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Journal articles on the topic 'Peg1'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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1

Assem, Yasser, Heba A. Mohamed, Rana Said, and Ahmed El-Masry. "Preparation of amphiphilic block copolymers (polyethylene adipate-block-polyethylene glycol) and its application in rotogravure ink formulations." Pigment & Resin Technology 47, no. 5 (2018): 415–23. http://dx.doi.org/10.1108/prt-02-2017-0020.

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Purpose The purpose of this paper is to prepare amphiphilic block copolymers polyethylene adipate-block-polyethylene glycol (PEA-b-PEG)s and study their performance as plasticizers in rotogravure ink formulations. Design/methodology/approach Series of amphiphilic block copolymers (PEA-b-PEG1), (PEA-b-PEG2), (PEA-b-PEG3), (PEA-b-PEG4) and (PEA-b-PEG5) were prepared by the reaction of adipic acid, ethylene glycol and polyethylene glycol of different molecular weights (300, 1,000, 2,000, 10,000 and 20,000 g/mol), respectively. Full characterization of the prepared copolymers was achieved using Fo

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Jung, Hwajin, Suk Kyung Lee, and Eek-hoon Jho. "Mest/Peg1 inhibits Wnt signalling through regulation of LRP6 glycosylation." Biochemical Journal 436, no. 2 (2011): 263–69. http://dx.doi.org/10.1042/bj20101512.

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Mest (mesoderm-specific transcript)/Peg1 (paternally expressed gene 1) is an imprinted gene that plays important roles in embryo development, although its biochemical role has not been determined. Ectopic expression of Mest/Peg1 inhibited Wnt-mediated reporter activity by enhancing the ubiquitination of β-catenin. The maturation and plasma membrane localization of the Wnt co-receptor LRP6 [LDLR (low-density lipoprotein receptor)-related protein 6], which are both necessary for Wnt signalling, were blocked by the expression of Mest/Peg1. Mest/Peg1 inhibited maturation of LRP6 by controlling the

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Obata, Y., T. Kaneko-Ishino, T. Koide, et al. "Disruption of primary imprinting during oocyte growth leads to the modified expression of imprinted genes during embryogenesis." Development 125, no. 8 (1998): 1553–60. http://dx.doi.org/10.1242/dev.125.8.1553.

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Parthenogenetic embryos, which contained one genome from a neonate-derived non-growing oocyte and the other from a fully grown oocyte, developed to day 13.5 of gestation in mice, 3 days longer than previously recorded for parthenogenetic development. To investigate the hypothesis that disruption of primary imprinting during oocyte growth leads to the modified expression of imprinted genes and this parthenogenetic phenotype, we have examined Peg1/Mest, Igf2, Peg3, Snrpn, H19, Igf2r and excess p57KIP2. We show that paternally expressed genes, Peg1/Mest, Peg3 and Snrpn, are expressed in the parth

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Charoenpanich, Jittima, Akio Tani, Naoko Moriwaki, Kazuhide Kimbara, and Fusako Kawai. "Dual regulation of a polyethylene glycol degradative operon by AraC-type and GalR-type regulators in Sphingopyxis macrogoltabida strain 103." Microbiology 152, no. 10 (2006): 3025–34. http://dx.doi.org/10.1099/mic.0.29127-0.

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The genes for polyethylene glycol (PEG) catabolism (pegB, C, D, A and E) in Sphingopyxis macrogoltabida strain 103 were shown to form a PEG-inducible operon. The pegR gene, encoding an AraC-type regulator in the downstream area of the operon, is transcribed in the reverse direction. The transcription start sites of the operon were mapped, and three putative σ 70-type promoter sites were identified in the pegB, pegA and pegR promoters. A promoter activity assay showed that the pegB promoter was induced by PEG and oligomeric ethylene glycols, whereas the pegA and pegR promoters were induced by P

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Marcelo, Gonçalo, Jessica Ariana-Machado, Maria Enea, et al. "Toxicological Evaluation of Luminescent Silica Nanoparticles as New Drug Nanocarriers in Different Cancer Cell Lines." Materials 11, no. 8 (2018): 1310. http://dx.doi.org/10.3390/ma11081310.

