Relevant bibliographies by topics / Pathobiont

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Pathobiont'

Author: Grafiati
Published: 7 July 2024
Last updated: 31 July 2025

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Journal articles on the topic "Pathobiont"

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Gilliland, A., Y. Chen, D. Tertigas, M. Surette, B. Bressler, and B. Vallance. "A24 AN ULCERATIVE COLITIS-ISOLATED PATHOBIONT CAN DEGRADE MUCUS PRODUCED BY UC PATIENT-DERIVED COLONOIDS." Journal of the Canadian Association of Gastroenterology 7, Supplement_1 (2024): 13–14. http://dx.doi.org/10.1093/jcag/gwad061.024.

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Abstract Background Inflammatory bowel disease (IBD) pathobionts are commensal microbes with pathogenic potential that may cause or exacerbate IBD symptoms. Some pathobionts (ex. Escherichia coli) reside at low levels in the lumen of a healthy gut but can rapidly grow in the inflamed colons of ulcerative colitis (UC) patients. To promote disease, these pathobionts must cross the colonic mucus barrier (comprised of MUC2) that separates the epithelium from luminal microbes. It is currently unclear how bacterial pathobionts cross the mucus barrier of UC patients. Aims Using healthy and UC patient
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Ryder, Mark I., Daniel H. Fine, and Annelise E. Barron. "From Global to Nano: A Geographical Perspective of Aggregatibacter actinomycetemcomitans." Pathogens 13, no. 10 (2024): 837. http://dx.doi.org/10.3390/pathogens13100837.

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The periodontal disease pathobiont Aggregatibacter actinomycetemcomitans (A. actinomycetemcomitans) may exert a range of detrimental effects on periodontal diseases in general and, more specifically, with the initiation and progression of Localized Stage III Grade C periodontitis (molar–incisor pattern). In this review of the biogeography of this pathobiont, the full range of geographical scales for A. actinomycetemcomitans, from global origins and transmission to local geographical regions, to more locally exposed probands and families, to the individual host, down to the oral cavity, and fin
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Jones, Josh, Qiaojuan Shi, Rahul R. Nath, and Ilana L. Brito. "Keystone pathobionts associated with colorectal cancer promote oncogenic reprograming." PLOS ONE 19, no. 2 (2024): e0297897. http://dx.doi.org/10.1371/journal.pone.0297897.

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Fusobacterium nucleatum (Fn) and enterotoxigenic Bacteroides fragilis (ETBF) are two pathobionts consistently enriched in the gut microbiomes of patients with colorectal cancer (CRC) compared to healthy counterparts and frequently observed for their direct association within tumors. Although several molecular mechanisms have been identified that directly link these organisms to features of CRC in specific cell types, their specific effects on the epithelium and local immune compartment are not well-understood. To fill this gap, we leveraged single-cell RNA sequencing (scRNA-seq) on wildtype mi
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Minton, Kirsty. "Pathobiont peacekeepers." Nature Reviews Immunology 18, no. 3 (2018): 152. http://dx.doi.org/10.1038/nri.2018.11.

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Catalan, Eduardo A., Emilio Seguel-Fuentes, Brandon Fuentes, et al. "Oral Pathobiont-Derived Outer Membrane Vesicles in the Oral–Gut Axis." International Journal of Molecular Sciences 25, no. 20 (2024): 11141. http://dx.doi.org/10.3390/ijms252011141.

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Oral pathobionts are essential in instigating local inflammation within the oral cavity and contribute to the pathogenesis of diseases in the gastrointestinal tract and other distant organs. Among the Gram-negative pathobionts, Porphyromonas gingivalis and Fusobacterium nucleatum emerge as critical drivers of periodontitis, exerting their influence not only locally but also as inducers of gut dysbiosis, intestinal disturbances, and systemic ailments. This dual impact is facilitated by their ectopic colonization of the intestinal mucosa and the subsequent mediation of distal systemic effects by
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Yang, H., H. Mirsepasi-Lauridsen, C. Struve, et al. "A21 ULCERATIVE COLITIS-ASSOCIATED E. COLI PATHOBIONTS POTENTIATE COLITIS IN SUSCEPTIBEL HOSTS." Journal of the Canadian Association of Gastroenterology 4, Supplement_1 (2021): 142–44. http://dx.doi.org/10.1093/jcag/gwab002.020.

