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1

Fish, F. J. Fish's clinical psychopathology: Signs and symptoms in psychiatry. 2nd ed. Bristol: J. Wright, 1985.

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2

Fish, Frank. Fish's Clinical psychopathology: Signs and symptoms in psychiatry. 2nd ed. Bristol: John Wright, 1985.

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3

Atre, Vaidya Nutan, ed. Descriptive psychopathology: The signs and symptoms of behavioral disorders. Cambridge: Cambridge University Press, 2009.

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4

Topical diagnosis in neurology: Anatomy, physiology, signs, symptoms. 2nd ed. Stuttgart: Thieme, 1989.

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5

Peter, Duus. Topical diagnosis in neurology: Anatomy, physiology, signs, symptoms. 3rd ed. Stuttgart: Thieme, 1998.

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6

Fish, F. J. Fish's clinical psychopathology: Signs and symptoms in psychiatry. 3rd ed. London: Gaskell, 2007.

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7

The obesity reality: A comprehensive approach to a growing problem. Lanham, Md: Rowman & Littlefield Publishers, 2012.

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8

Hicks, James Whitney. Fifty signs of mental illness: A guide to understanding mental health. New Haven: Yale University Press, 2005.

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9

Weinberg, Mea A. The dentist's quick guide to medical conditions. Ames, Iowa: John Wiley & Sons, Inc., 2014.

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10

E, Ulbricht Catherine, and Natural Standard (Firm), eds. Natural Standard medical conditions reference: An integrative approach. St. Louis: Mosby, 2009.

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11

1933-, Kidd C., and Morgan Trefor O, eds. Case-based medical physiology. Malden, Mass: Blackwell, 2005.

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12

Frazier, Margaret Schell. Essentials of human diseases and conditions. 4th ed. St. Louis, Mo: Saunders/Elsevier, 2009.

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13

A, Drzymkowski Jeanette, and Doty Sandra J, eds. Essentials of human diseases and conditions. Philadelphia: Saunders, 1996.

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14

Wist, Drzymkowski Jeanette, ed. Essentials of human diseases and conditions. 3rd ed. St. Louis, MO: Saunders, 2000.

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15

Frazier, Margaret Schell. Essentials of human diseases and conditions. 4th ed. St. Louis, Mo: Saunders/Elsevier, 2009.

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16

Cancer susceptibility: Methods and protocols. New York: Humana Press, 2010.

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17

Bradford, Roger. Children, families, and chronic disease: Psychological models and methods of care. London: Routledge, 1997.

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18

Alternatives in Jewish bioethics. Albany: State University of New York Press, 1997.

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19

Committee on the Use of Animals in Research (U.S.), National Academy of Sciences (U.S.), and Institute of Medicine (U.S.), eds. Science, medicine, and animals. Washington, D.C: National Academy Press, 1991.

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20

Roger, Herdman, and Institute of Medicine (U.S.). Division of Health Care Services., eds. Non-heart-beating organ transplantation: Medical and ethical issues in procurement. Washington, D.C: National Academy Press, 1997.

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21

An Introduction to the Symptoms and Signs of Clinical Medicine. A Hodder Arnold Publication, 2001.

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22

1949-, Gray David, and Toghill Peter J, eds. An introduction to the symptoms and signs of clinical medicine: A hands-on guide to developing core skills. London: Arnold, 2000.

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23

Tavares, Hermano. Assessment and Treatment of Pathological Gambling. Edited by Jon E. Grant and Marc N. Potenza. Oxford University Press, 2012. http://dx.doi.org/10.1093/oxfordhb/9780195389715.013.0091.

