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Journal articles on the topic 'Pancreatic necrosis'

Author: Grafiati

Published: 28 May 2022

Last updated: 31 July 2025

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1

Isenmann, Rainer, Markus BÜChler, Waldemar Uhl, Peter Malfertheiner, Markus Martini, and Hans G. Beger. "Pancreatic Necrosis." Pancreas 8, no. 3 (1993): 358–61. http://dx.doi.org/10.1097/00006676-199305000-00012.

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2

Branum, Gene, John Galloway, Wendy Hirchowitz, Morris Fendley, and John Hunter. "Pancreatic Necrosis." Annals of Surgery 227, no. 6 (1998): 870–77. http://dx.doi.org/10.1097/00000658-199806000-00010.

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3

Sano, Motohiko, and Nobuaki Okamoto. "Infectious Pancreatic Necrosis." Fish Pathology 52, no. 4 (2017): 177–80. http://dx.doi.org/10.3147/jsfp.52.177.

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4

BARON, TODD H., DESIREE E. MORGAN, SELWYN M. VICKERS, and AUDREY J. LAZENBY. "Organized Pancreatic Necrosis." Pancreas 19, no. 1 (1999): 105–8. http://dx.doi.org/10.1097/00006676-199907000-00016.

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5

Dionigi, Renzo, Francesca Rovera, Gianlorenzo Dionigi, Mario Diurni, and Salvatore Cuffari. "Infected Pancreatic Necrosis." Surgical Infections 7, supplement 2 (2006): s—49—s—52. http://dx.doi.org/10.1089/sur.2006.7.s2-49.

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6

Duininck, Troy M., Karen D. Libsch, Tatsuya Ueno, and Michael G. Sarr. "Infected Pancreatic Necrosis." Problems in General Surgery 19, no. 1 (2002): 72–80. http://dx.doi.org/10.1097/00013452-200203000-00011.

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7

Tudor, A., C. Molnar, C. Copotoiu, et al. "Pancreatico-Gastric Anastomosis with and without Sutures – Experimental Swine Model." Acta Medica Marisiensis 61, no. 2 (2015): 105–9. http://dx.doi.org/10.1515/amma-2015-0032.

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Abstract Objectives. The aim of our study is to identify a surgical technical that has the lowest rate of pancreatic fistulas in pancreatico-gastric anastomosis following duodenopancreatectomies. We studied pancreatico-gastric anastomosis performed with stitches compared to the ones performed without stitches. Methods. Our experimental model is based on ten piglets, which were divided into 2 groups. In the first group (n=5) the pancreatico-gastric anastomosis was done using double purse-string threads one passed through the gastric seromuscular layer and one through the gastric mucosa. In the

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8

Heo, Jun. "Infected Pancreatic Necrosis Mimicking Pancreatic Cancer." Case Reports in Gastroenterology 14, no. 2 (2020): 436–42. http://dx.doi.org/10.1159/000510161.

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Although infected pancreatic necrosis can develop as a result of rare conditions involving trauma, surgery, and systemic infection with an uncommon pathogen, it usually occurs as a complication of pancreatitis. Early phase of acute pancreatitis can be either edematous interstitial pancreatitis or necrotizing pancreatitis. The late complications of pancreatitis can be divided into pancreatic pseudocyst due to edematous interstitial pancreatitis or walled-off necrosis due to necrotizing pancreatitis. During any time course of pancreatitis, bacteremia can provoke infection inside or outside the p

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9

Chun, J. T., H. E. Akin, B. L. Enderson, and K. I. Maull. "PANCREATIC MARSUPIALIZATION FOR INFECTED PANCREATIC NECROSIS." Southern Medical Journal 83, Supplement (1990): 2S—76. http://dx.doi.org/10.1097/00007611-199009001-00303.

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10

Bittner, R., S. Block, M. B�chler, and H. G. Beger. "Pancreatic abscess and infected pancreatic necrosis." Digestive Diseases and Sciences 32, no. 10 (1987): 1082–87. http://dx.doi.org/10.1007/bf01300192.