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Abstract Background Ulcerative colitis (UC) is a chronic inflammatory condition linked to intestinal microbial dysbiosis, including the expansion of E. coli strains related to extra-intestinal pathogenic E. coli. These “pathobionts” exhibit pathogenic properties, but their potential to promote UC is unclear due to the lack of relevant animal models. Aims We explored the potential to establish a mouse model of GI infection by the UC-associated E. coli strain p19A, as well as characterize the pathogenic features of p19A. Methods We used a representative UC pathobiont strain (p19A), and mice lack
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Clayton, C., K. Ng, and C. Tropini. "A38 EXPLORING HOW BOWEL PREPARATION CAN AFFECT INFLAMMATORY BOWEL DISEASE VIA THE GUT MICROBIOTA." Journal of the Canadian Association of Gastroenterology 7, Supplement_1 (2024): 21–22. http://dx.doi.org/10.1093/jcag/gwad061.038.

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Abstract Background Inflammatory bowel disease (IBD) is a debilitating disorder that targets the gastrointestinal (GI) tract. Although its causes remain unknown, recent studies have identified changes to the gut microbiota associated with IBD. While most gut bacteria are essential for GI health, pathobionts are bacteria that are prevalent in IBD patients and that can act as pathogens and induce inflammation. IBD patients undergo routine endoscopies which require the administration of laxative-based bowel prep to clear out the luminal contents of the GI for the endoscope. It has been found that
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Kim, Byeongguk, and Nana Han. "Periodontal Pathobionts and Respiratory Diseases: Mechanisms of Interaction and Implications for Interdisciplinary Care." Biomedicines 13, no. 7 (2025): 1741. https://doi.org/10.3390/biomedicines13071741.

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Periodontitis is a prevalent chronic inflammatory disease that has been increasingly recognized for its systemic impacts, including its connection to respiratory diseases such as pneumonia, chronic obstructive pulmonary disease (COPD), Obstructive Sleep Apnea (OSA), asthma, lung cancer, and COVID-19. This review explores the potential role of periodontal pathobionts, particularly Porphyromonas gingivalis (Pg), Treponema denticola (Td), Fusobacterium nucleatum (Fn), Aggregatibacter actinomycetemcomitans (Aa), and Tannerella forsythia (Tf), in respiratory health. These pathobionts contribute to
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Chapman, Timothy J., Matthew C. Morris, Lei Xu, and Michael E. Pichichero. "Nasopharyngeal colonization with pathobionts is associated with susceptibility to respiratory illnesses in young children." PLOS ONE 15, no. 12 (2020): e0243942. http://dx.doi.org/10.1371/journal.pone.0243942.

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Some children are more susceptible to viral and bacterial respiratory infections in the first few years of life than others. However, the factors contributing to this susceptibility are incompletely understood. In a retrospective analysis of clinical samples collected from a prospectively-enrolled cohort of 358 children we sought associations between physician-attended illness visits and bacterial colonization in the first five years of life. A subset of children was identified by unsupervised clustering analysis as infection and allergy prone (IAP). Several respiratory infection- and allergy-
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Yang, Hyungjun, Hengameh Chloé Mirsepasi-Lauridsen, Carsten Struve, et al. "Ulcerative Colitis-associated E. coli pathobionts potentiate colitis in susceptible hosts." Journal of Immunology 202, no. 1_Supplement (2019): 192.3. http://dx.doi.org/10.4049/jimmunol.202.supp.192.3.

