Relevant bibliographies by topics / Paraneoplastic Syndromes/complications

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Academic literature on the topic 'Paraneoplastic Syndromes/complications'

Author: Grafiati
Published: 2 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "Paraneoplastic Syndromes/complications"

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Naqvi, Syed Mohammad, Hashim Talib Hashim, Syed Yaseen Naqvi, et al. "Small cell lung cancer progressing into fatal ascending motor and sensory polyneuropathy despite dramatic response to chemotherapy: A case report." Medicine 104, no. 1 (2025): e41172. https://doi.org/10.1097/md.0000000000041172.

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Rationale: Paraneoplastic syndromes occur in 10% to 20% of all malignancies, with paraneoplastic neurological syndromes in less than 1% of all malignancies. In small cell lung cancer (SCLC), paraneoplastic manifestations are more common, affecting up to 5% of cases. Common manifestations include syndrome of inappropriate antidiuretic hormone secretion, Cushing’s syndrome due to adrenocorticotropic hormone secretion, paraneoplastic cerebellar degeneration, and Lambert-Eaton myasthenic syndrome. Paraneoplastic acute motor and sensory polyneuropathy is an exceptionally rare but severe complicatio
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Maheshwari Nallur Siddaraju, Shivani B. Sonan, Kadeeja K S, et al. "Paraneoplastic Syndrome Associated Immune Complications: A Narrative Review of the Literature." Journal of Medical and Health Studies 5, no. 1 (2024): 10–16. http://dx.doi.org/10.32996/jmhs.2024.5.1.2.

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Paraneoplastic syndromes occur in cancer patients due to an alteration in their immune response. There are several factors that may result in the occurrence of paraneoplastic syndrome, including the presence of abnormal cytokines that cause widespread effects throughout the body. The paraneoplastic syndrome may be present in patients before a diagnosis of cancer, and thus, understanding it is crucial as it will help achieve a timely diagnosis, which may aid in improving the chance of treatment. It is associated with several complications/presentations in patients. In this review, we will discu
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Zekeridou, Anastasia. "Paraneoplastic Neurologic Disorders." CONTINUUM: Lifelong Learning in Neurology 30, no. 4 (2024): 1021–51. http://dx.doi.org/10.1212/con.0000000000001449.

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ABSTRACT OBJECTIVE This article reviews the clinical presentations, neural antibody associations, and oncologic accompaniments of paraneoplastic neurologic syndromes and neurologic autoimmunity in the context of immune checkpoint inhibitor (ICI) cancer immunotherapy. LATEST DEVELOPMENTS Neural antibody discovery has improved the diagnosis of paraneoplastic neurologic syndromes. Neural antibodies also delineate the underlying disease pathophysiology and thus inform outcomes and treatments. Neural antibodies specific for extracellular proteins have pathogenic potential, whereas antibodies specif
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Körber, Andreas. "Paraneoplastische Syndrome in der Dermatologie - eine Übersicht." Kompass Dermatologie 6, no. 4 (2018): 200–201. http://dx.doi.org/10.1159/000492631.

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A number of paraneoplastic syndromes have been described with gynecological cancers. These syndromes are induced by substances secreted by the tumor or by an immune response triggered by the cancer. Each system of the human body can be affected by different syndromes. Indeed, paraneoplastic syndromes occurring from tumors of the gynecologic tract were found to involve the nervous, ophthalmologic, dermatologic, rheumatologic, endocrine, hematologic and renal systems. These syndromes can manifest before, at the time, or after the diagnosis of cancer. They can also occur at the time of a recurren
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Hsu, Hui-Ching, Tzu-Yao Liao, Dave Wei-Chih Chen, Yu-Hsiang Juan, and Chuang-Chi Liaw. "Avascular Necrosis of Bone following Chemotherapy in Cancer Patients with Coagulopathy: Report of Two Cases." Case Reports in Oncology 11, no. 1 (2018): 185–90. http://dx.doi.org/10.1159/000488102.

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We report 2 cases of patients with solid tumors and coagulopathy who experienced avascular necrosis (AVN) of the bone following chemotherapy. Both cases exhibited nontraumatic bilateral AVN of the femoral heads, and one also showed bilateral AVN of the humeral heads. One case had multiple thromboembolic complications, including pulmonary obstructive syndrome and paraneoplastic pain. The other showed multiple paraneoplastic syndromes, with hypercalcemia and thrombocytosis. Groin pain and claudication of the lower extremities developed and persisted. Both patients eventually received bilateral h
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Amin, Anik, Enrique Chiu Han, Rumyar Ardakani, and Larry D. Anderson. "Unusual paraneoplastic neurological presentation associated with multiple myeloma." BMJ Case Reports 17, no. 12 (2024): e262469. https://doi.org/10.1136/bcr-2024-262469.

