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Journal articles on the topic 'Paralysis, history'

Author: Grafiati

Published: 4 June 2021

Last updated: 29 July 2025

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1

Chalissery, Albi Jose, Tudor Munteanu, Yvonne Langan, Francesca Brett, and Janice Redmond. "Diverse phenotype of hypokalaemic periodic paralysis within a family." Practical Neurology 18, no. 1 (2017): 60–65. http://dx.doi.org/10.1136/practneurol-2017-001677.

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Hypokalaemic periodic paralysis typically presents with intermittent mild-to-moderate weakness lasting hours to days. We report a case with an uncommon phenotype of late-onset myopathy without episodic paralytic attacks. Initial work-up including muscle biopsy was inconclusive. A subsequent review of the right deltoid biopsy, long exercise testing and repeated family history was helpful, followed by appropriate genetic testing. We identified a heterozygous pathogenic mutation in calcium ion channel (CACNA1S:c.1583G>A p.Arg528His) causing hypokalaemic periodic paralysis. Myopathy can present

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2

Kim, Youn Kyoung, and Sa-Yoon Kang. "Hyperkalemic Periodic Paralysis Caused by a Mutation in the Sodium Channel SCN4A Gene." Journal of Medicine and Life Science 11, no. 1 (2014): 5–7. http://dx.doi.org/10.22730/jmls.2014.11.1.5.

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Hyperkalemic periodic paralysis (HyperPP) is an autosomal dominant muscle sodium channelopathy characterized by recurrentepisode of reversible paralysis with concomitant hyperkalemia. The diagnosis of HyperPP is suggested by a history of attacks ofparalysis, positive family history, and the presence of myotonia. A 19-year-old man presented with recurrent generalized limbweakness since childhood. The paralysis often followed by fatigue, exercise, and fasting. His parents were clinically unaffectedand had never experienced paralytic symptoms. Electromyographic evaluation demonstrated myotonic di

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3

Rabinovich, Itamar. "Paralysis in Lebanon." Current History 89, no. 544 (1990): 73–76. http://dx.doi.org/10.1525/curh.1990.89.544.73.

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4

Vale, José, Nuno Canas, Alberto Leal, and Helena Vilar. "Paralisia periódica tireotóxica." Acta Médica Portuguesa 18, no. 5 (2012): 399–402. http://dx.doi.org/10.20344/amp.1056.

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Thyrotoxic periodic paralysis (TPP) is characterized by recurrent episodes of muscle weakness and hypokalemia associated with thyroid hyperactivity. Reported predominantly among individuals of Asian descent, TPP is a rare manifestation of hyperthyroidism in western populations. We describe a typical case of TPP in a 33-year-old portuguese male, who presented with acute tetraparesis associated with hypokalemia (K+ 2.6 mEq/L). The patient had a 8-week history of several episodes of paraparesis of variable intensity; these attacks were usually nocturnal, preceded by strenuous exercise and resolve

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5

Shreya, Kumar Nabin, Ahmad Ansari Fayaz, and Kumar Jha Anand. "Abstract Aim: To determine the impact of Botulinum Toxin on Cervical Dystonia and its influence on Quality of Life. Materials and Methods: This study was conducted in the Department of physical medicine and rehabilitation, ESIC Medical College, Bihta, Patna, Bihar, India. Male and female patients aged between 18 and 80 years who fulfilled the diagnostic criteria for primary CD, had good consciousness and comprehension of the HRQoL questionnaires, and could communicate in and understand Thai language were included in the study. All the patients were included and completed the study. To assess the disease-specific HRQoL, this study used the CDIP58 and CDQ-24. This study employed the SF-36 and the EQ-5D to assess patients' general HRQoL. Results: Mean duration of symptoms in enrolled patients was 5.0 years (range from 0 to 12 years). There were 16 cases of torticollis, two of laterocollis, one of anterocollis, and one of latrocollis included in the study. The previous mean number of botulinum toxin A injections received per patient was 8.85±5.32 injections, ranging from 1 to 22. After 24 weeks of treatment, the mean (SD) score of all eight domains of CDIP-58 including head and neck symptoms, pain and discomfort, upper limb activities, walking, sleep, annoyance, mood, and psychosocial functioning were significantly improved (P<0.001) (Table 2). The mean (SD) of each of the five categories of CDQ-24 was also significantly improved with P-values of 0.004, 0.001, 0.003, 0.001, and 0.0005 for stigma, emotional well-being, pain, activities of daily living, and social and family life, respectively (Table 2). After 24 weeks of treatment, the mean values of results of all eight domains of SF-36 did not significantly improved. The P-values for PF, RP, RE, VT, mental health, SF, BP, and GH are 0.726, 1.000, 0.505, 0.349, 0.115, 0.171, 0.427, and 0.380 respectively. The mean score of each category of EQ-5D including mobility, self-care, usual activities, pain/discomfort, and anxiety/depression revealed a nonsignificant change (P>0.05). Conclusion: In conclusion, the quality of life of CD patients improved after multiple botulinum toxin A injections. Each injection of botulinum toxin A seems to have cumulative effects in multiple-injection patients. The direct mechanism behind an improvement in physical health is well understood; however, that behind mental health is less obvious. A relief of dystonia could make a positive difference to patients' relaxation state and stress level, which would account for mental improvement." International Journal of Current Pharmaceutical Review and Research 15, no. 11 (2023): 790–94. https://doi.org/10.5281/zenodo.13234062.

