Relevant bibliographies by topics / Paracrine activities

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Paracrine activities'

Author: Grafiati
Published: 2 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "Paracrine activities"

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D'Andrea, S., S. Chousterman, J. E. Flechon, and C. La Bonnardiere. "Paracrine activities of porcine trophoblastic interferons." Reproduction 102, no. 1 (1994): 185–94. http://dx.doi.org/10.1530/jrf.0.1020185.

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Li, Feng, Noelle Whyte, and Christopher Niyibizi. "Differentiating multipotent mesenchymal stromal cells generate factors that exert paracrine activities on exogenous MSCs: Implications for paracrine activities in bone regeneration." Biochemical and Biophysical Research Communications 426, no. 4 (2012): 475–79. http://dx.doi.org/10.1016/j.bbrc.2012.08.095.

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Porter, D. C., E. Farmaki, S. Altilia, et al. "Cyclin-dependent kinase 8 mediates chemotherapy-induced tumor-promoting paracrine activities." Proceedings of the National Academy of Sciences 109, no. 34 (2012): 13799–804. http://dx.doi.org/10.1073/pnas.1206906109.

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Manoukian, A. S., K. B. Yoffe, E. L. Wilder, and N. Perrimon. "The porcupine gene is required for wingless autoregulation in Drosophila." Development 121, no. 12 (1995): 4037–44. http://dx.doi.org/10.1242/dev.121.12.4037.

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The Drosophila segment polarity gene wingless (wg) is required in the regulation of engrailed (en) expression and the determination of cell fates in neighboring cells. This paracrine wg activity also regulates transcription of wg itself, through a positive feedback loop including en activity. In addition, wg has a second, more direct autoregulatory requirement that is distinct from the en-dependent feedback loop. Four gene products, encoded by armadillo (arm), dishevelled (dsh), porcupine (porc) and zeste-white 3 (zw3), have been previously implicated as components of wg paracrine signaling. H
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Nawaz, Muhammad, Farah Fatima, Krishna C. Vallabhaneni, et al. "Extracellular Vesicles: Evolving Factors in Stem Cell Biology." Stem Cells International 2016 (2016): 1–17. http://dx.doi.org/10.1155/2016/1073140.

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Stem cells are proposed to continuously secrete trophic factors that potentially serve as mediators of autocrine and paracrine activities, associated with reprogramming of the tumor microenvironment, tissue regeneration, and repair. Hitherto, significant efforts have been made to understand the level of underlying paracrine activities influenced by stem cell secreted trophic factors, as little is known about these interactions. Recent findings, however, elucidate this role by reporting the effects of stem cell derived extracellular vesicles (EVs) that mimic the phenotypes of the cells from whi
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Gonzalez-Meljem, Jose Mario, and Juan Pedro Martinez-Barbera. "Adamantinomatous craniopharyngioma as a model to understand paracrine and senescence-induced tumourigenesis." Cellular and Molecular Life Sciences 78, no. 10 (2021): 4521–44. http://dx.doi.org/10.1007/s00018-021-03798-7.

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AbstractCellular senescence is a process that can prevent tumour development in a cell autonomous manner by imposing a stable cell cycle arrest after oncogene activation. Paradoxically, senescence can also promote tumour growth cell non-autonomously by creating a permissive tumour microenvironment that fuels tumour initiation, progression to malignancy and metastasis. In a pituitary tumour known as adamantinomatous craniopharyngioma (ACP), cells that carry oncogenic β-catenin mutations and overactivate the WNT signalling pathway form cell clusters that become senescent and activate a senescenc
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Allerstorfer, S., G. Sonvilla, H. Fischer, et al. "FGF5 as an oncogenic factor in human glioblastoma multiforme: autocrine and paracrine activities." Oncogene 27, no. 30 (2008): 4180–90. http://dx.doi.org/10.1038/onc.2008.61.

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Watabe, K., T. Fukuda, J. Tanaka, H. Honda, K. Toyohara, and O. Sakai. "Spontaneously immortalized adult mouse Schwann cells secrete autocrine and paracrine growth-promoting activities." Journal of Neuroscience Research 41, no. 2 (1995): 279–90. http://dx.doi.org/10.1002/jnr.490410215.

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Mesnard, D., M. Donnison, C. Fuerer, P. L. Pfeffer, and D. B. Constam. "The microenvironment patterns the pluripotent mouse epiblast through paracrine Furin and Pace4 proteolytic activities." Genes & Development 25, no. 17 (2011): 1871–80. http://dx.doi.org/10.1101/gad.16738711.

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Schütz, Eva, Rajinikanth Gogiraju, Maria Pavlaki, et al. "Age-Dependent and -Independent Effects of Perivascular Adipose Tissue and Its Paracrine Activities during Neointima Formation." International Journal of Molecular Sciences 21, no. 1 (2019): 282. http://dx.doi.org/10.3390/ijms21010282.

