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Relevant bibliographies by topics / PATHOLOGICAL VASCULAR REMODELING

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Academic literature on the topic 'PATHOLOGICAL VASCULAR REMODELING'

Author: Grafiati

Published: 10 March 2023

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Journal articles on the topic "PATHOLOGICAL VASCULAR REMODELING"

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Ali, Zaheer, Anthony Mukwaya, Antje Biesemeier, et al. "Intussusceptive Vascular Remodeling Precedes Pathological Neovascularization." Arteriosclerosis, Thrombosis, and Vascular Biology 39, no. 7 (2019): 1402–18. http://dx.doi.org/10.1161/atvbaha.118.312190.

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Uemura, Y., R. Shibata, K. Ohashi, et al. "An adipokine omentin prevents pathological vascular remodeling." European Heart Journal 34, suppl 1 (2013): P597. http://dx.doi.org/10.1093/eurheartj/eht307.p597.

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Han, Yue, Kai Huang, Qing-Ping Yao, and Zong-Lai Jiang. "Mechanobiology in vascular remodeling." National Science Review 5, no. 6 (2017): 933–46. http://dx.doi.org/10.1093/nsr/nwx153.

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Abstract Vascular remodeling is a common pathological process in cardiovascular diseases and includes changes in cell proliferation, apoptosis and differentiation as well as vascular homeostasis. Mechanical stresses, such as shear stress and cyclic stretch, play an important role in vascular remodeling. Vascular cells can sense the mechanical factors through cell membrane proteins, cytoskeletons and nuclear envelope proteins to initiate mechanotransduction, which involves intercellular signaling, gene expression, and protein expression to result in functional regulations. Non-coding RNAs, incl

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Wang, Zheng, Xiao Wu, Jiali Li, et al. "Potassium Dehydroandrograpolide Succinate Targets NRP1 Mediated VEGFR2/VE-Cadherin Signaling Pathway to Promote Endothelial Barrier Repair." International Journal of Molecular Sciences 24, no. 4 (2023): 3096. http://dx.doi.org/10.3390/ijms24043096.

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Impairment of vascular endothelial integrity is associated with various vascular diseases. Our previous studies demonstrated that andrographolide is critical to maintaining gastric vascular homeostasis, as well as to regulating pathological vascular remodeling. Potassium dehydroandrograpolide succinate (PDA), a derivative of andrographolide, has been clinically used for the therapeutic treatment of inflammatory diseases. This study aimed to determine whether PDA promotes endothelial barrier repair in pathological vascular remodeling. Partial ligation of the carotid artery in ApoE-/- mice was u

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Jia, Zhuangzhuang, Shuai Wang, Haifeng Yan, et al. "Pulmonary Vascular Remodeling in Pulmonary Hypertension." Journal of Personalized Medicine 13, no. 2 (2023): 366. http://dx.doi.org/10.3390/jpm13020366.

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Pulmonary vascular remodeling is the critical structural alteration and pathological feature in pulmonary hypertension (PH) and involves changes in the intima, media and adventitia. Pulmonary vascular remodeling consists of the proliferation and phenotypic transformation of pulmonary artery endothelial cells (PAECs) and pulmonary artery smooth muscle cells (PASMCs) of the middle membranous pulmonary artery, as well as complex interactions involving external layer pulmonary artery fibroblasts (PAFs) and extracellular matrix (ECM). Inflammatory mechanisms, apoptosis and other factors in the vasc

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Chan, Stefan, and Chen Yan. "PDE1 isozymes, key regulators of pathological vascular remodeling." Current Opinion in Pharmacology 11, no. 6 (2011): 720–24. http://dx.doi.org/10.1016/j.coph.2011.09.002.

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Esteban, Vanesa, Nerea Méndez-Barbero, Luis Jesús Jiménez-Borreguero, et al. "Regulator of calcineurin 1 mediates pathological vascular wall remodeling." Journal of Experimental Medicine 208, no. 10 (2011): 2125–39. http://dx.doi.org/10.1084/jem.20110503.

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Artery wall remodeling, a major feature of diseases such as hypertension, restenosis, atherosclerosis, and aneurysm, involves changes in the tunica media mass that reduce or increase the vessel lumen. The identification of molecules involved in vessel remodeling could aid the development of improved treatments for these pathologies. Angiotensin II (AngII) is a key effector of aortic wall remodeling that contributes to aneurysm formation and restenosis through incompletely defined signaling pathways. We show that AngII induces vascular smooth muscle cell (VSMC) migration and vessel remodeling i

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Esteban, Vanesa, Nerea Méndez-Barbero, Luis Jesús Jiménez-Borreguero, et al. "Regulator of calcineurin 1 mediates pathological vascular wall remodeling." Journal of Cell Biology 195, no. 1 (2011): i1. http://dx.doi.org/10.1083/jcb1951oia11.

