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Journal articles on the topic 'Pars planitis'

Author: Grafiati
Published: 4 June 2021
Last updated: 16 June 2025

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1

Malinowski, Susan M., James C. Folk, and Jose S. Pulido. "Pars planitis." Current Opinion in Ophthalmology 5, no. 3 (1994): 72–82. http://dx.doi.org/10.1097/00055735-199406000-00013.

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2

Raja, Sharath C., Douglas A. Jabs, J. P. Dunn, et al. "Pars planitis." Ophthalmology 106, no. 3 (1999): 594–99. http://dx.doi.org/10.1016/s0161-6420(99)90122-7.

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3

Lee, Andrew G. "Familial pars planitis." Ophthalmic Genetics 16, no. 1 (1995): 17–19. http://dx.doi.org/10.3109/13816819509057849.

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4

Kim, Yong-Kyu, Wontae Yoon, Jae Kyoun Ahn, and Sung Pyo Park. "Scleral Buckling for Rhegmatogenous Retinal Detachment Associated with Pars Planitis." Journal of Ophthalmology 2016 (2016): 1–5. http://dx.doi.org/10.1155/2016/4538193.

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Purpose. To evaluate the surgical outcome of scleral buckling (SB) in rhegmatogenous retinal detachment (RRD) patients associated with pars planitis.Methods. Retrospective review of RRD patients (32 eyes of pars planitis RRD and 180 eyes of primary RRD) who underwent SB. We compared primary and final anatomical success rates and visual outcomes between two groups.Results. Primary and final anatomical success were achieved in 25 (78.1%) and 31 (96.8%) eyes in the pars planitis RRD group and in 167 eyes (92.7%) and 176 eyes (97.7%) in primary RRD group, respectively. Both groups showed significant visual improvement (p<0.001) and there were no significant differences in final visual acuity. Pars planitis RRD group was associated with higher rate of postoperative proliferative vitreoretinopathy (PVR) development (12.5% versus 2.8%,p=0.031). Pars planitis and high myopia were significant preoperative risk factors and pseudophakia was borderline risk for primary anatomical failure after adjusting for various clinical factors.Conclusions. Pars planitis associated RRD showed inferior primary anatomical outcome after SB due to postoperative PVR development. However, final anatomical and visual outcomes were favorable. RRD cases associated with pars planitis, high myopia, and pseudophakia might benefit from different surgical approaches, such as combined vitrectomy and SB.
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5

Malalis, Julia F., Pooja Bhat, Michael Shapiro, and Debra A. Goldstein. "Retinoschisis in Pars Planitis." Ocular Immunology and Inflammation 25, no. 3 (2016): 344–48. http://dx.doi.org/10.3109/09273948.2015.1125511.

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6

MAGGON, RAKESH. "INTERMEDIATE UVEITIS PARS PLANITIS." Medical Journal Armed Forces India 57, no. 1 (2001): 84–85. http://dx.doi.org/10.1016/s0377-1237(01)80106-4.

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7

Bekir, Necdet A., and Ümit Darıcı. "PARS PLANIT." European Journal of Therapeutics 1, no. 2 (1990): 268–73. http://dx.doi.org/10.58600/eurjther.19900102-491.

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8

Garcia-Feijoo, Julian, Mario Martin-Carbajo, Jose M. Benitez del Castillo, and Julian Garcia-Sanchez. "Ultrasound Biomicroscopy in Pars Planitis." American Journal of Ophthalmology 121, no. 2 (1996): 214–15. http://dx.doi.org/10.1016/s0002-9394(14)70590-2.

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9

TESSLER, HOWARD H. "Pars Planitis and Autoimmune Endotheliopathy." American Journal of Ophthalmology 103, no. 4 (1987): 599. http://dx.doi.org/10.1016/s0002-9394(14)74294-1.

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10

Aaberg, Thomas M. "The Enigma of Pars Planitis." American Journal of Ophthalmology 103, no. 6 (1987): 828–30. http://dx.doi.org/10.1016/s0002-9394(14)74403-4.

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11

Khodadoust, Ali A., Yadolah Karnama, Kathleen M. Stoessel, and James E. Puklin. "Pars Planitis and Autoimmune Endotheliopathy." American Journal of Ophthalmology 102, no. 5 (1986): 633–39. http://dx.doi.org/10.1016/0002-9394(86)90537-4.

