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Relevant bibliographies by topics / Pathological Conditions

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Academic literature on the topic 'Pathological Conditions'

Author: Grafiati

Published: 4 June 2021

Last updated: 30 May 2022

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Journal articles on the topic "Pathological Conditions"

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KATERGARI, Simoni A., Athanasios MILOUSIS, Olga PAGONOPOULOU, Byron ASIMAKOPOULOS, and Nikos K. NIKOLETTOS. "Ghrelin in Pathological Conditions." Endocrine Journal 55, no. 3 (2008): 439–53. http://dx.doi.org/10.1507/endocrj.k07-106.

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Yang, William Y., Ying Shao, Jahaira Lopez-Pastrana, Jietang Mai, Hong Wang, and Xiao-feng Yang. "Pathological conditions re-shape physiological Tregs into pathological Tregs." Burns & Trauma 3 (May 28, 2015): 1–11. http://dx.doi.org/10.1186/s41038-015-0001-0.

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Abstract CD4+FOXP3+ regulatory T cells (Tregs) are a subset of CD4 T cells that play an essential role in maintaining peripheral immune tolerance, controlling acute and chronic inflammation, allergy, autoimmune diseases, and anti-cancer immune responses. Over the past 20 years, a significant progress has been made since Tregs were first characterized in 1995. Many concepts and principles regarding Tregs generation, phenotypic features, subsets (tTregs, pTregs, iTregs, and iTreg35), tissue specificity (central Tregs, effector Tregs, and tissue resident Tregs), homeostasis (highly dynamic and apoptotic), regulation of Tregs by receptors for PAMPs and DAMPs, Treg plasticity (re-differentiation to other CD4 T helper cell subsets, Th1, Th2, Tfh, and Th17), and epigenetic regulation of Tregs phenotypes and functions have been innovated. In this concise review, we want to briefly analyze these eight new progresses in the study of Tregs. We have also proposed for the first time a novel concept that “physiological Tregs” have been re-shaped into “pathological Tregs” in various pathological environments. Continuing of the improvement in our understanding on this important cellular component about the immune tolerance and immune suppression would lead to the future development of novel therapeutics approaches for acute and chronic inflammatory diseases, allergy, allogeneic transplantation-related immunity, sepsis, autoimmune diseases, and cancers.

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Agasarov, L. G., M. Yu Gerasimenko, T. V. Konchugova, A. A. Maryanovsky, V. A. Drobyshev, E. S. Vasilyeva, G. I. Safiullina, et al. "Pharmacopuncture in common pathological conditions*." Russian Journal of Physiotherapy, Balneology and Rehabilitation 17, no. 4 (June 1, 2020): 219–24. http://dx.doi.org/10.18821/1681-3456-2018-17-4-219-224.

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Clinical recommendations have been developed based on the analysis of Russian and international experience in the use of pharmacopuncture which is a contemporary medical technology combining the therapeutic possibilities of reflex therapy and clinical pharmacology. The first part of the recommendations sets out general provisions on the document structure and content that meet the requirements of GOST R 56034-2014 Clinical recommendations (treatment protocols). * Pharmacopuncture in common pathological conditions: clinical recommendations. M., 2017 p. 39. Available at: https://docplayer.ru/53018894-Klinicheskie-rekomendacii-farmakopunktura-pri-rasprostranennyh-patologicheskih-sostoyaniyah.html. Link active on 06/15/2018.

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Leocani, Letizia, and Giancarlo Comi. "EEG Coherence in Pathological Conditions." Journal of Clinical Neurophysiology 16, no. 6 (November 1999): 548. http://dx.doi.org/10.1097/00004691-199911000-00006.

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Bruno, David W. "Pathological conditions of wild salmonids." Fisheries Research 17, no. 1-2 (June 1993): 1–2. http://dx.doi.org/10.1016/0165-7836(93)90002-o.

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Wijgaerts, Anouck, and Kathleen Freson. "Megakaryopoiesis under normal and pathological conditions." Hématologie 20, no. 6 (November 2014): 319–28. http://dx.doi.org/10.1684/hma.2014.0970.

