Relevant bibliographies by topics / Pecan – Diseases

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Pecan – Diseases'

Author: Grafiati
Published: 4 June 2021
Last updated: 15 February 2022

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Journal articles on the topic "Pecan – Diseases"

1

Kilby, Michael W. "Characterization of the Pecan Industry in North America with Emphasis in the Southwest." HortScience 32, no. 6 (October 1997): 980c—980. http://dx.doi.org/10.21273/hortsci.32.6.980c.

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The pecan is native to North America and is cultured as a major crop in both the United States and Mexico. In the early part of this century, pecans were thought of as a secondary crop grown in the southern geographic section of the United States. Increased demand for use as a nutritious food has resulted in expansion of the industry into the desert Southwest and California. Adaptive cultivars and irrigation coupled with the lack of diseases and insects has been instrumental in industry development in the West. As the industry has matured during the latter part of the century, pecan culture has improved into a strong crop enterprise business. Orchard management technique and orchard development concepts have been refined, resulting in increased production and awareness. In recent years, production in Mexico has impacted the U.S. price structure and pecan industry economy. The alternate-bearing nature of pecans also impacts prices received by growers. The aging of pecan trees has resulted in serious dilemmas, such as increased tree size and shading. This situation requires techniques such as tree thinning or hedge pruning to enhance annual production and improve nut quality. Various ramifications and parameters of these management practices will be discussed.
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2

Herrera, Esteban A. "Pecan Growing in the Western United States." HortTechnology 5, no. 3 (July 1995): 200–201. http://dx.doi.org/10.21273/horttech.5.3.200.

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Almost 58,000 acres of pecans [Carya illinoinensis (Wangenh.) K. Koch] are planted in the western United States, which includes western Texas and southern areas of New Mexico, Arizona, and California. `Western Schley' is the main cultivar planted, with `Wichita' trees used as pollenizers. All orchards are flood-irrigated and almost no diseases are present. The pecan aphid complex is the predominant insect problem; however, orchard crowding is becoming a problem, and growers are thinning orchards and transplanting trees to new sites.
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3

Cason, K. M. T., and I. E. Yates. "APPRESSORIUM FORMATION IN THE PECAN SCAB FUNGUS." HortScience 27, no. 6 (June 1992): 627f—627. http://dx.doi.org/10.21273/hortsci.27.6.627f.

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Pecan scab, caused by the fungus Cladosporium caryigenum (Ell. et Lant) Gottwald, produces more damage to pecan than all other diseases and insects combined. Early events during infection are critical to disease establishment and to expression of host resistance, but have not been examined previously. Objectives of this research were to determine if there is regulation of appressorial formation and if it is related to resistance. Pre-infectional host-pathogen interactions were studied in vivo (on leaves) and in vitro (on callus, dialysis membrane, and agar) with light and electron microscopy. Leaves, callus tissue, dialysis membranes, and agar were inoculated with scab conidia and were incubated under conditions optimum for germination. Conidia germinate and produce a germ tube on agar and dialysis membrane, but appressoria are not formed. Appressoria form on pecan callus, but germ tubes are long. Long germ tubes are often associated with resistant disease reactions. In vivo, appressoria form readily, but germ tube length varies depending on the location of the spore on the leaf surface. Preliminary evidence indicates that surface topography affects induction of appressorium formation in the scab fungus.
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4

Sparks, Darrell. "Adaptability of Pecan as a Species." HortScience 40, no. 5 (August 2005): 1175–89. http://dx.doi.org/10.21273/hortsci.40.5.1175.

