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Journal articles on the topic 'Pcpe'

Author: Grafiati

Published: 4 June 2021

Last updated: 17 February 2022

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1

Cai, Mian, and Luying Xun. "Organization and Regulation of Pentachlorophenol-Degrading Genes in Sphingobium chlorophenolicum ATCC 39723." Journal of Bacteriology 184, no. 17 (September 1, 2002): 4672–80. http://dx.doi.org/10.1128/jb.184.17.4672-4680.2002.

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ABSTRACT The first three enzymes of the pentachlorophenol (PCP) degradation pathway in Sphingobium chlorophenolicum (formerly Sphingomonas chlorophenolica) ATCC 39723 have been characterized, and the corresponding genes, pcpA, pcpB, and pcpC, have been individually cloned and sequenced. To search for new genes involved in PCP degradation and map the physical locations of the pcp genes, a 24-kb fragment containing pcpA and pcpC was completely sequenced. A putative LysR-type transcriptional regulator gene, pcpM, and a maleylacetate reductase gene, pcpE, were identified upstream of pcpA. pcpE was found to play a role in PCP degradation. pcpB was not found on the 24-kb fragment. The four gene products PcpB, PcpC, PcpA, and PcpE were responsible for the metabolism of PCP to 3-oxoadipate in ATCC 39723, and inactivational mutation of each gene disrupted the degradation pathway. The organization of the pcp genes is unusual because the four PCP-degrading genes, pcpA, pcpB, pcpC, and pcpE, were found to be located at four discrete locations. Two hypothetical LysR-type regulator genes, pcpM and pcpR, have been identified; pcpM was not required, but pcpR was essential for the induction of pcpB, pcpA, and pcpE. The coinducers of PcpR were PCP and other polychlorinated phenols. The expression of pcpC was constitutive. Thus, the organization and regulation of the genes involved in PCP degradation to 3-oxoadipate were documented.

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2

Steiglitz, Barry M., Douglas R. Keene, and Daniel S. Greenspan. "PCOLCE2Encodes a Functional Procollagen C-Proteinase Enhancer (PCPE2) That Is a Collagen-binding Protein Differing in Distribution of Expression and Post-translational Modification from the Previously Described PCPE1." Journal of Biological Chemistry 277, no. 51 (October 20, 2002): 49820–30. http://dx.doi.org/10.1074/jbc.m209891200.

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The procollagen COOH-terminal proteinase enhancer (PCPE) is a glycoprotein that binds the COOH-terminal propeptide of type I procollagen and potentiates its cleavage by procollagen C-proteinases, such as bone morphogenetic protein-1 (BMP-1). Recently, sequencing of a human expressed sequence tag, which maps near the primary open angle glaucoma region on chromosome 3q21, showed it to encode a novel protein with only 43% identity with PCPE but with a similar domain structure. Here we show this novel protein to be a functional procollagen COOH-terminal proteinase enhancer with activity comparable with that of PCPE and thus propose the designations PCPE2 and PCPE1, respectively. PCPE2 is shown to have a much more limited distribution of expression than does PCPE1, with strong expression primarily in nonossified cartilage in developing tissues and at high levels in the adult heart. PCPE2 is shown to be a glycoprotein that differs markedly in the nature of its glycosylation from that of PCPE1. PCPE2 is also shown to have markedly stronger affinity for heparin than PCPE1, which may account for higher affinities for cell layers. Unexpectedly, both PCPE1 and PCPE2 were found to be collagen-binding proteins, capable of binding at multiple sites on the triple helical portions of fibrillar collagens and also capable of competing for such binding with procollagen C-proteinases. The latter observations may provide insights into the ways PCPEs affect the kinetics of the C-proteinase reaction and into the physical interactions that occur between procollagen C-proteinases and their substrates.

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3

Huelsman, L. P. "The PCPE CD-ROM." IEEE Circuits and Devices Magazine 22, no. 4 (July 2006): 3–38. http://dx.doi.org/10.1109/mcd.2006.1708368.

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4

Zhu, He, and Changcheng Huang. "IoT-B&B: Edge-Based NFV for IoT Devices with CPE Crowdsourcing." Wireless Communications and Mobile Computing 2018 (2018): 1–15. http://dx.doi.org/10.1155/2018/3027269.

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For embracing the ubiquitous Internet-of-Things (IoT) devices, edge computing and Network Function Virtualization (NFV) have been enabled in branch offices and homes in the form of virtual Customer-Premises Equipment (vCPE). A Service Provider (SP) deploys vCPE instances as Virtual Network Functions (VNFs) on top of generic physical Customer-Premises Equipment (pCPE) to ease administration. Upon a usage surge of IoT devices at a certain part of the network, vCPU, memory, and other resource limitations of a single pCPE node make it difficult to add new services handling the high demand. In this paper, we present IoT-B&B, a novel architecture featuring resource sharing of pCPE nodes. When a pCPE node has sharable resources available, the SP will utilize its free resources as a “bed-and-breakfast” place to deploy vCPE instances in need. A placement algorithm is also presented to assign vCPE instances to a cost-efficient pCPE node. By keeping vCPE instances at the network edge, their costs of hosting are reduced. Meanwhile, the transmission latencies are maintained at acceptable levels for processing real-time data burst from IoT devices. The traffic load to the remote, centralized cloud can be substantially reduced.

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5

Tessier, Agnès, Sandrine Vadon-Le Goff, Leena Bruckner-Tuderman, Alexander Nyström, and Catherine Moali. "Fonctions divergentes des « Procollagen C-Proteinase Enhancers », PCPE-1 et PCPE-2, dans la cicatrisation cutanée." Annales de Dermatologie et de Vénéréologie 143, no. 12 (December 2016): S430. http://dx.doi.org/10.1016/j.annder.2016.09.076.

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6

Potthoff, Bojarski, Kohut, Lipska, Liwo, Kessler, Ricard-Blum, and Samsonov. "Analysis of Procollagen C-Proteinase Enhancer-1/Glycosaminoglycan Binding Sites and of the Potential Role of Calcium Ions in the Interaction." International Journal of Molecular Sciences 20, no. 20 (October 10, 2019): 5021. http://dx.doi.org/10.3390/ijms20205021.

