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Journal articles on the topic 'Pancreatic steatosis'

Author: Grafiati

Published: 2 June 2025

Last updated: 31 July 2025

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1

Ramkissoon, Resham, and Timothy B. Gardner. "Pancreatic steatosis." Current Opinion in Gastroenterology 35, no. 5 (2019): 440–47. http://dx.doi.org/10.1097/mog.0000000000000566.

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Ramkissoon, Resham, and Timothy B. Gardner. "Pancreatic Steatosis." American Journal of Gastroenterology 114, no. 11 (2019): 1726–34. http://dx.doi.org/10.14309/ajg.0000000000000262.

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PAUL, Jayanta, and Ambalathu Veettil Hussain SHIHAZ. "PANCREATIC STEATOSIS: A NEW DIAGNOSIS AND THERAPEUTIC CHALLENGE IN GASTROENTEROLOGY." Arquivos de Gastroenterologia 57, no. 2 (2020): 216–20. http://dx.doi.org/10.1590/s0004-2803.202000000-27.

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ABSTRACT Fat infiltration in the pancreas is called pancreatic steatosis and it has several synonyms such as pancreatic lipomatosis, non-alcoholic fatty pancreatic disease, lipomatous pseudohypertrophy, fatty replacement, fatty pancreas and fatty infiltration. Pancreatic steatosis describes a disease ranging from infiltration of fat in the pancreas to pancreatic inflammation, and development of pancreatic fibrosis. There are multiple aetiologies of this condition, such as metabolic syndrome, alcohol intake, viral infections, toxins, congenital syndromes, etc. Pancreatic steatosis is usually di

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4

Chan, Chia-Hao, Chia-Chen Chang, and Yen-Chun Peng. "The Clinical Significance of Pancreatic Steatosis in Pancreatic Cancer: A Hospital-Based Study." Diagnostics 14, no. 19 (2024): 2128. http://dx.doi.org/10.3390/diagnostics14192128.

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Background/Objectives: Pancreatic cancer remains one of the deadliest malignancies worldwide with a pressing need for early detection and intervention strategies. Emerging evidence has suggested a potential link between pancreas steatosis, characterized by excessive pancreatic fat accumulation, and an increased risk of pancreatic cancer development. This retrospective imaging study aims to elucidate the association between pancreatic steatosis and the subsequent development of pancreatic cancer. In the study, we aimed to determine the characteristics of pancreatic cancer with pancreatic steato

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Mathur, A., N. J. Zyromski, T. E. Wade, S. Wang, D. A. Swartz-Basile, and H. A. Pitt. "EZETIMIBE AMELIORATES PANCREATIC STEATOSIS." Pancreas 37, no. 4 (2008): 484. http://dx.doi.org/10.1097/01.mpa.0000335336.97589.39.

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Altinmakas, Emre, Begum Guler, Sidar Copur, et al. "Determinants of Pancreatic Steatosis: A Retrospective Observational Study." Middle East Journal of Digestive Diseases 13, no. 4 (2021): 343–49. http://dx.doi.org/10.34172/mejdd.2021.245.

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BACKGROUND Metabolic syndrome affects 35% of the adult population in developed countries associated with non-alcoholic steatohepatitis, insulin resistance, and cardiovascular events. Fatty infiltration of the pancreas, or pancreatic steatosis, is a risk factor for acute pancreatitis, pancreatic malignancies, and diabetes mellitus, yet its relationship with metabolic syndrome is not well defined. METHODS We performed a single-centered retrospective observational study of 322 healthy subjects (subjects volunteering to be kidney transplant donors, mean age=46.3±13.5, 163 men and 159 women) in the

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Starodubova, A. V., S. D. Kosyura, E. N. Livantsova, Yu R. Varaeva, and A. A. Krasilova. "Diagnosing pancreatic steatosis in obese patients." Herald of Pancreatic Club 45, no. 4 (2019): 30–33. http://dx.doi.org/10.33149/vkp.2019.04.03.

