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Relevant bibliographies by topics / Pelvic inflammatory disease

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Academic literature on the topic 'Pelvic inflammatory disease'

Author: Grafiati

Published: 4 June 2021

Last updated: 30 July 2024

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Journal articles on the topic "Pelvic inflammatory disease"

1

Peterson, Herbert B., Edward I. Galaid, and Willard Gates. "Pelvic Inflammatory Disease." Emergency Medicine Clinics of North America 9, no. 2 (May 1991): 437–49. http://dx.doi.org/10.1016/s0733-8627(20)30495-8.

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Trent, M. "Pelvic Inflammatory Disease." Pediatrics in Review 34, no. 4 (April 1, 2013): 163–72. http://dx.doi.org/10.1542/pir.34-4-163.

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Popa, Elena, Ana-Aurelia Chiş-Şerban, Carmen Manole, Andrei Popa, and Adorata Coman. "Pelvic inflammatory disease." Medic.ro 6, no. 132 (2019): 40. http://dx.doi.org/10.26416/med.132.6.2019.2659.

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Pletcher, Jonathan R., and Gail B. Slap. "Pelvic Inflammatory Disease." Pediatrics in Review 19, no. 11 (November 1998): 363–67. http://dx.doi.org/10.1542/pir.19-11-363.

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Leyssens, A., F. M. Vanhoenacker, and C. Libeer. "Pelvic inflammatory disease." Journal of the Belgian Society of Radiology 94, no. 3 (March 11, 2011): 128. http://dx.doi.org/10.5334/jbr-btr.540.

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Melestean-Bratu, Madalina-Ioana, Antoine Edu, Mihaela Bujor, Stelian Conci, Florin Isopescu, Radu Mateescu, Mihai Dumitrascu, Florica Sandru, Andreea Carp-Veliscu, and Claudia Mehedintu. "Pelvic inflammatory disease." Romanian Journal of Medical Practice 16, S6 (December 15, 2021): 5–7. http://dx.doi.org/10.37897/rjmp.2021.s6.1.

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Pelvic inflammatory disease (PID) is a very common condition among women of reproductive age. It is also a common reason for presenting to the emergency room. Although it often presents with mild symptoms, it is very important to consider it in any sexually active patient in order to start treatment as soon as possible and avoid complications.

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Pletcher, Jonathan R., and Gail B. Slap. "Pelvic Inflammatory Disease." Pediatrics In Review 19, no. 11 (November 1, 1998): 363–67. http://dx.doi.org/10.1542/pir.19.11.363.

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Trent, Maria. "Pelvic Inflammatory Disease." Pediatrics In Review 34, no. 4 (April 1, 2013): 163–72. http://dx.doi.org/10.1542/pir.34.4.163.

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Soper, David E. "PELVIC INFLAMMATORY DISEASE." Infectious Disease Clinics of North America 8, no. 4 (December 1994): 821–40. http://dx.doi.org/10.1016/s0891-5520(20)30628-0.

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Rakha, Serena S. "Pelvic inflammatory disease." InnovAiT: Education and inspiration for general practice 11, no. 4 (March 2, 2018): 198–200. http://dx.doi.org/10.1177/1755738017750998.

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Pelvic inflammatory disease (PID) is a generalised term that describes infection of the upper genital tract in women. It is commonly caused by the sexually transmitted infections Chlamydia Trachomatis and Neisseria Gonorrhoea. Clinical features of PID include pelvic pain, deep dyspareunia, cervical motion tenderness and adnexal tenderness. Complications include infertility, ectopic pregnancy and chronic pelvic pain. Therefore, it is important for GPs to have a high index of suspicion when women present with symptoms of PID and to initiate antibiotic treatment before vaginal swab results are known.

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Dissertations / Theses on the topic "Pelvic inflammatory disease"

1

Simms, Ian David. "The epidemiology of pelvic inflammatory disease in England." Thesis, University College London (University of London), 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.407400.

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De, Silva H. J. "The pathogenesis of inflammation in pelvic ileal reservoirs." Thesis, University of Oxford, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.257941.

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Ueda, Hiroyuki. "Adnexal Masses Caused by Pelvic Inflammatory Disease : Appearance in MR Images." Kyoto University, 2003. http://hdl.handle.net/2433/148764.

