Relevant bibliographies by topics / PEG10

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  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'PEG10'

Author: Grafiati
Published: 2 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "PEG10"

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Yalçın, Arif Hakan, and Ercan Şimşir. "Production of Wood Pyrolysis Oil for Use as an Alternative Fuel in the Automotive Sector and Improvement of Its Physicochemical Properties." Journal of Materials and Mechatronics: A 6, no. 1 (2025): 262–73. https://doi.org/10.55546/jmm.1581683.

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Biomass resources have the potential to replace petroleum-based fuels. Biomass can be converted into pyrolysis oil by pyrolysis method and this oil is of interest as an alternative to fossil fuels used in many areas such as automotive sector. However, pyrolysis oil is difficult to use directly in diesel engines due to its low energy density, high viscosity and water content. The easiest solution is to create mixtures with high cetane content. In this study, polyethylene glycol 400 (PEG), Wood Pyrolysis oil (WPO), n-butanol (B) and 2-ethylhexyl nitrate (2-EHN) (PEG0/PY10/B85/2-EHN5) were obtain
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Campodonico, Will, Harihar M. Mohan, Phuoc T. Huynh, et al. "The gag-like gene RTL8 antagonizes PEG10-mediated virus like particles." PLOS ONE 19, no. 12 (2024): e0310946. https://doi.org/10.1371/journal.pone.0310946.

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PEG10 is a retroelement-derived Mart-family gene that is necessary for placentation and has been implicated in neurological disease. PEG10 resembles both retrotransposon and retroviral proteins and forms virus-like particles (VLPs) that can be purified using iodixanol ultracentrifugation. It is hypothesized that formation of VLPs is crucial to the biological roles of PEG10 in reproduction and neurological health. Here, we describe the regulation of PEG10 VLP formation and release in human cells with a role for the related Mart gene RTL8. RTL8 resembles a truncated form of PEG10 that shares hom
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Kim, Soojin, Daksh Thaper, Samir Bidnur, et al. "PEG10 is associated with treatment-induced neuroendocrine prostate cancer." Journal of Molecular Endocrinology 63, no. 1 (2019): 39–49. http://dx.doi.org/10.1530/jme-18-0226.

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Neuroendocrine (NE) differentiation of advanced prostate adenocarcinoma following androgen receptor (AR) axis-directed therapy is becoming increasingly recognized. Several models of this transdifferentiation provide insight into its molecular pathogenesis and have highlighted the placental gene PEG10 for further study. Using our unique model of enzalutamide resistance (ENZR) and NE differentiation, we studied PEG10/AR interplay in enzalutamide treatment-resistant cell lines 42DENZR and 42FENZR compared to LNCaP and castration-resistant 16DCRPC cells. ENZR cell lines with positive terminal NE m
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Xiong, Jie, Jian Qin, Yingcheng Zheng, Xiaofan Peng, Yixing Luo, and Xiangyu Meng. "PEG10 promotes the migration of human Burkitt’s lymphoma cells by up-regulating the expression of matrix metalloproteinase-2 and -9." Clinical & Investigative Medicine 35, no. 3 (2012): 117. http://dx.doi.org/10.25011/cim.v35i3.16587.

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Purpose: Paternally expressed gene 10 (PEG10) is important for apoptosis resistance in cancer cells; however, the effect of PEG10 on tumor cell migration remains poorly understood. In this study, we investigated the effects of PEG10 on proliferation, apoptosis, adhesion and migration in the Burkitt’s lymphoma cell line, Raji. 
 
 Methods: Apoptosis was induced by 5-fluorouracil (5-FU) in pcDNA3.0/PEG10 transiently transfected HEK293T cells and PEG10-suppressed Raji cells. siRNAPEG10 was used to inhibit PEG10 expression. Fluorescence-activated cell sorting (FACS) were performed to ana
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Kaneko-Ishino, Tomoko, and Fumitoshi Ishino. "Retrovirus-Derived RTL/SIRE Genes: Their Diverse Roles in the Current Eutherian Developmental System and Contribution to Eutherian Evolution." Biomolecules 13, no. 10 (2023): 1436. http://dx.doi.org/10.3390/biom13101436.