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11

Revencu, Sergiu, Alexandru Gaitur, Ruslan Pretula, et al. "PANCREATIC NECROSIS IN TERMINAL DISEASES WITHOUT SURGICAL IMPACT." Arta Medica 91, no. 2 (2024): 32–37. https://doi.org/10.5281/zenodo.13328393.

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<strong>Objectives. </strong>Pancreatic necrosis are encountered incidentally during the anatomopathological examination in terminal non-surgical conditions. Elucidation of their frequency, morphological aspects, and background pathologies constituted the purpose of the given study. <strong>Methods.</strong> From anatomopathological investigation protocols, collected from Municipal Clinical Hospital ”Saint Arhanghel Mihail”, during 2014 – 2020, of 550 cases of death, caused by diseases, in case of patients who did not undergo surgery, were selected 198 (36%) cases with which

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12

Manish, Shah, and Mandal Manish. "To Study the Surgical and Interventional Management and its Outcome in Severe Acute Pancreatitis." International Journal of Pharmaceutical and Clinical Research 15, no. 8 (2023): 1616–19. https://doi.org/10.5281/zenodo.11528801.

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<strong>Background:</strong> An auto-digestion of the gland causes acute pancreas inflammation, which is known as acute pancreatitis. The severity of acute pancreatitis can range from a moderate, self-limited illness to a severe, quickly progressing condition. The developments of pancreatic or peri-pancreatic necrosis, together with general and local consequences that contribute to a high death rate, characterize severe acute pancreatitis in 20–25% of cases. Therapy of sequelae, most notably the therapy of infected walled off necrosis, is the most frequent reason for intervention in

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13

Siby, Jayas, Preetha S, and Sindhu R.S. "Proportion of Pancreatic Necrosis among Patients with Acute Pancreatitis and Association between High-Density Lipoprotein (HDL Cholesterol) and Pancreatic Necrosis – A Prospective Observational Study from Kerala, South India." Journal of Evidence Based Medicine and Healthcare 8, no. 20 (2021): 1489–94. http://dx.doi.org/10.18410/jebmh/2021/282.

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BACKGROUND Acute pancreatitis is a very common disease and severe acute pancreatitis is life threatening and needs early identification. Current severity predicting indices in acute pancreatitis are cumbersome. There have been previous reports of low HDL cholesterol in life threatening acute pancreatitis. Previous studies in this regard were done in a single ethnicity population and needed to be validated in other ethnic groups. We wanted to estimate the proportion of pancreatic necrosis among the patients with acute pancreatitis attending Government Medical College, Thiruvananthapuram. We als

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14

Agarwal, Smita, Jennell E. Nelson, Seth R. Stevens, and Anita C. Gilliam. "An Unusual Case of Cutaneous Pancreatic Fat Necrosis." Journal of Cutaneous Medicine and Surgery 6, no. 1 (2002): 16–18. http://dx.doi.org/10.1177/120347540200600103.

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Background: Cutaneous pancreatic fat necrosis is a pathognomonic sign for pancreatic disease and usually presents as subcutaneous nodules in the pretibial region. Objective: A case of cutaneous pancreatic fat necrosis is presented in which the clinical presentation of diffuse erythema was unusual. This disease is discussed and its possible etiologies are reviewed. Methods: A MEDLINE search for cases of cutaneous pancreatic fat necrosis presenting as diffuse erythema without nodules was conducted. Results: Diffuse erythema is an unusual presentation of cutaneous pancreatic fat necrosis. Conclus

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15

Stamatakos, Michael. "Walled-off pancreatic necrosis." World Journal of Gastroenterology 16, no. 14 (2010): 1707. http://dx.doi.org/10.3748/wjg.v16.i14.1707.