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Abstract Ulcerative colitis (UC) is chronic inflammatory condition linked to intestinal microbial dysbiosis, including the expansion of E. coli strains related to extra-intestinal E. coli. These “pathobionts” exhibit pathogenic properties, but their potential to promote UC is unclear due to the lack of suitable animal models. Here, we established a mouse model using a representative UC pathobiont strain (p19A), and mice lacking single immunoglobulin and toll-interleukin 1 receptor domain (SIGIRR), a deficiency increasing susceptibility to gut infections. p19A was found to adhere to the cecal m
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Dissertations / Theses on the topic "Pathobiont"

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Jan, Alan. "Accélérer la restauration du microbiote intestinal pour renforcer la résistance à la colonisation contre les entérocoques résistants à la vancomycine." Electronic Thesis or Diss., université Paris-Saclay, 2023. http://www.theses.fr/2023UPASB087.

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Le tractus gastro-intestinal est un réservoir d'agents pathogènes opportunistes ou de pathobiontes, qui profitent d'une dysbiose pour proliférer chez les patients fragilisés. Les ERV proviennent du tractus gastro-intestinal, où leur prolifération précède la dissémination dans la circulation sanguine et peut conduire à une infection systémique. La compréhension des mécanismes responsables de la résistance à la colonisation intestinale par les ERV est essentielle pour le contrôle des infections. Peu d'études ont identifié les bactéries commensales qui renforcent la résistance à la colonisation d
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Larousse, Marie. "Étude de l’interaction entre Phytophthora parasitica et le microbiote rhizosphérique à la surface de la plante hôte Solanum lycopersicum." Thesis, Nice, 2016. http://www.theses.fr/2016NICE4038.

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Les oomycètes phytopathogènes ont co-évolué avec les microbiotes des plantes hôtes. Il en résulte la formation de biofilms et des réseaux complexes d’interactions dont nous commençons juste à comprendre l’incidence sur la biologie et la virulence des oomycètes. Déterminer la nature de ces interactions et leur rôle dans le contexte d’une infection est aujourd’hui un enjeu cognitif qui concerne la caractérisation des mécanismes moléculaires et communautaires sous-jacents. C’est également une opportunité en termes d’innovation pour élaborer des méthodes de lutte alternative à l’usage de fongicide
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Larousse, Marie. "Étude de l’interaction entre Phytophthora parasitica et le microbiote rhizosphérique à la surface de la plante hôte Solanum lycopersicum." Electronic Thesis or Diss., Nice, 2016. http://www.theses.fr/2016NICE4038.

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Les oomycètes phytopathogènes ont co-évolué avec les microbiotes des plantes hôtes. Il en résulte la formation de biofilms et des réseaux complexes d’interactions dont nous commençons juste à comprendre l’incidence sur la biologie et la virulence des oomycètes. Déterminer la nature de ces interactions et leur rôle dans le contexte d’une infection est aujourd’hui un enjeu cognitif qui concerne la caractérisation des mécanismes moléculaires et communautaires sous-jacents. C’est également une opportunité en termes d’innovation pour élaborer des méthodes de lutte alternative à l’usage de fongicide
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Romano, Sara. "Dynamique des populations et communautés bactériennes au cours de l’hospitalisation et des infections associées aux soins : cas particulier de la chirurgie cardiaque." Thesis, Montpellier, 2015. http://www.theses.fr/2015MONT3515.

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Les microbiotes humains sont considérés comme des organes supplémentaires impliqués dans des pathologies diverses, y compris infectieuses. Les déséquilibres des microbiotes, ou dysbioses, créent des niches écologiques pathologiques ou pathobiomes. Ce nouveau paradigme de l'infection s'applique tout particulièrement aux infections opportunistes. Dans ce travail, nous considérons des infections associées aux soins (IAS), les infections du site opératoire en chirurgie cardiaque, comme le résultat d'une pathologie de niche et nous étudions la dynamique des communautés et des populations microbienn
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Pauvert, Charlie. "Comparaison et évaluation d’approches bioinformatiques et statistiques pour l'analyse du pathobiome des plantes cultivées." Thesis, Bordeaux, 2019. http://www.theses.fr/2019BORD0214/document.