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In this study, we characterize the interactions between the extracellular matrix protein, procollagen C-proteinase enhancer-1 (PCPE-1), and glycosaminoglycans (GAGs), which are linear anionic periodic polysaccharides. We applied molecular modeling approaches to build a structural model of full-length PCPE-1, which is not experimentally available, to predict GAG binding poses for various GAG lengths, types and sulfation patterns, and to determine the effect of calcium ions on the binding. The computational data are analyzed and discussed in the context of the experimental results previously obtained using surface plasmon resonance binding assays. We also provide experimental data on PCPE-1/GAG interactions obtained using inhibition assays with GAG oligosaccharides ranging from disaccharides to octadecasaccharides. Our results predict the localization of GAG-binding sites at the amino acid residue level onto PCPE-1 and is the first attempt to describe the effects of ions on protein-GAG binding using modeling approaches. In addition, this study allows us to get deeper insights into the in silico methodology challenges and limitations when applied to GAG-protein interactions.

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7

Coeler, Matthias, Vanessa van Laack, Frederieke Langer, Annegret Potthoff, Sören Höhn, Sebastian Reuber, Katharina Koscheck, and Mareike Wolter. "Infiltrated and Isostatic Laminated NCM and LTO Electrodes with Plastic Crystal Electrolyte Based on Succinonitrile for Lithium-Ion Solid State Batteries." Batteries 7, no. 1 (February 3, 2021): 11. http://dx.doi.org/10.3390/batteries7010011.

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We report a new process technique for electrode manufacturing for all solid-state batteries. Porous electrodes are manufactured by a tape casting process and subsequently infiltrated by a plastic crystal polymer electrolyte (PCPE). With a following isostatic lamination process, the PCPE was further integrated deeply into the porous electrode layer, forming a composite electrode. The PCPE comprises the plastic crystal succinonitrile (SN), lithium conductive salt LiTFSI and polyacrylonitrile (PAN) and exhibits suitable thermal, rheological (ƞ = 0.6 Pa s @ 80 °C 1 s−1) and electrochemical properties (σ > 10−4 S/cm @ 45 °C). We detected a lowered porosity of infiltrated and laminated electrodes through Hg porosimetry, showing a reduction from 25.6% to 2.6% (NCM infiltrated to laminated) and 32.9% to 4.0% (LTO infiltrated to laminated). Infiltration of PCPE into the electrodes was further verified by FESEM images and EDS mapping of sulfur content of the conductive salt. Cycling tests of full cells with NCM and LTO electrodes with PCPE separator at 45 °C showed up to 165 mAh/g at 0.03C over 20 cycles, which is about 97% of the total usable LTO capacity with a coulomb efficiency of between 98 and 99%. Cycling tests at 0.1C showed a capacity of ~128 mAh/g after 40 cycles. The C-rate of 0.2C showed a mean capacity of 127 mAh/g. In summary, we could manufacture full cells using a plastic crystal polymer electrolyte suitable for NCM and LTO active material, which is easily to be integrated into porous electrodes and which is being able to be used in future cell concepts like bipolar stacked cells.

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8

Astakhov, Evgeny. "«Eurocommunism» and the split of the Communist movement in Spain." Cuadernos Iberoamericanos, no. 4 (December 28, 2017): 7–15. http://dx.doi.org/10.46272/2409-3416-2017-4-7-15.

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In the period post Franco were created more favorable conditions for left parties, ﬁrst of all for Communist party. However, «eurocommunists» leadership of the Communist party of Spain (KPI) led her to a deep crisis. The creation in January 1984 of the new Communist party of the people of Spain (PCPE), despite the difﬁculties of institutional development, the complicated ﬁnancial situation, lack of personnel, became a signiﬁcant factor in the national political ﬁeld. After many years of political and ideological disarmament of the left forces in Spain appeared a party, acting with genuine class positions. At the same time, PCPE played the role of catalyst of processes oriented to shift to the left axis of the political life of the country. However, the current situation in the Spanish communist movement, the whole objective situation in Spain dictated the need for the uniﬁcation of the communists. That goal was answered by the creation of a left electoral coalition «United left».

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9

Weiss, Tali, Marina Brusel, Patricia Rousselle, and Efrat Kessler. "The NTR domain of procollagen C-proteinase enhancer-1 (PCPE-1) mediates PCPE-1 binding to syndecans-1, -2 and -4 as well as fibronectin." International Journal of Biochemistry & Cell Biology 57 (December 2014): 45–53. http://dx.doi.org/10.1016/j.biocel.2014.09.023.

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10

Chen, Li, Yimei Zhao, Xinyu Sun, Jun Jiang, Fengshou Wu, and Kai Wang. "Synthesis, singlet oxygen generation and DNA photocleavage of β,β′-conjugated polycationic porphyrins." Journal of Porphyrins and Phthalocyanines 23, no. 06 (May 28, 2019): 655–63. http://dx.doi.org/10.1142/s1088424619500378.

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In this paper, three [Formula: see text],[Formula: see text]-conjugated cationic porphyrin compounds were designed and synthesized. The structure of the intermediates and desired porphyrins were confirmed by UV, IR, 1H NMR, MS and elemental analysis. The interaction modes between these porphyrins and ct-DNA were studied by UV-vis spectroscopy and fluorescence emission spectroscopy. The results showed that PCP 1 had an external binding mode with DNA at low DNA concentration and could intercalate DNA with the increase of concentration. PCP 2 interacted with DNA through an external binding mode, and PCP 3 could insert into DNA. The binding constants ([Formula: see text] between PCP1[Formula: see text]PCP3 and ct-DNA were calculated to be 8.41 × 104, 7.33 × 104 and 4.14 × 104 M[Formula: see text], respectively. The singlet oxygen (1O[Formula: see text] generation of PCP1[Formula: see text]PCP3 was determined by the 1,3-diphenylisobenzofuran (DPBF) method using tetrapyridylporphyrin (H2TMPyP) as a reference. The 1O2 generation rate of PCP1[Formula: see text]PCP3 followed the order of PCP2 >PCP1>H2TMPyP >PCP3. Subsequently, the photocleavage effect of porphyrins on pBR322 plasmid DNA was studied by gel electrophoresis. At 10.0 [Formula: see text]M, PCP1 and PCP2 could cleave DNA completely. At 2.0 [Formula: see text]M, the cleavage rate of DNA by PCP3 was 57.5%, which was significantly higher than that of H2TMPyP (38.8%). These results verified that the amount of cationic ions in the porphyrin structure could affect the binding modes of porphyrins with DNA and their cleavage ability of DNA.

