models were fitted for overall, disease-specific and relapse-free survival to determine the value of significant prognostic variables in risk stratification. The models were fitted in a logical order using a careful strategy with step by step interpretation of the results. Immunohistochemistry demonstrated increased sFRP4 membranous expression (&gt 10%) in 49/95 PC specimens and this correlated with improved overall survival (HR:0.99;95%CI:0.97-6.40;LRchi2=134.75; 1df; ??&lt 0.001). Increased sFRP4 cytoplasmic staining (&gt 2/3) in 46/85 patients increased the disease-specific survival (HR:0.52;95%CI:0.31-0.89;LR test statistic =248.40;1df;??&lt 0.001). Increasing ??-catenin membranous expression (&lt _60%) in 26/116 patients was associated with an increased risk of overall death (HR:3.18;95%CI:1.14-8.89;LR test statistic =4.61;1df,??&lt 0.05). Increasing cytoplasmic expression in 65/114 patients was protective and was associated with prolonged survival on univariate, but not multivariate analysis (Disease specific survival HR:0.75;95%CI:0.56-1.00;logrank chi2=3.91;1df; ??=0.05). Increased nuclear ??-catenin expression in 65/114 patients was associated with prolonged survival (disease-specific HR:0.92;95%CI:0.83-1.02; LR test statistic= 49.72;1df;??&lt 0.001). At the conclusion, 12 patients (8.6%) remained alive, 122 died of their disease (68 males versus 54 females). They were followed for a median of 8.7 months (range 1.0-131.3) months. The median age was 66.5 years (range 34.4-96.0, standard deviation 10.9) years. Pancreatic resection was achieved in 79 patients with 46.8% achieving RO resection. The 30 day post-operative mortality was 2.1%. The overall 1 year survival rate was (33.7% ; 95%CI: 25.78-33.79) with a 5 year survival of (2.87%, 95%CI: 2.83-6.01) and a median survival of (8.90 months; 95%CI: 7.5-10.2). The median disease-specific survival was (9.40; 95%CI: 7.9-10.5 months) and the median time to relapse was 1.2 months (95%CI 1.0-1.2 months). A central tenet of contemporary cancer research is that an understanding of the genetic and molecular abnormalities that accompany the development and progression of cancer is critical to further advances in diagnosis, treatment and eventual prevention. High throughput tissue microarrays were used to study expression of two novel tumour markers in a cohort of pancreatic cancer patients and identified sFRP4 and ??-catenin as potential novel prognostic markers.
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Books on the topic "Pancreas Pancreatitis"

1

Chamberlain, Ronald S., and Avram M. Cooperman. The pancreas revisited: Diagnosis, chronic pancreatitis. W.B. Saunders, 2001.

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Achkasov, Evgeniy, Andrey Pugaev, Maksim Zabelin, and Vladislav Posudnevskiy. Acute pancreatitis: clinic, diagnosis, treatment. INFRA-M Academic Publishing LLC., 2019. http://dx.doi.org/10.12737/995531.

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The textbook consistently highlights the issues of anatomy and physiology of the pancreas, etiology, pathogenesis, classification, clinical picture, diagnosis and treatment of acute pancreatitis. Special attention is paid to determining the severity and prognosis of the disease. Modern approaches to treatment taking into account the severity of the disease, features of suppression of secretory activity of the pancreas and the role of nutritional support in the complex treatment of acute pancreatitis are presented. Attention is drawn to the timing of minimally invasive interventions for uninfected and infected postnecrotic fluid formations, as well as methods of surgical treatment in the phase of purulent-necrotic complications of acute pancreatitis. For the first time in the educational edition psychological aspects of rehabilitation of surgical patients are presented. Mastering the material of the textbook is facilitated by test tasks and questions for self-control.&#x0D; Meets the requirements of the Federal state educational standards of higher education of the last generation.&#x0D; It is intended for students of medical universities, clinical residents and doctors studying in the system of additional professional education, specialty "Surgery".
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Gardner, Timothy B., and Christopher E. Forsmark. Prediction and management of severe acute pancreatitis. Springer, 2015.

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Antonio, Tiengo, ed. Diabetes secondary to pancreatopathy: Proceedings of the Post EASD International Symposium on Diabetes Secondary to Pancreatopathy, Padova, Italy, 21-22 September 1987. Excerpta Medica, 1988.

