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Journal articles on the topic 'Paraparesie'

Author: Grafiati

Published: 4 June 2021

Last updated: 5 February 2022

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1

Aljaafari, Danah, and Noman Ishaque. "Paraparetic Variant of Guillain-Barré Syndrome in First 24 Hours of Postpartum Period: A case report." Sultan Qaboos University Medical Journal [SQUMJ] 20, no. 2 (June 28, 2020): 227. http://dx.doi.org/10.18295/squmj.2020.20.02.015.

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Guillain-Barré syndrome (GBS) is a heterogeneous disorder with a diverse clinical presentation ranging from weakness of certain body regions to tetraparesis with autonomic dysfunction and respiratory failure. Paraparetic GBS is a variant of GBS which is characterised by weakness limited to the lower limbs only. It is crucial to identify such topographical presentations, as a delay in diagnosis can lead to delayed initiation of specific treatment, which can negatively impact the outcome. We report a 29-year-old female patient who presented to the King Fahd Hospital of the University, Al Khobar, Saudi Arabia, in 2017 with rapid onset asymmetrical weakness of lower extremities associated with bladder dysfunction during the immediate postpartum period. The weakness spared cranial nerves and arms and imaging studies of the spine was unremarkable. Cerebrospinal fluid investigations showed cyto-albuminologic dissociation and nerve conduction studies showed features of demyelination. The patient was diagnosed with a paraparetic variant of GBS and treated with intravenous immunoglobulin. She had almost recovered completely at the two–month follow-up.Keywords: Paraparesis; Guillain-Barré Syndrome; Demyelination; Postpartum Period; Case Report; Saudi Arabia.

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2

Marco Orsini, Marco Orsini, Marcos RG de Feitas, Mariana P. Mello, Reny de Souza Antonioli, Nelson Kale, Júlia Fernandes Eigenheer, Carlos Henrique Melo Reis, and Osvaldo JM Nascimento. "Hidroterapia no gerenciamento da espasticidade nas paraparesias espásticas de várias etiologias." Revista Neurociências 18, no. 1 (March 31, 2001): 81–86. http://dx.doi.org/10.34024/rnc.2010.v18.8507.

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A espasticidade é uma manifestação clínica comum resultante da lesão do neurônio motor superior no sistema nervoso central. É uma condição que afeta adultos e crianças com uma grande variedade de patologias agudas e crônicas, sendo a característica predominante de diversas condições. Um dos aspectos mais incapacitantes das doenças que cursam com paraparesia espástica refere-se ao comprometimento progressivo da marcha, podendo levar aos indivíduos a permanecerem confinados a cadeira de rodas. A hidroterapia, por suas propriedades de suporte, assistência e resistência, constitui uma alternativa para a reabilitação física de pacientes neurológicos. Com base nisto este artigo tem como objetivo discutir os principais efeitos da hidroterapia na minimização das deficiências e incapacidades funcionais decorrentes da espasticidade em pacientes com paraparesias espásticas.

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3

Hassan, Anhar, Shivam Om Mittal, William T. Hu, Keith A. Josephs, Eric J. Sorenson, and J. Eric Ahlskog. "Natural History of “Pure” Primary Lateral Sclerosis." Neurology 96, no. 17 (February 26, 2021): e2231-e2238. http://dx.doi.org/10.1212/wnl.0000000000011771.

