Relevant bibliographies by topics / PDE2A

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'PDE2A'

Author: Grafiati
Published: 6 July 2024
Last updated: 31 July 2025

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Journal articles on the topic "PDE2A"

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Skryabin, Egor B., Kirstie A. De Jong, Hariharan Subramanian, et al. "CRISPR/Cas9 Knock-Out in Primary Neonatal and Adult Cardiomyocytes Reveals Distinct cAMP Dynamics Regulation by Various PDE2A and PDE3A Isoforms." Cells 12, no. 11 (2023): 1543. http://dx.doi.org/10.3390/cells12111543.

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Cyclic nucleotide phosphodiesterases 2A (PDE2A) and PDE3A play an important role in the regulation of cyclic adenosine monophosphate (cAMP) and cyclic guanosine monophosphate (cGMP)-to-cAMP crosstalk. Each of these PDEs has up to three distinct isoforms. However, their specific contributions to cAMP dynamics are difficult to explore because it has been challenging to generate isoform-specific knock-out mice or cells using conventional methods. Here, we studied whether the CRISPR/Cas9 approach for precise genome editing can be used to knock out Pde2a and Pde3a genes and their distinct isoforms
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Ivey, F. Douglas, Lili Wang, Didem Demirbas, Christina Allain, and Charles S. Hoffman. "Development of a Fission Yeast-Based High-Throughput Screen to Identify Chemical Regulators of cAMP Phosphodiesterases." Journal of Biomolecular Screening 13, no. 1 (2007): 62–71. http://dx.doi.org/10.1177/1087057107312127.

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Cyclic nucleotide phosphodiesterases (PDEs) comprise a superfamily of enzymes that serve as drug targets in many human diseases. There is a continuing need to identify high-specificity inhibitors that affect individual PDE families or even subtypes within a single family. The authors describe a fission yeast-based high-throughput screen to detect inhibitors of heterologously expressed adenosine 3′,5′-cyclic monophosphate (cAMP) PDEs. The utility of this system is demonstrated by the construction and characterization of strains that express mammalian PDE2A, PDE4A, PDE4B, and PDE8A and respond a
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Xu, *Ying. "CYCLIC NUCLEOTIDE PHOSPHODIESTERASE 2: A NOVEL TARGET FOR THE TREATMENT OF DEMENTIAS ASSOCIATED WITH ALZHEIMER’ S DISEASE." International Journal of Neuropsychopharmacology 28, Supplement_1 (2025): i290—i291. https://doi.org/10.1093/ijnp/pyae059.514.

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Abstract Background The failure to find effective therapies in the past two decades underscores the challenge and the urgent need for identifying novel targets against Alzheimer's disease (AD). Cyclic nucleotide phosphodiesterase 2 (PDE2) is a "druggable target" which has more advantage than currently developed PDE4 and PDE5 inhibitors due to fewer side effects. Aims & Objectives The overall objective of this proposal is to investigate the role of PDE2 in mediating mitochondrial dysfunction and neurodegeneration, which should provide the rationale for future research toward developing pote
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Manns, J. M., K. J. Brennan, S. B. Sheth, and R. W. Colman. "Differential Regulation of Human Platelet Responses by cGMP Inhibited and Stimulated cAMP Phosphodiesterases." Thrombosis and Haemostasis 87, no. 05 (2002): 873–79. http://dx.doi.org/10.1055/s-0037-1613099.

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SummaryPlatelets contain two cAMP phosphodiesterases (PDEs) which regulate intracellular cAMP levels, cGMP-inhibited cAMP PDE (PDE3A) and cGMP-stimulated PDE (PDE2A). Using the PDE3 inhibitor, milrinone and the PDE2 inhibitor, erythro-9-(2-hydroxyl-3-nonyl)adenine (EHNA), we have explored the contribution of each PDE to the regulation of platelet function. Inhibition of PDE2 resulted in higher levels of intracellular cAMP than inhibition of PDE3A suggesting this PDE may be the more important regulator of cAMP in human platelets. However, a concentration-dependent inhibition of agonist-induced
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Carvalho, Thays Maria da Conceição Silva, Silvia Cardarelli, Mauro Giorgi, Andrea Lenzi, Andrea M. Isidori, and Fabio Naro. "Phosphodiesterases Expression during Murine Cardiac Development." International Journal of Molecular Sciences 22, no. 5 (2021): 2593. http://dx.doi.org/10.3390/ijms22052593.

