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Relevant bibliographies by topics / Parkinson's disease; Neuroscience / Journal articles

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Journal articles on the topic 'Parkinson's disease; Neuroscience'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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1

Dauer, William, and Serge Przedborski. "Parkinson's Disease." Neuron 39, no. 6 (2003): 889–909. http://dx.doi.org/10.1016/s0896-6273(03)00568-3.

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2

Allain, Hervé, Danièle Bentué-Ferrer, and Yvette Akwa. "Disease-modifying drugs and Parkinson's disease." Progress in Neurobiology 84, no. 1 (2008): 25–39. http://dx.doi.org/10.1016/j.pneurobio.2007.10.003.

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3

Wider, Christian, Sarah J. Lincoln, Michael G. Heckman, et al. "Phactr2 and Parkinson's disease." Neuroscience Letters 453, no. 1 (2009): 9–11. http://dx.doi.org/10.1016/j.neulet.2009.02.009.

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4

Agid, Y., and F. Javoy-Agid. "Peptides and Parkinson's disease." Trends in Neurosciences 8 (January 1985): 30–35. http://dx.doi.org/10.1016/0166-2236(85)90012-8.

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5

William Langston, J. "MPTP and parkinson's disease." Trends in Neurosciences 8 (January 1985): 79–83. http://dx.doi.org/10.1016/0166-2236(85)90031-1.

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6

Robbins, Trevor W., and Roshan Cools. "Cognitive deficits in Parkinson's disease: A cognitive neuroscience perspective." Movement Disorders 29, no. 5 (2014): 597–607. http://dx.doi.org/10.1002/mds.25853.

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7

Dekker, M. "Mutations in the hemochromatosis gene (HFE), Parkinson's disease and parkinsonism." Neuroscience Letters 348, no. 2 (2003): 117–19. http://dx.doi.org/10.1016/s0304-3940(03)00713-4.

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8

Venhovens, J., J. Meulstee, B. R. Bloem, and W. I. M. Verhagen. "Neurovestibular analysis and falls in Parkinson's disease and atypical parkinsonism." European Journal of Neuroscience 43, no. 12 (2016): 1636–46. http://dx.doi.org/10.1111/ejn.13253.

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9

Vanhauwaert, Roeland, and Patrik Verstreken. "Flies with Parkinson's disease." Experimental Neurology 274 (December 2015): 42–51. http://dx.doi.org/10.1016/j.expneurol.2015.02.020.

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10

Okai, David. "Neuropsychiatric sequelae of Parkinson's disease: what every clinician needs to know." BJPsych Advances 26, no. 6 (2020): 343–45. http://dx.doi.org/10.1192/bja.2020.70.

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SUMMARYJones et al's review of the assessment and management of the neuropsychiatric manifestations of Parkinson's disease serves as a practical guide to clinicians. This commentary outlines some of the underlying neuroscience and psychological sequelae of this range of conditions, offering a takeaway message to the clinician with an interest in Parkinson's neuropsychiatry.

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11

Pan, Weidong, Shin Kwak, Yun Liu, et al. "Traditional Chinese Medicine Improves Activities of Daily Living in Parkinson's Disease." Parkinson's Disease 2011 (2011): 1–7. http://dx.doi.org/10.4061/2011/789506.

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We evaluated the effects of a traditional Chinese medicine (TCM), named Zeng-xiao An-shen Zhi-chan 2 (ZAZ2), on patients with Parkinson's disease (PD). Among 115 patients with idiopathic PD enrolled (mean age, 64.7 ± 10.2 years old), 110 patients (M = 65, F = 45; mean age, 64.9 ± 10.7 years old) completed the study. Patients took either ZAZ2 () or placebo granule () in a blind manner for 13 weeks while maintaining other anti-Parkinson medications unchanged. All participants wore a motion logger, and we analyzed the power-law temporal autocorrelation of the motion logger records taken on 3 occa

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12

Koçoğlu, Koray, Gülden Akdal, Berril Dönmez Çolakoğlu, et al. "The effect of directional social cues on saccadic eye movements in Parkinson’s disease." Experimental Brain Research 239, no. 7 (2021): 2063–75. http://dx.doi.org/10.1007/s00221-021-06034-7.