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Eutherians have 11 retrotransposon Gag-like (RTL)/sushi-ichi retrotransposon homolog (SIRH) genes presumably derived from a certain retrovirus. Accumulating evidence indicates that the RTL/SIRH genes play a variety of roles in the current mammalian developmental system, such as in the placenta, brain, and innate immune system, in a eutherian-specific manner. It has been shown that the functional role of Paternally Expressed 10 (PEG10) in placental formation is unique to the therian mammals, as are the eutherian-specific roles of PEG10 and PEG11/RTL1 in maintaining the fetal capillary network a
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Yin, Dong, Xinyi Yao, Jingyuan Zhang, et al. "Abstract 5399: Placental gene PEG10 promotes onco-fetal metabolic reprogramming in hepatocellular carcinoma." Cancer Research 85, no. 8_Supplement_1 (2025): 5399. https://doi.org/10.1158/1538-7445.am2025-5399.

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Abstract Tumorigenesis is a complex biological process, accompanied by cellular dedifferentiation and metabolic reprogramming, which similarities to the metabolic characteristics of embryonic development stages. In our research, we focused particularly on the RNA-binding protein PEG10, which is highly expressed in the placenta and found to be similarly overexpressed in liver cancer cells, playing a key role in the process of aerobic glycolysis in tumor cells. This discovery suggests that there may be a phenomenon of fetal-like metabolic reprogramming in hepatocellular carcinoma (HCC), which is
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Kainz, Birgit, Martin Bilban, Medhat Shehata, et al. "The Paternally Expressed Gene 10 (PEG10) on Chromosome 7q21 Is Overexpressed and Imprinted in High-Risk B-CLL." Blood 106, no. 11 (2005): 1221. http://dx.doi.org/10.1182/blood.v106.11.1221.1221.

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Abstract Introduction: To establish surrogate markers for IgVH mutation status in B-CLL, we performed microarray analysis with mutated and unmutated patient samples. Among the most differentially expressed genes was PEG10, a maternally imprinted gene located on chromosome 7q21. PEG10 is known to be expressed during placental development and in hepatocellular carcinoma. Here we have investigated the association of PEG10 with B-CLL subtypes and risk factors as well as its transcriptional regulation. In addition, we studied its expression in other hematologic malignancies. Methods and Results: In
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Liu, Fengjie, Yumei Gao, Bufang Xu, et al. "PEG10 amplification at 7q21.3 potentiates large-cell transformation in cutaneous T-cell lymphoma." Blood 139, no. 4 (2022): 554–71. http://dx.doi.org/10.1182/blood.2021012091.

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Abstract Mycosis fungoides (MF), the most common form of cutaneous T-cell lymphoma, undergo large-cell transformation (LCT) in the late stage, manifesting aggressive behavior, resistance to treatments, and poor prognosis, but the mechanisms involved remain unclear. To identify the molecular driver of LCT, we collected tumor samples from 133 MF patients and performed whole-transcriptome sequencing on 49 advanced-stage MF patients, followed by integrated copy number inference and genomic hybridization. Tumors with LCT showed unique transcriptional programs and enriched expressions of genes at ch
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Byrne, Raphael M., Rebecca Ruhl, Christian Lanciault, Sudarshan Anand, Abhinav Nellore, and Vassiliki Liana Tsikitis. "Age-related differences in gene expression in colorectal cancer (CRC)." Journal of Clinical Oncology 36, no. 4_suppl (2018): 654. http://dx.doi.org/10.1200/jco.2018.36.4_suppl.654.

