Relevant bibliographies by topics / Parietal pericardium

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Academic literature on the topic 'Parietal pericardium'

Author: Grafiati
Published: 5 June 2025
Last updated: 16 July 2025

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Journal articles on the topic "Parietal pericardium"

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Lamping, K. G., C. D. Rios, J. A. Chun, H. Ooboshi, B. L. Davidson, and D. D. Heistad. "Intrapericardial administration of adenovirus for gene transfer." American Journal of Physiology-Heart and Circulatory Physiology 272, no. 1 (1997): H310—H317. http://dx.doi.org/10.1152/ajpheart.1997.272.1.h310.

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Gene transfer to the heart has been accomplished with intravascular administration of adenoviral vectors into the pericardial sac, by increasing the duration of exposure to the adenovirus, would result in gene expression in the pericardium and perhaps myocardium and therefore might provide an alternative method to intravascular administration for gene transfer. We injected a replication-deficient adenovirus (average 1 x 10(12) particles/ml in 3% sucrose; 1 x 10(10) plaque forming units/ml containing cDNA encoding a nuclear-targeted bacterial beta-galactosidase into the pericardial sac of dogs.
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Napolitano, Giuseppe, Josephine Pressacco, and Eleonore Paquet. "Imaging Features of Constrictive Pericarditis: Beyond Pericardial Thickening." Canadian Association of Radiologists Journal 60, no. 1 (2009): 40–46. http://dx.doi.org/10.1016/j.carj.2009.02.034.

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Constrictive pericarditis is caused by adhesions between the visceral and parietal layers of the pericardium and progressive pericardial fibrosis that restricts diastolic filling of the heart. Later on, the thickened pericardium may calcify. Despite a better understanding of the pathophysiologic basis of the imaging findings in constrictive pericarditis and the recent advent of magnetic resonance imaging (MRI) technology, which has dramatically improved the visualization of the pericardium, the diagnosis of constrictive pericarditis remains a challenge in many cases. In patients with clinical
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Guglielmini, Carlo, Carla Civitella, Daniela Malatesta, and Chiara Palmieri. "Metastatic Pericardial Tumors in a Dog With Equivocal Pericardial Cytological Findings." Journal of the American Animal Hospital Association 43, no. 5 (2007): 284–87. http://dx.doi.org/10.5326/0430284.

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A metastatic tumor associated with pericardial effusion was diagnosed in a 6-year-old, female, mixed-breed dog. Echocardiography identified multiple echogenic masses adherent to both visceral and parietal pericardium, while results of pericardial fluid cytology were non-diagnostic. The distribution pattern of the masses is remarkable in that they protruded from both pericardial surfaces, rather than one, and demonstrated an oscillatory motion during the cardiac cycle. Pathological examination confirmed the diagnosis of multiple metastatic tumors of the pericardium, with the primary tumor being
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Sun, Qianhui, Liang Ma, and Peng Teng. "A Rare Constrictive Pericarditis with Complete Separation between the Visceral and Parietal Pericardium: A Case Report." Heart Surgery Forum 23, no. 4 (2020): E546—E548. http://dx.doi.org/10.1532/hsf.3021.

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Background: Pericarditis is the most common form of pericardial disease, while constrictive pericarditis is challenging in diagnosis and is easily overlooked. Case report: A 30-year-old female presented with abdominal distension and mild lower extremity edema for 3 months. The patient was initially suspected of having cirrhosis caused by Wilson Disease. Following liver biopsy and multiple investigation, thickened, calcified pericardium was detected by echocardiography and chest computed tomography. The patient was finally diagnosed with chronic constrictive pericarditis and received pericardie
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Para, Ombretta, Eleonora Blasi, Martina Finocchi, et al. "Management of pericarditis." Italian Journal of Medicine 13, no. 3 (2019): 161–68. http://dx.doi.org/10.4081/itjm.2019.1153.