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Milosevic, Dusanka, Ljiljana Janosevic, Ranko Dergenc, and Milan Vasic. "Pathological conditions associated with rhinitis medicamentosa." Srpski arhiv za celokupno lekarstvo 132, no. 1-2 (2004): 14–17. http://dx.doi.org/10.2298/sarh0402014m.

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Rhinitis medicamentosa (nose-drop-nose") is a term used for pathological condition of the nasal mucous membrane that results from long-term abuse with intranasal vasoconstrictors. The aim of this work was to examine what lead the patients with nosedropnose rhinitis to the initial usage of intranasal vasoactive drugs, in this prospective study, 92 patients with rhinitis medicamentosa were included. The evaluation of all study subjects comprised the history, ORL, microbiological and radiological examination, skin prick tests with a battery of routine respiratory and nutritive allergens and nasal cytology. The results of this study showed that the pathological conditions for initial use of intranasal vasoactive drugs were: acute upper respiratory infections in 293%, vasomotor rhinitis in 21.7%, allergic rhinitis in 16.3%, deviated nasal septum in 13.0%, nasal polyposis in 12%, rhinitis induced by mechanical trauma in 4.4%, and hormonal rhinitis in 3.3% of patients with rhinitis medicamentosa. In conclusion, the most common pathological conditions for developing rhinitis medicamentosa were chronic inflammatory and structural diseases manifested by permanent nasal obstruction as well as acute upper respiratory infections are.

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McCoy, J. Michael, and Daniel Oreadi. "Diagnosis and Management of Pathological Conditions." Journal of Oral and Maxillofacial Surgery 75, no. 8 (August 2017): e224-e263. http://dx.doi.org/10.1016/j.joms.2017.04.024.

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Carlson, Eric R., Ghali E. Ghali, and Kathleen E. Herb-Brower. "Diagnosis and Management of Pathological Conditions." Journal of Oral and Maxillofacial Surgery 70, no. 11 (November 2012): e232-e271. http://dx.doi.org/10.1016/j.joms.2012.07.037.

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Piombino-Mascali, Dario, Justina Kozakaitė, Algirdas Tamošiūnas, Ramūnas Valančius, Stephanie Panzer, and Rimantas Jankauskas. "Skeletal pathological conditions of Lithuanian mummies." Papers on Anthropology 23, no. 1 (July 1, 2014): 118. http://dx.doi.org/10.12697/poa.2014.23.1.10.

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Dissertations / Theses on the topic "Pathological Conditions"

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Moore, Robert James. "Vascularization of the intervertebral disc in pathological conditions /." Title page, contents and abstract only, 1995. http://web4.library.adelaide.edu.au/theses/09PH/09phm8233.pdf.

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Zhu, Chunni. "The Blood-brain barrier in normal and pathological conditions." Title page, abstract and contents only, 2001. http://web4.library.adelaide.edu.au/theses/09PH/09phz637.pdf.

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Bibliography: leaves 318-367. Examines the blood-brain barrier in normal and pathological conditions induced by intravascular and extravascular insults. Intravascular insults were induced by administration of Clostridium perfringens prototoxin; extravascular insults were induced by an impact acceleration model for closed head injury to induce traumatic brain injury. Also examines the integrity of the blood-brain barrier ultrastructurally and by its ability to exclude endogenous and exogenous tracers. Also studies the expression of 2 blood-brain barrier specific proteins, endothelial barrier antigen (EBA) and glucose transporter 1 (GLUT1)

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Khalefa, Baled Ibrahim Noufal [Verfasser]. "Opioid receptor efficacy during normal and pathological conditions / Baled Khalefa." Berlin : Freie Universität Berlin, 2014. http://d-nb.info/1046563831/34.

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Keller, Matthias [Verfasser]. "Formation of Intracardiac Electrograms under Physiological and Pathological Conditions / Matthias Keller." Karlsruhe : KIT Scientific Publishing, 2014. http://www.ksp.kit.edu.

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Bull, Sarah-Jane. "Mechanisms promoting myelin formation and maintenance under normal and pathological conditions." Thesis, McGill University, 2012. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=106240.