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Logan, Susan. "Screening for Chlamydia trachomatis in obstetrics and gynaecology." Thesis, University of Aberdeen, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.288266.

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In 1996, a RCOG Study Group reporting on the prevention of pelvic infection highlighted the considerable role C. trachomatis played in female reproductive morbidity and the potential advantages of DNA based assays. A national screening programme was suggested, as Sweden and the USA had demonstrated that screening women could decrease prevalence and pelvic inflammatory disease rates. In the UK, out with genito-urinary medicine clinics, awareness of the infection and screening was virtually non-existent. Women attending obstetric and gynaecology-affiliated clinics were at increased risk of ascending infection compared to the general public and ideally placed for opportunistic screening. However, patients were TESTED only if symptomatic, by specimens taken from the endocervix for culture or antigen detection assay. It was from this background that the studies commenced. The thesis comprises of: - A questionnaire survey assessing sexually active women’s knowledge of C. trachomatis infection and perceived acceptability of different methods and settings for screening. Women attending induced abortion and family planning clinics in Aberdeen and Leeds were recruited. - A prevalence study, aiming to identify who should undergo screening. Sexually active women attending six different clinical settings in Aberdeen’s Obstetrics & Gynaecology department were screened for Chlamydia. - A study assessing test performance and acceptability of four different screening approaches (enzyme immunoassay of endocervical specimens and ligase chain reaction assay of endocervical, clinician-collected vulva!, and urine specimens) to opportunistically screen pregnant and non-pregnant women, under 25 years of age. - A study evaluating patient-collected vulval swabs, as an alternative to non-invasive screening by urine. Women under 25 years of age attending a family planning clinic were opportunistically screened and test performance and acceptability evaluated. - A study determining whether the measurement of chlamydial IgG antibodies alone or in combination with medical history and/or transvaginal ultrasound can predict tubal infertility in subfertile women.

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Molander, Pontus. "Diagnosis and management of patients with clinically suspected acute pelvic inflammatory disease." Helsinki : University of Helsinki, 2003. http://ethesis.helsinki.fi/julkaisut/laa/kliin/vk/molander/.

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Davies, Bethan. "The association between Chlamydia trachomatis and pelvic inflammatory disease : findings from observational studies." Thesis, Imperial College London, 2014. http://hdl.handle.net/10044/1/42879.

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Estimates of the cost-effectiveness of chlamydia control interventions are highly sensitive to the risk of progression from genital chlamydia infection to pelvic inflammatory disease (PID). There is no consensus for the risk of PID following asymptomatic chlamydia infections detected through population-based testing. The aim of this thesis is to generate improved estimates of this risk of PID that can be used to parameterise mathematical models and inform chlamydia control policy. We have determined the risk of PID following a positive chlamydia test in three cohorts: a small historic prospective clinical cohort of sex workers (Praed Street Project (PSP)); a large population-based retrospective cohort from Manitoba, Canada established for this research; and a large nationally representative retrospective cohort from Denmark. The risk of PID was higher in women with a positive chlamydia test compared to women who tested negative (PSP: adjusted hazard ratio (AHR) 2.03 (95%CI 0.75-5.49); Manitoba: 1.55 (95%CI 1.43-1.70); Denmark: 1.42 (95%CI 1.32-1.53)). There was heterogeneity in this risk: 13-23% higher following a repeat infection; up to four-fold higher in younger women (Manitoba: AHR 4.55 (95%CI 3.59-5.78) in 12-15 compared to 30-40 years); two-fold higher following previous gonorrhoea (PSP: 2.28 (95%CI 1.14-4.56)). The increased risk following a positive test lasted considerably longer than the likely duration of infection and fewer than 10% of PID diagnoses within 12 months of a test could be attributed to a positive result. This suggests that there are other important causes of PID. Individual-based risks of progression that capture this heterogeneity may improve the accuracy of estimates from mathematical models and therefore their utility to policy makers. Further research is needed to fully characterise the aetiology of PID to inform the design of chlamydia control interventions. In the meantime, interventions should focus on young women and those at risk of repeat chlamydia infection.