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Frequency of pancreatic steatosis in adults of general population is up to 35%. Causes of triglyceride accumulation in the pancreas include aging, obesity, type 2 diabetes mellitus, metabolic syndrome, hypertriglyceridemia (genetically determined or secondary one). Nomenclature by M. M. Smits is used for diagnosing, which sets out pancreatic steatosis, lipomatous pseudohypertrophy of the pancreas, fatty replacement of the pancreas, fatty infiltration of the pancreas, non-alcoholic fatty pancreatic disease, non-alcoholic pancreatic steatosis, non-alcoholic steatopancreatitis. Pancreatic s

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Gubergrits, N. B., N. V. Byelyayeva, G. M. Lukashevich, et al. "News of European Pancreatology (based on the materials of the 52nd meeting of the European Pancreatic Club)." Herald of Pancreatic Club 49, no. 4 (2020): 6–20. http://dx.doi.org/10.33149/vkp.2020.04.01.

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The 52nd meeting of the European Pancreatic Club was organized and conducted online in Paris on July 2–3, 2020. It is the first time the Congress took place online, with about a thousand of pancreatologists having participated in it. The high scientific, organizational and technical level of the congress should be noted. This review presents the most interesting research results in the following sections: anatomy of the pancreas, acute early chronic, autoimmune pancreatitis, cancer and other malignant pancreatic diseases, functional pancreatic insufficiency, enzyme replacement therapy, p

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Marin-Serrano, Eva, Noemi González Pérez De Villar, Ana Barbado Cano, Rubén Fernández-Martos, Jose Maria Mostaza Prieto, and Maria Dolores Martín-Arranz. "Pancreatic steatosis and cardiovascular risk." Pancreatology 24 (December 2024): e173-e174. https://doi.org/10.1016/j.pan.2024.05.464.

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Melitas, Constantine, and Mick Meiselman. "Metabolic Pancreatitis: Pancreatic Steatosis, Hypertriglyceridemia, and Associated Chronic Pancreatitis in 3 Patients with Metabolic Syndrome." Case Reports in Gastroenterology 12, no. 2 (2018): 331–36. http://dx.doi.org/10.1159/000490042.

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Obesity, insulin resistance, and metabolic syndrome continue to increase in prevalence. Hypertriglyceridemia is commonly associated and represents a valuable marker of metabolic syndrome. An increase in subcutaneous fat deposition places patients at risk for visceral adipose deposition in sites such as the liver, heart, and pancreas. Pancreatic steatosis in the setting of metabolic syndrome is a rapidly emerging entity whose clinical spectrum remains to be defined. Hypertriglyceridemia is an accepted cause of acute pancreatitis but its role in chronic pancreatic injury remains to be explored.

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11

Mathur, Abhishek, Nicholas J. Zyromski, Henry A. Pitt, et al. "Pancreatic Steatosis Promotes Dissemination and Lethality of Pancreatic Cancer." Journal of the American College of Surgeons 208, no. 5 (2009): 989–94. http://dx.doi.org/10.1016/j.jamcollsurg.2008.12.026.

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12

Kromrey, Marie-Luise, Nele Friedrich, Ralf-Thorsten Hoffmann, et al. "Pancreatic Steatosis Is Associated With Impaired Exocrine Pancreatic Function." Investigative Radiology 54, no. 7 (2019): 403–8. http://dx.doi.org/10.1097/rli.0000000000000554.

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13

Pivtorak, K., I. Fedzhaga, and N. Pivtorak. "NAFLD, pancreatic steatosis and atherosclerotic process." Atherosclerosis 355 (August 2022): 199–200. http://dx.doi.org/10.1016/j.atherosclerosis.2022.06.802.

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14

Dholakia, S., E. J. Sharples, R. J. Ploeg, and P. J. Friend. "Significance of steatosis in pancreatic transplantation." Transplantation Reviews 31, no. 4 (2017): 225–31. http://dx.doi.org/10.1016/j.trre.2017.08.001.