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AbstractAim: To determine the etiological spectrum of hypokalaemia periodic paralysis.Material and methods: This study was conducted in the department of Medicine, JNKTMCH, Madhepura, Bihar,India for one year. All patients admitted in the neurology ward with acute flaccid paralysis with hypokalaemia(serum potassium <3.5) involving two or more limbs without sphincter and sensory disturbances were includedin the study. Medical history, previous episodes of similar weakness, hyperthyroidism, diarrhoea, vomiting, renaldisease, and drug intake were noted. Any history of similar illness in the fa

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6

Binder, Devin K. "A History of Todd and His Paralysis." Neurosurgery 54, no. 2 (2004): 480–87. http://dx.doi.org/10.1227/01.neu.0000103490.49537.37.

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7

Cabrera-Gómez, J. A., N. Echazabal-Santana, Y. Real-González, et al. "Hereditary Melkersson-Rosenthal syndrome and multiple sclerosis." Multiple Sclerosis Journal 11, no. 3 (2005): 364–66. http://dx.doi.org/10.1191/1352458505ms1164cr.

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The revision of MEDLINE from 1966 to 2003 did not report any association between multiple sclerosis (MS) and Melkersson—Rosenthal syndrome (MRS). This is a case report of a 51-year-old woman, with history of four recurrent Bell’s palsies. In 1999 she developed a right facial paralysis due to a supranuclear pyramidal lesion with right monoparesis. The family history showed five relatives with recurrent Bell’s paralysis and plicata tongue. Physical examination: right Bell’s paralysis, left supranuclear facial paralysis, furrowed tongue, right hemiparesis with pallor of the optic disks. Brain mag

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8

Kesavan T M, Ananda, Sreejith Kumar K C, and Sreya Raghunath. "Hypokalemic Peroidic Paralysis: The Unusual Ones in Pediatrics." Indian Journal of Trauma and Emergency Pediatrics 14, no. 1 (2022): 25–27. http://dx.doi.org/10.21088/ijtep.2348.9987.14122.3.

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Hypokalaemic periodic paralysis is an uncommon but potentially life threatening clinical syndrome. It should consider in any child with recurrent motor paralysis. A positive family history with a low potassium level during attacks, will confirm the diagnosis If recognized and treated appropriately, patients recover without any clinical squeal. No extensive investigations are needed. KCL, acetazolamide, spiranolactone and lifestyle modificationswill help them to bring to normal activity.

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9

Metri, Shrikanth, and Srinivas B. "PERIODIC PARALYSIS IN THYROTOXICOSIS." International Journal of Advanced Research 10, no. 04 (2022): 589–91. http://dx.doi.org/10.21474/ijar01/14587.

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Thyrotoxic periodic paralysis (TPP) is one of the rare manifestation thyrotoxicosis. In this case report, we present a 36-year-old man, who had lower limb weakness for one day on waking up.He has no family history of thyroid or neurological problems.Any cause of thyrotoxicosis maybe sufficient to trigger attacks in susceptible patients. In order to permanently repair the problem, the underlying hyperthyroid status must be rectified.

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10

Borg, Tiffanie-Marie, Amy Sarah Warwick, and Mas Ahmed. "The child with facial nerve palsy." InnovAiT: Education and inspiration for general practice 14, no. 12 (2021): 742–48. http://dx.doi.org/10.1177/17557380211046283.

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In this article we summarise the educational aspects regarding the clinical presentation, diagnosis and management of children presenting with facial nerve paralysis, highlighting the importance of family history. We report two cases of hereditary facial nerve paralysis managed by the authors: one a child with familial facial nerve palsy, and the other a child with Melkersson–Rosenthal syndrome. The history in both cases revealed multiple family members with the same disorder.

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11

Chiwome, Lawman, Daniel Yidana, and Sai Man Mung. "Hyperkalemic Paralysis Due to Addison’s Disease." Journal of the Endocrine Society 5, Supplement_1 (2021): A135. http://dx.doi.org/10.1210/jendso/bvab048.272.

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Abstract A 60-year-old woman with type 1 diabetes mellitus, past history of breast cancer, degenerative disc disease, hypothyroidism and asthma gave a 5-day history of flaccid paralysis and a 5-month history of back pain and worsening mobility. Investigations revealed hypercalcemia, hyperkalemia and hyponatremia which were initially thought to be due to hypercalcemia of malignancy on the assumption that her breast cancer had recurred. Imaging of the spine did not reveal cauda equina syndrome. There was no clinical and biochemical improvement in spite of fluid resuscitation. Hypoadrenalism was

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12

Widjajanti, Anik, and S. M. Agustini. "HIPOKALEMIK PERIODIK PARALISIS." INDONESIAN JOURNAL OF CLINICAL PATHOLOGY AND MEDICAL LABORATORY 12, no. 1 (2018): 19. http://dx.doi.org/10.24293/ijcpml.v12i1.836.