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Luminescent mesoporous silica nanoparticles, CdTeQDs@MNs@PEG1, SiQDs@Isoc@MNs and SiQDs@Isoc@MNs@PEG2, were successfully synthetized and characterized by SEM, TEM, XRD, N2 nitrogen isotherms, 1H NMR, IR, absorption, and emission spectroscopy. Cytotoxic evaluation of these nanoparticles was performed in relevant in vitro cell models, such as human hepatoma HepG2, human brain endothelial (hCMEC/D3), and human epithelial colorectal adenocarcinoma (Caco-2) cell lines. None of the tested nanoparticles showed significant cytotoxicity in any of the three performed assays (MTT/NR/ LDH) compared with t

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6

Tahar, Benaniba Mohamed, and Aouachria Kamira. "Thermal and Dynamic Mechanical Analyses of Poly(Lactic Acid)/Poly(Ethylene Glycol) Blends." Academic Perspective Procedia 1, no. 1 (2018): 526–35. http://dx.doi.org/10.33793/acperpro.01.01.102.

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Blends of poly(lactic acid) (PLA) and poly(ethylene glycol) (PEG) with various contents (0, 5, 10, 15, 20 and 30 weight %) and with different molecular weights (M&amp;macr;w = 1000, 4000 and 6000 g/mol), called respectively PEG1, PEG2, and PEG3 were prepared by melt blending. Since glass transition temperature (Tg), T? and loss factor (tan ?) are relevant indicators of polymer chain mobility, plasticization has been studied by dynamic mechanical analysis (DMA) and differential scanning calorimetry (DSC). Low molecular weight (LMW) PEG enable increased miscibility with PLA and more efficien

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Stouder, Christelle, and Ariane Paoloni-Giacobino. "Transgenerational effects of the endocrine disruptor vinclozolin on the methylation pattern of imprinted genes in the mouse sperm." REPRODUCTION 139, no. 2 (2010): 373–79. http://dx.doi.org/10.1530/rep-09-0340.

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Endocrine-disrupting chemicals (EDCs), among which is the antiandrogen vinclozolin (VCZ), have been reported to affect the male reproductive system. In this study, VCZ was administered to pregnant mice at the time of embryo sex determination, and its possible effects on the differentially methylated domains (DMDs) of two paternally (H19 and Gtl2) and three maternally (Peg1, Snrpn, and Peg3) imprinted genes were tested in the male offspring. The CpGs methylation status within the five gene DMDs was analyzed in the sperm, tail, liver, and skeletal muscle DNAs by pyrosequencing. In the sperm of c

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8

Motsch, Christiane, S. Kahl, and Kathrin Nebelung. "Grundlagen der enteralen Ernährung, Sondentechniken, PEG1." Laryngo-Rhino-Otologie 80, no. 8 (2001): 449–57. http://dx.doi.org/10.1055/s-2001-16429.

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9

Denomme, Michelle M., Carlee R. White, Carolina Gillio-Meina, et al. "Compromized Fertility Disrupts Peg1 but Not Snrpn and Peg3 Imprinted Methylation Acquisition in Mouse Oocytes." Biology of Reproduction 87, Suppl_1 (2012): 273. http://dx.doi.org/10.1093/biolreprod/87.s1.273.

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10

Chiron, Stéphane, Alyona Bobkova, Haowen Zhou, and Michael P. Yaffe. "CLASP regulates mitochondrial distribution in Schizosaccharomyces pombe." Journal of Cell Biology 182, no. 1 (2008): 41–49. http://dx.doi.org/10.1083/jcb.200712147.

