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Relevant bibliographies by topics / Pathogenic Microbiology

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Academic literature on the topic 'Pathogenic Microbiology'

Author: Grafiati

Published: 4 June 2021

Last updated: 4 February 2022

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Journal articles on the topic "Pathogenic Microbiology"

1

Balows, Albert. "Clinical and pathogenic microbiology." Diagnostic Microbiology and Infectious Disease 9, no. 3 (March 1988): 195–96. http://dx.doi.org/10.1016/0732-8893(88)90030-2.

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Kunin, Calvin M. "Book ReviewClinical and Pathogenic Microbiology." New England Journal of Medicine 318, no. 10 (March 10, 1988): 649. http://dx.doi.org/10.1056/nejm198803103181027.

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Rouault, T. A. "MICROBIOLOGY: Enhanced: Pathogenic Bacteria Prefer Heme." Science 305, no. 5690 (September 10, 2004): 1577–78. http://dx.doi.org/10.1126/science.1102975.

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Xu, Zhenbo, Xingyong Xu, Da Qi, Ling Yang, Bing Li, Lin Li, Xiaoxi Li, and Dingqiang Chen. "Effect of aminoglycosides on the pathogenic characteristics of microbiology." Microbial Pathogenesis 113 (December 2017): 357–64. http://dx.doi.org/10.1016/j.micpath.2017.08.053.

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De Backer, Marianne D., Mitch Raponi, and Greg M. Arndt. "RNA-mediated gene silencing in non-pathogenic and pathogenic fungi." Current Opinion in Microbiology 5, no. 3 (June 2002): 323–29. http://dx.doi.org/10.1016/s1369-5274(02)00319-3.

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Łanocha-Arendarczyk, Natalia, Danuta Kosik-Bogacka, Wojciech Zaorski, Karolina Kot, Katarzyna Galant, and Aleksandra Łanocha. "PATHOGENIC FREE-LIVING AMOEBA." Postępy Mikrobiologii - Advancements of Microbiology 56, no. 1 (2019): 106–12. http://dx.doi.org/10.21307/pm-2017.56.1.106.

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Pennisi, E. "MICROBIOLOGY: Environmentally Sensitive Protein Proves Key to Making Yeast Pathogenic." Science 312, no. 5773 (April 28, 2006): 515. http://dx.doi.org/10.1126/science.312.5773.515.

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Korting, H. C. "Dimorphism in Human Pathogenic and Apathogenic Yeasts. Contributions to Microbiology." Mycoses 44, no. 9-10 (November 2001): 433. http://dx.doi.org/10.1046/j.1439-0507.2001.00683.x.

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Vyas, Ishan K., Melissa Jamerson, Guy A. Cabral, and Francine Marciano-Cabral. "Identification of Peptidases in Highly Pathogenic vs. Weakly Pathogenic Naegleria fowleri Amebae." Journal of Eukaryotic Microbiology 62, no. 1 (August 21, 2014): 51–59. http://dx.doi.org/10.1111/jeu.12152.

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Wasylnka, Julie A., Megan I. Simmer, and Margo M. Moore. "Differences in sialic acid density in pathogenic and non-pathogenic Aspergillus species." Microbiology 147, no. 4 (April 1, 2001): 869–77. http://dx.doi.org/10.1099/00221287-147-4-869.

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Dissertations / Theses on the topic "Pathogenic Microbiology"

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Kearney, Theresa Elizabeth. "Survival of pathogenic bacteria in anaerobic digesters." Thesis, Queen's University Belfast, 1991. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.334706.

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Collins, Cathleen A. "Ubiquitin in host defense against pathogenic mycobacteria." Diss., Search in ProQuest Dissertations & Theses. UC Only, 2009. http://gateway.proquest.com/openurl?url_ver=Z39.88-2004&rft_val_fmt=info:ofi/fmt:kev:mtx:dissertation&res_dat=xri:pqdiss&rft_dat=xri:pqdiss:3359543.

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Oki, Aminat. "Characterization of the Effects of Iron on Neisseria Gonorrhoeae Surface Protein Modulation and Host Cell Interactions." VCU Scholars Compass, 2014. http://scholarscompass.vcu.edu/etd/3534.