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12

PIVETTI-PEZZI, PAOLA, and STEFANO TAMBURI. "Pars Planitis and Autoimmune Endotheliopathy." American Journal of Ophthalmology 104, no. 3 (1987): 311–12. http://dx.doi.org/10.1016/0002-9394(87)90433-8.

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13

Tejada, P., A. Sanz, and D. Criado. "Pars planitis in a family." International Ophthalmology 18, no. 2 (1994): 111–13. http://dx.doi.org/10.1007/bf00919250.

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14

Przeździecka-Dołyk, Joanna, Agnieszka Węgrzyn, Anna Turno-Kręcicka, and Marta Misiuk-Hojło. "Immunopathogenic Background of Pars Planitis." Archivum Immunologiae et Therapiae Experimentalis 64, no. 2 (2015): 127–37. http://dx.doi.org/10.1007/s00005-015-0361-y.

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15

Greiner, K. H., D. W. L. Wilson, J. Thomson, D. J. Kilmartin, S. J. Urbaniak, and J. V. Forrester. "Genetic Polymorphism of HLA DR in a Scottish Population of Patients with Pars Planitis." European Journal of Ophthalmology 13, no. 5 (2003): 433–38. http://dx.doi.org/10.1177/112067210301300503.

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Purpose Human leucocyte antigen (HLA) class II influences the immunological susceptibility for a variety of diseases including many types of non-infectious intraocular inflammation. Previous studies on North American patients with pars planitis, a subtype of intermediate uveitis, reported an increased prevalence of HLA DR15 in this population. In contrast, two European studies could not find an association between HLA DR2 or its allelic subtype DR15 and various forms of intermediate uveitis. We therefore investigated the genotype frequency of HLA DR alleles in a Scottish population of patients with typical pars planitis. Methods Twenty patients with pars planitis were identified from the uveitis database of Grampian Universitiy Hospitals. Only patients with bilateral vitritis and snowbanks in at least one eye in the absence of systemic disease were included in the study. Fifteen patients and 34 healthy controls underwent HLA DR genotyping for all DRB genes using PCR sequence specific primers. Results HLA DR15 was found in 13% of patients with pars planitis and in 24% of controls. There was no statistically significant difference between these two groups. Furthermore, the frequencies of HLA DR 1, 3–14, and 16 did not differ significantly between patients and controls. Conclusions There appears to be no association between the occurrence of pars planitis and the HLA DR15 or other known HLA DR genotypes in Scottish patients. However, the small sample size limits the power of this study.
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16

Duinkerke-Eerola, K. U., A. Pinckers, and J. R. M. Cruysberg. "Pars planitis in father and son." Ophthalmic Paediatrics and Genetics 11, no. 4 (1990): 305–8. http://dx.doi.org/10.3109/13816819009015718.

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17

Phillips, William B., Robert L. Bergren, and J. Arch McNamara. "Pars Planitis Presenting With Vitreous Hemorrhage." Ophthalmic Surgery, Lasers and Imaging Retina 24, no. 9 (1993): 630–31. http://dx.doi.org/10.3928/1542-8877-19930901-15.

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18

García, L. Arellanes, G. Ortiz-Ponce, and C. Recillas-Gispert. "Peripheral Corneal Endotheliopathy and Pars Planitis." Ocular Immunology and Inflammation 4, no. 3 (1996): 135–38. http://dx.doi.org/10.3109/09273949609079646.

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19

Özdemir, Hakan, Murat Karaçorlu, Serra Arf Karaçorlu, and Fevzi Şentürk. "Serous Macular Detachment in Pars Planitis." Türk Oftalmoloji Dergisi 40, no. 3 (2010): 184–86. http://dx.doi.org/10.4274/tod.40.184.

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20

Okinami, Satoshi, Mitsuko Sunakawa, Ikki Arai, Masayoshi lwaki, Mika Nihira, and Nobuchika Ogino. "Treatment of Pars planitis with Cryotherapy." Ophthalmologica 202, no. 4 (1991): 180–86. http://dx.doi.org/10.1159/000310192.

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21

Mehta, Sonia, Luxme Hariharan, Allen C. Ho, and John H. Kempen. "Peripapillary choroidal neovascularization in pars planitis." Journal of Ophthalmic Inflammation and Infection 3, no. 1 (2013): 13. http://dx.doi.org/10.1186/1869-5760-3-13.