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11

Granados, Jersson, Juan Carlos Uribe, and Osmany Blanco. "Expresión de receptor tipo toll 4 en células deciduales estromales asociadas a complicaciones del embarazo." Revista Facultad de Ciencias de la Salud UDES 4, no. 2.S1 (June 30, 2017): 22. http://dx.doi.org/10.20320/rfcsudes.v4i2.s1.r09.

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Introducción: la asociación entre infección e inflamación se considera la causa principal de complicaciones como la pre-eclampsia. Las células deciduales estromales (DSC) constituyen el principal componente celular de la decidua. Las DSC son las principales protectoras frente a bacterias Gram negativas y señales de peligro que emanan de inflamación severa con o sin infección. Por medio de la expresión de TLR4 las DSC regulan la repuesta inmune durante la infección. Objetivo: Determinar la expresión de receptores TLR4, así como citoquinas producidas por las DSC de pacientes con pre-eclampsia, que podrían actuar como marcadores diagnósticos de activación inmunológica. Materiales y métodos: Estudio descriptivo. Las DSC fueron aisladas y cultivadas a partir de tejido placentario, de pacientes con parto vaginal sin complicaciones (PVSC, n=6), parto por cesárea electiva (PCE, n=6) y parto por cesárea con pre-eclampsia (PCPE, n=6). La caracterización fenotípica de las DSC se efectuó mediante análisis por citometría de flujo. La determinación de citoquinas se realizó mediante ELISAs. La expresión génica de TLR4 se valoró por qRT-PCR. Resultados: la pureza de las DSC se determinó por la expresión de antígenos característicos como: CD10, CD44, CD73 y CD90. La ausencia de CD45 y CD62p, descartó la contaminación con células leucocitarias y células epiteliales. La secreción de IL-6 por las DSC fue mayor en PVSC, que en PCE y PCPE. La expresión génica del TLR4 fue similar en las DSC. Conclusiones: Se confirmó la pureza de las DSC. La expresión génica de TLR4 en las DSC de PVSC, PCE y PCPE, no presentó diferencias significativas. Las DSC secretan IL-6 e IL-10.

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12

Calvo, Guillermo A., and Jacob A. Frenkel. "From Centrally Planned to Market Economy: The Road from CPE to PCPE." Staff Papers - International Monetary Fund 38, no. 2 (June 1991): 268. http://dx.doi.org/10.2307/3867100.

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13

Osinski, Géraldine. "Le pcpe : un nouveau dispositif pour les personnes en situation de handicap." Le Journal des psychologues 373, no. 1 (2020): 74. http://dx.doi.org/10.3917/jdp.373.0074.

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14

Frenkel, Jacob A., and Guillermo Calvo. "From Centrally-Planned to Market Economies: The Road from CPE to PCPE." IMF Working Papers 91, no. 17 (1991): 1. http://dx.doi.org/10.5089/9781451843446.001.

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15

Agrahari, Sunil K., Sangita D. Kumar, and Ashwini K. Srivastava. "Development of a Carbon Paste Electrode Containing Benzo-15-Crown-5 for Trace Determination of the Uranyl Ion by Using a Voltammetric Technique." Journal of AOAC INTERNATIONAL 92, no. 1 (January 1, 2009): 241–47. http://dx.doi.org/10.1093/jaoac/92.1.241.

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Abstract The interaction of macrocyclic compounds like crown ethers and UO22+ has been studied by electrochemical methods. A modified carbon paste electrode incorporating benzo-15-crown-5 (B15C5) was used to evaluate the electron transfer reaction of UO22+ by cyclic voltammetry, differential pulse voltammetry, and electrochemical impedance spectroscopy. Electrochemical impedance studies showed that charge transfer resistance was less for the B15C5-modified electrode than for the plain carbon paste electrode (PCPE). On the basis of these observations, a UO22+-sensitive crown ether chemically modified electrode (CME) for trace analysis was fabricated and investigated in aqueous solutions. It was found that a 5 B15C5CME for UO22+ showed a better voltammetric response than did the PCPE. UO22+ could be quantified at sub-μg/mL levels by differential pulse voltammetry with a detection limit of 0.03 μg/mL. By differential pulse adsorptive stripping voltammetry, UO22+ could be quantified in the working range of 0.002-0.2 μg/mL, with a detection limit of 1.1 μg/L. Simultaneous determination of UO22+, Pb2+, and Cd2+ was possible. The method was successfully applied to the determination of UO22+ in synthetic, as well as real, samples; the results were found to be comparable to those obtained by inductively coupled plasma-atomic emission spectroscopy.

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16

Weiss, Tali, Sylvie Ricard-Blum, Laura Moschcovich, Eitan Wineman, Shlomit Mesilaty, and Efrat Kessler. "Binding of Procollagen C-Proteinase Enhancer-1 (PCPE-1) to Heparin/Heparan Sulfate." Journal of Biological Chemistry 285, no. 44 (August 21, 2010): 33867–74. http://dx.doi.org/10.1074/jbc.m110.141366.