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Hong, Xuechong, and Wenduo Gu. "Plasticity of vascular resident mesenchymal stromal cells during vascular remodeling." Vascular Biology 1, no. 1 (2019): H67—H73. http://dx.doi.org/10.1530/vb-19-0022.

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Vascular remodeling is a complex and dynamic pathological process engaging many different cell types that reside within the vasculature. Mesenchymal stromal/stem cells (MSCs) refer to a heterogeneous cell population with the plasticity to differentiate toward multiple mesodermal lineages. Various types of MSC have been identified within the vascular wall that actively contribute to the vascular remodeling process such as atherosclerosis. With the advances of genetic mouse models, recent findings demonstrated the crucial roles of MSCs in the progression of vascular diseases. This review aims to

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Jin, Xin, Guo-xiang Fu, Xiao-dong Li, Ding-liang Zhu, and Ping-jin Gao. "Expression and Function of Osteopontin in Vascular Adventitial Fibroblasts and Pathological Vascular Remodeling." PLoS ONE 6, no. 9 (2011): e23558. http://dx.doi.org/10.1371/journal.pone.0023558.

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Dissertations / Theses on the topic "PATHOLOGICAL VASCULAR REMODELING"

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Hendel, Alon. "Granzyme B in vascular remodeling and pathological angiogenesis." Thesis, University of British Columbia, 2013. http://hdl.handle.net/2429/44782.

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Granzyme B (GZMB) is a serine protease that is expressed by a variety of immune cells and is abundant in a large number of chronic inflammatory disorders. GZMB is highly expressed in cytotoxic lymphocytes where it serves as the main effector molecule of the granule exocytosis pathway by which cytotoxic immune cells mediate target cell death through intracellular delivery of GZMB, leading to activation of apoptotic signaling cascades. GZMB can also accumulate extracellularly during inflammation, where it can cleave a range of extracellular matrix (ECM) proteins that may disrupt cell-matrix inte

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Minami, Takeya. "Reciprocal expression of MRTF-A and myocardin is crucial for pathological vascular remodeling in mice." Kyoto University, 2013. http://hdl.handle.net/2433/174774.

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Keuylian, Zela Talar. "The implication of adenylyl cyclase isoform 8 and its regulation by the Notch pathway in vascular smooth muscle cell transdifferentiation and pathological vesel remodeling." Paris 6, 2013. http://www.theses.fr/2013PA066110.

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L’athérosclérose se caractérise par le rétrécissement de la taille de la lumière des vaisseaux artériels, appelé “sténose”, par la formation de plaques d’athérome dans la paroi des artères. L’un des facteurs majeurs qui contribue à la progression de la formation des lésions est le changement phénotypique des cellules musculaires lisses médiales. Ce processus permet leur transition d’un phénotype quiescent/contractile à un phénotype sécrétoire, prolifératif et migratoire. Mes travaux consistaient à élucider une partie des mécanismes qui régule ce changement. Nous avons montré que l’expression d

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Hsieh, Nan-Kuang, and 謝楠光. "Vascular Pathological Changs and Remodeling In Various Segments of Cerebral Arteries Following Chronic Nitric Oxide Blockade: A Comparison Between Hypertensive and Normotensive Rats." Thesis, 2004. http://ndltd.ncl.edu.tw/handle/38443779512911708563.

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博士<br>國防醫學院<br>醫學科學研究所<br>92<br>Stroke and coronary arterial disease are two major cardiovas-cular events in hypertensive patients. Cerebrovascular diseases and stroke remain the main causes of mortality and morbidity in elderly hypertensive patients. Eedogenous nitric oxide (NO) exerts critical and diverse functions in the cardiovascular system, and abnormalities in NO production is associated with a number of cardiovascular diseases. In a series of studies, we carried out the analysis of blood pressure and vascular structure in spontaneously hypertensive rats（SHR）and normotensive

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PACCOSI, SARA. "CARATTERIZZAZIONE DI CELLULE DENDRITICHE UMANE NEL RIMODELLAMENTO VASCOLARE PATOLOGICO PER L'INDIVIDUAZIONE DI BERSAGLI FARMACOLOGICI." Doctoral thesis, 2013. http://hdl.handle.net/2158/796857.

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Maio, Twofoot Maria Tina. "CLINICAL AND EXPERIMENTAL EVIDENCE FOR THE PATHOLOGICAL MECHANISMS UNDERLYING ASPECTS OF SEXUAL DYSFUNCTION: IMPACT OF ADIPOSITY AND CHRONIC KIDNEY DISEASE." Thesis, 2013. http://hdl.handle.net/1974/8366.