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Cardiovascular risk factors may act by modulating the composition and function of the adventitia. Here we examine how age affects perivascular adipose tissue (PVAT) and its paracrine activities during neointima formation. Aortic tissue and PVAT or primary aortic smooth muscle cells from male C57BL/6JRj mice aged 52 weeks (“middle-aged”) were compared to tissue or cells from mice aged 16 weeks (“adult”). Vascular injury was induced at the carotid artery using 10% ferric chloride. Carotid arteries from the middle-aged mice exhibited smooth muscle de-differentiation and elevated senescence marker
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Dissertations / Theses on the topic "Paracrine activities"

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Modrowski, Dominique. "Activites autocrine et paracrine du granulocyte-macrophage colony-stimulating factor dans les cellules osteoblastiques humaines ; regulation par les proteoglycannes." Paris 7, 1997. http://www.theses.fr/1997PA077253.

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Ce travail a pour but de mieux connaitre les mecanismes qui participent a la regulation locale de la formation osseuse par certaines cytokines produites par les cellules osteoblastiques dans l'os adulte. Pour cela, nous avons utilise des cellules osteoblastiques humaines obtenues a partir de travees osseuses prelevees chez des adultes ne presentant pas de maladie metabolique osseuse (cellules hob) ou des cellules hob immortalisees. Nous avons examine l'activite mitogene des interleukines-1 et -6 (il-1, il-6), du tumor necrosis factor (tnf) et des prostaglandines e2 endogenes dans les cellules
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Books on the topic "Paracrine activities"

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Walter, Helen Jane. The spatio-temporal modulation of the insulin-like growth factor[s] axis in the lesioned central nervous system: Implications for endocrine, paracrine and autocrine activities. University of Birmingham, 1996.

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Book chapters on the topic "Paracrine activities"

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Pflüger, K. H., A. Grüber, M. Welslau, H. Köppler, and K. Havemann. "Transferrin Derivatives with Growth Factor Activities in Acute Myeloblastic Leukemia:An Autocrine/Paracrine Pathway." In Acute Leukemias II. Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-74643-7_16.

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Bang, Peter, and Kerstin Hall. "Insulin-like growth factors as endocrine and paracrine hormones." In The Insulin-like Growth Factors. Oxford University PressOxford, 1992. http://dx.doi.org/10.1093/oso/9780198542704.003.0007.

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Abstract Three decades ago, Salmon and Daughaday (1957) proposed that the growth-promoting action of growth hormone was mediated through secondary substances. The search for such factors led to the discovery of the insulin-like growth factors (IGFs). These were discovered as three different biological activities in plasma; the growth hormone-regulated, sulphation factor activity, the non-suppressible insulin-like activity, and multiplication-stimulating activity (Daughaday et al. 1972, 1987).
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Akhurst, Rosemary J. "Localization of growth factor mRNA in tissue sections by in situ hybridization." In Growth Factors. Oxford University PressOxford, 1993. http://dx.doi.org/10.1093/oso/9780199633609.003.0007.

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Abstract It is becoming clear that, in vivo, the majority of growth factor bio-activities are effective locally, exerted via autocrine, paracrine, and (possibly?) intracrine mechanisms. To investigate molecular mechanisms of action it is therefore important to identify both sites of synthesis and sites of action for each growth factor. The development of in situ hybridization for detection of cellular mRNAs in tissue sections has provided a very powerful and precise tool with which to address the first of these issues. Using this technique, individual cells possessing RNA transcripts for a giv
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McInnes, Iain B. "Cytokines." In Oxford Textbook of Medicine. Oxford University Press, 2010. http://dx.doi.org/10.1093/med/9780199204854.003.0403.

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Cytokines are small glycoprotein mediators that are involved in every facet of immune effector function and regulation. More than 200 cytokines have been identified, which may usefully be classified in structurally related superfamilies. They can (1) function through binding to specific receptors that in turn signal via complex transduction pathways to regulate gene expression, thereby mediating positive and negative regulatory activities; (2) operate as soluble mediators in the extracellular domain or within cells, where they may also traffic to the nucleus and exhibit dual function as transc
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Liu, Shujuan, and Ahmed Ashour Ahmed. "Growth factors and uncontrolled proliferation." In Oxford Textbook of Oncology. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199656103.003.0002.

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A mandatory requirement of normal growth and development is the tight spatial and temporal synchronization of cellular proliferation within a specific tissue type and between tissue types that make up an organ, a process that is governed by intrinsic and extrinsic regulatory pathways. For a cell to synchronize its growth with that of other cells it requires a network of proteins that can sense external cues, send internal messages, and produce extracellular messengers that feed back information to neighbouring cells. Growth factors act as cellular messengers to feed back regulatory signals tha
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Liu, Shujuan, and Ahmed Ashour Ahmed. "Growth factors and uncontrolled proliferation." In Oxford Textbook of Oncology. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199656103.003.0002_update_001.

