Academic literature on the topic 'Papovaviridae Infections'

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Journal articles on the topic "Papovaviridae Infections"

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Le Calloch, R., J. C. Ianotto, C. Berthou, and A. Tempescul. "Hemorrhagic Cystitis due to BK Reactivation in a Young Female Treated for Hodgkin-Disease." Case Reports in Hematology 2011 (2011): 1–2. http://dx.doi.org/10.1155/2011/592470.

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Hodgkin's lymphoma is a disease with a high rate of curability under classic chemo-radiotherapy regimes. Complications due to chemotherapy could include viral reactivation due to chronic lymphopenia. BK virus (BKV) is a polyoma virus belonging to the Papovaviridae family with antibody seroprevalences in healthy populations varying from 60% to 80%. Initial infections are asymptomatic usually occur in early childhood, after which the viruses remain latent in the kidneys or urothelium. Reactivation of BKV occurs in individuals with severe immunosuppression during HIV infections, transplantation or, exceptionally, after classical chemotherapy. BKV incidence is approximately 0% to 5% in immunocompetent individuals. Reactivation is associated with nephropathy and haemorrhagic cystitis. Herein, we present a case of a haemorrhagic cystitis due to BKV reactivation in a patient with Hodgkin's disease treated with chemotherapy.
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Panjkovic, Milana, and Tatjana Ivkovic-Kapicl. "Etiology and pathogenesis of precancerous lesions and invasive cervical carcinoma." Medical review 61, no. 7-8 (2008): 364–68. http://dx.doi.org/10.2298/mpns0808364p.

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Cervical cancer is the second most common gynecological malignancy in the world. Human papilloma virus (HPV) infection is the leading ethiologic agent in the development of premalignant and malignant cervical diseases. HPV is a member of the Papovaviridae family and until now over 100 types have been recognized. There are two types of viral infection: latent and productive. Virus induced oncogenesis is the result of interaction between virus oncoproteins E6 and E7 and tumor supresor host genes p53 and Rb. Many cofactors such as immunosuppression, early sexual relationship, multiple sexual partners, other sexualy transsmited infections and smooking are contributing factors of the precancerous and invasive cervical lesions. According to the oncogenic potential HPV are divided into three groups: low, intermediate and high oncogenic risk viruses. Molecular technics which are used for the virus detection are: In situ hibridisation,, Hybrid capture test and polymerasa chain reaction. Human papilloma virus testing has an important role in the follow up and treatment of women with 'atypical squamous cells of unknown significant' changes in cervical smears and low-grade squamous intraepithelial lesions, changes in punch biopsy.
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Brewer, Matthew G., Stephanie R. Monticelli, Mary C. Moran, Benjamin L. Miller, Lisa A. Beck, and Brian M. Ward. "Conditions That Simulate the Environment of Atopic Dermatitis Enhance Susceptibility of Human Keratinocytes to Vaccinia Virus." Cells 11, no. 8 (April 14, 2022): 1337. http://dx.doi.org/10.3390/cells11081337.

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Individuals with underlying chronic skin conditions, notably atopic dermatitis (AD), are disproportionately affected by infections from members of the herpesviridae, papovaviridae, and poxviridae families. Many patients with AD experience recurrent, widespread cutaneous viral infections that can lead to viremia, serious organ complications, and even death. Little is known about how the type 2 inflammatory environment observed in the skin of AD patients impacts the susceptibility of epidermal cells (keratinocytes) to viral pathogens. Herein, we studied the susceptibility of keratinocytes to the prototypical poxvirus, vaccinia virus (VV)—the causative agent of eczema vaccinatum—under conditions that simulate the epidermal environment observed in AD. Treatment of keratinocytes with type 2 cytokines (IL-4 and -13) to simulate the inflammatory environment or a tight junction disrupting peptide to mirror the barrier disruption observed in AD patients, resulted in a differentiation-dependent increase in susceptibility to VV. Furthermore, pan JAK inhibition was able to diminish the VV susceptibility occurring in keratinocytes exposed to type 2 cytokines. We propose that in AD, the increased viral susceptibility of keratinocytes leads to enhanced virus production in the skin, which contributes to the rampant dissemination and pathology seen within patients.
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Howell, D. N., and S. E. Miller. "Polyomaviruses As Emerging Pathogens." Microscopy and Microanalysis 7, S2 (August 2001): 168–69. http://dx.doi.org/10.1017/s143192760002691x.