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Iron is an essential nutrient that is sequestered by iron-binding proteins in the human host resulting in a hostile environment for microbes. Neisseria gonorrhoeae, however, can utilize numerous iron-binding proteins such as transferrin and lactoferrin to acquire this nutrient. During initial infection, gonococci have access to transferrin and lactoferrin present in semen and vaginal fluids, as well as to hemoglobin present in blood during menses or disseminated infections. Consequently, the gonococcus likely encounters conditions of high iron at some stages in the course of natural infection. Potential contributions of iron to gonococcal invasion have however been largely over looked in the field as most studies investigating invasion represent iron depleted environments. Considering the link between menses in women and ascending gonococcal infections, we hypothesized that high iron concentrations present at this time triggers the induction of membrane proteins that enhance gonococcal pathogenesis. Here, we report the gonococcal iron-induced surface proteome and show evidence of post-translational modification of many of these proteins. We also present evidence of an iron enhanced, Opa-independent invasion mechanism. Finally, we investigated the role of NspA, TdfJ and NGO1063 on Opa-independent iron induced invasion. Our studies underscore the importance of investigating the effect of iron on gonococcal host cell interactions. Given the potential clinical relevancy of this phenomenon, data from our studies represent a solid framework for further investigation of gonococcal pathogenesis.

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Toney, Denise Marie. "Mechanisms of Complement Resistance by Pathogenic Naegleria fowleri Amoebae." VCU Scholars Compass, 1993. http://scholarscompass.vcu.edu/etd/5063.

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The genus Naegleria is composed of a distinct group of free-living amoeboﬂagellates that include both pathogenic and nonpathogenic species. N. fowleri, the only pathogenic species of Naegleria to be isolated from humans, is the etiological agent of primary amoebic meningoencephalitis, a rare but rapidly fatal disease of the central nervous system in humans and in laboratory animals. The mechanisms of pathogenicity and the determinants of virulence of N. fowleri are unknown. Both pathogenic and nonpathogenic Naegleria activate the alternative complement pathway, however pathogenic N. fowleri are complement-resistant and nonpathogenic N. gruberi are complement-sensitive. The ability to resist complement-mediated lysis may be an important determinant of virulence of N. fowleri. These studies demonstrate that pathogenic N. fowleri possess at least two mechanisms for resisting complement lysis. Pathogenic N. fowleri synthesize a surface associated protein which appears to possess structural as well as functional homology to the human complement regulatory glycoprotein, CD59. Also, other surface glycoproteins appear to play a role in regulating complement lysis either directly or by indirect inhibitory mechanisms. In addition to complement regulatory glycoproteins, pathogenic N. fowleri possess the ability to remove membrane deposited complement proteins, C5b-C9, from their cell surface by membrane vesiculation. The presence of complement regulatory proteins and the ability to vesiculate in response to serum complement, alone or in combination, serves to protect pathogenic N. fowleri from complement-mediated damage. Nonpathogenic N. gruberi do not appear to possess surface complement regulatory proteins or the ability to vesiculate in response to serum complement. Additional studies demonstrate that growth medium modulates complement resistance and virulence. More importantly, by using changes in growth medium, an in vitro model was developed for differentially expressing proteins associated with the complement-resistant state. The induction of these de novo synthesized proteins may serve as markers of virulence and complement resistance in pathogenic N. fowleri amoebae.

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Burda, Whittney. "Exploring the Pathogenic and Drug Resistance Mechanisms of Staphylococcus aureus." Scholar Commons, 2015. http://scholarcommons.usf.edu/etd/5650.