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ObjectiveTo assess whether primary lateral sclerosis (PLS), classified as pure when the EMG is normal, converts to amyotrophic lateral sclerosis (ALS) after longitudinal follow-up.MethodsRetrospective chart review was performed of patients with pure PLS at Mayo Clinic in Rochester, MN (1990–2016). Inclusion criteria required a normal EMG during the first 4 years of symptoms.ResultsForty-three patients had pure PLS (25 female, 58%) with a median onset age of 50 years (range 38–78 years) and median follow-up at 9 years’ disease duration (range 4–36 years). The ascending paraparesis phenotype (n = 30, 70%) was most common, followed by hemiparetic onset (n = 9, 21%) and bulbar onset (n = 4, 9%). Among the 30 paraparetic-onset cases, bladder symptoms (n = 18, 60%) and dysarthria (n = 15, 50%) were more common than pseudobulbar affect (n = 9, 30%) and dysphagia (n = 8, 27%). By the last follow-up, 17 of 30 (56%) used a cane and 6 (20%) required a wheelchair. The paraparetic variant, compared with hemiparetic and bulbar onset, had the youngest onset (48 vs 56 vs 60 years, respectively; p = 0.02). Five patients died; 1 patient required a feeding tube; and none required permanent noninvasive ventilation. Two patients developed an idiopathic multisystem neurodegenerative disorder, which surfaced after 19 and 20 years. Two patients developed minor EMG abnormalities. The remainder 39 had persistently normal EMGs.ConclusionsPure PLS did not convert to ALS after a median of 9 years’ disease duration follow-up in our study population. The ascending paraparetic phenotype was most common, with earlier onset and frequent bladder involvement. After years of pure PLS, <5% develop a more pervasive neurodegenerative disorder.

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4

Carletti, Beatrice Enrica, Irene Espadas, and Daniel Sanchez-Masian. "Thoracic vertebral canal stenosis due to articular process hypertrophy in two cats treated by hemilaminectomy with partial osteotomy of the spinous process." Journal of Feline Medicine and Surgery Open Reports 5, no. 2 (July 2019): 205511691986317. http://dx.doi.org/10.1177/2055116919863176.

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Case summary A 9-year-old neutered female British Shorthair cat (case 1) and a 13-year-old neutered male domestic shorthair cat (case 2) showed signs of chronic T3–L3 myelopathy, which progressed over 6 and 12 months, respectively. On presentation, case 1 had moderate pelvic limb proprioceptive ataxia and ambulatory paraparesis, and case 2 was non-ambulatory paraparetic and had urinary incontinence. Bilateral enlargement of the articular process joints at T11–T12 in case 1 and T3–T4 in case 2 causing dorsolateral extradural spinal cord compression was shown on MRI. Surgical decompression by a unilateral approach through hemilaminectomy with partial osteotomy of the spinous process was performed in both cases. The side of the approach was chosen based on the severity of the cord compression. Surgery resulted in a satisfactory outcome with short hospitalisation times. On discharge, case 1 showed mild postural reaction deficits on both pelvic limbs. Case 2 had regained urinary continence and could ambulate unassisted, although it remained severely ataxic. The 6 month follow-up showed very mild paraparesis and proprioceptive ataxia in both cats. No chronic medical treatment was required. Relevance and novel information This is the first report to describe clinical presentation, imaging features, surgical treatment and outcomes of thoracic vertebral canal stenosis owing to bilateral articular process hypertrophy in cats with no adjacent spinal diseases. Thoracic articular process hypertrophy should be included in the differential diagnosis of adult cats with chronic progressive myelopathy. Hemilaminectomy with partial osteotomy of the spinous process might be an appropriate surgical technique in these cases.

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5

Chaudhuri, Arunima, SamirK Hazra, AmitK Bandopadhyay, and Soma Datta. "Postspinal paraparesis." Journal of Obstetric Anaesthesia and Critical Care 2, no. 1 (2012): 54. http://dx.doi.org/10.4103/2249-4472.99333.

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6

Tribus, Clifford B. "Transient Paraparesis." Spine 26, no. 9 (May 2001): 1086–89. http://dx.doi.org/10.1097/00007632-200105010-00021.

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7

Fattahi, Arash, Seyed Mohammad Reza Mohajeri, Abdolhadi Daneshi, and Ardeshir Shahivand. "Hyperextension thoracic spine fracture with complete neurological recovery after surgical fixation: A case report." Surgical Neurology International 11 (May 30, 2020): 137. http://dx.doi.org/10.25259/sni_226_2020.