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3′-5′ cyclic nucleotide phosphodiesterases (PDEs) are a large family of enzymes playing a fundamental role in the control of intracellular levels of cAMP and cGMP. Emerging evidence suggested an important role of phosphodiesterases in heart formation, but little is known about the expression of phosphodiesterases during cardiac development. In the present study, the pattern of expression and enzymatic activity of phosphodiesterases was investigated at different stages of heart formation. C57BL/6 mice were mated and embryos were collected from 14.5 to 18.5 days of development. Data obtained by
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Chen, Ling, Suying Cui, Haiyang Yu, et al. "Reduced phosphodiesterase-2 activity in the amygdala results in anxiolytic-like effects on behavior in mice." Journal of Psychopharmacology 33, no. 5 (2019): 568–76. http://dx.doi.org/10.1177/0269881119832753.

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Background: Phosphodiesterase-2 (PDE2) is a cyclic nucleotide phosphodiesterase and is highly expressed in the amygdala, which suggests its important role in anxiety-like behavior. Aims: The present study examined whether reduced PDE2A expression in the central nucleus of the amygdala (CeA) produces anxiolytic-like effects in mice. Methods: PDE2A knockdown in amygdaloid (AR5) cells or the CeA was established using a lentiviral vector-based siRNA system. The anxiety-like behaviors were detected by the elevated plus maze (EPM) and hole-board tests in mice. The related proteins involved in cAMP/c
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Chen, Lin, Jinchi Zhou, Zifeng Zhao, et al. "Low Expression of Phosphodiesterase 2 (PDE2A) Promotes the Progression by Regulating Mitochondrial Morphology and ATP Content and Predicts Poor Prognosis in Hepatocellular Carcinoma." Cells 12, no. 1 (2022): 68. http://dx.doi.org/10.3390/cells12010068.

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Phosphodiesterase 2 (PDE2A) modulates the levels of cAMP/cGMP and was recently found to be involved in mitochondria function regulation, closely related to multiple types of tumor progression. This study aimed to estimate the prognostic significance and biological effects of PDE2A on hepatocellular carcinoma (HCC). We comprehensively analyzed the PDE2A mRNA expression in HCC based on The Cancer Genome Atlas (TCGA) database and investigated the effects of PDE2A on the proliferation and metastatic capacity of HCC cells. PDE2A was downregulated in 25 cancer types, including HCC. Lower PDE2A expre
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Barbagallo, Federica, Valentina Rotilio, Maria Rita Assenza, et al. "PDE2A Is Indispensable for Mouse Liver Development and Hematopoiesis." International Journal of Molecular Sciences 21, no. 8 (2020): 2902. http://dx.doi.org/10.3390/ijms21082902.

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Phosphodiesterase 2A (PDE2A) is a cAMP-cGMP hydrolyzing enzyme essential for mouse development and the PDE2A knockout model (PDE2A−/−) is embryonic lethal. Notably, livers of PDE2A−/− embryos at embryonic day 14.5 (E14.5) have extremely reduced size. Morphological, cellular and molecular analyses revealed loss of integrity in the PDE2A−/− liver niche that compromises the hematopoietic function and maturation. Hematopoietic cells isolated from PDE2A−/− livers are instead able to differentiate in in vitro assays, suggesting the absence of blood cell-autonomous defects. Apoptosis was revealed in
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Matthiesen, Karina, and Jacob Nielsen. "Binding of cyclic nucleotides to phosphodiesterase 10A and 11A GAF domains does not stimulate catalytic activity." Biochemical Journal 423, no. 3 (2009): 401–9. http://dx.doi.org/10.1042/bj20090982.