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We have previously identified σS, an ECF sigma factor that is important in the virulence and stress response of S. aureus. Transcriptional profiling of sigS revealed that it is differentially regulated in a variety of laboratory and clinical strains of S. aureus, suggesting that there exists a regulatory network that modulates its expression. In order to identify direct regulators of sigS expression, we performed a biotin pull down assay in tandem with mass spectrometry. We identified CymR as a direct regulator and observed that sigS expression is increased in cells lacking cymR. In addition, transposon mutagenesis was performed to identify regulators of sigS expression. We identified insertions in genes that are transcriptional regulators, and elements involved in amino acid biosynthesis and DNA replication, recombination and repair as influencing sigS expression. Finally, methyl nitro-nitrosoguanidine mutagenesis in conjunction with whole genome sequencing was employed and revealed mutations in the lactose repressor, lacR, and the membrane sensor histidine kinase, kdpD, as negatively effecting sigS expression. EMSAs revealed that LacR is an indirect regulator of sigS expression, while the response regulator KdpE is a direct repressor. These results indicate that a complex regulatory network is in place for sigS that modulates its expression. In a continuation of studies on σS regulation, we next explored interplay with the products of genes conserved within the sigS locus. We determined that this region is conserved amongst all the sequenced staphylococci, and includes four genes: SAUSA300_1721 (a conserved hypothetical protein), as well as sigS, ecfX, and ecfY. In order to investigate the relationship between EcfX and σS we performed protein pull down assays and observed that these two protein interact. Further to this, transcriptional analysis of sigS in an ecfX mutant reveal that expression of sigS is decreased, indicating that it is an activator. Architectural analysis of the sigS locus via RNAseq revealed that the majority of transcription in this region comes from ecfY, a gene that is downstream and divergent to sigS. We demonstrate that inactivation of ecfY leads to a significant increase in sigS expression, and that ecfY null strains are more resistant to DNA damaging agents such as UV, H2O2, MMS, and ethidium bromide, which we have previously demonstrated that a sigS mutant is highly sensitive to. Our studies also revealed that an ecfY null strain is better able to survive intracellularly following phagocytosis by RAW 264.7 cell and demonstrates increased survival in whole-human blood, which is again opposed to that previously observed for sigS deficient strains. Because the ecfY null strain overexpresses sigS, we investigated the regulon of this sigma factor using this mutant in conjunction with RNAseq analysis. We identified that genes putatively under the control of σS are involved in DNA damage and repair, virulence, amino acid starvation and nucleic acid biosynthesis. Collectively, our results indicate that σS is regulated via a unique mechanism: positively through an apparent need for an activator protein (EcfX) and negatively via RNA-RNA interaction (the 3’ UTR of ecfY). We suggest that the evidence presented here greatly adds not only to our understanding of the regulatory circuits extant within S. aureus, but also to alternative sigma factor biology in general. Finally, we evaluated the efficacy of a novel library of quinazoline-based compounds against a highly drug resistant strain of S. aureus. We performed structure activity and structure property relationship assays in order to identify lead compounds. These methods lead to the identification of N2,N4-disubstituted quinazoline-2,4-diamines that had low minimum inhibitory concentrations, along with favorable physiochemical properties. Evaluation of their biological activity demonstrated limited potential for resistance of to our quinazoline based compounds, low toxicity to human epithelial cells, and strong efficacy in vivo. Taken together, our findings support the use of quinazoline derivatives as potential new antimicrobials against multidrug resistant S. aureus.

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Islam, Shahidul Md. "Studies on gene expression in a pathogenic bacterium." Thesis, University of Birmingham, 2011. http://etheses.bham.ac.uk//id/eprint/1729/.

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The activity of the major pathogenicity determinant of enterohaemorrhagic Escherichia coli is primarily coordinated by expression of the LEE1 operon which is part of the locus of enterocyte effacement (LEE). The LEE1 operon regulatory region has been dissected. LEE-encoded transcription factor, GrlA, activates the LEE1 P1 promoter by binding to a target located within the 18 base pair spacer between the promoter 10 and 35 elements. Shortening this spacer to 17 base pairs increases P1 promoter activity and short-circuits GrlA dependent activation, suggesting that GrlA functions like MerR family transcription activators. It was also found that the LEE1 P1 promoter is overlapped by a cryptic promoter, designated P1A. A single base substitution in the P1 consensus -35 element unmasks P1A promoter activity. In contrast, P1A activity is much less when P1 is inactivated by a mutation in its -10 hexamer element. Hence, even when P1 is inactive, the consensus -35 element sequesters RNA polymerase and prevents its access to the P1A promoter. The LEE1 leader sequence also contains a mini-gene that encodes a dipeptide. Genetic studies showed that expression of this mini-gene is important for optimal expression of downstream genes.

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Brownell, Abigael C. "The Roles of Microcystin and Sulfide in Physiology and Tactic Responses of Pathogenic and Non-Pathogenic Mat-Forming Cyanobacteria." FIU Digital Commons, 2014. http://digitalcommons.fiu.edu/etd/1364.

