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Journal articles on the topic 'Pentraxina 3'

Author: Grafiati

Published: 4 June 2021

Last updated: 11 March 2023

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1

El Wakeel, Maged A., Rania N. Sabry, Ghada M. El-kassas, et al. "Pentraxin 3: A Potential Novel Predictor for Neonatal Pulmonary Hypertension." Open Access Macedonian Journal of Medical Sciences 7, no. 15 (2019): 2424–27. http://dx.doi.org/10.3889/oamjms.2019.638.

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BACKGROUND: Persistent pulmonary hypertension of the newborn (PPHN) is a serious neonatal problem which has a high mortality rate even with advanced modes of mechanical ventilation. Pentraxin 3 is one of the long pentraxins, which plays an essential role in regulation of cell proliferation and angiogenesis. AIM: This study aims to assess serum pentraxin 3 levels in neonates with pulmonary arterial hypertension and compare them in those who have other congenital heart diseases and healthy neonates. Also, we intended to evaluate serum levels of CRP as a mediator of inflammation in the stud

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2

Kunes, P., Z. Holubcova, M. Kolackova, and J. Krejsek. "Pentraxin 3(PTX 3): An Endogenous Modulator of the Inflammatory Response." Mediators of Inflammation 2012 (2012): 1–10. http://dx.doi.org/10.1155/2012/920517.

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Inflammatory or anti-inflammatory? That is the question as far as the acute-phase response and its mediators, the pentraxins, are concerned. Only some ten years ago, the classical or short pentraxin C-reactive protein and the newly discovered long pentraxin PTX3 were considered to exert most of the detrimental effects of acute inflammation, whether microbial or sterile in origin. However, accumulating evidence suggests an at least dichotomous, context-dependent outcome attributable to the pentraxins, if not a straightforward anti-inflammatory nature of the acute-phase response. This paper is f

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3

Alieva, Amina M., Natalia V. Teplova, Maxim A. Batov, et al. "Pentraxin-3 – a promising biological marker in heart failure: literature review." Consilium Medicum 24, no. 1 (2022): 53–59. http://dx.doi.org/10.26442/20751753.2022.1.201382.

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According to many studies, inflammation plays a very significant role in the pathogenesis of heart failure. Many studies have demonstrated an increase in circulating levels of inflammatory markers and cytokines such as C-reactive protein, tumor necrosis factor-a (TNF-a), and interleukins. C-reactive protein is produced in the liver in response to stimulation by various cytokines, mainly interleukin-6, and is a member of the pentraxin superfamily. Pentraxin-3, which is a long pentraxin, has a C-terminal domain of pentraxin similar to the classic short pentraxins, but differs from them in the pr

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4

Lucarelli, G., G. S. Netti, F. Spadaccino, et al. "La Pentraxina 3 (PTX3) Modula l’immunoflogosi nel microambiente tumorale ed è un fattore prognostico in pazienti con carcinoma renale a cellule chiare." European Urology Open Science 20 (October 2020): S63—S64. http://dx.doi.org/10.1016/s2666-1683(20)35398-2.

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5

Bonacina, Fabrizia, Andrea Baragetti, Alberico Luigi Catapano, and Giuseppe Danilo Norata. "Long Pentraxin 3: Experimental and Clinical Relevance in Cardiovascular Diseases." Mediators of Inflammation 2013 (2013): 1–10. http://dx.doi.org/10.1155/2013/725102.

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Pentraxin 3 (PTX3) is an essential component of the humoral arm of innate immunity and belongs, together with the C-reactive protein (CRP) and other acute phase proteins, to the pentraxins' superfamily: soluble, multifunctional, pattern recognition proteins. Pentraxins share a common C-terminal pentraxin domain, which in the case of PTX3 is coupled to an unrelated long N-terminal domain. PTX3 in humans, like CRP, correlates with surrogate markers of atherosclerosis and is independently associated with the risk of developing vascular events. Studies addressing the potential physiopathological r

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6

Introna, M., VV Alles, M. Castellano, et al. "Cloning of mouse ptx3, a new member of the pentraxin gene family expressed at extrahepatic sites." Blood 87, no. 5 (1996): 1862–72. http://dx.doi.org/10.1182/blood.v87.5.1862.1862.