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Pecan [Carya illinoinensis (Wangenh.) C. Koch] is indigenous to the Mississippi River drainage system of the United States. Climate in the native pecan region ranges from humid to semiarid and from mild to harsh winters. Rainfall is bimodal with peaks in March to April and in August to September. Pecan is site specific and is the climax tree species on loamy, well drained, first bottom river land with a relatively high water table. Detrimental effects from pecan's shade intolerance from its more vigorous, sympatric species are minimized as these species are specific to differ sites. Pecan's deep and phreatophytic rooting habit ensures soil moisture during drought periods and facilitates pecan's survival in semiarid regions. Root development in the humus-surface layer ensures nutrient uptake from the most nutrient rich layer of the soil and, when the lower soil profile is saturated, aeration for the roots and water and nutrient uptake. The bimodal rain pattern replenishes soil profile moisture and its timing ensures seed germination, stand establishment, well-developed seed, and minimal drought stress. Natural selection for freeze tolerance and for minimum fruit development time allows survival in areas with harsh winters and short growing seasons. Regulation of seed germination and budbreak by heating and chilling results in pecan being native in cold and warm climates, greatly increasing the native range. The northern limit for pecan is dictated by heat units; the southern limit is restricted by lack of bimodal rains and vivipary. Reproductive stress is caused by the high lipid content of seed, but is counteracted by a long juvenile growth period of the seedling, by a small nut size and low percentage kernel, and by “off” production years of the tree. Nut and percentage kernel decrease as the growing season decreases which contributes to species survival in geographical regions with a short growing season. Selection for small nuts with low percentage kernel is enhanced by predators. Tree reserves are depleted by heavy production during “on” years and are replenished during “off”years. Perpetuation of pecan forests is apparently from sib/half sib seedlings following predator satiation while dissemination into new areas may be mainly by predators. Pecan and its pests successfully co-exist. Major defense against fruit feeders is escape in time, leaf feeders by biological associations and accommodation, and leaf diseases by confrontation. Heterozygous progenies from cross-pollination provide ample genetic diversity for continuous pecan selection to endure pressures imposed throughout a wide climatic range. Ecological adaptions within native pecan forests should be used in developing and maintaining commercial pecan orchards.
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Peña, Jose G. "Pecan Production Trends: A Comparison of Production in the Southeastern and Southwestern United States." HortTechnology 5, no. 3 (July 1995): 202–5. http://dx.doi.org/10.21273/horttech.5.3.202.

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The United States pecan industry experienced dynamic production changes during the last 25 years. Production in Georgia, the leading state, experienced serious problems during the late 1980s and early 1990s as a result of orchard crowding, old orchards, high incidence of diseases, and other problems. During the same 25-year period, plantings and production shifted to the southwestern United States to new production centers in Texas, New Mexico, Arizona, and California under a drier, more-favorable growing environment. Although the southeastern region continues to lead the nation in annual pecan production due to the high number and concentration of orchards with improved varieties, production in the southwestern region eventually may dominate the industry.
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Ozcariz-Fermoselle, María Virginia, Raúl Fraile-Fabero, Tomás Girbés-Juan, Oscar Arce-Cervantes, Juan Andrés Oria de Rueda-Salgueiro, and Anabela Marisa Azul. "Use of lignocellulosic wastes of pecan (Carya illinoinensis) in the cultivation of Ganoderma lucidum." Revista Iberoamericana de Micología 35, no. 2 (April 2018): 103–9. http://dx.doi.org/10.1016/j.riam.2017.09.005.

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Zhang, C. Q., and B. C. Xu. "First Report of Canker on Pecan (Carya cathayensis) Caused by Botryosphaeria dothidea in China." Plant Disease 95, no. 10 (October 2011): 1319. http://dx.doi.org/10.1094/pdis-05-11-0457.

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In the late 1990s, sporadic occurrence of Botryosphaeria canker on Carya cathayensis was recorded in Zhejiang Province, China. From 2005 to 2009, nearly 90% of orchards in Zhejiang and Anhui provinces were seriously affected by this disease. Symptoms were similar to those of canker of C. illinoinensis (2); small, elliptical lesions that developed on the bark at points of infection and then enlarged to form large, sunken, elongated cankers. The cankers coalesced, forming large diffuse areas of blighted tissue, which turned black. Tissue samples from the margin of trunk lesions from 35 different diseased trees from five counties were surface sterilized with 1.5% sodium hypochlorite for 3 min, plated on 2% potato dextrose agar (PDA), and incubated at 25°C in the dark for 1 week. Gray-black mycelia and colorless, aseptate, thin-walled conidia, 17.3 ± 0.8 long and 4.5 ± 0.5 μm wide, were produced. On the basis of these morphological characteristics, the fungus was identified as Botryosphaeria dothidea (Moug. ex Fr.) Ces. & De Not (1). The internal transcribed spacer (ITS) region was amplified with primers ITS1/ITS4 from DNA extracted from mycelium produced on PDA and was recorded as GenBank Accession Nos. HQ731442 and HQ731443. The results of BLAST showed that it had more than 98% similarity to records for B. dothidea. Uninfected twigs and stems of C. cathayensis were wounded with a scalpel and then sprayed with a conidia suspension of 106 conidia per ml in distilled water as inoculum or distilled water only to provide an noninoculated control, wrapped in plastic bags to retain moisture, and incubated for 48 h. For each isolate, five twigs and stems per tree and a total of 10 trees were inoculated. After 2 weeks, 14 of 15 isolates caused lesions on inoculated stems and twigs, whereas no symptoms developed on the noninoculated controls. Cultures isolated from lesions and cultured on PDA exhibited morphological characteristics identical to those of B. dothidea, confirming completion of Koch's postulates. Currently, the distribution of Botryosphaeria canker of C. cathayensis is confined to Zhejiang and Anhui provinces. The identification of the pathogen now allows for appropriate forest management measures. To our knowledge, this is the first report of Botryosphaeria canker of pecan (C. cathayensis) in China. References: (1) S. Denman et al. Stud. Mycol. 45:129, 2000. (2) W. A. Sinclair and H. H. Lyon. Diseases of Trees and Shrubs. 2nd ed. Cornell University Press, Ithaca, NY, 2005.
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Sanderlin, R. S. "Evidence That Xylella fastidiosa Is Associated with Pecan Fungal Leaf Scorch." Plant Disease 82, no. 2 (February 1998): 264. http://dx.doi.org/10.1094/pdis.1998.82.2.264a.