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To date eleven human PDE (3′,5′-cyclic nucleotide phosphodiesterase) families have been identified. Of these, five families contain non-catalytic tandem GAF (cGMP-specific and -stimulated phosphodiesterases, Anabaenaadenylate cyclases and Escherichia coliFhlA) domains, GAFa and GAFb, in the N-terminal part of the enzyme. For PDE2A, PDE5A and PDE6 the GAF domains have been shown to bind cGMP with high affinity. For PDE2A and PDE5A this ligand binding has been shown to stimulate the catalytic activity of the enzyme. PDE10A and PDE11A are the two most recently described PDEs and it has been sugge
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Ritawidya, Ludwig, Briel, Brust, and Scheunemann. "Synthesis and In Vitro Evaluation of 8-Pyridinyl-Substituted Benzo[e]imidazo[2,1-c][1,2,4]triazines as Phosphodiesterase 2A Inhibitors." Molecules 24, no. 15 (2019): 2791. http://dx.doi.org/10.3390/molecules24152791.

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Phosphodiesterase 2A (PDE2A) is highly expressed in distinct areas of the brain, which are known to be related to neuropsychiatric diseases. The development of suitable PDE2A tracers for Positron Emission Tomography (PET) would permit the in vivo imaging of the PDE2A and evaluation of disease-mediated alterations of its expression. A series of novel fluorinated PDE2A inhibitors on the basis of a Benzoimidazotriazine (BIT) scaffold was prepared leading to a prospective inhibitor for further development of a PDE2A PET imaging agent. BIT derivatives (BIT1–9) were obtained by a seven-step synthesi
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Dissertations / Theses on the topic "PDE2A"

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Lobo, Miguel Gonçalo de Oliveira Jones Ferrão. "Role of PDE2A in cAMP/PKA signaling compartmentalization." Thesis, University of Oxford, 2018. http://ora.ox.ac.uk/objects/uuid:e647c600-48e4-4222-85e7-019f91745608.

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<b>Background and Aims :</b> cAMP/PKA signaling is compartmentalized within the cell, with tight spatial and temporal control of signal propagation underpinning specificity of response. The cAMP-degrading enzymes, phosphodiesterases (PDEs), are key regulators of this process. They are situated in specific subcellular domains within which they control local cAMP levels. Several components of the cAMP/PKA cascade have been identified in different mitochondrial compartments, including isoform 2 of PDE2A and multiple A-kinase anchoring proteins, suggesting the presence of multiple cAMP/PKA signali
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Delhaye, Sébastien. "Rôle de la phosphodiestérase 2A dans la physiopathologie du syndrome de l’X fragile." Electronic Thesis or Diss., Université Côte d'Azur, 2021. http://theses.univ-cotedazur.fr/2021COAZ6014.

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Le Syndrome de l’X Fragile (SXF) est la forme la plus fréquente de déficience intellectuelle héréditaire. Le phénotype des patients SXF est complexe. En effet, ils présentent aussi des traits autistiques, de l’hyperactivité et un déficit de l’attention. Au niveau du cerveau, le phénotype majeur est la présence d’épines dendritiques plus nombreuses, plus longues et plus fines. Ces anomalies morphologiques sont associées à des formes altérées de plasticité synaptique chez le modèle murin de SXF, la souris Fmr1-KO. Le SXF est causé par l’expansion (plus de 200 fois) du triplet nucléotidique CGG d
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Law, Robert. "PDE3A signalling in blood platelets." Thesis, University of Hull, 2016. http://hydra.hull.ac.uk/resources/hull:13761.

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Cyclic 3’, 5’ adenosine monophosphate (cAMP) signalling downstream of prostacyclin (PGI₂) is a key inhibitory pathway in blood platelets. This pathway is dynamically regulated by phosphodiesterase 3A (PDE3A), which hydrolyses cAMP into metabolically inactive AMP. Although PDE3A is an established drug target in anti-platelet therapies, the molecular mechanisms that underlie its function in platelets remain unclear. Therefore, the major aim of this study was to further explore PDE3A signalling in human platelets. Using a combination of cell fractionation and immunoblotting we identified two PDE3
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Wilson, Moira Ann. "Characterisation and analysis of PDE4A phosphodiesterase isoforms." Thesis, University of Glasgow, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.307182.

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Johnston, Lee Ann. "The investigation of two novel PDE4A enzymes." Thesis, University of Glasgow, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.400759.

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Livie, Craig. "Determining the role of PDE2 within the mitochondria." Thesis, University of Glasgow, 2015. http://theses.gla.ac.uk/6683/.