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AbstractThere is growing interest in how social processes and behaviour might be affected in Parkinson’s disease. A task which has been widely used to assess how people orient attention in response to social cues is the spatial cueing task. Socially relevant directional cues, such as a picture of someone gazing or pointing to the left or the right have been shown to cause orienting of visual attention in the cued direction. The basal ganglia may play a role in responding to such directional cues, but no studies to date have examined whether similar social cueing effects are seen in people with

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13

Praamstra, P., and F. M. Plat. "Failed Suppression of Direct Visuomotor Activation in Parkinson's Disease." Journal of Cognitive Neuroscience 13, no. 1 (2001): 31–43. http://dx.doi.org/10.1162/089892901564153.

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The response times in choice-reaction tasks are faster when the relative spatial positions of stimulus and response match than when they do not match, even when the spatial relation is irrelevant to response choice. This spatial stimulus—response (S-R) compatibility effect (i.e., the Simon effect) is attributed in part to the automatic activation of spatially corresponding responses, which need to be suppressed when the spatial location of stimulus and correct response do not correspond. The present study tested patients with Parkinson's disease and healthy control subjects in a spatial S-R co

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14

Rosenthal, Arnon. "Auto Transplants for Parkinson's Disease?" Neuron 20, no. 2 (1998): 169–72. http://dx.doi.org/10.1016/s0896-6273(00)80445-6.

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15

Kingwell, Katie. "New leads for Parkinson's disease." Nature Reviews Neuroscience 11, no. 10 (2010): 667. http://dx.doi.org/10.1038/nrn2918.

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16

Rees, Richard Nathaniel, Alastair John Noyce, and Anette Schrag. "The prodromes of Parkinson's disease." European Journal of Neuroscience 49, no. 3 (2018): 320–27. http://dx.doi.org/10.1111/ejn.14269.

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17

Golbe, Lawrence I. "The Genetics of Parkinson's Disease." Reviews in the Neurosciences 4, no. 1 (1993): 1–16. http://dx.doi.org/10.1515/revneuro.1993.4.1.1.

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18

Barker, Roger. "Environmental toxins and Parkinson's disease." Trends in Neurosciences 12, no. 5 (1989): 182. http://dx.doi.org/10.1016/0166-2236(89)90066-0.

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19

Cools, Alexander Rudolf. "Synaptic homeostasis and Parkinson's disease." Trends in Neurosciences 14, no. 5 (1991): 182–83. http://dx.doi.org/10.1016/0166-2236(91)90100-9.

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20

Vázquez-Vélez, Gabriel E., and Huda Y. Zoghbi. "Parkinson's Disease Genetics and Pathophysiology." Annual Review of Neuroscience 44, no. 1 (2021): 87–108. http://dx.doi.org/10.1146/annurev-neuro-100720-034518.

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Parkinson's disease (PD) is a common neurodegenerative disorder characterized by degeneration of the substantia nigra pars compacta and by accumulation of α-synuclein in Lewy bodies. PD is caused by a combination of environmental factors and genetic variants. These variants range from highly penetrant Mendelian alleles to alleles that only modestly increase disease risk. Here, we review what is known about the genetics of PD. We also describe how PD genetics have solidified the role of endosomal, lysosomal, and mitochondrial dysfunction in PD pathophysiology. Finally, we highlight how all thre

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21

Smith, Lisa M., and Louise C. Parr-Brownlie. "A neuroscience perspective of the gut theory of Parkinson's disease." European Journal of Neuroscience 49, no. 6 (2018): 817–23. http://dx.doi.org/10.1111/ejn.13869.

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22

Collier, T. "Primate models of Parkinson's disease." Experimental Neurology 183, no. 2 (2003): 258–62. http://dx.doi.org/10.1016/s0014-4886(03)00246-2.