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A 7-year-old boy of Java origin, visited the doctor with progressive weakness everytime he ate food containing Monosodium Glutamat (MSG). Progressive weakness began from his legs and spreaded to the arms as well (sometimes also to the neck). He could neither walk nor do anything for 5–6 hours then began to resolve spontaneously.. We suspected the diagnosis of hypokalaemic periodic paralysis upon the history of episodes of flacid paralysis and low serum concentration of potassium (< 3,5 mmol/L) during the attacts. The clinical examination showed that this boy is in good general and nutrition

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13

Mitra, Manasij, Nupur Biswas, Kumar Shailendra, Anil Chandra Jain, and Maitraye Basu. "A UNIQUE CASE OF BILATERAL VOCAL FOLD PARALYSIS FOLLOWING SPINAL ANAESTHESIA." EUREKA: Health Sciences 3 (May 31, 2020): 3–5. http://dx.doi.org/10.21303/2504-5679.2020.001315.

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Cranial nerve palsies are potential but rare complications of spinal anaesthesia. Most of the literatures support upper cranial nerve palsies like VI, IV and III cranial nerve palsies. Intrathecal hypotension resulting in tractional injury of the cranial nerves is the likely mechanism of injury. As on date, some cases of unilateral vocal fold paralysis and very little bilateral vocal fold paralysis have been described in case reports. We have described a patient who developed hoarseness and dysphagia 7 days after receiving spinal anaesthesia for fixation of inter-trochanteric fracture femur. T

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14

Manasij, Mitra, Biswas Nupur, Shailendra Kumar, Chandra Jain Anil, and Basu Maitraye. "A UNIQUE CASE OF BILATERAL VOCAL FOLD PARALYSIS FOLLOWING SPINAL ANAESTHESIA." EUREKA: Health Sciences, no. 3 (May 31, 2020): 3–5. https://doi.org/10.21303/2504-5679.2020.001315.

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Cranial nerve palsies are potential but rare complications of spinal anaesthesia. Most of the literatures support upper cranial nerve palsies like VI, IV and III cranial nerve palsies. Intrathecal hypotension resulting in tractional injury of the cranial nerves is the likely mechanism of injury. As on date, some cases of unilateral vocal fold paralysis and very little bilateral vocal fold paralysis have been described in case reports. We have described a patient who developed hoarseness and dysphagia 7 days after receiving spinal anaesthesia for fixation of inter-trochanteric fracture femur. T

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15

Agbotsou, Komi Igneza, Damelan Kombate, Christopher Mehri, et al. "Carotid Dissection and Isolated Paralysis of Ipsilateral Half Tongue: Clinical Cases." Case Reports in Neurological Medicine 2020 (December 17, 2020): 1–6. http://dx.doi.org/10.1155/2020/8853206.

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Objective. We report two cases of carotid dissection revealed by isolated paralysis of the ipsilateral half tongue. Observations. First patient, 52 years old, with no particular medical or surgical history, presented with isolated paralysis of the left half tongue preceded by two weeks of moderate-intensity cervicalgia and having been the subject to cervical manipulation. MRI revealed dissection of the left internal carotid artery in its prepetrous portion. The evolution after 6 weeks of platelet aggregating inhibitors treatment was favorable. The second patient, 74 years old, with no particul

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16

Bocklud, Brooke, and Gauri Mankekar. "Stylohyoid fracture resulting in facial paralysis." BMJ Case Reports 16, no. 7 (2023): e254938. http://dx.doi.org/10.1136/bcr-2023-254938.

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A female patient in her early 30s was referred to the otology clinic for progressive left facial paralysis. Two weeks prior to the onset of the paralysis, she had heard a loud cracking sound when drinking water. A CT scan showed a left styloid fracture near the stylomastoid foramen, compressing the facial nerve. Varicella IgG and IgM were negative, but the patient was started on a tapering course of steroids and valacyclovir. Approximately 1 month after the original incident, her left facial nerve paralysis improved. This report describes the unusual cause of facial paralysis and discusses the

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17

Jackson, Ian, Yazan Addasi, Moeed Ahmed, et al. "Hypokalemic Periodic Paralysis Precipitated by Thyrotoxicosis and Renal Tubular Acidosis." Case Reports in Endocrinology 2021 (June 21, 2021): 1–4. http://dx.doi.org/10.1155/2021/4529009.

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Background. Hypokalemic periodic paralysis is a rare neuromuscular disorder characterized by transient episodes of flaccid paralysis due to a defect in muscle ion channels. Most cases are hereditary, but it can be acquired. We present a case of acquired hypokalemic periodic paralysis associated with hyperthyroidism and renal tubular acidosis. Clinical Case. A 38-year-old female with a history of Graves’ disease presented to the emergency department with generalized weakness and associated nausea, vomiting, and weight loss. Examination was significant for diffuse weakness in all extremities. La

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18

Singleton, Neal, Matthew Bowman, and David Bartle. "Resolution of Right Hemidiaphragm Paralysis following Cervical Foraminotomies." Case Reports in Orthopedics 2018 (2018): 1–5. http://dx.doi.org/10.1155/2018/6195179.