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654 Background: Colorectal cancer (CRC) in young patients is increasing in incidence and is associated with worse outcomes than CRC in older patients. While distinct molecular subtypes of CRC have been recently characterized, it is unclear whether there are molecular differences between the tumors of young and old patients. We sought to identify differences in gene expression of CRC between these two groups. Our discovery analysis identified a gene signature of several differentially expressed RNAs, from which we validated PEG10. The PEG10 gene on chromosome 7q21.3 has been implicated in liver
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Segel, Michael, Blake Lash, Jingwei Song, et al. "Mammalian retrovirus-like protein PEG10 packages its own mRNA and can be pseudotyped for mRNA delivery." Science 373, no. 6557 (2021): 882–89. http://dx.doi.org/10.1126/science.abg6155.

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Eukaryotic genomes contain domesticated genes from integrating viruses and mobile genetic elements. Among these are homologs of the capsid protein (known as Gag) of long terminal repeat (LTR) retrotransposons and retroviruses. We identified several mammalian Gag homologs that form virus-like particles and one LTR retrotransposon homolog, PEG10, that preferentially binds and facilitates vesicular secretion of its own messenger RNA (mRNA). We showed that the mRNA cargo of PEG10 can be reprogrammed by flanking genes of interest with Peg10’s untranslated regions. Taking advantage of this reprogram
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Dissertations / Theses on the topic "PEG10"

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Kim, Soojin. "Elucidation of AR impact on the paternally expressed Gene 10 (PEG10) in Enzalutamide-resistant prostate cancer." Thesis, University of British Columbia, 2016. http://hdl.handle.net/2429/59301.

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The full abstract for this thesis is available in the body of the thesis, and will be available when the embargo expires.<br>Medicine, Faculty of<br>Experimental Medicine, Division of<br>Medicine, Department of<br>Graduate
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Brown, Laura-Anne. "Functional characterisation of PEX10 in Arabidopsis thaliana." Thesis, University of Leeds, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.445377.

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Haupenthal, Katharina [Verfasser]. "Über PEX10 als Regulator der de novo Peroxisomenbiogenese / Katharina Haupenthal." Lübeck : Zentrale Hochschulbibliothek Lübeck, 2019. http://d-nb.info/1191804119/34.

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Anderson, Neil Fraser. "The role of the imprinted gene Peg1 in mammalian development." Thesis, University of Cambridge, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.596095.

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The paternally expressed gene 1 (Peg1), also known as mesodermally expressed specific transcript (Mest), is one of approximately 60 genes so far discovered, the expression of which is dependent on the parental origin of each gene copy. Human Peg1 and its highly conserved mouse counterpart are expressed primarily from the paternal genome. Initial investigations into the distribution of <i>Peg1</i> expression have concluded that its mRNA is found exclusively in tissues of mesodermal origin and that expression levels decline substantially after birth. A lack of <i>Peg1</i> expression in mice has
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Prestele, Jakob. "Funktionelle Charakterisierung der pflanzlichen Zink-RING-Finger-Peroxine PEX10, PEX2 und PEX12 in Arabidopsis thaliana." kostenfrei, 2009. http://d-nb.info/998641693/34.

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Channathodiyil, Prasanna. "Characterisation of genetic and epigenetic aberrations in paediatric high grade glioma." Thesis, University of Wolverhampton, 2016. http://hdl.handle.net/2436/617784.

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Paediatric high grade glioma (HGG), including diffuse intrinsic pontine glioma (DIPG) are highly aggressive tumours with no effective cures. Lack of understanding of the molecular biology of these tumours, in part due to lack of well-characterised pre-clinical models, is a great challenge in the development of novel therapies. Analysis of paired cell culture/biopsy samples in this study revealed that paediatric HGG short-term cell cultures retain many of the tumour characteristics in vivo. Using a genome-wide approach, copy number, gene and miRNA expression, and methylation changes were charac
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Elves, Rachel Leigh. "Consequences of mitotic loss of heterozygosity on genomic imprinting in mouse embryonic stem cells." Thesis, University of British Columbia, 2008. http://hdl.handle.net/2429/1564.