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Bhuiya, Sumita. "The impact of inflammatory genetics on oocyte viability and embryo development in women with chronic pelvic inflammatory disease." Thesis, University of Leeds, 2013. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.581454.

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Infection by Chlamydia trachomatis is the leading cause of tubal disease and accounts for a significant proportion of the physical, psychosocial and economic burden of infertility. As it is frequently asymptomatic and opportunities for early treatment are missed, repeated/chronic infection leads to permanent/irreversible inflammation- induced tubal damage, which may extend as far as the ovary. However, not all infected women develop tubal pathology, possibly due to inherent host-specific and environment-induced differences in immune response. This study investigated the impact of putative cytokine-based gene and protein-mediated influences governing the host immune response to C. trachomatis infection and its effects on tubal pathology and ovarian function. The study population comprised 367 women whose infection status was determined serologically. Genetic predisposition to tubal disease was assessed by profiling single nucleotide polymorphism allelic variants in nine cytokines. Ovarian follicular fluid and circulatory cytokine profiles were determined by fluid-phase multiplex immunoassay, while serum antl-Mullerlan hormone levels were measured by enzyme-linked immunosorbent assay. No genetic or circulatory markers of susceptibility to tubal pathology were identified. Furthermore, tubal disease had no significant impact on ovarian function (based on ovarian reserve and follicular cytokine levels). The failure to identify genetic susceptibility markers of tubal disease may reflect the fact that the disease is under polygenic influence with superimposed environmental predisposing factors. Together with the fact that local inflammatory processes may not be reflected systemically, putative changes in cytokine profile between initial infection and the study period could explain why no serum biomarkers of tubal disease could be identified. Finally, the observation that tubal disease had no measurable effect on ovarian pathophysiology is likely to be due either to the small number of women with severe disease recruited to the study or due to other confounding factors eliminating the contribution of the infective inflammatory responses. Further investigation is thus warranted.

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Cover, Natasha Faith. "A Novel Device and Nanoparticle-Based Approach for Improving Diagnosis and Treatment of pelvic Inflammatory Disease." Scholar Commons, 2012. http://scholarcommons.usf.edu/etd/4020.

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Pelvic Inflammatory Disease (PID) is one of the most common causes of morbidity in women. PID is a polymicrobial infection of the female reproductive tract, and is associated with pelvic pain, abnormal uterine bleeding, and tubal damage that can lead to ectopic pregnancies and infertility. It is curable but the effects of PID can be permanent if not properly diagnosed and treated. PID presents as a spectrum of disease and is often missed at early stages; even acute PID can be difficult to diagnose, as there is no single conclusive diagnostic test. Currently, PID is identified and treated syndromically because pelvic pain is the only consistent clinical finding. The Center for Disease Control and Prevention (CDC) recommends doxycycline, a broad-spectrum antibiotic, for treatment but doxycycline can cause gastrointestinal irritation and local inflammation leading to an incomplete treatment. Most cases of PID are polymicrobial infections of the tubes and endometrium, which are not accessible to culture due to the difficulty of procuring samples above the naturally contaminated vagina and distal cervix. Given the difficulty of properly diagnosing PID and the limitations and side effects of the current treatments, there is an urgent need for new approaches for improving the accuracy for diagnosis and treatment of PID. We propose a new and practical approach to collect sterile specimen samples from the endometrium for more accurate PID diagnosis, and to treat the reproductive tract locally using doxycycline-loaded nanoparticles. The proposed research presents a novel sterile uterine sampler cover (SUSC) device that can safely and effectively collect uncontaminated specimen samples from the uterus, and also deliver nano-encapsulated drugs directly to the site of infection. The analysis of uncontaminated endometrium samples is expected to provide an understanding of uterine flora in symptomatic and asymptomatic women, and will lead to the identification of infective microbes in symptomatic women for pathogen-specific treatment. The use of nano-encapsulated doxycycline will enable localized drug delivery to lower drug dosage and minimize side effects for the patient. The doxycycline-loaded nanoparticles are characterized and evaluated based on their drug release properties, size distribution, and tissue response in vitro. This research will lead towards a more effective approach for the diagnosis and treatment of PID while freeing women from prolonged systemic treatments and their adverse effects. Moreover, this research will increase our understanding of the uterine biome under various hormonal and pathologic conditions, in symptomatic and asymptomatic women.