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As gambling becomes more popular, more people will be exposed to it; thus, the prevalence of and demand for gambling-related treatments are expected to increase. Pathological gambling (PG) is the most severe level of gambling compromise, characterized by unrestrained gambling to the point of financial and psychosocial harm. Classified among the impulse control disorders, PG resembles other addictive disorders. A host of scales for screening and diagnosing PG are available for both the specialist and the general practitioner. The diagnosis of PG, like that of other addictions, is based upon signs of loss of control over the target behavior (i.e., gambling), dose escalation (increasing amounts wagered to get the same excitement as in previous bets), withdrawal-like symptoms, psychosocial harm, persistent desire, and persistent betting despite the negative consequences. Its treatment requires thorough assessment of psychiatric related conditions, motivational intervention, gambling-focused psychotherapy, relapse prevention, and support for maintenance of treatment gains. Psychopharmacological tools to treat craving and gambling recurrence are an incipient but promising field.
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24

Markman, John D. Diagnostic and Clinical Scales for Peripheral Neuropathy. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0120.

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Due to the absence of a definitive pathological finding, objective biomarker, or imaging correlate, neuropathic pain syndromes may be graded as possible or probable depending on the results of neurological assessment. It is important to acknowledge the diagnostic uncertainty inherent in such a grading system based on probability in a condition for which there is no “gold standard” upon which to base validation studies. Neuropathic pain is a multidimensional entity, and specific syndromes may have distinct sensory profiles (i.e. different combinations of sensory signs and symptoms). Clinical suspicion for an underlying neuropathic mechanism increases when pain is characterized by features such as numbness, paresthesias, and allodynia and when the symptoms are generally resistant to standard over-the-counter and prescribed analgesics. In this chapter a variety pain scales are reviewed.
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25

Hambrick, Linda D. Affective Disorders: Epidemiology, Signs / Symptoms and Prognoses. Nova Science Publishers, Incorporated, 2013.

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26

Your Guide to Symptoms Signs & Conditions. Caxton Editions, 2002.

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27

Hudgins, Louanne, Helga V. Toriello, Gregory M. Enns, and H. Eugene Hoyme, eds. Signs and Symptoms of Genetic Conditions. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199930975.001.0001.

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28

Duus' Topical Diagnosis in Neurology: Anatomy, Physiology, Signs, Symptoms. Thieme Medical Publishers, Incorporated, 2012.

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29

Signs and Symptoms of Genetic Conditions: A Handbook. Oxford University Press, 2014.

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30

Language: Structure, Processing, and Disorders (Issues in the Biology of Language and Cognition). The MIT Press, 1992.

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31

Language: Structure, Processing, and Disorders (Issues in the Biology of Language and Cognition). The MIT Press, 1996.

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32

Compston, Alastair. Multiple sclerosis and other demyelinating diseases. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569381.003.0871.

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The oligodendrocyte–myelin unit subserves saltatory conduction of the nerve impulse in the healthy central nervous system. At one time, many disease processes were thought exclusively to target the structure and function of myelin. Therefore, they were designated ‘demyelinating diseases’. But recent analyses, based mainly on pathological and imaging studies, (re)emphasize that axons are also directly involved in these disorders during both the acute and chronic phases. Another ambiguity is the extent to which these are inflammatory conditions. Here, distinctions should be made between inflammation, as a generic process, and autoimmunity in which rather a specific set of aetiological and mechanistic conditions pertain. And there are differences between disorders that are driven primarily by immune processes and those in which inflammation occurs in response to pre-existing tissue damage.With these provisos, the pathological processes of demyelination and associated axonal dysfunction often account for episodic neurological symptoms and signs referable to white matter tracts of the brain, optic nerves, or spinal cord when these occur in young people. This is the clinical context in which the possibility of ‘demyelinating disease’ is usually considered by physicians and, increasingly, the informed patient. Neurologists will, with appropriate cautions, also be prepared to diagnose demyelinating disease in older patients presenting with progressive symptoms implicating these same pathways even when there is no suggestive past history. Both in its typical and atypical forms multiple sclerosis remains by far the commonest demyelinating disease. But acute disseminated encephalomyelitis, the leucodystrophies, and central pontine myelinolysis also need to be considered in particular circumstances; and multiple sclerosis itself has a differential diagnosis in which the relapsing-remitting course is mimicked by conditions not associated with direct injury to the axon–glial unit. Since our understanding of the cause, pathogenesis and features of demyelinating disease remains incomplete, classification combines aspects of the aetiology, clinical features, pathology, and laboratory components. Whether the designation ‘multiple sclerosis’ encapsulates one or more conditions is now much debated. We anticipate that a major part of future studies in demyelinating disease will be further to resolve this question of disease heterogeneity leading to a new taxonomy based on mechanisms rather than clinical empiricism. But, for now, the variable ages of onset, unpredictable clinical course, protean clinical manifestations, and non-specific laboratory investigations continue to make demyelinating disease one of the more challenging diagnostic areas in clinical neurology.
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33