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16

Thomson, John-Edwin, Sven M. van Dijk, Martin Brand, Hjalmar C. van Santvoort, and Marc G. Besselink. "Managing Infected Pancreatic Necrosis." Chirurgia 113, no. 3 (2018): 291. http://dx.doi.org/10.21614/chirurgia.113.3.291.

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17

Gardner, Timothy B., Christopher A. Olenec, Jocelyn D. Chertoff, Todd A. Mackenzie, and Douglas J. Robertson. "Hemoconcentration and Pancreatic Necrosis." Pancreas 33, no. 2 (2006): 169–73. http://dx.doi.org/10.1097/01.mpa.0000226885.32957.17.

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18

Wu, B. U., V. K. Singh, R. Maurer, D. L. Conwell, and P. A. Banks. "HEMOCONCENTRATION AND PANCREATIC NECROSIS." Pancreas 37, no. 4 (2008): 501–2. http://dx.doi.org/10.1097/01.mpa.0000335415.53906.0f.

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19

Gloor, Beat, Waldemar Uhl, and Markus W. Büchler. "Debridement of pancreatic necrosis." Operative Techniques in General Surgery 4, no. 2 (2002): 116–23. http://dx.doi.org/10.1053/otgn.2002.33143.

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20

Gardner, Timothy B., Christopher A. Olenec, Todd A. Mackenzie, and Douglas J. Robertson. "HEMOCONCENTRATION AND PANCREATIC NECROSIS." American Journal of Gastroenterology 99 (October 2004): S48—S49. http://dx.doi.org/10.14309/00000434-200410001-00146.

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21

White, E. M., J. Wittenberg, P. R. Mueller, et al. "Pancreatic necrosis: CT manifestations." Radiology 158, no. 2 (1986): 343–46. http://dx.doi.org/10.1148/radiology.158.2.3510442.

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22

Aldridge, M. C. "Diagnosis of pancreatic necrosis." British Journal of Surgery 75, no. 2 (1988): 99–100. http://dx.doi.org/10.1002/bjs.1800750203.

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23

Litvin, A., S. Rumovskaya, S. Listopad, F. Paramzin, A. Kropinov, and A. Kapustina. "Differentiating infected pancreatic necrosis from sterile pancreatic necrosis with CT radiomics features." Pancreatology 21 (July 2021): S31. http://dx.doi.org/10.1016/j.pan.2021.05.083.

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24

Pal, K. M., Pashtoon Murtaza Kasi, Mohammad Tayyeb, S. M. Faisal Mosharraf, and Zafar Fatmi. "Correlates of Morbidity and Mortality in Severe Necrotizing Pancreatitis." ISRN Surgery 2012 (July 9, 2012): 1–5. http://dx.doi.org/10.5402/2012/215193.

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Acute severe pancreatitis is associated with a high morbidity and mortality and frequently is accompanied by underlying pancreatic parenchymal necrosis. Patients with pancreatic necrosis must be identified, because the morbidity and mortality rate in this subgroup is much higher. Our objective was to compare the clinical outcomes of these patients based on the degree of pancreatic necrosis. A total of 35 patients were noted to have pancreatic necrosis. These were divided into 2 groups based on extent of necrosis: group A had less than 50% necrosis and group B had more than 50% necrosis. The ra

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25

Miwa, Toshiki, Hajime Tanaka, and Toshiaki Shiojiri. "Infected pancreatic necrosis and retroperitoneal abscess associated with Lactobacillus paracasei." BMJ Case Reports 14, no. 8 (2021): e243936. http://dx.doi.org/10.1136/bcr-2021-243936.

