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Relevant bibliographies by topics / Pancytopenie

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Academic literature on the topic 'Pancytopenie'

Author: Grafiati

Published: 4 June 2021

Last updated: 10 February 2022

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Journal articles on the topic "Pancytopenie"

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Polák, Pavel, Jan Kamelander, Jana Michalcová, Jiřina Zavřelová, Libor Červinek, Tomáš Rohan, Zuzana Pazdičová, Václav Odstrčilík, and Miroslav Penka. "Pancytopenia, panhypopituitarism and liver cirrhosis: analysis of a difficult clinical case." Vnitřní lékařství 65, no. 11 (November 1, 2019): 715–20. http://dx.doi.org/10.36290/vnl.2019.124.

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Polák, Pavel, Leoš Křen, Jana Lindušková, Andrea Pavlíková, Michael Doubek, Markéta Šejnohová, and Miroslav Penka. "Pancytopenia as the main manifestation of human immunodeficiency virus infection in the AIDS stage." Vnitřní lékařství 67, no. 4 (June 29, 2021): E26—E28. http://dx.doi.org/10.36290/vnl.2021.065.

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Raja, Shruti, Febe Renjitha Suman, Julius Xavier Scott, M. S. Latha, Aruna Rajenderan, and Ethican Abhirami. "Pancytopenia – (?) An obstacle in the diagnosis and outcome of pediatric acute lymphoblastic leukemia." South Asian Journal of Cancer 04, no. 02 (April 2015): 068–71. http://dx.doi.org/10.4103/2278-330x.155648.

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Abstract Context: Acute lymphoblastic leukemia (ALL) ranks first among pediatric malignancies. 8-12% of ALL present with pancytopenia and 2% with hypocellular marrow a. Diagnosis of ALL in the background of pancytopenia and aplastic bone marrow is difficult. Aims: This study was aimed to compare the clinicopathologic, genetic, and outcome of paediatric ALL patients with and without pancytopenia. Settings and Design: This is a retrospective cross-sectional study. Susbjects and Methods: The study included all ALL patients presenting with pancytopenia. The control group included equal number of randomly selected patients with ALL without pancytopenia treated during the same period. Ethics committee approved this study. The demographic, laboratory, and treatment-related details were retrieved from the records and entered in an Excel sheet. Statistical Analysis Used: Data was analyzed with Chi-square test with IBM SPSS statistics 16 software. Results: Diagnosis by peripheral smear is significantly lower (P = 0.015) in comparison with the control group. There is no significant difference in diagnosis between the groups by bone marrow aspirate (P = 0.731) and biopsy (P = 0.849). The diagnosis of leukemia is misdiagnosed as hypo cellular/aplastic marrow in 10% of the pancytopenic patients. Flow cytometry yielded the diagnosis in all the pancytopenic patients. Though cytogenetic abnormalities are more common in pancytopenic group, it is not statistically significant (P = 0.106). There is no significant difference in treatment outcome between the groups (P = 0.0827%). Conclusions: Clinical expertise is highly essential to evaluate a case of pancytopenia to diagnose leukemia. Pancytopenia is an obstacle in the diagnosis of ALL without immunophenotyping. There is no significant difference in the outcome between the two groups.

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Tazi Mezalek, Zoubida, Wafaa Ammouri, Mouna Maamar, Meriem Bourkia, Hicham Harmouche, Mohammed Adnaoui, Meryem Alaoui, and Nisrine Bouigua. "Vitamine B12 Deficiency Presenting As Severe Pancytopenia in Adult Patients. a Monocentric Study of 104 Cases." Blood 128, no. 22 (December 2, 2016): 4812. http://dx.doi.org/10.1182/blood.v128.22.4812.4812.

