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Books on the topic 'Parenchymal'

Author: Grafiati

Published: 5 June 2025

Last updated: 2 August 2025

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1

Piracha, Kashif. Diffuse Pulmonary Parenchymal Disease. Springer Nature Switzerland, 2023. http://dx.doi.org/10.1007/978-3-031-36037-4.

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Fervenza, Fernando C., Julie Lin, Sanjeev Sethi, and Ajay K. Singh, eds. Core Concepts in Parenchymal Kidney Disease. Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-8166-9.

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Poletti, Venerino, ed. Transbronchial cryobiopsy in diffuse parenchymal lung disease. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-14891-1.

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4

Jepson, Mark Andrew. Physiological and pathological implications of hepatic parenchymal heterogeneity. University of Birmingham, 1988.

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5

Bush, Andrew, and Jane C. Davies. Paediatric respiratory disease: Parenchymal diseases : an atlas of investigation and management. Clinical Pub., 2011.

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6

Quistorff, Bjørn. Metabolic heterogeneity of liver parenchymal cells; investigated by digitonin perfusion techniques. Luna-Tryk, 1994.

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7

MD) Hales Lung Conference (1st 2009 Baltimore. Proceedings of 2009 Hales Lung Conference: Clinical and pathophysiolgic aspects of diffuse parenchymal disease, April 27, 2009, Baltimore, MD : clinical and pathophysiologic aspects of diffuse parenchymal lung disease. BoBitField, 2011.

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8

Brown, Laurence Andrew. A functional and morphological investigation of the mechanisms of cerebral blood flow-metabolism coupling in parenchymal microvessels. University of Birmingham, 2000.

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9

Sahay, Manisha. Diseases of renal parenchyma. Intech, 2012.

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10

Delcourt, Candice, and Craig Anderson. Management of parenchymal haemorrhage. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0237.

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Parenchymal intracerebral haemorrhage (ICH) affects several million people in the world each year, most of whom reside in developing countries. ICH accounts for 10-40% of strokes and is the least treatable form of stroke with a 30-day mortality of 30-55%, with half of these deaths occurring within the first few days of onset. . High blood pressure is both a causal and prognostic factor for ICH, with early control of hypertension being the only medical treatment which may improve recovery and the level of residual functioning. The role of surgery remains controversial. Management is largely sup

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11

Costabel, U., R. M. du Bois, and J. J. Egan, eds. Diffuse Parenchymal Lung Disease. S. Karger AG, 2007. http://dx.doi.org/10.1159/isbn.978-3-318-01377-1.

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12

Diffuse parenchymal lung disease. Karger, 2007.

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13

Fervenza, Fernando C., Julie Lin, and Sanjeev Sethi. Core Concepts in Parenchymal Kidney Disease. Springer, 2013.

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14

Fervenza, Fernando C., Ajay K. Singh, Julie Lin, and Sanjeev Sethi. Core Concepts in Parenchymal Kidney Disease. Springer London, Limited, 2013.

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15

Fervenza, Fernando C., Ajay K. Singh, Julie Lin, and Sanjeev Sethi. Core Concepts in Parenchymal Kidney Disease. Springer, 2015.

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16

Fervenza, Fernando C., Ajay K. Singh, Julie Lin, and Sanjeev Sethi. Core Concepts in Parenchymal Kidney Disease. Springer, 2013.

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17

Poletti, Venerino. Transbronchial Cryobiopsy in Diffuse Parenchymal Lung Disease. Springer International Publishing AG, 2020.

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18

Poletti, Venerino. Transbronchial cryobiopsy in diffuse parenchymal lung disease. Springer, 2019.

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19

Diffuse Parenchymal Lung Disease (Progress in Respiratory Research). S. Karger AG (Switzerland), 2007.

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20

Kuiper, Johan. Cellular communication between parenchymal, Kupffer and endothelial liver cells. 1989.

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21

THOMAS. Hepatic Non Parenchymal Cells And Their Role In Disease. Chapman & Hall, 1996.

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22

Davey, Patrick, Sherif Gonem, and David Sprigings. Interstitial lung disease. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0139.

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The interstitial lung diseases, also known as the diffuse or diffuse parenchymal lung diseases, are a broad group of pulmonary disorders which mainly affect the lung parenchyma as opposed to the airways. By convention, infectious and malignant conditions are excluded from this definition. Thus, the interstitial lung diseases comprise a group of conditions characterized by variable degrees of inflammation and fibrosis, centred on the lung interstitium and alveolar airspaces.

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23

Charidimou, Andreas, Eric Jouvent, and Susanne J. Van Veluw, eds. Cerebral Small Vessel Diseases: From Vessel Alterations to Cortical Parenchymal Injury. Frontiers Media SA, 2020. http://dx.doi.org/10.3389/978-2-88963-587-0.