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Infected pancreatic necrosis is a postpancreatitis complication that is mainly caused by Enterobacteriaceae and Enterococci. Here, we have reported a very rare case of Lactobacillus paracasei bacteraemia associated with infected pancreatic necrosis and retroperitoneal abscess. In addition to the diagnosis of diabetic ketoacidosis, blood test results revealed a high inflammatory status. CT of the abdomen revealed pancreatic walled-off necrosis. Blood culture and aspiration fluid culture revealed positivity for L. paracasei, leading to the diagnosis of infected pancreatic necrosis. The abscess h

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26

Darvin, V. V., S. V. Onishchenko, E. V. Loginov, and A. A. Kabanov. "Severe acute pancreatitis: risk factors of adverse outcomes and their correction." Annaly khirurgicheskoy gepatologii = Annals of HPB surgery 23, no. 2 (2018): 76–83. http://dx.doi.org/10.16931/1995-5464.2018276-83.

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Aim. To analyze management of severe acute destructive pancreatitis and to determine the main tactical, anatomical and pathophysiological factors determining risks of adverse outcomes. Material and methods. 3581 patients with acute pancreatitis were enrolled. Retrospectively, 239 patients were assigned to severe pancreatic necrosis; invasive surgical techniques were applied in 210 cases. Twenty-nine patients with pancreatic necrosis underwent endoscopic papillosphincterotomy with main pancreatic duct stenting. Results. Overall mortality in patients with severe pancreatic necrosis was 32.3%, in

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27

Akayzin, E. S., A. S. Metelev, and A. E. Akayzina. "INDICATORS OF VOLATILE FATTY ACIDS IN DIFFERENTIAL DIAGNOSTICS OF INFECTED AND STERILE PANCREONECROSIS." Russian Clinical Laboratory Diagnostics 64, no. 11 (2019): 644–48. http://dx.doi.org/10.18821/0869-2084-2019-64-11-644-648.

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The aim of the study was to assess the information content of volatile fatty acid parameters for the differential diagnosis of infected and sterile pancreatic necrosis. The work is based on the results of examination and treatment of 34 patients with pancreatic necrosis. The analysis of concentrations of volatile fatty acids: acetic, propionic, butyric and isovaleric was carried out on an automated gas chromatograph «Crystallux-4000» with a capillary column «HP-FFAP» and flame ionization detector. The indicators of acetic, propionic, butyric, isovaleric acid and the sum of volatile fatty acids

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28

Baron, Todd H. "Treatment of Pancreatic Pseudocysts, Pancreatic Necrosis, and Pancreatic Duct Leaks." Gastrointestinal Endoscopy Clinics of North America 17, no. 3 (2007): 559–79. http://dx.doi.org/10.1016/j.giec.2007.05.013.

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29

Frost, Fabian, Laura Schlesinger, Mats L. Wiese, et al. "Infection of (Peri-)Pancreatic Necrosis Is Associated with Increased Rates of Adverse Events during Endoscopic Drainage: A Retrospective Study." Journal of Clinical Medicine 11, no. 19 (2022): 5851. http://dx.doi.org/10.3390/jcm11195851.

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Pancreatic necroses are a major challenge in the treatment of patients with pancreatitis, causing high morbidity. When indicated, these lesions are usually drained endoscopically using plastic or metal stents. However, data on factors associated with the occurrence of failure or adverse events during stent therapy are scarce. We retrospectively analyzed all adverse events and their associated features which occurred in patients who underwent a first-time endoscopic drainage of pancreatic necrosis from 2009 to 2019. During the observation period, a total of 89 eligible cases were identified. Ad

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30

Ammori, B. J. "Laparoscopic transgastric pancreatic necrosectomy for infected pancreatic necrosis." Surgical Endoscopy -1, no. 1 (2003): 1. http://dx.doi.org/10.1007/s00464-002-4202-9.

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31

Логинов, Е. В., and В. В. Дарвин. "PANCREATIC DUCT STENTING IN PATIENTS WITH PANCREATIC NECROSIS." Vestnik SurGU. Meditsina 17, no. 1 (2024): 26–33. http://dx.doi.org/10.35266/2949-3447-2024-1-3.

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The article reviews Russian and foreign literature on the treatment of patients with acute pancreatitis according to the relevance, as well as increase in disease frequency, high mortality rate, and persistent disability of patients with severe pancreatitis. Pancreatic duct stenting is one of the most promising treatments for pancreatic necrosis, and it can help improve the treatment of such patients with a severe disease.