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Abstract Vitamin B12 deficiency should be suspected in all patients with unexplained anemia and/or neuropsychiatric symptoms, and special attention should be paid to patients at risk (the elderly, vegetarians, patients with autoimmune thyroiditis or vitiligo, patients receiving proton pump inhibitors or biguanides for prolonged periods). Documented symptomatic pancytopenia related to cobalamin deficiency is very rare representing less than 5% cases in different series. We aim to report an important series of pancytopenia related to cobalamin deficiency to analyze the clinical, biological and outcomes of those patients. Methods : Consecutive patients presented with B12 deficiency and pancytopenia were included. Charts were retrospectively reviewed from an internal medicine department during a period of 15 years (2000-2015). Diagnosis of cobalamine deficiency used the vitamin B12 dosage bellow 200 pg/ml. Assessment included clinical features, blood count and morphological review and a statistical analysis of those parameters comparing pancytopenic and non-pancytopenic patients. Results The medical records of 268 consecutive patients hospitalized for cobalamin deficiency from January 2000 to December 2015 were identified and retrospectively analyzed. Among them, 104 patients had pancytopenia (38.8%). The median age was 55+17 years and the sex ratio : 1. Pancytopenia revealed vitamin B12 deficiency in all cases. Neurological manifestations were associated in 13 cases and digestive involvement in 16 cases. Twenty five patients presented with febrile neutropenia. Fourteen patients (13.4%) presented with "pseudo" thrombotic microangiopathy. The mean hemoglobin level was 58+19 g/L, the neurtophils count was 1188+579/µl and median platelets count was 69.616+34.379/µl. Macrocytosis was present in 80.7% of patient with mean MCV was 106+11 fl, but was normal (<95 fl) in 20 patients. The mean serum vitamine B12 levels was 80+11 pg/ml. The median serum lactate dehydrogenase level was high (3.204 IU/L). The causes of B12 vitamin deficiency were mainly food cobalamin malabsorption (63.4%) and pernicious anemia (28.8%). Correction of the hematological abnormalities was achieved in all patients treated with either intramuscular, subcutaneous or oral cyanocobalamin. Significant differences appears comparing patients with or without pancytopenia. Pancytopenic patients had significantly lower MCV mean (p=0.001), lower polynuclear neutrophils count and lower platelets count. Atrophic gastritis was also significantly associated with pancytopenia (p=0.005). There was no significant difference in mean haemoglobin level (p=0.3), serum cobalamin level (p=0.17). Looking at other variables that could explain the high prevalence of pancytopenia in our series, there were no difference in ferritinemia level or number of patients with of iron deficiency . Conclusion In this study, based a single institution with a large number of consecutive patients with well-documented cobalamin deficiency, we found a high prevalence (38,8%) of severe pancytopenia. We don't find yet an explanation for those findings. Those results can also remind us that severe vitamin B12 deficiency may present with findings mimicking malign hematologic disorder and timely recognition and supplementation lead to resolution of symptoms and blood abnormalities. Disclosures No relevant conflicts of interest to declare.

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Mupamombe, Charles T., Felix M. Reyes, Derek B. Laskar, and Joseph Gorga. "Myxedema Coma Complicated by Pancytopenia." Case Reports in Medicine 2019 (July 14, 2019): 1–6. http://dx.doi.org/10.1155/2019/2320751.

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Hypothyroidism is common, with an extreme manifestation of myxedema coma if untreated. Hematologic consequences of myxedema coma include mild leukopenia and anemia, rarely pancytopenia. We present a patient with typical symptoms of myxedema coma, but found to be pancytopenic, with sustained response to levothyroxine and blood transfusion for anemia.

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Chikan, Nadia Shafi, Qazi Mohammad Iqbal, and Altaf Hussain Mir. "Clinico-haematological profile of adult pancytopenia patients at a tertiary care institute in South India." International Journal of Research in Medical Sciences 9, no. 2 (January 29, 2021): 521. http://dx.doi.org/10.18203/2320-6012.ijrms20210435.