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Viberga, Ilze. "The Clinical Appearance of Pelvic Inflammatory Disease in Relation to Use of Intrauterine Device in Latvia : A Study with Special Emphasis on Factors Influencing the Clinical Course of PID in IUD Users." Doctoral thesis, Uppsala : Acta Universitatis Upsaliensis, 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-6458.

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Armitage, Trudi. "Development of improved diagnostics for acute and persistent Chlamydia trachomatis infections." Queensland University of Technology, 2007. http://eprints.qut.edu.au/16584/.

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The asymptomatic nature of chlamydial infection renders the differential diagnosis of acute and chronic infection difficult. An untreated Chlamydia trachomatis infection can become chronic, result in disease sequelae such as salpingitis and pelvic inflammatory disease (PID), and ultimately culminate in tubal occlusion and infertility. Diagnostic tests for C. trachomatis such as nucleic acid amplification testing (PCR), antigen detection and serological methods have variable performance capabilities with respect to sensitivity, specificity and stage of infection. The use of PCR as a diagnostic tool is somewhat limited, as specimen collection is routinely sampled from the lower genital tract; hence, infections in the fallopian tube where inflammatory damage is most significant, escape detection. Furthermore, PCR can only detect selected Chlamydia DNA sequences from readily accessible sites of the genital tract, and therefore cannot differentiate between acute and chronic infection. Other serological assays aim to discriminate the various stages of C. trachomatis infection through identification of key antigens. The efficacy of these assays however is impeded due to cross-reactivity between chlamydial species and the subsequent antibody response against the target antigen is not restricted to patients with a specific stage of infection. To identify antibody responses capable of differentiating various states of chlamydial infection, samples were collected from both men and women given the variability of immune responses between the two genders. Samples were assigned to a patient group according to infection status and then probed against protein extracts of HEp-2 cells infected with C. trachomatis serovar L2 and HEp-2 cells pre-treated with IFN-γ and infected with C. trachomatis serovar L2. (persistence cell culture) Serological analysis revealed the presence of five antigens (denoted bands A, B, C, D and M) which were shown to be differential between patient groups. Identification of bands B and C by N-terminal sequencing provided two possible candidates for each antigen, ie. CT727 and CT396 (band B) and CT157 and CT423 (band C). In contrast, band M which was unique to males was a PmpB (probable outer membrane protein B) fragment. The four target antigens (CT157, CT423, CT727 and CT396) were expressed as recombinant proteins using autoinduction media and were subsequently probed by both male and female sera to evaluate their diagnostic potential. Results showed that two chlamydial antigenic targets (CT157 and CT727) have the potential to discriminate between acute and chronic C. trachomatis infection. However, since only a small number of samples (n = 3) were used for this aspect of the study, the findings should simply be viewed as preliminary. In females, sensitivity and specificity values were derived using various combinations of the four target antigens into a panel format for the purpose of detecting chronic C. trachomatis infections. The preferred format was B + C with a sensitivity and specificity of 80% and 84% respectively. Using the IFN-γ-mediated persistence model, only two of the five antigenic targets were shown to be differentially expressed. PmpB in males and CT157 (the most likely band C candidate) in females were shown to be up-regulated to varying degrees in samples across the patient groups. We also demonstrated that no other chlamydial antigens are up-regulated during a persistent C. trachomatis infection. In conclusion, although combinations of bands A, B, C, D and M differentiate between male and female patient groups under normal chlamydial growth conditions, during IFN-γ-induced persistence, only bands C (CT157) and M (CT413 - PmpB) are up-regulated thus suggesting a potential role in chronic C. trachomatis infection.

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Books on the topic "Pelvic inflammatory disease"

1

O'Donnell, Judith A. Pelvic inflammatory disease. Philadelphia: Chelsea House Publishers, 2005.

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Landers, Daniel V., and Richard L. Sweet, eds. Pelvic Inflammatory Disease. New York, NY: Springer New York, 1997. http://dx.doi.org/10.1007/978-1-4612-0671-2.