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Falk, Symposium (83rd 1995 Bolzano Italy). Advances in hepatobiliary and pancreatic diseases: Special clinical topics : proceedings of the Falk Symposium no. 83, held in Bolzano, Italy, April 7-8, 1995. Kluwer Academic Publishers, 1995.

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1940-, Carter David C., and Warshaw Andrew L, eds. Pancreatitis. Churchill Livingstone, 1989.

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(Editor), Andrew L. Warshaw, ed. Pancreatitis. Churchill Livingstone, 1989.

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Diseases of the pancreas: Acute pancreatitis, chronic pancreatitis, neoplasms of the pancreas. 2nd ed. Karger, 2004.

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Malfertheiner, Peter, Waldemar Uhl, and Michael G. Sarr. Diseases of the Pancreas: Acute Pancreatitis, Chronic Pancreatitis, Tumours of the Pancreas. 2nd ed. S. Karger Publishers (USA), 2004.

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Grammatikopoulos, Tassos. The pancreas. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198759928.003.0050.

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The chapter on the pancreas includes some basic facts in regards to anatomy and physiology, but then goes on to focus on the diagnosis and management of pancreatitis, hereditary pancreatitis, and autoimmune pancreatitis. It covers the recent advances in genetics, nutritional management, interventional diagnostic or therapeutic procedures, and recommended treatment regimens.
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Book chapters on the topic "Pancreas Pancreatitis"

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Lowenfels, Albert B., and Patrick Maisonneuve. "Chronic Pancreatitis." In The Pancreas. John Wiley & Sons, Ltd, 2018. http://dx.doi.org/10.1002/9781119188421.ch50.

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Aßfalg, Volker, Norbert Hüser, and Helmut Friess. "Chronic Pancreatitis." In The Pancreas. John Wiley & Sons, Ltd, 2018. http://dx.doi.org/10.1002/9781119188421.ch61.

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Ito, Tetsuhide, Masayuki Hijioka, and Tooru Shimosegawa. "Early Chronic Pancreatitis." In The Pancreas. John Wiley & Sons, Ltd, 2018. http://dx.doi.org/10.1002/9781119188421.ch44.

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Shelton, Celeste, and David C. Whitcomb. "Hereditary Chronic Pancreatitis." In The Pancreas. John Wiley & Sons, Ltd, 2018. http://dx.doi.org/10.1002/9781119188421.ch45.

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Klöppel, Günter. "Histopathology of Acute Pancreatitis." In The Pancreas. John Wiley & Sons, Ltd, 2018. http://dx.doi.org/10.1002/9781119188421.ch19.

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Lowe, Mark E., and Véronique D. Morinville. "Acute Pancreatitis in Children." In The Pancreas. John Wiley & Sons, Ltd, 2018. http://dx.doi.org/10.1002/9781119188421.ch23.

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Dudeja, Vikas, Rajinder Dawra, and Ashok K. Saluja. "Pathophysiology of Experimental Pancreatitis." In The Pancreas. John Wiley & Sons, Ltd, 2018. http://dx.doi.org/10.1002/9781119188421.ch6.

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Kamisawa, Terumi, and Tooru Shimosegawa. "Epidemiology of Autoimmune Pancreatitis." In The Pancreas. John Wiley & Sons, Ltd, 2018. http://dx.doi.org/10.1002/9781119188421.ch63.

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Zen, Yoh. "Pathogenesis of Autoimmune Pancreatitis." In The Pancreas. John Wiley & Sons, Ltd, 2018. http://dx.doi.org/10.1002/9781119188421.ch64.

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Gupta, Rajib, and Vikram Deshpande. "Histology of Autoimmune Pancreatitis." In The Pancreas. John Wiley & Sons, Ltd, 2018. http://dx.doi.org/10.1002/9781119188421.ch65.

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Conference papers on the topic "Pancreas Pancreatitis"

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Moura, DB, N. Nunes, M. Flor de Lima, et al. "Recurrent Acute Pancreatitis In Pediatrics – Pancreas Divisum." In ESGE Days 2021. Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1724948.

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LAYER, PETER, and JUTTA KELLER. "HUMAN EXOCRINE PANCREATIC SECRETION IN NORMAL PANCREAS AND CHRONIC PANCREATITIS." In Proceedings of the 92nd Course of the International School of Medical Sciences. WORLD SCIENTIFIC, 1999. http://dx.doi.org/10.1142/9789814447249_0010.