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Polyomaviruses are double-stranded DNA viruses of the family Papovaviridae. They replicate in cell nuclei, where they form 40-50-nm, non-enveloped, icosahedral nucleocapsids that exit the host cell by lysis (Fig. 1A-C). Polyomaviruses with pathogenic potential in both human and non-human hosts have been known for many decades. in recent years, however, new pathogenic polyomaviruses have been described in man, non-human primates, and other animals. New forms of acquired and iatrogenic immunocompromise have also led to changes in the prevalence and pattern of disease caused by well-characterized polyomaviruses. This presentation provides an overview of emerging polyomavirus diseases, illustrated with vignettes from our diagnostic practices. in many cases, the detection and monitoring of polyomavirus infections has been facilitated by ultrastructural examination.Two well-characterized polyomaviruses cause disease in humans, almost invariably in the setting of immunosuppression. JC virus, described in 1971, is the causative agent for progressive multifocal leukoencephalopathy (PML), a generally fatal disorder characterized by patchy, progressive destruction of the cerebral white matter.
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Rodas Neira, Edgar, Betty Pazmino Gòmez, Roberto Coello Peralta, Julio Bermudez Bermudez, Karen Rodas Pazmino, Luis Cagua Montano, Alicia Cercado Mancero, et al. "Detección serológica del Virus Papiloma Humano en mujeres mayores de 20 años. Caso sector “Gary Esparza”, Babahoyo, Ecuador // Serological detection of human papilloma virus in women over 20 years. “Gary Esparza” sector, Babahoyo, Ecuador." Ciencia Unemi 10, no. 23 (October 4, 2017): 112. http://dx.doi.org/10.29076/issn.2528-7737vol10iss23.2017pp112-115p.

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El Virus del Papiloma Humano (HPV) tiene un genoma ADN bicatenario perteneciente a la familia de los Papovaviridae, tiene capacidad oncogénica en una variedad de mamíferos, en especial el hombre, en el que se conoce más de 200 serotipos y representa una de las enfermedades de transmisión sexual más común, por su relación de patogenia oncológica. Se clasifica en tipos de alto y bajo riesgo oncológico (IARC), también se ha descrito otros tipos de infecciones como las orofaríngeas y amigdalitis. El objetivo de este estudio fue determinar el cribado de detección de anticuerpos IgG contra los genotipos 6, 11, 16 y 18 del virus mencionado, mediante la técnica serológica de Microelisa. Mediante un estudio descriptivo, prospectivo y transversal, realizado en el primer semestre del año 2015 en el Sector Gary Esparza del cantón Babahoyo, provincia de Los Ríos, Ecuador, de un universo de 250 habitantes se tomaron muestras sanguíneas a 97 mujeres mayores de 20 años y determinó 8 casos positivos (8.25%), con ello se estableció el primer diagnóstico serológico en el país, lo que constituye una herramienta de detección preliminar o screening de un gran número de muestras. ABSTRACT The Human Papilloma Virus (HPV) is a virus that has a double-stranded DNA genome belonging to the Papovaviridae family. It has oncogenic ability in a variety of mammals, especially man, in which more than 200 serotypes are known and represents one of the most common sexually transmitted diseases because of its relation to oncological pathogenesis. It is classified into high and low cancer types (IARC), other types of infections such as oropharyngeal and tonsillitis have also been described. The objective of this study was to determine the screening of IgG antibodies against Human Papilloma Virus genotypes 6, 11, 16 and 18 using the Microelisa serological technique. A descriptive, prospective and cross-sectional study was carried out during the first semester of 2015 in the Gary Esparza Sector of Babahoyo, Los Ríos province, with a population of 250 habitants. Blood samples were taken from 97 women with 20 years old, being determined 8 positive cases (8.25%), establishing the first serological diagnosis in the country, which is a tool for preliminary screening or screening of a large number of samples.
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İzmir Güner, Şebnem, Mustafa Teoman Yanmaz, Ekrem Güner, Pınar Seymen, Melike Nalbant Moray, and Işın Kılıçarslan. "Acute Proximal Tubular Necrosis Due to Cidofovir Usage in BK Virus Related Hemorrhagic Cystitis in an Allogeneic Stem Cell Transplanted Patient." Grand Journal of Urology, 2021. http://dx.doi.org/10.5222/gju.2021.25743.

