Relevant bibliographies by topics / PCSK7

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'PCSK7'

Author: Grafiati
Published: 9 March 2023
Last updated: 31 July 2025

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Journal articles on the topic "PCSK7"

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Parvaz, Najmeh, and Zahra Jalali. "Molecular evolution of PCSK family: Analysis of natural selection rate and gene loss." PLOS ONE 16, no. 10 (2021): e0259085. http://dx.doi.org/10.1371/journal.pone.0259085.

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Proprotein convertases subtilisin kexins are serine endoproteases, playing critical roles in the biological functions, including lipid, glucose, and bile acid metabolism, as well as cell proliferation, migration, and metastasis. Experimental studies have demonstrated the physiological functions of PCSKs and their association with diseases; however, studies on the evolutionary history and diversification of these proteins are missing. In the present research, a bioinformatics study was conducted on the molecular evolution of several PCSKs family members and gene loss events across placental mam
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Porcheron, Chloé, Mailys Le Devehat, Anna Roubtsova, et al. "Blockade of colon cancer metastasis via single and double silencing ofPCSK7/PCSK9: enhanced T cells cytotoxicity in mouse and human." Journal for ImmunoTherapy of Cancer 13, no. 6 (2025): e011364. https://doi.org/10.1136/jitc-2024-011364.

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BackgroundImmunotherapy approaches based on T cells provided breakthroughs in cancer treatment but could cause many immune-related adverse events, and their efficacy is limited for many cancers with an acquired dysfunction/exhaustion of T cells. The present study presents a novel role in immunity for proprotein convertase subtilisin-kexin 7 (PCSK7), the seventh proprotein convertase of the 9-membered secretory proprotein convertase subtilisin-kexin (PCSK)-family.MethodsWe analyzed cell surface levels of various immune checkpoint proteins in human and mouse cell models in the presence or absenc
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Dongiovanni, Paola, Marica Meroni, Guido Baselli, et al. "PCSK7 gene variation bridges atherogenic dyslipidemia with hepatic inflammation in NAFLD patients." Journal of Lipid Research 60, no. 6 (2019): 1144–53. http://dx.doi.org/10.1194/jlr.p090449.

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Dyslipidemia and altered iron metabolism are typical features of nonalcoholic fatty liver disease (NAFLD). Proprotein convertase subtilisin/kexin type 7 (PCSK7) gene variation has been associated with circulating lipids and liver damage during iron overload. The aim of this study was to examine the impact of the PCSK7 rs236918 variant on NAFLD-related traits in 1,801 individuals from the Liver Biopsy Cohort (LBC), 500,000 from the UK Biobank Cohort (UKBBC), and 4,580 from the Dallas Heart Study (DHS). The minor PCSK7 rs236918 C allele was associated with higher triglycerides, aminotransferases
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Malakootian, Mahshid, Parisa Naeli, Seyed Javad Mowla, and Nabil G. Seidah. "Post-Transcriptional Effects of miRNAs on PCSK7 Expression and Function: miR-125a-5p, miR-143-3p, and miR-409-3p as Negative Regulators." Metabolites 12, no. 7 (2022): 588. http://dx.doi.org/10.3390/metabo12070588.

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The regulatory mechanism of PCSK7 gene is still unknown, although its encoded protein PC7 is the most ancient and highly conserved of all proprotein convertases and exhibits enzymatic and non-enzymatic functions in liver triglyceride regulation. Bioinformatics algorithms were used to predict regulatory microRNAs (miRNAs) of PCSK7 expression. This led to the identification of four miRNAs, namely miR-125a-5p, miR-143-3p, miR-409-3p, and miR-320a-3p, with potential binding sites on the 3′-untranslated region (3′-UTR) of human PCSK7 mRNA. The expression patterns of these miRNAs and PCSK7 mRNA were
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Buch, Stephan, Aneesh Sharma, Eleanor Ryan, et al. "Variants in PCSK7, PNPLA3 and TM6SF2 are risk factors for the development of cirrhosis in hereditary haemochromatosis." Alimentary Pharmacology & Therapeutics 53, no. 7 (2021): 830–43. http://dx.doi.org/10.1111/apt.16252.