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Epigenetic differences between maternally inherited and paternally inherited chromosomes, such as CpG methylation, render the maternal and paternal genome functionally inequivalent, a phenomenon called genomic imprinting. This functional inequivalence is exemplified with imprinted genes, whose expression is parent-of-origin specific. The dosage of imprinted gene expression is disrupted in cells with uniparental disomy (UPD), which is an unequal parental contribution to the genome. I have derived mouse embryonic stem (ES) cell sub-lines with maternal UPD (mUPD) for mouse chromosome 6 (MMU6) to
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Chen, Hsiang-Chi, and 陳湘琦. "The Expression and Biological Functions of PEG10 in Human Esophageal Carcinoma." Thesis, 2005. http://ndltd.ncl.edu.tw/handle/71781111126132857650.

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碩士<br>國立陽明大學<br>公共衛生研究所<br>93<br>PEG10 is a paternally expressed gene, which was reported to be overexpressed in hepatoma. By RT-PCR, we identified that PEG10 was highly expressed in the human esophageal carcinoma tissues, but was very low or even undetectable in the adjacent normal tissues. By immunohistochemical staining, strong nuclear and cytoplasmic staining of PEG10 was detected in esophageal carcinoma tissues. To test the effects of PEG10 gene on growth of esophageal carcinoma cells, we infected esophageal carcinoma cell lines with a replication-deficient adenovirus expressing PEG10 (Ad
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Chen, Gui-May, and 陳貴美. "Developmental study of Peg10, a paternally expressed gene, in the nucleus accumbens of mouse forebrain." Thesis, 2016. http://ndltd.ncl.edu.tw/handle/68724383549593264558.

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碩士<br>國立陽明大學<br>神經科學研究所<br>105<br>Peg10 (Paternally Expressed Gene 10) is expressed in early stages of mouse embryos. Peg10 is expressed in the mouse brain, but its detailed expression pattern and function in developing mouse brain are yet unkown. The main focus of my thesis was to study the expression pattern and function of Peg10 in developing mouse forebrain. In the first part of my thesis study, we investigated the expression pattern of Peg10 in developing mouse forebrain. Western blotting showed that Peg10-RF1 isoform, but not Peg10-RF1/2 isoform, was expressed in E13.5 developing mouse f
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Prestele, Jakob [Verfasser]. "Funktionelle Charakterisierung der pflanzlichen Zink-RING-Finger-Peroxine PEX10, PEX2 und PEX12 in Arabidopsis thaliana / Jakob Prestele." 2009. http://d-nb.info/998641693/34.

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Books on the topic "PEG10"

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Dobbins, James, Howard Shatz, and Ali Wyne. Russia Is a Rogue, Not a Peer; China Is a Peer, Not a Rogue: Different Challenges, Different Responses. RAND Corporation, 2019. http://dx.doi.org/10.7249/pe310.

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Book chapters on the topic "PEG10"

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Shimozawa, Nobuyuki, Tomoko Nagase, Yasuhiko Takemoto, Yasuyuki Suzuki, and Naomi Kondo. "Genetic Heterogeneity in Japanese Patients with Peroxisome Biogenesis Disorders and Evidence for a Founder Haplotype for the Most Common Mutation in PEX10 Gene." In Advances in Experimental Medicine and Biology. Springer US, 2003. http://dx.doi.org/10.1007/978-1-4419-9072-3_10.

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Rosa, Laís. "No meio do caminho, a gente dança." In TEPe 2022 - Encontro Internacional sobre a Cidade, o Corpo e o Som. INET-md, Faculdade de Motricidade Humana, Universidade de Lisboa, 2022. http://dx.doi.org/10.53072/ilic8040/per10.