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Several different leaf scorch symptomatologies occur on the foliage of pecan (Carya illinoinensis). The causes of these different scorch symptoms have been associated with environmental stresses, nutritional imbalances, mites, and pathogens. One type of leaf scorch is characterized by necrosis beginning at the tips or margins of the leaflets and progressing toward the midrib and base of the leaflets. The most distinguishing feature of this type of leaf scorch is a dark brown, black, or purplish band of tissue at the interface of the necrotic and green leaflet tissue. This band does not occur on all affected leaflets, but is a consistent feature with this symptomatology. Leaflets with this leaf scorch usually abscise before the entire leaflet becomes necrotic. Affected leaflets will abscise from a compound leaf, while leaflets without symptoms remain on the rachis. When this scorch is severe, the entire leaf including the rachis may abscise. The symptoms are often confined to one area of the tree. Symptoms of this disease appear as early as June but often begin in July. Incidence and severity increase through the remainder of summer and into fall. Over the past 25 years, several different genera of fungi have been reported as being associated with this leaf scorch (2). The phenomenon was named fungal leaf scorch (FLS) because of the association with fungi and the observation that some fungicides reduced the severity of leaf scorch. Genera of fungi implicated in the development of FLS were Pestalotia, Epicoccum, Curvularia, and Fusarium. Recent work has indicated that the disease could be caused by a Phomopsis sp. or Glomerella cingulata or both (1). The symptoms and epidemiology of the FLS are similar to other leaf scorch diseases of hardwood caused by the fastidious xylem-limited bacterium Xylella fastidiosa. A commercial enzyme-linked immunosorbent assay (Agdia, Elkhart, IN) was used to screen for this organism in association with FLS. Assays were conducted by extracting from a composite sample of 9 to 12 sections (approximately 3 cm long) taken from 3 to 4 rachises of each tree tested. Positive reaction for the presence of X. fastidiosa in rachises was recorded from 10 of 10 trees with symptoms of FLS on the Cape Fear cultivar. One of two trees was positive from Cape Fear leaves without symptoms. Symptomless foliage from two less susceptible cultivars, Stuart (two trees) and Sumner (one tree), assayed negative for the bacterium. The association of X. fastidiosa with symptoms of FLS provides evidence that this organism is involved in the etiology of pecan leaf scorch; however, further work is needed to determine the exact etiology. References: (1). A. J. Latham et al. Plant Dis. 79:182, 1995. (2) R. H. Littrell and R. E. Worley. Phytopathology 62:805, 1972.
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Woodward, J. E., D. B. Langston, J. H. Brock, R. C. Kemerait, T. B. Brenneman, and G. H. Beard. "First Demonstration of Koch's Postulates for Lasiodiplodia theobromae Fruit Spot on Eggplant (Solanum melongena)." Plant Disease 89, no. 6 (June 2005): 687. http://dx.doi.org/10.1094/pd-89-0687a.