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SummaryBackgroundCirrhosis develops in <10% of individuals homozygous for the C282Y variant in the homeostatic iron regulator (HFE) gene. Carriage of PCSK7:rs236918 is associated with an increased risk of cirrhosis in this population.AimTo determine if genetic variants significantly associated with the risk of alcohol‐ and NAFLD‐related cirrhosis also modulate the cirrhosis risk in C282Y homozygotes.MethodsVariants in PCSK7, PNPLA3, TM6SF2, MBOAT7 and HSD17B13 were genotyped in 1319 C282Y homozygotes, from six European countries, of whom 171 (13.0%) had cirrhosis. Genotypic and allelic asso
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Kupcinskas, Juozas, Irena Valantiene, Greta Varkalaitė, et al. "PNPLA3 and RNF7 Gene Variants are Associated with the Risk of Developing Liver Fibrosis and Cirrhosis in an Eastern European Population." Journal of Gastrointestinal and Liver Diseases 26, no. 1 (2017): 37–43. http://dx.doi.org/10.15403/jgld.2014.1121.261.pnp.

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Background & Aims: Genome-wide association studies have revealed an association between the risk of developing liver fibrosis or cirrhosis and the single nucleotide polymorphisms (SNPs) of the PNPLA3, RNF7, MERTK and PCSK7 genes. We aimed to validate these results in an Eastern European population.Methods: We evaluated the associations between the PNPLA3 (rs738409), RNF7 (rs16851720), MERTK (rs4374383) and PCSK7 (rs236918) variants and liver fibrosis and cirrhosis in a series of consecutive patients recruited at the Department of Gastroenterology, Lithuanian University of Health Sciences H
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Vargas-Alarcón, Gilberto, Oscar Pérez-Méndez, Héctor González-Pacheco, et al. "The rs508487, rs236911, and rs236918 Genetic Variants of the Proprotein Convertase Subtilisin–Kexin Type 7 (PCSK7) Gene Are Associated with Acute Coronary Syndrome and with Plasma Concentrations of HDL-Cholesterol and Triglycerides." Cells 10, no. 6 (2021): 1444. http://dx.doi.org/10.3390/cells10061444.

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Dyslipidemia has a substantial role in the development of acute coronary syndrome (ACS). Previous reports, including genome-wide associations studies (GWAS), have shown that some genetic variants of the proprotein convertase subtilisin–kexin type 7 (PCSK7) gene are associated with plasma lipid levels. In the present study, we evaluated whether PCSK7 gene polymorphisms are significantly associated with the plasma lipid profile and ACS. Three PCSK7 gene polymorphisms (rs508487 T/C, rs236911 C/A, and rs236918 C/G) were determined using TaqMan genotyping assays in a group of 603 ACS patients and 6
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Ashraf, Yahya, Stéphanie Duval, Vatsal Sachan, et al. "Proprotein convertase 7 (PCSK7) reduces apoA‐V levels." FEBS Journal 287, no. 16 (2020): 3565–78. http://dx.doi.org/10.1111/febs.15212.

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Tobiasch, Moritz, Benedikt Schaefer, André Viveiros, Herbert Tilg, Ivo Graziadei, and Heinz Zoller. "Survival in HFE hemochromatosis: influence of polymorphisms in HSD17B13, GNPAT, and PCSK7." Journal of Hepatology 73 (August 2020): S551—S552. http://dx.doi.org/10.1016/s0168-8278(20)31575-0.

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Schwienbacher, Christine, Alice Serafin, Alessandra Zanon, Peter P. Pramstaller, Irene Pichler, and Andrew A. Hicks. "Involvement of proprotein convertase PCSK7 in the regulation of systemic iron homeostasis." Hepatology 58, no. 5 (2013): 1860–61. http://dx.doi.org/10.1002/hep.26392.