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Mano, Shoji, Kazumi Hikino, and Masatake Kanai. "Ubiquitination on the Peroxisomal Membrane for Protein Transport in Plants." In Modifications in Biomacromolecules. IntechOpen, 2024. http://dx.doi.org/10.5772/intechopen.112092.

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Peroxisomes are ubiquitous organelles present in most eukaryotic cells that have important biological functions related to fatty acid metabolism and detoxification of reactive oxygen species. Disruption of peroxisomal function affects the survival of cells and organisms. Peroxisomes do not have their own genome, and peroxisomal proteins are encoded in the nuclear genome. Therefore, efficient and accurate posttranslational transport of peroxisomal proteins is necessary to maintain peroxisomal function. In mammals, yeast, and plants, many factors involved in protein transport to peroxisomes have
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Conference papers on the topic "PEG10"

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Scorzo, Augustino V., Caleb Y. Kwon, Rendall R. Strawbridge, and Scott C. Davis. "Comparison of a fast-acting contrast agent for fluorescence-guided neurosurgery with co-administered ALA-PpIX and second-window ICG using fluorescence cryotomography." In Optical Molecular Probes, Imaging and Drug Delivery. Optica Publishing Group, 2025. https://doi.org/10.1364/omp.2025.ow2e.1.

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A promising new untargeted, fast-acting and persistent contrast agent (TMR-PEG1k) for fluorescence-guided surgery was evaluated alongside two clinically tested fluorescent contrast agents for glioma resection (ALA-PpIX and second-window ICG) using fluorescence cryotomography of whole mice.
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Shapovalova, Mariya. "Abstract 1145: Biomarker-driven molecular imaging of prostate cancer using the PEG10 promoter." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-1145.

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Shapovalova, Mariya. "Abstract 1145: Biomarker-driven molecular imaging of prostate cancer using the PEG10 promoter." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-1145.

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Kelly, Marcus P., Achim A. Jungbluth, Bor-Wen Wu, John Bomalaski, Gerd Ritter, and Lloyd J. Old. "Abstract 4519: Small cell lung cancers lack expression of argininosuccinate synthase (ASS) and are sensitive to arginine deprivation using arginine deiminase-PEG20 (ADI-PEG20)." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-4519.

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Khadeir, Ramsay S., Melissa M. Phillips, Mandeep Sagoo, Victoria Cohen, Caroline Thuang, and Peter W. Szlosarek. "Abstract 5569: The impact of ADI-PEG20 on PDL1 expression in ASS1 deficient uveal melanoma." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-5569.

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Yang, Rui. "Research on Quantitative Methylation Action of PEG1 in Assisted Reproductive Technologies by Network Video Consultation Platform." In 2015 7th International Conference on Information Technology in Medicine and Education (ITME). IEEE, 2015. http://dx.doi.org/10.1109/itme.2015.139.

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Gupta, Sounak, Mir Maria, John Bomalaski, et al. "Abstract 11: Arginine deprivation therapy using ADI-PEG20 as a novel therapeutic modality in the treatment of bladder cancer." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-11.

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Szlosarek, Peter W., Fiona Luong, Andrew Clear, et al. "Abstract 4067: Pegylated arginine deiminase (ADI-PEG20) as a potential novel therapy for argininosuccinate synthetase-deficient acute myeloid leukemia." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-4067.

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Allen, Michael, Nelofer Syed, Tim Crook, et al. "Abstract A23: Frequent ASS1 deficiency in bladder cancer and sensitivity to pegylated arginine deiminase (ADI-PEG20): A potential novel therapeutic strategy." In Abstracts: AACR International Conference on Translational Cancer Medicine-- Jul 11-14, 2010; San Francisco, CA. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1078-0432.tcmusa10-a23.

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Sowers, Rebecca, John Bomalaski, and Richard Gorlick. "Abstract 1692: Expression of argininosuccinate synthetase protein and correlation with response to the novel therapeutic pegylated arginine deiminase (ADI-PEG20) in osteosarcoma." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-1692.

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