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Background: Pancytopenia is not a disease by itself; rather it describes simultaneous presence of anemia, leukopenia and thrombocytopenia resulting from a number of disease processes. Varieties of hematological and non-hematological disorders may affect bone marrow either primarily or secondarily, resulting in the manifestation of pancytopenia. The incidence of various hematological disorders causing pancytopenia varies due to geographical distribution and genetic predisposition. This study highlights the spectrum of causes, clinical presentation and bone marrow morphology of pancytopenia.Methods: This prospective observational study was conducted for a period of two years at Al-Ameen Medical College, Bijapur, Bangalore. During this period, fifty patients with a hematological diagnosis of pancytopenia were studied during period in the department of pathology.Results: Among the 50 cases studied, 35 were males and 15 were females. Most of the patients presented with generalized weakness and fever. The commonest physical finding was pallor, followed by splenomegaly and hepatomegaly. Dimorphic anemia was predominant blood picture. Bone marrow study showed 72% hypercellular marrow, 12% normocellular and 16% hypocellular marrow. The commonest cause for pancytopenia was megaloblastic anemia followed by iron deficiency anaemia and malaria.Conclusions: The present study concludes that detailed hematological investigations along with bone marrow examination in pancytopenic patients is helpful to diagnose or rule out the causes of pancytopenia.

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Girardi, Angela Ferronato, Amanda Noéli da Silva Campos, Caroline Argenta Pescador, Arleana Do Bom Parto Ferreira de Almeida, Adriane Jorge Mendonça, Luciano Nakazato, Anderson Castro Soares de Oliveira, and Valeria Régia Franco Sousa. "Quantitative analysis of bone marrow in pancytopenic dogs." Semina: Ciências Agrárias 38, no. 6 (November 23, 2017): 3639. http://dx.doi.org/10.5433/1679-0359.2017v38n6p3639.

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The pancytopenia can be associated with intra and extra medullary disorders. When the etiology is not obvious, the examination of bone marrow is necessary. The study aims to report and discuss quantitative amendments in bone marrow and their causes in dogs with pancytopenia. Bone marrow aspirate was obtained from 65 dogs with pancytopenia over a period of 13 consecutive months for preparation of smears stained by Giemsa and observed in light microscopy. Five hundred cells differential count was held; and assessed myeloid:erythroid ratio, cellularity, megakaryocytes and direct parasitological examination. The data were evaluated by the Chi-square statistical test. Blood tests of 3120 canines with several clinical changes were analyzed to identify pancytopenia, which accounted for 167 (5.4%) dogs. Interpretation of quantitative characteristics was carried out from the bone marrow smear in 65 pancytopenic dogs and the etiology was established in 40 (61.5%) of these, which included infection by E. canis and L. chagasi, idiopathic aplastic anemia, chronic renal failure and co-infections. In 17 (26.2%) animals were not observed medullary changes. The most bone marrow change was observed myeloid and erythroid hypoplasia in 17 (26.2%) dogs, followed by myeloid and erythroid hyperplasia (24.6%). The myelogram association to the blood cell count allowed the identification of medullary findings and its participation on the occurrence of pancytopenia cases. The number of cases resulting from infectious diseases was 38 (58.5%) in canine assessed: significant number due to its endemic characteristic of the study region.

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Rahman, ATM Ataur, ARM Saifuddin Ekram, Mohammed Shahadat Hossain, Jayanta Kumar Saha, Fahreen Rahman, Muhammad Kamruzzaman, Ahmed Manadir Hossain, and MM Shahin Ul Islam. "Aetiology and Clinical Presentation of Pancytopenia in a Teaching Hospital." Faridpur Medical College Journal 13, no. 2 (October 20, 2019): 62–65. http://dx.doi.org/10.3329/fmcj.v13i2.43622.

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Pancytopenia is a morphological description of the peripheral blood picture irrespective of its cause & it denotes simultaneous presence of anaemia, leucopenia and thrombocytopenia. The study was done to observe the demographic profile & the clinical presentations of pancytopenic patients and to find out the aetiology of pancytopenia. This cross sectional study was carried out in Medicine ward of Rajshahi Medical College Hospital from August 2005 to July 2006. A uniform protocol was followed in all cases, to have appropriate history, physical findings & laboratory investigations. The commonest cause of pancytopenia was aplastic anaemia (48%) which was followed by hypersplenism (22%) & acute leukemia (16%). The majority cases of aplastic anaemia were idiopathic (57.69%), only 42.31% cases were due to secondary causes. The majority (94%) of patients was in the age range of 14-60 years and up to 54% was in the age group of 20-40 years. The ratio of male to female in patients of pancytopenia was 1.5:1 and 3:1 in aplastic anaemia. In secondary causes of aplastic anaemia, insecticides and viral hepatitis was found to be causative agent in 45.45% cases of each followed by pregnancy in 9.10% of cases. The common presenting complains of aplastic anaemia were generalized weakness (100%), gum bleeding (76.92%), fever (76.92%) and pallor (96.15%). Faridpur Med. Coll. J. Jul 2018;13(2): 62-65