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Dissertations / Theses on the topic "PCSK7"

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GRENI, FEDERICO. "Variabilità fenotipica nell'emocromatosi: studio di due potenziali modificatori genetici in PCSK7 e GNPAT." Doctoral thesis, Università degli Studi di Milano-Bicocca, 2017. http://hdl.handle.net/10281/140994.

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Introduction and aim: Hereditary hemochromatosis (HH) is a genetic disease characterized by a progressive iron overload in different tissues. Homozygosity for the p.C282Y mutation is the most frequent genotype associated with the disease and it is directly responsible for an inappropriate production of hepcidin, the main regulator of iron homeostasis. Several evidences indicated that p.C282Y homozygous genotype has an incomplete penetrance due to the combined action of genetic and acquired modifier factors. Recently, the attention was focused on GNPAT rs11558492 and PCSK7 rs236918 single nucle
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Bhat, Mamatha. "Expression of PCSK9 in Hepatocellular Carcinoma." Thesis, McGill University, 2012. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=106271.

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Hepatocellular carcinoma (HCC) is an often fatal condition due to late diagnosis, resistance to existing anticancer agents, as well as underlying liver disease that can limit the use of hepatotoxic chemotherapy. Proprotein convertases (PCs) are serine proteases that convert a variety of growth factors, cell surface glycoproteins, receptors and metalloproteinases into their active forms, thus regulating the biological activity of these proteins. PCs have been found to be upregulated in various malignancies. Growth factors implicated in HCC, such as IGF-1, HGF, VEGF and PDGF, have all been shown
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DA, DALT LORENZO. "IMPACT OF PCSK9 ON EXTRAHEPATIC TISSUES." Doctoral thesis, Università degli Studi di Milano, 2021. http://hdl.handle.net/2434/813080.

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Introduzione e scopo: la proproteina convertasi subtilisina kexin tipo 9 (PCSK9) è una glicoproteina di 692 amminoacidi che appartiene alla famiglia delle proproteine convertasi. È prodotta principalmente dal fegato dal quale viene secreta nel torrente circolatorio. PCSK9 interagisce con diversi recettori della famiglia dell’LDLr, inclusi VLDLr, LRP1 ma anche con CD36, e guida la loro degradazione lisosomiale. La mancanza di PCSK9 determina quindi una maggiore espressione dei recettori della famiglia LDLr e favorisce l'accumulo di lipidi nei tessuti extraepatici. L’eccesso di lipidi cellulari
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Stefan, Elias. "Familjär hyperkolesterolemi (FH) – analys av prevalens i Stockholm och hälsoekonomiska konsekvenser av tidigdiagnostik och behandling." Thesis, Uppsala universitet, Institutionen för farmaci, 2021. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-434844.

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Background: Familial hypercholesterolemia (FH) is a genetic disorder estimated to affect 0,4 % of the world's population (1 in 250). Patients with FH have abnormally high LDL-cholesterol.  Aim: The aim of this study was to estimate the prevalence of FH in Stockholm County and to evaluate the health economic impact of diagnosing people with FH early in life. Methods: Two algorithms were used to estimate the number of people with high LDLcholesterol. The first method applied data on cholesterol measurements from patients in Stockholm County between 2006-2008 and a modified version of Dutch Lipid
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Giunzioni, I. "MACROPHAGE EXPRESSION OF PCSK9 INFLUENCES ATHEROSCLEROSIS DEVELOPMENT." Doctoral thesis, Università degli Studi di Milano, 2014. http://hdl.handle.net/2434/229332.

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Proprotein Convertase Subtilisin/Kexin 9 (PCSK9) regulates low‐density lipoprotein (LDL) cholesterol levels by binding and degrading hepatic LDL receptor (LDLR), thus promoting atherosclerosis. Little is known of PCSK9 effect in macrophages and whether this contributes to the development of the atheroma. To test the effect of human (h) PCSK9 expression on atherosclerosis we developed transgenic mice expressing hPCSK9 on wild type (WT), LDLR‐/‐ or apoE‐/‐ background. We first demonstrated that both mPCSK9 and hPCSk9 are expressed at the mRNA level and secreted in the culture medium by MPM.
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Kourimate, Sanae. "Pcsk9 : régulation et implication dans le syndrome métabolique." Nantes, 2008. https://archive.bu.univ-nantes.fr/pollux/show/show?id=4ac185ba-f999-45ff-9241-4278a9699b5c.