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24

Török, M. Estée, Fiona J. Cooke, and Ed Moran. Neurological infections. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199671328.003.0019.

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This chapter covers both acute bacterial and viral, and chronic, meningitis, as well as tuberculous, cryptococcal, coccidioidal, and Histoplasma meningitis, describing meningeal symptoms (headache, neck stiffness, vomiting, photophobia) and cerebral dysfunction (confusion, coma). The chapter also covers neurocysticercosis (including parenchymal and extra-parenchymal cysts), encephalitis (an inflammatory process in the brain characterized by cerebral dysfunction), as well as brain abscess, cerebritis, subdural empyema, epidural abscess, and cerebrospinal fluid shunt infections.

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25

Katritsis, Demosthenes G., Bernard J. Gersh, and A. John Camm. Secondary hypertension. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199685288.003.0502_update_002.

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The causes of secondary hypertension and their therapy are discussed. Considered conditions are secondary hypertension, renovascular hypertension, renal parenchymal disease, primary aldosteronism, phaeochromocytoma, adrenal incidentaloma, and other causes of secondary hypertension.

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26

Kortgen, Andreas, and Michael Bauer. Hepatic function in the critically ill. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0175.

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The liver with its parenchymal and non-parenchymal cells plays a key role in the organism with manifold functions of metabolism, synthesis, detoxification, excretion, and host response. This requires a portfolio of different tests to obtain an overview of hepatic function. In the critically ill hepatic dysfunction is common and potentially leading to extrahepatic organ dysfunctions culminating in multi-organ failure. Conventional laboratory measures are used to evaluate hepatocellular damage, cholestasis, or synthesis. They provide valuable (differential) diagnostic data and can yield prognost

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27

Hather, Jon G. Archaeological Parenchyma. Taylor & Francis Group, 2016.

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28

Hather, Jon G. Archaeological Parenchyma. Routledge, 2016. http://dx.doi.org/10.4324/9781315434490.

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29

Archaeological Parenchyma. Archetype Publications Ltd, 2000.

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30

Hather, Jon G. Archaeological Parenchyma. Taylor & Francis Group, 2017.

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31

Archaeological Parenchyma. Taylor & Francis Group, 2016.

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32

Lambert, Heather. Primary vesicoureteric reflux and reflux nephropathy. Edited by Adrian Woolf. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0355_update_001.

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Vesicoureteric reflux (VUR) describes the flow of urine from the bladder into the upper urinary tract when the ureterovesical junction fails to perform as a one-way valve. Most commonly, VUR is primary, though it can be secondary to bladder outflow obstruction and can occur in several multiorgan congenital disorders. There is good evidence of a genetic basis with a greatly increased risk of VUR in children with a family history of VUR. VUR is a congenital disorder, which largely shows improvement or complete resolution with age. Fetal VUR may be associated with parenchymal developmental defect

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33

Millar, Professor Ann B., Dr Richard Leach, Dr Rebecca Preston, et al. Respiratory diseases and respiratory failure. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199565979.003.0005.

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Chapter 5 covers respiratory diseases and respiratory failure, including clinical presentations of respiratory disease, assessment of diffuse lung disease, hypoxaemia, respiratory failure, and oxygen therapy, pneumonia, mycobacterial infection, asthma, chronic obstructive pulmonary disease (COPD), lung cancer, mediastinal lesions, pneumothorax, pleural disease, asbestos-related lung disease, diffuse parenchymal (interstitial) lung disease, sarcoidosis, pulmonary hypertension, acute respiratory distress syndrome, bronchiectasis and cystic fibrosis, bronchiolitis, eosinophilic lung disease, airw

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34

Elwell, Christine, and Kufre Sampson. Neurological tumours. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0237.

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Neurological tumours are categorized by the WHO as follows: neuroepithelial tumours (gliomas, oligodendrogliomas, ependymomas, pineal parenchymal tumours, medulloblastoma, neuronal and neuroglial tumours); cranial and paraspinal nerve tumours (schwannoma, neurofibromas); meningeal tumours (meningiomas); lymphomas; germ cell tumours (germinoma, teratoma); sellar region tumours (cranipharyngioma); and metastases. The tumours are classified according to grade. The WHO histological grading scheme used for astrocytomas is based on mitoses, nuclear pleomorphism, necrosis, and endothelial proliferati

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35

Aksamit, Timothy R. Diffuse Lung Disease. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199755691.003.0617.