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15

Yang, Wenjuan, Jinhang Gao, Yang Tai, et al. "Betaine Attenuates Alcohol-Induced Pancreatic Steatosis." Pancreas 45, no. 6 (2016): 836–45. http://dx.doi.org/10.1097/mpa.0000000000000557.

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16

Dağdeviren, Murat, Mustafa Altay, and Emra Nalbant. "Pancreatic steatosis: diagnosis and clinical significance." Journal of Contemporary Medicine 7 (February 6, 2017): 1. http://dx.doi.org/10.16899/gopctd.290190.

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17

Smits, Mark M., and Erwin J. M. van Geenen. "The clinical significance of pancreatic steatosis." Nature Reviews Gastroenterology & Hepatology 8, no. 3 (2011): 169–77. http://dx.doi.org/10.1038/nrgastro.2011.4.

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18

Truong, Emily, Stephen Pandol, and Christie Jeon. "Uniting Epidemiology and Experimental Models: Pancreatic Steatosis and Pancreatic Cancer." eBioMedicine 79 (May 2022): 103996. http://dx.doi.org/10.1016/j.ebiom.2022.103996.

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19

Gubergrits, N. B., N. V. Byelyayeva, T. L. Mozhyna, N. E. Monogarova, K. N. Borodiy, and A. V. Yuryeva. "Pancreatic steatosis: a giant step forward or recognition of ignorance." Herald of Pancreatic Club 49, no. 4 (2020): 21–41. http://dx.doi.org/10.33149/vkp.2020.04.02.

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This review analyzes the terminology of pancreatic steatosis, the data about the prevalence of non-alcoholic fatty pancreas disease (NAFPD) were given. The etiological factors of NAFPD are usually subdivided into hereditary, metabolic, and toxic ones. The main etiopathogenetic factor of pancreatic fatty infiltration is obesity: it causes pancreatic infiltration by adipocytes, leading to the development of NAFPD. The pathogenesis of the disease is associated with adipocytic tissue dysfunction, which induces local and systemic inflammatory response with corresponding clinical consequences.

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20

Byelyayeva, N., N. Gubergrits, and G. Lukashevich. "Frequency of combined hepatic and pancreatic steatosis." Pancreatology 21 (July 2021): S110. http://dx.doi.org/10.1016/j.pan.2021.05.294.

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21

Orellana, Fernando, Fernando Torres, Rolando Cocio, et al. "Pancreatic steatosis: A frequently ignored radiological finding." Pancreatology 17, no. 4 (2017): S30. http://dx.doi.org/10.1016/j.pan.2017.07.102.

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22

Фоминых, Юлия Александровна, Юрий Павлович Успенский, Ирина Анатольевна Горбачева, and Ольга Александровна Кизимова. "Steatosis of pancreas as cluster of the metabolic syndrome." University therapeutic journal, no. 2 (May 11, 2022): 31–41. http://dx.doi.org/10.56871/3032.2022.22.42.004.

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В данной статье представлен анализ отечественной и зарубежной литературы, посвященный вопросу неалкогольной жировой болезни поджелудочной железы в детской и взрослой популяции. В настоящее время центральное ожирение становится серьезной проблемой общественного здравоохранения среди всех возрастных групп, поэтому особое внимание все больше сосредоточено на патологии поджелудочной железы. Имеющиеся данные указывают на то, что неалкогольная жировая болезнь поджелудочной железы в значительной степени связана с повышенным риском развития метаболического синдрома и его компонентов. В большинстве слу

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23

Cortés, Pedro, Alfred Nelson, Fernando Stancampiano, et al. "Tu1476 PANCREATIC STEATOSIS AND POST-OPERATIVE PANCREATIC FISTULA IN PATIENTS WITH MAJOR PANCREATIC SURGERY." Gastroenterology 166, no. 5 (2024): S—1908. http://dx.doi.org/10.1016/s0016-5085(24)04880-7.