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Proprotein convertase subtilisin/kexin type 9 (PCSK9) est une serine protéase, jouant un rôle important dans la régulation des niveaux de cholestérol circulant. Les mutations gains de fonctions de PCSK9 sont associées à une hypercholestérolémie autosomale dominante due à une élévation des concentrations en Low Density Lipoprotein associées au cholestérol (LDLc). A l'inverse, des mutations perte de fonction de PCSK9, sont associées à une diminution de ces concentrations et à une réduction de 88% du risque d'apparition de maladies coronariennes. Dans le réticulum endoplasmique, Pro-PCSK9 subit u
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CANCLINI, LAURA. "PROPROTEIN CONVERTASE SUBTILISIN/KEXIN TYPE 9 PREFERENTIALLY ASSOCIATES WITH A SPECIFIC LDL SUBFRACTION: A DETAILED CHARACTERIZATION AND STUDY OF THE EFFECTS OF ANTI-PCSK9 MABS TREATMENT." Doctoral thesis, Università degli Studi di Milano, 2022. https://hdl.handle.net/2434/947250.

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BACKGROUND AND AIM: Proprotein Convertase Subtilysin/Kexin Type 9 is a key regulator of LDL-C levels. Nevertheless, its physiological modulation is not fully understood. It is unclear whether PCSK9 has biological effects other than degrading the LDLR; consensus is lacking also on how PCSK9 is transported in the bloodstream, and whether this influences PCSK9 function. There are several conflicting data about PCSK9’s possible association with different lipoprotein subtypes. The biological function of the lipoprotein-bound PCSK9, also, is still a matter of debate. Due to its role in modulating pl
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Dewpura, Thilina. "Importance of phosphorylation in PCSK9 processing, stability and function." Thesis, University of Ottawa (Canada), 2010. http://hdl.handle.net/10393/28604.

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Proprotein convertase subtilisin/kexin type 9 (PCSK9) is a secreted glycoprotein regulating the degradation of low density lipoprotein receptor. Single nucleotide polymorphisms in PCSK9 associate with both hyper- and hypo-cholesterolemia; studies show significant reduction in risk of coronary heart disease for 'loss of function' PCSK9 carriers. We used a combination of mass spectrometry and radiolabeling to report that PCSK9 is phosphorylated at two sites, Ser47 in its propeptide, and Ser688 in its C-terminus. Site directed mutagenesis (SDM) demonstrated that a Golgi casein kinase-like kinase
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Roubtsova, Anna. "Partial hepatectomy and liver regeneration in PCSK9 knockout mice." Thesis, McGill University, 2008. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=112356.

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The proprotein convertase subtilisin/kexin type 9, PCSK9, belongs to the proprotein convertase (PC) family. Human mutations in the gene encoding PCSK9 lead to either familial hyper- or hypocholesterolemia, resulting from a gain or loss of function, respectively. Mice lacking PCSK9 are viable and show a 42% decrease in plasma cholesterol levels. The enzyme triggers the degradation of the low density lipoprotein receptor (LDLR) through a partially unknown mechanism.<br>PCSK9 is very abundant in the liver and intestine during development and adulthood. Hepatocytes have a capacity to reproduce the
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Torrinha, José Maria de Queiroz e. Lencastre Fleming. "Inibidores PCSK9: nova estratégia para o tratamento da hipercolesterolemia." Master's thesis, [s.n.], 2015. http://hdl.handle.net/10284/5293.