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Abstract Pentraxins, which include C reactive protein (CRP) and serum amyloid P component (SAP), are prototypic acute phase reactants that serve as indicators of inflammatory reactions. Here we report genomic and cDNA cloning of mouse ptx3 (mptx3), a member of the pentraxin gene family and characterize its extrahepatic expression in vitro and in vivo. mptx3 is organized into three exons on chromosome 3: the first (43 aa) and second exon (175 aa) code for the signal peptide and for a protein portion with no high similarity to known sequences the third (203 aa) for a domain related to classical

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7

Introna, M., VV Alles, M. Castellano, et al. "Cloning of mouse ptx3, a new member of the pentraxin gene family expressed at extrahepatic sites." Blood 87, no. 5 (1996): 1862–72. http://dx.doi.org/10.1182/blood.v87.5.1862.bloodjournal8751862.

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Pentraxins, which include C reactive protein (CRP) and serum amyloid P component (SAP), are prototypic acute phase reactants that serve as indicators of inflammatory reactions. Here we report genomic and cDNA cloning of mouse ptx3 (mptx3), a member of the pentraxin gene family and characterize its extrahepatic expression in vitro and in vivo. mptx3 is organized into three exons on chromosome 3: the first (43 aa) and second exon (175 aa) code for the signal peptide and for a protein portion with no high similarity to known sequences the third (203 aa) for a domain related to classical pentraxin

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8

Nagai, Kei, Atsushi Ueda, Chie Saito, Asako Zempo-Miyaki, and Kunihiro Yamagata. "Annual Decline in Pentraxin 3 Is a Risk of Vascular Access Troubles in Hemodialysis Patients." International Journal of Nephrology 2014 (2014): 1–7. http://dx.doi.org/10.1155/2014/297954.

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Pentraxin 3 (PTX3), a multifunctional modulator of the innate immunoinflammatory response, is higher in patients undergoing hemodialysis than healthy control. Our study focused on annual change in PTX3 levels in patients with chronic hemodialysis, because regularly undergoing hemodialysis for many years modifies vascular inflammatory status. To demonstrate whether annual change in PTX3 is associated with vascular events, we measured blood levels of pentraxins (PTX3 and high-sensitivity C-reactive protein (hsCRP)) at baseline and in the next year in 76 hemodialysis patients and observed 20 pati

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9

Lech, M., C. Rommele, and H. J. Anders. "Pentraxins in nephrology: C-reactive protein, serum amyloid P and pentraxin-3." Nephrology Dialysis Transplantation 28, no. 4 (2012): 803–11. http://dx.doi.org/10.1093/ndt/gfs448.

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10

Szymkowiak, Martyna, Piotr Surmiak, and Małgorzata Baumert. "Pentraxin 3 – possible uses in neonatology and paediatrics." Pediatria i Medycyna Rodzinna 16, no. 3 (2020): 247–50. http://dx.doi.org/10.15557/pimr.2020.0045.

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Pentraxin 3 (PTX3) is a multifunctional acute phase protein belonging to the family of long pentraxins, which is synthesised in numerous cells of the body under the influence of proinflammatory factors and locally at the site of inflammation. Under physiological conditions, PTX3 is stored in neutrophil granules, where there is a constant pool of glycoproteins. Increased pentraxin 3 levels in blood serum are observed as early as 1 hour after a damaging stimulus. Elevation of PTX3 serum levels can be used to diagnose fertility disorders in women as well as in pregnancy pathology, women at risk o

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11

Golconda, Umamaheshwari, Richard Sobonya, and Stephen Klotz. "Do Pentraxins Bind to Fungi in Invasive Human Gastrointestinal Candidiasis?" Journal of Fungi 4, no. 3 (2018): 111. http://dx.doi.org/10.3390/jof4030111.

