Relevant bibliographies by topics / Parvalbumin positive interneuron

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Academic literature on the topic 'Parvalbumin positive interneuron'

Author: Grafiati
Published: 3 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "Parvalbumin positive interneuron"

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Cooke, James E., Martin C. Kahn, Edward O. Mann, Andrew J. King, Jan W. H. Schnupp, and Ben D. B. Willmore. "Contrast gain control occurs independently of both parvalbumin-positive interneuron activity and shunting inhibition in auditory cortex." Journal of Neurophysiology 123, no. 4 (2020): 1536–51. http://dx.doi.org/10.1152/jn.00587.2019.

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We investigated whether contrast gain control is mediated by shunting inhibition from parvalbumin-positive interneurons in auditory cortex. We performed extracellular and intracellular recordings in mouse auditory cortex while presenting sensory stimuli with varying contrasts and manipulated parvalbumin-positive interneuron activity using optogenetics. We show that while parvalbumin-positive interneuron activity modulates the gain of cortical responses, this activity is not the primary mechanism for contrast gain control in auditory cortex.
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Woodruff, Alan R., and Pankaj Sah. "Inhibition and Synchronization of Basal Amygdala Principal Neuron Spiking by Parvalbumin-Positive Interneurons." Journal of Neurophysiology 98, no. 5 (2007): 2956–61. http://dx.doi.org/10.1152/jn.00739.2007.

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Using mice that express enhance green fluorescent protein (EGFP) under the control of the parvalbumin promoter, we made paired recordings from interneurons and principal neurons in the basal amygdala. In synaptically connected pairs, we show that single action potentials in a parvalbumin expressing interneuron can inhibit spiking in the synaptically connected principal neuron. When principal neurons were provided with suprathreshold oscillatory drive via a somatic patch pipette, action potentials in the interneuron inhibited spiking in principal neurons only when the interneuron spike occurred
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Giesers, Naomi K., and Oliver Wirths. "Loss of Hippocampal Calretinin and Parvalbumin Interneurons in the 5XFAD Mouse Model of Alzheimer’s Disease." ASN Neuro 12 (January 2020): 175909142092535. http://dx.doi.org/10.1177/1759091420925356.

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The deposition of amyloid-β peptides in the form of extracellular plaques and neuronal degeneration belong to the hallmark features of Alzheimer’s disease (AD). In addition, impaired calcium homeostasis and altered levels in calcium-binding proteins seem to be associated with the disease process. In this study, calretinin- (CR) and parvalbumin- (PV) positive gamma-aminobutyric acid-producing (GABAergic) interneurons were quantified in different hippocampal subfields of 12-month-old wild-type mice, as well as in the transgenic AD mouse models 5XFAD and Tg4-42. While, in comparison with wild-typ
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Howard, MacKenzie A., and Scott C. Baraban. "Synaptic integration of transplanted interneuron progenitor cells into native cortical networks." Journal of Neurophysiology 116, no. 2 (2016): 472–78. http://dx.doi.org/10.1152/jn.00321.2016.

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Interneuron-based cell transplantation is a powerful method to modify network function in a variety of neurological disorders, including epilepsy. Whether new interneurons integrate into native neural networks in a subtype-specific manner is not well understood, and the therapeutic mechanisms underlying interneuron-based cell therapy, including the role of synaptic inhibition, are debated. In this study, we tested subtype-specific integration of transplanted interneurons using acute cortical brain slices and visualized patch-clamp recordings to measure excitatory synaptic inputs, intrinsic pro
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Hameed, Mustafa Q., Tsung-Hsun Hsieh, Leon Morales-Quezada, et al. "Ceftriaxone Treatment Preserves Cortical Inhibitory Interneuron Function via Transient Salvage of GLT-1 in a Rat Traumatic Brain Injury Model." Cerebral Cortex 29, no. 11 (2018): 4506–18. http://dx.doi.org/10.1093/cercor/bhy328.