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Introduction. Hemidiaphragm paralysis secondary to phrenic nerve palsy is a well-recognised medical condition. There are few case reports in the literature documenting resolution of hemidiaphragm paralysis following cervical spine surgery. This case report documents our experience with one such case. Case Presentation. A 64-year-old man was referred to the orthopaedic service with right hemidiaphragm paralysis. He had a previous history of asbestos exposure and polio and was initially seen and investigated by the respiratory physicians. He also reported intermittent neck pain and an MRI scan s

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19

Zbar, Ross I. S., and Richard J. H. Smith. "Vocal fold Paralysis in Infants Twelve Months of Age and Younger." Otolaryngology–Head and Neck Surgery 114, no. 1 (1996): 18–21. http://dx.doi.org/10.1016/s0194-59989670277-2.

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Seventeen cases of unilateral or bilateral vocal fold paralysis were diagnosed in infants younger than 12 months from 1991 to 1994 at the University of Iowa Hospitals and Clinics. Eight (47%) children with left vocal fold paralysis had a history of prior thoracic surgery –- two to repair complex congenital anomalies and six to ligate a patent ductus arteriosus. During the study period, a total of 81 patent ductus arteriosus ligations were performed, yielding a 7.4% postoperative incidence of vocal fold paralysis. Seven (41%) children had idiopathic vocal fold paralysis (3 right, 1 left, 3 bila

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20

Karosi, T., T. Rácz, É. Szekanecz, Á. Tóth, and I. Sziklai. "Recurrent laryngeal nerve paralysis due to subclinical Lyme borreliosis." Journal of Laryngology & Otology 124, no. 3 (2009): 336–38. http://dx.doi.org/10.1017/s0022215109990867.

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AbstractObjective:We report an extremely rare case of recurrent laryngeal nerve paralysis due to subclinical Lyme borreliosis.Method:Case report presenting a 15-year-old girl referred with hoarseness and soft voice.Results:Right-sided recurrent laryngeal nerve paralysis was observed using videolaryngoscopy. Imaging was used to exclude intracranial, cervical and intrathoracic embryological lesions, vascular malformations and tumours. Laboratory and electrophysiological investigations were used to exclude inflammatory and paraneoplastic processes, endocrinopathy and metabolic disorders. Serologi

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21

Muhammad, Zeeshan Dr Faiza Fazal Dr. Rimza Abid. "CEREBRAL PALSY PRESENTING AS RECURRENT PNEUMONIA." INDO AMERICAN JOURNAL OF PHARMACEUTICAL SCIENCES o6, no. 08 (2019): 15108–14. https://doi.org/10.5281/zenodo.3375184.

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<strong><em>Background:</em></strong><em> Cerebral paralysis is a typical reason for youth bleakness and mortality especially in creating nations like Pakistan. Despite the fact that it generally introduces as gross engine formative postponement, nonetheless, one of its normal yet under the announced method of introduction is intermittent pneumonia.</em> <strong><em>Objective: </em></strong><em>To determine the clinical and radiological profile of children with cerebral palsy presenting as recurrent pneumonia.</em> <strong><em>Patient & Methods:</em></strong><em> This research was carried

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22

Carvalho, Ana Catarina, Joana Matos, Susana Lira, and Ana Reis. "Parésia Hipocaliemica em Adolescente: Um Caso Clínico." Acta Médica Portuguesa 32, no. 5 (2019): 400. http://dx.doi.org/10.20344/amp.10497.

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Familial hypokalaemic periodic paralysis is a rare autosomal dominant neuromuscular disease characterized by episodic attacks of flaccid paralysis with concomitant hypokalaemia. We present a case of a 15-year-old male adolescent observed in the pediatric emergency department by flaccid paresis of the 4 limbs of sudden onset and progressive worsening. In the anamnesis, corticosteroid and antihistamine intake were observed on the previous day for urticaria and family history of transient episodes of flaccid paralysis in adolescence, asymptomatic after the fourth decade of life, without an establ

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23

DEVRIESE, P. P., and W. H. MOESKER. "THE NATURAL HISTORY OF FACIAL PARALYSIS IN HERPES ZOSTER." Clinical Otolaryngology 13, no. 4 (1988): 289–98. http://dx.doi.org/10.1111/j.1365-2273.1988.tb01134.x.

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24

Muthanna, Ali Obaid. "Unilateral Vocal Fold Paralysis in Adults: Etiological Factors in Yemen." مجلة جامعة صنعاء للطب والعلوم الصحية 18, no. 1 (2024): 35–40. http://dx.doi.org/10.59628/jchm.v18i1.728.