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Diffuse lung disease includes a wide range of parenchymal lung diseases that have infectious, inflammatory, malignant, drug, occupational or environmental, and other causes. Although many identifiable causes are recognized, the cause of most cases of diffuse lung disease in many published series is idiopathic. The clinical course may be acute or prolonged and may progress rapidly to life-threatening respiratory failure with death, or it may be indolent over many years. In most instances, a differential diagnosis can readily be formulated by obtaining the medical history, with emphasis on the n

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36

Haghighi, Afshin Borhani, and Bernadette Kalman. Other Proven and Putative Autoimmune Disorders of the CNS. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0094.

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Behcet’s Disease (BD) is a multiorgan disorder characterized by oral and genital ulceration, uveitis, and dermatological symptoms. BD is most prevalent in the Mediterranean countries and East Asia, but also occurs in Europe and North America. The etiology remains unknown. Evidence suggests that BD is an autoimmune disorder with complex traits. Neuro-Behcet’s Syndome (NBS) develops in about 5% to 30% of patients with BD and presents with parenchymal or nonparenchymal pathology. The course of NBS is highly variable. Treatment strategies include modulations of the immune response and tissue degen

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37

McCabe, Sam, Christopher Harnain, and Grigory Rozenblit. Use of an Elongated Radiopaque Gelatin Sponge Plug for Tract Occlusion After Hepatic Interventions. Edited by S. Lowell Kahn, Bulent Arslan, and Abdulrahman Masrani. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199986071.003.0088.

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This chapter describes the use of an elongated radiopaque gelatin sponge plug for tract occlusion after percutaneous biliary, portal venous, or hepatic venous access or intervention. Often, embolization coils or a gelatin sponge “slurry” is sufficient for hemostasis following liver intervention. This technique offers an inexpensive, temporary, and readily available option for achieving hemostasis following liver interventions. A contrast-soaked Gelfoam plug is loaded into a delivery cylinder that is advanced into the access sheath and positioned and deployed under fluoroscopic guidance. This t

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38

Sahay, Manisha, ed. Diseases of Renal Parenchyma. InTech, 2012. http://dx.doi.org/10.5772/2484.

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39

Deltas, Constantinos, Elena Levtchenko, Mariya Severova, and Veeraish Chauhan. Diseases of Renal Parenchyma. DI Press, 2022.

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40

Siebert, Stefan, Sengupta Raj, and Alexander Tsoukas. Complications of axial spondyloarthritis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198755296.003.0009.

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In addition to the well-recognized extra-articular manifestations (EAMs) of ankylosing spondylitis (AS), this condition can also be associated with a number of clinically important complications. While EAMs are considered part of the spondyloarthritis (SpA), the complications are generally a consequence of having the disease. Patients with AS are at increased risk of osteoporosis and spinal fractures. The latter may occur after seemingly minor trauma and may lead to significant neurological compromise. Other potential neurological complications include atlantoaxial subluxation and compressive

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41

Lam, Diana L., and Habib Rahbar. Non-Mass Enhancement on MRI. Edited by Christoph I. Lee, Constance D. Lehman, and Lawrence W. Bassett. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190270261.003.0031.

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Breast cancer presents on MRI as an enhancing finding on post-contrast T1-weighted images that is distinct from normal background parenchymal enhancement (BPE), and these enhancing lesions can be further described as a focus, mass, or non-mass enhancement (NME). Each enhancing lesion, with the exception of a focus, can be described further with specific morphological features that are defined by the ACR BI-RADS Atlas. This chapter reviews the key imaging and clinical features, imaging protocols and pitfalls, differential diagnoses, and management recommendations of a focus of enhancement and n

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42

Plotnik, Adam N., and Stephen Kee. Advancing the TIPS Sheath Through a Difficult Cirrhotic Liver: Pay It Forward Off the Balloon. Edited by S. Lowell Kahn, Bulent Arslan, and Abdulrahman Masrani. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199986071.003.0076.

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The “pay it forward off the balloon” technique for advancing the transjugular intrahepatic portosystemic shunt (TIPS) sheath may be employed during a difficult TIPS case when the interventionalist has already accessed the portal vein but cannot get the standard 10 Fr TIPS sheath through the fibrotic tract into the portal vein to thereby allow placement of a standard TIPS covered stent. In some patients, the fibrotic recoil of the parenchymal hepatic tract can be so severe that the basic balloon dilation maneuver fails. The pay it forward off the balloon technique employs the use of a 6 mm × 4-

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43

Felbaum, Daniel R., Jonathan H. Sherman, and Walter C. Jean. Pineal Tumors. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190696696.003.0003.

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Pineal region tumors can include a variety of histologies including pineal parenchymal tumor, germ cell tumor, glial tumor, metastasis and meningioma. The workup for pineal region tumors includes standard magnetic resonance imaging for anatomic imaging, as well as cerebrospinal fluid markers to assess for certain germ cell tumors. Cerebrospinal fluid diversion may be necessary if patients present with hydrocephalus. If surgical resection is indicated based on the suspected diagnosis, magnetic resonance venogram is an important study that influences the surgical trajectory. This chapter reviews

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44

Araujo, Abelardo Q.-C. Neurological Manifestations of the Human T-lymphotropic Virus Type 1. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0161.

