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Books on the topic 'Pattern recognition receptor'

Author: Grafiati

Published: 4 June 2021

Last updated: 26 July 2025

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1

Shan, Libo, and Ping He, eds. Plant Pattern Recognition Receptors. Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-6859-6.

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2

Kutikhin, Anton G., and Arseniy E. Yuzhalin. Genomics of Pattern Recognition Receptors. Springer Basel, 2013. http://dx.doi.org/10.1007/978-3-0348-0688-6.

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3

Kutikhin, Anton G. Genomics of pattern recognition receptors: Applications in oncology and cardiovascular diseases. Springer, 2013.

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4

Tran, Thi Hoai Nguyen. Pattern recognition receptors as key players in adrenal gland dysfunction during sepsis. Universitätsbibliothek Johann Christian Senckenberg, 2012.

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5

J, Ishii Ken, and Akira S, eds. Nucleic acids in innate immunity. CRC Press, 2008.

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6

Kutikhin, Anton G., and Arseniy E. Yuzhalin, eds. Pattern Recognition Receptors and Cancer. Frontiers Media SA, 2015. http://dx.doi.org/10.3389/978-2-88919-674-6.

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7

Wiersinga, W. Joost, and Tom van der Poll. The host response to infection in the critically ill. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0303.

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Infection continues to be a leading cause of intensive care unit death. The host response to infection can be seen as a pattern recognition receptor (PRR)-mediated dysregulation of the immune system following pathogen invasion in which a careful balance between inflammatory and anti-inflammatory responses is vital. A measured and rapid response to microbial invasion is essential to health. The same immunological and coagulation systems that protect against localized infection can act to our disadvantage when these systems are activated systemically during generalized microbial infection. Toll-

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8

Kutikhin, Anton G., and Arseniy E. Yuzhalin. Genomics of Pattern Recognition Receptors: Applications in Oncology and Cardiovascular Diseases. Springer Basel AG, 2015.

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9

Badimon, Lina, and Gemma Vilahur. Atherosclerosis and thrombosis. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0040.

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Atherosclerosis is the main underlying cause of heart disease. The continuous exposure to cardiovascular risk factors induces endothelial activation/dysfunction which enhances the permeability of the endothelial layer and the expression of cytokines/chemokines and adhesion molecules. This results in the accumulation of lipids (low-density lipoprotein particles) in the extracellular matrix and the triggering of an inflammatory response. Accumulated low-density lipoprotein particles suffer modifications and become pro-atherogenic, enhancing leucocyte recruitment and further transmigration across

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10

Badimon, Lina, and Gemma Vilahur. Atherosclerosis and thrombosis. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199687039.003.0040_update_001.

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Atherosclerosis is the main underlying cause of heart disease. The continuous exposure to cardiovascular risk factors induces endothelial activation/dysfunction which enhances the permeability of the endothelial layer and the expression of cytokines/chemokines and adhesion molecules. This results in the accumulation of lipids (low-density lipoprotein particles) in the intimal layer and the triggering of an inflammatory response. Accumulated low-density lipoprotein particles attached to the extracellular matrix suffer modifications and become pro-atherogenic, enhancing leucocyte recruitment and

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11

Badimon, Lina, and Gemma Vilahur. Atherosclerosis and thrombosis. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199687039.003.0040_update_002.

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Atherosclerosis is the main underlying cause of heart disease. The continuous exposure to cardiovascular risk factors induces endothelial activation/dysfunction which enhances the permeability of the endothelial layer and the expression of cytokines/chemokines and adhesion molecules. This results in the accumulation of lipids (low-density lipoprotein particles) in the intimal layer and the triggering of an inflammatory response. Accumulated low-density lipoprotein particles attached to the extracellular matrix suffer modifications and become pro-atherogenic, enhancing leucocyte recruitment and

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12

Geri, Guillaume, and Jean-Paul Mira. Host–pathogen interactions in the critically ill. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0306.

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Infection by a pathogenic micro-organism triggers a coordinated activation of both innate and adaptive immune responses. The innate immune response quickly triggers an antimicrobial response that will initiate development of a pathogen-specific, long-lasting adaptive immune response. Accurate recognition of microbial-associated molecular patterns by pattern-recognition receptors (PRRs) is the cornerstone of this immediate response. Most studied PRRs are Toll-like receptors (TLRs) and their kinase signalling cascades that activate nuclear transcription factors, and induce gene expression and cy

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13

Voll, Reinhard E., and Barbara M. Bröker. Innate vs acquired immunity. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0048.

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The innate and the adaptive immune system efficiently cooperate to protect us from infections. The ancient innate immune system, dating back to the first multicellular organisms, utilizes phagocytic cells, soluble antimicrobial peptides, and the complement system for an immediate line of defence against pathogens. Using a limited number of germline-encoded pattern recognition receptors including the Toll-like, RIG-1-like, and NOD-like receptors, the innate immune system recognizes so-called pathogen-associated molecular patterns (PAMPs). PAMPs are specific for groups of related microorganisms

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14

Ishii, Ken J., and Shizuo Akira. Nucleic Acids in Innate Immunity. Taylor & Francis Group, 2008.

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15

Ishii, Ken J., and Shizuo Akira. Nucleic Acids in Innate Immunity. Taylor & Francis Group, 2008.

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16

Ishii, Ken J. Nucleic Acids in Innate Immunity. Taylor & Francis Group, 2008.

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17

Ishii, Ken J., and Shizuo Akira. Nucleic Acids in Innate Immunity. Taylor & Francis Group, 2019.

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18

(Editor), Ken J. Ishii, and Shizuo Akira (Editor), eds. Nucleic Acids in Innate Immunity. CRC, 2008.

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