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Pericarditis is an inflammatory syndrome involving pericardium, which is a double-walled sac consisting of two leaves, a serous visceral layer in contact with the myocardium (pericardium) and a parietal fibrous one, delimiting a cavity (pericardial cavity) containing pericardial fluid. Pericarditis may occur isolated or as a manifestation of a systemic disorder. Diagnosis and correct management of pericarditis can be difficult and its natural history is often characterized by a lot of relapses. Treatment of acute pericarditis should target the underlying etiology. The diagnosis is based on cha
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6

Subtaweesin, Thaworn. "Excision of visceral pericardium for chronic effusive constrictive pericarditis." Asian Cardiovascular and Thoracic Annals 28, no. 1 (2019): 65–67. http://dx.doi.org/10.1177/0218492319874905.

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Chronic effusive-constrictive pericarditis was successfully treated by removal of the visceral pericardium in a 15-year-old Thai female. The constrictive visceral pericardium was barely detected by noninvasive studies. This case highlights the importance of removal of the visceral pericardium in this condition. If only the parietal pericardium is removed, the patient will not improve clinically.
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Townsley, M. I., D. Negrini, and J. L. Ardell. "Regional blood flow to canine parietal pleura and internal intercostal muscle." Journal of Applied Physiology 70, no. 1 (1991): 97–102. http://dx.doi.org/10.1152/jappl.1991.70.1.97.

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Transcapillary Starling forces in the parietal pleura and the underlying interstitium may potentially contribute to the exchange of fluid across this barrier. However, the extent of blood flow to the parietal pleura has not been measured. Thus, using standard microsphere techniques, we compared blood flow to the parietal pleura, including the subpleural interstitium, with blood flow to the adjacent internal intercostal muscle, as well as with flows to other serous tissues, including mediastinal pleura, pericardium, and parietal peritoneum, in anesthetized dogs that were either breathing sponta
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Patel, Parth, Sudhendu Patel, and Vidhi Shah. "Colchicine for pericarditis due to COVID-19: a case report." International Journal of Advances in Medicine 9, no. 4 (2022): 498. http://dx.doi.org/10.18203/2349-3933.ijam20220793.

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The pericardium is a fibroelastic sac made up of visceral and parietal layers separated by a (potential) space, the pericardial cavity.The most troublesome complication of acute pericarditis is the development of recurrent episodes of pericardial inflammation, occurring in 15% to 32% of cases. Therapeutic modalities are nonspecific and include non-steroidal anti-inflammatory drugs (NSAIDs) and corticosteroids. Here we present a case of a patient presenting with pericarditis due to COVID-19. He was successfully treated with colchicine. To our knowledge acute pericarditis due to severe acute res
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Liwan, Armand Setiady, Ni Putu Veny Kartika Yantie, and Eka Gunawijaya. "Challenges in diagnosing pediatric pericarditis and the etiology in remote areas during the COVID-19 pandemic: a socio-clinical dilemma." Paediatrica Indonesiana 64, no. 2 (2024): 184–92. http://dx.doi.org/10.14238/pi64.2.2024.184-92.

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Inflammation of the visceral and parietal surfaces of the pericardium is defined as pericarditis. It can evolve to excessive production of pericardial effusion if the speed of fluid accumulation is faster than the absorption. Acute pericarditis is rare in children but it can lead to circulatory collapse and death. It accounts for <0.2% of the emergency visits of children without prior heart diseases in tertiary pediatric emergency settings. The etiology of acute pericarditis varies depending on geography, and the most common etiology in children are bacterial infection, viral pericarditis,
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Lee, M. C., Y. C. Fung, R. Shabetai, and M. M. LeWinter. "Biaxial mechanical properties of human pericardium and canine comparisons." American Journal of Physiology-Heart and Circulatory Physiology 253, no. 1 (1987): H75—H82. http://dx.doi.org/10.1152/ajpheart.1987.253.1.h75.