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Les interactions entre micro-organismes sous-tendent de nombreux services écosystémiques, y compris la régulation des maladies des plantes cultivées. Un acteur de cette régulation est le pathobiome, défini comme le sous-ensemble des micro-organismes associés à une plante hôte en interaction avec un agent pathogène. L'un des défis actuels consiste à reconstruire les pathobiomes à partir de données de metabarcoding, pour identifier des agents potentiels de biocontrôle et pour surveiller en temps réel leurs réponses aux changements environnementaux. Plusieurs verrous méthodologiques doivent cepen
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Tröger, Hanno [Verfasser]. "Wirkungsweise von Pathogenen, Pathobionten und Probiotika auf die epitheliale Transport- und Barrierefunktion des Darms / Hanno Tröger." Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2017. http://d-nb.info/1123071632/34.

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Chow, Janet. "A Pathobiont of the Mammalian Microbiota Balances Intestinal Inflammation and Colonization." Thesis, 2011. https://thesis.library.caltech.edu/6368/1/Chow%2C_Janet_Thesis.pdf.

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Humans and mammals are colonized by a multitude of microbial organisms that have co‐evolved with their hosts for millions of years. The majority of these microbes reside in the gastrointestinal (GI) tract as a complex and dynamic consortium. Though most associations with the host are symbiotic or commensal, some resident bacteria have the potential to cause disease under certain conditions. We refer to these bacteria as ‘pathobionts.’ Pathobionts are distinct from opportunistic pathogens, which are often acquired from the environment and cause acute infections. Bacterial type VI secretion syst
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Books on the topic "Pathobiont"

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Fine, Daniel H., ed. <em>Aggregatibacter actinomycetemcomitans: </em>A D-3 (Dysbiosis, Damage, Disease) Periodontal Pathobiont. MDPI, 2025. https://doi.org/10.3390/books978-3-7258-3064-0.

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Spatz, Linda Ann, Judith A. James, and Gregg Joshua Silverman, eds. Pathogens, Pathobionts and Autoimmunity. Frontiers Media SA, 2021. http://dx.doi.org/10.3389/978-2-88971-546-6.

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Book chapters on the topic "Pathobiont"

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Dimkić, Ivica, Aleksandra Jelušić, Katarina Kruščić, and Tamara Janakiev. "Pathobiome and Microbial Community Shifts Associated with Vegetable, Fruit, and Cereal Crops." In Plant Pathogen Interaction. Springer Nature Singapore, 2023. http://dx.doi.org/10.1007/978-981-99-4890-1_9.

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Bez, Cristina, Hang Dinh Thuy, Minh Nguyen Hong, Iris Bertani, and Vittorio Venturi. "Pathobiome Studies as a Way to Identify Microbial Co-operators and/or Antagonists of the Incoming Plant Pathogen." In Innovations in Land, Water and Energy for Vietnam’s Sustainable Development. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-51260-6_6.

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Zaborina, Olga, Alexander Zaborin, Robert Keskey, and John Alverdy. "Enhancing Survival Following High-Risk Surgery by Modulating Actionable Items within the Pathobiome." In Gut Microbiota - A Key Player in Overall Human Pathologies [Working Title]. IntechOpen, 2025. https://doi.org/10.5772/intechopen.1010335.