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22

KHODADOUST, ALI A., YADSLAH KARNAMA, KATHLEEN M. STOESSEL, and JAMES E. PUKLIN. "Pars Planitis and Autoimmune Endotheliopathy: Reply." American Journal of Ophthalmology 103, no. 4 (1987): 599–600. http://dx.doi.org/10.1016/s0002-9394(14)74295-3.

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23

Michelson, Joseph B., Mitchell H. Friedlaender, and Robert A. Nozik. "Lens Implant Surgery in Pars Planitis." Ophthalmology 97, no. 8 (1990): 1023–26. http://dx.doi.org/10.1016/s0161-6420(90)32470-3.

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24

Khodadoust, Ali A., Yadolah Karnama, Kathleen M. Stoessel, and James E. Puklin. "Pars Planitis and Autoimmune Endotheliopathy: Reply." American Journal of Ophthalmology 104, no. 3 (1987): 312. http://dx.doi.org/10.1016/0002-9394(87)90434-x.

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25

Holland, Gary N. "The Enigma of Pars Planitis, Revisited." American Journal of Ophthalmology 141, no. 4 (2006): 729–30. http://dx.doi.org/10.1016/j.ajo.2005.12.049.

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26

SUH, DONNY W., JOSE S. PULIDO, LEE M. JAMPOL, LAWRENCE P. CHONG та MATHEW THOMAS. "COATSʼ-LIKE RESPONSE IN PARS PLANITIS". Retina 19, № 1 (1999): 79–80. http://dx.doi.org/10.1097/00006982-199901000-00015.

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27

Henderly, Dale E., Robert S. Haymond, Narsing A. Rao, and Ronald E. Smith. "The Significance of the Pars Plana Exudate in Pars Planitis." American Journal of Ophthalmology 103, no. 5 (1987): 669–71. http://dx.doi.org/10.1016/s0002-9394(14)74327-2.

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28

Haimovici, Robert, Susan L. Lightman, and Alan C. Bird. "Familial pars planitis and dominant optic atrophy." Ophthalmic Genetics 18, no. 1 (1997): 43–45. http://dx.doi.org/10.3109/13816819709057883.

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29

KALINA, PAUL H., JOHN M. PACH, HELMUT BUETTNER, and DENNIS M. ROBERTSON. "NEOVASCULARIZATION OF THE DISC IN PARS PLANITIS." Retina 10, no. 4 (1990): 269–73. http://dx.doi.org/10.1097/00006982-199010000-00008.

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30

Kaufman, Adam H., and C. Stephen Foster. "Cataract Extraction in Patients with Pars Planitis." Ophthalmology 100, no. 8 (1993): 1210–17. http://dx.doi.org/10.1016/s0161-6420(93)31503-4.

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31

Rafael, Torres dos Santos1* Lucas Magno Goedert1 Fernanda Romagnole Pugliese4 Ian Caldeira Ruppen2 Leandro Hideki Otani3 Felicia Satie Ibuki Otani3 Alana Reigota da Costa Rosa2 Guilherme Enzo Giovanelli Mansano2 Vitor Augusto Olivari do Carmo2 Luana Schiavon Dib2 Giovanna Gregório Gonçalves Pereira2 Heloise de Arruda2 Karolina Bogusch Cava2 Matheus Bitetti Paiva2 Victor Corrêa de Oliveira Neto2 and Yasmin Cavatorta Jannani2. "Pars Planitis Associated with Cystoid Macular Edema with Visual Acuity Preservation: A Case Report." Mega Journal of Case Reports 8, no. 2 (2025): 2001–5. https://doi.org/10.5281/zenodo.14935046.

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<strong>Abstract</strong> Pars Planitis, a form of intermediate uveitis, is characterized by idiopathic inflammation of the posterior segment of the eye. Among common complications, cystoid macular edema (CME) can occur, causing structural changes in the retina, though it does not always significantly impact visual acuity. This article discusses possible pathophysiological explanations, available therapeutic options, and the importance of clinical follow-up, emphasizing cases where visual function is maintained. The review is based on recent clinical and experimental studies, offering a critical analysis of the challenges in managing the disease. <strong>Keywords</strong><strong>:</strong> Pars Planitis; Macular Edema; Uveitis; Inflammation
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32

Flores-Huerta, Nadia, Karla Tovar-Hernández, and Matilde Ruiz-Cruz. "Pars planitis: una patología ocular que debe informarse." Revista Ciencia UANL 28, no. 129 (2025): 44–53. https://doi.org/10.29105/cienciauanl28.129-6.