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The two-dimensional mechanical properties of human pericardium from seven males undergoing coronary artery bypass surgery were studied. A 25-mm square piece of parietal pericardium overlying the right ventricle was excised. An approximately 8-mm square target was marked at the center, and its dimension was measured electrooptically. When immersed in physiological saline at 37 degrees C, the specimen was subjected to biaxial isotropic loading. Large deformations developed in the beginning of the loading; the pericardium became increasingly stiffer when load was increased, and then became almost
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Books on the topic "Parietal pericardium"

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Adlam, David. Pericardial disease. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0109.

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The pericardium forms a continuous sac around the heart, analogous to the pleura surrounding the lungs, and the peritoneum surrounding the abdominal viscera. Between the parietal and visceral layers of the serous pericardium is the pericardial space, which normally contains a small volume of pericardial fluid. The clinical spectrum of pericardial diseases can be divided into: pericarditis, caused by acute inflammation; pericardial effusion, or fluid accumulation in the pericardial space, leading to tamponade; and constrictive pericarditis, caused by chronic infiltration or inflammation leading
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Alexander, Kevin. Myocarditis and Pericarditis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0019.

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Infectious myocarditis is a primary, inflammatory cardiomyopathy that can lead to cardiomyocyte toxicity via direct myocyte invasion, toxin production, and/or stimulation of a chronic inflammatory response through antigenic mimicry. Its incidence is difficult to determine due to significant disease heterogeneity and the lack of a noninvasive gold standard for diagnosis. Often, the causative pathogen is not identified; in cases where it is, appropriate anti-infective agents may be used. Treatment is primarily supportive. Acute infectious pericarditis involves inflammation of the parietal and vi
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Book chapters on the topic "Parietal pericardium"

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Yucelsen, Can. "Approach to Pericarditis." In Thoracic Infections. Nobel Tip Kitabevleri, 2024. http://dx.doi.org/10.69860/nobel.9786053358930.11.

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Pericarditis is the inflammation of the parietal and visceral layers of the pericardium, presenting with characteristic chest pain, electrocardiographic changes, and a pericardial friction rub. It is the most frequent form of pericardial disease and a common cause of chest pain, particularly affecting males between 20-50 years. The etiology includes viral, bacterial, fungal, and parasitic infections, autoimmune diseases, trauma, and other conditions like uremia and hypothyroidism. The pathophysiology involves the activation of the inflammatory cascade leading to pericardial effusion, which can range from serous to hemorrhagic in nature. Acute pericarditis is characterized by intense inflammation and effusion, while chronic pericarditis may lead to fibrosis and constrictive pericarditis. Diagnosis relies on clinical evaluation, typical chest pain, EKG changes, and detection of pericardial effusion via echocardiography. Laboratory tests help rule out other conditions, and imaging techniques like CT and MRI can confirm the diagnosis. Treatment is generally symptomatic for viral/idiopathic cases, utilizing NSAIDs, colchicine, and sometimes corticosteroids. Purulent pericarditis requires surgical drainage and antibiotics, while tuberculous pericarditis needs specific anti-tuberculosis treatment. Pericardiocentesis is indicated for tamponade, and pericardiectomy for constriction. The prognosis is typically benign for idiopathic and viral pericarditis, although recurrent episodes can affect the quality of life. Acute pericarditis usually resolves without long-term damage, but complications such as cardiac tamponade and constrictive pericarditis can occur.
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Ross, Elizabeth M., Charles L. Mclntosh, and William C. Roberts. "“Massive” Calcification of a Right Ventricular Outflow Parietal Pericardial Patch in Tetralogy of Fallot." In Case Reports in Cardiology. CRC Press, 2023. http://dx.doi.org/10.1201/9781003409342-35.

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Silver, Marc A., Philip R. Orenburg, and William C. Roberts. "Severe Mitral Regurgitation Immediately After Mitral Valve Replacement with a Parietal Pericardial Bovine Bioprosthesis." In Case Reports in Cardiology. CRC Press, 2023. http://dx.doi.org/10.1201/9781003409281-28.