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Chand, Ramesh, and Nutan Singh. "Clinic-etiological profile of pancytopenia in children: a tertiary care center based study of Kumaun region, India." International Journal of Contemporary Pediatrics 5, no. 6 (October 22, 2018): 2173. http://dx.doi.org/10.18203/2349-3291.ijcp20184275.

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Background: Pancytopenia is not a disease entity but a triad of findings in which all blood cell lineages i.e leukocytes, erythrocytes and platelets are reduced in blood .It is consequence of various medical conditions. Present study was conducted to assess the etiology, clinical profile and bone marrow morphology of pancytopenia.Methods: A cross sectional prospective study was carried out among 42 children of age between 1 to 15 year with pancytopenia. Blood samples of the patients were analyzed for complete blood count and peripheral smear along with presence and absence of immature cells and abnormal cells. In bone marrow examination, morphology of all cell’s lineage, cellularity, parasite and abnormal cells were scrutinized. Trephine biopsy was done if indicated. Special investigations were done to confirm the diagnosis.Results: Among the 42 cases studied, age of the patients ranged from 1 to 15 years with a slight male predominance. Most of the patients presented with generalized weakness and weight loss and fever. The commonest physical finding was pallor, followed by splenomegaly and hepatomegaly. The commonest cause for pancytopenia was megaloblastic anemia Among the non-haematological causes kala azar 5 (11.9%) is the leading cause in this study.Conclusions: The present study concludes that detailed primary haematological investigations along with bone marrow examination in pancytopenic patients are helpful for diagnosis and management. This study also suggests that megaloblastic anaemia, dimorphic anaemia and kala-azar should also be included in differential diagnosis of pancytopenia in this geographical area.

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Premkumar, M., N. Gupta, T. Singh, and T. Velpandian. "Cobalamin and Folic Acid Status in Relation to the Etiopathogenesis of Pancytopenia in Adults at a Tertiary Care Centre in North India." Anemia 2012 (2012): 1–12. http://dx.doi.org/10.1155/2012/707402.

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Background. Pancytopenia has multiple etiologies like megaloblastic anemia, aplastic anemia, leukemia, and various infections. We investigated the clinical, etiological and hematological profile including bone marrow morphology of patients with pancytopenia in relation to their vitamin B12 and folic acid status at a tertiary care referral hospital in north India.Methods. A total of 140 consecutive patients with pancytopenia were selected from June 2007 to December 2008. Bone marrow examination and other tests were carried out as warranted, including serum cobalamin and folate assays using liquid chromatography mass spectroscopy (LC MS/MS).Results. The study population consisted of 92 males and 48 females with a mean age of 32.8 years. Megaloblastic anemia 60.7%, aplastic anemia (7.8%), and leukemia (9.2%) were common causes. Infectious causes (16.4% of all cases) included leishmaniasis, HIV–AIDS, malaria and tuberculosis. Severe cobalamin deficiency (B12 < 100 pg/mL) was seen in 81% of all patients including 91.6% of patients with MA. In contrast, only 7.14% of all pancytopenic patients were folate deficient. Folate deficiency (<5 ng/mL) was seen in just 5% MA patients. Combined cobalamin and folate deficiency was seen in 5 patients (3.51%).Conclusion. Cobalamin deficiency was found to be more common in our setting and is largely underdiagnosed in the age of folate supplementation. Infectious diseases like tuberculosis, leishmaniasis, and increasingly HIV are important and treatable causes of pancytopenia. This is in contrast with the developed nations where the bulk of disease is due to malignancy or marrow aplasia.