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La pars planitis (PP) es una inflamación del ojo (uveítis intermedia) caracterizada por presentar queratopatía en banda, inflamación en la cámara anterior y vítreo, condensaciones vítreas (“copos de nieve”), exudados en la pars plana (“bancos de nieve”), así como vasculitis de retina e inflamación del nervio óptico.
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33

Listernick, Robert. "A 12-Year-Old Boy with Pars Planitis." Pediatric Annals 44, no. 11 (2015): 462–64. http://dx.doi.org/10.3928/00904481-20151112-02.

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34

Potter, Michael J., Stephanie O. Myckatyn, Alan L. Maberley, and Ann S. Lee. "Vitrectomy for pars planitis complicated by vitreous hemorrhage." American Journal of Ophthalmology 131, no. 4 (2001): 514–15. http://dx.doi.org/10.1016/s0002-9394(00)00844-8.

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35

Grzybowski, Andrzej, Piotr Kanclerz, and Uwe Pleyer. "Challenges with cataract surgery in pars planitis patients." Graefe's Archive for Clinical and Experimental Ophthalmology 255, no. 8 (2017): 1483–84. http://dx.doi.org/10.1007/s00417-017-3698-6.

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36

Kosmaczewska, Agata, Joanna Przeździecka-Dołyk, Anna Turno-Kręcicka та ін. "Imbalance in PB IL-17-Secreting and Regulatory Cells in Pars Planitis Is Associated with Dysregulation of IFN-γ-Secreting Cells, Especially in Patients with Clinical Complications". Mediators of Inflammation 2020 (31 липня 2020): 1–9. http://dx.doi.org/10.1155/2020/9175083.

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Background/Purpose. Pars planitis (PP) is a chronic intermediate uveitis of undefined cause, although autoimmune etiology is strongly suggested. This study was undertaken to evaluate circulating pro- and anti-inflammatory lymphocyte subpopulations in patients with PP. A better understanding of immune alterations in PP may contribute to the development of an appropriate biologic treatment approach. Material and Methods. In our case-control study, samples of peripheral blood from 15 patients with pars planitis and 17 healthy subjects were collected. The patients underwent a full ophthalmological evaluation including macular optical coherence tomography (OCT) and submacular choroid (with enhanced deep imaging) with the SD-OCT Spectralis system. By flow cytometry, we assessed lymphocyte subpopulations as follows: Th1 subset: CD3+CD8-IFN-γ+ and CCR4-CXCR3+IFN-γ+ cells, Th17 subset: CD3+CD8-IL-17+ and CCR4+CCR6+IL-17+ cells, T regulatory (Treg): CD4+CD25hiCD127- cells, T suppressor (Tsup): CD8+CD28-FOXP3+ cells, and B regulatory (Breg): CD19+CD24hiCD38hi cells. The results are presented as the median percentage of immune cells in comparison to those obtained from the healthy control group. Results. In patients, an increase in the population of Th17-secreting cells negatively correlated with the abundance of both IFN-γ-producing and T regulatory as well as suppressor cells, regarding all the phenotypes studied. Although a strong dependence of the PB Th1 cell compartment on the duration of the disease was observed, it was limited to the subgroup of patients with macular edema only. The frequency of B regulatory cells was unchanged compared to controls. Conclusions. In pars planitis, the alterations in lymphocyte cell distribution affect primarily the T cell repertoire. The imbalance in PB Th1/Th17/Treg cells creates proinflammatory conditions, strengthening the suggestion that the immune background may play a role in pars planitis pathogenesis. Also, circulating Th1 level may be of potential clinical relevance in terms of prediction of a more severe course of the disease.
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37

Tugal-Tutkun, Ilknur, PinarCakar Ozdal, and Nilufer Berker. "Pars planitis: Epidemiology, clinical characteristics, management and visual prognosis." Journal of Ophthalmic and Vision Research 10, no. 4 (2015): 469. http://dx.doi.org/10.4103/2008-322x.176897.