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24

Dite, Petr, Martin Blaho, Martina Bojkova, Petr Jabandziev, and Lumir Kunovsky. "Nonalcoholic Fatty Pancreas Disease: Clinical Consequences." Digestive Diseases 38, Suppl. 2 (2019): 143–49. http://dx.doi.org/10.1159/000505366.

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Metabolic syndrome and its components such as obesity, hypertriglyceridemia, type-2 diabetes mellitus (DM-T2), and arterial hypertension are unequivocally serious problems for every society. This is especially true in economically developed countries where the imbalance in lifestyle between caloric intake and caloric output still gets greater and greater. This fact is not only a concern for the adult population but for children as well. However, metabolic syndrome does not only affect society and health in regards to cardiovascular diseases, it significantly concerns gastroenterology where it

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25

Freeman, Hugh J. "Hepatobiliary Tract and Pancreatic Disorders in Celiac Disease." Canadian Journal of Gastroenterology 11, no. 1 (1997): 77–81. http://dx.doi.org/10.1155/1997/160201.

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A number of hepatobiliary tract and pancreatic disorders have been documented in patients with celiac disease. Some disorders have shared immunological or genetic factors, including chronic hepatitis, primary biliary cirrhosis and sclerosing cholangitis. Other hepatic or pancreatic pathological changes in celiac disease have been documented with severe malnutrition and malabsorption, including hepatic steatosis and pancreatic insufficiency, sometimes with pancreatic calcification. Finally, celiac disease may be associated with other very rare hepatic complications, such as hepatic T cell lymph

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26

Kharchenko, N. V., H. V. Beketova, and V. P. Shipulin. "Reviews of the monograph “Pancreatic steatosis in children”." GASTROENTEROLOGY 54, no. 2 (2020): 124–25. http://dx.doi.org/10.22141/2308-2097.54.2.2020.206231.

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27

Tahtaci, Mustafa, Oktay Algin, Tarkan Karakan, et al. "Can pancreatic steatosis affect exocrine functions of pancreas?" Turkish Journal of Gastroenterology 29, no. 5 (2018): 588–94. http://dx.doi.org/10.5152/tjg.2018.17696.

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28

Bi, Ye, Hai-Yan Lin, Ming-Long Li, Jie Zhou, and Xiang-Lan Sun. "The Association Between Pancreatic Steatosis and Metabolic Syndrome." Pancreas 51, no. 8 (2022): 1000–1006. http://dx.doi.org/10.1097/mpa.0000000000002138.

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29

Costa, Nívia Rézio, Eurípides Barsanulfo Borges dos Reis, and Kelly Cristina Ferreira de Moraes Paes. "PANCREATIC STEATOSIS: A NEW AND CHALLENGING CLINICAL ENTITY." Journal of Coloproctology 44, S 01 (2024): S1—S138. https://doi.org/10.1055/s-0045-1808811.

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30

Tanaeva, T. A., and L. A. Ivanov. "Correlation between type 2 diabetes mellitus and pancreatic steatosis." Perm Medical Journal 40, no. 2 (2023): 5–10. http://dx.doi.org/10.17816/pmj4025-10.

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Objective. To study the features of endocrine function in non-alcoholic fatty disease of the pancreas in patients with chronic pancreatitis in remission. Materials and methods. There were examined 62 patients with chronic pancreatitis without exacerbation, who were admitted to the day hospital department of the City Clinical Hospital № 9 (Izhevsk). The data of laboratory and instrumental research methods, the features of the pancreatic endocrine function in patients with chronic pancreatitis, accompanied by fatty degeneration of the organ were analyzed. Results. Pancreatic steatosi

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31

Chen, Xu, Jinghe Xiao, Juan Pang, Shen Chen, Qing Wang та Wenhua Ling. "Pancreatic β-Cell Dysfunction Is Associated with Nonalcoholic Fatty Liver Disease". Nutrients 13, № 9 (2021): 3139. http://dx.doi.org/10.3390/nu13093139.