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Tissue from 13 autopsy cases with invasive gastrointestinal candidiasis was studied for the binding of the pentraxins, C-reactive protein (CRP), pentraxin 3 (PTX3), and serum amyloid P component (SAP) to fungal surfaces. Invasive candidal infection was demonstrated using a hematoxylin and eosin stain and a Gomori methenamine silver stain (GMS). Immunohistochemistry was performed with CRP and PTX3 monoclonal antibodies and did not demonstrate CRP or PTX3 bound to fungi (0 of 13 cases), although CRP was extensively deposited on human tissue. A polyclonal antibody to SAP showed that SAP was bound

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12

Kathariya, Rahul, Hansa Jain, Dnyneshwari Gujar, Archana Singh, Himanshu Ajwani, and Devendra Mandhyan. "Pentraxins as Key Disease Markers for Periodontal Diagnosis." Disease Markers 34, no. 3 (2013): 143–51. http://dx.doi.org/10.1155/2013/259273.

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Periodontal diseases are characterized by a complex set of biologic interactions between a diverse and dynamic microbial ecosystem and the host’s multifaceted and responsive immune and inflammatory machinery. Such interactions between microbial pathogens and various host response systems play a critical role in the development and progression of periodontal disease via the release of inflammatory and immune mediators. Advances in periodontal disease diagnostic are moving toward methods whereby periodontal risk can be identified and quantified by detecting such inflammatory mediators in its seq

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13

Gursu, M., S. Ozturk, Z. Aydin, et al. "Is Pentraxin-3 Stronger Than C-Reactive Protein to Determine Inflammation in Peritoneal Dialysis Patients?" European Journal of Inflammation 10, no. 3 (2012): 289–95. http://dx.doi.org/10.1177/1721727x1201000305.

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Pentraxin-3 (PTX-3) is the prototype of long pentraxins and is produced by many tissues and organs including vascular endothelial cells in response to pro-inflammatory signals. It is thought to be an independent indicator of disease activity. We analyzed the correlation of PTX-3 with other markers of inflammation in peritoneal dialysis (PD) patients. Non-diabetic patients on chronic PD program who meet the dialysis adequacy criteria and who had no active infectious/inflammatory disease were included. Demographic and clinical parameters were recorded as well as hsCRP, fibrinogen, interleukin-6

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14

Battal, Fatih, Özgül Emel Bulut, Şule Yıldırım, Hakan Aylanç, Nazan Kaymaz, and Sema Uysal. "Serum Pentraxin 3 Concentration in Neonatal Sepsis." Journal of Pediatric Infectious Diseases 14, no. 05 (2019): 219–22. http://dx.doi.org/10.1055/s-0039-1688930.

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Objective Neonatal sepsis is one of the most important causes of neonatal morbidity and mortality. Symptoms and signs of neonatal sepsis can be silent; therefore, laboratory investigation is necessary in cases of doubt or if there are risk factors. Early diagnosis is important for early intervention and treatment. The most valuable method for diagnosis is blood culture; however, false positivity due to contamination or false negativity despite ongoing fatal bacterial infections can be seen. Pentraxin 3 is a prototype of the long pentraxin family. It has some differences from C-reactive protein

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15

Roy, Nitai, Katsuki Ohtani, Yoshihiko Hidaka, et al. "Three pentraxins C-reactive protein, serum amyloid p component and pentraxin 3 mediate complement activation using Collectin CL-P1." Biochimica et Biophysica Acta (BBA) - General Subjects 1861, no. 2 (2017): 1–14. http://dx.doi.org/10.1016/j.bbagen.2016.11.023.