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Movement of mitochondria in Schizosaccharomyces pombe depends on their association with the dynamic, or plus ends, of microtubules, yet the molecular basis for this interaction is poorly understood. We identified mmd4 in a screen of temperature-sensitive S. pombe strains for aberrant mitochondrial morphology and distribution. Cells with the mmd4 mutation display mitochondrial aggregation near the cell ends at elevated temperatures, a phenotype similar to mitochondrial defects observed in wild-type cells after microtubule depolymerization. However, microtubule morphology and function appear nor

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Horii, T., M. Kimura, S. Morita, Y. Nagao, and I. Hatada. "222 LOSS OF IMPRINTS OF PARTHENOGENETIC EMBRYONIC STEM CELLS IN MURINE CHIMERAS." Reproduction, Fertility and Development 19, no. 1 (2007): 228. http://dx.doi.org/10.1071/rdv19n1ab222.

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Mammalian parthenotes with the 2 maternal genomes cannot develop to term. By contrast, chimeras produced by parthenogenetic and normal embryos can develop to term. However, parthenogenetic cells contribute to restricted cells and body weights of the chimeras are reduced. These effects are due to aberrant expressions of imprinted genes, with complete methylation of the maternally methylated genes and complete loss of the paternally methylated genes. On the other hand, parthenogenetic ES (PGES) chimeras show more normal tissue contribution of donor cells and body weight compared to parthenogenet

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12

Hashimoto, Hiroshi, Masahiro Sugawara, Hiroshi Tsuda, and Shunichi Hirose. "Lipo PEG1 Therapy for Vascular Disturbances in SLE." Japanese Journal of Clinical Immunology 9, no. 3 (1986): 157–64. http://dx.doi.org/10.2177/jsci.9.157.

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13

Reule, M., R. Krause, M. Hemberger, and R. Fundele. "Analysis of Peg1/Mest imprinting in the mouse." Development Genes and Evolution 208, no. 3 (1998): 161–63. http://dx.doi.org/10.1007/s004270050168.

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14

Lee, Soo Young, Young Ho Cho, Ju Yong Youn, et al. "Preparation and Characterization of Porous Membrane for Drug Release." Key Engineering Materials 342-343 (July 2007): 485–88. http://dx.doi.org/10.4028/www.scientific.net/kem.342-343.485.

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To develop osmotic granule with semi-permeable membranes, we prepared the semipermeable membranes with different pore forming agent by using solvent casting method. The membrane was consisted of cellulose acetate, Eudragit® RS, hydroxypropylcellulose (HPC), and triethylcitrate (TEC) in the presence of PEG200, PEG1, 000, or dibutylsebacate(DBS) as a pore forming agent. The produced membranes were white and elastic and exhibited soft property on touch. The release amount of pore forming agent from membrane with different pore forming agent was measured in water dissolution media and the order wa

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15

McMinn, J., M. Wei, Y. Sadovsky, H. M. Thaker, and B. Tycko. "Imprinting of PEG1/MEST Isoform 2 in Human Placenta." Placenta 27, no. 2-3 (2006): 119–26. http://dx.doi.org/10.1016/j.placenta.2004.12.003.

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16

Kosaki, Kenjiro, Rika Kosaki, William J. Craigen, and Nobutake Matsuo. "Isoform-Specific Imprinting of the Human PEG1/MEST Gene." American Journal of Human Genetics 66, no. 1 (2000): 309–12. http://dx.doi.org/10.1086/302712.

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17

Kobayashi, S., T. Kohda, N. Miyoshi, et al. "Human PEG1/MEST, an Imprinted Gene on Chromosome 7." Human Molecular Genetics 6, no. 5 (1997): 781–86. http://dx.doi.org/10.1093/hmg/6.5.781.

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18

Lefebvre, L., S. Viville, S. C. Barton, F. Ishino, and M. A. Surani. "Genomic Structure and Parent-of-Origin-Specific Methylation of Peg1." Human Molecular Genetics 6, no. 11 (1997): 1907–15. http://dx.doi.org/10.1093/hmg/6.11.1907.

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19

Piorecka, Kinga, Jan Kurjata, and Wlodzimierz A. Stanczyk. "Novel Polyhedral Silsesquioxanes [POSS(OH)32] as Anthracycline Nanocarriers—Potential Anticancer Prodrugs." Molecules 26, no. 1 (2020): 47. http://dx.doi.org/10.3390/molecules26010047.