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Oligodendrocytes (OLs) are the myelinating cells of the central nervous system (CNS). They develop from oligodendrocyte precursor cells and differentiate into mature myelin forming OLs. Once myelin is formed, myelinated axons are segregated into different domains around the myelin-devoid nodes of Ranvier. Maintenance of these domains is essential for efficient saltatory conduction, and several myelin proteins contribute to the maintenance of myelin structure and function. Netrin-1 and its receptor DCC were shown to be involved in the maintenance of paranodal axoglial junctions in vitro, but their role in vivo is not established. Myelin is also a major source of inhibition in the injured CNS, and several myelin proteins have been shown to inhibit axonal regeneration. In this thesis, different aspects of OL biology will be considered. First, signaling pathways involved in OL differentiation will be studied. I demonstrate a synergistic combination of growth factors that promotes late stages of OL differentiation via the PI3K/Akt/mTor pathway in vitro. In the second part, I present in vivo evidence of the involvement of DCC in paranodal and myelin maintenance, and in myelin protein composition. Furthermore, absence of DCC expression by OLs leads to the development of a balance and coordination deficit in mice. These results show that expression of DCC by OLs is required for proper maintenance and stability of myelin in vivo. Finally, I investigate the source of netrin-1 expression in the injured CNS. Our findings demonstrate that netrin-1, in addition to being a potential myelin-associated inhibitor, is also expressed by fibroblasts and some reactive astrocytes in the injured CNS. Netrin-1 expression might contribute to the inhibition of regeneration and failure of remyelination in the injured CNS. Understanding the mechanisms of myelin formation and maintenance will help to develop therapeutic strategies for the treatment of demyelinating diseases like multiple sclerosis, and to promote functional recovery following CNS injury.
Les oligodendrocytes (OLs) sont les cellules myélinisantes du système nerveux central (SNC). Les OLs matures formant la myéline sont dérivés des précurseurs d'OLs, qui se différencient pendant le développement. La myélinisation engendre la formation de différents domaines axonaux autour des nœuds de Ranvier, zones non-myélinisées de l'axone. Le maintien de ces domaines est essentiel pour la conduction axonale saltatoire, et plusieurs protéines de la myéline contribuent au maintien de la structure et de la fonction de la myéline. La nétrine-1 et son récepteur DCC sont impliqués dans le maintien des jonctions paranodales in vitro, mais leur rôle in vivo n'est pas encore établi. La myéline est également une source majeure d'inhibition après une lésion du SNC. Dans cette thèse, différents aspects de la biologie des OLs seront couverts. Premièrement, les voies de signalisation impliquées dans la differentiation des OLs seront étudiées. Dans la première partie, je démontre une synergie de facteurs de croissance sur la voie de signalisation PI3K/Akt/mTor, agissant sur la différentiation morphologique des OLs in vitro. Dans la seconde partie, en utilisant des souris knockout conditionnelles, je présente des preuves de l'implication de DCC dans le maintien des paranodes, de la myéline, et de la composition protéique de la myéline. De plus, l'absence d'expression de DCC par les OLs mène au développement d'un déficit moteur de coordination et d'équilibre. Ces résultats montrent que l'expression de DCC par les OLs est requise pour le bon maintien et la stabilité de la myéline in vivo. Finalement, nous avons investigué la source de l'expression de la nétrine-1 dans les lésions du SNC. Nos résultats démontrent que la nétrine-1, en plus d'être potentiellement un inhibiteur associé à la myéline, est aussi exprimée par les fibroblastes et certains astrocytes réactifs dans les lésions de la moelle épinière. L'expression de nétrine-1 pourrait donc contribuer à l'inhibition de la regénération et l'échec de la remyélinisation après une lésion du SNC. Une meilleure compréhension des mécanismes de formation et de maintien de la myéline peut contribuer à développer des stratégies thérapeutiques pour le traitement de maladies démyélinisantes comme la sclérose en plaques, ou à promouvoir le rétablissement des fonctions après les lésions du SNC.

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Le, Gris Masha. "Mitochondrial protein expression in the developing brain and in pathological conditions." Thesis, University of Oxford, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.670248.