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Cardiovascular disease (CVD) and erectile dysfunction (ED) have common etiologies, such as increased adiposity and chronic diseases. Incident ED is known to be a sentinel of CVD, providing a unique opportunity for early lifestyle interventions to attenuate the progression of disease. The internal pudendal artery (IPA) plays an important role in controlling resistance to penile blood flow and thereby erections. Although morphological and functional disturbances in the IPA have been associated with ED, few studies have characterized changes in the IPA as it relates to increased adiposity and chr

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Books on the topic "PATHOLOGICAL VASCULAR REMODELING"

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1945-, Hori M., Janicki Joseph S, and Maruyama Yukio 1941-, eds. Cardiac-vascular remodeling and functional interaction. Springer, 1997.

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Hori, Masatsugu, Yukio Maruyama, and Joseph S. Janicki. Cardiac-Vascular Remodeling and Functional Interaction. Springer London, Limited, 2013.

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Maruyama, Yukio. Cardiac-Vascular Remodeling and Functional Interaction. Springer, 2013.

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Hori, Masatsugu, Yukio Maruyama, and Joseph S. Janicki. Cardiac-Vascular Remodeling and Functional Interaction. Springer, 2014.

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Yang, Zhihong, and Xiu-Fen Ming. Adventitia and perivascular adipose tissue—the integral unit in vascular disease. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755777.003.0020.

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Obesity and obesity-associated metabolic disorders are highly associated with cardiovascular disease. Abnormal ectopic deposition and accumulation of adipose tissue in organs, including perivascular space (perivascular adipose tissue, PVAT) in obesity are emerging to contribute to vascular disease development through pathological paracrine and/or endocrine secretion of cytokines, namely adipokines, which are vasoactive factors including vascular relaxing and contracting factors, smooth muscle growth promoting and inhibiting factors, and pro- and anti-inflammatory factors. In obesity, productio

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Book chapters on the topic "PATHOLOGICAL VASCULAR REMODELING"

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Kawashima, Seinosuke, Ken-ichi Hirata, and Mitsuhiro Yokoyama. "Nitric Oxide and the Heart: Implications in Physiological and Pathological Conditions." In Cardiac-Vascular Remodeling and Functional Interaction. Springer Japan, 1997. http://dx.doi.org/10.1007/978-4-431-67041-4_28.

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Cui, Jiaxin, Mariluz Rojo Domingo, Ryan Konno, et al. "Impact of Pathological Vascular Remodelling on Right Ventricular Mechanics." In Computational Physiology. Springer Nature Switzerland, 2023. http://dx.doi.org/10.1007/978-3-031-25374-4_7.

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AbstractPulmonary arterial hypertension (PAH) is a rare disorder characterized by elevated blood pressure and pulmonary vascular resistance, often followed by right ventricular hypertrophy and heart failure. The effect of PAH and its treatments on the mechanics, function, and remodelling of the right ventricle (RV) is currently not well understood. To study cardiac biomechanics and functionality as PAH progresses, we implemented a computational model of the heart simulating right ventricular maladaptive remodelling. Our Windkessel-based model, which accounts for direct ventricular interaction and the presence of the pericardium, is utilized to simulate various disease stages of PAH. We find that the pericardium has a larger effect on heart performance than ventricular interaction through the septum.We also examined the effectiveness of two treatments, ventricular assist device (RVAD) and atrial septostomy, on diseased hearts. We show that while both pulsatile and continuous RVADs restore cardiac function, pulsatile RVAD improves cardiac output 29.4% more than continuous RVAD. We also demonstrate that atrial septostomy improves cardiac output by 19.5%. Our model can be further extended by simulating the heart&#x2019;s response to other treatments such as extracorporeal membrane oxygenation (ECMO), and by incorporating ventricular remodelling growth simulations and finite-element ventricular modelling.

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Renna, Nicolas F., Rodrigo Garcia, Jesica Ramirez, and Roberto M. Miatello. "Vascular Repair and Remodeling: A Review." In Physiologic and Pathologic Angiogenesis - Signaling Mechanisms and Targeted Therapy. InTech, 2017. http://dx.doi.org/10.5772/67485.

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Robinson, Chapman. "Pulmonary hypertension (PHT)." In Oxford Handbook of Respiratory Medicine, edited by Stephen J. Chapman, Grace V. Robinson, Rahul Shrimanker, Chris D. Turnbull, and John M. Wrightson. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780198837114.003.0038.