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Background: Nonalcoholic fatty liver disease (NAFLD) is associated with decreased insulin sensitivity. However, the association between NAFLD and pancreatic β-cell function is still ambiguous. Here, we assessed whether pancreatic β-cell function is associated with NAFLD. Method: The data of NHANES III from 1988 to 1994 were used. NAFLD was diagnosed when subjects had ultrasonographically hepatic steatosis without other liver diseases. Disposition index (DI) was employed to assess pancreatic β-cell function. A total of 6168 participants were included in this study. Results: NAFLD participants h

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32

Cherrie Fung, Hay Ching, Juan Pablo Meneses, Nirusha Surendran, et al. "Assessing Pancreatic Fat and Its Correlation with Liver Fat in Suspected MASLD Patients Using Advanced Deep Learning Techniques from MRI Images." Applied Sciences 14, no. 24 (2024): 11924. https://doi.org/10.3390/app142411924.

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Pancreatic steatosis and metabolic-dysfunction-associated steatotic liver disease are characterised by fat accumulation in abdominal organs, but their correlation remains inconclusive. Recently proposed deep learning (DL) for proton density fat fraction (PDFF) estimation, which quantifies organ fat, has primarily been assessed for quantifying liver fat. This study aims to validate DL models for pancreatic PDFF quantification and compare pancreas and liver fat content. We evaluated three DL models—Non-Linear Variables Neural Network (NLV-Net), U-Net, and Multi-Decoder Water-Fat separation Netwo

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Gullo, Lucio, Eugenio Salizzoni, Carla Serra, Lucia Calculli, Luciana Bastagli, and Marina Migliori. "Can Pancreatic Steatosis Explain the Finding of Pancreatic Hyperenzymemia in Subjects With Dyslipidemia?" Pancreas 33, no. 4 (2006): 351–53. http://dx.doi.org/10.1097/01.mpa.0000240603.26312.2a.

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Vlăduț, C., D. M. Bilous, R. Prodan, et al. "Pancreatic steatosis in pancreatic cystic neoplasms: insights from a matched case-control study." Endoscopy 57, S 02 (2025): S224. https://doi.org/10.1055/s-0045-1805550.

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35

Rasineni, Karuna, Jacy L. Kubik, Carol A. Casey, and Kusum K. Kharbanda. "Inhibition of Ghrelin Activity by Receptor Antagonist [d-Lys-3] GHRP-6 Attenuates Alcohol-Induced Hepatic Steatosis by Regulating Hepatic Lipid Metabolism." Biomolecules 9, no. 10 (2019): 517. http://dx.doi.org/10.3390/biom9100517.

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Alcoholic steatosis, characterized by an accumulation of triglycerides in hepatocytes, is one of the earliest pathological changes in the progression of alcoholic liver disease. In our previous study, we showed that alcohol-induced increase in serum ghrelin levels impair insulin secretion from pancreatic β-cells. The consequent reduction in the circulating insulin levels promote adipose-derived fatty acid mobilization to ultimately contribute to hepatic steatosis. In this study, we determined whether inhibition of ghrelin activity in chronic alcohol-fed rats could improve hepatic lipid homeost

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36

van Raalte, Daniël H., Nynke J. van der Zijl, and Michaela Diamant. "Pancreatic steatosis in humans: cause or marker of lipotoxicity?" Current Opinion in Clinical Nutrition and Metabolic Care 13, no. 4 (2010): 478–85. http://dx.doi.org/10.1097/mco.0b013e32833aa1ef.

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37

Sumiyoshi, S., Y. Kobayashi, K. Souda, Y. Takehara, and H. Nakamura. "Hepatobiliary and pancreatic: Insulinoma metastases with focal hepatic steatosis." Journal of Gastroenterology and Hepatology 20, no. 4 (2005): 650. http://dx.doi.org/10.1111/j.1440-1746.2005.03871.x.

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38

Yang, Wenjuan, Jing Li, and Chengwei Tang. "Su1893 Betaine Alleviates Alcohol-Induced Pancreatic Steatosis in Rats." Gastroenterology 146, no. 5 (2014): S—495. http://dx.doi.org/10.1016/s0016-5085(14)61782-0.