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Projeto de Pós-Graduação/Dissertação apresentado à Universidade Fernando Pessoa como parte dos requisitos para obtenção do grau de Mestre em Ciências Farmacêuticas<br>As doenças cardiovasculares são uma das principais causas de morbilidade e mortalidade a nível mundial. Neste enquadramento, um dos principais fatores de risco associado às doenças cardiovasculares é a hipercolesterolemia. As opções farmacológicas existentes para o tratamento e prevenção desta dislipidemia centram-se, sobretudo, no uso de fármacos como as estatinas, a ezetimiba, os fibratos, o ácido nicotínico e as resinas seques
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Books on the topic "PCSK7"

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Tuakli-Wosornu, Yetsa Adebodunde. Molecular and clinical chracterization of loss-of-function mutations in proprotein convertase subtilisin/kexin 9 (PCSK₉) and the genetic absence of PCSK₉. 2007.

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Xu, Weiming. New Cardiovascular Research: PCSK9 As a New Therapeutic Target for Cardiovascular Disease. Independently Published, 2021.

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Muller, Jurgen. Regeln Mit Simatic: Praxisbuch Fr Regelungen Mit Simatic S7 Und Simatic PCs7. Not Avail, 2003.

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Controlling with SIMATIC: Practice Book for SIMATIC S7 and SIMATIC PCS7 Control Systems. Wiley-VCH, 2005.

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Müller, Jürgen, Bernd-Markus Pfeiffer, and Roland Wieser. Regeln Mit SIMATIC: Praxisbuch Für Regelungen Mit SIMATIC und SIMATIC S7 PCS7 Für Die Prozessautomatisierung. Publicis MCD Werbeagentur GmbH, 2020.

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Müller, Jürgen, Bernd-Markus Pfeiffer, and Roland Wieser. Regeln Mit SIMATIC: Praxisbuch Für Regelungen Mit SIMATIC und SIMATIC S7 PCS7 Für Die Prozessautomatisierung. Publicis MCD Werbeagentur GmbH, 2012.

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Regeln mit SIMATIC: Praxisbuch für Regelungen mit SIMATIC S7 und SIMATIC PCS7 für die Prozessautomatisierung. Wiley & Sons, Limited, John, 2011.

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Atta-ur-Rahman and M. Iqbal Choudhary, eds. Frontiers in Cardiovascular Drug Discovery: Volume 4. BENTHAM SCIENCE PUBLISHERS, 2019. http://dx.doi.org/10.2174/97816810839951180401.

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Frontiers in Cardiovascular Drug Discovery is an eBook series devoted to publishing the latest advances in cardiovascular drug design and discovery. Each volume brings reviews on the biochemistry, in-silico drug design, combinatorial chemistry, high-throughput screening, drug targets, recent important patents, and structure-activity relationships of molecules used in cardiovascular therapy. The eBook series should prove to be of great interest to all medicinal chemists and pharmaceutical scientists involved in preclinical and clinical research in cardiology. The fourth volume of the series cov
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High Cholesterol: Education for Patients and the Public. Exon Publications, 2025. https://doi.org/10.36255/high-cholesterol.

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High cholesterol is a common health condition that increases the risk of heart disease, stroke, and other cardiovascular problems. This article provides a comprehensive guide on high cholesterol, including its causes, symptoms, diagnosis, treatment, and long-term management. It begins with an explanation of what cholesterol is and how excessive levels can harm the body. The article highlights the widespread prevalence of high cholesterol, noting how lifestyle choices and genetic factors, including mutations in the LDLR, APOB, and PCSK9 genes, play a role in cholesterol regulation. The symptoms
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Atherosclerosis: Education for Patients and the Public. Exon Publications, 2025. https://doi.org/10.36255/atherosclerosis.

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Atherosclerosis is a condition in which fatty deposits build up inside the arteries, causing them to narrow and harden, leading to reduced blood flow. This article provides a detailed guide on atherosclerosis, explaining its causes, symptoms, diagnosis, treatment, and long-term management. It begins by defining atherosclerosis and describing how plaque accumulates in the arteries over time, increasing the risk of heart attacks, strokes, and other cardiovascular problems. The article discusses how common this condition is and highlights genetic factors such as the APOE and PCSK9 genes that may
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Book chapters on the topic "PCSK7"

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Wright, R. Scott. "PCSK9 Inhibiting siRNA." In Stroke Revisited: Dyslipidemia in Stroke. Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-16-3923-4_12.