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Anthracyclines belong to the anticancer drugs that are widely used in chemotherapy. However, due to their systemic toxicity they also exert dangerous side effects associated mainly with cardiovascular risks. The pathway that is currently often developed is their chemical and physical modification via formation of conjugated or complexed prodrug systems with a variety of nanocarriers that can selectively release the active species in cancer cells. In this study, six new nanoconjugates were synthesized with the use of polyhedral oligosilsesquioxanes [POSS(OH)32] as nanocarriers of the anticancer

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20

Yonekura, Shinichi, Masaki Ohata, Megumi Tsuchiya, Hitomi Tokita, Moeko Mizusawa, and Yukako Tokutake. "Peg1/Mest , an imprinted gene, is involved in mammary gland maturation." Journal of Cellular Physiology 234, no. 2 (2018): 1080–87. http://dx.doi.org/10.1002/jcp.27219.

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21

Jung, Hwajin, Suk Kyung Lee, and Eek-hoon Jho. "P112. MEST/Peg1 inhibits Wnt signaling via regulating maturation of LRP6." Differentiation 80 (November 2010): S54. http://dx.doi.org/10.1016/j.diff.2010.09.118.

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22

Seo, Bong Jong, Hyun Sik Jang, Hyuk Song, et al. "Generation of Mouse Parthenogenetic Epiblast Stem Cells and Their Imprinting Patterns." International Journal of Molecular Sciences 20, no. 21 (2019): 5428. http://dx.doi.org/10.3390/ijms20215428.

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Pluripotent stem cells can be established from parthenogenetic embryos, which only possess maternal alleles with maternal-specific imprinting patterns. Previously, we and others showed that parthenogenetic embryonic stem cells (pESCs) and parthenogenetic induced pluripotent stem cells (piPSCs) progressively lose the bimaternal imprinting patterns. As ESCs and iPSCs are naïve pluripotent stem cells, parthenogenetic primed pluripotent stem cells have not yet been established, and thus, their imprinting patterns have not been studied. Here, we first established parthenogenetic epiblast stem cells

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23

Nakamura, Akie, Koji Muroya, Hiroko Ogata-Kawata, et al. "A case of paternal uniparental isodisomy for chromosome 7 associated with overgrowth." Journal of Medical Genetics 55, no. 8 (2018): 567–70. http://dx.doi.org/10.1136/jmedgenet-2017-104986.

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BackgroundPaternal uniparental disomy for chromosome 7 (upd(7)pat) is extremely rare, and only four cases have been previously reported. As these cases were accompanied by autosomal-recessive disorders which are likely to be involved in growth restriction, the relevance of upd(7)pat to the overgrowth phenotype remains unclear. Here we describe one case of upd(7)pat with no additional genetic diseases, which may answer the question.MethodsA 5-year-old Japanese boy presented with a tall stature of unknown causes. To detect the genetic cause of the tall stature, we performed Sanger sequencing, ta

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Stouder, Christelle, and Ariane Paoloni-Giacobino. "Specific transgenerational imprinting effects of the endocrine disruptor methoxychlor on male gametes." REPRODUCTION 141, no. 2 (2011): 207–16. http://dx.doi.org/10.1530/rep-10-0400.

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Endocrine-disrupting chemicals (EDCs), among which methoxychlor (MXC), have been reported to affect the male reproductive system. This study evaluates the possible deleterious effects of MXC on imprinted genes. After administration of the chemical in adult male mice or in pregnant mice we analyzed by pyrosequencing possible methylation defects in two paternally imprinted (H19 and Meg3 (Gtl2)) and three maternally imprinted (Mest (Peg1), Snrpn, and Peg3) genes in the sperm and in the tail, liver, and skeletal muscle DNAs of the adult male mice and of the male offspring. MXC treatment of adult m

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25

Riesewijk, Anne M., Nadya Blagitko, Albert A. Schinzel, et al. "Evidence against a major role of PEG1/MEST in Silver–Russell syndrome." European Journal of Human Genetics 6, no. 2 (1998): 114–20. http://dx.doi.org/10.1038/sj.ejhg.5200164.