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23

Koller, William C. "Neuroprotective Therapy for Parkinson's Disease." Experimental Neurology 144, no. 1 (1997): 24–28. http://dx.doi.org/10.1006/exnr.1996.6383.

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24

Barcia, Carlos, Andrés Fernández Barreiro, Máximo Poza, and María-Trinidad Herrero. "Parkinson's disease and inflammatory changes." Neurotoxicity Research 5, no. 6 (2003): 411–17. http://dx.doi.org/10.1007/bf03033170.

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25

Van Laar, Victor S., and Sarah B. Berman. "Mitochondrial dynamics in Parkinson's disease." Experimental Neurology 218, no. 2 (2009): 247–56. http://dx.doi.org/10.1016/j.expneurol.2009.03.019.

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26

Fales, Christina L., Zeba F. Vanek, and Barbara J. Knowlton. "Backward inhibition in Parkinson's disease." Neuropsychologia 44, no. 7 (2006): 1041–49. http://dx.doi.org/10.1016/j.neuropsychologia.2005.11.002.

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27

Brundin, Patrik, and J. William Langston. "Journal of Parkinson's Disease." Journal of Parkinson's Disease 1, no. 1 (2011): 1. http://dx.doi.org/10.3233/jpd-2011-10022.

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28

Ishizaki, F., T. Harada, and S. Nakamura. "Diurnal blood pressure variation in Parkinson's disease and vascular parkinsonism." Journal of the Autonomic Nervous System 43 (April 1993): 87. http://dx.doi.org/10.1016/0165-1838(93)90237-o.

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29

Vance, Jeffery M., Syed Ali, Walter G. Bradley, Carlos Singer, and Donato A. Di Monte. "Gene–environment interactions in Parkinson's disease and other forms of parkinsonism." NeuroToxicology 31, no. 5 (2010): 598–602. http://dx.doi.org/10.1016/j.neuro.2010.04.007.

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30

Haehner, Antje, Thomas Hummel, and Heinz Reichmann. "Olfactory Loss in Parkinson's Disease." Parkinson's Disease 2011 (2011): 1–6. http://dx.doi.org/10.4061/2011/450939.

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Impairment of olfaction is a characteristic and early feature of Parkinson's disease. Recent data indicate that >95% of patients with Parkinson's disease present with significant olfactory loss. Deficits in the sense of smell may precede clinical motor symptoms by years and can be used to assess the risk for developing Parkinson's disease in otherwise asymptomatic individuals. This paper summarizes the available information about olfactory function in Parkinson's disease, indicating the advantageous use of olfactory probes in early and differential diagnosis.

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31

Scholz, O. B., and M. Sastry. "Memory Characteristics in Parkinson's Disease." International Journal of Neuroscience 27, no. 3-4 (1985): 229–34. http://dx.doi.org/10.3109/00207458509149768.

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32

Sandyk, Reuven, and Robert P. Iacono. "The hypothalamus in parkinson's disease." International Journal of Neuroscience 33, no. 3-4 (1987): 257–59. http://dx.doi.org/10.3109/00207458708987409.

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33

Sandyk, Reuven. "Autoimmune Mechanisms in Parkinson's Disease." International Journal of Neuroscience 48, no. 3-4 (1989): 369–72. http://dx.doi.org/10.3109/00207458909002183.

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34

Raskin, Sarah A., Joan C. Borod, Jeanette Wasserstein, Ivan Bodis-Wollner, Lisa Coscia, and Melvin D. Yahr. "Visuospatial Orientation in Parkinson's Disease." International Journal of Neuroscience 51, no. 1-2 (1990): 9–18. http://dx.doi.org/10.3109/00207459009000503.

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35

Sandyk, Reuven. "Delusional Depression in Parkinson's Disease." International Journal of Neuroscience 70, no. 1-2 (1993): 69–74. http://dx.doi.org/10.3109/00207459309000562.