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Hemorrhagic cystitis (HS) is a frequently seen complication of bone marrow transplantation. This condition occurs depending on the preparatory regimen of bone marrow transplantation. The BK virus (BKV), a human polyomavirus, is a small double helix DNA virüs belonging to the Papovaviridae family. It is commonly found in societies as an occult infection. Whereas, cidofovir (CDV), an acyclic nucleoside phosphonate, is used as a proven antiviral agent against polyomaviruses. In this case report, acute proximal tubular necrosis due to cidofovir used in hemorrhagic cystitis caused by BK virus, and its treatment in a patient diagnosed with T-lymphoblastic lymphoma in remission, and underwent allogeneic stem cell transplantation, was presented.
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Dissertations / Theses on the topic "Papovaviridae Infections"

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Mellin, Hanna. "Human papillomavirus in tonsillar cancer /." Stockholm : Karolinska Univ. Press, 2002. http://diss.kib.ki.se/2002/91-7349-366-X/.

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Mandelson, Margaret T. "Cervical cancer : associations with HPV infection, barrier contraceptive use, and Pap smear screening /." Thesis, Connect to this title online; UW restricted, 1994. http://hdl.handle.net/1773/10919.

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Friesland, Signe. "Factors of importance for radiosensitivity of tonsillar carcinoma /." Stockholm, 2002. http://diss.kib.ki.se/2002/91-7349-211-6.

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Delmas, Véronique. "Structure et proprietes biologiques du papovavirus de hamster." Paris 6, 1986. http://www.theses.fr/1986PA066550.

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Le papovavirus de hamster (hapv) possede un tropisme restreint in vivo vis a vis des keratinocytes et des lymphocytes. Il se replique dans les tumeurs cutanes qui apparaissent chez des hamsters syriens, et induit egalement des lymphomes chez le hamster. L'organisation genetique du hapv deduite de sa sequence a montre qu'il appartient a la famille des polyomavirus. Le hapv est present dans les lymphomes sous forme de multiples copies libres possedant toujours une deletion localisee dans la meme region du genome. Les signaux de transcription precoce du hapv semblent etre actives par la region precoce de ce virus
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Books on the topic "Papovaviridae Infections"

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Jane, Sterling, and Tyring Stephen K, eds. Human papillomaviruses: Clinical and scientific advances. London: Arnold, 2001.

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A, Stanley Margaret, and International Workshop on HPV Immunology (2nd : 1993 : Cambridge, England), eds. Immunology of human papillomaviruses. New York: Plenum Press, 1994.

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Fabiano, Franco Eduardo Luiz, and Monsonégo Joseph, eds. New developments in cervical cancer screening and prevention. Oxford [England]: Blackwell Science, 1997.

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(Editor), Jane C. Sterling, and Stephen K. Tyring (Editor), eds. Human Papillomaviruses: Clinical and Scientific Advances. A Hodder Arnold Publication, 2001.

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Syrjänen, Kari J., and Stina M. Syrjänen. Papillomavirus Infections in Human Pathology. Wiley & Sons, Incorporated, John, 2001.

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Syrjänen, Kari J., and Stina M. Syrjänen. Papillomavirus Infections in Human Pathology. Wiley, 2000.

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Renzo, Barrasso, and Gross G, eds. Human papilloma virus infection: A clinical atlas. Berlin: Ullstein Mosby, 1997.

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G, Gross, and Krogh Geo von, eds. Human papillomavirus infections in dermatovenereology. Boca Raton: CRC Press, 1997.

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W, Tindle Robert, ed. Vaccines for human papillomavirus infection and anogenital disease. Austin: R.G. Landes, 1999.

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Everything You Need to Know About Human Papillomavirus. Rosen Publishing Group, 2001.

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Book chapters on the topic "Papovaviridae Infections"

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Pfister, Herbert. "Papovaviridae: The Papillomaviruses." In Laboratory Diagnosis of Infectious Diseases Principles and Practice, 301–16. New York, NY: Springer New York, 1988. http://dx.doi.org/10.1007/978-1-4612-3900-0_16.

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Arthur, Ray R., and Keerti V. Shah. "Papovaviridae: The Polyomaviruses." In Laboratory Diagnosis of Infectious Diseases Principles and Practice, 317–23. New York, NY: Springer New York, 1988. http://dx.doi.org/10.1007/978-1-4612-3900-0_17.

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"Papovaviridae." In Virus Infections of Ruminants, 187. Elsevier, 1990. http://dx.doi.org/10.1016/b978-0-444-87312-5.50028-x.

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