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3’,5’-cyclic adenosine monophosphate (cAMP) is a near ubiquitous second messenger responsible for the regulation of a myriad of physiological processes. It is produced by the adenylyl cyclases (AC) and degraded by phosphodiesterases (PDEs). The primary effector of cAMP is protein kinase A (PKA). In order to overcome cross-contamination of separate cAMP-mediated processes within the same cell strict spatiotemporal control is required. Compartmentalisation sculpts cAMP gradients allowing targeted cAMP/PKA action with the cell. This is achieved by the tethering of unique isoforms of AC, PKA and P
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Begg, Fiona A. "Cloning and biochemical characterisation of two novel PDE4A cAMP phosphodiesterases." Thesis, University of Glasgow, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.394925.

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Sekharan, Monica R. "Structural studies of the cGMP-binding GAF domain of PDE5A /." Thesis, Connect to this title online; UW restricted, 2004. http://hdl.handle.net/1773/8502.

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Ercu, Maria [Verfasser]. "Molecular mechanisms underlying PDE3A-caused hypertension with brachydactyly (HTNB) / Maria Ercu." Berlin : Freie Universität Berlin, 2020. http://d-nb.info/1222513862/34.

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Lounas, Amel. "Fonction et localisation de la PDE8A dans les cellules ovariennes porcines et son implication dans la stéroïdogenèse." Master's thesis, Université Laval, 2017. http://hdl.handle.net/20.500.11794/27731.

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Les nucléotides cycliques sont des seconds messagers intracellulaires possédant une grande importance dans la signalisation cellulaire du follicule ovarien. Les niveaux intracellulaires en nucléotides cycliques tel que l'adénosine monophosphate cyclique (AMPc) dépendent de leur synthèse, assurée par l'adenylyl-cyclase (AC) ainsi que leur dégradation par les phosphodiéstérases (PDE). Ces dernières appartiennent à la superfamille des métalophosphohydrolases, elles hydrolysent le groupement phosphate en 3' des nucléotides cycliques pour produire un nucléotide 5' phosphate. Dans les cellules ovari
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Books on the topic "PDE2A"

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Sewell, Granville. Solving Partial Differential Equation Applications with PDE2D. John Wiley & Sons, Inc., 2018. http://dx.doi.org/10.1002/9781119507918.

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Sewell, Granville. Solving Partial Differential Equation Applications with PDE2D. Wiley & Sons, Limited, John, 2018.

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Sewell, Granville. Solving Partial Differential Equation Applications with PDE2D. Wiley & Sons, Incorporated, John, 2018.

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Sewell, Granville. Solving Partial Differential Equation Applications with PDE2D. Wiley, 2018.

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Sewell, Granville. Solving Partial Differential Equation Applications with PDE2D. Wiley & Sons, Incorporated, John, 2018.

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Book chapters on the topic "PDE2A"

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Lobo, Miguel J., and Manuela Zaccolo. "PDE2A." In Encyclopedia of Signaling Molecules. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-67199-4_101603.

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Lobo, Miguel J., and Manuela Zaccolo. "PDE2A." In Encyclopedia of Signaling Molecules. Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4614-6438-9_101603-1.

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Zahid, Sarwar, Kari Branham, Dana Schlegel, et al. "PDE6A." In Retinal Dystrophy Gene Atlas. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-10867-4_54.

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Petersen-Jones, Simon M., Laurence M. Occelli, Martin Biel, and Stylianos Michalakis. "Advancing Gene Therapy for PDE6A Retinitis Pigmentosa." In Retinal Degenerative Diseases. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-27378-1_17.

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Pardasani, R. T., and P. Pardasani. "Effective magnetic moment of [Co(pdea)2(BF4)2]⋅4H2O." In Magnetic Properties of Paramagnetic Compounds. Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-23675-4_3057.

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Meins, M., A. Janecke, C. Marschke, et al. "Mutations in PDE6A, the Gene Encoding the α-Subunit of Rod Photoreceptor Cgmp-Specific Phosphodiesterase, are Rare in Autosomal Recessive Retinitis Pigmentosa." In Degenerative Retinal Diseases. Springer US, 1997. http://dx.doi.org/10.1007/978-1-4615-5933-7_26.