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Schepis, T., A. Tringali, T. Voiosu, et al. "PANCREAS DIVISUM AND RECURRENT PANCREATITIS: LONG-TERM RESULTS OF MINOR PAPILLA SPHINCTEROTOMY." In ESGE Days 2019. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1681602.

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Ashraf, S., K. Lam, and G. Hu. "When Your Pancreas Breaks Your Heart: A Case of Pancreatitis Induced Takotsubo Cardiomyopathy." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a3568.

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Nguyen, Thien Ly, Purba Singh, Parash Parajuli, et al. "Abstract A38: Twist1-driven fatty pancreas formation facilitates pancreatitis and pancreatic ductal adenocarcinoma progression." In Abstracts: AACR Special Conference: Advances in Modeling Cancer in Mice: Technology, Biology, and Beyond; September 24-27, 2017; Orlando, Florida. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.mousemodels17-a38.

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Kopantzev, Eugene P., Eugenia Usova, Marina Kopantseva, et al. "Abstract 4277: Comparative gene expression analysis of proliferating stromal cells from pancreatic ductal adenocarcinoma, pancreatitis and normal pancreas." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-4277.

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Espinel, J., E. Pinedo, V. Cano, R. Pérez, and M. Jiménez. "Minor Papilla Sphincterotomy And Biodegradable Pancreatic Stent In The Management Of Pancreas Divisum With Recurrent Pancreatitis In A Patient." In ESGE Days 2021. Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1724838.

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MK, Melzer, J. Gout, S. Schirge, et al. "The role of the transcription factor Tbx3 in the embryonic development of the murine pancreas and regeneration of acute pancreatitis." In DGVS Digital: BEST OF DGVS. © Georg Thieme Verlag KG, 2020. http://dx.doi.org/10.1055/s-0040-1716145.

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Xie, Jiarong, and Lei Xu. "IDDF2018-ABS-0086 Non-alcoholic fatty pancreas disease as an independent predictor of acute pancreatitis: a retrospective study of 264 patients." In International Digestive Disease Forum (IDDF) 2018, Hong Kong, 9–10 June 2018. BMJ Publishing Group Ltd and British Society of Gastroenterology, 2018. http://dx.doi.org/10.1136/gutjnl-2018-iddfabstracts.101.

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SILVA, WILLAMS ALVES DA, KELLY GUEDES DA SILVA, LAIZE MEIRELLY ALVES SILVA, et al. "CONTRIBUIÇÃO DO FARMACÊUTICO NA MELHORIA DA QUALIDADE DE VIDA DO PACIENTE DIABÉTICO." In Brazilian Congress. brazco, 2020. http://dx.doi.org/10.51162/brc.health2020-00038.

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A Diabetes mellitus (DM) faz parte do grupo de doencas metabolicas que resulta em defeitos na secrecao ou na acao do hormonio insulina, que e produzido no pancreas, pelas chamadas celulas beta pancreaticas. Portando, este trabalho tem por objetivo demonstrar a importancia do profissional farmaceutico na melhoria da qualidade de vida dos pacientes diabeticos, garantindo resultado no tratamento farmacologico. Trata-se de uma revisao narrativa da literatura, onde foram utilizadas as seguintes bases de dados: Scielo, Medline, Lilacs, Pubmed. Foram utilizados os artigos que se encontraram disponiveis na integra, publicados entre os anos de 2011 a 2020. E comum a nao aceitacao ao tratamento por parte de alguns pacientes, ocasionando em um descontrole nos niveis de glicemia e aumenta as chances de mortalidade. Com isso ha um aumento dos riscos de doencas cardiovasculares, neuropatia, retinopatia e hospitalizacoes. Desta forma, e importante no cuidado farmaceutico a identificacao de problemas relacionados ao uso do medicamento, como por exemplo, a efetividade da farmacoterapia, as reacoes adversas que o paciente pode apresentar e a polifarmacia (o uso concomitante de quatro medicamento ou mais), que e bastante comum em pacientes com diabetes,
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Reports on the topic "Pancreas Pancreatitis"

1

Research, Gratis. Gallstone Pancreatitis. Gratis Research, 2020. http://dx.doi.org/10.47496/gr.blog.08.

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