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The human T cell lymphotropic virus type 1 (HTLV-1) is a retrovirus that infects about 20 million individuals worldwide. Its typical neurological presentation is of a chronic, slowly progressive myelopathy named “HTLV-1-associated myelopathy/tropical spastic paraparesis” (HAM/TSP). HAM/TSP emerges as the tip of the iceberg among numerous other neurological clinical syndromes caused by this virus, such as inflammatory myopathies, polyneuropathies, ALS-like syndromes, dysautonomia, etc. HAM/TSP designates a spastic paraparesis with neurogenic bladder, and minor sensory signs. Pathologically, HAM

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45

Ince, Can, and Alexandre Lima. Monitoring the microcirculation in the ICU. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0142.

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The microcirculation is the key physiological compartment of the cardiovascular system where oxygen is delivered by convection and diffusion to respiring parenchymal cells to support cellular, and thereby organ, function. The microcirculation consists of microvessels less than 100 µmin diameter consisting of arterioles, capillaries, and venules. The smallest vessels (<6 µm) are the capillaries where most oxygen leaves the circulation by passive diffusion to cells. The critical role of the microcirculation has long been recognized, although it has recently been possible to image its function

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46

Cullinan, Paul, and Joanna Szram. Occupational lung disease. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0142.

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Some occupational lung diseases are defined by their clinical or pathological nature (e.g. occupational asthma or mesothelioma), while others are defined by their specific etiology (e.g. silicosis, farmer’s lung). Most fall into one of three categories. The first is airways disease, including occupational asthma (induced by a workplace agent), work-exacerbated asthma (preexisting asthma provoked by one or more agents at work), and irritant-induced asthma (initiated by a single, toxic exposure to a respiratory irritant); COPD and obliterative bronchiolitis may arise from workplace exposures, an

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47

McCabe, Sam, Christopher Harnain, and Grigory Rozenblit. Transmesenteric Method of TIPS Placement Using Portal Access via Mini-Laparotomy. Edited by S. Lowell Kahn, Bulent Arslan, and Abdulrahman Masrani. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199986071.003.0079.

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Transmesenteric portal access via mini-laparotomy may be used as a salvage technique when standard transjugular intrahepatic portosystemic shunt (TIPS) is unsuccessful due to difficult anatomy or portal vein thrombosis. This technique allows for precise determination of both the portal and the hepatic vein branch involved in the TIPS. This method usually involves the cooperation of a surgeon, who performs a mini-laparotomy and exposes a small bowel loop in the interventional suite. A mesenteric venous branch is then cannulated, providing direct access to the portal venous system. In distinctio

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48

Glockner, James F., Kazuhiro Kitajima, and Akira Kawashima. Magnetic resonance imaging. Edited by Christopher G. Winearls. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0015_update_001.

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Magnetic resonance imaging (MRI) provides excellent anatomic detail and soft tissue contrast for the evaluation of patients with renal disease. MRI needs longer scan time than computed tomography (CT); however, no radiation is involved. Gadolinium-based contrast agents (GBCAs) are used to help provide additional image contrast during MRI. MRI is indicated for characterization of renal mass, staging of malignant renal neoplasms, and determination of vena cava involvement by the renal tumour. Magnetic resonance (MR) angiography is widely accepted as a non-invasive imaging work-up of renal artery

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49

Solomon, Tom. Meningitis. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569381.003.0969.

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Meningitis is defined as inflammation of the brain meninges, characterized clinically by inflammatory cells in CSF. When there is concurrent parenchymal brain involvement the term meningoencephalitis is used, meningoencephalomyelitis implies that there is spinal cord involvement too.Although increased cellularity in the CSF, or pleocytosis, is traditionally considered the hallmark of meningitis, some organisms, particularly fungi, can cause meningitis without a pleocytosis, especially in the immunocompromised. The advent of more sensitive methods of detecting viral nucleic acid in the CSF such

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50

Stocchetti, Nino, and Andrew I. R. Maas. Causes and management of intracranial hypertension. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0233.

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Intracranial hypertension may damage the brain in two ways—it causes tissue distortion and herniation, and reduces cerebral perfusion. The many different pathologies that can result in intracranial hypertension include subarachnoid haemorrhage, spontaneous intra-parenchymal haemorrhage, malignant cerebral hemispheric infarction, and acute hydrocephalus. The pathophysiology and specific treatment of intracranial hypertension may be different and depend on aetiology. In patients with subarachnoid haemorrhage a specific focus is on treating secondary hydrocephalus and maintaining adequate cerebra

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