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Planktothricoides raciborskii and Roseofilum reptotaenium are physiologically similar, yet ecologically distinct organisms found in a hot spring outflow and coral black band disease (BBD), respectively. The aim of this study was to elucidate the relationship between R. reptotaenium and sulfide in BBD, to compare microcystin (MC) production in response to environmental factors, and to determine chemotactic responses to MC and sulfide by the two organisms. Results showed that the pathogenicity of R. reptotaenium in BBD is dependent on sulfate-reducing bacteria as secondary pathogens. Roseofilum reptotaenium produced significantly more MC than P. raciborskii, as measured using ELISA. Roseofilum reptotaenium was negatively chemotactic to sulfide, determined using horizontal and vertical gradients in agar, while P. raciborskii was not affected. Neither cyanobacterium was chemotactic to MC in the agar assays. The ecophysiology of P. raciborskii and R. reptotaenium in relation to MC production and response to sulfide reflected their pathogenic versus non-pathogenic status.

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West, Patrick William John. "Studies on the identification and pathogenic potential of Streptococcus mitis." Thesis, University of Salford, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.308265.

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Cockburn, Chelsea. "Acid sphingomyelinase is essential for vacuolar development of A. phagocytophilum." VCU Scholars Compass, 2018. https://scholarscompass.vcu.edu/etd/5684.

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Obligate intracellular bacteria are significant causes of morbidity and mortality with over two hundred and fifty million infections worldwide annually. One such bacterium, Anaplasma phagocytophilum is the etiologic agent of human granulocytic anaplasmosis (HGA), a tick-transmitted febrile illness. Previous studies have shown that A. phagocytophilum lacks genes for cholesterol biosynthesis and solely relies on Niemann Pick protein type C (NPC)1-mediated low density lipoprotein (LDL)-derived cholesterol to complete its infection cycle.Acid sphingomyelinase (ASMase) is a lysosomal enzyme that is essential for diverse cellular processes including liberation of LDL-derived cholesterol from the lysosome. By first studying A. phagocytophilum, we found that functional inhibitors of acid sphinogmyelinase (FIASMAs) arrest the bacterium’s infection cycle in a dose-dependent manner. FIASMAs inhibit vacuole maturation, conversion to the infectious form, and eliminate the production of infectious progeny. NPC1-mediated LDL-derived cholesterol traffic to the ApV is abrogated in the presence of FIASMAs. Similar to the in vitro model, A. phagocytophilum cannot establish a productive infection in both ASMase-/-and FIASMA treated mice. Furthermore, we extended our studies to Coxiella burnetti (Q fever), and Chlamydia spp. (STD, infectious blindness, pneumonia). FIASMA treatment has a rapid bacteriocidal effect on C. burnettiwithin host cells. Additionally, FIASMA treatment inhibits C. trachomatis and C. pneumoniae inclusion expansion and infectious progeny generation, with C. pneumoniae being more severely impacted. These data highlight the critical, yet distinct roles that ASMase plays in these pathogens’ infection cycles. Furthermore, these results signify the therapeutic potential of FIASMAs for treating diseases caused by these pathogens.

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Holmes, Ashleigh. "Characterising virulence factors from pathogenic bacteria using fluorescent reporters." Thesis, University of Glasgow, 2012. http://theses.gla.ac.uk/3317/.