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32

Dronov, O. I., I. O. Kovalska, T. Yu Ivanets, Ye S. Kozachuk, L. V. Levchenko, and U. V. Stasiuk. "The role of endogenous insulin in patients with acute pancreatitis." INTERNATIONAL JOURNAL OF ENDOCRINOLOGY (Ukraine) 20, no. 8 (2025): 607–12. https://doi.org/10.22141/2224-0721.20.8.2024.1468.

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Background. Acute pancreatitis is an inflammatory disease of the exocrine part of the pancreas, accompanied by various complications and further development of necrosis of the gland and parapancreatic tissue with possible secondary infection. The study of experimental models shows that the acinar cells surrounding the islets of Langerhans remain relatively intact, indicating that endogenous insulin is a protective factor for the pancreatic parenchyma. The purpose of the study was to determine the dependence of endogenous insulin levels on the extent of pancreatic parenchymal damage. Materials

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33

Shah, Devank, Rupesh Pokharna, Sudhir Maharshi, Kamleshkumar Sharma, and Prerak Maniar. "Pancreatitis, Polyarthritis and Panniculitis (PPP) Syndrome - A Rare Presentation." ACG Case Reports Journal 12, no. 2 (2025): e01601. https://doi.org/10.14309/crj.0000000000001601.

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ABSTRACT Pancreatitis, panniculitis, and polyarthritis (PPP) syndrome is a rare manifestation (prevalence 2%–3%) of underlying pancreatic pathology which is characterized by fat necrosis in subcutaneous tissue and joints. The widely accepted etiological hypothesis is systemic dissemination of pancreatic lipase. It has a mortality rate approaching 25%. We present a case of pancreatitis, panniculitis, and polyarthritis syndrome in a 52-year-old man with chronic pancreatitis complicated by pancreatico-pericardial fistula.

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34

Mauda Shadi, L. A., A. A. Litvin, O. G. Jarikov, and G. A. Senchuk. "USE OF THE PROCALCITONIN TEST FOR DIAGNOSTICS OF INFECTED PANCREATIC NECROSIS." Health and Ecology Issues, no. 4 (December 28, 2007): 30–34. http://dx.doi.org/10.51523/2708-6011.2007-4-4-6.

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The significance of plasma procalcitonin concentration for the differential of sterile and infected pancreatic necrosis was evaluated. Twenty-five patients with acute severe pancreatitis were examined. Plasma procalcitonin concentrations were notably increased in patients with infected pancreatic necrosis. These data indicate that measurement of procalcitonin concentration is the optimal laboratory test for the differential diagnosis of sterile and infected pancreatic necrosis.

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35

Boyko, V. V., V. M. Lykhman, A. O. Merkulov, et al. "DIFFERENTIAL DIAGNOSIS OF STERILE AND I NFECTED PANCREONECROSIS." Kharkiv Surgical School, no. 1 (March 20, 2021): 31–35. http://dx.doi.org/10.37699/2308-7005.1.2021.06.

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Summary. Introduction. The search for objective methods for diagnosing the form and severity of acute pancreatitis, accurate prediction and timely prevention of infectious complications in this surgical pathology is of great practical importance for modern medicine. Materials and methods. According to clinical, morphological and bacteriological data, groups of patients were selected. The first group included 33 patients with sterile pancreatic necrosis (SP), the second group consisted of 29 patients with infected pancreatic necrosis (IP). Results and discussion. The program of diff

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36

CHIKAEV, VYACHESLAV F. "MINIMALLY INVASIVE TECHNIQUES TO TREAT THE COMPLICATIONS OF ACUTE PANCREATITIS." Bulletin of Contemporary Clinical Medicine 16, suppl.1 (2023): 97–100. http://dx.doi.org/10.20969/vskm.2023.16(suppl.1).97-100.