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National Institute of Allergy and Infectious Diseases (U.S.), ed. Pelvic inflammatory disease. [Bethesda, Md: National Institute of Allergy and Infectious Diseases, 1992.

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National Institutes of Health (U.S.), ed. Pelvic inflammatory disease. Bethesda, Md: National Institutes of Health, 1988.

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National Institutes of Health (U.S.), ed. Pelvic inflammatory disease. Bethesda, Md: National Institutes of Health, 1989.

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L, Sweet Richard, and Wiesenfeld Harold, eds. Pelvic inflammatory disease. London: Taylor & Francis, 2006.

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National Institute of Allergy and Infectious Diseases (U.S.), ed. Pelvic inflammatory disease. [Bethesda, Md: National Institute of Allergy and Infectious Diseases, 1992.

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Hayhurst, Chris. Pelvic inflammatory disease. New York: Rosen, 2009.

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V, Landers Daniel, and Sweet Richard L, eds. Pelvic inflammatory disease. New York: Springer, 1997.

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S, Berger Gary, and Weström Lars 1930-, eds. Pelvic inflammatory disease. New York: Raven Press, 1992.

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Book chapters on the topic "Pelvic inflammatory disease"

1

Greydanus, Donald E., Kevin W. Cates, and Nina Sadigh. "Pelvic Inflammatory Disease." In Sexually Transmitted Infections in Adolescence and Young Adulthood, 69–86. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-20491-4_6.

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Gordon, Alan G., and B. Victor Lewis. "Pelvic Inflammatory Disease." In Gynaecological Endoscopy, 57–71. Boston, MA: Springer US, 1988. http://dx.doi.org/10.1007/978-1-4899-3240-2_5.

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de Camargo Penteado, César Amaral, Geraldo Souza Pinho Alves, and Harley De Nicola. "Pelvic Inflammatory Disease." In Atlas of Imaging in Infertility, 33–40. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-13893-0_4.

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Sprang, M. LeRoy. "Pelvic Inflammatory Disease." In Office Gynecology, 76–96. New York, NY: Springer New York, 1993. http://dx.doi.org/10.1007/978-1-4612-4340-3_7.

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Augustin, Goran, and Maja Prutki. "Pelvic Inflammatory Disease." In CT Scan in Abdominal Emergency Surgery, 199–206. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-48347-4_17.

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McNicholas, Colleen, and Jeffrey F. Peipert. "Pelvic Inflammatory Disease." In Practical Pediatric and Adolescent Gynecology, 361–65. Oxford, UK: Blackwell Publishing Ltd., 2013. http://dx.doi.org/10.1002/9781118538555.ch51.

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Goldmeier, David, and Simon Barton. "Pelvic Inflammatory Disease." In Sexually Transmitted Diseases, 110–14. London: Springer London, 1987. http://dx.doi.org/10.1007/978-1-4471-1432-1_11.

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Gronthoud, Firza Alexander. "Pelvic Inflammatory Disease." In Practical Clinical Microbiology and Infectious Diseases, 285–87. First edition. | Boca Raton : CRC Press, 2020.: CRC Press, 2020. http://dx.doi.org/10.1201/9781315194080-4-46.

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Buy, Jean Noel, and Michel Ghossain. "Pelvic Inflammatory Disease." In Gynecological Imaging, 759–83. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-31012-6_33.

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van Gunst, S. G., and V. G. Pigmans. "Pelvic inflammatory disease." In NHG-standaarden voor de praktijkassistente 2014, 359–62. Houten: Bohn Stafleu van Loghum, 2013. http://dx.doi.org/10.1007/978-90-368-0485-1_58.

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Conference papers on the topic "Pelvic inflammatory disease"

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Xianfang Xu and Yugai Gao. "Notice of Retraction: The Physical Therapy of pelvic inflammatory disease." In 2010 International Conference on Computer Application and System Modeling (ICCASM 2010). IEEE, 2010. http://dx.doi.org/10.1109/iccasm.2010.5620234.

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Basaraba, I., and E. C. Hoffman. "Severe Complicated Pelvic Inflammatory Disease: A Presentation of Unusual Polymicrobial Pathogens." In American Thoracic Society 2024 International Conference, May 17-22, 2024 - San Diego, CA. American Thoracic Society, 2024. http://dx.doi.org/10.1164/ajrccm-conference.2024.209.1_meetingabstracts.a3595.