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Various neurological complications can occur in multiple myeloma. We report a unique paraneoplastic neurological presentation that led to a multiple myeloma diagnosis. A man in his mid-40s experienced a 6-month progressive decline in vision and gait. Examination demonstrated an upper motor neuron pattern, while MRI showed bilateral corticospinal tractopathy and optic neuropathy. Workup led to a diagnosis of multiple myeloma, while other causes (eg, primary central nervous system demyelination, toxic-metabolic, degenerative) were ruled out. Paraneoplastic antibody panels were negative. There wa
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SUTTON, Ian, and John B. WINER. "The immunopathogenesis of paraneoplastic neurological syndromes." Clinical Science 102, no. 5 (2002): 475–86. http://dx.doi.org/10.1042/cs1020475.

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Paraneoplastic neurological syndromes are rare non-metastatic complications of cancer that have an immune-mediated aetiology. The central and peripheral nervous systems are considered to be immune-privileged sites, since the presence of the ‘blood-brain/nerve barrier’ means that antigens sequestered within the nervous system do not normally induce an immune response. Aberrant expression of a neuronal antigen by a tumour arising outside this barrier can lead to the breakdown of immune tolerance to the nervous system. However, in many cases the immune mechanisms that result in neurological dysfu
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Mastropietro, Fabiola, Alessandra Piccini, Giulia Lucignani, Alfonso Rubino, and Giancarlo Fiermonte. "Atypical presentation of Non-Hodgkin Lymphoma (NHL): a case report." Clinical Management Issues 8, no. 4 (2014): 103–7. http://dx.doi.org/10.7175/cmi.v8i4.942.

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Lymphomas infrequently cause peripheral nerve complications. These syndromes mostly occur by direct compression or infiltration of nerves (neurolymphomatosis), but may also be due to a remote effect as paraneoplastic syndromes, neurotoxic complications of chemotherapy, antibody-mediated or autoimmune mechanisms.We report the case of a 60-year-old woman who presented with a complex peripheral nervous system involvement as initial manifestation of Non-Hodgkin Lymphoma (NHL). This case sheds light on “protean” mechanism of peripheral nerve complications during the course of NHL and related diagno
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Popławska-Domaszewicz, Karolina, Jolanta Florczak-Wyspiańska, Wojciech Kozubski, and Sławomir Michalak. "Paraneoplastic movement disorders." Reviews in the Neurosciences 29, no. 7 (2018): 745–55. http://dx.doi.org/10.1515/revneuro-2017-0081.

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Abstract Paraneoplastic movement disorders are rare, autoimmune-mediated, nonmetastatic complications of malignant neoplasms. Common paraneoplastic movement disorders include paraneoplastic chorea, dystonia, cerebellar degeneration, different types of encephalitis, opsoclonus-myoclonus syndrome, stiff person syndrome, and neuromyotonia. Syndromes usually develop before tumor diagnosis, have subacute onset, and are associated with serum or cerebrospinal fluid antibodies. Two types of antibodies can be distinguished: antibodies against nuclear and cytoplasmic neuronal antigens (anti-Hu, anti-Ri,
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Betcherman, Laura, and Angela Punnett. "Paraneoplastic Syndromes in Children with Hodgkin Lymphoma." Oncology & Hematology Review (US) 13, no. 01 (2017): 41. http://dx.doi.org/10.17925/ohr.2017.13.01.41.

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Paraneoplastic syndromes (PNS) refer to a phenomenon whereby certain malignancies manifest as symptoms not directly related to the tumor itself. PNS has been described in association with Hodgkin lymphoma (HL) in adults and children and may affect a number of organ systems. The pathophysiology is variable and in many cases is not well understood. Specific paraneoplastic antibodies have been isolated in some syndromes, though are not required for the diagnosis. The two best described for HL are the anti-Tr and anti-mGluR5 antibodies identified in some cases of limbic encephalitis and cerebellar
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Books on the topic "Paraneoplastic Syndromes/complications"

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Watson, Max, Caroline Lucas, Andrew Hoy, and Jo Wells. Endocrine and metabolic complications of advanced cancer. Oxford University Press, 2010. http://dx.doi.org/10.1093/med/9780199234356.003.0020.