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Ahmed, Zain, Prerak Juthani, Megan Lee, and Nihar R. Desai. "PCSK9 Inhibiting Monoclonal Antibodies." In Stroke Revisited: Dyslipidemia in Stroke. Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-16-3923-4_11.

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Desnick, Robert J., Orlando Guntinas-Lichius, George W. Padberg, et al. "FHBL due to Defective PCSK9." In Encyclopedia of Molecular Mechanisms of Disease. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_8889.

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Choquet, Hélène, Pieter Stijnen, and John W. M. Creemers. "Genetic and Functional Characterization of PCSK1." In Methods in Molecular Biology. Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61779-204-5_13.

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Wang, Zuo, Zhi-Han Tang, Yun-Chen Lv, Lu-Shan Liu, and Zhi-Sheng Jiang. "Bioinformatic Analysis of PCSK9 Related Caspase3 Activation." In Recent Advances in Computer Science and Information Engineering. Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-25778-0_73.

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Ooi, Teik Chye, and Hussein Abujrad. "PCSK9 as a Biomarker of Cardiovascular Disease." In Biomarkers in Cardiovascular Disease. Springer Netherlands, 2016. http://dx.doi.org/10.1007/978-94-007-7678-4_20.

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Ooi, Teik Chye, and Hussein Abujrad. "PCSK9 as a Biomarker of Cardiovascular Disease." In Biomarkers in Cardiovascular Disease. Springer Netherlands, 2015. http://dx.doi.org/10.1007/978-94-007-7741-5_20-1.

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Farnier, Michel. "Statins and PCSK9 Inhibitors: Defining the Correct Patients." In Combination Therapy In Dyslipidemia. Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-20433-8_9.

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Fradeneck, Katherine, Merna Mikhail, and Robert Danoff. "Novel Non Statin Therapy for Hyperlipidemia-PCSK9-2018." In Top Articles in Primary Care. Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-25620-2_37.

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Innocenti, Francesca, Valentina Di Maria, Alice Poggi, and Riccardo Pini. "Biomarkers of Sepsis and a Focus on PCSK9." In Biomarkers in Trauma, Injury and Critical Care. Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-07395-3_40.

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Conference papers on the topic "PCSK7"

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Pang, Qinkang, Yuxuan Cai, and Yunchong Long. "Design of Integrated Control System for Industrial Boilers Based on PCS7." In 2024 3rd International Symposium on Sensor Technology and Control (ISSTC). IEEE, 2024. https://doi.org/10.1109/isstc63573.2024.10824054.

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Stickel, F., S. Buch, E. Ryan, et al. "TM6SF2 rs58542926 und PCSK7 rs236918 sind genetische Risikoloci einer Leberzirrhose bei hereditärer Hämochromatose." In Viszeralmedizin 2017. Georg Thieme Verlag KG, 2017. http://dx.doi.org/10.1055/s-0037-1604986.

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Buch, S., F. Stickel, H. Zoller, et al. "Variants in TM6SF2, PNPLA3 and PCSK7 are risk factors for the development of cirrhosis in people with genetic haemochromatosis." In 35. Jahrestagung der Deutschen Arbeitsgemeinschaft zum Studium der Leber. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0038-1677064.

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Coelho, Nathália Greco, Luana Bernardes Xavier, Gabriela Lopes Martins, et al. "Avaliação dos polimorfismos rs505151 e rs562556 da pró-proteína convertase subtilisina kexina do tipo 9 relacionados aos níveis dos parâmetros do perfil lipídico em indivíduos dislipidêmicos." In Resumos do 56º Congresso Brasileiro de Patologia Clínica/Medicina Laboratorial. Zeppelini Editorial e Comunicação, 2024. https://doi.org/10.5327/1516-3180.142s1.11100.