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Pierce, Kenneth E., Paul G. Curran, Christopher P. Zelinka, Andy J. Fischer, Simon M. Petersen-Jones, and Joshua T. Bartoe. "Sildenafil Administration in Dogs Heterozygous for a Functional Null Mutation in Pde6a: Suppressed Rod-Mediated ERG Responses and Apparent Retinal Outer Nuclear Layer Thinning." In Retinal Degenerative Diseases. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-27378-1_61.

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"Introduction to PDE2D." In Solving Partial Differential Equation Applications with PDE2D. John Wiley & Sons, Inc., 2018. http://dx.doi.org/10.1002/9781119507918.ch0.

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Martinez, Sergio E. "PDE2 Structure and Functions." In Cyclic Nucleotide Phosphodiesterases in Health and Disease. CRC Press, 2006. http://dx.doi.org/10.1201/9781420020847-4.

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Martinez, Sergio. "PDE2 Structure and Functions." In Cyclic Nucleotide Phosphodiesterases in Health and Disease. CRC Press, 2006. http://dx.doi.org/10.1201/9781420020847.ch4.

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Conference papers on the topic "PDE2A"

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Doummar, D., C. Dentel, V. Bouilleret, et al. "Biallelic PDE2A Mutations: A New Cause of Intellectual Disability with Paroxysmal Dyskinesia and/or Epilepsy." In Abstracts of the 47th Annual Meeting of the SENP (Société Européenne De Neurologie Pédiatrique). Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1685434.

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Rentsendorj, Otgonchimeg, Franco D'Alessio, Aigul Moldobaeva, Y. Eto, and David B. Pearse. "LPS Induced INOS Expression Is Negatively Regulated By Phosphodiesterase 2A (PDE2A) In Lung And Peritoneal Macrophages." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a5062.

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Rentsendorj, Otgonchimeg, Mahendra Damarla, Michael T. Crow, Ji-Young Choi, Franco D'Alessio, and David B. Pearse. "Phosphodiesterase 2A (PDE2A) Upregulates Inducible Nitric Oxide Synthase (INOS) In Lung Injury From Intra-Tracheal LPS And Large Tidal Volume Ventilation In Mice." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a2107.

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Wu, Xiaoyun, Timothy Lewis, Luc de Waal, et al. "Abstract 2028: PDE3A modulation for cancer therapy." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-2028.

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Li, Gang, and Songqi Huang. "PDEA target weight determination methods based on GRA / AHP." In 2009 IEEE International Conference on Grey Systems and Intelligent Services (GSIS 2009). IEEE, 2009. http://dx.doi.org/10.1109/gsis.2009.5408342.

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Tergau, Katharina, Moritz Gröner, Rebecca Firneburg, et al. "The cGMP-induced PDE2 stimulation as a novel antiarrhythmic strategy." In cGMP: Generators, Effectors and Therapeutic Implications. ScienceOpen, 2024. http://dx.doi.org/10.14293/cgmp.000015.v1.

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Tergau, Katharina, Moritz Gröner, Rebecca Firneburg, et al. "The cGMP-induced PDE2 stimulation as a novel antiarrhythmic strategy." In cGMP: Generators, Effectors and Therapeutic Implications. ScienceOpen, 2024. http://dx.doi.org/10.14293/cgmp.24000073.v1.

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Wu, Xiaoyun, Malvina Papanastasiou, Gavin Schnitzler, et al. "Abstract 1219: Deep mutational scanning of PDE3A identifies residues required for DNMDP response." In Proceedings: AACR Annual Meeting 2021; April 10-15, 2021 and May 17-21, 2021; Philadelphia, PA. American Association for Cancer Research, 2021. http://dx.doi.org/10.1158/1538-7445.am2021-1219.

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Hou, Qingfeng, Xiaobo Zheng, Donghong Guo, et al. "PDEA-Based Amphiphilic Polymer Enables pH-Responsive Emulsions for a Rapid Demulsification." In SPE International Conference on Oilfield Chemistry. Society of Petroleum Engineers, 2019. http://dx.doi.org/10.2118/193640-ms.

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Jiao, Ai-Ying, and Jun-Hai Ma. "An Empirical Research of Bohai Rim Container Terminal Based on PDEA Model." In 2008 4th International Conference on Wireless Communications, Networking and Mobile Computing (WiCOM). IEEE, 2008. http://dx.doi.org/10.1109/wicom.2008.1610.

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