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Huang, Charlie Chia Wei. "Regulation of Cat-1 gene transcription during physiological and pathological conditions." Case Western Reserve University School of Graduate Studies / OhioLINK, 2010. http://rave.ohiolink.edu/etdc/view?acc_num=case1270242874.

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Weinrich, Kendra S. "Oral Pathological Conditions in Early Postcontact Guale, St. Catherines Island, Georgia." The Ohio State University, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=osu1587568057924649.

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Pritchard, Ronald Douglas. "Magnetic resonance spectroscopy studies of the myocardium under physiological and pathological conditions." Thesis, Imperial College London, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.362882.

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Morotti, Stefano <1984&gt. "Computational Modeling of Cardiac Excitation-Contraction Coupling in Physiological and Pathological Conditions." Doctoral thesis, Alma Mater Studiorum - Università di Bologna, 2013. http://amsdottorato.unibo.it/5427/.

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The cardiomyocyte is a complex biological system where many mechanisms interact non-linearly to regulate the coupling between electrical excitation and mechanical contraction. For this reason, the development of mathematical models is fundamental in the field of cardiac electrophysiology, where the use of computational tools has become complementary to the classical experimentation. My doctoral research has been focusing on the development of such models for investigating the regulation of ventricular excitation-contraction coupling at the single cell level. In particular, the following researches are presented in this thesis: 1) Study of the unexpected deleterious effect of a Na channel blocker on a long QT syndrome type 3 patient. Experimental results were used to tune a Na current model that recapitulates the effect of the mutation and the treatment, in order to investigate how these influence the human action potential. Our research suggested that the analysis of the clinical phenotype is not sufficient for recommending drugs to patients carrying mutations with undefined electrophysiological properties. 2) Development of a model of L-type Ca channel inactivation in rabbit myocytes to faithfully reproduce the relative roles of voltage- and Ca-dependent inactivation. The model was applied to the analysis of Ca current inactivation kinetics during normal and abnormal repolarization, and predicts arrhythmogenic activity when inhibiting Ca-dependent inactivation, which is the predominant mechanism in physiological conditions. 3) Analysis of the arrhythmogenic consequences of the crosstalk between β-adrenergic and Ca-calmodulin dependent protein kinase signaling pathways. The descriptions of the two regulatory mechanisms, both enhanced in heart failure, were integrated into a novel murine action potential model to investigate how they concur to the development of cardiac arrhythmias. These studies show how mathematical modeling is suitable to provide new insights into the mechanisms underlying cardiac excitation-contraction coupling and arrhythmogenesis.
Il cardiomiocita è un sistema biologico complesso in cui molti meccanismi interagiscono non linearmente nel processo che accoppia l'eccitazione elettrica alla contrazione meccanica. Lo sviluppo di modelli matematici è quindi fondamentale nel settore dell'elettrofisiologia cardiaca, dove l'uso di strumenti computazionali è diventato complementare alla classica sperimentazione. La mia attività di ricerca si è concentrata sullo sviluppo di tali modelli allo scopo di investigare la regolazione dell'accoppiamento eccitazione-contrazione nella cellula ventricolare. In particolare, questa tesi presenta le seguenti attività: 1) Studio delle inaspettate deleterie conseguenze della somministrazione di un bloccante del canale sodio ad un paziente affetto da sindrome del QT lungo di tipo 3. I risultati sperimentali sono stati usati per riprodurre con un modello di corrente sodio gli effetti di mutazione e trattamento farmacologico, al fine di studiare come questi influenzino il potenziale d'azione umano. La nostra ricerca ha suggerito che l'analisi del fenotipo clinico non è sufficiente per somministrare un farmaco a pazienti che presentano mutazioni con indefinite proprietà elettrofisiologiche. 2) Sviluppo di un modello di inattivazione del canale calcio di tipo L nel cardiomiocita di coniglio allo scopo di riprodurre fedelmente i contributi di inattivazione voltaggio e calcio-dipendente. Il modello, applicato all'analisi delle cinetiche di tale corrente durante normale ed anormale ripolarizzazione, ha predetto lo sviluppo di attività aritmica in caso di inibizione del meccanismo calcio-dipendente, il cui effetto è predominante in condizioni fisiologiche. 3) Analisi delle conseguenze aritmogene dell'interazione tra le vie di segnalazione di stimolazione beta-adrenergica e proteina chinasi calcio-calmodulina dipendente. Le descrizioni dei due sistemi regolatori, entrambi aumentati in condizioni di insufficienza cardiaca, sono state integrate in un nuovo modello di potenziale d'azione murino, al fine di studiare come questi concorrono nell'insorgenza di aritmie. Questi studi mostrano come la modellistica matematica permetta di investigare i meccanismi che regolano l'accoppiamento eccitazione-contrazione e l'aritmogenesi.