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Kaneko-Ishino, Tomoko, Yoshimi Kuroiwa, Naoki Miyoshi, et al. "Peg1/Mest imprinted gene on chromosome 6 identified by cDNA subtraction hybridization." Nature Genetics 11, no. 1 (1995): 52–59. http://dx.doi.org/10.1038/ng0995-52.

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27

Vidal, A. C., N. M. Henry, S. K. Murphy, et al. "PEG1/MEST and IGF2 DNA methylation in CIN and in cervical cancer." Clinical and Translational Oncology 16, no. 3 (2013): 266–72. http://dx.doi.org/10.1007/s12094-013-1067-4.

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Izraelit, A., V. Ten, G. Krishnamurthy, and V. Ratner. "Neonatal Cyanosis: Diagnostic and Management Challenges." ISRN Pediatrics 2011 (December 29, 2011): 1–4. http://dx.doi.org/10.5402/2011/175931.

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Neonatal central cyanosis is always a sign of serious pathological processes and may involve diverse organs and impose a significant diagnostic and therapeutic challenge. Here, we report an unusual presentation of Ebstein's anomaly, a rare congenital heart malformation, as the cause of central cyanosis in a one-week-old full-term infant. Initiation of PEG1 therapy in neonates with Ebstein's anomaly always needs a very careful consideration because of a high risk for the development of a “circular shunt” leading to severe deterioration of multiple organs perfusion.

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Russo, S., M. F. Bedeschi, F. Cogliati, et al. "Maternal chromosome 7 hetero/isodisomy in Silver-Russell syndrome and PEG1 biallelic expression." Clinical Dysmorphology 9, no. 3 (2000): 157–62. http://dx.doi.org/10.1097/00019605-200009030-00001.

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Kataoka, H., S. Nakano, M. Oshimura, Y. Kunimoto, and H. Kitano. "S081 Loss of Imprinting of PEG1/MEST, IGF2 in Head and Neck Cancer." Archives of Otolaryngology–Head & Neck Surgery 132, no. 8 (2006): 857. http://dx.doi.org/10.1001/archotol.132.8.857-c.

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31

Takahashi, Mayumi, Yasutomi Kamei, and Osamu Ezaki. "Mest/Peg1 imprinted gene enlarges adipocytes and is a marker of adipocyte size." American Journal of Physiology-Endocrinology and Metabolism 288, no. 1 (2005): E117—E124. http://dx.doi.org/10.1152/ajpendo.00244.2004.

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Obesity is a common and serious metabolic disorder in the developed world that is occasionally accompanied by type II diabetes, atherosclerosis, hypertension, and hyperlipidemia. We have found that mesoderm-specific transcript (Mest)/paternally expressed gene 1 (Peg1) gene expression was markedly enhanced in white adipose tissue of mice with diet-induced and genetically caused obesity/diabetes but not with streptozotocin-induced diabetes, which does not cause obesity. Administration of pioglitazone, a drug for type II diabetes and activator of peroxisome proliferator-activated receptor (PPAR)γ

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Paul, R. N., and G. H. Egley. "Cuticular penetration of Sesbania exaltata STEM tissue by penetration pegs from appressoria of the plant pathogenic fungus: Colletotrichum truncatum." Proceedings, annual meeting, Electron Microscopy Society of America 50, no. 1 (1992): 838–39. http://dx.doi.org/10.1017/s0424820100124598.

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Colletotrichum truncatum is a potentially useful pathogen for the biocontrol of the weed Sesbania exaltata [(Raf.) Cory]. Information on the infection process is important in optimizing the use of this pathogen as a mycoherbicide. Successful infection of a host species depends upon the fungus’ ability to penetrate the plant’s defenses. Some fungi bypass the host plant cuticle by invading through stomates or other openings on the epithelial surface. Colletotrichum is capable of breaching the cuticle by means of a penetration peg1 produced from an appressorium. We investigated this process by ex

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Pedersen, I. S. "Promoter switch: a novel mechanism causing biallelic PEG1/MEST expression in invasive breast cancer." Human Molecular Genetics 11, no. 12 (2002): 1449–53. http://dx.doi.org/10.1093/hmg/11.12.1449.