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Dissertations / Theses on the topic "Pancytopenie"

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Heijden, Michiel Simon van der. "The Fanconi anemia/BRCA2 pathway in pancreatic cancer." [S.l. : Amsterdam : s.n.] ; Universiteit van Amsterdam [Host], 2005. http://dare.uva.nl/document/79702.

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Penchenat, Olivier. "Les formes pancytopéniques de la tuberculose aigue͏̈." Bordeaux 2, 1990. http://www.theses.fr/1990BOR25044.

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Artaud, Patricia Gris. "Mastocytose osseuse révélée par une pancytopénie : à propos d'un cas." Montpellier 1, 1989. http://www.theses.fr/1989MON11084.

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LE, PANS NICOLE. "La maladie de pearson : un exemple de cytopathie mitochondriale ; a propos d'une observation personnelle et revue de la litterature." Rennes 1, 1992. http://www.theses.fr/1992REN1M057.

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Greco, Frédéric. "Pancytopénie révélant une carence en vitamine B12 à propos d'un cas et revue de la littérature." Montpellier 1, 1992. http://www.theses.fr/1992MON11119.

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Colleri-Leduc, Xavier. "Connaissances actuelles sur la carence en folates chez l'éthylique chronique : à propos d'un cas de pancytopénie." Bordeaux 2, 1990. http://www.theses.fr/1990BOR25006.

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Bannwarth, E. "Pancytopénie au méthotrexate à faible dose dans la polyarthrite rhumatoi͏̈de : à propos d'un cas et revue de la littérature." Bordeaux 2, 1992. http://www.theses.fr/1992BOR2M203.

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Diouf-Lasserre, Marie-Catherine. "Manifestations hématologiques au cours de la brucellose septicémique : à propos de deux cas, purpura thrombopénique, pancytopénie." Bordeaux 2, 1992. http://www.theses.fr/1992BOR2M151.

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Bell, Charlotte Rosie. "The pathogenesis of Bovine Neonatal Pancytopenia." Thesis, University of Edinburgh, 2014. http://hdl.handle.net/1842/17857.

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Bovine Neonatal Pancytopenia (BNP) is a disease of calves, characterised by peripheral blood and bone marrow depletion, which emerged in Europe in 2007. A strong epidemiological association between BNP and the administration of a particular inactivated Bovine Viral Diarrhoea (BVD) vaccine (Pregsure BVD, Pfizer Animal Health) to the dams of affected calves has been reported. Early studies suggested that BNP is mediated by the transfer of alloantibodies in colostrum and that these alloantibodies recognise major histocompatibility complex (MHC) class I molecules. This led to the hypothesis that Pregsure contains bovine MHC I molecules, originating from the MDBK line cell used in vaccine production, and that this is responsible for the generation of alloantibodies in particular cows injected with the vaccine. This project aimed to investigate the mechanisms by which BNP arises and develops. In particular to gain an understanding of the molecular basis of the syndrome and how this influences the number of cows and calves affected and the specificity of the pathology for the haematopoietic system. Haematological analysis of clinically normal calves born on a BNP-affected farm demonstrated that 15% of calves had profoundly abnormal haematology and could be described as affected by subclinical BNP. BNP was reproduced experimentally by feeding pooled colostrum to neonatal calves, confirming the role of colostrum in mediating the condition. Detailed analysis of serial haematology and bone marrow pathology from these calves demonstrated variable alloantibody damages to different haematopoietic lineages. In vitro cellular assays using a panel of MHC I-defined bovine leukocyte cell lines and mouse cells individually transfected with the MDBK-MHC I alleles demonstrated that Pregsure vaccinated cows have significantly higher titres of functionally active MHC I alloantibodies than BVDV unvaccinated cows or cows vaccinated with alternative BVDV vaccines. The alloantibody response was found to be heterogeneous in individual Pregsure vaccinated cows. MHC I expression levels on peripheral blood and bone marrow cells, assessed by flow cytometry, was shown to correlate with levels of in vitro and in vivo alloantibody damage. Overall, the results of this project demonstrate that the pathogenesis of BNP is mediated by the transfer of MHC I-specific alloantibodies via colostrum that cause rapid destruction of peripheral blood and bone marrow cells, and which is dependent on the titre of alloantibody produced by an individual cow, its specificity for the MHC I alleles present, and density of MHC I expression on the specific cells.