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16

Nabrdalik, Katarzyna, Artur Chodkowski, Wojciech Bartman, et al. "Pentraxin 3 and atherosclerosis among type 2 diabetic patients." Open Life Sciences 12, no. 1 (2017): 92–98. http://dx.doi.org/10.1515/biol-2017-0010.

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AbstractType 2 diabetes is contemporarily a major social and epidemiological problem and among others is a strong risk factor for cardiovascular diseases. Pentraxin 3, a potential early biomarker of atherosclerosis, is an acute-phase reactant produced by the peripheral tissues where the inflammation takes place. In this study we examined a group of patients with type 2 diabetes with and without cardiovascular complications compared to persons with normal glucose tolerance (patients with cardiovascular complications and healthy volunteers). Plasma pentraxin 3 concentration as well as some basic

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17

Aygun, O., and R. Yildiz. "Evaluation of thrombomodulin and pentraxin-3 as diagnostic biomarkers in calves with sepsis." Veterinární Medicína 63, No. 7 (2018): 313–20. http://dx.doi.org/10.17221/159/2017-vetmed.

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Early diagnosis and treatment of sepsis in patients are crucial for their survival and can help reduce mortality rates. Novel biomarkers, such as thrombomodulin and pentraxin-3, have been used as diagnostic, prognostic and mortality indicators in patients with sepsis. Plasma thrombomodulin is a vascular endothelial membrane-bound glycoprotein and pentraxin-3 is an acute-phase protein. In the present study, thrombomodulin and pentraxin-3 levels were determined in calves with sepsis, to determine their diagnostic values as well as usefulness as indicators of health status. To this end, 20 neonat

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18

Zhu, Min, Hongli Yu, Ying Sun, and Wenli Yu. "Pentraxin-3 in the Spinal Dorsal Horn Upregulates Nectin-1 Expression in Neuropathic Pain after Spinal Nerve Damage in Male Mice." Brain Sciences 12, no. 5 (2022): 648. http://dx.doi.org/10.3390/brainsci12050648.

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Purpose: Neuropathic pain often originates from nerve injury or diseases of the somatosensory nervous system. However, its specific pathogenesis remains unclear. The requirement for excitatory synaptic plasticity in pain-related syndromes has been demonstrated. A recent study reported that pentraxin-3 is important in glutamatergic synaptic formation and function. Meanwhile, nectin-1 mediates synaptogenesis in neurological disorders. The present study aimed to evaluate whether pentraxin-3 and nectin-1 modulate spinal nerve damage-related neuropathic pain in male mice. Methods: L4 spinal nerve l

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19

Zhang, Jun, Haili Wang, Boming Xia, and Lun Dong. "Brief overview of Pentraxin 3." American Journal of Emergency Medicine 38, no. 8 (2020): 1692. http://dx.doi.org/10.1016/j.ajem.2020.01.018.

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20

Kontny, Frederic, Thomas Andersen, Thor Ueland, et al. "Pentraxin-3 vs C-reactive protein and other prognostic biomarkers in acute coronary syndrome: A substudy of the Platelet Inhibition and Patients Outcomes (PLATO) trial." European Heart Journal: Acute Cardiovascular Care 9, no. 4 (2019): 313–22. http://dx.doi.org/10.1177/2048872619846334.

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Aims: We investigated the dynamics, associations with patient characteristics, other biomarkers, and clinical outcomes of pentraxin 3 in acute coronary syndrome. Methods and results: In multivariate analyses, pentraxin 3 measured in 5154 patients randomised in the Platelet Inhibition and Patients Outcomes (PLATO) trial (NCT00391872) was compared with leukocytes, high-sensitivity C-reactive protein, interleukin-6, cystatin C, N-terminal prohormone brain natriuretic peptide, high-sensitivity troponin T and growth differentiation factor 15 concerning prediction of clinical outcome. Pentraxin 3 pe

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21

Inoue, Kenji. "The Cardioprotective Role of Pentraxin 3." Journal of Atherosclerosis and Thrombosis 22, no. 4 (2015): 335–37. http://dx.doi.org/10.5551/jat.ed011.