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34

LI, Li, Tong JIA, Tong-Shan WU, and Shou-Quan ZHANG. "Studies on SNP and genomic imprinting of the PEG1 gene in swine." Hereditas (Beijing) 33, no. 7 (2011): 738–42. http://dx.doi.org/10.3724/sp.j.1005.2011.00738.

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35

Nye, Monica D., Cathrine Hoyo, and Susan K. Murphy. "In vitro lead exposure changes DNA methylation and expression of IGF2 and PEG1/MEST." Toxicology in Vitro 29, no. 3 (2015): 544–50. http://dx.doi.org/10.1016/j.tiv.2015.01.002.

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Suzuki, Shunsuke, Marilyn B. Renfree, Andrew J. Pask, et al. "Genomic imprinting of IGF2, p57KIP2 and PEG1/MEST in a marsupial, the tammar wallaby." Mechanisms of Development 122, no. 2 (2005): 213–22. http://dx.doi.org/10.1016/j.mod.2004.10.003.

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37

Cucerea, Manuela, Marta Simon, Elena Moldovan, Marcela Ungureanu, Raluca Marian, and Laura Suciu. "Congenital Heart Disease Requiring Maintenance of Ductus Arteriosus in Critically Ill Newborns Admitted at A Tertiary Neonatal Intensive Care Unit." Journal of Critical Care Medicine 2, no. 4 (2016): 185–91. http://dx.doi.org/10.1515/jccm-2016-0031.

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AbstractIntroduction: Congenital heart diseases (CHD) have been reported to be responsible for 30 to 50% of infant mortality caused by congenital disabilities. In critical cases, survival of newborns with CHD depends on the patency of the ductus arteriosus (PDA), for maintaining the systemic or pulmonary circulation. The aim of the study was to assess the efficacy and side effects of PGE (prostaglandin E) administration in newborns with critical congenital heart disease requiring maintenance of the ductus arteriosus.Material and method: All clinical and paraclinical data of 66 infants admitted

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Lee, Young Jae, Chang Won Park, Yoonsoo Hahn, et al. "Mit1 /Lb9 and Copg2 , new members of mouse imprinted genes closely linked to Peg1 /Mest." FEBS Letters 472, no. 2-3 (2000): 230–34. http://dx.doi.org/10.1016/s0014-5793(00)01461-7.

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Maaß, Christian, Jose Ricardo Avelar Rivas, Ali Asgar Attarwala, et al. "Physiologically based pharmacokinetic modeling of 18F-SiFAlin-Asp3-PEG1-TATE in AR42J tumor bearing mice." Nuclear Medicine and Biology 43, no. 4 (2016): 243–46. http://dx.doi.org/10.1016/j.nucmedbio.2016.01.001.

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Kawakami, Takashige, Hiroyuki Sugimoto, Rie Furuichi, et al. "Cadmium reduces adipocyte size and expression levels of adiponectin and Peg1/Mest in adipose tissue." Toxicology 267, no. 1-3 (2010): 20–26. http://dx.doi.org/10.1016/j.tox.2009.07.022.

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Hayashida, Soichiro, Kentaro Yamasaki, Yuki Asada, Eiichi Soeda, Norio Niikawa, and Tatsuya Kishino. "Construction of a Physical and Transcript Map Flanking the Imprinted MEST/PEG1 Region at 7q32." Genomics 66, no. 2 (2000): 221–25. http://dx.doi.org/10.1006/geno.2000.6206.

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Schöherr, Nadine, Susanne Jäger, Michael B. Ranke, Hartmut A. Wollmann, Gerhard Binder, and Thomas Eggermann. "No evidence for isolated imprinting mutations in the PEG1/MEST locus in Silver–Russell patients." European Journal of Medical Genetics 51, no. 4 (2008): 322–24. http://dx.doi.org/10.1016/j.ejmg.2008.05.001.

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Peng, Wei, Ying Chen, Xin Luo, et al. "DNA methylation-associated repression of MEST/PEG1 expression contributes to the invasion of extravillous trophoblast cells." Placenta 46 (October 2016): 92–101. http://dx.doi.org/10.1016/j.placenta.2016.08.093.