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Abstract Traumatic brain injury (TBI) results in a decrease in glutamate transporter-1 (GLT-1) expression, the major mechanism for glutamate removal from synapses. Coupled with an increase in glutamate release from dead and dying neurons, this causes an increase in extracellular glutamate. The ensuing glutamate excitotoxicity disproportionately damages vulnerable GABAergic parvalbumin-positive inhibitory interneurons, resulting in a progressively worsening cortical excitatory:inhibitory imbalance due to a loss of GABAergic inhibitory tone, as evidenced by chronic post-traumatic symptoms such a
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Sherwood, Chet C., Mary Ann Raghanti, Cheryl D. Stimpson, et al. "Inhibitory interneurons of the human prefrontal cortex display conserved evolution of the phenotype and related genes." Proceedings of the Royal Society B: Biological Sciences 277, no. 1684 (2009): 1011–20. http://dx.doi.org/10.1098/rspb.2009.1831.

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Inhibitory interneurons participate in local processing circuits, playing a central role in executive cognitive functions of the prefrontal cortex. Although humans differ from other primates in a number of cognitive domains, it is not currently known whether the interneuron system has changed in the course of primate evolution leading to our species. In this study, we examined the distribution of different interneuron subtypes in the prefrontal cortex of anthropoid primates as revealed by immunohistochemistry against the calcium-binding proteins calbindin, calretinin and parvalbumin. In additi
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Yekhlef, Latefa, Gian Luca Breschi, Laura Lagostena, Giovanni Russo, and Stefano Taverna. "Selective activation of parvalbumin- or somatostatin-expressing interneurons triggers epileptic seizurelike activity in mouse medial entorhinal cortex." Journal of Neurophysiology 113, no. 5 (2015): 1616–30. http://dx.doi.org/10.1152/jn.00841.2014.

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GABAergic interneurons are thought to play a critical role in eliciting interictal spikes (IICs) and triggering ictal discharges in temporal lobe epilepsy, yet the contribution of different interneuronal subtypes to seizure initiation is still largely unknown. Here we took advantage of optogenetic techniques combined with patch-clamp and field recordings to selectively stimulate parvalbumin (PV)- or somatostatin (SOM)-positive interneurons expressing channelrhodopsin-2 (CHR-2) in layers II–III of adult mouse medial entorhinal cortical slices during extracellular perfusion with the proconvulsiv
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Negwer, Moritz, Karol Piera, Rick Hesen, et al. "EHMT1 regulates Parvalbumin-positive interneuron development and GABAergic input in sensory cortical areas." Brain Structure and Function 225, no. 9 (2020): 2701–16. http://dx.doi.org/10.1007/s00429-020-02149-9.

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AbstractMutations in the Euchromatic Histone Methyltransferase 1 (EHMT1) gene cause Kleefstra syndrome, a rare form of intellectual disability (ID) with strong autistic traits and sensory processing deficits. Proper development of inhibitory interneurons is crucial for sensory function. Here we report a timeline of Parvalbumin-positive (PV+) interneuron development in the three most important sensory cortical areas in the Ehmt1+/− mouse. We find a hitherto unreported delay of PV+ neuron maturation early in sensory development, with layer- and region-specific variability later in development. T
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Singh, Yajuvinder, Henri Leinonen, Feroze Fazaludeen, et al. "Loss of Cln5 leads to altered Gad1 expression and deficits in interneuron development in mice." Human Molecular Genetics 28, no. 19 (2019): 3309–22. http://dx.doi.org/10.1093/hmg/ddz165.