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During October 2004, diseased eggplant fruit from a commercial farm in Colquitt County, Georgia, developed circular, tan, water-soaked lesions. Gray, septate mycelia quickly covered the fruit. Diseased fruit became shriveled, spongy, and mummified. Disease incidence in the field was approximately 1%. Lasiodiplodia theobromae (Pat.) Griffon & Maubl. (synonym Botryodiplodia theobromae Pat.) (2) was isolated from the margins of lesions and cultured on acidified potato dextrose agar. The fungus produced grayish colonies with aerial hyphae and black ostiolate pycnidia massed into stroma. Mature elliptical conidia (25.8 × 15.6 μm) were brown, had a single septation, and longitudinal striations. Isolates obtained from peanut and pecan were included in the pathogenicity tests. Mature fruit cv. Nightshade were surface disinfested for 30 s in 70% ethanol, followed by 60 s in 0.5% sodium hypochlorite, rinsed twice in sterile distilled water, and allowed to dry. Inoculations were made by placing an agar plug containing L. theobromae mycelial side down on the surface of the fruit or wounding with a sterile toothpick containing mycelium of the fungus. Fruit similarly inoculated with agar plugs or sterile toothpicks served as controls. There were a total of three replicates. Fruit were placed in plastic containers lined with moistened paper towels. Containers were placed in a dew chamber and incubated (28°C, relative humidity >95%) for 3 days, and then evaluated. Symptoms identical to those observed on naturally infected fruit developed on inoculated fruit. Controls remained disease free. L. theobromae was reisolated from all symptomatic tissue, satisfying Koch's postulates. Disease damage on wounded fruit was twice that of nonwounded fruit. However, seven of nine inoculations with agar plugs containing L. theobromae resulted in infection. Lesion lengths from wound inoculations were 9.8, 7.3, and 5.2 cm for isolates from peanut, pecan, and eggplant, respectively. Generally, L. theobromae is considered a facultative wound pathogen or a secondary invader (3). However, this study suggests that direct infection can occur. Although fruit spot has been reported previously on eggplant (1), to our knowledge, this is the first report verifying L. theobromae as the causal agent. References: (1) S. A. Alfieri et al. Index of Plant Diseases in Florida. Fla. Dep. Agric. Consum. Serv. Bull. 11, 1984. (2) H. L. Barnett and B. B. Hunter. Illustrated Guide of Imperfect Fungi. 4th ed. The American Phytopathological Society St. Paul, MN, 1998. (3) P. M. Phipps and D. M. Porter. Plant Dis. 82:1205, 1998.
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10

Oliver, J. E., P. A. Cobine, and L. De La Fuente. "Xylella fastidiosa Isolates from Both subsp. multiplex and fastidiosa Cause Disease on Southern Highbush Blueberry (Vaccinium sp.) Under Greenhouse Conditions." Phytopathology® 105, no. 7 (July 2015): 855–62. http://dx.doi.org/10.1094/phyto-11-14-0322-fi.

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Xylella fastidiosa is a xylem-limited gram-negative plant pathogen that affects numerous crop species, including grape, citrus, peach, pecan, and almond. Recently, X. fastidiosa has also been found to be the cause of bacterial leaf scorch on blueberry in the southeastern United States. Thus far, all X. fastidiosa isolates obtained from infected blueberry have been classified as X. fastidiosa subsp. multiplex; however, X. fastidiosa subsp. fastidiosa isolates are also present in the southeastern United States and commonly cause Pierce’s disease of grapevines. In this study, seven southeastern U.S. isolates of X. fastidiosa, including three X. fastidiosa subsp. fastidiosa isolates from grape, one X. fastidiosa subsp. fastidiosa isolate from elderberry, and three X. fastidiosa subsp. multiplex isolates from blueberry, were used to infect the southern highbush blueberry ‘Rebel’. Following inoculation, all isolates colonized blueberry, and isolates from both X. fastidiosa subsp. multiplex and X. fastidiosa subsp. fastidiosa caused symptoms, including characteristic stem yellowing and leaf scorch symptoms as well as dieback of the stem tips. Two X. fastidiosa subsp. multiplex isolates from blueberry caused more severe symptoms than the other isolates examined, and infection with these two isolates also had a significant impact on host mineral nutrient content in sap and leaves. These findings have potential implications for understanding X. fastidiosa host adaptation and expansion and the development of emerging diseases caused by this bacterium.
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Dissertations / Theses on the topic "Pecan – Diseases"

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Olsen, M., S. Rasmussen, and C. Nischwitz. "Effect of fungicide treatments on incidence of powdery mildew of pecan and on pecan nut quality." College of Agriculture, University of Arizona (Tucson, AZ), 2002. http://hdl.handle.net/10150/226097.