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Background: Unilateral vocal fold paralysis is common finding in otolaryngology practice. It is not a diagnosis by itself. The exact incidence of unilateral vocal fold paralysis has been difficult to find out because of multiple reasons. Various etiologies known to cause this condition are neck surgery, cancer, neck trauma, and neurological disorders. Objective: The aim of this study was to determine the possible etiologies of unilateral vocal fold paralysis in adult patients. Methods: A hospital-based study was conducted at OtoLaryngology Department, Al-Thawra Teaching Hospital, Sana'a, Yemen

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25

COCKS, JOAN. "From Politics to Paralysis." Political Theory 24, no. 3 (1996): 518–37. http://dx.doi.org/10.1177/0090591796024003010.

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26

Poduval, Jayita. "Percutaneous facial nerve stimulation." Journal of Medical Research 1, no. 2 (2015): 68–69. http://dx.doi.org/10.31254/jmr.2015.1209.

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Introduction: Facial nerve paralysis is a commonly encountered clinical entity. Bell’s palsy is the most frequently observed manifestation of facial paralysis, and is diagnosed and treated mainly on the basis of history and clinical examination. Some other cases of facial paralysis would however require more objective, intensive and reliable means of evaluation in order to facilitate optimum management. Method: When the diagnosis of facial paralysis is in doubt, electrodiagnostic tests are a useful method of evaluation. This paper discusses the technique by which these tests are performed usin

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Oishi, Tatsuya, Jennifer Pagano, Cody Sellers, and Nivedita U. Jerath. "Two Cases of Periodic Paralysis Associated With MCM3AP Variants." Journal of Clinical Neuromuscular Disease 25, no. 1 (2023): 36–41. http://dx.doi.org/10.1097/cnd.0000000000000454.

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Abstract Objectives: Periodic paralysis is a rare genetic condition characterized by episodes of neuromuscular weakness, often provoked by electrolyte abnormalities, physiologic stress, physical exertion, and diet. In addition to mutations in genes coding for skeletal muscle ion channels, in 2019, Gustavasson et al discovered that the MCM3AP gene could be responsible for periodic paralysis. In this study, we present 2 individuals with clinical episodes of periodic paralysis who have variants in the MCM3AP gene. Methods: Two unrelated probands were independently evaluated with clinical, genetic

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Pokalkar, Apeksha. "Monsoon fasiculation paralysis syndrome: revisited." International Journal of Research in Medical Sciences 13, no. 2 (2025): 885–87. https://doi.org/10.18203/2320-6012.ijrms20250275.

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Cases of sudden onset external ophthalmoplegia with bulbar and skeletal muscle weakness and fasciculations are noted in this region during the monsoon season. The neurological deficit peaks within 2-3 days with potential risk of respiratory paralysis. Total recovery takes place recovery within 4-8 days if respiratory crises is taken care of. Two such cases who presented with acute onset of flaccid quadriplegia with relative preservation deep tendon jerks are reported here. There was no history of any bites or exposure to known toxins. The patients made spontaneous recovery in one week time.

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Holm-Yildiz, Sonja, Nanna Witting, Julia Dahlqvist, et al. "Permanent muscle weakness in hypokalemic periodic paralysis." Neurology 95, no. 4 (2020): e342-e352. http://dx.doi.org/10.1212/wnl.0000000000009828.

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ObjectiveTo map the phenotypic spectrum in 55 individuals with mutations in CACNA1S known to cause hypokalemic periodic paralysis (HypoPP) using medical history, muscle strength testing, and muscle MRI.MethodsAdults with a mutation in CACNA1S known to cause HypoPP were included. Medical history was obtained. Muscle strength and MRI assessments were performed.ResultsFifty-five persons were included. Three patients presented with permanent muscle weakness and never attacks of paralysis. Seventeen patients presented with a mixed phenotype of periodic paralysis and permanent weakness. Thirty-one p

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Bawa, Rohit, Hassan H. Ramadan, and Stephen J. Wetmore. "Bilateral Vocal Cord Paralysis with Shy-Drager Syndrome." Otolaryngology–Head and Neck Surgery 109, no. 5 (1993): 911–14. http://dx.doi.org/10.1177/019459989310900521.

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Shy-Drager syndrome consists of progressive autonomic nervous system failure with Parkinson's disease-like symptoms and orthostatic hypotension. It can also result in airway compromise from bilateral vocal cord paralysis. Fewer than 30 cases of severe bilateral vocal cord paresis or paralysis associated with the Shy-Drager syndrome have been reported in the English literature. We present a case of a 72-year-old man who had a 2-year history of orthostatic hypotension, neurogenic bladder, impotence, anhydrosis, and extremity weakness and paresthesias. Hoarseness and dyspnea with stridor develope

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Reddy Bendaram, Snigdha, Misbah Azmath, Pooja Luthra, and Latha Dulipsingh. "ODP614 Two cases of Graves Disease Associated Hypokalemic Periodic Paralysis." Journal of the Endocrine Society 6, Supplement_1 (2022): A794. http://dx.doi.org/10.1210/jendso/bvac150.1643.