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Books on the topic "Pathological Conditions"

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Kernberg, Otto F. Borderline conditions and pathological narcissism. Northvale,N.J: Jason Aronson, 1990.

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Borderline conditions and pathological narcissism. New York: J. Aronson, 1985.

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Kernberg, Otto F. Borderline conditions and pathological narcissism. Northvale, NJ: Jason Aronson, 2002.

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J, Putschar Walter G., ed. Identification of pathological conditions in human skeletal remains. City of Washington: Smithsonian Institution Press, 1985.

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Ortner, Donald J. Identification of pathological conditions in human skeletal remains. Toronto: Custom Publishing Service, University of Toronto Bookstores, 1999.

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Human plasma proteins: Their investigation in pathological conditions. 2nd ed. Chichester: Wiley, 1987.

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Identification of pathological conditions in human skeletal remains. 2nd ed. Amsterdam: Academic Press, 2003.

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Schoutens, A., J. Arlet, J. W. M. Gardeniers, and S. P. F. Hughes, eds. Bone Circulation and Vascularization in Normal and Pathological Conditions. Boston, MA: Springer US, 1993. http://dx.doi.org/10.1007/978-1-4615-2838-8.

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Society for Matrix Research. International Congress. Normal matrix and pathological conditions: 2nd International Congress of the Society for Matrix Research. Stuttgart: G. Fischer, 1992.

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Therapists in the community: Changing the conditions that produce psychopathology. Northvale, N.J: J. Aronson, 1994.

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Book chapters on the topic "Pathological Conditions"

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Flora, Swaran J. S. "Arsenic in Pathological Conditions." In Encyclopedia of Metalloproteins, 123–31. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-1533-6_438.

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Paranjpe, Vikram, and Anat Galor. "The Tear Film: Pathological Conditions." In Ocular Fluid Dynamics, 347–71. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-25886-3_15.

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Vannotti, A. "Iron Metabolism in Pathological Conditions." In Ciba Foundation Symposium - Isotopes in Biochemistry, 90–95. Chichester, UK: John Wiley & Sons, Ltd., 2008. http://dx.doi.org/10.1002/9780470715161.ch9.

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Cheniclet, C., C. Bernard-Dagan, and G. Pauly. "Terpene Biosynthesis Under Pathological Conditions." In Mechanisms of Woody Plant Defenses Against Insects, 117–30. New York, NY: Springer New York, 1988. http://dx.doi.org/10.1007/978-1-4612-3828-7_6.

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Wang, Dayong. "Toxicity Assessment Under the Pathological Conditions." In Exposure Toxicology in Caenorhabditis elegans, 653–82. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-6129-0_21.

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White, Anne, and David Ray. "ACTH Precursors in Different Pathological Conditions." In The Acth Axis: Pathogenesis, Diagnosis and Treatment, 85–107. Boston, MA: Springer US, 2003. http://dx.doi.org/10.1007/978-1-4615-0501-3_5.

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Azarpazhooh, Amir. "Radiographic Analysis of Acquired Pathological Dental Conditions." In Endodontic Radiology, 153–65. Chichester, UK: John Wiley & Sons, Ltd, 2017. http://dx.doi.org/10.1002/9781119421689.ch10.