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Background: Hyperextension thoracic spine fractures (HTSFs) typically involve the anterior ligamentous complex of the spine. These patients often present with paraplegia and warrant early surgical reduction/fixation even though few deficits resolve. Here, we present the unusual case of a 40-year-old male whose paraparetic deficit resolved following reduction/fixation of a T7-T8 HTSF. Case Description: A 40-year-old male presented with a thoracic computed tomography (CT) documented T7- T8 HTSF following a motor vehicle accident. His neurological examination revealed severe paraparesis, but without a sensory level (ASIA motor score 78). The chest CT angiogram scan revealed a hypodensity in the aorta, representing a small traumatic aortic dissection responsible for the patient’s right hemothorax; 450 ml of blood was removed on chest tube placement. He underwent urgent/emergent thoracic spine reduction and fixation at the T7-T8 level. Within 5 postoperative months, he recovered fully neurological function (ASIA motor score 100). Conclusion: We recommend urgent/emergent surgical reduction/stabilization for patients with thoracic HTSF to decrease offer the potential for neurological recovery and avoid secondary injury due to continued compression.

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8

Crowe, Yvette C., Georgina Child, Richard Lam, and Ross McGregor. "Congenital block vertebrae and intervertebral disc protrusion in a young cat." Journal of Feline Medicine and Surgery Open Reports 5, no. 2 (July 2019): 205511691986803. http://dx.doi.org/10.1177/2055116919868037.

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Case summaryA 6-month-old domestic shorthair cat was evaluated for proprioceptive ataxia of the pelvic limbs. Over 2 months, the cat became poorly ambulatory, paraparetic with proprioceptive ataxia and developed a firm, distended bladder with intermittent overflow urinary incontinence. Block vertebrae (T1–3, T4–5 and T10–11) and lordosis were identified on radiographs of the vertebral column. MRI revealed T3/4 intervertebral disc protrusion with severe extradural compression, secondary syringohydromyelia caudal to the protrusion and generalised intervertebral disc disease throughout the cervical and thoracic vertebrae. Dorsal laminectomy at T3/4 resulted in resolution of paraparesis and marked improvement in coordination and strength. Block vertebrae are usually considered an incidental finding. In this patient, angular deformation (lordosis) and adjacent segment disease probably contributed to clinically significant intervertebral disc degeneration and protrusion.Relevance and novel informationThere are few case reports in the literature of multiple congenital vertebral malformations causing neurological deficits in cats. This is the first reported case of successful surgical management of intervertebral disc protrusion, possibly secondary to block vertebrae and lordosis in a cat.

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9

Raj, Anil K., Peter D. Neuhaus, Adrien M. Moucheboeuf, Jerryll H. Noorden, and David V. Lecoutre. "Mina: A Sensorimotor Robotic Orthosis for Mobility Assistance." Journal of Robotics 2011 (2011): 1–8. http://dx.doi.org/10.1155/2011/284352.

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While most mobility options for persons with paraplegia or paraparesis employ wheeled solutions, significant adverse health, psychological, and social consequences result from wheelchair confinement. Modern robotic exoskeleton devices for gait assistance and rehabilitation, however, can support legged locomotion systems for those with lower extremity weakness or paralysis. The Florida Institute for Human and Machine Cognition (IHMC) has developed the Mina, a prototype sensorimotor robotic orthosis for mobility assistance that provides mobility capability for paraplegic and paraparetic users. This paper describes the initial concept, design goals, and methods of this wearable overground robotic mobility device, which uses compliant actuation to power the hip and knee joints. Paralyzed users can balance and walk using the device over level terrain with the assistance of forearm crutches employing a quadrupedal gait. We have initiated sensory substitution feedback mechanisms to augment user sensory perception of his or her lower extremities. Using this sensory feedback, we hypothesize that users will ambulate with a more natural, upright gait and will be able to directly control the gait parameters and respond to perturbations. This may allow bipedal (with minimal support) gait in future prototypes.