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Kasonta, Rahel [Verfasser]. "On the aetiology of bovine neonatal pancytopenia (BNP) / Rahel Kasonta." Mainz : Universitätsbibliothek Mainz, 2014. http://d-nb.info/1059427060/34.

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Books on the topic "Pancytopenie"

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P, Alter Blanche, ed. Aplastic anemia, acquired and inherited. Philadelphia: Saunders, 1994.

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Publications, ICON Health. Pancytopenia - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References. ICON Health Publications, 2004.

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MacCallum, Niall S. Management of oncological complications in the ICU. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0376.

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Important treatment complications relevant to critical care are discussed. Cancer-related pain is complex and requires multidisciplinary care, particularly in the peri-operative setting. Chemotherapeutic complications include pancytopenia, cardiac, pulmonary, renal, gastrointestinal, hepatic, and neurotoxicity. Radiotherapy complications include cardiac, pulmonary, and gastrointestinal toxicity. In general, management includes assessing the risk-benefit to cytotoxic therapy withdrawal and supportive care. There is a paucity of proven treatment options for most complications, althoughcertain therapies are used to prevent and/or treat complications (e.g. tumour lysis syndrome). Thromboembolic disease is a common cause of mortality and morbidity; low molecular weight heparin therapy may be superior to oral anticoagulation.

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Book chapters on the topic "Pancytopenie"

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Sills, R. H., and A. Deters. "Pancytopenia." In Practical Algorithms in Pediatric Hematology and Oncology, 12–13. Basel: KARGER, 2003. http://dx.doi.org/10.1159/000069577.

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Annamalai, Aniyizhai. "Pancytopenia." In Medical Management of Psychotropic Side Effects, 197–99. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-51026-2_30.

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Maier, Sarah Lena, and Kai Lehmberg. "Severe Pancytopenia." In Pediatric Immunology, 395–402. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-21262-9_76.

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Beck, Norman. "Pancytopenia and Bicytopenia." In Diagnostic Hematology, 297–308. London: Springer London, 2009. http://dx.doi.org/10.1007/978-1-84800-295-1_13.

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Hay, Deborah. "Pancytopenia and febrile neutropenia." In Acute Medicine - A Practical Guide to the Management of Medical Emergencies, 5th Edition, 577–83. Chichester, UK: John Wiley & Sons, Ltd, 2017. http://dx.doi.org/10.1002/9781119389613.ch101.

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Pandey, Soumya, and Terry Harville. "Liver Transplantation Who Developed Pancytopenia Post-transplantation." In Pediatric Autoimmunity and Transplantation, 295–303. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-26280-8_52.

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"Pancytopenia." In Diagnostic Pathology: Blood and Bone Marrow, 172–79. Elsevier, 2018. http://dx.doi.org/10.1016/b978-0-323-39254-9.50042-8.

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Bixby, Dale. "Pancytopenia." In The Saint-Chopra Guide to Inpatient Medicine, edited by Sanjay Saint and Vineet Chopra, 323–28. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190862800.003.0059.

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"Pancytopenia." In Encyclopedia of Genetics, Genomics, Proteomics and Informatics, 1436. Dordrecht: Springer Netherlands, 2008. http://dx.doi.org/10.1007/978-1-4020-6754-9_12225.

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"Fanconi Pancytopenia." In Encyclopedia of Cancer, 1695. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-46875-3_100886.