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Objetivo: Avaliar os polimorfismos do gene da pró-proteína convertase subtilisina kexina do tipo 9 (PCSK9), proteína associada à degradação de receptores de LDL, e a sua associação com o perfil lipídico em indivíduos dislipidêmicos. Método: Este estudo incluiu um total de 216 indivíduos adultos, dos quais 109 eram dislipidêmicos e 107 normolipêmicos correspondentes ao grupo controle, todos avaliados por exames clínicos e laboratoriais. As análises moleculares dos polimorfismos rs505151 e rs562556 no gene da PCSK9 foram realizadas por PCR em Tempo Real, utilizando-se o TaqMan SNP. As análises e
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Tent, Michiel. "Oral PCSK9 inhibitor significantly lowers LDL-C." In ACC 2023 Scientific Session, edited by Marc Bonaca. Medicom Medical Publishers, 2023. http://dx.doi.org/10.55788/629cbf29.

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Martin Plagaro, Cesar, Kepa B. Uribe, Asier Benito Vicente, et al. "Hiperkolesterolemia Familiarra: PCSK9 aldaeren karakterizazioa tratamendu pertsonalizaturako." In III. Ikergazte. Nazioarteko ikerketa euskaraz. UEU arg, 2019. http://dx.doi.org/10.26876/ikergazte.iii.04.10.

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Niemann, B., L. Li, F. Knapp, R. Schulz, and S. Rohrbach. "Modifying Epicardial PCSK9 Expression to Protect Cardiac Function?" In 48th Annual Meeting German Society for Thoracic, Cardiac, and Vascular Surgery. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1678923.

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8

Sha, Xiangtong, and Yueqiang Wang. "PCSK1 Variants and Obesity: Relationship in Different Population." In 2021 International Conference on Public Art and Human Development ( ICPAHD 2021). Atlantis Press, 2022. http://dx.doi.org/10.2991/assehr.k.220110.187.

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9

Ferrer Machín, A., S. Martin Rodriguez, J. Vilar Rodriguez, et al. "4CPS-178 Effect of pcsk9 inhibitors on hypercholesterolemia." In 28th EAHP Congress, Bordeaux, France, 20-21-22 March 2024. British Medical Journal Publishing Group, 2024. http://dx.doi.org/10.1136/ejhpharm-2024-eahp.282.

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10

Lecis, M., E. Viglione, S. Strobino, and G. Ceravolo. "5PSQ-017 PCSK-9 inhibitors: real world effectiveness." In 25th EAHP Congress, 25th–27th March 2020, Gothenburg, Sweden. British Medical Journal Publishing Group, 2020. http://dx.doi.org/10.1136/ejhpharm-2020-eahpconf.334.

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Reports on the topic "PCSK7"

1

Zhang, Lingnan, Fang Zhang, Xinwei Jia, et al. Targeting PCSK9 in Heterozygous familial hypercholesterolemia: a meta-analysis of randomized controlled trials. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2024. http://dx.doi.org/10.37766/inplasy2024.3.0095.

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2

Yu, Yani, Lei Chen, Honghong Zhang, et al. Racial differences in the safety and efficacy of PCSK9 inhibitors in the treatment of hyperlipidemia：A Systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2021. http://dx.doi.org/10.37766/inplasy2021.11.0047.

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3

Niu, Xiaowei, and Shuwen Hu. Efficacy and safety of PCSK9 inhibitors and statin lipid-lowering therapy in coronary atherosclerosis: A meta-analysis of randomized trials. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.12.0019.

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4

Kao, Guoying, Chuan Chen, Ying Zhang, Yi Xu, and Gang Xu. Efficacy and Safety of PCSK-9 Inhibitors in Patients with Acute Coronary Syndrome: A Systematic Review and Network Meta-Analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2025. https://doi.org/10.37766/inplasy2025.7.0037.

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5

Sun, Jing-Chao. The intense lipid-lowering strategies of PCSK9 inhibitor or ezetimibe for cardiovascular events in patients with coronary heart disease: a systemic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2023. http://dx.doi.org/10.37766/inplasy2023.4.0040.

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You might also be interested in the extended bibliographies on the topic 'PCSK7' for particular source types:
Journal articles Dissertations / Theses
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