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10

Ferreira, Adelmo, Bonfim Alves Silva Jr, F. Menezes Braga, Núbia M. Noschese Gargiulo, and J. Norberto Stávale. "Paraparesia por gota." Arquivos de Neuro-Psiquiatria 47, no. 4 (December 1989): 479–83. http://dx.doi.org/10.1590/s0004-282x1989000400017.

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Os autores apresentam um caso de bloqueio de canal raquidiano por tofo em paciente de 23 anos de idade com gota tofácea crônica e artrite gotosa, que desenvolveu paraparesia crural. Submetido a laminectomia, com exérese de acúmulos de cristais junto ao ligamento amarelo, não houve melhora do déficit motor. A revisão da literatura mostrou 9 casos previamente relatados.

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11

Herrero Antón, María José, José Cardona Serra, and José Luis Parajuá Pozo. "Paraparesia espástica tropical." FMC - Formación Médica Continuada en Atención Primaria 19, no. 6 (June 2012): 385. http://dx.doi.org/10.1016/s1134-2072(12)70415-2.

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12

Zaninovic, Vladimir. "TROPICAL SPASTIC PARAPARESIS." Lancet 330, no. 8553 (August 1987): 280. http://dx.doi.org/10.1016/s0140-6736(87)90870-1.

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13

Phoncharoensri, Dittana, Rawiphan Witoonpanich, Supoch Tunlayadechanont, and Jiraporn Laothamatas. "Confusion and paraparesis." Lancet 363, no. 9425 (June 2004): 1954. http://dx.doi.org/10.1016/s0140-6736(04)16410-6.

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14

Coleman, R. J., and J. W. Sander. "Tropical spastic paraparesis." Annals of Neurology 19, no. 6 (June 1986): 613. http://dx.doi.org/10.1002/ana.410190626.

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15

Zelnik, N., E. Leshinsky, and E. H. Kolodny. "Familial Spastic Paraparesis." Pediatric Neurosurgery 23, no. 4 (1995): 225–26. http://dx.doi.org/10.1159/000120963.

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16

Bruyn, R. P. M., J. van Deutekom, R. R. Frants, and G. W. Padberg. "Hereditary spastic paraparesis." Clinical Neurology and Neurosurgery 95, no. 2 (June 1993): 125–29. http://dx.doi.org/10.1016/0303-8467(93)90006-3.

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17

Román, Gustavo C., and Lydia N. Román. "Tropical spastic paraparesis." Journal of the Neurological Sciences 87, no. 1 (October 1988): 121–38. http://dx.doi.org/10.1016/0022-510x(88)90059-7.

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18

Setyaningrum, Dewi. "Logotherapy to Increase Self Esteem of Patient With Paraparese Inferiors." International Journal of Psychosocial Rehabilitation 24, no. 1 (January 20, 2020): 1526–35. http://dx.doi.org/10.37200/ijpr/v24i1/pr200250.

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19

Dhoot, Roshni, Katelyn Payne, John K. Fink, and Robert M. Pascuzzi. "An Unexplained Case of Progressive Spastic Paraparesis in an Individual with Known DiGeorge Syndrome." Case Reports in Neurology 12, no. 2 (June 9, 2020): 165–68. http://dx.doi.org/10.1159/000507954.

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DiGeorge syndrome (22q11.2 deletion) is associated with several neurologic disorders including structural abnormalities involving brain and spine, movement disorders, and epilepsy. Progressive spastic paraparesis has not been reported with DiGeorge syndrome. We report an individual in which DiGeorge syndrome was associated with progressive spastic paraparesis. This report extends the clinical phenotype of DiGeorge syndrome and presents the differential diagnosis of progressive spastic paraparesis in individuals with DiGeorge syndrome which provides insight into the clinical evaluation of such individuals.