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Chu, Xiang-Ping, and Zhi-Gang Xiong. "Acid-Sensing Ion Channels in Pathological Conditions." In Advances in Experimental Medicine and Biology, 419–31. Boston, MA: Springer US, 2012. http://dx.doi.org/10.1007/978-1-4614-4756-6_36.

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Roberts, Charlotte. "Pathological Conditions and Anomalies in Archaeological Investigations." In Encyclopedia of Global Archaeology, 5823–29. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4419-0465-2_145.

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Rodríguez-Martín, Conrado. "Pathological Conditions and Anomalies in Forensic Contexts." In Encyclopedia of Global Archaeology, 5829–35. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4419-0465-2_2249.

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Conference papers on the topic "Pathological Conditions"

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Seliktar, R., J. Mizrahi, T. Vachranukunkiet, M. Besser, and D. Kuenzig. "Gait performance under pathological conditions." In Proceedings of the Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 1988. http://dx.doi.org/10.1109/iembs.1988.94787.

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Sghaier, A. B., L. Romdhane, and F. B. Ouezdou. "Index finger system force capabilities under simulated pathological conditions." In 2010 IEEE/RSJ International Conference on Intelligent Robots and Systems (IROS 2010). IEEE, 2010. http://dx.doi.org/10.1109/iros.2010.5650428.

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SILVA, C., R. BERENGUER, Y. COUTINHO, A. SÁ, F. FERREIRA, R. OLIVEIRA, E. MONTEIRO, and A. CARNEIRO. "Analysis of pathological manifestations of a residential building - Case study." In 9th International Conference On Concrete Under Severe Conditions - Environment and Loading. MENVIA, 2019. http://dx.doi.org/10.31808/5ca6e03c5ca4f0d406ac8894.

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Rani, Priya, Priyadarshini N., Rajkumar E. R., and Kumar Rajamani. "Retinal vessel segmentation under pathological conditions using supervised machine learning." In 2016 International Conference on Systems in Medicine and Biology (ICSMB). IEEE, 2016. http://dx.doi.org/10.1109/icsmb.2016.7915088.

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DOUVEN, A. R., R. BERENGUER, A. SÁ, Y. COUTINHO, F. FERREIRA, R. OLIVEIRA, E. MONTEIRO, and A. CARNEIRO. "Analysis of pathological manifestations present in a slab from an old construction - Case study." In 9th International Conference On Concrete Under Severe Conditions - Environment and Loading. MENVIA, 2019. http://dx.doi.org/10.31808/5ca6e03c5ca4f0d406ac8895.

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Wang, C., P. Beyerlein, G. Petznick, A. Krause, C. Nugent, and W. Dubitzky. "Relevance of the KCNH2 protein stoichiometry to pathological conditions underlying QT abnormality." In 2008 35th Annual Computers in Cardiology Conference. IEEE, 2008. http://dx.doi.org/10.1109/cic.2008.4749107.

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Warrick, Amanda E., J. Douglas Swarts, and Samir N. Ghadiali. "Fluid Structure Interactions in the Eustachain Tube Under Normal and Pathological Conditions." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-175328.

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Cleft Palate is a craniofacial syndrome in which the two plates that form the hard palate are not completely joined. As a result, the soft tissue anatomy of the Eustachian Tube (ET) is altered. The ET is a collapsible tube which connects the middle ear (ME) with the nasopharynx (NP). The ET must be periodically opened to equalize ME and NP pressures and drain ME fluids. In healthy adults, ET openings occur during swallowing, where muscle contraction deforms the surrounding soft tissue. However, changes in tissue anatomy may lead to ET dysfunction (i.e. closure during swallowing) and the development of ME disorders such as Otitis Media (OM)[1]. These disorders are especially problematic in infants with cleft palate as they hinder speech, hearing and psychosocial development. Although surgical procedures can be used to repair a cleft palate, these procedures do not typically account the possible development of ET dysfunction and/or OM.

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Murai, Toshiyuki, and Hiroto Kawashima. "Tools for analysis of cell adhesion molecules under physiological and pathological conditions." In 2008 International Symposium on Micro-NanoMechatronics and Human Science (MHS). IEEE, 2008. http://dx.doi.org/10.1109/mhs.2008.4752473.