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This chapter covers paraneoplastic syndromes (Hypercalcaemia, Cushing’s syndrome, syndrome of inappropriate antidiuresis, hypoglycaemia (non-islet cell), carcinoid syndrome) before turning to non-paraneoplastic complications, including diabetes mellitus.
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Bower, Mark, Louise Robinson, and Sarah Cox. Endocrine and metabolic complications of advanced cancer. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199656097.003.0142.

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Cancer produces endocrine and metabolic complications in two ways. Firstly, the primary tumour or its metastases may interfere with the function of endocrine glands, kidneys, or liver by invasion or obstruction. Secondly, tumours may give rise to remote effects without local spread and these actions are termed paraneoplastic manifestations of malignancy. Generally, these paraneoplastic syndromes arise from secretion by tumours of hormones, cytokines, and growth factors, but also occur when normal cells secrete products in response to the presence of tumour. This chapter reviews the pathogenesi
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Jordan, Nerissa. Non-metastatic neurological manifestations of malignancy. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0238.

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Neurological complications of systemic malignancy are frequent. They may reflect direct local effects of the tumour; CNS infection; side effects of chemotherapy or radiotherapy; nutritional or metabolic derangements; or a paraneoplastic syndrome. The paraneoplastic neurological syndromes are a group of disorders associated with a malignancy outside the nervous system. The pathophysiology is immune-mediated, with the tumour’s expression of neuronal proteins invoking antibody formation, which in turn results in neurological symptoms. This chapter will mainly focus on these syndromes.
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Internal malignancy and the skin: Paraneoplastic and cancer treatment-related cutaneous disorders. Saunders, 2008.

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Caraceni, Augusto, Cinzia Martini, and Fabio Simonetti. Neurological problems in advanced cancer. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199656097.003.0141.

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Neurological complications are frequent in populations with advanced cancer. An adequate neurological assessment is always important in addressing pain, cognitive symptoms, and peripheral and central nervous system complications. This chapter discusses a variety of neurological problems found in advanced cancer together with their clinical aspects and management, including some suggested regimens for pharmacological therapy. Complications that are discussed include intracranial hypertension, seizures in patients with advanced illness (including a suggested algorithm for the management of statu
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Book chapters on the topic "Paraneoplastic Syndromes/complications"

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Rosenfeld, Myrna R., and Josep Dalmau. "Paraneoplastic Syndromes of the Nervous System as Complications of Cancer." In Cancer Neurology in Clinical Practice. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-57901-6_13.

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Deangelis, Lisa M., and Jerome B. Posner. "Paraneoplastic Syndromes." In Neurologic Complications of Cancer. Oxford University Press, 2008. http://dx.doi.org/10.1093/med/9780195366747.003.0015.

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Rees, Jeremy. "Paraneoplastic neurological syndromes." In Oxford Textbook of Medicine, edited by Christopher Kennard. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198746690.003.0616.

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Paraneoplastic neurological syndromes are disorders caused by the presence of an underlying tumour, but not due to either direct or metastatic invasion, or to recognized metabolic or endocrine complications. They are thought to arise from an autoimmune response to ‘onconeural’ tumour antigens which are also expressed by cells of the central or peripheral nervous systems. Paraneoplastic neurological syndromes are rare but important because (1) they often develop before the cancer has been identified, (2) serological testing for specific antineuronal (onconeural) antibodies may identify a neurol
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Ryan, Richella, and Ruth Casey. "Endocrine and metabolic complications of advanced cancer." In Oxford Textbook of Palliative Medicine, edited by Nathan I. Cherny, Marie T. Fallon, Stein Kaasa, Russell K. Portenoy, and David C. Currow. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780198821328.003.0084.

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This chapter reviews the epidemiology, pathogenesis, clinical features, and management of endocrine and metabolic complications of advanced cancer. Broadly, these complications may arise in three ways due to (1) direct tumour invasion of organs or tissues; (2) non-metastatic systemic effects resulting from secretion of hormones, cytokines, or growth factors either by the tumour itself or by normal cells in response to tumour invasion (‘paraneoplastic effects’); and (3) treatments targeted at cancer or its symptoms. The chapter is divided into four sections. First, it describes the three most c
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Vernino, Dr Steven. "Paraneoplastic autonomic neuropathy and limbic encephalitis." In Fifty Neurologic Cases From Mayo Clinic. Oxford University PressNew York, NY, 2004. http://dx.doi.org/10.1093/oso/9780195177442.003.0009.