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A mandatory requirement of normal growth and development is the tight spatial and temporal synchronization of cellular proliferation within a specific tissue type and between tissue types that make up an organ, a process that is governed by intrinsic and extrinsic regulatory pathways. For a cell to synchronize its growth with that of other cells it requires a network of proteins that can sense external cues, send internal messages, and produce extracellular messengers that feed back information to neighbouring cells. Growth factors act as cellular messengers to feed back regulatory signals tha
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Baikampady, Savitri Vasudev, C. S. Hiremath, Reeta Varyani, and Venketesh. "Role of Vyana Vayu in CardioVascular System, Etiopathogenesis and Therapeutic Strategies: An Ayurveda Perspective." In Advancements in Cardiovascular Research and Therapeutics: Molecular and Nutraceutical Perspectives. BENTHAM SCIENCE PUBLISHERS, 2022. http://dx.doi.org/10.2174/9789815050837122010009.

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A systems approach to health is the hallmark of Ayurveda. It believes in preventing disease and maintaining and restoring health. The entire concept stands on three fundamental functional units-Vata, Pitta and Kapha, where Vata, mobilizes the other two units. Depending on their locations, Vata (Vayu) is classified into five subtypes, where each has its distinct role to perform. Vyana Vayu (VV), an important subtype of Vata, is synthesized in myocytes and responsible for the genesis of the action potential. A key regulator in contractile functions, VV propels out nutrients from the heart. It no
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Naylor, M. S., and F. R. Balkwill. "The role of cytokines in tumour progression." In Cell Proliferation in Cancer. Oxford University PressOxford, 1995. http://dx.doi.org/10.1093/oso/9780198547914.003.0005.

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Abstract Cytokines make up the fourth major class of soluble intercellular signalling mole cules alongside hormones, neurotransmitters, and prostaglandins/leukotrienes. They possess a number of common features despite the variety of molecular structures and properties. Cytokines are all polypeptides of low molecular weight (generally less than 80 kDa) that bind to high affinity cell surface receptors and cause changes in macromoleculer synthesis in target cells. Cytokines may act in a paracrine, autocrine (including intracellular autocrine loops), and juxtacrine manner (i.e., cytokines express
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Funder, John W. "Hormones and receptors: fundamental considerations." In Oxford Textbook of Endocrinology and Diabetes. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199235292.003.1022.

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The original endocrine physiologists viewed hormones as responses to homoeostatic challenge, any signal a call to arms; the word is thus derived from the classical Greek ωρμαειν‎—‘to arouse’. In the twenty-first century a hormone is a molecule—small or large, protein or lipid—secreted in a regulated fashion from one organ and acting on another. The definition is firmly based on the anatomy of the seventeenth century, the histology of the nineteenth, and the physiology of the twentieth. It has been shaped by convention and clinical specialization: gut hormones are the marches between endocrinol
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Declercq, W., M. Logghe, W. Fiers, and R. Contreras. "Cloning and expression of cytokine genes." In Cytokine Molecular Biology. Oxford University PressOxford, 2000. http://dx.doi.org/10.1093/oso/9780199638581.003.0001.

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Abstract Cytokines are proteins secreted into the extracellular fluid or are membrane exposed by a cell. There they exert their effects on the same cells (autocrine activity) or on neighbouring cells (paracrine activity) by interacting with specific receptors. Most cytokine proteins have, on a molar basis, very high biological activities (for instance, colony-stimulating factors are active at 10-11 to 10-13 M concentrations) which can usually be assayed in rapid, sensitive, and fairly specific in vitro test systems. These fast and simple detection systems have played a crucial role in cloning
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Conference papers on the topic "Paracrine activities"

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Pinzetta, Giulia, Allan Alcará, Isadora Ghilardi, Vitoria Pimentel, et al. "Modulation of the expression of the SLC12A2 gene that encodes the cationic co- transporter NKCC1 in epileptic animals treated with mesenchymal stem cells." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.689.

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Introduction: Temporal Lobe Epilepsy (TLE) can be identified by synchronized and rhythmic firing of neuronal populations that results in spontaneous and recurrent seizures in individuals affected by it1 . This type of epilepsy is clinically relevant because of its high incidence and refractoriness rate2,3. Thus, the search for therapeutic alternatives becomes important. Due to its benefits and less invasive administration, the mesenchymal stem cells (MSCs)4 appears as a possible therapeutic alternative, because can stimulate and provide a favorable niche for recovery based on their paracrine a
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