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Filipiuc, Silvia, Ramona Gabriela Ursu, Dragos Nemescu, Diana Costin, and Luminita Smaranda Iancu. "qPCR detection of Neisseria Gonorrhoeae in Patients with Clinic Pelvic Inflammatory Disease." In 2019 E-Health and Bioengineering Conference (EHB). IEEE, 2019. http://dx.doi.org/10.1109/ehb47216.2019.8970010.

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Oakeshott, P. "S04.4 Evidence based approaches to preventing pelvic inflammatory disease (PID) and its sequelae." In Abstracts for the STI & HIV World Congress, July 14–17 2021. BMJ Publishing Group Ltd, 2021. http://dx.doi.org/10.1136/sextrans-2021-sti.31.

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Trent, Maria, Jamie Perin, Pamela Matson, and Charlotte Gaydos. "P595 Vaginal microbiota among adolescent and young adult women with pelvic inflammatory disease." In Abstracts for the STI & HIV World Congress (Joint Meeting of the 23rd ISSTDR and 20th IUSTI), July 14–17, 2019, Vancouver, Canada. BMJ Publishing Group Ltd, 2019. http://dx.doi.org/10.1136/sextrans-2019-sti.665.

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Ranasinghe, Samindra, and Luciana Rubinstein. "P99 Haemophilus influenzae as the cause of pelvic inflammatory disease: a rare case presentation." In BASHH 2022 Abstracts. BMJ Publishing Group Ltd, 2022. http://dx.doi.org/10.1136/sextrans-bashh-2022.144.

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Bittleston, H., J. Coombe, M. Temple-Smith, D. Bateson, J. Hunady, L. Sanci, J. Hocking, and J. Goller. "P088 Australian general practitioners’ consideration of pelvic inflammatory disease in women diagnosed with an STI, and barriers to providing pelvic examinations." In Abstracts for the STI & HIV World Congress, July 14–17 2021. BMJ Publishing Group Ltd, 2021. http://dx.doi.org/10.1136/sextrans-2021-sti.218.

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Lewis, Joanna, Paddy Horner, and Peter White. "P603 Estimating population burden of pelvic inflammatory disease due tomycoplasma genitaliumin england: an evidence synthesis." In Abstracts for the STI & HIV World Congress (Joint Meeting of the 23rd ISSTDR and 20th IUSTI), July 14–17, 2019, Vancouver, Canada. BMJ Publishing Group Ltd, 2019. http://dx.doi.org/10.1136/sextrans-2019-sti.671.

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Jenkins, Rhian, Clare Tavener, Justine Orme, Natalie Edelman, Colin Fitzpatrick, and Daniel Richardson. "P103 History of sexual assault and domestic violence in patients diagnosed with pelvic inflammatory disease." In BASHH 2023 Abstracts. BMJ Publishing Group Ltd, 2023. http://dx.doi.org/10.1136/sextrans-bashh-2023.142.

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Davies, Bethan, Katy Me Turner, Berit Andersen, Henrik Westh, and Helen Ward. "P3.25 Quantification of the risk of pelvic inflammatory disease following achlamydia trachomatistest by diagnostic test type." In STI and HIV World Congress Abstracts, July 9–12 2017, Rio de Janeiro, Brazil. BMJ Publishing Group Ltd, 2017. http://dx.doi.org/10.1136/sextrans-2017-053264.262.

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Reports on the topic "Pelvic inflammatory disease"

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Lo, Shyh-Ching. Mycoplasma Infections and Non-Gonococcal Urethritis and Pelvic Inflammatory Disease in Women Patients. Fort Belvoir, VA: Defense Technical Information Center, October 1995. http://dx.doi.org/10.21236/ada332972.

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Zhu, Rui, Mingle Li, and Xueman Ma. Guizhi Fuling Combined with Antibiotic Therapy for Pelvic Inflammatory Disease: A Systematic Review and Meta-Analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, October 2020. http://dx.doi.org/10.37766/inplasy2020.10.0035.