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Lilly, Leonard S. "Pericardial Disease." In The Brigham Intensive Review of Internal Medicine. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199358274.003.0081.

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The pericardium is a two-layered sac that surrounds the heart. It is composed of an outer stiff fibrous coat (the parietal pericardium) and a thin inner membrane that is adherent to the external surface of the heart (the visceral pericardium). The visceral pericardium reflects back at the level of the great vessel origins to form the inner lining of the parietal layer. The space between these two layers normally contains 15–50 mL of serous pericardial fluid, which permits the heart to contract in a minimum-friction environment. The major diseases of the pericardium are acute pericarditis, cardiac tamponade, and constrictive pericarditis.
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Melduni, Rowlens M. "Pericardial Disease." In Mayo Clinic Cardiology, 5th ed., edited by Joseph G. Murphy, Nandan S. Anavekar, Barry A. Boilson, Margaret A. Lloyd, Rekha Mankad, and Raymond C. Shields. Oxford University PressNew York, 2024. https://doi.org/10.1093/med/9780197599532.003.0075.

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Abstract The pericardium is a fibromembranous inelastic sac less than 2 mm in thickness that comprises 2 distinct layers. The outer inelastic fibrous layer (fibrous pericardium) anchors the heart in the mediastinum with attachments anterior to the manubrium and xiphoid process, posterior to the vertebral column, and inferior to the diaphragm. The inner serous double layer (serous pericardium) is divided into the visceral pericardium (epicardium) and the parietal pericardium.
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Klarich, Kyle W. "Pericardial Disease and Cardiac Tumors." In Mayo Clinic Internal Medicine Board Review. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190464868.003.0009.

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The space between the visceral and parietal pericardium normally contains 15 to 25 mL of clear fluid. The pericardium functions to prevent cardiac distention, limit cardiac displacement (by its attachment to neighboring structures), and protect the heart from inflammation. The chest pain of pericarditis is aggravated by movement of the trunk, inspiration, and coughing. The pain can be relieved by sitting up. Low-grade fever and malaise may occur.
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Klarich, Kyle W. "Pericardial Disease and Cardiac Tumors." In Mayo Clinic Internal Medicine Board Review, 12th ed., edited by Christopher M. Wittich, Thomas J. Beckman, Sara L. Bonnes, et al. Oxford University PressNew York, 2019. http://dx.doi.org/10.1093/med/9780190938369.003.0009.

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Abstract The pericardium consists of a fibrous sac and a serous membrane. The space between visceral and parietal pericardium normally contains 15 to 25 mL of clear fluid. The pericardium functions to prevent cardiac distention, limit cardiac displacement (by its attachment to neighboring structures), and protect the heart from inflammation. Metastatic tumors are far more common than primary cardiac tumors (>40-fold). The most frequent metastases to the heart are melanoma, lymphoma, and breast, lung, and esophageal tumors. Patients with metastatic malignancies have metastatic disease to the heart up to 15% of the time. More than one-half of patients with malignant melanoma have metastases to the heart. Metastatic disease to the heart carries a poor prognosis.
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Martinello, Richard A., and Michael Cappello. "Acute pericarditis." In Schlossberg's Clinical Infectious Disease, edited by Cheston B. Cunha. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780190888367.003.0038.

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This chapter investigates acute pericarditis. The pericardium serves to protect the heart from physiologic changes in intracardiac pressure related to respiration and postural change, and it may also augment the mechanical function of the cardiac chambers. The pericardium is composed of a visceral layer that directly adheres to the epicardium and an outer parietal layer. These two layers are separated by 10 to 35 mL of serous fluid. Pericarditis refers to the inflammation of these tissues. It may be acute, recurrent, or chronic, and can be due to a wide array of etiologies. Both infectious and noninfectious processes have been identified as causes of pericarditis. Most cases are caused by viruses, are self-limited, and the specific pathogen is often unidentified. Urgent drainage of pericardial fluid should be considered in any patient with a possible diagnosis of purulent pericarditis or if hemodynamic compromise is identified. The outcome in these patients is generally poor without drainage, even when appropriate antibiotics are administered.
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Grandhi, Ravi K., and Alaa Abd-Elsayed. "Cardiac Tamponade." In Advanced Anesthesia Review, edited by Alaa Abd-Elsayed. Oxford University PressNew York, 2023. http://dx.doi.org/10.1093/med/9780197584521.003.0129.