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Abstract Introduction Periodic paralysis (PP) is a rare neuromuscular disorder due to a defect in ion channels in muscle, characterized by episodes of painless muscle weakness or paralysis, which may be precipitated by heavy exercise or high-carbohydrate meals. Hypokalemic PP (HPP) is most common of the periodic paralyses, however is still very rare, with an estimated prevalence of 1 in 100,000. An acquired form of hypokalemic PP can be seen in thyrotoxicosis of any cause. We present 2 cases of HPP due to thyrotoxicosis. Case 1 27 year old Asian male presented to the ED due to sudden onset of

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Shabbir, Sana, Arisha Sana Memon, Gulshad Wagan, Sadia Effendi, Fahmida Gul, and Kanwal Abbas Bhatti. "Clinical Study on the Causes and Outcome of Facial Nerve Palsy at Liaquat University Hospital Hyderabad." Pakistan Journal of Medical and Health Sciences 16, no. 11 (2022): 639–41. http://dx.doi.org/10.53350/pjmhs20221611639.

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Background and Aim: A facial paralysis can be enormously distressing for a person. In order to restore and rehab face symmetry, it is essential to understand the source and type of nerve damage. Lower motor neuron facial paralysis was investigated in the current study to assess its various causes. Patients and Method: This prospective study was carried out on 48 facial nerve palsy (FNP) in the Department of Medicine, Liaquat University Hospital, Hyderabad from January 2021 to August 2022. Patients diagnosed with peripheral facial nerve paralysis of any age and both genders were enrolled. Patie

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Anderson, Karolina, and Melissa Wellons. "ODP525 Thyrotoxic Periodic Paralysis." Journal of the Endocrine Society 6, Supplement_1 (2022): A785—A786. http://dx.doi.org/10.1210/jendso/bvac150.1624.

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Abstract Thyrotoxic periodic paralysis manifests with painless muscle weakness, mostly in the proximal lower extremities. It is often precipitated byexercise, high carbohydrate meals, and occasionally fasting. Though most cases are hereditary, and affect Asian men disproportionately, some cases may occur sporadically. This thyrotoxicosis is most often attributable to Graves disease and patients typically have symptoms of thyrotoxicosis for months leading up to the episodes of paralysis. This is an 18-year-old Asian man with a history of Autism Spectrum Disorder who presents for evaluation of l

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Klyachkin, G. A. "Case of Periodic Oculomotor Nerve Palsy." Neurology Bulletin V, no. 1 (2020): 107–14. http://dx.doi.org/10.17816/nb46644.

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Periodically recurring paralysis of the oculomotor nerve (migraine ophthalmoplegique French authors) is not often observed. In foreign literature, I managed to collect 24 cases, and in Russian literature, as far as I know, there is only one observation by prof. L.O.Darkshevich. In view of this, I think it is interesting to give a brief history of the illness of one patient, in whom, during the last day, I observed periodic paralysis of the oculomotor nerve

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Imashuku, Shinsaku, Tomoko Teramura-Ikeda, Naoko Kudo, Shigehiro Kaneda, and Toshihiro Tajima. "Concurrence of thyrotoxicosis and Gitelman’s syndrome-associated hypokalemia-induced periodic paralysis." Pediatric Reports 4, no. 2 (2012): 18. http://dx.doi.org/10.4081/pr.2012.e18.

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A 16-year-old Japanese boy with a history of truancy had been treated at a psychiatric clinic. When the patient was referred to us for hypokalemia-associated paralysis, the diagnosis of thyrotoxic hypokalemic periodic paralysis was made, common in Asian men. Subsequently, the patient was found to have persistently high plasma renin and aldosterone levels. Thus, solute carrier family 12 member 3 gene (<em>SLC12A3</em>) analysis was performed. A novel missense homozygous mutation CTC-&gt;CAC at codon 858 (L858H) was found for which the patient was homozygous and his non-consangui

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Chung, D. Y., and J. Hardman. "Prolonged Paralysis following Mivacurium Administration." Anaesthesia and Intensive Care 30, no. 3 (2002): 360–63. http://dx.doi.org/10.1177/0310057x0203000316.

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Mivacurium is a benzylisoquinolone, choline-like, non-depolarizing muscle relaxant. Its onset of action is similar to that of atracurium but its duration of action is shorter (approximately 10–15 minutes). Mivacurium is metabolized by plasma cholinesterases at approximately 70% of the rate of metabolism of suxamethonium1. Deficiency or abnormality of plasma cholinesterase may cause the duration of action of both suxamethonium2,3 and mivacurium2,4,5,6 to be greatly prolonged. We describe a case of prolonged mivacurium paralysis after day surgery. Laboratory investigations showed a genetic tende

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Gaitan-Quintero, Gustavo, Loida Camargo-Camargo, Norman López-Velásquez, and Miguel González. "Presentation of Bilateral Facial Paralysis in Melkersson–Rosenthal Syndrome." Case Reports in Neurological Medicine 2021 (January 6, 2021): 1–4. http://dx.doi.org/10.1155/2021/6646115.