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PHT is a haemodynamic and pathophysiological state that can be found in multiple clinical conditions. Each group has differing characteristic pathological features, but vasoconstriction, remodelling of the pulmonary vessel wall, medial hypertrophy of distal pulmonary arteries ± fibrotic change, and thrombosis lead to raised pulmonary vascular resistance and ultimately right heart failure. The symptoms of PHT are primarily due to RV dysfunction. The symptoms are non-specific, often leading to a delay in diagnosis from first symptoms, which include exertional breathlessness, due to the inability to increase cardiac output with exercise. WHO functional assessment classification is used to quantify the condition. Other symptoms include chest pain (right heart angina), fatigue and weakness, syncope or pre-syncope, due to a fall in systemic BP on exercise, palpitations, peripheral oedema and other signs of right-sided fluid overload.

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Conference papers on the topic "PATHOLOGICAL VASCULAR REMODELING"

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Song Hu. "Multi-parametric photoacoustic microscopy of pathological remodeling in vascular anatomy and function." In 2015 IEEE Photonics Conference (IPC). IEEE, 2015. http://dx.doi.org/10.1109/ipcon.2015.7323619.

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McGah, Patrick M., Alberto Aliseda, Daniel F. Leotta, and Kirk W. Beach. "Effects of Wall Distensibility on the Numerical Simulation of Arteriovenous Fistulae." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14183.

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Arteriovenous fistulae are created surgically to provide an adequate access for dialysis in patients with End-Stage Renal Disease (ESRD). Producing an autogenous shunt linking an artery and a vein in the peripheral circulation bypasses the high resistance capillary bed in order to provide the necessary flow rates at sites easily accessible for dialysis. It has long been recognized that hemodynamics constitute the primary external influence on the remodeling process of anastomosed vascular tissue [1, 2]. The high flow rate, together with the exposure of the venous tissue to the high arterial pr

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Sheidaei, A., S. C. Hunley, L. G. Raguin, and S. Baek. "Simulation of Aneurysm Growth With Stepwise Updating of Hemodynamic Loads Using an MRI-Based Geometric Model." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-205499.

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Computer simulations of vascular tissue adaptation under various physiological and pathological conditions have emerged as a new area of research and aided researchers in their understanding of stress-mediated growth and remodeling (G&R) in these structures. With advances in computational biomechanics and biomedical imaging techniques, combinations of these advanced methods will provide promising tools for medical diagnosis and surgical planning in the future (e.g., [1]). Recently Figueroa et al. [2] presented a new computational framework that brings advances in computational biosolid and

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Baek, Seungik, C. Alberto Figueroa, Charles A. Taylor, and Jay D. Humphrey. "A Framework for Fluid-Solid-Growth Modeling and its Application to Understanding the Enlargement of a Fusiform Aneurysm." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192805.

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Blood vessels adapt in response to changes in their biomechanical and biochemical environment under various physiological and pathological conditions. While advances in computational hemodynamics and arterial wall mechanics have been spectacular, such advances have been achieved separately; there is, therefore, a pressing need for coupling biosolid and biofluid mechanics within a computationally efficient framework to study effects of fluid-solid interactions (FSI) in growth and remodeling (G&R) of the vessel wall. Toward this end, we built a fluid-solid-growth (FSG) modeling framework [1]

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Haskett, Darren, Marie Fouts, Urs Utzinger, Doug Larson, Mohamad Azhar, and Jonathan Vande Geest. "The Effects of Angiotensin II Infusion on the Mechanical Response and Microstructural Organization of Mouse Aorta." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19635.

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Vascular diseases such as aneurysm and aortic dissection account for almost 16,000 deaths in the United States annually. In both of these diseases vascular inflammation is a common pathogenic factor. Another common pathologic feature of vascular disease includes structural matrix remodeling. It is also increasingly believed that inflammation may play a key role in the formation and progression of atherosclerotic vascular disease. Angiotensin II (AngII), a potent vasopressor, is also a strong inducer of vascular inflammation and aortic remodeling in atherosclerosis-prone mice. Based on this kno

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Graham, BB, L. Zhang, MM Mentink-Kane, et al. "Physiologic and Pathologic Analysis of a Murine Model of Schistosomiasis Pulmonary Vascular Remodeling." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a1796.

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Coyle, K., M. Ratsep, K. Laverty та ін. "Natural Killer Cell TGFβ Signalling Influences Pulmonary Vascular Development and Pathological Vascular Remodelling in a Mouse Model of Pulmonary Arterial Hypertension". У American Thoracic Society 2022 International Conference, May 13-18, 2022 - San Francisco, CA. American Thoracic Society, 2022. http://dx.doi.org/10.1164/ajrccm-conference.2022.205.1_meetingabstracts.a4723.

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Liu, Fei, Margarita M. Suarez Velandia, Emeka Ifedigbo, et al. "Pathologic Matrix Stiffening Promotes Cyclooxygenase (COX)-2-Dependent Mechanobiological Feedback Amplification Of Vascular Remodeling In Pulmonary Arterial Hypertension." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a2622.

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