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Books on the topic 'Pancreatic necrosis'

Author: Grafiati

Published: 28 May 2022

Last updated: 31 July 2025

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1

Munro, A. L. S. Infectious pancreatic necrosis (IPN). Department of Agriculture and Fisheries for Scotland, Marine Laboratory, 1988.

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2

Zhou, Chengfeng. Detection of infectious pancreatic necrosis virus by polymerase chain reaction. National Library of Canada, 1992.

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3

Zhou, Chengfeng. Detection of infectious pancreatic necrosis virus by polymerase chain reaction. University of Prince Edward Island, 1992.

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4

Souter, B. W. Infectious pancreatic necrosis virus in Atlantic Salmon (Salmo salar), and Brook Trout (Salvelinus fontinalis), in Central Newfoundland. Central and Arctic Region, Dept. of Fisheries and Oceans, 1992.

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5

Canada. Dept. of Fisheries and Oceans. Western Region. Infectious pancreatic necrosis virus in adult Arctic charr, Salvelinus alpinus (L.), in rivers in the MacKenzie Delta region and Yukon Territory. Western Region, Dept. of Fisheries and Oceans, 1986.

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6

Keshav, Satish, and Alexandra Kent. Pancreatic disease. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0201.

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Acute pancreatitis is an acute inflammatory process of the pancreas and is potentially reversible. It is characterized by oedema and necrosis of peripancreatic fat and may progress to necrosis of glandular and surrounding tissue. Activation of pancreatic enzymes leads to pancreatic autodigestion and systemic effects.

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7

Kwon, Rachel J. Minimally Invasive Approach to Infected Pancreatic Necrosis. Edited by Danny Sherwinter. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199384075.003.0020.

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This chapter provides a summary of a landmark study in abdominal surgery. Does a minimally invasive, “step-up” approach to necrotizing pancreatitis reduce mortality and major complications as compared to open necrosectomy? Starting with that question, it describes the basics of the study, including funding, year study began, year study was published, study location, who was studied, who was excluded, how many patients, study design, study intervention, follow-up, endpoints, results, and criticism and limitations. The chapter briefly reviews other relevant studies and information, gives a summa

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8

Ireland, Northern. Infected Waters (Infectious Pancreatic Necrosis) Order (Northern Ireland) 2003. Stationery Office, The, 2003.

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9

Munro, A. L. S. Infectious Pancreatic Necrosis (IPN) (Aquaculture Information Series: 2/1988). The Stationery Office Books (Agencies), 1988.

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10

Ireland, Northern. Infected Waters (Infectious Pancreatic Necrosis) Revocation Order (Northern Ireland) 2003. Stationery Office, The, 2003.

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11

Ireland, Northern. Infected Waters (Infectious Pancreatic Necrosis) (Revocation) Order (Northern Ireland) 2006. Stationery Office, The, 2006.

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12

Ireland, Northern. Infected Waters (Infectious Pancreatic Necrosis) (No. 2) Order (Northern Ireland) 2003. Stationery Office, The, 2003.

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13

Wechsler, Sally Janet. Importance of infectious pancreatic necrosis virus in striped bass, Morone saxatilis. 1986.

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14

Maret, Joseph J. Virulence characteristics of infectious pancreatic necrosis virus (IPNV) in brook trout, Salvelinus fontinalis, fry. 1997.

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15

Manning, Donald Scott. Deletion mapping and expression of the large genomic segment of infectious pancreatic necrosis virus. 1988.

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16

Barrie, Rosemary Jean. Characterization of an immunoreactive region of the major capsid protein for infectious pancreatic necrosis virus. 1988.

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17

Fitz, Reginald Heber. Acute Pancreatitis: A Consideration of Pancreatic Hemorrhage, Hemorrhagic, Suppurative, and Gangrenous Pancreatitis, and of Disseminated Fat-Necrosis. Creative Media Partners, LLC, 2018.

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18

Huang, Manley T. F. Molecular cloning of infectious pancreatic necrosis virus and characterization of the coding capacity of the complementary DNA. 1985.

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19

Carter, R., and C. W. Imrie. Hepatobiliary surgery. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198510567.003.0008.

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Introduction 306Investigational procedures 308Pancreatic mobilisation 310Management of tumours 312Management of pancreatic necrosis 316Management of pseudocyst 318Management of chronic pancreatitis 320Common pancreatic complications 322As the morbidity and mortality associated with pancreatic surgery are amongst the highest in surgical practice, management of patients should take place within the context of multi-disciplinary team and they should be in optimal condition pre-operatively. They may be suffering from exocrine failure, altered glucose tolerance, or altered coagulation due to post-h

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20

Mason, Carla L. Molecular characterization of the proteinase and RNA-dependent RNA polymerase of Infectious Pancreatic Necrosis Virus, a fish birnavirus. 1992.

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21

Skipworth, James R. A., and Stephen P. Pereira. Pathophysiology, diagnosis, and assessment of acute pancreatitis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0190.

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The incidence of acute pancreatitis continues to increase, but the attendant mortality has not decreased for >30 years. The pathogenesis remains poorly understood, but the initial mechanism appears to be intracellular activation of pancreatic enzymes, with micro- and macrovascular dysfunction, in conjunction with a systemic inflammatory response acting as a key propagating factor and determinant of severity. A multitude of causes or initiators exist, but there is a common pathophysiological pathway. The use of conventional scoring systems, combined with repeated clinical and laboratory asse

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22

Johnson, Steven B. Pathophysiology and management of abdominal injury. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0334.

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Abdominal injuries are common following blunt and penetrating trauma. They can result in a spectrum of severity from benign to potentially life-threatening conditions. Soon after injury, haemorrhage is the predominant concern, and leading cause of morbidity and mortality. Active haemorrhage resulting in shock requires emergent operative intervention and aggressive haemostatic resuscitation. However haemodynamically-stable patients benefit from non-operative management of solid organ injuries with or without angiographic embolization. Sepsis usually occurs as a result of intra-abdominal infecti

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23

Rajendram, Rajkumar. Management of acute pancreatitis in the critically ill. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0191.

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The major causes of morbidity and mortality in acute pancreatitis are organ dysfunction and infection of necrotic tissue. Management should aim to prevent, or to diagnose and treat, the complications of pancreatic inflammation, and any predisposing factors to avoid recurrence. Medical management is essentially supportive with oxygen, intravenous fluids, analgesia, enteral or parenteral nutrition, and correction of metabolic abnormalities. Patients with severe acute pancreatitis are unlikely to resume prompt oral intake so nutritional support is also required. Post-pyloric feeding is not requir

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