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Abstract Cardiac tamponade is a medical emergency that requires close attention. It can be acute, subacute, occult, or regional. Etiologies of pericardial tamponade vary based on whether they are inflammatory or noninflammatory (1). The pericardium is composed of two layers of tissue, the serous visceral and fibrous parietal layers, which have less than 50 mL of serous fluid. Presentation will depend on the amount and rate of accumulation of fluid. Diagnosis can be made by history, physical examination, and imaging studies. Treatment will typically require evacuation of the fluid to reduce the pressure around the heart. Treatment goals include maintaining preload, afterload, and contractility.
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Atkinson, Martin E. "The heart, pericardium, and mediastinum." In Anatomy for Dental Students. Oxford University Press, 2013. http://dx.doi.org/10.1093/oso/9780199234462.003.0019.

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The heart, the arteries and veins leaving and entering the heart which are usually referred to as the great vessels, the trachea and bronchi, the oesophagus, and the vagus and phrenic nerves and sympathetic chains occupy the mediastinum , the area in the middle of the thoracic cavity between the two pleural sacs. The anteroposterior dimension of the thorax is narrowest in the mediastinum because of the presence of the thoracic vertebrae posteriorly. Laterally, the pleural sacs enclosing the lungs extend much further back alongside the vertebrae in the areas known as the paravertebral gutters. The great vessels enter and leave the superior aspect of the heart. The large veins draining the head, neck, and arms lie most superficially; they unite to form the superior vena cava that enters the right atrium of the heart. These veins overlie the two large arteries exiting the heart, the aorta, and pulmonary trunk. The aorta has a short ascending part, then forms the aortic arch passing backwards and to the left before continuing down the posterior wall of the thorax as the descending thoracic aorta. The subclavian and common carotid arteries, supplying blood to the arms and head and neck, respectively, arise from the aortic arch. The oesophagus is the deepest structure lying on the vertebrae and the trachea and main bronchi lie superficial to it. The sympathetic chains lie lateral to the vertebral bodies and the vagus and phrenic nerves are in intermediate positions. All these structures will be described in more detail in the rest of this chapter. The mediastinum is divided, for descriptive convenience, into the superior and inferior mediastinum. Figure 12.1 shows the imaginary line of division joining the sternal angle and the intervertebral disc below T4 that demarcates the boundaries of the superior and inferior of the mediastinum. The superior mediastinum occupies the space between the thoracic inlet above and the imaginary horizontal plane. The inferior mediastinum lies below that line and extends as far as the diaphragm. The lateral borders of both subdivisions of the mediastinum are the parietal pleura covering the medial aspect of the lungs, the mediastinal pleura.
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Conference papers on the topic "Parietal pericardium"

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Fonseca, Abner Lourenço da. "Constrictive Pericarditis." In II INTERNATIONAL SEVEN MULTIDISCIPLINARY CONGRESS. Seven Congress, 2023. http://dx.doi.org/10.56238/homeinternationalanais-079.

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Abstract The pericardium is a fibrous membrane that surrounds the heart and is composed of two components: visceral pericardium and parietal pericardium. Under physiological conditions, it performs important functions, such as lubrication, which minimizes friction between the organ and adjacent structures, limits intrathoracic cardiac motion, aids in filling the cardiac chambers, and participates in the balancing between the right and left ventricles during diastole and systole interactions. Constrictive Pericarditis occurs when the pericardium is thickened, fibrotic and often calcified, signi
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