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17

Symoens, Sofie, Marjolijn Renard, Christelle Bonod-Bidaud, Delfien Syx, Elisabeth Vaganay, Fransiska Malfait, Sylvie Ricard-Blum, et al. "Identification of binding partners interacting with the α1-N-propeptide of type V collagen." Biochemical Journal 433, no. 2 (December 22, 2010): 371–81. http://dx.doi.org/10.1042/bj20101061.

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The predominant form of type V collagen is the [α1(V)]2α2(V) heterotrimer. Mutations in COL5A1 or COL5A2, encoding respectively the α1(V)- and α2(V)-collagen chain, cause classic EDS (Ehlers−Danlos syndrome), a heritable connective tissue disorder, characterized by fragile hyperextensible skin and joint hypermobility. Approximately half of the classic EDS cases remain unexplained. Type V collagen controls collagen fibrillogenesis through its conserved α1(V)-N-propeptide domain. To gain an insight into the role of this domain, a yeast two-hybrid screen among proteins expressed in human dermal fibroblasts was performed utilizing the N-propeptide as a bait. We identified 12 interacting proteins, including extracellular matrix proteins and proteins involved in collagen biosynthesis. Eleven interactions were confirmed by surface plasmon resonance and/or co-immunoprecipitation: α1(I)- and α2(I)-collagen chains, α1(VI)-, α2(VI)- and α3(VI)-collagen chains, tenascin-C, fibronectin, PCPE-1 (procollagen C-proteinase enhancer-1), TIMP-1 (tissue inhibitor of metalloproteinases-1), MMP-2 (matrix metalloproteinase 2) and TGF-β1 (transforming growth factor β1). Solid-phase binding assays confirmed the involvement of the α1(V)-N-propeptide in the interaction between native type V collagen and type VI collagen, suggesting a bridging function of this protein complex in the cell-matrix environment. Enzymatic studies showed that processing of the α1(V)-N-propeptide by BMP-1 (bone morphogenetic protein 1)/procollagen C-proteinase is enhanced by PCPE-1. These interactions are likely to be involved in extracellular matrix homoeostasis and their disruption could explain the pathogenetic mechanism in unresolved classic EDS cases.

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18

Kessler-Icekson, Gania, Hadassa Schlesinger, Sarit Freimann, and Efrat Kessler. "Regulation of procollagen C-proteinase (PCP) and its enhancer protein (PCPE) in the remodeling myocardium." Journal of Molecular and Cellular Cardiology 34, no. 6 (June 2002): A33. http://dx.doi.org/10.1016/s0022-2828(02)90858-8.

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19

Zhu, Qin, Wei Guo, Shengjie Zhang, Yang Feng, Xiao Wang, Daniel S. Greenspan, Libin Zhou, and Guo‐Ru Huang. "Synergistic effect of PCPE 1 and sFRP 2 on the processing of procollagens via BMP1." FEBS Letters 593, no. 7 (March 21, 2019): 760. http://dx.doi.org/10.1002/1873-3468.13363.

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20

Lagoutte, Priscillia, Emmanuel Bettler, Sandrine Vadon-Le Goff, and Catherine Moali. "Procollagen C-proteinase enhancer-1 (PCPE-1), a potential biomarker and therapeutic target for fibrosis." Matrix Biology Plus 11 (August 2021): 100062. http://dx.doi.org/10.1016/j.mbplus.2021.100062.

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21

Humppi, Tarmo. "Synthesis of polychlorinated phenoxyphenols (PCPP), phenoxyanisoles (PCPA), dibenzo-p-dioxins (PCDD), dibenzofurans (PCDF) and diphenyl ethers (PCDE)." Chemosphere 15, no. 9-12 (January 1986): 2003–6. http://dx.doi.org/10.1016/0045-6535(86)90502-3.

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22

Hassoun, Eyal, Mary Safrin, Hana Ziv, Sarah Pri-Chen, and Efrat Kessler. "Procollagen C-Proteinase Enhancer 1 (PCPE-1) as a Plasma Marker of Muscle and Liver Fibrosis in Mice." PLOS ONE 11, no. 7 (July 26, 2016): e0159606. http://dx.doi.org/10.1371/journal.pone.0159606.

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23

Huang, Yan, Randy Xun, Guanjun Chen, and Luying Xun. "Maintenance Role of a Glutathionyl-Hydroquinone Lyase (PcpF) in Pentachlorophenol Degradation by Sphingobium chlorophenolicum ATCC 39723." Journal of Bacteriology 190, no. 23 (September 26, 2008): 7595–600. http://dx.doi.org/10.1128/jb.00489-08.

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ABSTRACT Pentachlorophenol (PCP) is a toxic pollutant. Its biodegradation has been extensively studied in Sphingobium chlorophenolicum ATCC 39723. All enzymes required to convert PCP to a common metabolic intermediate before entering the tricarboxylic acid cycle have been characterized. One of the enzymes is tetrachloro-p-hydroquinone (TeCH) reductive dehalogenase (PcpC), which is a glutathione (GSH) S-transferase (GST). PcpC catalyzes the GSH-dependent conversion of TeCH to trichloro-p-hydroquinone (TriCH) and then to dichloro-p-hydroquinone (DiCH) in the PCP degradation pathway. PcpC is susceptible to oxidative damage, and the damaged PcpC produces glutathionyl (GS) conjugates, GS-TriCH and GS-DiCH, which cannot be further metabolized by PcpC. The fate and effect of GS-hydroquinone conjugates were unknown. A putative GST gene (pcpF) is located next to pcpC on the bacterial chromosome. The pcpF gene was cloned, and the recombinant PcpF was purified. The purified PcpF was able to convert GS-TriCH and GS-DiCH conjugates to TriCH and DiCH, respectively. The GS-hydroquinone lyase reactions catalyzed by PcpF are rather unusual for a GST. The disruption of pcpF in S. chlorophenolicum made the mutant lose the GS-hydroquinone lyase activities in the cell extracts. The mutant became more sensitive to PCP toxicity and had a significantly decreased PCP degradation rate, likely due to the accumulation of the GS-hydroquinone conjugates inside the cell. Thus, PcpF played a maintenance role in PCP degradation and converted the GS-hydroquinone conjugates back to the intermediates of the PCP degradation pathway.