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Chikamoto, Akira, Daisuke Hashimoto, Yuki Kitano, et al. "Analysis of hepatic steatosis after surgery for pancreatic carcinoma." Pancreatology 16, no. 4 (2016): S78. http://dx.doi.org/10.1016/j.pan.2016.06.281.

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40

De Lucia, Sara Sofia, Enrico Celestino Nista, Maria Assunta Zocco, et al. "Pancreatic steatosis: metabolic implications and correlation with ultrasound findings." Pancreatology 23 (November 2023): e77. http://dx.doi.org/10.1016/j.pan.2023.06.600.

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41

Hristich, T. N. "Important role of fat tissue hormones in development of comorbid chronic pancreatitis and obesity." Bulletin of the Club of Pancreatologists 37, no. 3 (2017): 30–35. http://dx.doi.org/10.33149/vkp.2017.03.03.

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Aim of this paper is to consider the role of hormones of the adipose tissue in mechanisms of obesity, metabolic syndrome, type 2 diabetes mellitus upon chronic pancreatitis. Materials and methods. The literature review indicates the value of visceral fat in the development of insulin resistance, dyslipidemia, including atherogenic one, taking into account the possible infiltration of pancreatic tissue by adipocytes. Participation of some adipocytokines of adipose tissue in the development of obesity upon chronic pancreatitis is highlighted. It is shown that in some cases the hormones of

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42

D’Cruz, V., A. De Zutter, M. Van den Broecke, et al. "Prevalence of metabolic dysfunction-associated fatty liver disease after pancreatic surgery in a historical Belgian cohort and review of the literature." Acta Gastro Enterologica Belgica 87, no. 3 (2024): 373–80. http://dx.doi.org/10.51821/87.3.10078.

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Background and objectives: Metabolic dysfunction-associated fatty liver disease (MAFLD) has been reported as a complication after pancreatic surgery. The aim of this study is to assess this phenomenon in a Belgian population, specifically in a period in time when less perioperative chemotherapy was given. Methods: We performed a retrospective monocentric cohort study with 124 selected patients who underwent pancreatic surgery – pancreaticoduodenectomy (PD), distal pancreatectomy (DP) or total pancreatectomy – between 2005 and 2014. Steatosis was assessed radiologically, using Hounsfield units

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Gómez-Zorita, Saioa, Iñaki Milton-Laskíbar, Leixuri Aguirre, Alfredo Fernández-Quintela, Jianbo Xiao, and María P. Portillo. "Effects of Pterostilbene on Diabetes, Liver Steatosis and Serum Lipids." Current Medicinal Chemistry 28, no. 2 (2020): 238–52. http://dx.doi.org/10.2174/0929867326666191029112626.

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: Pterostilbene, a phenolic compound derived from resveratrol, possesses greater bioavailability than its parent compound due to the presence of two methoxyl groups. In this review, the beneficial effects of pterostilbene on diabetes, liver steatosis and dyslipidemia are summarized. Pterostilbene is a useful bioactive compound in preventing type 1 diabetes, insulin resistance and type 2 diabetes in animal models. Concerning type 1 diabetes, the main mechanisms described to justify the positive effects of this phenolic compound are increased liver glycogen content and hepatic glucokinase and ph

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Rech, Leslie, Peter Zahradka, and Carla G. Taylor. "Marginal Zinc Deficiency Promotes Pancreatic Islet Enlargement While Zinc Supplementation Improves the Pancreatic Insulin Response in Zucker Diabetic Fatty Rats." Nutrients 16, no. 12 (2024): 1819. http://dx.doi.org/10.3390/nu16121819.