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20

Lupescu, IC, S. Iacob, IG Lupescu, L. Gheorghe, and AO Dulamea. "From cirrhosis to paraparesis." Romanian Journal of Neurology 18, no. 4 (December 30, 2019): 211–14. http://dx.doi.org/10.37897/rjn.2019.4.9.

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21

Guillem, James. "Clinical conundrum: Hindlimb paraparesis." BSAVA Companion 2018, no. 4 (April 1, 2018): 8–11. http://dx.doi.org/10.22233/20412495.0418.08.

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22

Lee, Philip, Isabel Smith, Alina Piesowicz, and David Brenton. "Spastic paraparesis after anaesthesia." Lancet 353, no. 9152 (February 1999): 554. http://dx.doi.org/10.1016/s0140-6736(98)10090-9.

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23

Claret Teruel, G., R. Garrido Romero, A. Fernández López, and C. Luaces Cubells. "Paraparesia aguda no traumática." Anales de Pediatría 60, no. 2 (2004): 186–88. http://dx.doi.org/10.1016/s1695-4033(04)78245-4.

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24

de Castro García, F. J., J. Á. Santos Sánchez, P. García Iñigo, and M. T. Adeva Bartolomé. "Paraparesia en adulto joven." SEMERGEN - Medicina de Familia 31, no. 10 (November 2005): 486–88. http://dx.doi.org/10.1016/s1138-3593(05)72974-9.

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25

Shaw, Brian A. "Pott's Disease with Paraparesis." New England Journal of Medicine 334, no. 15 (April 11, 1996): 958–59. http://dx.doi.org/10.1056/nejm199604113341505.

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26

Pottecher, Pierre, Pierre Martz, and Paul Ornetti. "Paraparesis Revealing Tophaceous Gout." Arthritis & Rheumatology 70, no. 6 (April 23, 2018): 942. http://dx.doi.org/10.1002/art.40434.

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27

Bird, T. D. "Idiopathic progressive spastic paraparesis." JAMA: The Journal of the American Medical Association 274, no. 15 (October 18, 1995): 1191b—1191. http://dx.doi.org/10.1001/jama.274.15.1191b.

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28

Garcés, J. M., M. A. Santana, M. I. Alonso, and J. Villar. "Paraparesia espástica tropical importada." Revista Clínica Española 207, no. 6 (June 2007): 311. http://dx.doi.org/10.1157/13106859.

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29

Rider, Eric, and Carl A. Gold. "Young Man With Paraparesis." Annals of Emergency Medicine 72, no. 3 (September 2018): e19-e20. http://dx.doi.org/10.1016/j.annemergmed.2018.03.043.

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30

Kaiser, P. "Endemische spastische Paraparese (Konzo)." Der Nervenarzt 73, no. 10 (October 1, 2002): 946–51. http://dx.doi.org/10.1007/s00115-002-1380-6.

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31

Gupta, Dhananjay, Pradeep R, Anish Mehta, Mahendra Javali, Purshottam T. Acharya, and Rangasetty Srinivasa. "Acute Onset Asymmetric Sensorimotor Paraparesis: Not Always Spinal!" Journal of Stroke Medicine 3, no. 1 (June 2020): 40–42. http://dx.doi.org/10.1177/2516608520927198.

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Objective: To report an atypical case of acute onset sensorimotor paraparesis secondary to bilateral cerebral stroke. Background: Acute onset paraparesis or paraplegia is usually secondary to a spinal cord disease. Central or cerebral causes of paraparesis are rare and include parasagittal and bilateral precentral lesions. Design/Methods: Case report and literature review. Results: A 65-year-old man presented with acute onset weakness of both lower limbs, associated with pins and needle sensation. On examination, he was found to have paraparesis (grade 2/5, both legs) and an asymmetric sensory loss in both legs and thighs. Spinal magnetic resonance imaging ruled out any compressive or noncompressive etiology. Magnetic resonance imaging of the brain showed an acute infarction in the bilateral cerebral hemisphere in both the pre- and postcentral gyrus. An angiogram of the brain revealed an aplastic right ACA-A1 with left ACA-A1 feeding bilateral A2. There was distal left ACA-A1 stenosis seen, the probable cause of bilateral stroke in this patient. The patient was treated conservatively and showed symptomatic improvement during the course of stay at the hospital. Conclusion: This case of acute paraparesis secondary to bilateral cerebral infarction demonstrates the need to always look for a cerebral cause. In patients with cerebral infarction, who present early to a hospital, it may provide a window for thrombolytic or endovascular therapy.