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Abstract. Introduction. Hollow organ perforations accompanying pancreatic necrosis are a severe complication. In this case, surgical intervention is limited to a degree due to the extensive process in the upper abdomen and the patient’s functional condition. Aim. To share our experience in the minimally invasive repair of gastric perforation in a case series of severely infected pancreatic necroses and retroperitoneal phlegmons. Materials and Methods. We analyzed a case series in our clinic over 5 years, including the necrotizing forms of acute pancreatitis with complications, such as acute pe

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37

Boyko, V. V., А. O. Merkulov, О. M. Shevchenko, et al. "APPLICATION OF MINIMALLY INVASIVE ENDOSCOPIC INTERVENTIONS IN PATIENTS WITH PANCREANECROSIS." Kharkiv Surgical School, no. 4-5 (September 30, 2023): 14–18. http://dx.doi.org/10.37699/2308-7005.4-5.2023.02.

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Summary. Improvements in the methods of diagnosis and pathogenetic complex treatment of pancreatic necrosis in recent years have made it possible to significantly improve the results of treatment, while the issues of diagnosis, indications and treatment tactics of severe, common forms of pancreatic necrosis and its post-necrotic complications still remain far from a final solution . 30 patients with pancreatic necrosis were observed. All patients were comparable in terms of age, sex, and etiology. Sixteen patients of the main study group (53 %) underwent various endoscopic interventions on the

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38

Hamir, A. N., P. L. Habecker, and C. Tillman. "Pancreatic necrosis in a llama." Veterinary Record 142, no. 23 (1998): 644–45. http://dx.doi.org/10.1136/vr.142.23.644.

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39

Mowery, Nathan T., Brandon R. Bruns, Heather G. MacNew, et al. "Surgical management of pancreatic necrosis." Journal of Trauma and Acute Care Surgery 83, no. 2 (2017): 316–27. http://dx.doi.org/10.1097/ta.0000000000001510.

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40

Abu Dayyeh, Barham K., and Mark Topazian. "Endoscopic management of pancreatic necrosis." American Journal of Gastroenterology 113, no. 9 (2018): 1269–73. http://dx.doi.org/10.1038/s41395-018-0060-5.

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41

Beger, Hans G., Reinhard Bittner, Silvia Block, and Markus Büchler. "Bacterial contamination of pancreatic necrosis." Gastroenterology 91, no. 2 (1986): 433–38. http://dx.doi.org/10.1016/0016-5085(86)90579-2.

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42

Beat, Gloor, Schmidt Oliver, Uhl Waldemar, and Markus W. Büchler. "Prophylactic antibiotics and pancreatic necrosis." Current Gastroenterology Reports 3, no. 2 (2001): 109–14. http://dx.doi.org/10.1007/s11894-001-0006-7.

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43

Larvin, Mike. "Management of infected pancreatic necrosis." Current Gastroenterology Reports 10, no. 2 (2008): 107–14. http://dx.doi.org/10.1007/s11894-008-0030-y.

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44

Chaudhary, Adarsh, Ajay Sachdev, and Sanjay Negi. "Biliary Complications of Pancreatic Necrosis." International Journal of Gastrointestinal Cancer 29, no. 3 (2001): 129–32. http://dx.doi.org/10.1385/ijgc:29:3:129.

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45

Fotoohi, Mehran, and L. William Traverso. "Management of severe pancreatic necrosis." Current Treatment Options in Gastroenterology 10, no. 5 (2007): 341–46. http://dx.doi.org/10.1007/s11938-007-0034-y.

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46

Tirnanic, Tanja, Tatjana Radevic, Andrea Djordjevic, Nenad Petrov, and Zeljko Mijuskovic. "Pancreatic panniculitis associated with periampullary duodenal diverticulum: A case report." Vojnosanitetski pregled, no. 00 (2023): 43. http://dx.doi.org/10.2298/vsp230626043t.