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Chistiakov, Valerii Vladimirovich, and Kseniia Evgenevna Bezukh. "Modern Approaches to the Study of Man in Normal and Pathological Conditions." In International Research-to-practice conference. TSNS Interaktiv Plus, 2019. http://dx.doi.org/10.21661/r-508881.

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Vinokurov, A. A., D. V. Belik, and E. M. Voronin. "Biotechnical system for evaluating the spectrum of the human voice in normal conditions and in pathological conditions." In 2012 IEEE 11th International Conference on Actual Problems of Electronics Instrument Engineering (APEIE). IEEE, 2012. http://dx.doi.org/10.1109/apeie.2012.6629036.

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Reports on the topic "Pathological Conditions"

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Liu, Xianqiang, WX Ye, H. Fu, SR Zhu, W. Fu, Yinshuo Han, and Qin Wang. Potential link of neutrophil to lymphocyte ratio with pathological liver conditions in chronic hepatitis B patients: a meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, May 2021. http://dx.doi.org/10.37766/inplasy2021.5.0020.

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Liu, Xiaopei, Dan Liu, and Cong’e Tan. Gut microbiome-based machine learning for diagnostic prediction of liver fibrosis and cirrhosis: a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, May 2022. http://dx.doi.org/10.37766/inplasy2022.5.0133.

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Review question / Objective: The invasive liver biopsy is the gold standard for the diagnosis of liver cirrhosis. Other non-invasive diagnostic approaches, have been used as alternatives to liver biopsy, however, these methods cannot identify the pathological grade of the lesion. Recently, studies have shown that gut microbiome-based machine learning can be used as a non-invasive diagnostic approach for liver cirrhosis or fibrosis, while it lacks evidence-based support. Therefore, we performed this systematic review and meta-analysis to evaluate its predictive diagnostic value in liver cirrhosis or fibrosis. Condition being studied: Liver fibrosis and cirrhosis. Liver fibrosis refers to excessive deposition of liver fibrous tissue caused by various pathogenic factors, such as hepatitis virus, alcohol, and drug-induced chemical injury. Continuous progression of liver fibrosis can lead to liver cirrhosis.

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Chejanovsky, Nor, Diana Cox-Foster, Victoria Soroker, and Ron Ophir. Honeybee modulation of infection with the Israeli acute paralysis virus, in asymptomatic, acutely infected and CCD colonies. United States Department of Agriculture, December 2013. http://dx.doi.org/10.32747/2013.7594392.bard.

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Honey bee (Apis mellifera) colony losses pose a severe risk to the food chain. The IAPV (Israeli acute paralysis virus) was correlated with CCD, a particular case of colony collapse. Honey bees severely infected with IAPV show shivering wings that progress to paralysis and subsequent death. Bee viruses, including IAPV, are widely present in honey bee colonies but often there are no pathological symptoms. Infestation of the beehive with Varroa mites or exposure to stress factors leads to significant increase in viral titers and fatal infections. We hypothesized that the honey bee is regulating/controlling IAPV and viral infections in asymptomatic infections and this control is broken through "stress" leading to acute infections and/or CCD. Our aims were: 1. To discover genetic changes in IAPV that may affect tissue tropism in the host, and/or virus infectivity and pathogenicity. 2. To elucidate mechanisms used by the host to regulate/ manage the IAPV-infection in vivo and in vitro. To achieve the above objectives we first studied stress-induced virus activation. Our data indicated that some pesticides, including myclobutanil, chlorothalonil and fluvalinate, result in amplified viral titers when bees are exposed at sub lethal levels by a single feeding. Analysis of the level of immune-related bee genes indicated that CCD-colonies exhibit altered and weaker immune responses than healthy colonies. Given the important role of viral RNA interference (RNAi) in combating viral infections we investigated if CCD-colonies were able to elicit this particular antiviral response. Deep-sequencing analysis of samples from CCD-colonies from US and Israel revealed high frequency of small interfering RNAs (siRNA) perfectly matching IAPV, Kashmir bee virus and Deformed wing virus genomes. Israeli colonies showed high titers of IAPV and a conserved RNAi pattern of targeting the viral genome .Our findings were further supported by analysis of samples from colonies experimentally infected with IAPV. Following for the first time the dynamics of IAPV infection in a group of CCD colonies that we rescued from collapse, we found that IAPV conserves its potential to act as one lethal, infectious factor and that its continuous replication in CCD colonies deeply affects their health and survival. Ours is the first report on the dominant role of IAPV in CCD-colonies outside from the US under natural conditions. We concluded that CCD-colonies do exhibit a regular siRNA response that is specific against predominant viruses associated with colony losses and other immune pathways may account for their weak immune response towards virus infection. Our findings: 1. Reveal that preventive measures should be taken by the beekeepers to avoid insecticide-based stress induction of viral infections as well as to manage CCD colonies as a source of highly infectious viruses such as IAPV. 2. Contribute to identify honey bee mechanisms involved in managing viral infections.