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24

Hassoun, Eyal, Mary Safrin, Hana Ziv, Sarah Pri-Chen, and Efrat Kessler. "Correction: Procollagen C-Proteinase Enhancer 1 (PCPE-1) as a Plasma Marker of Muscle and Liver Fibrosis in Mice." PLOS ONE 11, no. 9 (September 6, 2016): e0162747. http://dx.doi.org/10.1371/journal.pone.0162747.

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25

Massoudi, Dawiyat, Colin J. Germer, Jeffrey M. Glisch, and Daniel S. Greenspan. "Procollagen C-proteinase enhancer 1 (PCPE-1) functions as an anti-angiogenic factor and enhances epithelial recovery in injured cornea." Cell and Tissue Research 370, no. 3 (September 21, 2017): 461–76. http://dx.doi.org/10.1007/s00441-017-2689-6.

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26

Hassoun, Eyal, Mary Safrin, Eitan Wineman, Peretz Weiss, and Efrat Kessler. "Data comparing the plasma levels of procollagen C-proteinase enhancer 1 (PCPE-1) in healthy individuals and liver fibrosis patients." Data in Brief 14 (October 2017): 777–81. http://dx.doi.org/10.1016/j.dib.2017.08.047.

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27

Massunari, Loiane, Renata Zoccal Novais, Márcio Teixeira Oliveira, Diego Valentim, Eloi Dezan Junior, and Cristiane Duque. "Antimicrobial Activity and Biocompatibility of the Psidium cattleianum Extracts for Endodontic Purposes." Brazilian Dental Journal 28, no. 3 (June 2017): 372–79. http://dx.doi.org/10.1590/0103-6440201601409.

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Abstract Psidium cattleianum (PC) has been displaying inhibitory effect against a variety of microorganisms, but this effect has not yet been tested against endodontic pathogens. The aim of this study was to evaluate the antimicrobial activity and biocompatibility of the aqueous (PCAE) and hydroethanolic (PCHE) extracts from Psidium cattleianum (PC) leaves. Minimum inhibitory concentration (MIC) and minimum lethal concentration (MLC) were determined using the microdilution broth method in order to analyze the antimicrobial effect against Enterococcus faecalis, Pseudomonas aeruginosa, Actinomyces israelii and Candida albicans in planktonic conditions. Biofilm assays were conducted only with the extracts that were able to determine the MLC for microorganisms in planktonic conditions. Immediate and late tissue reactions against PC extracts were evaluated using edemogenic test and histological analysis of subcutaneous implants in Wistar rats. The results showed that the MIC and MLC values ranged between 0.25 and 4 mg/mL. The MLC obtained for PCHE inhibited 100% growth of all the tested strains, except for C. albicans. PCAE had the same effect for E. faecalis and P. aeruginosa. Both PC extracts were able to eliminate E. faecalis biofilms and only the PCHE eliminated P. aeruginosa biofilms. The positive controls inhibited the growth of all tested strains in MIC and MLC essays, but no CHX tested concentrations were able to eliminate A. israelii biofilm. PCAE caused a discrete increase in the edema over time, while PCHE caused a higher initial edema, which decreased progressively. Both PCAE and PCHE extracts were biocompatible, but PCHE showed better results with slight levels of inflammation at 28 days. In conclusion, PCHE was biocompatible and presented better antimicrobial effect against important pathogens associated with persistent endodontic infections

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Xun, Luying, Sara M. Belchik, Randy Xun, Yan Huang, Huina Zhou, Emiliano Sanchez, ChulHee Kang, and Philip G. Board. "S-Glutathionyl-(chloro)hydroquinone reductases: a novel class of glutathione transferases." Biochemical Journal 428, no. 3 (May 27, 2010): 419–27. http://dx.doi.org/10.1042/bj20091863.

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Sphingobium chlorophenolicum completely mineralizes PCP (pentachlorophenol). Two GSTs (glutathione transferases), PcpC and PcpF, are involved in the degradation. PcpC uses GSH to reduce TeCH (tetrachloro-p-hydroquinone) to TriCH (trichloro-p-hydroquinone) and then to DiCH (dichloro-p-hydroquinone) during PCP degradation. However, oxidatively damaged PcpC produces GS-TriCH (S-glutathionyl-TriCH) and GS-DiCH (S-glutathionyl-TriCH) conjugates. PcpF converts the conjugates into TriCH and DiCH, re-entering the degradation pathway. PcpF was further characterized in the present study. It catalysed GSH-dependent reduction of GS-TriCH via a Ping Pong mechanism. First, PcpF reacted with GS-TriCH to release TriCH and formed disulfide bond between its Cys53 residue and the GS moiety. Then, a GSH came in to regenerate PcpF and release GS–SG. A TBLASTN search revealed that PcpF homologues were widely distributed in bacteria, halobacteria (archaea), fungi and plants, and they belonged to ECM4 (extracellular mutant 4) group COG0435 in the conserved domain database. Phylogenetic analysis grouped PcpF and homologues into a distinct group, separated from Omega class GSTs. The two groups shared conserved amino acid residues, for GSH binding, but had different residues for the binding of the second substrate. Several recombinant PcpF homologues and two human Omega class GSTs were produced in Escherichia coli and purified. They had zero or low activities for transferring GSH to standard substrates, but all had reasonable activities for GSH-dependent reduction of disulfide bond (thiol transfer), dehydroascorbate and dimethylarsinate. All the tested PcpF homologues reduced GS-TriCH, but the two Omega class GSTs did not. Thus PcpF homologues were tentatively named S-glutathionyl-(chloro)hydroquinone reductases for catalysing the GSH-dependent reduction of GS-TriCH.

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Porcionato, Marco Aurélio de Felicio, Telma Teresinha Berchielli, Gumercindo Loriano Franco, Pedro de Andrade, Roselene Nunes da Silveira, and Weber Vilas Bôas Soares. "Digestibilidade, degradabilidade e concentração amoniacal no rúmen de bovinos alimentados com polpa cítrica peletizada normal ou queimada." Revista Brasileira de Zootecnia 33, no. 1 (February 2004): 258–66. http://dx.doi.org/10.1590/s1516-35982004000100030.