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Zinc deficiency has been associated with the worsening of diabetes while zinc supplementation has been proposed to ameliorate diabetes. This study examined the effects of marginal zinc deficiency (MZD) and zinc supplementation (ZS) on obesity, glycemic control, pancreatic islets, hepatic steatosis and renal function of Zucker diabetic fatty (ZDF) rats. Male ZDF rats were fed an MZD, zinc control (ZC) or ZS diet (4, 30 and 300 mg Zn/kg diet, respectively), and lean Zucker rats were fed a ZC diet for 8 weeks. MZD and ZS did not alter body weight or whole-body composition in ZDF rats. MZD ZDF rat

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45

Zvyagin, A. A., N. Yu Fateyeva, L. V. Moshurova, T. V. Chubarov, M. A. Denisova, and V. A. Ermolovskaya. "Magnetic resonance imaging in the diagnosis of steatosis in metabolic-associated fatty liver disease in adolescents." Pediatria. Journal named after G.N. Speransky 103, no. 4 (2024): 74–80. http://dx.doi.org/10.24110/0031-403x-2024-103-4-74-80.

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Incidence of overweight and obesity is increasing among the child population Worldwide. The prevalence of fatty liver disease is increasing as well. The transition to the new term “metabolically associated fatty liver disease” (MAFLD) implies the identification of hepatic steatosis and its quantitative assessment. Magnetic resonance imaging (MRI) has significant advantages for assessing hepatic steatosis, but is rarely used in pediatric practitioners’ routine. The purpose of this research was to improve the quality of MAFLD diagnosis in adolescents with obesity by identifying steatosis and liv

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46

Jaghutriz, Benjamin Assad, Róbert Wagner, Martin Heni, et al. "Metabolomic Characteristics of Fatty Pancreas." Experimental and Clinical Endocrinology & Diabetes 128, no. 12 (2019): 804–10. http://dx.doi.org/10.1055/a-0896-8671.

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Abstract Objective Pancreatic steatosis is associated with impaired beta cell function in patients with prediabetes. The pathomechanisms underlying this association still remain to be elucidated. Recent data show that adipocytes are situated within the pancreatic parenchyma and therefore give raise to hypothesize that pancreatic fat together with known and unknown metabolites such as hepatokines affect insulin secretion. Applying a targeted metabolomic approach we investigated possible circulating markers of pancreatic fat in order to better understand its role in the pathophysiology of impair

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47

Zigmond, Ehud, Sarah W. Zangen, Orit Pappo та ін. "β-Glycosphingolipids improve glucose intolerance and hepatic steatosis of the Cohen diabetic rat". American Journal of Physiology-Endocrinology and Metabolism 296, № 1 (2009): E72—E78. http://dx.doi.org/10.1152/ajpendo.90634.2008.

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A link between altered levels of various gangliosides and the development of insulin resistance was described in transgenic mice. Naturally occurring glycosphingolipids were shown to exert immunomodulatory effects in a natural killer T (NKT) cell-dependent manner. This study examined whether glycosphingolipid-induced modulation of the immune system may reduce pancreatic and liver steatosis and stimulate insulin secretion in the Cohen diabetes-sensitive (CDS) rat, a lean model of non-insulin-resistant, nutritionally induced diabetes. Four groups of CDS rats fed a diabetogenic diet were treated

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48

Stepanov, Yu M., N. Yu Zavgorodnyaya, and O. Yu Lukianenko. "Pancreatic steatosis in children. Part 1. Etiology, epidemiology and pathogenesis." GASTROENTEROLOGY 51, no. 1 (2017): 46–55. http://dx.doi.org/10.22141/2308-2097.51.1.2017.97871.

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Lee, Y., I. Lingvay, L. S. Szczepaniak, M. Ravazzola, L. Orci, and R. H. Unger. "Pancreatic steatosis: harbinger of type 2 diabetes in obese rodents." International Journal of Obesity 34, no. 2 (2009): 396–400. http://dx.doi.org/10.1038/ijo.2009.245.

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Li, Jing, Yi Xie, Fang Yuan, Bin Song, and Chengwei Tang. "873 The Possible Genetic Background of Alcohol-Induced Pancreatic Steatosis." Gastroenterology 142, no. 5 (2012): S—150—S—151. http://dx.doi.org/10.1016/s0016-5085(12)60569-1.

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