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38

Romero, Ricardo, Jesus Peralta, Elena Sendagorta, and Jose Abelairas. "Pars Planitis in Children: Epidemiologic, Clinical, and Therapeutic Characteristics." Journal of Pediatric Ophthalmology and Strabismus 44, no. 5 (2007): 288–93. http://dx.doi.org/10.3928/01913913-20070901-03.

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39

Prieto, J. F., E. Dios, J. M. Gutierrez, A. Mayo, M. Calonge, and J. M. Herreras. "Pars planitis: epidemiology, treatment, and association with multiple sclerosis." Ocular Immunology and Inflammation 9, no. 2 (2001): 93–102. http://dx.doi.org/10.1076/ocii.9.2.93.3975.

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40

Chen, P. P., and L. P. Chong. "Coats'-like response in a patient with pars planitis." British Journal of Ophthalmology 80, no. 7 (1996): 675–76. http://dx.doi.org/10.1136/bjo.80.7.675.

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41

Oruc, Sema, Brian F. Duffy, Thalachallour Mohanakumar, and Henry J. Kaplan. "The association of HLA class II with pars planitis." American Journal of Ophthalmology 131, no. 5 (2001): 657–59. http://dx.doi.org/10.1016/s0002-9394(00)00863-1.

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42

Carmen Jimenez-Martinez, Maria, Marisela Linares, Stephanie Voorduin, Miguel Pedroza, Veronica Zaga, and Yonathan Garfias. "Su.132. Activation of Nkt Cells in Pars Planitis." Clinical Immunology 119 (January 2006): S205. http://dx.doi.org/10.1016/j.clim.2006.04.559.

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43

POLLACK, ARYEH L., H. RICHARD McDONALD, ROBERT N. JOHNSON, et al. "PERIPHERAL RETINOSCHISIS AND EXUDATIVE RETINAL DETACHMENT IN PARS PLANITIS." RETINA 22, no. 6 (2002): 719–24. http://dx.doi.org/10.1097/00006982-200212000-00006.

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44

Park, Susan E. "2Peripheral Scatter Photocoagulation for Neovascularization Associated With Pars Planitis." Archives of Ophthalmology 113, no. 10 (1995): 1277. http://dx.doi.org/10.1001/archopht.1995.01100100065030.

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45

Dev, Sundeep, William F. Mieler, Jose S. Pulido, and Robert A. Mittra. "Visual outcomes after pars plana vitrectomy for epiretinal membranes associated with pars planitis." Ophthalmology 106, no. 6 (1999): 1086–90. http://dx.doi.org/10.1016/s0161-6420(99)90247-6.

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46

Androudi, Sofia, Muna Ahmed, Periklis Brazitikos, and C. Stephen Foster. "Valsalva retinopathy: diagnostic challenges in a patient with pars-planitis." Acta Ophthalmologica Scandinavica 83, no. 2 (2005): 256–57. http://dx.doi.org/10.1111/j.1600-0420.2005.00456.x.

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47

Thomas, Akshay S., Amro Ali, Sruthi Arepalli, and Eric B. Suhler. "Inferior Corneal Haze and Inflammatory Endotheliopathy Related to Pars Planitis." Ocular Immunology and Inflammation 28, no. 5 (2019): 798–801. http://dx.doi.org/10.1080/09273948.2019.1629603.

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48

Malinowski, Susan M., Jose S. Pulido, and James C. Folk. "Long-term Visual Outcome and Complications Associated with Pars Planitis." Ophthalmology 100, no. 6 (1993): 818–25. http://dx.doi.org/10.1016/s0161-6420(93)31567-8.

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49

González Rubio-Medina, E., and M. Pedroza-Seres. "Pacientes con pars planitis tratados con fotocoagulación selectiva: curso clínico." Archivos de la Sociedad Española de Oftalmología 88, no. 8 (2013): 298–301. http://dx.doi.org/10.1016/j.oftal.2012.09.032.

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50

Salcedo-Villanueva, G., L. Arellanes-García, J. Fromow-Guerra, and E. Hernández-Quintela. "Asociación de membranas epirretinianas con edema macular en pars planitis." Archivos de la Sociedad Española de Oftalmología 89, no. 1 (2014): 22–26. http://dx.doi.org/10.1016/j.oftal.2013.09.008.

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