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Conference papers on the topic "Pancytopenie"

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RODRIGUES, PATRICK FONTES, LUIZ PIERRE HUNING, ANA PAULA ADAME, and MARCIO AUGUSTO NOGUEIRA. "ENTEROPATHIC SPONDYLITIS: TREATMENT, PANCYTOPENIA AND PORTAL HYPERTENSION IDIOPATHIC." In 36º Congresso Brasileiro de Reumatologia. São Paulo: Editora Blucher, 2019. http://dx.doi.org/10.5151/sbr2019-092.

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Nunes, Diego, Caio Rutichelli Cardoso, Raissa Dudienas Domingues Pereira, Vinícius Verlangieri Soubihe, Lilian Tereza Lavras Costalla, and Simone Appenzeller. "DIFFERENTIAL DIAGNOSIS OF PANCYTOPENIA IN SYSTEMIC LUPUS ERYTHEMATOUS." In Congresso Brasileiro de Reumatologia 2020. Sociedade Brasileira de Reumatologia, 2021. http://dx.doi.org/10.47660/cbr.2020.17575.

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Ciofoaia, G. A., and J. Murati. "Community Acquired Adenovirus Pneumonia Complicated with Pancytopenia in Immunocompetent Host." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a5204.

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Meher, J., M. S. Siddiqui, K. B. Nagpure, R. Rokkam, M. K. Nayak, and S. Biswal. "Bone Marrow Eosinophilia Causing Pancytopenia in Pulmonary Tuberculosis: A Curious Case." In American Thoracic Society 2021 International Conference, May 14-19, 2021 - San Diego, CA. American Thoracic Society, 2021. http://dx.doi.org/10.1164/ajrccm-conference.2021.203.1_meetingabstracts.a1378.

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Mull, E. S., N. S. Achenjang, and K. Krivchenia. "Drug Induce Fever and Pancytopenia: A Case of Piperacillin-Tazobactam Hypersensitivity." In American Thoracic Society 2021 International Conference, May 14-19, 2021 - San Diego, CA. American Thoracic Society, 2021. http://dx.doi.org/10.1164/ajrccm-conference.2021.203.1_meetingabstracts.a3373.

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Hsu, D., S. Wilde, and J. Velotta. "Thymoma-Associated Immunodeficiency: A Rare Case of Good's Syndrome and Severe Pancytopenia." In American Thoracic Society 2021 International Conference, May 14-19, 2021 - San Diego, CA. American Thoracic Society, 2021. http://dx.doi.org/10.1164/ajrccm-conference.2021.203.1_meetingabstracts.a2409.

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Alatassi, E., A. Mahmoud, A. Mohamed, A. Gomaa, and I. Ismail-Sayed. "Unexplained Lactic Acidosis and Digital Cyanosis in a Patient with Acute Myeloid Leukemia and Pancytopenia." In American Thoracic Society 2021 International Conference, May 14-19, 2021 - San Diego, CA. American Thoracic Society, 2021. http://dx.doi.org/10.1164/ajrccm-conference.2021.203.1_meetingabstracts.a2049.

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Yahşi, Aysun, Tuğba Erat, Halil Özdemir, Tuğçe Tural Kara, Reyhan Erol, Fatma Tuba Eminoğlu, Elif Ince, et al. "P274 An unexpected disease in an infant with pancytopenia and pulmonary abscess: glycogen storage disease type 1b." In 8th Europaediatrics Congress jointly held with, The 13th National Congress of Romanian Pediatrics Society, 7–10 June 2017, Palace of Parliament, Romania, Paediatrics building bridges across Europe. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2017. http://dx.doi.org/10.1136/archdischild-2017-313273.362.

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Kuppuswamy, V., and J. A. Haspel. "A 60 Year Old Female with Rheumatoid Arthritis and Psoriatic Arthritis Presents with New Onset Pancytopenia and Coffee Ground Emesis." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a4855.

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Wiesen, Jonathan, Moshe Ornstein, Daniel A. Culver, Carol F. Farver, and Joseph G. Parambil. "Pancytopenia And Elevated Pancreatic Enzymes: An Atypical Presentation Of The Granulomatous Lesions Of Unknown Significance (GLUS) Syndrome Treated Successfully With Steroids And Pentoxyfilline." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a6588.

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