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Protein translocation systems are invaluable to pathogenic bacteria, facilitating the display of virulence factors on their surface or their release into the extracellular environment. Some protein export systems are ubiquitous and essential to cell survival whereas others are horizontally acquired on prophages or pathogenicity islands (PAI), in many cases providing the bacterium with pathogenic advantages. For the majority of the known protein export systems, their structure, function and secreted substrates have been characterised, yet some proteins have been identified that are secreted via unknown mechanisms. Enterohaemorrhagic Escherichia coli (EHEC) O157:H7 is an important cause of human foodborne disease worldwide. The pathogenesis of this bacterium is mainly attributed to the secretion of toxins and the presence of a Type III Secretion System (T3SS). The T3SS can translocate bacterial proteins, known as effectors, into the host cell which mediate an effect culminating in the formation of a characteristic attaching and effacing (A/E) lesion. This system is encoded on a horizontally acquired PAI termed the locus of enterocyte effacement (LEE). The LEE not only encodes the T3SS apparatus but also several effectors secreted by the system and transcription factors which regulate its expression. However, it was recently found that T3SS not only secretes LEE encoded effectors but can also secrete proteins encoded on other prophages present in the EHEC genome. Characterisation of these non-LEE encoded effectors is ongoing and this study investigates the expression, regulation and function of non-LEE encoded effector H1 (NleH1) and H2 (NleH2). NleH1 and NleH2 are secreted by the T3SS but are encoded on different prophages. This study demonstrates that expression of NleH1 and NleH2 is induced in the same in vitro conditions which stimulate the expression of the LEE but is diminished upon initial host cell contact in vitro. Transcription of nleH1 and nleH2 is dependant upon factors specific to E. coli O157:H7 and these factors are regulated by LEE encoded regulators Ler and GrlA, as they have a positive effect on nleH transcription. NleH1 and H2 are predicted serine/threonine protein kinases and are able to autophosphorylate. Yeast two hybrid screening and 2D differential gel electrophoresis did not elucidate a eukaryotic protein binding partner of NleH1 or NleH2. Transfection assays show that they do not have a significant effect upon NF-κB activation in vitro. Determining the expression, regulation and function of non-LEE encoded effectors contributes towards further understanding of how this pathogen causes disease. Streptococcus pneumoniae, also known as the pneumococcus, is another globally important human pathogen. It is a very diverse pathogen, with over 90 capsular serotypes and is naturally competent for DNA uptake. Pneumococcal pathogenesis is facilitated by the production of a pore-forming toxin, pneumolysin. Pneumolysin’s activities in pneumococcal pathogenesis extend beyond its cytolytic function as it can also activate the complement pathway and modulate the host cytoskeleton. Pneumolysin is a member of a conserved family of toxins known as the cholesterol dependant cytolysins but differs due to the lack of a secretion signal peptide within its sequence. This indicates that it is not secreted from the bacterium however it has been reported that some strains can release pneumolysin in a cell lysis-independent manner. Additional to this, pneumolysin can also localise to the cell wall, and this localisation is not strain dependent. This study characterised codon-optimised N-terminally labelled pneumolysin constructs and applied them to assess the localisation of pneumolysin. In addition, the importance of autolysin and genes which are co-transcribed with Ply upon the localisation/secretion of pneumolysin was investigated by construction of a pneumococcal strain carrying an autolysin-pneumolysin fusion which naturally occurs in equine strains. These genes were not required for the translocation of pneumolysin or its association with the cell wall. Growth of this strain, and its isogenic parent, in vitro at a low density and low temperature resulted in the pneumolysin being detected in the broth culture. This indicates that pneumolysin can be released from the cell wall and that this action is not dependant upon the genes which were deleted in the mutant. The distribution of pneumolysin on the pneumococcal surface was assessed with immunofluorescence, and LumioTM substrate fluorescence, microscopy and found to have a general distribution. As a contribution to future pneumococcal research, codon-optimised fluorescent protein reagents were developed and can be used as reporters for gene expression and protein localisation.

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Books on the topic "Pathogenic Microbiology"

1

Stolen, Joanne S. Pathogenic microbiology: Emerging and reemerging diseases. Fair Haven, NJ: SOS Publications, 1998.

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Dipti, Agrawal, ed. A concise manual of pathogenic microbiology. Hoboken, N.J: Wiley-Blackwell, 2012.

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Mishra, Saroj K., and Dipti Agrawal. A Concise Manual of Pathogenic Microbiology. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2012. http://dx.doi.org/10.1002/9781118301234.

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Wang, Nian. Virulence mechanisms of plant-pathogenic bacteria. St. Paul, Minnesota: American Phytopathological Society, 2015.

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Manual of practical medical microbiology and parasitology. New York: Wiley, 1985.

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Karlström, Åsa. Collagen-like proteins in horse pathogenic streptococcus equi. Uppsala, Sweden: Swedish University of Agricultural Sciences, 2005.

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Kaneko, Mitsumi, and Shinji Izumiyama. Suidō no byōgen biseibutsu taisaku. Tōkyō: Maruzen, 2006.