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Powdery mildew of pecan, caused by Microsphaera ulni, was observed on pecan shucks by the latter part of June 2000 in a commercial pecan orchard near Sahuarita, Arizona. Results of 1999 studies indicated that infection does not reduce nut quality. In order to determine effects of fungicide treatments and to substantiate results from 1999, preventive applications of micronized sulfur and azoxystrobin were initiated on June 8, 2000 in selected clusters in both Wichita and Western varieties. Trials were established in plots that had a high incidence of powdery mildew in 1999. Whole nut weights, kernel weights, or color ratings were not significantly different among clusters of nuts that were treated with fungicides and untreated nuts that were infected with powdery mildew. Percent disease incidence was 100% in untreated clusters, 0% in clusters treated with azoxystrobin every two weeks, and 5.3% (Wichita) and 8.8% (Western) in clusters treated with sulfur three times early in the season. Results indicate that disease did not affect nut weight or quality and that early preventive fungicide treatments are effective in controlling infections.
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Olsen, M., S. Rasmussen, C. Nischwitz, and M. Kilby. "Effect of Powdery Mildew on Pecan Nut Weight and Quality." College of Agriculture, University of Arizona (Tucson, AZ), 2000. http://hdl.handle.net/10150/223843.

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Powdery mildew of pecan, caused by Microsphaera ulni, results in discoloration of pecan shucks, but its effects on yield and quality of kernels are not known. In 1999, powdery mildew was observed on pecan shucks by the latter part of June in a commercial pecan orchard near Sahuarita, Arizona. The fungus continued to be active throughout the summer. However, results of a field test comparing diseased and healthy nuts from two varieties of pecans indicate that powdery mildew did not affect nut weight or quality. Whole nut weights, kernel weights, color ratings or percentage of discarded nuts were not significant between paired clusters of nuts that were treated with fungicides and remained disease free and untreated nuts that were infected with powdery mildew. Although shucks may have a high percentage of area covered by powdery mildew, results from this trial indicate that fungicide treatments are not warranted.
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El, Rayess Mohamed Aghar. "Genetic variation in the PECAM-1 gene and its role in atherosclerosis and coronary heart disease." Thesis, University College London (University of London), 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.404401.

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Demuth, Julia Elisabeth. "Einfluss von freien Fettsäuren und Triglyceriden auf die Expression von proinflammatorischen Mediatoren und Adhäsionsmolekülen in Hepatozyten und Kupffer-Zellen (der Ratte)." Doctoral thesis, 2009. http://hdl.handle.net/11858/00-1735-0000-0006-AF6A-1.

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Books on the topic "Pecan – Diseases"

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D, Solomon J. A guide to the insect borers, pruners, and girdlers of pecan and hickory. New Orleans, La: U.S. Dept. of Agriculture, Forest Service, Southern Forest Experiment Station, 1986.

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Estrach, María Teresa. Pecas, manchas y verrugas. México: Editorial Diana, 1993.

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Yizhar, Y. Hadbarat maziḳim, maḥalot ṿa-ʻasavim be-maṭaʻim subṭropiyim uve-peḳan. [Israel]: Miśrad ha-ḥaḳlaʻut, Sherut ha-hadrakhah ṿeha-miḳtsoʻa, ha-Maḥlaḳah la-haganat ha-tsomeaḥ, 1988.

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Wang, Xuewei, and Guilan Zhao. Guan xin bing yong yao yu pei can =: Guanxinbing yongyao yu peican. [Jilin]: Jilin ke xue ji shu chu ban she, 2006.

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Pecan pest management. [Texas?]: Harris & Dean, 1997.

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Rand, Frederick Vernon. Pecan Rosette, Its Histology, Cytology, and Relation to Other Chlorotic Diseases. Franklin Classics Trade Press, 2018.

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Chihuahua (Mexico : State). Dirección General de Desarrollo Rural., ed. Campaña fitosanitaria '93. [Chihuahua, Mexico]: Gobierno del Estado, Dirección General de Desarrollo Rural, 1993.