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Kerjean, A. "Establishment of the paternal methylation imprint of the human H19 and MEST/PEG1 genes during spermatogenesis." Human Molecular Genetics 9, no. 14 (2000): 2183–87. http://dx.doi.org/10.1093/hmg/9.14.2183.

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45

Hahn, Yoonsoo, Seung Kyoung Yang, and Jae Hoon Chung. "Structure and expression of the zebrafish mest gene, an ortholog of mammalian imprinted gene PEG1/MEST." Biochimica et Biophysica Acta (BBA) - Gene Structure and Expression 1731, no. 2 (2005): 125–32. http://dx.doi.org/10.1016/j.bbaexp.2005.09.004.

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Chen, S. L., H. Y. Zheng, X. Y. Shi, Y. Q. Wu, and L. L. Wang. "DNA methylation status of imprinted H19, PEG1, KvDMR1 genes in babies conceived by assisted reproductive technology." Fertility and Sterility 96, no. 3 (2011): S177—S178. http://dx.doi.org/10.1016/j.fertnstert.2011.07.692.

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47

Kamei, Yasutomi, Takayoshi Suganami, Takashi Kohda, et al. "Peg1/Mestin obese adipose tissue is expressed from the paternal allele in an isoform-specific manner." FEBS Letters 581, no. 1 (2006): 91–96. http://dx.doi.org/10.1016/j.febslet.2006.12.002.

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48

Blayney, Douglas W., Stephan Ogenstad, Yuankai Shi, et al. "Plinabulin (Plin) combined with half-dose pegfilgrastim (Peg) compared with full-dose peg alone for chemotherapy- induced-neutropenia (CIN): Neutrophil, bone pain, and immunosuppressive effects." Journal of Clinical Oncology 37, no. 15_suppl (2019): e12017-e12017. http://dx.doi.org/10.1200/jco.2019.37.15_suppl.e12017.

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e12017 Background: Peg is used for CIN prevention. Plin, a novel, non-G-CSF agent is in Phase (Ph) 3 trials for CIN prevention and as an anticancer (NSCLC) agent. In contrast to Peg, Plin does not cause bone pain and maintains absolute neutrophil counts (ANC) within the normal range. Furthermore, Peg, but not Plin, increases Neutrophil-to-Lymphocyte Ratio (NLR) and Lymphocyte-to-Monocyte Ratio (LMR) to immune suppressive values; i.e. NLR>5 and LMR<3.2 (Blayney, ASCO 2018, ESMO 2018, ASCO-SITC 2018). We tested the effects on CIN, bone pain and immune-suppressive profile (i.e. NLR> 5 an

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Miozzo, M., F. R. Grati, G. Bulfamante, et al. "Post-Zygotic Origin of Complete Maternal Chromosome 7 Isodisomy and Consequent Loss of Placental PEG1/MEST Expression." Placenta 22, no. 10 (2001): 813–21. http://dx.doi.org/10.1053/plac.2001.0728.

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Li, N., T. Xiang, A. Ledgard, J. Peterson, D. Wells, and R. Lee. "226 GREATER DYSREGULATION OF GENE EXPRESSION IN PRE-IMPLANTATION CLONED OVINE COMPARED WITH CLONED BOVINE CONCEPTUSES." Reproduction, Fertility and Development 17, no. 2 (2005): 263. http://dx.doi.org/10.1071/rdv17n2ab226.

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Abstract:

In our experience, the cloning of sheep by somatic cell nuclear transfer has been less successful than with cattle (5 v. 10% live births from embryo transfer). Here, we compare data collected over many years on the pre-implantation development of nuclear transfer (NT) ovine and bovine embryos with contemporary in vitro produced (IVP) embryos at the elongation (bovine Day 16–18 v. ovine Day 14) and allantois formation (bovine Day 26/27 v. ovine Day 21) stages. Sheep NT conceptuses were generated from three fibroblast types (skin, kidney, and lung) derived from a Day 100 female fetus. Bovine NT

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