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Abstract Neurologic paraneoplastic syndromes are rare but severe neuroimmunologic complications of malignancy. The tumors most commonly associated with these syndromes are small cell lung carcinoma, ovarian carcinoma, breast carcinoma, and thymoma. At surgery, a 1.0 x 1.0 x 1.0-cm grade 3 (of 4) squamous cell carcinoma was identified. These syndromes usually have a subacute onset and a relentlessly progressive clinical course leading to profound morbidity. Typically, the neurologic presentation antedates the diagnosis of malignancy, and the cancer, when found, tends to be localized and respons
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Bansal, Varun, and Margaret M. Johnson. "Pulmonary Malignancy." In Mayo Clinic Critical and Neurocritical Care Board Review, 2nd ed., edited by Eelco F. M. Wijdicks, Rodrigo Cartin-Ceba, William D. Freeman, Alice Gallo de Moraes, and Richard A. Oeckler. Oxford University Press, 2025. https://doi.org/10.1093/med/9780197628638.003.0024.

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Abstract Non–small cell lung cancer accounts for 75% of primary lung cancers, and most cases are diagnosed at an advanced stage because symptoms are absent in the early stages of the disease. Paraneoplastic syndromes include hypercalcemia, Lambert-Eaton myasthenic syndrome, syndrome of inappropriate antidiuretic hormone secretion, Cushing syndrome, and superior vena cava syndrome. Metastatic disease in the thorax can lead to life-threatening complications from malignant pleural or pericardial effusion and lymphangitic cancer. Cardiopulmonary complications may result from various types of chemo
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Watson, Dr Max, Ann Barrett, Roy AJ Spence, and Chris Twelves. "Patients with lung cancer." In Oncology. Oxford University PressOxford, 2006. http://dx.doi.org/10.1093/oso/9780198567578.003.0008.

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Abstract Between 10 and 20% of patients may present with, or develop complications of, the non-metastatic paraneoplastic syndromes associated with lung cancer. These are particularly well documented for small cell carcinoma and large cell undifferentiated carcinoma, both of which tend to have many neuroendocrine features. However, they do occur to a lesser degree with squamous cell carcinoma and adenocarcinoma. Many of these syndromes are mediated by peptides, which mimic active portions of known hormones.
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Caraceni, Augusto, Fabio Simonetti, and Cinzia Martini. "Neurological problems in advanced cancer." In Oxford Textbook of Palliative Medicine, edited by Nathan I. Cherny, Marie T. Fallon, Stein Kaasa, Russell K. Portenoy, and David C. Currow. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780198821328.003.0083.

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Neurological complications are frequent in patients with advanced cancer. This chapter discusses a variety of neurological problems found in advanced cancer together with their clinical aspects and management, including some suggested regimens for pharmacological therapy. Complications discussed include intracranial hypertension, seizures in patients with advanced illness (including a suggested algorithm for the management of status epilepticus), delirium, brain metastases, spinal cord compression (including an algorithm for the evaluation of back pain), leptomeningeal metastases, base of the
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Wendler, Julia, Gerald Illerhaus, and Lakshmi Nayak. "Neurological complications of lymphoid malignancies." In Oxford Textbook of Neurohaematology, edited by Tracy Batchelor, Joshua P. Klein, Andrés José María Ferreri, and Lisa M. DeAngelis. Oxford University PressOxford, 2024. http://dx.doi.org/10.1093/med/9780198884903.003.0007.

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Abstract Lymphoid malignancies can involve both the central and peripheral nervous systems and may present in various ways. The central and peripheral nervous systems can be affected directly by primary central nervous system lymphoma or metastatic or local spread of systemic lymphoma. Indirect effects include spinal cord compression, paraneoplastic syndromes, and vascular involvement. Moreover, various treatment strategies for lymphoid malignancies can negatively affect the central and peripheral nervous systems. Early and adequate diagnosis of neurological complications can be challenging bu
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"14 Non-tumor complications and syndromesIt’s Not Just the Tumor: CNS Paraneoplastic Syndromes and Cerebrovascular Complications of Cancers." In Brain Tumor Imaging, edited by Rajan Jain and Marco Essig. Georg Thieme Verlag, 2016. http://dx.doi.org/10.1055/b-0035-122305.

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Journal articles
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