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G, Scott Elvyn, Finegold Sydney M. 1921-, and Baron Ellen Jo, eds. Bailey and Scott's Diagnostic microbiology. 7th ed. St. Louis: Mosby, 1986.

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R, O'Brian Mark, and SpringerLink (Online service), eds. Molecular Aspects of Iron Metabolism in Pathogenic and Symbiotic Plant-Microbe Associations. Dordrecht: Springer Netherlands, 2013.

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Laboratory text in introductory microbiology for health sciences students. Englewood Cliffs, N.J: Prentice-Hall, 1985.

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Book chapters on the topic "Pathogenic Microbiology"

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Hyde, Richard M. "Review of Pathogenic Microbiology." In Oklahoma Notes, 119–74. New York, NY: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4684-0288-9_4.

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Hyde, Richard M. "Review of Pathogenic Microbiology." In Oklahoma Notes, 119–74. New York, NY: Springer US, 1989. http://dx.doi.org/10.1007/978-1-4684-0340-4_3.

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Hyde, Richard M. "Review of Pathogenic Microbiology." In Oklahoma Notes, 120–74. New York, NY: Springer US, 1992. http://dx.doi.org/10.1007/978-1-4684-0433-3_4.

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Hyde, Richard M. "Review of Pathogenic Microbiology." In Oklahoma Notes, 129–218. New York, NY: Springer New York, 1995. http://dx.doi.org/10.1007/978-1-4613-8439-7_3.

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Boch, J., and U. Bonas. "Gram-Negativ Plant Pathogenic Bacteria." In Contributions to Microbiology, 186–96. Basel: KARGER, 2001. http://dx.doi.org/10.1159/000060411.

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Biran, Dvora, and Eliora Z. Ron. "Extraintestinal Pathogenic Escherichia coli." In Current Topics in Microbiology and Immunology, 149–61. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/82_2018_108.

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Lebaron, Philippe, Benoit Cournoyer, Karine Lemarchand, Sylvie Nazaret, and Pierre Servais. "Environmental and Human Pathogenic Microorganisms." In Environmental Microbiology: Fundamentals and Applications, 619–58. Dordrecht: Springer Netherlands, 2014. http://dx.doi.org/10.1007/978-94-017-9118-2_15.

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Visvesvara, Govinda S. "Pathogenic and Opportunistic Free-Living Amebae." In Manual of Clinical Microbiology, 2387–98. Washington, DC, USA: ASM Press, 2015. http://dx.doi.org/10.1128/9781555817381.ch139.

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Seifert, Horst S. H., Sabine Giercke-Sygusch, and Helge Boehnel. "Identification of Pathogenic Bacteria by Headspace Gas Chromatography." In Analytical Microbiology Methods, 125–36. Boston, MA: Springer US, 1990. http://dx.doi.org/10.1007/978-1-4899-3564-9_8.

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Delaquis, Pascal, Julie Brassard, and Alvin Gajadhar. "Pathogenic Viruses and Protozoa Transmitted by Soil." In Manual of Environmental Microbiology, 3.3.1–1–3.3.1–14. Washington, DC, USA: ASM Press, 2015. http://dx.doi.org/10.1128/9781555818821.ch3.3.1.

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Conference papers on the topic "Pathogenic Microbiology"

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Leite, Caroline Junqueira Barcellos, Jossana Pereira de Sousa, Kataryne Árabe Rimá de Oliveira, Geany Targino de Souza, Danilo Elias Xavier, Maria Lucia da Conceição, Vivyanne dos Santos Falcão Silva, and Evandro Leite de Souza. "Antimicrobial Activity of Cymbopogon Citratus (Dc) Stapf (Lemongrass) Essential Oil on Pathogenic Bacteria From Fruit Juices." In XII Latin American Congress on Food Microbiology and Hygiene. São Paulo: Editora Edgard Blücher, 2014. http://dx.doi.org/10.5151/foodsci-microal-327.