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Sun, Lina, Yanan Han, Hua Wang, Huanyu Liu, Shan Liu, Hongbin Yang, Xiaoxia Ren, and Ying Fang. MicroRNAs as Potential Biomarkers for the Diagnosis of Inflammatory Bowel Disease: A Systematic Review and Meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, February 2022. http://dx.doi.org/10.37766/inplasy2022.2.0027.

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Review question / Objective: The purpose of this systematic review was to systematically review the clinical studies regarding miRNAs as diagnostic biomarkers for inflammatory bowel disease and assess the overall diagnostic accuracy of miRNAs. Condition being studied: The symptoms of inflammatory bowel disease (IBD) are highly variable. The diagnosis of IBD must be made through medical history, physical, laboratory, radiologic, endoscopic, and histological examinations. However, these diagnostic techniques are not specific and sometimes even equivocal. Therefore, reliable biomarkers are urgently needed in the diagnosis of IBD. Several clinical and preclinical researches have shown that dysregulated microRNAs (miRNAs) play a crucial role in IBD development. miRNAs, as single-stranded noncoding RNAs that contain 22-24 nucleotides, can post-transcriptionally regulate gene expression by blocking mRNA translation or degrading target mRNAs. miRNAs are widely involved in physiological and pathological cellular processes, such as differentiation, proliferation and apoptosis. Besides, they are stable, noninvasive, and resistant to degradation by ribonucleases, making them valuable targets in the diagnosis, monitoring, prognosis, and treatment of diseases. To date, inconsistent results have been found about miRNA expression profiling in the patients with IBD. Moreover, the diagnostic accuracy of miRNAs for IBD has not been reported in any meta-analysis.

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Cao, Xianling, Xuanyou Zhou, Naixin Xu, Songchang Chang, and Chenming Xu. Association of IL-4 and IL-10 Polymorphisms with Preterm Birth Susceptibility: A Systematic Review and Meta-Analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, April 2022. http://dx.doi.org/10.37766/inplasy2022.4.0044.

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Review question / Objective: The aim of our systematic review and meta-analysis was to summarize the effects of IL-4 and IL-10 gene polymorphism and clarify their possible association with PTB. Condition being studied: World Health Organization (WHO) defines preterm birth (PTB) as babies born alive before 37 weeks of pregnancy are completed. The new estimates show that the prevalence of PTB during 2014 ranged from 8.7% to13.4% of all live births, about 15 million preterm babies born each year. Besides, PTB is the leading cause of death worldwide for children below 5 years of age. Babies born preterm are at an increased risk of short-term and long-term complications attributed to immaturity of multiple organ systems, such as cerebral palsy, intellectual disabilities, vision and hearing impairments, and impaired cognitive development. PTB has become a worldwide public health problem, but its etiology remains unclear. Accumulating evidence shows that PTB is a syndrome that can be attributed to a variety of pathological processes(5). Inflammatory diseases and genetic background are known risk factors for PTB, many studies had shown that genetic variations in proinflammatory cytokines such as tumor necrosis factor-α (TNF-α) and interleukin-1 α (IL-1 α) are associated with increased risk of PTB, but the relationship between genetic polymorphism in anti-inflammatory cytokines and risk of PTB remains controversial.

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