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22

Hill, A. L., D. A. Lowes, N. R. Webster, C. C. Sheth, N. A. R. Gow, and H. F. Galley. "Regulation of pentraxin-3 by antioxidants." British Journal of Anaesthesia 103, no. 6 (2009): 833–39. http://dx.doi.org/10.1093/bja/aep298.

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23

Lannergård, Anders, Fredrik Rosenström, Erik Normann, and Anders Larsson. "Serum pentraxin 3 concentrations in neonates." Upsala Journal of Medical Sciences 119, no. 1 (2014): 62–64. http://dx.doi.org/10.3109/03009734.2013.878770.

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24

Zhang, Yingze, and John F. McDyer. "Pentraxin 3 in Primary Graft Dysfunction." American Journal of Respiratory and Critical Care Medicine 186, no. 6 (2012): 475–77. http://dx.doi.org/10.1164/rccm.201207-1158ed.

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25

Korzonek-Szlacheta, Ilona, Bartosz Hudzik, Aleksander Danikiewicz, et al. "Pentraxin-3 and coronary artery disease." Experimental Gerontology 102 (February 2018): 1–2. http://dx.doi.org/10.1016/j.exger.2017.11.016.

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26

Larsson, Anders, Maria Palm, Johanna Helmersson, and Ove Axelsson. "Pentraxin 3 Values During Normal Pregnancy." Inflammation 34, no. 5 (2010): 448–51. http://dx.doi.org/10.1007/s10753-010-9252-x.

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27

Baumert, Małgorzata, Piotr Surmiak, Martyna Szymkowiak, and Agnieszka Janosz. "The Assessment of Pentraxin 3: A Novel Biomarker in Early Detection of Infection in Newborns." BioMed Research International 2021 (June 30, 2021): 1–8. http://dx.doi.org/10.1155/2021/6638622.

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Introduction. As the clinical manifestation of neonatal infection is nonspecific and characterised by varied clinical features, it is highly problematic to establish an early diagnosis. Recently, hopes have been raised by the new acute-phase protein—pentraxin 3 (PTX3). PTX3 belongs to the family of long pentraxins, which is synthesized in numerous cells like endothelial cells, macrophages, and monocytes infiltrating sites of inflammation. Material and Methods. In our research, we have enrolled 29 newborns with infection as the study group and 47 healthy ones as the control group, as well as th

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Ceylan, Mustafa, Omer Faruk Bayraktutan, Sinan Becel, Ömer Atis, Ahmet Yalcin, and Dilcan Kotan. "Serum levels of pentraxin-3 and other inflammatory biomarkers in migraine: Association with migraine characteristics." Cephalalgia 36, no. 6 (2015): 518–25. http://dx.doi.org/10.1177/0333102415598757.

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Background Several studies have been conducted on the inflammatory aspects of migraine. Pentraxins are a novel and important part of innate immunity as a superfamily of acute phase proteins. In our study, we aimed to demonstrate the relationship between migraine and the serum levels of pentraxin-3 (PTX-3), C-reactive protein (CRP), fibrinogen and D-dimer. Methods We recruited 30 migraine patients (in both the attack and interictal period) and 30 healthy controls. Serum samples were obtained from all participants, and a brain MRI performed in the last six months was assessed regarding the prese

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El-Kassas, Ghada M., Maged A. El Wakeel, Mona A. Elabd, et al. "Vitamin D Status in Neonatal Pulmonary Infections: Relationship to Inflammatory Indicators." Open Access Macedonian Journal of Medical Sciences 7, no. 23 (2019): 3970–74. http://dx.doi.org/10.3889/oamjms.2019.592.