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Abstract The Finnish-variant late infantile neuronal ceroid lipofuscinosis, also known as CLN5 disease, is caused by mutations in the CLN5 gene. Cln5 is strongly expressed in the developing brain and expression continues into adulthood. CLN5, a protein of unknown function, is implicated in neurodevelopment but detailed investigation is lacking. Using Cln5−/− embryos of various ages and cells harvested from Cln5−/− brains we investigated the hitherto unknown role of Cln5 in the developing brain. Loss of Cln5 results in neuronal differentiation deficits and delays in interneuron development duri
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Selten, Martijn, Hans van Bokhoven, and Nael Nadif Kasri. "Inhibitory control of the excitatory/inhibitory balance in psychiatric disorders." F1000Research 7 (January 8, 2018): 23. http://dx.doi.org/10.12688/f1000research.12155.1.

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Neuronal networks consist of different types of neurons that all play their own role in order to maintain proper network function. The two main types of neurons segregate in excitatory and inhibitory neurons, which together regulate the flow of information through the network. It has been proposed that changes in the relative strength in these two opposing forces underlie the symptoms observed in psychiatric disorders, including autism and schizophrenia. Here, we review the role of alterations to the function of the inhibitory system as a cause of psychiatric disorders. First, we explore both
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Dissertations / Theses on the topic "Parvalbumin positive interneuron"

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Cornford, Jonathan. "Computational aspects of parvalbumin-positive interneuron function." Thesis, University College London (University of London), 2017. http://discovery.ucl.ac.uk/10039927/.

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The activity of neurons is dependent on the manner in which they process synaptic inputs from other cells. In the event of clustered synaptic input, neurons can respond in a nonlinear manner through synaptic and dendritic mechanisms. Such mechanisms are well established in principal excitatory neurons throughout the brain, where they increase neuronal computational ability and information storage capacity. In contrast for parvalbumin-positive (PV+) interneurons, the most common cortical class of in- hibitory interneuron, synaptic integration is thought to be either linear or sub-linear in natu
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Foggetti, Angelica. "Anatomical and functional study of parvalbumin-positive interneurons in the hippocampal formation." Thesis, University of Aberdeen, 2014. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=227103.

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It has long been acknowledged that inhibitory interneurons play a crucial role in regulating the input-output functions of principal cells in the hippocampus. The investigations we conducted focus on one specific population of interneurons, expressing the protein parvalbumin. The thesis describes three different studies, aimed to characterize anatomical and functional aspects of parvalbumin positive interneurons in the mouse hippocampal formation. The first study examines long-range projections of these neurons from CA1 and subiculum to distant regions of the brain, finding potential targets m
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Holzbecher, André Jörg. "Function of interneuronal gap junctions in hippocampal sharp wave-ripples." Doctoral thesis, Humboldt-Universität zu Berlin, 2018. http://dx.doi.org/10.18452/19364.

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Eine einzigartige experimentelle Beobachtung, welche die Basis für eine ganzheitliche, neurowissentschafliche Theorie für Gedächtnis darstellen könnte, sind sharp wave-ripples (SWRs). SWRs werden in lokalen Neuronennetzwerken erzeugt und sind wichtig für Gedächtniskonsolidierung; SWRs sind charakteristische Ereignisse der lokalen Feldpotentiale im Hippocampus des Säugetiers, die in Phasen von Schlaf und Ruhe vorkommen. Eine SWR besteht aus einer sharp wave, einer ≈ 100 ms langen Auslenkung des Feldpotentials, welche mit ripples, 110–250 Hz Oszillationen, überlagert ist. Jüngste Experimente be
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Douceau, Sara. "Implication du tPA cérébral dans la régulation des comportements et le remodelage des perineuronal nets Tissue-type plasminogen activator expressed in PKC-δ positive GABAergic neurons within the central amygdala modulates motor and emotional responses tPA originating from parvalbumin interneurons controls the remodeling of perineuronal nets". Thesis, Normandie, 2020. http://www.theses.fr/2020NORMC423.