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Hao, Hongjuan, Xiao Chen, Zhaohua Wang, Li Feng, and Xiaoli Zhao. Which patients with hydrosalpinges will benefit more from reproductive surgery to improve natural pregnancy outcomes?-System evaluation and Meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, September 2022. http://dx.doi.org/10.37766/inplasy2022.9.0105.

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Review question / Objective: To evaluate natural pregnancy outcomes of hydrosalpinx and different grades of hydrosalpinx,which perform reproductive surgery. Information sources: Electronic searches of Pubmed, Embase, Cochrane Library, Web of science, and Clinical Trails. All literature on hydrosalpinx and reproductive surgery were retrieved. The Mesh subject words and free words are: “Salpingitis, Salpingitides, hydrosalpin*, distal tubal occlusion, pelvic inflammatory disease, Reproductive surgical procedure, tubal surgery, microsurg *, laparoscopic surgery ,salpingostomy , salpingectomy”. References of the original and reviewed articles were manually searched to include the relevant literature.

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Frost, Jennifer J., Mia R. Zolna, Lori F. Frohwirth, Ayana Douglas-Hall, Nakeisha Blades, Jennifer Mueller, Zoe H. Pleasure, and Shivani Kochhar. Publicly Supported Family Planning Services in the United States: Likely Need, Availability and Impact, 2016. Guttmacher Institute, October 2019. http://dx.doi.org/10.1363/2019.30830.

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Key Points Key Points In 2016, 20.6 million U.S. women were likely in need of public support for contraceptive services and supplies. Between 2010 and 2016, the number of women likely in need of public support for contraceptive services and supplies rose 8% overall. Among women below 250% of federal poverty guidelines, there was a 12% increase; among adolescents, there was a 5% decline. Between 2013 and 2016, the number of women likely in need of public support for contraceptive services who had neither public nor private health insurance fell more than one-third (36%), from 5.6 million to 3.6 million. States that implemented the Affordable Care Act’s Medicaid expansion experienced particularly large declines. Between 2010 and 2016, the overall number of women receiving publicly supported contraceptive services remained stable at about nine million women. However, the number of women served by different types of providers shifted dramatically over this period. While Title X–funded sites continued to serve the largest segment of women receiving publicly supported care, their patient load fell by 25%, from 4.7 million in 2010 to 3.5 million in 2016. The number of contraceptive patients served by other public clinics that do not receive Title X funding rose by 29% and the number of women receiving Medicaid-funded contraceptive services from private providers rose by 19%. In 2016, women who obtained contraceptive services from all publicly supported providers were able to postpone or avoid two million pregnancies that they would have been unable to prevent without access to publicly supported care. Women who obtained contraceptives from Title X–funded clinics avoided 755,000 pregnancies. Screening and vaccination services provided at family planning visits with all publicly supported providers helped patients avoid more than 12,000 cases of pelvic inflammatory disease and nearly 2,000 cases of cervical cancer in 2016. More than 100,000 chlamydia infections, 18,000 gonorrhea infections and 800 cases of HIV were prevented among the partners of women obtaining publicly funded contraceptive care.

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The Copper T 380 Intrauterine Device: A Summary of Scientific Data. Population Council, 1992. http://dx.doi.org/10.31899/cbr1992.1000.

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Intrauterine devices are the most widely used of all reversible contraceptive methods worldwide. Modern IUDs are among the most effective and long-acting methods of family planning and are acceptable options for many women. This monograph presents highlights of the clinical performance of the Copper T 380 over eight years, including the latest data on effectiveness, expulsions, and continuation rates. It incorporates data from Population Council and World Health Organization studies, and work by scientists at Family Health International and in several countries. Clinical studies of the three models of the Copper T 380 have provided substantial evidence of the safety, effectiveness, convenience, acceptability, and long-acting quality of this IUD. The bulk of the material in this document presents preclinical and clinical performance, including mechanisms of action, effectiveness, outcome of accidental pregnancy, rates of expulsion and ectopic pregnancy, side effects, continuation rates, return to fertility, and lactation and IUD use. There is also discussion of the data on IUD use and pelvic inflammatory disease, and the importance of performing skilled insertions under aseptic conditions.

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