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AIM: The study aimed to evaluate serum vitamin D concentrations among neonates with pneumonia. METHODS: This case-control study enrolled 33 neonates with pneumonia in addition to 30 healthy controls. CBC, CRP, Serum vitamin D and Pentraxin 3 levels were measured for all participants. RESULTS: There was significant difference between patients and controls regarding Hemoglobin levels, TLC and CRP (p value < 0.01, = 0.002, < 0.01 respectively). Patients with pneumonia showed significant lower levels of Vit. D (9 ± 2.1) compared to controls (14.1 ± 2.8), P value < 0.01. Howeve

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Petterson, Stine Asferg, Mia Dahl Sørensen, and Bjarne Winther Kristensen. "Expression Profiling of Primary and Recurrent Glioblastomas Reveals a Reduced Level of Pentraxin 3 in Recurrent Glioblastomas." Journal of Neuropathology & Experimental Neurology 79, no. 9 (2020): 975–85. http://dx.doi.org/10.1093/jnen/nlaa088.

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Abstract Glioblastomas (GBM) are highly infiltrative tumors and despite intensive treatment tumor recurrence is inevitable. The immune microenvironment in recurrent GBM is poorly characterized, but it is potentially influenced by therapeutic interventions with surgery, radiotherapy, and chemotherapy. The aim of this study was to obtain a deeper insight in the immune microenvironment in primary and recurrent GBM. Primary and recurrent glioblastoma samples from 18 patients were identified and expression profiling of 770 myeloid innate immune-related markers was performed. Leukemia inhibitory fac

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31

Augusto, Jean-François, Caroline Poli, Céline Beauvillain, et al. "Anti-pentraxin antibodies in autoimmune systemic diseases: Focus on anti-pentraxin-3 autoantibodies." International Reviews of Immunology 36, no. 3 (2017): 145–53. http://dx.doi.org/10.1080/08830185.2017.1284210.

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32

Barazzoni, R., M. Zanetti, M. Giuricin, et al. "PP219-SUN GASTRIC BY-PASS DIFFERENTIALLY MODULATES CIRCULATING SHORT AND LONG PENTRAXINS – HIGHER PLASMA PENTRAXIN-3 AFTER GASTRIC BY-PASS-INDUCED WEIGHT LOSS." Clinical Nutrition 32 (September 2013): S105—S106. http://dx.doi.org/10.1016/s0261-5614(13)60264-2.

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33

Turkmen, O., S. Mollaoglu, G. Goynumer, and B. Isbilen. "Plasma pentraxin 3 levels in preeclamptic patients." Clinical and Experimental Obstetrics & Gynecology 42, no. 2 (2015): 220–23. http://dx.doi.org/10.12891/ceog1825.2015.

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34

Dickerson, Faith, Cassie Stallings, Andrea Origoni, et al. "Pentraxin 3 is reduced in bipolar disorder." Bipolar Disorders 17, no. 4 (2014): 409–14. http://dx.doi.org/10.1111/bdi.12281.

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35

Agilli, Mehmet, and Fevzi Nuri Aydin. "Evaluation of pentraxin-3 in POEMS syndrome." Journal of Neuroimmunology 278 (January 2015): 136. http://dx.doi.org/10.1016/j.jneuroim.2014.12.015.

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Razvina, Olga, Shuying Jiang, Koichi Matsubara, et al. "Differential expression of pentraxin 3 in neutrophils." Experimental and Molecular Pathology 98, no. 1 (2015): 33–40. http://dx.doi.org/10.1016/j.yexmp.2014.11.009.

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Guo, Tang-Meng, Li Ke, Xiu-E. Zhang, Ling Li, Li-Juan Xiong, and Bei Cheng. "Reply: Pentraxin-3 and coronary artery disease." Experimental Gerontology 103 (March 2018): 87. http://dx.doi.org/10.1016/j.exger.2018.01.004.

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38

Angeli, Fabio, Enrica Angeli, Monica Trapasso, and Paolo Verdecchia. "Pentraxin-3 and the pathogenesis of preeclampsia." Hypertension Research 43, no. 9 (2020): 979–81. http://dx.doi.org/10.1038/s41440-020-0466-5.