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32

Kaur, Jaspreet, Gautam Jesrani, Monica Gupta, and Sarabmeet Singh Lehl. "Spastic paraparesis associated with advanced liver cirrhosis: a condition obscure in terms of treatment and prognosis." BMJ Case Reports 13, no. 6 (June 2020): e235090. http://dx.doi.org/10.1136/bcr-2020-235090.

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Hepatic myelopathy or spastic paraparesis of liver disease is an insidious onset condition with pure motor spastic paraparesis without sensory, bladder or bowel involvement in patients with chronic liver disease, in which the neurological dysfunction cannot be explained by other causes. It is a rare, relentlessly progressive and mostly irreversible neurological complication resulting from portosystemic shunts occurring spontaneously, created surgically or due to ‘functional shunting’. In some cases, no evidence of shunting is elicitable due to difficulty in locating the hidden collaterals. We report this rare case of a 33-year-old man with chronic liver disease presenting with spastic paraparesis after 11 months of resolution of an episode of hepatic encephalopathy.

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33

Watanabe, Akira, Kinya Nakanishi, and Kazuo Kataoka. "Intradural spinal arachnoid cyst contributing to sudden paraparesis." Surgical Neurology International 10 (June 19, 2019): 102. http://dx.doi.org/10.25259/sni-246-2019.

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Background: Spinal arachnoid cysts are cystic lesions filled with cerebrospinal fluid that contributes to neurological deficits depending on their size/location within the spinal canal. Here, we report a patient with a spinal subarachnoid cyst who suddenly developed paraparesis. Case Description: A 37-year-old female with a thoracic spinal arachnoid cyst at the T7 level suddenly developed lower abdominal pain followed by immediate paraparesis. Two weeks following the onset of symptoms, she underwent a T6-T8 laminectomy; this included with full cyst excision. By the 4th postoperative week, her signs/symptoms fully resolved. Conclusions: A 37-year-old female with a T7 thoracic spinal subarachnoid cyst who presented with acute paraparesis regained normal function 2 weeks following a T6-T8 laminectomy.

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34

Brar, Rahat, Abhishek Prasad, Shaleen Rana, and Vaibhav Pratap Singh. "Extramedullary haematopoiesis presenting with paraparesis." Acta Neurologica Belgica 114, no. 4 (October 9, 2013): 311–12. http://dx.doi.org/10.1007/s13760-013-0254-y.

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35

Rodgers-Johnson, Pamela, OWENSTC Morgan, Vladimir Zaninovic, D. Carleton Gajdusek, RalphM Garruto, SandraA Larsen, and DanielS Graham. "TREPONEMATOSES AND TROPICAL SPASTIC PARAPARESIS." Lancet 327, no. 8484 (April 1986): 809. http://dx.doi.org/10.1016/s0140-6736(86)91826-x.

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36

Utku, Ufuk, Talip Asil, Kemal Balcı, İlkay Uzunca, and Yahya Çelik. "Hepatic myelopathy with spastic paraparesis." Clinical Neurology and Neurosurgery 107, no. 6 (October 2005): 514–16. http://dx.doi.org/10.1016/j.clineuro.2004.10.002.

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37

Cárdenas, Graciela, Erik Guevara, Rosa Delgado-Hernández, and José Luís Soto-Hernández. "Skin infection and progressive paraparesis." Lancet Infectious Diseases 12, no. 5 (May 2012): 422. http://dx.doi.org/10.1016/s1473-3099(12)70062-2.