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L’activateur tissulaire du plasminogène (tPA) est une sérine protéase initialement décrite dans le compartiment vasculaire pour son rôle dans les processus de fibrinolyse. Le tPA est également retrouvé dans le parenchyme cérébral où il est exprimé par différents types cellulaires et notamment par les neurones. Cette protéase est impliquée dans de nombreuses fonctions cérébrales telles que la plasticité synaptique, la modulation de la neurotransmission glutamatergique et régule également les processus cognitifs et émotionnels. L’implication du tPA dans ces différentes fonctions est dépendante d
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Ehtiati, Amir Ali [Verfasser], Peer Christian [Akademischer Betreuer] Wulff, and Martina [Gutachter] Böttner. "Strukturelle Plastizität von parvalbumin-positiven Interneuronen des Gyrus dentatus / Amir Ali Ehtiati ; Gutachter: Martina Böttner ; Betreuer: Peer Christian Wulff." Kiel : Universitätsbibliothek Kiel, 2020. http://d-nb.info/1219507989/34.

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Balough, Elizabeth Maier. "Parvalbumin-Positive Interneurons' Orchestration of Episodic Memory in Health and Disease." Thesis, 2020. https://doi.org/10.7916/d8-y0d9-p514.

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Our lives unfold in space and time—we are able to be aware not only of the present instant but also to recollect the past and imagine the future, and our memories tend to be not instantaneous snapshots but rather bear a temporal, sequential dimension. This faculty of time travel allows us to adjust our current actions in light of what we have previously learned and with respect to what we aspire to become. It depends upon faithful records of our personal experiences, termed episodic memory. While over the last century we have learned a great deal about the molecular changes that support this k
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Huang, Tzu-Hsuan, and 黃子瑄. "Investigate the Effect of Enriched Environment on the Gene Expression of Parvalbumin- and Somatostatin-Positive Interneurons in Mice." Thesis, 2018. http://ndltd.ncl.edu.tw/handle/2qc6by.

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碩士<br>國立陽明大學<br>腦科學研究所<br>106<br>Background: Inhibitory interneurons are essential for the organization and regulation of neural networks. The two major types of inhibitory interneuron, Parvalbumin-positive (PV+) and Somatostatin-positive (SST+) interneurons are essential for the information processing and memory formation in the hippocampus. Learning and memory are mediated by the experience-dependent plasticity in the neuronal circuits. However, the molecular mechanism of PV+ and SST+ interneurons during the learning process remained unclear. Here, we utilized enriched environment (EE) as a
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Books on the topic "Parvalbumin positive interneuron"

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Balough, Elizabeth Maier. Parvalbumin-Positive Interneurons' Orchestration of Episodic Memory in Health and Disease. [publisher not identified], 2020.

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Book chapters on the topic "Parvalbumin positive interneuron"

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Behrens, M. Margarita. "Studying Schizophrenia in a Dish: Use of Primary Neuronal Cultures to Study the Long-Term Effects of NMDA Receptor Antagonists on Parvalbumin-Positive Fast-Spiking Interneurons." In Animal Models of Schizophrenia and Related Disorders. Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61779-157-4_6.

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Fasching, Liana, Melanie Brady, and Flora M. Vaccarino. "Cellular and Molecular Pathology in Tourette Syndrome." In Tourette Syndrome, 2nd ed., edited by Liana Fasching, Melanie Brady, and Flora M. Vaccarino. Oxford University Press, 2022. http://dx.doi.org/10.1093/med/9780197543214.003.0012.

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Abstract This chapter summarizes the available literature and data on pathological findings in Tourette syndrome. In severe, unremitting Tourette syndrome, there are decreases in somatostatin-positive/nitric oxide synthase–positive interneurons, fast spiking parvalbumin-positive/γ-aminobutyric acid-ergic interneurons, as well as tonically active cholinergic interneurons in the caudate nucleus and putamen. There is also a prominent increase in inflammation throughout the basal ganglia along with activation of microglial cells. Overall, neuroimaging studies suggest that the basal ganglia, a set
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