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Menon, P. S. N. "Childhood Obesity, Metabolic Syndrome and Pentraxin-3." Indian Journal of Pediatrics 82, no. 1 (2014): 3–4. http://dx.doi.org/10.1007/s12098-014-1644-8.

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Iwagaitsu, Shiho, and Taio Naniwa. "Improvement of Arterial Wall Lesions in Parallel with Decrease of Plasma Pentraxin-3 Levels in a Patient with Refractory Takayasu Arteritis after Treatment with Tocilizumab." Case Reports in Rheumatology 2017 (2017): 1–5. http://dx.doi.org/10.1155/2017/4580967.

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A 19-year-old Japanese woman with active Takayasu arteritis despite multiple conventional immunosuppressive therapies with glucocorticoids in combination with intravenous cyclophosphamide, azathioprine, or infliximab with methotrexate and tacrolimus was successfully treated by switching from infliximab to intravenous tocilizumab. Worsening of claudication of the legs and elevated acute phase reactants, including plasma pentraxin-3 levels, were observed during combination therapy with infliximab. Computed tomography demonstrated increased wall thickening with contrast enhancement in the preexis

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Rauten, Anne Marie, Isabela Silosi, Stefan Ioan Stratul, et al. "Expression of Pentraxin 3 and Thrombospondin 1 in Gingival Crevicular Fluid during Wound Healing after Gingivectomy in Postorthodontic Patients." Journal of Immunology Research 2016 (2016): 1–7. http://dx.doi.org/10.1155/2016/4072543.

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Background. Wound healing is a tissue repair process after an injury, and two of its main components are inflammation and angiogenesis, in which course a cascade of mediators is involved. The aim of this research was to evaluate the involvement of Pentraxin 3 and Thrombospondin 1 in wound healing after periodontal surgery (gingivectomy) for gingival overgrowth during orthodontic treatment with or without magnification devices, by assessing their levels in GCF.Methods. From 19 patients with gingival overgrowth as a result of fixed orthodontic treatment, the overgrown gingiva was removed by ging

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Mahmudov, R. M., V. S. Mammadov, and L. R. Mirzakhanova. "Association of blood level of inflammatory mediators and morphology of coronary artery stenosis in patients with stable angina." Kazan medical journal 95, no. 3 (2014): 331–34. http://dx.doi.org/10.17816/kmj1507.

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Aim. To study the association of morphology of coronary artery disease and pentraxin-3, tumor necrosis factor α blood levels in patients with stable angina who undergo coronary artery bypass surgery. Methods. The study included 92 patients aged 44-73 years with stable angina of II-III functional class. The coronary artery disease type was classified by AHA/ACC criteria: type A - concentric stenosis with flat plague and plague length less than 10 mm, type B - eccentric stenosis or blunt-edged stenosis with moderate calcinosis or signs of mural trombosis, with length not exceeding 200 mm, type C

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Lee, Kyong-No, Kyo Hoon Park, Yu Mi Kim, Iseop Cho, and Tae Eun Kim. "Prediction of emergency cerclage outcomes in women with cervical insufficiency: The role of inflammatory, angiogenic, and extracellular matrix-related proteins in amniotic fluid." PLOS ONE 17, no. 5 (2022): e0268291. http://dx.doi.org/10.1371/journal.pone.0268291.

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Objective We aimed to determine whether various novel inflammatory, angiogenic, and extracellular matrix-related mediators in amniotic fluid (AF) can independently predict emergency cerclage outcomes in women with acute cervical insufficiency (CI). Methods This was a retrospective cohort study conducted among 50 singleton pregnant women (18–25 weeks) who underwent emergency cerclage for CI and were subjected to amniocentesis. The AF samples were assayed for endoglin, endostatin, haptoglobin, insulin-like growth factor-binding protein (IGFBP)-3, -4, kallistatin, lumican, macrophage colony-stimu

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Mairuhu, Albert T. A., Giuseppe Peri, Tatty E. Setiati, et al. "Elevated plasma levels of the long pentraxin, pentraxin 3, in severe dengue virus infections." Journal of Medical Virology 76, no. 4 (2005): 547–52. http://dx.doi.org/10.1002/jmv.20397.