Duus' Topical Diagnosis In Neurology: Anatomy, Physiology, Signs, Symptoms (Thieme Flexibook). 4th ed. Thieme Medical Publishers, 2005.

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34

The Myth of Pain (Philosophical Psychopathology). The MIT Press, 1999.

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35

The Myth of Pain (Philosophical Psychopathology). The MIT Press, 2001.

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36

A, Davis Mark, Greenough P. Gregg, and Votey Scott R, eds. Signs & symptoms in emergency medicine: Literature-based guide to emergent conditions. St. Louis: Mosby, 1999.

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37

D, Gruskin Karen, ed. Signs and symptoms in pediatrics: Literature-based approach to pediatric conditions. Philadelphia, Pa: Mosby, 2005.

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38

Mark A., M.D. Davis (Editor), Gregg Greenough (Editor), Scott R., M.D. Votey (Editor), and P. Gregg, M.D. Greenough (Editor), eds. Signs & Symptoms in Emergency Medicine: Literature-Based Guide to Emergent Conditions. C.V. Mosby, 1999.

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39

Waldmann, Carl, Neil Soni, and Andrew Rhodes. Neurological disorders. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199229581.003.0022.

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Agitation and confusion 360Status epilepticus 362Meningitis 364Intracerebral haemorrhage 366Subarachnoid haemorrhage 368Ischaemic stroke 370Guillain–Barre syndrome 372Myasthenia gravis 374ICU neuromuscular disorders 376Tetanus 378Botulism 380Neurorehabilitation 382Hyperthermias 384Agitation and confusion are common features in critical illness. Agitation is a symptom or sign of numerous acute and chronic disease states that include pain, anxiety and delirium. Agitation is present in around half of ICU patients, with 15% experiencing severe agitation. Confusion may also be chronic or acute and arise from an overlapping set of pathological processes that includes hypoxia, hypotension, hypoglycaemia and dementia. It is possible to be agitated and not confused, and vice versa. Recognition and treatment of the underlying condition is of utmost importance, rather than treating the symptoms alone....
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40

R, Votey Scott, and Davis Mark A, eds. Signs and symptoms in emergency medicine: Literature-based approach to emergent conditions. 2nd ed. Philadelphia, PA: Mosby Elsevier, 2006.

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41

Weinberg, Mea A., Stuart L. Segelnick, Joseph S. Insler, and Samuel Kramer. Dentist's Quick Guide to Medical Conditions. Wiley & Sons, Incorporated, John, 2015.

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42

Weinberg, Mea A., Stuart L. Segelnick, Joseph S. Insler, and Samuel Kramer. Dentist's Quick Guide to Medical Conditions. Wiley & Sons, Incorporated, John, 2015.

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43

The Hot Brain: Survival, Temperature, and the Human Body. The MIT Press, 2000.

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44

Gisolfi, Carl V., and Mora Francisco. Hot Brain: Survival, Temperature, and the Human Body. MIT Press, 2003.

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45

Gisolfi, Carl V., and Francisco Mora Teruel. Hot Brain: Survival, Temperature, and the Human Body. MIT Press, 2002.

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46

Ferrozzi, Francesco. Ct Of Metastases. Springer, 2000.

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47

CT of Metastases. Springer, 2011.

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48

Essentials of Human Diseases and Conditions. Elsevier - Health Sciences Division, 2012.

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49

Essentials of Human Diseases and Conditions. Elsevier - Health Sciences Division, 2020.

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50

Workbook for Essentials of Human Diseases and Conditions. 4th ed. Saunders, 2008.

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