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Book chapters on the topic "Pecan – Diseases"

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Dutcher, James D., Lenny Wells, Timothy B. Brenneman, and Michael G. Patterson. "Integration of Insect and Mite Management With Disease and Weed Control in Pecan Production." In Integrated Management of Arthropod Pests and Insect Borne Diseases, 133–62. Dordrecht: Springer Netherlands, 2010. http://dx.doi.org/10.1007/978-90-481-8606-8_6.

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Ortiz-Quezada, Ana G., Leonardo Lombardini, and Luis Cisneros-Zevallos. "Antioxidants in Pecan Nut Cultivars [Carya illinoinensis (Wangenh.) K. Koch]." In Nuts and Seeds in Health and Disease Prevention, 881–89. Elsevier, 2011. http://dx.doi.org/10.1016/b978-0-12-375688-6.10104-5.

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Haddad, Ella H. "Health Effects of a Pecan [Carya illinoinensis (Wangenh.) K. Koch] Nut-rich Diet." In Nuts and Seeds in Health and Disease Prevention, 891–98. Elsevier, 2011. http://dx.doi.org/10.1016/b978-0-12-375688-6.10105-7.

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Danielski, Renan, Gabriela Polmann, and Jane Mara Block. "Valorization of Native Nuts from Brazil and Their Coproducts." In Innovation in the Food Sector Through the Valorization of Food and Agro-Food By-Products [Working Title]. IntechOpen, 2020. http://dx.doi.org/10.5772/intechopen.95056.

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The consumption of nuts as part of a healthy diet and active lifestyle has long been associated with chronic disease prevention. Nuts and their coproducts such as oil, cake, plant-based milk substitutes, flour, and shell are rich in lipids, proteins, phenolics, and other bioactive compounds. Nut flour also presents interesting physical properties, such as water or oil holding capacity, foam properties, emulsifying activity, and emulsion stability. These biological and physical properties make these products commercially attractive as organic ingredients in several foods such as spreads, bakery products, and cereal bars. In this chapter, the nutritional and bioactive profiles, as well as the evidenced health-promoting effects of nuts originating from Brazil, will be discussed. The focus will be on commercial nuts such as cashews, pecan, and Brazil nuts, along with some underexplored and relatively unknown indigenous species, such as sapucaia, chichá, monguba, and pracaxi. The knowledge of these Brazilian native nuts and their coproducts is important for stimulating their consumption among the population and their large-scale commercialization.
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Conference papers on the topic "Pecan – Diseases"

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Lishnevsky, Marta, Steven J. Woods, William A. Muller, David W. Riches, and Alan R. Schenkel. "Comparative Analysis Of Bleomycin In Pulmonary Disease Susceptible Pecam Deficient Mice." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a6001.

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Kastan, Michael B. "Abstract IA08: Ataxia-telangiectasia: Broad implications from the study of a rare disease." In Abstracts: AACR Special Conference: Pediatric Cancer at the Crossroads: Translating Discovery into Improved Outcomes; November 3-6, 2013; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.pedcan-ia08.

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Agustina, Wiwik, and Agus E Purnomo. "MENURUNNYA KADAR HEMOGLOBIN PADA PENDERITA END STAGE RENAL DISEASE (ESRD) YANG MENJALANI HEMODIALISIS DI KOTA MALANG." In Regulasi Peran Tenaga Perawat Dalam Mendukung Program indonesia Sehat Dengan Pendekatan Keluarga (PIS - PK). Universitas Muhammadiyah Jember, 2019. http://dx.doi.org/10.32528/psn.v0i0.1733.

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Simko, Stephen J., Marie-Luise Berres, Karen Phaik-Har Lim, Tricia Peters, Jeremy Price, Philip J. Lupo, M. John Hicks, et al. "Abstract B79: Detectable BRAF-V600E mutation in circulating peripheral blood of patients with Langerhans cell histiocytosis correlates with risk organ involvement and residual disease." In Abstracts: AACR Special Conference: Pediatric Cancer at the Crossroads: Translating Discovery into Improved Outcomes; November 3-6, 2013; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.pedcan-b79.

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Tichenor, Lisa W., Mac T. Tichenor, and Neil Moseley. "Abstract IA28: What advocates can do to accelerate your research and enhance patient care: The perspective of memorial foundation focusing on a specific disease." In Abstracts: AACR Special Conference: Pediatric Cancer at the Crossroads: Translating Discovery into Improved Outcomes; November 3-6, 2013; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.pedcan-ia28.

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