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38

Aschenbroich, S., A. Woolcock, and D. R. Rissi. "Paraparesis in a Golden Retriever." Veterinary Pathology 51, no. 5 (November 5, 2013): 996–99. http://dx.doi.org/10.1177/0300985813509385.

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39

Dham, Bhavpreet S., Daniel M. Kwa, and Joseph V. Campellone. "Postpartum paraparesis from spinal neurofibroma." Spine Journal 12, no. 7 (July 2012): e5-e8. http://dx.doi.org/10.1016/j.spinee.2012.07.006.

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40

Takaya, S., M. Hiroki, T. Sawada, and H. Fukuyama. "Paraparesis due to capsular hemorrhages." Neurology 62, no. 6 (March 22, 2004): 967. http://dx.doi.org/10.1212/01.wnl.0000115395.82421.54.

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41

McMonagle, P., M. Hutchinson, and B. Lawlor. "Hereditary spastic paraparesis and psychosis." European Journal of Neurology 13, no. 8 (August 2006): 874–79. http://dx.doi.org/10.1111/j.1468-1331.2006.01379.x.

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42

Kumar, AP Naveen, Lalitha, M. Srinivas, and A. Patnaik. "P22 Ochronosis presenting as paraparesis." Indian Journal of Rheumatology 3, no. 3 (November 2008): S36. http://dx.doi.org/10.1016/s0973-3698(10)60466-9.

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43

Savage, Henry Oluwasefunmi, Malin Roesner, and David Cohen. "Spastic Paraparesis Following Cocaine Inhalation." Case Reports in Medicine 2009 (2009): 1–3. http://dx.doi.org/10.1155/2009/465968.

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Cocaine use is reaching epidemic proportions in the UK and the consequences are a number of debilitating effects. Strokes may result from a number of mechanisms related to cocaine use. This report describes a case of cocaine induced stroke in an apparently healthy young man with unusual patterns of radiological findings on his brain MRI.

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44

Adlakha, Arun, and Henry J. Schultz. "Spastic Paraparesis of Insidious Onset." Hospital Practice 29, no. 12 (December 15, 1994): 49–53. http://dx.doi.org/10.1080/21548331.1994.11443121.

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45

Savini, Paolo. "Paraparesis induced by extramedullary haematopoiesis." World Journal of Radiology 3, no. 3 (2011): 82. http://dx.doi.org/10.4329/wjr.v3.i3.82.

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46

Zaninovic, Vladimir, Cesar Arango, Robin Biojo, Carios Mora, Pamela Rodgers-Johnson, Mauricio Concha, Raul Corral, et al. "Tropical spastic paraparesis in colombia." Annals of Neurology 23, S1 (1988): S127—S132. http://dx.doi.org/10.1002/ana.410230730.

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47

Wells, Christine R. "Familial Spastic Paraparesis and Deafness." Archives of Neurology 43, no. 9 (September 1, 1986): 943. http://dx.doi.org/10.1001/archneur.1986.00520090071021.

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Tenembaum, Silvia N., Ricardo C. Reisin, Ana L. Taratuto, and Natalio Fejerman. "Spastic paraparesis and sensory neuropathy." Muscle & Nerve 19, no. 5 (May 1996): 649–53. http://dx.doi.org/10.1002/(sici)1097-4598(199605)19:5<649::aid-mus14>3.0.co;2-i.

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Garg, R. K., B. Karak, and S. Misra. "Acute paraparesis with tuberculous meningitis." Postgraduate Medical Journal 74, no. 871 (May 1, 1998): 269–71. http://dx.doi.org/10.1136/pgmj.74.871.269.

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Kumar, Dinesh, Prasanth Varghese, and Percival Gilvaz. "A RARE CASE OF PARAPARESIS." Journal of Evolution of Medical and Dental Sciences 5, no. 69 (August 29, 2016): 5047–48. http://dx.doi.org/10.14260/jemds/2016/1146.

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