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36

Hammerstad, J. P., K. Elliott, E. Mak, M. Schulzer, S. Calne, and D. B. Calne. "Tendon jerks in Parkinson's disease." Journal of Neural Transmission - Parkinsons Disease and Dementia Section 8, no. 1-2 (1994): 123–30. http://dx.doi.org/10.1007/bf02250923.

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37

Hattori, Nobutaka. "Mitochondrial dysfucntion in Parkinson's disease." Neuroscience Research 58 (January 2007): S15. http://dx.doi.org/10.1016/j.neures.2007.06.088.

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38

vinh quôc Luong, Khanh, and Lan Thi Hoàng Nguyên. "Vitamin D and Parkinson's disease." Journal of Neuroscience Research 90, no. 12 (2012): 2227–36. http://dx.doi.org/10.1002/jnr.23115.

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39

Sherer, Todd B., Ranjita Betarbet, and J. Timothy Greenamyre. "Environment, Mitochondria, and Parkinson's Disease." Neuroscientist 8, no. 3 (2002): 192–97. http://dx.doi.org/10.1177/1073858402008003004.

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40

Huang, Yue, Linda Cheung, Dominic Rowe, and Glenda Halliday. "Genetic contributions to Parkinson's disease." Brain Research Reviews 46, no. 1 (2004): 44–70. http://dx.doi.org/10.1016/j.brainresrev.2004.04.007.

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41

Horellou, Philippe, and Jacques Mallet. "Gene therapy for Parkinson's disease." Molecular Neurobiology 15, no. 2 (1997): 241–56. http://dx.doi.org/10.1007/bf02740636.

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42

Potič, Jagoda, and Nikola Volf. "Compression neuropathy in Parkinson's disease." Electroencephalography and Clinical Neurophysiology 61, no. 3 (1985): S110. http://dx.doi.org/10.1016/0013-4694(85)90436-5.

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43

Barbato, L., S. Rinalduzzi, M. Laurenti, S. Ruggieri, and N. Accornero. "Color VEPs in Parkinson's disease." Electroencephalography and Clinical Neurophysiology/Evoked Potentials Section 92, no. 2 (1994): 169–72. http://dx.doi.org/10.1016/0168-5597(94)90057-4.

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44

Vinish, Monika, and Jennie Milstein. "Non-motor aspects of Parkinson's disease." Annals of Neurosciences 16, no. 4 (2009): 176–79. http://dx.doi.org/10.5214/ans.0972-7531.0916410.

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45

Vinish, Monika, and Jennie Milstein. "Non-motor aspects of Parkinson's disease." Annals of Neurosciences 16, no. 4 (2009): 176–79. http://dx.doi.org/10.5214/ans.0972.7531.2009.160410.

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46

Jenner, Peter. "Parkinson's disease, pesticides and mitochondrial dysfunction." Trends in Neurosciences 24, no. 5 (2001): 245–46. http://dx.doi.org/10.1016/s0166-2236(00)01789-6.

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47

Westerlund, Marie, Barry Hoffer, and Lars Olson. "Parkinson's disease: Exit toxins, enter genetics." Progress in Neurobiology 90, no. 2 (2010): 146–56. http://dx.doi.org/10.1016/j.pneurobio.2009.11.001.

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48

Haelterman, Nele A., Wan Hee Yoon, Hector Sandoval, Manish Jaiswal, Joshua M. Shulman, and Hugo J. Bellen. "A Mitocentric View of Parkinson's Disease." Annual Review of Neuroscience 37, no. 1 (2014): 137–59. http://dx.doi.org/10.1146/annurev-neuro-071013-014317.

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49

Davis, Nick. "REDUCED INTRACORTICAL INHIBITION IN PARKINSON'S DISEASE." Neuroreport 12, no. 15 (2001): A87—A88. http://dx.doi.org/10.1097/00001756-200110290-00003.

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50

Engelender, Simone, and Ole Isacson. "The Threshold Theory for Parkinson's Disease." Trends in Neurosciences 40, no. 1 (2017): 4–14. http://dx.doi.org/10.1016/j.tins.2016.10.008.

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