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45

Inoue, Kenji, Tatsuhiko Kodama, and Hiroyuki Daida. "Pentraxin 3: A Novel Biomarker for Inflammatory Cardiovascular Disease." International Journal of Vascular Medicine 2012 (2012): 1–6. http://dx.doi.org/10.1155/2012/657025.

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Numerous studies have recently examined the role of pentraxin 3 (PTX3) in clinical situations. The pentraxin family includes C-reactive protein (CRP); however, unlike CRP, PTX3 is expressed predominantly in atherosclerotic lesions that involve macrophages, neutrophils, dendritic cells, or smooth muscle cells. Interestingly, PTX3 gene expression in human endothelial cells is suppressed to a greater extent by pitavastatin than the expression of 6,000 other human genes that have been examined, suggesting that PTX3 may be a novel biomarker for inflammatory cardiovascular disease. The expression an

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Tornyigah, Bernard, Samuel Odarkwei Blankson, Rafiou Adamou, et al. "Specific Combinations of Inflammatory, Angiogenesis and Vascular Integrity Biomarkers Are Associated with Clinical Severity, Coma and Mortality in Beninese Children with Plasmodium Falciparum Malaria." Diagnostics 12, no. 2 (2022): 524. http://dx.doi.org/10.3390/diagnostics12020524.

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Malaria-related deaths could be prevented if powerful diagnostic and reliable prognostic biomarkers were available to allow rapid prediction of the clinical severity allowing adequate treatment. Using quantitative ELISA, we assessed the plasma concentrations of Procalcitonin, Pentraxine-3, Ang-2, sTie-2, suPAR, sEPCR and sICAM-1 in a cohort of Beninese children with malaria to investigate their potential association with clinical manifestations of malaria. We found that all molecules showed higher levels in children with severe or cerebral malaria compared to those with uncomplicated malaria (

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Bilgin, Huseyin, Murat Haliloglu, Ali Yaman, et al. "Sequential Measurements of Pentraxin 3 Serum Levels in Patients with Ventilator-Associated Pneumonia: A Nested Case-Control Study." Canadian Journal of Infectious Diseases and Medical Microbiology 2018 (2018): 1–8. http://dx.doi.org/10.1155/2018/4074169.

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Purpose. The main purpose of this study was to investigate the dynamics of pentraxin 3 (PTX3) compared with procalcitonin (PCT) and C-reactive protein (CRP) in patients with suspicion of ventilator-associated pneumonia (VAP). Materials and Methods. We designed a nested case-control study. This study was performed in the Surgical Intensive Care Unit of a tertiary care academic university and teaching hospital. Ninety-one adults who were mechanically ventilated for >48 hours were enrolled in the study. VAP diagnosis was established among 28 patients following the 2005 ATS/IDSA guidelines. Res

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Górka-Dynysiewicz, Joanna, and Jolanta Zuwała-Jagiełło. "Pentraxin 3 and its role in tissue repair." Farmacja Polska 76, no. 2 (2020): 65–72. http://dx.doi.org/10.32383/farmpol/118530.

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Mantovani, A. "Pentraxin-3 in COPD: innocent bystander or amplifier?" European Respiratory Journal 39, no. 4 (2012): 795–96. http://dx.doi.org/10.1183/09031936.00198111.

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Imai, Naofumi, Shinichi Nishi, Kazuhiro Yoshita, et al. "Pentraxin-3 expression in acute renal allograft rejection." Clinical Transplantation 26 (June 29, 2012): 25–31. http://dx.doi.org/10.1111/j.1399-0012.2012.01641.x.

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