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Introduction. Pancreatic panniculitis is a rare type of lobular panniculitis that manifests as painful erythematous nodules on the skin of the lower extremities. Subcutaneous fat necrosis caused by the release of pancreatic enzymes is the underlying cause of the disease, affecting around 2 to 3% of patients with pancreatic diseases. Case report. We present a case of a 58-year-old male patient who exhibited painful erythematous nodules on the lower extremities and trunk. Laboratory results revealed increased levels of pancreatic enzymes, amylase, and lipase, as well as heightened levels of gluc

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47

Stepchenkov, Roman Petrovich. "The principles of providing emergency care for patients with pancreatic necrosis." Spravočnik vrača obŝej praktiki (Journal of Family Medicine), no. 7 (July 1, 2021): 59–64. http://dx.doi.org/10.33920/med-10-2107-08.

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At the present stage, the treatment of patients with pancreatic necrosis is one of the most urgent surgical tasks. Pancreatic necrosis is a destructive lesion of the pancreas, often leading to the development of multiple organ failure. The pathogenesis is based on a violation of the mechanism of counteraction to proteolytic enzymes produced by the pancreas. Pancreatic necrosis is a formidable complication of pancreatitis, with mortality ranging from 30 to 70 %. Today, in terms of the frequency of occurrence in surgical practice, this pathology ranks second after acute appendicitis. The leading

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48

Rana, Surinder Singh, and Rajesh Gupta. "Nomenclature of Pancreatic Fluid Collections following Acute Pancreatitis: Need to Further Revise the Atlanta Classification System!" Journal of Digestive Endoscopy 13, no. 04 (2022): 257–59. http://dx.doi.org/10.1055/s-0042-1759512.

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AbstractA better understanding of the disease pathophysiology, improved imaging modalities, and the development of minimally invasive interventions led to the revision of the Atlanta classification and new terminologies for the pancreatic fluid collections (PFCs) occurring in acute pancreatitis. Peripancreatic necrosis (PPN) or extra-pancreatic necrosis (EPN) has been reported as a distinct morphological entity with a better outcome than combined pancreatic and peripancreatic necrosis as well as pancreatic parenchymal necrosis alone and slightly worse than acute interstitial pancreatitis. In t

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49

Lankisch, Paul G., Karl Struckmann, and Dirk Lehnick. "Presence and extent of extrapancreatic fluid collections are indicators of severe acute pancreatitis." International journal of pancreatology : official journal of the International Association of Pancreatology 26, no. 3 (1999): 131. https://doi.org/10.5281/zenodo.4555015.

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BACKGROUND It has been suggested that early localization of both necrosis and extrapancreatic fluid collections by contrast-enhanced computed tomography (CT) can predict the outcome in severe acute pancreatitis. These two assumptions were evaluated. PATIENTS AND METHODS This study comprises 228 patients with a first attack of acute pancreatitis admitted to our clinic from 1987 to 1995 and for whom the prognostic value of a contrast-enhanced CT obtained within 72 h of admission was prospectively evaluated. These CTs were retrospectively re-evaluated for the localization of pancreatic necrosis a

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50

Dr., Sheyon Yohannan; Dr. Dhruv Kumar Sharma. "Can Organ Failure and Infected Pancreatic Necrosis be Used to Forecast Mortality in Patients with Gallstone-Induced Pancreatitis?" International Journal of Innovative Science and Research Technology 8, no. 2 (2023): 849–53. https://doi.org/10.5281/zenodo.7677954.

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Early identification of high-risk patientsin a common emergency like acute gallstone-induced pancreatitis can be difficult. In patients with acute gallstone-induced pancreatitis, persistent organ failure is considered to be the most important cause of mortality. This study strives to understand the relationship between persistent organ failure with infected pancreatic necrosis and its relationship with mortality.  Design We performed a prospective observational study of 219 patients diagnosed with acute gallstone-induced pancreatitis who were admitted to the Department of General Surgery, IGM

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