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Objetivou-se avaliar a digestibilidade e a degradabilidade de dois tipos de polpa cítrica peletizada: normal (PCPN) e queimada (PCPQ), bem como o potencial de produção de nitrogênio amoniacal com dois níveis de inclusão na ração (40 e 60%). O delineamento experimental utilizado foi de blocos casualizados, em esquema fatorial 2 x 2 (dois tipos de polpa e dois níveis de inclusão) e testemunha. A inclusão de polpa cítrica peletizada (PCP) nas dietas, normal ou queimada, aumentou os coeficientes de digestibilidade dos nutrientes, quando comparados com a ração testemunha, exceto para a proteína bruta (PB). Com relação aos coeficientes de digestibilidade para os dois tipos de PCP, verificou-se maior digestibilidade dos nutrientes da PCPN em relação à PCPQ, principalmente em relação a PB, FDN e FDA, pressupondo que a utilização da PCPQ pode comprometer a qualidade da ração, ocasionando perdas na produtividade dos animais. A PCPN apresentou maiores taxas de degradação da MS e FDN, quando comparada à PCPQ. Verificou-se que o potencial de degradação foi alcançado com 48 horas de incubação para MS e FDN, para todos os tratamentos. Para produção de N-NH3 não foi observada diferença significativa entre os tratamentos. Contudo, a PCPQ pode ser utilizada na alimentação de bovinos, sendo sua utilização pautada na relação custo benefício.

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Sun, Jianlong, David J. Berg, and Brendan Twamley. "Bis(9,10-phenanthrenocyclopentadienyl)yttrium complexes: synthesis and solution behaviour." Canadian Journal of Chemistry 91, no. 12 (December 2013): 1281–87. http://dx.doi.org/10.1139/cjc-2013-0343.

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The synthesis of a number of yttrium metallocenes based on the phenanthrene-fused Cp ligand PCpR (R = H, Me, Ph) is reported. Acid−base (σ-bond metathesis) reactions between the parent HPCpR ligands and Y(CH2SiMe3)3(THF)2 afford the monomeric alkyl complexes Y(PCpR)2(CH2SiMe3)(THF) (R = H, 1; Me, 2; Ph, 3). Salt metathesis between Li+PCpMe− and YCl3 in THF similarly affords the monomeric chloride complex Y(PCpMe)2(Cl)(THF) (4), which reacts further with methyllithium to generate the bridging “ate” complex Y(PCpMe)2(μ-Me)2Li(THF)2 (5). Complex 3 undergoes rapid hydrogenolysis in the presence of phenylsilane to afford the crystalline bridging hydride dimer [Y(PCpPh)2]2(μ-H)2 (6). The X-ray structures of complexes 2 and 6 are reported along with the solution behaviour of 2.

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MORIMOTO, H., J. WADA, B. FONT, J. MOTT, D. HULMES, T. OOKOSHI, H. NAIKI, A. YASUHARA, A. NAKATSUKA, and K. FUKUOKA. "Procollagen C-proteinase enhancer-1 (PCPE-1) interacts with β2-microglobulin (β2-m) and may help initiate β2-m amyloid fibril formation in connective tissues." Matrix Biology 27, no. 3 (April 2008): 211–19. http://dx.doi.org/10.1016/j.matbio.2007.11.005.

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Moschcovich, Laura, and Efrat Kessler. "Data comparing the kinetics of procollagen type I processing by bone morphogenetic protein 1 (BMP-1) with and without procollagen C-proteinase enhancer 1 (PCPE-1)." Data in Brief 9 (December 2016): 883–87. http://dx.doi.org/10.1016/j.dib.2016.10.027.

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Marsh, E. E., E. K. Pearson, H. Ishikawa, S. Reierstad, and S. E. Bulun. "Type I procollagen COOH-terminal proteinase enhancer (PCOLCE) and its protein product procollagen C-endopeptidase enhancer-1 (PCPE-1) are differentially expressed in leiomyoma versus myometrium." Fertility and Sterility 90 (September 2008): S172. http://dx.doi.org/10.1016/j.fertnstert.2008.07.899.

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34

Dai, MingHua, Julie Bull Rogers, Joseph R. Warner, and Shelley D. Copley. "A Previously Unrecognized Step in Pentachlorophenol Degradation in Sphingobium chlorophenolicum Is Catalyzed by Tetrachlorobenzoquinone Reductase (PcpD)." Journal of Bacteriology 185, no. 1 (January 1, 2003): 302–10. http://dx.doi.org/10.1128/jb.185.1.302-310.2003.

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ABSTRACT The first step in the pentachlorophenol (PCP) degradation pathway in Sphingobium chlorophenolicum has been believed for more than a decade to be conversion of PCP to tetrachlorohydroquinone. We show here that PCP is actually converted to tetrachlorobenzoquinone, which is subsequently reduced to tetrachlorohydroquinone by PcpD, a protein that had previously been suggested to be a PCP hydroxylase reductase. pcpD is immediately downstream of pcpB, the gene encoding PCP hydroxylase (PCP monooxygenase). Expression of PcpD is induced in the presence of PCP. A mutant strain lacking functional PcpD has an impaired ability to remove PCP from the medium. In contrast, the mutant strain removes tetrachlorophenol from the medium at the same rate as does the wild-type strain. These data suggest that PcpD catalyzes a step necessary for degradation of PCP, but not for degradation of tetrachlorophenol. Based upon the known mechanisms of flavin monooxygenases such as PCP hydroxylase, hydroxylation of PCP should produce tetrachlorobenzoquinone, while hydroxylation of tetrachlorophenol should produce tetrachlorohydroquinone. Thus, we proposed and verified experimentally that PcpD is a tetrachlorobenzoquinone reductase that catalyzes the NADPH-dependent reduction of tetrachlorobenzoquinone to tetrachlorohydroquinone.