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Infection-related complications are common among surgical patients and result in morbidity and mortality. Despite the use of antibiotics, infection-related complications continue to occur. While the main sources of postoperative infections have been assumed to be due to some types of perioperative external contamination, an increasing number of studies have suggested an endogenous source when intestinal pathogens disseminate to cause the syndrome of “gut-origin sepsis.” In this context, the composition and function of the intestinal microbiome appear to play a crucial role. Patients are at inc
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Tonev, Anton. "Perspective Chapter: The Parasitic Component." In Bacterial, Viral and Fungal Coinfections [Working Title]. IntechOpen, 2024. http://dx.doi.org/10.5772/intechopen.114391.

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There is no tissue or organ in the body of plants, animals and man in which some parasitic species have not adapted to live. Blood, brain, liver and striated and cardiac muscles, which we are used to in classical microbiology to perceive, a priori, as sterile organs, are in fact often the home of some parasitic species. This ecological idyll often terrifies clinicians and owners, especially when it comes to parasitic species utilizing humans as final, intermediate or potential hosts. The parasites are well adapted to pass the barriers that the body puts in front of other microorganisms, thus c
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Stewart, Jane E., Mee-Sook Kim, Bradley Lalande, and Ned B. Klopfenstein. "Pathobiome and microbial communities associated with forest tree root diseases." In Forest Microbiology. Elsevier, 2021. http://dx.doi.org/10.1016/b978-0-12-822542-4.00004-8.

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Bateman, Kelly S., Stephen W. Feist, John P. Bignell, David Bass, and Grant D. Stentiford. "Marine pathogen diversity and disease outcomes." In Marine Disease Ecology. Oxford University Press, 2020. http://dx.doi.org/10.1093/oso/9780198821632.003.0001.

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This chapter provides an introduction to important marine pathogens, providing an overview of the diversity of pathogen types and how they affect different hosts in the marine environment. The chapter focuses on wild and cultured species and highlights that single infections are relatively rare, with co- and secondary infections being commonplace. The authors highlight the importance of understanding “normal” host tissue structure prior to interpreting pathological changes and outline the role of histology to assess pathogenicity of emerging diseases, linking presence of individual pathogens a
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Coates, Christopher J. "Diseases of chelicerates." In Invertebrate Pathology. Oxford University Press, 2022. http://dx.doi.org/10.1093/oso/9780198853756.003.0009.

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Chelicerates represent one of the oldest and second most speciose groups within the Phylum Arthropoda. Often referred to as ‘living fossils’, extant chelicerates inhabit terrestrial and aquatic ecosystems. Spiders, scorpions, ticks, and mites are usually mistaken for insects, just as horseshoe crabs are misidentified as crustaceans. The biological and commercial importance of chelicerates cannot be overstated; members represent vectors of devastating animal and plant diseases (acarids), wielders of poisons and venoms (arachnids), and critical for the detection of bacterial contaminants in phar
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Conference papers on the topic "Pathobiont"

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Azzouz, Doua F., Jill P. Buyon, and Gregg J. Silverman. "AI-26 Intestinal IGA as pathfinders to identify microbiome pathobiont candidates in SLE." In Abstracts of the Third Biannual Scientific Meeting of the North and South American Lupus Community, Armonk, New York, USA, September 29 – October 1, 2016. Lupus Foundation of America, 2016. http://dx.doi.org/10.1136/lupus-2016-000179.26.

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Silverman, Gregg J., Macintosh Cornwell, Peter Izmirly, Jill Buyon, Doua F. Azzouz, and Kelly Ruggles. "1103 Lupus clinical flares in patients with gut pathobiont blooms share a novel peripheral blood transcriptomic immune activation profile." In LUPUS 21ST CENTURY 2022 CONFERENCE, Abstracts of Sixth Scientific Meeting of North American and European Lupus Community, Tucson, AZ, USA – September 20–23, 2022. Lupus Foundation of America, 2022. http://dx.doi.org/10.1136/lupus-2022-lupus21century.68.