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Carvalho, Rayssa Julliane de, Geany Targino de Souza, Neyrijane Targino de Souza, Danilo Elias Xavier, Maria Lúcia da Conceição, Marciane Magnani, and Evandro Leite de Souza. "Antimicrobial Activity of Thymus Vulgaris L. Essential Oil Against Coalho Cheese-Related Pathogenic and Technological Bacteria." In XII Latin American Congress on Food Microbiology and Hygiene. São Paulo: Editora Edgard Blücher, 2014. http://dx.doi.org/10.5151/foodsci-microal-333.

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Ratti, R. Priscila, A. C. M. Toledo Piza, A. C. Granato, C. O. Hokka, and C. Paiva de Sousa. "Isolation and antimicrobial activity of Streptomyces tubercidicus against pathogenic bacteria and fungi." In Proceedings of the III International Conference on Environmental, Industrial and Applied Microbiology (BioMicroWorld2009). WORLD SCIENTIFIC, 2010. http://dx.doi.org/10.1142/9789814322119_0129.

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Agbakwuru, Chinedu William. "An Investigation on Heterotrophic and Pathogenic Gastrointestinal Bacteria in Otamiri River, Nigeria." In Proceedings of the II International Conference on Environmental, Industrial and Applied Microbiology (BioMicroWorld2007). WORLD SCIENTIFIC, 2009. http://dx.doi.org/10.1142/9789812837554_0033.

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Martín-González, A., M. T. García, C. Pelaz, and J. C. Gutiérrez. "Microbial Pandora's box : Interactions of free living protozoa with human pathogenic bacteria." In Proceedings of the II International Conference on Environmental, Industrial and Applied Microbiology (BioMicroWorld2007). WORLD SCIENTIFIC, 2009. http://dx.doi.org/10.1142/9789812837554_0064.

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de Cara, M., D. Palmero, C. Durán, C. Lacasa, M. Santos, M. D. Coffey, and J. C. Tello. "Phytophthora parasitica showing host specificity and pathogenic ability on tomato and sweet pepper." In Proceedings of the III International Conference on Environmental, Industrial and Applied Microbiology (BioMicroWorld2009). WORLD SCIENTIFIC, 2010. http://dx.doi.org/10.1142/9789814322119_0023.

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Palmero, D., J. M. Rodríguez, M. de Cara, F. Camacho, C. Iglesias, and J. C. Tello. "Prevalence of plant pathogenic isolates of airborne Fusarium species in south east coast of Spain." In Proceedings of the III International Conference on Environmental, Industrial and Applied Microbiology (BioMicroWorld2009). WORLD SCIENTIFIC, 2010. http://dx.doi.org/10.1142/9789814322119_0026.

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Souza, Geany Targino de, Rayssa Julliane de Carvalho, Neyrijane Targino de Souza, Jessica Bezerra dos Santos Rodrigues, Danilo Elias Xavier, Evandro Leite de Souza, and Marciane Magnani. "Antimicrobial Activity of the Origanum Vulgare L. Essential Oil Against Pathogenic and Starter Bacteria Species Related To Brazilian Semi-Hard Cheese (Coalho)." In XII Latin American Congress on Food Microbiology and Hygiene. São Paulo: Editora Edgard Blücher, 2014. http://dx.doi.org/10.5151/foodsci-microal-325.

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Ordax, M., E. Marco-Noales, M. M. López, and E. G. Biosca. "Pathogenic bacteria can produce exopolysaccharides and use them as carbon source under stress conditions: the case of Erwinia amylovora." In Proceedings of the III International Conference on Environmental, Industrial and Applied Microbiology (BioMicroWorld2009). WORLD SCIENTIFIC, 2010. http://dx.doi.org/10.1142/9789814322119_0021.

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Guerra, Ingrid Conceição Dantas, Rayssa Julliane de Carvalho, Estefânia Fernandes Garcia, Marta Suely Madruga, Miriane Moreira Fernandes dos Santos, Priscila Dinah Lima Oliveira, and Evandro Leite de Souza. "Post-Harvest Treatment Using Coatings of Chitosan and And Mentha X Villosa Huds. Essential Oil Maintains the Quality of Tomatoes (Lycopersicum Esculentum) and Delays the Growth of Post-Harvest Pathogenic Fungi." In XII Latin American Congress on Food Microbiology and Hygiene. São Paulo: Editora Edgard Blücher, 2014. http://dx.doi.org/10.5151/foodsci-microal-306.

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