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Paasivirta, Jaakko, Juhani Tarhanen, and Jaakko Soikkeli. "Occurence and fate of polychlorinated aromatic ethers (PCDE, PCA, PCV, PCPA and PCBA) in environment." Chemosphere 15, no. 9-12 (January 1986): 1429–33. http://dx.doi.org/10.1016/0045-6535(86)90421-2.

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36

Rogers, Cheryl, and Simon Lemaire. "Characterization of [3H] desmethylimipramine binding in bovine adrenal medulla: interactions with σ- and (or) phencyclidine-receptor ligands." Canadian Journal of Physiology and Pharmacology 70, no. 11 (November 1, 1992): 1508–14. http://dx.doi.org/10.1139/y92-214.

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High-affinity binding sites (apparent KD 2.87 nM) for [3H]desmethylimipramine ([3H]DMI), have been demonstrated and characterized in membrane preparations of bovine adrenal medulla. The binding of [3H]DMI improved upon pretreatment of the membrane with KCl and was saturable, sodium dependent, and potently inhibited by nisoxetine and imipramine. [3H]DMI binding was also inhibited by various phencyclidine (PCP)- and (or) σ-receptor ligands, with the following order of potency: haloperidol > rimcazole > (−)-butaclamol > dextromethorphan > MK-801 > (+)-3-(3-hydroxyphenyl)-N-(1-propyl)piperidine((+)-3-PPP) > PCP > N-(2-thienyl)cyclohexyl-3,4-piperidine (TCP) > (+)-SKF-10047 > (−)-SKF-10047. The inhibition produced by σ ligands was not attributed to stimulation of either σ1- or σ2-receptors, owing to inactivity of the selective σ-receptor ligands (+)-pentazocine and 1,3-di(2-tolyl)guanidine (DTG). The inhibition of [3H]DMI binding by σ- and PCP-receptor ligands was not attributed to PCP1- or PCP2-receptor stimulation, owing to the decreased potency (100-fold) of these ligands in [3H]DMI assays compared with the affinity for brain PCP1 sites, and the ineffectiveness of the PCP2-ligand N-(1-(2-benzo(b)thiophenyl)cyclohexyl)piperidine (BTCP). Scatchard analysis of the inhibition by the σ-ligands haloperidol and (+)-3-PPP, as well as the PCP1 receptor ligand MK-801, demonstrated noncompetitive interaction with the site bound by [3H]DMI. These studies indicate that bovine adrenomedullary membranes possess a specific receptor for the noradrenaline uptake inhibitor [3H]DMI, which is sensitive to allosteric modulation produced by PCP and σ-ligands.Key words: desmethylimipramine, σ-receptor, phencyclidine, noradrenaline uptake, adrenal medulla.

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37

Polit, Justyna Teresa, Janusz Maszewski, and Marzena Rosiak. "lAA and BAP affect protein phosphorylation-dependent processes during sucrose-mediated G1 to S and G2 to M transitions in root meristem cells of Vicia faba." Acta Societatis Botanicorum Poloniae 73, no. 1 (2011): 17–22. http://dx.doi.org/10.5586/asbp.2004.003.

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In carbohydrate-starved root meristems of <em>Vicia faba </em>subsp. minor, the expression of two Principal Control Points located at the final stages of the G1 (PCP1) and G2 (PCP2) phases has been found to be correlated with a marked decrease of protein phosphorylation within cell nuclei, nucleoli and cytoplasm. Adopting the same experimental model in our present studies, monoclonal FITC conjugated antibodies that recognize phosphorylated form of threonine (αT<sup>P</sup>ab-FITC) were used to obtain an insight about how the indole-3-acetic acid (IAA), benzyl-6-aminopurine (BAP), and the mixture of both phytohormones influence the time-course changes in an overall protein phosphorylation during sucrose-mediated PCP1→S and PCP2→M transitions. Unsuspectedly, neither IAA, BAP, nor the mixture of both phytohormones supplied in combination with sucrose did up-regulate protein phosphorylation. However using the block-and-release method, it was shown that root meristems of Vicia provided with sucrose alone indicated higher levels of αT<sup>P</sup>ab-FITC. Contrarily, phytohormones supplied in combination with sucrose induced apparent decline in phosphorylation of cell proteins, which - when compared with the influence of sucrose alone - became increasingly evident in time. Thus, it seems probable, that a general decline in the amount of αT<sup>P</sup>ab-FITC labeled epitopes may overlay specific phosphorylations and dephosphorylations governed by the main cell cycle kinases and phosphatases.

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Hamdy, F. C., and M. Rouprêt. "L’étude PCPT." Progrès en Urologie 18 (April 2008): 40–43. http://dx.doi.org/10.1016/s1166-7087(08)70512-8.

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39

Kirby, RS. "PCPD editorial." Prostate Cancer and Prostatic Diseases 1, no. 5 (September 1998): 227. http://dx.doi.org/10.1038/sj.pcan.4500254.

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40

Ben‐Sasson, Eli, Oded Goldreich, Prahladh Harsha, Madhu Sudan, and Salil Vadhan. "Robust PCPs of Proximity, Shorter PCPs, and Applications to Coding." SIAM Journal on Computing 36, no. 4 (January 2006): 889–974. http://dx.doi.org/10.1137/s0097539705446810.

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Dinur, Irit, and Prahladh Harsha. "Composition of Low-Error 2-Query PCPs Using Decodable PCPs." SIAM Journal on Computing 42, no. 6 (January 2013): 2452–86. http://dx.doi.org/10.1137/100788161.

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42

Guo, J., and P. Waldron. "Deterioration of PCPV concrete." Nuclear Engineering and Design 198, no. 3 (June 2000): 211–26. http://dx.doi.org/10.1016/s0029-5493(99)00341-6.

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43

Urbanek, Petr, Herbert Bock, and Calin Vicol. "Percutaneous Cardiopulmonary Support (PCPS)." Cardiology 84, no. 3 (1994): 216–21. http://dx.doi.org/10.1159/000176401.