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Introduction. Melkersson–Rosenthal syndrome (MRS) is a neuromucocutaneous disorder characterized by the following classic symptom triad: peripheral facial paralysis, orofacial edema, and scrotal or fissured tongue. It is rare, and since most of the patients are oligo- or monosymptomatic, it makes it difficult to diagnose. Clinical Case. We present a 26-year-old male patient with a history of sickle cell trait, untreated snoring, and left peripheral facial paralysis when he was 11 years old. This was an overall 20-day clinical profile that started with left peripheral facial paralysis, which wa

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Mitrovic, Slobodan, Gordana Mumovic, Rajko Jovic, and Vladimir Kljajic. "Unilateral laryngeal paralysis." Medical review 56, no. 1-2 (2003): 59–62. http://dx.doi.org/10.2298/mpns0302059m.

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Introduction Phoniatric rehabilitation is mainly aimed at restoring satisfactory phonation. Voice quality depends on the capacity of intact vocal cords to compensate the deficiency involved, as well as on automatism of phonation. Material and methods The study included 50 patients. All subjects underwent history taking, reported symptoms that urged them to visit a phoniatrician; they were submitted to a clinical otorhinolaryngologic and phoniatric examinations, voice assessment by subjective acoustic analysis spectral analysis by digital sonography and laryngostroboscopy. All patients underwen

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Boon, Tay Seng, Tan Yen Yun, and Gerard Jason Mathews. "A CASE REPORT OF THYROTOXIC PERIODIC PARALYSIS." Journal of the ASEAN Federation of Endocrine Societies 40, S1 (2025): 48. https://doi.org/10.15605/jafes.040.s1.078.

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INTRODUCTION/BACKGROUND Thyrotoxic periodic paralysis (TPP) is a potentially life-threatening clinical manifestation of thyrotoxicosis predominantly seen in those of Asian descent between the ages of 20 and 40 years. The attack is characterised by acute and reversible severe muscle weakness with hypokalemia that resolves with the treatment of hyperthyroidism. CASE A 22-year-old Chinese male with no previous medical illness presented to the emergency department with sudden onset bilateral lower limb weakness associated with intermittent palpitations for the past month. Lower limbs neurological

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Shergill, Luvleen, Jasir Nayati, Reshma Nair, and Alan R. Hirsch. "43 Tinnitus as an Aura for Sleep Paralysis." CNS Spectrums 24, no. 1 (2019): 199. http://dx.doi.org/10.1017/s1092852919000361.

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AbstractObjectiveTo understand that tinnitus may be an aura for sleep paralysis.BackgroundSleep paralysis is a transient-paralysis which occurs during awakening or falling asleep (Wilson, 1928). Those affected experience symptoms including visual, auditory, and haptic hallucinations, voluntary motor paralysis with intact ocular and respiratory motor movements, and diffuse or localized paresthesias. Sleep paralysis associated with tinnitus as an aura, has not heretofore been described.MethodsA 34 year-old, right-handed female presented with a 13 year history of sleep paralysis. One month prior,

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Islam, AKM Monwarul, Sharadindu Shekhar Roy, Shahana Zaman, Farhana Ahmed, Muhammad Abdur Rahim, and Humayra Jesmin. "Hypokalemic Periodic Paralysis: Reports of Two Cases and Brief Review." BIRDEM Medical Journal 9, no. 1 (2019): 74–79. http://dx.doi.org/10.3329/birdem.v9i1.39729.

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Hypokalemic periodic paralysis (HPP) is a rare autosomal dominant channelopathy characterized by skeletal muscle weakness or paralysis when there is a fall in potassium levels in blood. Weakness may be mild and limited to certain muscle groups or more severe causing generalized paralysis. During an attack, reflexes may be diminished or absent. Attacks may last for a few hours or persist for several days, ultimately resulting in complete recovery. Some patients may develop chronic muscle weakness later in life. Recurrent muscle weakness accompanied by hypokalemia and exclusion of other causes h

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Varma-Doyle, Aditi Vian, Kristen Garvie, Seema Walvekar, Mae Igi, and Radha Mayuri Garikepati. "Ascending Paralysis in a 36-Year-Old Woman With Bipolar Disorder and Recent Aspiration Pneumonia." Journal of Investigative Medicine High Impact Case Reports 8 (January 2020): 232470962093164. http://dx.doi.org/10.1177/2324709620931649.

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Guillain-Barré syndrome (GBS) is an acute inflammatory demyelinating polyneuropathy affecting both motor and sensory peripheral nerves. Typically presenting after a gastrointestinal or a respiratory tract infection, it manifests as ascending paralysis with concomitant areflexia in patients. Cytoalbuminologic dissociation is a supportive finding on cerebrospinal fluid (CSF) analysis. Due to variability in presentation, misdiagnosis and delay in treatment can occur, and consequently, GBS can become life threatening due to respiratory failure. We report ascending paralysis in a 36-year-old woman

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Ding, Joy Zhuo, and Hugh J. McMillan. "“Owl’s Eye” Sign in Acute Flaccid Paralysis." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 46, no. 6 (2019): 756–57. http://dx.doi.org/10.1017/cjn.2019.250.