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Pereira, M. S., K. Stuhlträger, N. Effelsberg, et al. "OP0047 FASTING MODULATES THE HUMAN GUT MICROBIOME AND REDUCES AN IL-17+/IFNG+ T CELL-INDUCING GUT PATHOBIONT IN PATIENTS WITH RHEUMATOID ARTHRITIS." In EULAR 2024 European Congress of Rheumatology, 12-15 June. Vienna, Austria. BMJ Publishing Group Ltd and European League Against Rheumatism, 2024. http://dx.doi.org/10.1136/annrheumdis-2024-eular.3302.

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Khomyakova, T. I., and Yu N. Khomyakov. "PATHOBIOM: STAGES OF FORMATION AND WAYS OF MANAGEMENT." In INNOVATIVE TECHNOLOGIES IN SCIENCE AND EDUCATION. DSTU-Print, 2020. http://dx.doi.org/10.23947/itno.2020.306-310.

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An increase in the incidence and mortality of the population from pathologies associated with the formation of the pathobiome as a stable community of microorganisms occurs simultaneously with an increase in the proportion of antibiotic-resistant microorganisms in these communities. The formation of a pathobiome inevitably leads to the development of an immunodeficiency state and an increase in sensitivity to viral infections. The paper describes the stages of pathobiome formation and its main characteristics. Approaches have been formulated to prevent its pathobiome formation or reduce its st
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Li, L., M. Mac Aogáin, T. Xu, et al. "Neisseria species as pathobionts in bronchiectasis." In ERS International Congress 2022 abstracts. European Respiratory Society, 2022. http://dx.doi.org/10.1183/13993003.congress-2022.1566.

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Jaggi, T., M. Mac Aogáin, H. R. Keir, et al. "Neisseria Species as Novel Respiratory Pathobionts in Bronchiectasis." In American Thoracic Society 2021 International Conference, May 14-19, 2021 - San Diego, CA. American Thoracic Society, 2021. http://dx.doi.org/10.1164/ajrccm-conference.2021.203.1_meetingabstracts.a3903.

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Aogáin, Micheál Mac, Li Liang, Tengfei Xu, et al. "Characterizing Neisseria spp. as novel respiratory pathobionts in bronchiectasis." In ERS International Congress 2021 abstracts. European Respiratory Society, 2021. http://dx.doi.org/10.1183/13993003.congress-2021.pa2060.

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Fernandez-Barat, L., R. López-Aladid, V. Alcaraz, et al. "The Mucoid Pathobiome and Its Clinical Implications in Non-Cystic Fibrosis Bronchiectasis." In American Thoracic Society 2022 International Conference, May 13-18, 2022 - San Francisco, CA. American Thoracic Society, 2022. http://dx.doi.org/10.1164/ajrccm-conference.2022.205.1_meetingabstracts.a2001.

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"Actinidia host genetic background dictates oomycete pattern in the Kiwifruit Vine Decline Syndrome pathobiome." In IS-MPMI Congress. IS-MPMI, 2023. http://dx.doi.org/10.1094/ismpmi-2023-12.

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Kiwifruit Vine Decline Syndrome (KVDS) is the most important soil-borne disease within Italian kiwifruit industry, causing €300,000 of economic losses in 2020 alone. So far, the KVDS’ aetiological agents belong primarily to the oomycetes. Since no effective management strategies exist yet, the most promising approach to overcoming KVDS is therefore the use of resistant species as rootstocks or for breeding programmes. Several Actinidia genotypes showing different resistance’ rates to KVDS were grown in disease-promoting soils. A metabarcoding approach was set up to identify the oomycetes assoc
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Slater, Rachael, Alessandra Frau, Umer Zeeshan Ijaz, et al. "P297 The presence of oral bacterial pathobionts and their volatile organic compound markers in the stool of colorectal cancer patients." In Abstracts of the BSG Annual Meeting, BSG LIVE’25, 23–26 June 2025, SEC Glasgow. BMJ Publishing Group Ltd and British Society of Gastroenterology, 2025. https://doi.org/10.1136/gutjnl-2025-bsg.430.

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