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44

Brunner, Laureline, Marina Canepa Allen, Mary Malebranche, Catherine Hudon, Nicolas Senn, Olivier Hugli, Francis Vu, Christina Akré, and Patrick Bodenmann. "Qualitative evaluation of primary care providers’ experiences caring for frequent users of the emergency department." BMJ Open 11, no. 6 (June 2021): e044326. http://dx.doi.org/10.1136/bmjopen-2020-044326.

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ObjectivesMany interventions have been developed over the years to offer frequent users of the emergency department (FUEDs) better access to quality coordinated healthcare. Despite recognising the role primary care physicians (PCPs) play in FUEDs’ care, to date their perceptions of case management, the most studied intervention, have rarely been assessed. Furthermore, a gap regarding PCPs’ experience of caring for FUEDs persists. Thus, this study aimed to explore PCPs’ perceptions of the care provided to FUEDs in emergency and primary care settings, their views on the local case management team (CMT), and their suggestions to improve FUEDs’ care.DesignQualitative study using in-depth semistructured interviews and inductive thematic analysis.SettingCanton of Vaud, Switzerland.ParticipantsThirty PCPs participated, 16 in private practice (PP-PCPs) and 14 based at the Lausanne University Centre of General Medicine and Public Health (Unisanté—U-PCPs).ResultsU-PCPs and PP-PCPs thought that most FUEDs’ emergency department (ED) visits were legitimate, but questioned ED adequacy to meet FUEDs’ needs. Yet, both PCP groups reported encountering many challenges in FUEDs’ care themselves. In this context, PP-PCPs seemed more satisfied of the care they provided to FUEDs than U-PCPs. Generally, U-PCPs seemed to find more value in the CMT to help them care for FUEDs than PP-PCPs. To enhance FUEDs’ care, U-PCPs and PP-PCPs suggested enhancing collaboration with other healthcare providers. U-PCPs also wished to increase their availability, and some PP-PCPs considered outpatient clinics, larger group practices or medical centres most appropriate to handle FUEDs’ needs.ConclusionsThis study highlights the many challenges PCPs face in caring for FUEDs, that a CM intervention has the potential to mitigate, and provides ways forward in improving FUEDs’ care, including reinforced communication with the CMT and ED physicians, and structural changes to their own way of delivering care to FUEDs.

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45

Guan, Jiazhen, Yuan Luo, and Bradley M. Denker. "Purkinje cell protein-2 (Pcp2) stimulates differentiation in PC12 cells by Gβγ-mediated activation of Ras and p38 MAPK." Biochemical Journal 392, no. 2 (November 22, 2005): 389–97. http://dx.doi.org/10.1042/bj20042102.

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Purkinje cell protein-2 (Pcp2 or L7) is highly expressed in cerebellar Purkinje cells and retinal bipolar neurons and interacts with the Gαi/o family of G-proteins. Although the expression pattern of Pcp2 in the developing central nervous system suggests a role in differentiation, its function remains unknown. We established Tet-off inducible expression of Pcp2 in PC12 cells (rat pheochromocytoma cells) to determine whether Pcp2 regulates neuronal differentiation. Utilizing a polyclonal antibody, Pcp2 was localized in the cell body and throughout neurites of differentiated PC12 cells, similar to its localization in cerebellar Purkinje cells. Pcp2 expression in PC12 cells stimulated process formation (5-fold) and NGF (nerve growth factor)-stimulated neurite length (2-fold). Under basal conditions, Pcp2-PC12 cells demonstrated a 5-fold increase in Ras activation relative to non-induced PC12 cells and there was no change in extracellular-signal-regulated kinase 1/2 activity with Pcp2 expression. However, Pcp2 induction led to a >3-fold increase in basal p38 MAPK (mitogen-activated protein kinase) activity and the addition of NGF significantly stimulated both Ras and p38 MAPK in Pcp2-PC12 cells relative to the controls. Pretreatment of Pcp2-PC12 cells with the p38-specific inhibitor SB203580 blocked both the increased neurite formation and NGF-stimulated neurite growth. Pertussis toxin treatment had no effect on neurite growth in control cells, but completely blocked Pcp2-mediated increased neurite growth. Transient transfection of the β-adrenergic receptor kinase C-terminus to prevent signalling through Gβγ in Pcp2-PC12 cells also inhibited the Pcp2-induced phenotype and reduced the Pcp2-stimulated Ras activation. Taken together, these findings demonstrate that Pcp2 induces differentiation in PC12 cells, in part through Gβγ-mediated Ras and p38 MAPK activation and suggest the potential for similar signalling mechanisms in Purkinje cells.

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MASAI, TAKASHI. "Percutaneous Cardiopulmonary Support System(PCPS)." Japanese journal of extra-corporeal technology 19, no. 1 (1993): 1–5. http://dx.doi.org/10.7130/hokkaidoshakai.19.1.

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KANO, NAOYUKI. "Effectiveness of hemodialysis in PCPS." Japanese journal of extra-corporeal technology 24, no. 1 (1997): 72–75. http://dx.doi.org/10.7130/hokkaidoshakai.24.72.

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Thoma, Yann, Alberto Dassatti, Daniel Molla, and Enrico Petraglio. "FPGA-GPU communicating through PCIe." Microprocessors and Microsystems 39, no. 7 (October 2015): 565–75. http://dx.doi.org/10.1016/j.micpro.2015.02.005.

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49

Dinur, Irit. "PCPs with small soundness error." ACM SIGACT News 39, no. 3 (September 2008): 41–57. http://dx.doi.org/10.1145/1412700.1412713.

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Hung, Dorothy Y., Thomas G. Rundall, Deborah J. Cohen, Alfred F. Tallia, and Benjamin F. Crabtree. "Productivity and Turnover in PCPs." Medical Care 44, no. 10 (October 2006): 946–51. http://dx.doi.org/10.1097/01.mlr.0000220828.43049.32.

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