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A 4-year-old boy presented with asymmetric acute flaccid paralysis (AFP) of his right arm and both legs. He was alert with no oculobulbar weakness or incontinence. He had fever and diarrhea 5 days earlier. He was fully immunized with no travel history.

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Tyszkiewicz, Thomas, and Isam Atroshi. "Bilateral anterior interosseous nerve syndrome with 6-year interval." SAGE Open Medical Case Reports 6 (January 1, 2018): 2050313X1877741. http://dx.doi.org/10.1177/2050313x18777416.

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Flexor pollicis longus paralysis related to idiopathic anterior interosseous nerve syndrome is well known, but few reports exist on bilateral disease. A 24-year-old man with no personal or family history of neurological disease developed isolated total loss of active flexion of the right thumb’s interphalangeal joint after undergoing a wrist arthroscopy. Surgical exploration 5 weeks after onset showed flexor pollicis longus tendon to be intact; anterior interosseous nerve decompression was done with no abnormalities found. Because of persistent paralysis, electromyography was performed showing

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Saraniti, Carmelo, and Barbara Verro. "Reanimation Techniques of Peripheral Facial Paralysis: A Comprehensive Review Focusing on Surgical and Bioengineering Approaches." Journal of Clinical Medicine 13, no. 20 (2024): 6124. http://dx.doi.org/10.3390/jcm13206124.

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Peripheral facial paralysis represents a disabling condition with serious psychological and social impact. Patients with peripheral facial paralysis have a disfigurement of the face with loss of harmony and symmetry and difficulties in everyday facial functions such as speaking, drinking, laughing, and closing their eyes, with impairment of their quality of life. This paralysis leads to impairment of facial expression, which represents one of the first means of communication, an important aspect of human interaction. This review aims to explore the reanimation techniques for managing periphera

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Blessy, Rachal Boban, K. J. Cillamol, and Cheruvil |. Sheffin Thomas |. Tony Abraham Elena. "Hypokalemic Periodic Paralysis A Case Report." International Journal of Trend in Scientific Research and Development 3, no. 3 (2019): 216–17. https://doi.org/10.31142/ijtsrd21658.

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Hypokalemic periodic paralysis HPP is a medical emergency with prevalence of 1 in 100,000 . Rapid management is very important since, very low potassium levels can lead to cardiac complications . In this case, a twenty four year old female without a similar history in the family, having hypokalemia periodic paralysis attack is presented. This case report study has been presented for the consideration of the rare HPP in patients presenting with sudden muscle weakness. Blessy Rachal Boban | Cillamol K. J | Elena Cheruvil | Sheffin Thomas | Tony Abraham "Hypokalemic Periodic Paralysis: A Cas

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Shah, Urmil, Kumar Abhishek, Rushikesh Shukla, et al. "1043 Prevalence of Sleep Paralysis in Medical Students Across India- a Cross Sectional Study." SLEEP 47, Supplement_1 (2024): A448—A449. http://dx.doi.org/10.1093/sleep/zsae067.01043.

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Abstract Introduction Sleep paralysis is a condition that has been on the rise in recent years, occurring during the pre or post sleep stages. While there is no known direct cause of sleep paralysis, studies have shown a number of potential causes, including poor quality of sleep, alcohol use, exposure to stressful events, anxiety disorders, and a family history of the illness. The purpose of this study was to assess the prevalence of sleep paralysis among medical college students in India and identify factors associated with the condition. Methods A cross-sectional study involving 155 medical

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Mani, Utsav Anand, Mukesh Kumar, Haider Abbas, and Pranay Gupta. "Delayed Neurotoxic Paralysis with Confounding Hypokalaemia." Journal of the Association of Physicians of India 1, no. 1 (2023): 26–28. http://dx.doi.org/10.5005/njem-11015-0002.

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Snakebite is a major health issue in India and developing nations across the globe. Common krait bites are associated with delayed paralysis and locked-in syndrome. Locked-in syndrome has been documented among patients of snakebite. Our case is unique due to the confounding effect of hypokalaemia which may confuse the physician as hypokalaemic periodic paralysis (HPP), a rare genetic condition which can also be precipitated by a stressful event such as snakebite. To the best of our knowledge, we could not associate the presence of hypokalaemia in patients of neurotoxic snakebite. As krait bite

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King, Ericka F., Marike Zwienenberg-Lee, Steve Maturo, Peter Siao Tick Chong, Christopher Hartnick, and David J. Brown. "Natural history of vocal fold paralysis in Arnold-Chiari malformation." International Journal of Pediatric Otorhinolaryngology Extra 6, no. 4 (2011): 256–60. http://dx.doi.org/10.1016/j.pedex.2010.11.008.

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Kuruvilla, George, Shirley Perry, Beverly Wilson, and Hamdy El-Hakim. "The Natural History of Vincristine-Induced Laryngeal Paralysis in Children." Archives of Otolaryngology–Head & Neck Surgery 135, no. 1 (2009): 101. http://dx.doi.org/10.1001/archoto.2008.514.

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